More than 15% of the population are literally useless.
This won’t end well.
Most of this hovering percentage (depending where you draw the line, with machine replacement) are male.
Society has no answer for this. There is no logical way to pander to them and save society from the thing sinking it aka them.
Einstein really hated white men, because the public introduction of nukes (muh publication and global display) has made it so we can’t cull our own useless eaters (the angry gamma types that hate authority, women and standards).
This is the death sentence of not cutting out cancer because it’s technically your own cells.
That’s what this has done. This is the start of dysgenics, nothing else.
I’m sure cancer cells would call for Muh Loyalty so the Titanic doesn’t patch its own hull and bail out water.
Breeding only comes a while after considerations of birth (i.e. the fact you must be born to be a parental candidate). Regression to the mean also pummels every population. This isn’t a one-time sweep.
“A grand total of two points. Don’t build your spaceships to escape the cooties just yet.”
And even there, look at the other half. Literally. Everything to the left of 1SD.
“When it comes to geniuses, these guys are trying to claim they’re Einstein by association.
That is retarded. Claim to be Hercules at least, he was male too. Aim high in your delusion.”
They hate authority because, being too stupid to be able to complete instructions, they assume the instructor hates them. They hate women because we’re sensitive to avoid any indicator of retarded offspring (ultimate waste of investment, more than being barren and caring for relatives). They hate standards because they know, deep down, in any society they’d be inferior and it would be out of reach. This makes them spiteful e.g. the anti-merit of barring women from colleges when they qualify. If you really think women are dumber, what do you care if you’d get in? Logically, they’d want weaker aka dumber people as their competition (why white SJWs want non-white SJWs in college). They think everyone cheats (college scandal) because they have to. Academia is now run by cheats (replication crisis) and its reputation under conservatives is no longer present.
This will lead to dystopia, which we’re already seeing moderately ripe fruits of.
They feel threatened by anyone because everyone is, statistically, an intellectual threat.
This explains chimp-outs at completely normal people and whatever the white version is, the mantrum?
The omegas are quiet and fine, the classic manual labourers. They mix the drinks, pick the food, clean the guns. Gammas have delusions of grandeur (supported by equalism) and when they fail, they’re the revenge fantasists. The low IQ/aggression link comes through there.
The SJWism started in the military when they forgot their purpose – cull the people society cannot feed.
This no below-X requirement is convenient for SJWs, who tend to be lower on the IQ scale themselves and oddly fearful of a draft that hasn’t happened since the last century.
The military have been pussies since world wars one and two, when, wouldn’t you know it, men started to go soft.
Yeah, because you practiced a dysgenic cull. In war, there’s always a cull. Introducing exemptions on age or fitness or IQ or mental malingering or having Daddy Moneybags will kill the country more surely than the cause.
Men went weak because of this. They over-compensate now with machines, all the gadgets in the world won’t help when your national IQ is declining. America needed Operation Paperclip or we’d all be speaking Russian now, because those non-white poor huddled masses from the previous 50 years had overbred.
Zero sum funding, the smart or the stupid. All the money to ‘rehabilitate’ criminals, to prison PS4s, to the translator in a primary school is money away from real art, real culture, real innovation, actual intelligence. It is costing you already.
Dumb men being arse-covered by greedy ones. There are far, far more male criminals, aren’t there?
In terms of a genetic product, that is a quality issue. The SJWs sense this but draw the wrong conclusions. We already have touchy-feely men, they’re in politics keeping the stupid mass of men going, for fun and profit.
You can’t sell junk food to kids living with both married parents eating home-cooked fare.
Football matches used to be a lot cheaper and had white, native players. Beer used to be cheaper and better. Clothes used to be non-plastic and retain a shape. You’ve lost everything your sex used to value. What happened to the trades? The technical colleges? The pubs?
The list goes on, classism feeds into it. Grow the poor with their feeblest like the reverse of making a thoroughbred, how they organise their own class breeding. You end up with fat Americans in a walmart blubbering along oohing and aahing in the playmobil mobile.
You were betrayed by your fellow man, women have got shit to do with that. We were often banned outright from those positions, it’s literally impossible. The main qualification was a dick, IQ didn’t matter to you, Mr Boys Club “have you met my shitbag of a son?“, so you kinda deserve this. If you wanna bring up YKW, well, Cromwell invited them back in. This blindspot shows how dumb the average white nationalist is.
None of you wanna bring in anti-nepotism law that requires the hire was of AVERAGE competence for the role.
or this shit happens
This saccharine naive libertarian “my fellow male would never betray me” – when competition and deceit is THE point of capitalism! THE point! The only one!
That’s the pleasant theoretical version, actual volume is worse.
After WW2 in particular, the remaining men (most undeserving cowards, frauds etc) were left a plethora of women to breed with. These military restrictions are r-selected and treasonous to national interests.
No coincidence their children were druggy hippies. Quelle surprise. The shittiest cultural movement in human history. To match the biggest dysgenic cull 20-30 years prior.
I posted this before as “where did all the good men go?” dead, mostly.
A gamma at this point would pipe up: why not the women?
Well, women didn’t evolve to fight, for one. It’s fairer to force 50yo Harold than 20yo Cindy Lou.
Ultimately, it must be this way around though, since men can’t butt heads like rams anymore to sexually compete or duel, the civilized equivalent (that gangs, sports, the gym seek to emulate, weakly).
Bringing back duelling to the death between two consenting parties (like boxing minus gloves) would have the same eugenic effect on a smaller scale. It’s a check and balance also backed by the death penalty for various heinous crimes, that also, wouldn’t you know it, was taken off the books by weaklings.
Rules in sport prevent death – but that is the purpose of sport. Today, only narcissists enter sport, knowing they will not die. Further, they get to bully and ‘fight’ aka play fight, like a child. There are fewer restraints on children fighting, think about that. We also mix the schools to blur gender, so weak boys can hit girls. That sounds like a society that can recover its civilization. Men also had all sorts of evolved responses in a fight, many male animals try to tear or bite one another’s nuts off. I can’t imagine many ‘well hard’ MMA bitches wanting to enter a ring if THAT were an option.
But that’s a real fight. There are no rules, except try not to die.
The modern Olympics is a joke, people went to watch the athletes compete, really compete, which means possibly die.
Women have fake tits, men have fake balls. That’s why modern women don’t respect gym bros. Even gym girls cheat on them. That’s why. Boys with sometimes implanted muscles or chemically fake muscles who’d shriek like a bitch if you ‘broke the rules’.
What rules? Real fighting has no sodding rules, nancy boy. Go back even to the 1950s and they’d all be considered flaming effeminate cuntmuffins. They’re all ponces in their little shorts with their waxed chest and homoerotic bragging. Why don’t women want kids anymore? Well… look at the options.
I asked a military bloke if he wanted to go running: “nah, it’s too cold.”
There was a light drizzle and it was about 12C. That counts as warm here.
I tried to take the piss and the excuse was ‘he couldn’t get sick’. Sure. Fucking sure.
A lot of these sports people are fake outdoorsy, they’re scared of rain and cold.
No coincidence they move from colder places to LA or Australia. Bloody wimps.
‘but what about the women?’ – gammas who imagine women have it easy because they’re really that dumb
The low IQ women don’t breed anyway, either because no man wants to marry it (in civilized places where marriage happens before spawn) or because their stupidity kills their kids with the wrong food or neglect (r-parenting, including abortion).
Peterson tells you half the truth, that’s the rest.
What do we do with the dipshits?
Good question. Don’t mention most of them are men, that demo might get triggered.
Past a certain point of genetic deterioration and cultural degeneration, even the lowliest woman will calculate men are too weak to even risk breeding with. They wait until after the next cull – if not war, famine, disease etc. like a capitalist retaining their productivity under socialism. EXACTLY like that, it’s the other sex’s method of preservation of energy. Look at the surveys predicting (women hoping) everything gets worse. For a ‘baby boom’ you need a war first to sweep out the dregs of men, that’s a rule of history and this is why.
Eliminate outgroup race (30-40% in some areas), from that remainder then eliminate other religions or no religion (90%) and then not mental (50% or so remaining probably on meds that hide their psychotic tendencies) and who is left?
That’s without looking into high IQ (105+) because the past two generations either aborted to afford a few more holidays or they never found marriage material dating back to the post-WW2 shit tier bullshit.
You killed the golden goose. Globalization is a horseman in better threads.
This kinda shit annoys me because it isn’t Gen X/Y/Z’s actual fault.
Credit: Master Brew
Kids cost too much! We’re already funding immigrant ones! Plus lazy retirees! Plus foreign aid! We can barely afford rent! Still, I see birth rate memes.
How’s about pro-marriage memes for men?
The controlled ops wouldn’t like that, would probably fill it with mixed couples or IVFs. Therein lies the problem. How many of you arseholes have actually sat down and read the Race Relations Act? US or UK? That’s when the SJWs won. When you cannot legally walk away. That document cements forced multiculturalism aka white genocide.
Below a certain level, I don’t think an adult can actually BE a parent, yet we don’t require an IQ test for a breeding license because feefees. Human rights are granted automatically at 18, even if their mental age is always 14.
Aw, but morons who believe in man cards don’t want to be objectively independently tested, we’re meant to just trust their word they’re superior yet oppressed! Bring back SATs, FFS, before it’s too late!
Especially broflakes feefees online, where they’re all magically 130IQ but can’t construct a sentence where I don’t wish to scratch my eyes out. Shoot teachers who help students cheat, I’m serious. Do you want a dense surgeon or a thicko pilot? If education is serious, cheating it is cheating society aka treason. Meritocracy requires you punish cheats severely.
A child can just be punished by omission aka permanent record, nobody will hire, booted out of school.
No “oh my Bobby’s a rebel” bullshit from weak patriarchs.
A purely “white is right” West has never existed because it would’ve been swarmed by its own idiots long before any success. There need to be strong discussions of what to do with the subhuman (neurologically, that’s what low IQ is, like a brain atavism from genetic load, their brain never completes adult development) in our own populations (certainly no incentives or celebrations!) and those we imported from the hillbilly nations of Europe e.g. Poland, with their child thieves and mother-whores bankrupting the NHS maternity wards at “fellow white’s” existential expense.
YOU have to live with this.
We have a dysgenic breeding pool, a eugenic economy, open borders, high crime and a huge debt state.
This won’t end pleasantly.
But fine – ignore me. Fine by me. No skin off my nose.
see section 31 They’ll blame children, independent thinkers and vaccine failure. It’s gene therapy, failure doesn’t happen. Population immunity is based on natural exposure and has never had anything to do with vaccines. Ever. In a century plus of biology. Over a year of exposure, we’re already immune naturally.
section 32 most of the Third Wave dead will be the fully vaccinated go ahead, check 70% of deaths, they say
deaths “larger than peak” from Jan 2021 warned in section 35
section 56 quote “vaccinated, but, nevertheless, not protected against death” that’s how stupid they are, stupid and malicious and under-50 deaths were minimal, under-60s a little higher, over-70s the only group at-risk, if you remember back to last year, which already feels like a century
They go on “This is NOT the result of vaccines being ineffective, merely uptake being so high”. That makes zero logical sense, either it protects you in reality or doesn’t. The map is not the territory, your model isn’t real.
If it doesn’t work, it doesn’t work. Then by definition, it’s ineffective. The purpose of uptake was prevention, which relies on effectiveness. They’re connected variables, idiot.
Look at the death charts on the right of page 18. The more vaccine, the more death.
I SEE SEAS OF BLUE, YOU CAN HAVE THEM TOO, A GENE THERAPY FOR ME, A GENE THERAPY FOR YOU, AND I THINK TO MYSELF, THIS IS BILL GATES’ WORLD, YES I THINK TO MYSELF, THIS IS BILL GATES’ WORLD…
section 60 is a ray of sunshine
“The widespread transmission of a variant that circumvents vaccine induced and naturally acquired immunity is a realistic possibility.” amusing of you to lump them in
“a large section of the population would be susceptible, whether they’ve been vaccinated or not”
“models here used assume….” a lot of shit, frankly, a lot of bullshit
The models used here assume that the effectiveness of vaccines remains high and they do not consider the impact of new variants of concern. < – neither has been proven, not even a little bit
“62 If a variant of concern were to reduce vaccine effectiveness against severe disease and death” Yes, death would be an unfortunate side effect….
63 “next generation of vaccines” forever and ever, medical martial law
“64 new vaccines will be required in the medium term” thin end of a wedge, this is “preparation for this REQUIREMENT…..”
66 waning immunity “optimistic assumption” no shit?
Appendix 2 lists, among other things, “vaccine reduction in risk of death” pretty sure it’s still 100% guys
A cheeky footnote reads “16 Very low, low and higher efficacy assumptions for Warwick are based on weighted averages of the two vaccines (30% PF, 70% AZ)” A third effective? Are you joking? And that was the average so the true range went lower. Note: always check the footnotes, they always hide shit in the footnotes. Nobody ever reads the footnotes, except for me. Because I’m often the cheeky little shit hiding things in them.
It’s strange how Asian areas of the UK are getting ‘new variants’ and ‘outbreaks’ when they have the largest numbers of doctors, who were among the earliest to get the gene therapy…..
Keywords: cytokine storm, live shedding, mortality rates per age group for nation.
If your employer is forcing you to obtain a medical treatment, have them sign paperwork making them financially responsible for all your medical treatments as a result of any and all damage from the experiment. They’ll change their tune. Medical segregation is also super illegal.
A debate has arisen regarding the validity of racial/ethnic categories for biomedical and genetic research. Some claim ‘no biological basis for race’ while others advocate a ‘race-neutral’ approach, using genetic clustering rather than self-identified ethnicity for human genetic categorization. We provide an epidemiologic perspective on the issue of human categorization in biomedical and genetic research that strongly supports the continued use of self-identified race and ethnicity.
“Instead, we looked at DNA from modern humans belonging to African populations and searched for unusual regions in the genome.” And they were older than modern. Imagine my shock. “What we do know is that the sequences of those forms, even the Neanderthals, are not that different from modern humans,” he said. -BECAUSE THEY THE SAME SPECIES. Sorry, broke into spontaneous ebonics because of the topic. “They have certain characteristics that make them different from modern DNA.” No shit?
Could be better or worse depending on the variant. They refuse to clone Neanderthals because no company could own them (human rights are based on demonstration of human IQ) and they’d be the smartest human on the planet. It would show us all up. I want it to happen. Neanderthals were more civilized than us, we learnt funerary rites from them. Please clone the African ancestors too, see what happens.
“Then we asked ourselves what does the general pattern of variation look like in the DNA that we sequenced in those African populations, and we started to look at regions that looked unusual,” Hammer said. “We discovered three different genetic regions fit the criteria for being archaic DNA still present in the genomes of sub–Saharan Africans. Interestingly, this signature was strongest in populations from central Africa.” I’ll fly high on this one and say no comment.
“We are talking about something that happened between 20,000 and 60,000 years ago – not that long ago in the scheme of things,” Hammer said. “If interbreeding occurs, it’s going to bring in a whole chromosome, and over time, recombination events will chop the chromosome down to smaller pieces. And those pieces will now be found as short, unusual fragments. By looking at how long they are we can get an estimate of how far back the interbreeding event happened.” Outbreeding depression…. it isn’t inter-breeding, it’s out-breeding…. “We think there were probably thousands of interbreeding events,” Hammer said. “It happened relatively extensively and regularly.”
He can’t be saying, they were literally fucking monkeys? No. …No? No. Are they technically humans they were screwing or not? At best, they mixed races. At worst, bestiality. I read a conspiracy that human anatomy is so weird because we’re the result of primates mating with hogs. That’s why pig hearts and such work on us. It was surprisingly detailed and I couldn’t think of anything to counter it.* I am also scared now.
I’m making the odd low/no effort post when I can be asked.
Damage to FSH and LH could make someone permanently infertile including women. Those are key for supporting a pregnancy. Infertility in women can be indicated. Children of Men is set in England….
The characteristic sloughing of the endometrium caused by fucking with hCG levels is called a decidual cast. It’s the reason some women (not all) with endometriosis can miscarry so often. They’ve weaponised it.
h/t Anonymous Conservative for gene therapy (convid) link, where I still comment sometimes to clarify, despite obstacles in my personal life preventing me from posting much here for personal safety.
“After reading hundreds of testimonies, the Instagram user learned many women experienced desidual casts, the lining of the uterus falling out. A quick Google search of the term showed a spike in search results in January 2021, when the Covid-19 vaccines were being introduced.”
Endometriosis is a regenerative medical issue which involves the appearance of endometrial tissue outside the uterine cavity, causing pelvic discomfort to be felt by the patient and in some cases, infertility. It has been discovered that the progesterone treatment used for endometriosis oftentimes leads to the formation of a decidual cast.
One of the most common contraceptives, forced on minors in the UK. They’re even telling virgins they ‘need’ it ‘just in case’, I had personal experience of shutting down those lies as a teen. I’m not taking shit with side effects and no longitudinal studies on fertility because my doctor assumes I’ll magically turn into a whore. That’s just insulting. Their only excuse after that was ‘period pains’ ….yes they were STILL trying to push it….. I was like…. I’ll stick to Ibuprofen, thanks? Progesterone causes PMT so if you’re wondering why so many modern women are bitchy….. it’s the progesterone only pill. I’ve had to drop friends after they started taking it. They became raging banshees. They suddenly hated men too. Pretty sure most SJWs are on it. Men on xenoestrogens exhibit PMT like symptoms, also those on steroids. So it isn’t just women being poisoned.
So HCG issues + progesterone = …….
Without endometriosis, it still occurs
The use of consolidated oral preventative pills may prompt the formation of decidual casts. There have been cases of dysfunctional uterine draining where the patient was prescribed a monophasic oral preventative, and an extensive decidual cast was discovered amid the treatment regimen. The same will happen in cases of treating oral pills for an adolescent menorrhagia. Medroxyprogesterone acetic acid derivation (DMPA) is an injectable type of prophylactic which has been demonstrated to cause the development and conveyance of decidual casts as well.
Things the men commenting on this wouldn’t know. Use it wisely.
1995 evidence of precedent for deceptive vector of transmission, official contamination reportage and hence, UN contravention of genocide law established in the 1940s, section (d) and (c):
“PIP: A priest, president of Human Life International (HLI) based in Maryland, has asked Congress to investigate reports of women in some developing countries unknowingly receiving a tetanus vaccine laced with the anti-fertility drug human chorionic gonadotropin (hCG). If it is true, he wants Congress to publicly condemn the mass vaccinations and to cut off funding to UN agencies and other involved organizations. The natural hormone hCG is needed to maintain pregnancy. The hormone would produce antibodies against hCG to prevent pregnancy. In the fall of 1994, the Pro Life Committee of Mexico was suspicious of the protocols for the tetanus toxoid campaign because they excludedallmalesandchildren and called for multipleinjections of the vaccine in onlywomenofreproductiveage. Yet, one injection provides protection for at least 10 years. The Committee had vials of the tetanus vaccine analyzed for hCG. It informed HLI about the tetanus toxoid vaccine. HLI then told its World Council members and HLI affiliates in more than 60 countries. Similar tetanus vaccines laced with hCG have been uncovered in the Philippines and in Nicaragua. In addition to the World Health Organization (WHO), other organizations involved in the development of an anti-fertility vaccine using hCG include the UNPopulationFund, the UN Development Programme, the World Bank, the PopulationCouncil, the RockefellerFoundation, the US National Institute of Child Health and Human Development, the All India Institute of Medical Sciences, and Uppsala, Helsinki, and Ohio State universities. The priest objects that, if indeed the purpose of the mass vaccinations is to prevent pregnancies, women are uninformed, unsuspecting, and unconsenting victims.”
“Vaccines are under development for the control of fertility in males and females. This review discusses developments in anti-fertility vaccines at the National Institute of Immunology, New Delhi, India. A single injection procedure for the sterilization or castration of male animals depending on the site at which the injection is given, has passed through fieldtesting and is expected to be on the market in the nearfuture. Vaccines inducing antibodiesagainstthehuman chorionic gonadotropin have gone through phase I trials with satisfactoryresults. A vaccine producing a consistentlybioeffectiveantibody response against gonadotropin-releasing hormone is ready for phase I/II clinical trials in patients of carcinoma of prostate after due experimentation in animals and toxicology studies. Research to identify sperm antigens for incorporation into second generation vaccines is in progress.”
Control, like farm animals. Single injection for castration of male animals possible.
Look forward to the “drop of testosterone levels”. At least you got to drink some bitch tits beer with the ‘boys’. Don’t come crying to me. You wanted to be ‘male animals’.
Evaluation of human papilloma virus in semen as a risk factor for low sperm quality and poor in vitro fertilization outcomes: a systematic review and meta-analysis
A review of the literature regarding ART outcomes showed an association between HPV infection and decreased PR, and an even stronger association between HPV infection and increased MR.
-increased miscarriage rate, lower odds of conceiving
Conclusion: Our meta-analysis shows a negative effect of HPV on sperm concentration, motility, and morphology. Further subgroup and categorical analysis confirmed the clinical significance of impaired sperm motility in HPV-infected sperm, although the sperm count and morphology must be carefully analyzed. The studies reviewed reported lower PR and increased MR in couples with HPV-infected sperm. As most studies had a moderate risk of bias, these observations warrant further large, well-designed studies before introducing clinical management recommendations.
Human papillomavirus infection and fertility alteration: a systematic review
Results: HPV infections are shown to be significantly associated to many adverse effects in the reproductive function. These adverse effects were reported in different levels from cells production to pregnancy and may be related to the infecting genotype.
Conclusions: It appears from this study that HPV detection and genotyping could be of great value in infertility diagnosis at least in idiopathic infertility cases. Like for the risk of carcinogenesis, another classification of HPV regarding the risk of fertility alteration may be considered after deep investigations.
Human Papilloma Virus (HPV) and Fertilization: A Mini Review
Sorry but if something makes you less virile, you’re less of a man.
Human papilloma virus (HPV) is one of the most prevalent viral sexually transmitted diseases. The ability of HPV to induce malignancy in the anogenital tract and stomato-pharyngeal cavity is well documented. Moreover, HPV infection may also affect reproductive health and fertility. Although, the impact of HPV on female fertility has not been thoroughly studied it has been found also to have an impact on semen parameters. Relative information can be obtained from studies investigating the relationship between HPV and pregnancy success. Furthermore, there is an ongoing debate whether HPV alters the efficacy of assisted reproductive technologies. An association between HPV and assisted reproductive technologies (ART) programs has been reported. Nevertheless, due to conflicting data and the small number of existing studies further research is required. It remains to be clarified whether HPV detection and genotyping could be included in the diagnostic procedures in couples undergoing in vitro fertilization (IVF)/intrauterine insemination (IUI) treatments. Vaccination of both genders against HPV can reduce the prevalence of HPV infection and eliminate its implications on human fertility. The aim of the present mini-review is to reiterate the association between HPV and human fertility through a systematic literature review.
The role of human papillomavirus on sperm function
I love how many yanks pull a Henry 8th and blame women for their own infertility, in this century.
Recent findings: HPVs are agents of the most common sexually transmitted disease and can lead to warts and cancers both in men and women. A high incidence of HPV infection has been demonstrated in sperm from sexually active men with and without risk factors for HPV and from infertile patients.
Semen infection is associated to an impairment of sperm parameters suggesting a possible role in male infertility. – really???
Interestingly, it has been demonstrated that when HPV is present in semen only a percentage of total cells are infected
-only? a? 100% is a percentage too…
and the virus can be localized in sperm or in exfoliated cells with different impact on sperm motility. Moreover, infected sperm are able to penetrate the oocyte, to deliver HPV genome in the oocyte and HPV genes can be actively transcribed by the fertilized oocyte.
-wouldn’t it be ironic if it made the kids or grandkids infertile instead? because they were conceived with it, a polluted germline
Recently an increased risk of pregnancy loss has been demonstrated in couples undergoing in-vitro fertilization and particularly when HPV DNA was present in semen samples of male partners.
– no blaming women this time, unless women haz sperm?
Summary: To date, no effective treatment, control strategy and prevention is provided for men despite the reported high incidence of HPV semen infection.
– no hurt their feefees? NAW
Because this infection in men is also a problem for partners, and because growing evidence suggests that semen infection may cause infertility and early miscarriage, more attention should be paid to male HPV infection. This study reviews the more recent literature about the role of HPV infection on sperm function and human reproduction.
– Manosphere fears this topic and all male degenerate accountability.
semen infection may cause infertility and early miscarriage
High-risk human papillomavirus in semen is associated with poor sperm progressive motility and a high sperm DNA fragmentation index in infertile men
Does the presence of human papillomavirus (HPV) in semen impact seminal parameters and sperm DNA quality in white European men seeking medical help for primary couple’s infertility?
>STD >DNA quality >in the germline of >white men
Never talk about it, I’m sure it’ll be fine.
HPV seminal infections involving high-risk (HR) genotypes are associated with impaired sperm progressive motility and sperm DNA fragmentation (SDF) values.
HPV is commonly present in semen samples.
No? F no it’s not. Stop sparing slutty blushes.
The overall rate of HPV positivity was 15.5%
so 1 in 7, uncommon at best. No normalizing pathology please.
And it varies majorly by race and sexuality. Not sex because it’s sexual, obviously.
Sperm progressive motility was significantly lower (P = 0.01) while SDF values were higher (P = 0.005) in HPV+ men compared to those with no HPV. In particular, HR HPV+ men had lower sperm progressive motility (P = 0.007) and higher SDF values (P = 0.003) than those with a negative HPV test. Univariable analysis showed that HR HPV+ was associated with impaired sperm progressive motility (P = 0.002) and SDF values (P = 0.003). In the multivariable analysis, age, FSH levels and testicular volume were significantly associated with impaired sperm progressive motility (all P ≤ 0.04). Conversely BMI, CCI, smoking habits and HPV status were not. Only age (P = 0.02) and FSH (P = 0.01) were significantly associated with SDF, after accounting for BMI, CCI, testicular volume, smoking habits and HPV status.
Impact of human papillomavirus infection in semen on sperm progressive motility in infertile men: a systematic review and meta-analysis
Background: Human papillomavirus (HPV) has been considered as one of the most common sexually transmitted viruses that may be linked to unexplained infertility in men. The possible mechanisms underlying correlation between HPV infection and infertility could be related to the altered sperm parameters. Current studies have investigated the effect of HPV seminal infection on sperm quality in infertile men, but have shown inconsistent results.
Methods: We systematically searched PubMed, Embase, Web of Science and CNKI for studies that examined the association between HPV seminal infection and sperm progressive motility. Data were pooled using a random-effects model. Outcomes were the sperm progressive motility rate. Results are expressed as standardised mean difference (SMD) with 95% confidence interval (CI). Heterogeneity was evaluated by the I-square (I2) statistic.
Results: Ten studies were identified, including 616 infertile patients with HPV seminal infection and 2029 infertile controls without HPV seminal infection. Our meta-analysis results indicated that sperm progressive motility was significantly reduced in HPV-infected semen samples compared with non-infected groups [SMD:-0.88, 95% CI:-1.17 ~ – 0.59]. There existed statistical heterogeneity (I2 value: 86%) and the subgroup analysis suggested that study region might be the causes of heterogeneity.
Conclusions: HPV semen infection could significantly reduce sperm progressive motility in infertile individuals. There were some limitations in the study such as the differences in age, sample sizes and the number of HPV genotypes detected. Further evidences are needed to better elucidate the relationship between HPV seminal infection and sperm quality.
The prevalence of Human Papilloma Virus (HPV) infection in the oligospermic and azoospermic men
The current study shows that HPV infection can affect on sperm count and motility and decrease count of sperm cell and decrease motility capability of these cells.
Among 50 confirmed oligospermic male, 15 were HPV DNA positive (30%).
In azoospemic group we had 8 HPV DNA positive (40%) and in normal group just 3 of 20(15%) samples were positive.
-what r the odds?
we found statistical significant relationship for sperm count (p<0.05) and sperm motility (slow) (p<0.05) in oligospermic group positive samples compared with negative. In the present study, 13 HPV genotypes were detected among positive samples. HPV genotypes 16, 45 in the high risk group and 6,11,42 in the low risk group were more frequent than the others.
Semen washing procedures do not eliminate human papilloma virus sperm infection in infertile patients
had HPV DNA on sperm and exfoliated cells. Sperm washing centrifugation showed no changes in the number of infected samples and in the percentage of infected cells. Ficoll and swim-up protocols induced a slight reduction in the number of infected samples (30 and 26, respectively).
no muh scientism and IVF cope
This study demonstrated that conventional sperm selection rarely eliminates HPV sperm infection. More attention should be paid to the reproductive health of infected patients because, not only can HPV be transmitted, but it may also have a negative effect on development of the fetus.
a negative effect on development of the fetus
so even if they all married a virgin waifu, they’d infect her and have defective babies comedy GOLD, 24K.
Is HPV the Novel Target in Male Idiopathic Infertility? A Systematic Review of the Literature
Infertility is an important health problem that affects up to 16% of couples worldwide.
1 in 7, where have I heard THAT before….? [scroll up]
Male infertility is responsible for about 50% of the cases,
–NAY, men are never responsible for their own in/fertility, have you been online recently?
and the various causes of male infertility may be classified in pre-testicular (for example hypothalamic diseases), testicular, and post-testicular (for example obstructive pathologies of seminal ducts) causes. Sexually transmitted infections (STI) are increasingly widely accepted by researchers and clinicians as etiological factors of male infertility. In particular, several recent reports have documented the presence of HPV in seminal fluid and observed that sperm infection can also be present in sexually active asymptomatic male and infertile patients.
In this review, we aimed to perform a systematic review of the whole body of literature exploring the impact of HPV infection in natural and assisted fertility outcomes, from both an experimental and a clinical point of view. Starting from in-vitro studies in animals up to in-vivo studies in humans, we aimed to study and evaluate the weight of this infection as a possible cause of idiopathic infertility in males with any known cause of conception failure.
Significant Correlation between High-Risk HPV DNA in Semen and Impairment of Sperm Quality in Infertile Men
guess the result
go on think
A total of 140 subjects participated in the current study. Among 70 confirmed infertile males, only 8 (11.43%) cases tested positive for high-risk HPV and all fertile men were HPV-negative. This data revealed a significant association between high-risk HPV and male infertility (P=0.03). The percentage of normal sperm morphology and sperm motility rate significantly declined in men infected with HPV (P<0.001).
and all fertile men were HPV-negative
oof and the sluts of both sexes are dying out, I am distraught. The genetics of the future are fairing brighter than you’d think.
Conclusion: There was a significantly higher prevalence of high-risk HPV in infertile men than fertile men. HPV infection seemed to be a risk factor for male infertility. Additional, larger studies should be conducted to confirm the impact of HPV on male infertility.
Player burnout shall henceforth be dubbed HPV-driven infertility?
Association between human papillomavirus infection and sperm quality: A systematic review and a meta-analysis
Human papillomavirus (HPV) has a high incidence rate in both males and females.
-maybe where you live
HPV infection in women has been shown to affect fertility and lead to foetal death and pregnancy loss. However, research on HPV infection in men is limited.
-well the husbands are freshly infecting the wives so–
-Ashley Madison wasn’t full of women stepping out, was it?
The aim of this study was to study the effect of HPV infection in semen on sperm quality and present the findings of previous studies through a meta-analysis. Databases including PubMed, MEDLINE, EMBASE, Web of Science, Cochrane Library, WanFang data and China National Knowledge Infrastructure were searched for relevant studies. A systematic review and meta-analysis were performed, and 17 studies were included for analyses based on a set criterion. Meta-analyses indicated that HPV infection in semen significantly reduced sperm concentration (SMD = -0.12, 95% CI: -0.21 to -0.03, p = .009), sperm motility (SMD = -0.55, 95% CI: -0.780 to -0.33, p = .000), sperm viability (SMD = -0.55, 95% CI: -0.780 to -0.33, p = .000) and sperm morphology (SMD = -0.34, 95% CI: -0.61 to -0.07, p = .015). The high-risk HPV (HrHPV) infection could significantly reduce sperm count (SMD = -0.65, 95% CI: -1.11 to -0.18, p = .007) compared with high-risk HPV (LrHPV) infection.
In conclusion, HPV infection in semen significantly reduced sperm quality, and the HrHPV infection could significantly reduce sperm count compared with LrHPV.
tick tock goes your biological clock, nobody can wait as long as they want NOBODY
Male sperm quality and risk of recurrent spontaneous abortion in Chinese couples: A systematic review and meta-analysis
Conclusions: The results of this analysis support an association of sperm density, sperm viability, sperm progressive motility rate, normal sperm morphology rate, sperm deformity rate, as well as sperm DFI with RSA.
IF you conceived, magically, it would kill your baby. REPEATEDLY.
Semen parameters and sperm morphology in men in unexplained recurrent spontaneous abortion, before and during a 3 year follow-up period
Baby death aborts the defective DNA, HPV fucks with your sperm’s DNA. Water is wet.
HPV makes you biologically unfit. According to the ultimate test, the womb.
To investigate the role of the ‘male factor’ in the pathogenesis of recurrent spontaneous abortion (RSA), especially sperm morphology abnormalities, 120 previously selected couples with unexplained RSA were studied for sperm parameters retrospectively and prospectively. The patients were subdivided into three subgroups, depending on their reproductive outcome during the 3 years of follow-up study: (i) 48 RSA couples who achieved a successful pregnancy; (ii) 39 RSA couples who experienced further abortions, and (iii) 33 RSA couples who experienced infertility during the follow-up period. A semen analysis was performed twice at the time of inclusion in this study, and twice again during the 3 year follow-up period. No significant differences in semen parameters were observed between RSA males and fertile controls. Instead, significant differences were observed between the group of RSA couples who experienced infertility during the follow-up and the other two groups (RSA couples who achieved successful pregnancy and RSA couples who experienced miscarriages and no live birth during the follow-up) for sperm concentration (P < 0.01 and P < 0.01 respectively), sperm motility (P < 0.01 and P < 0.01 respectively) and sperm morphology abnormalities (P < 0.01 and P < 0.01 respectively).
Sperm DNA fragmentation in couples with unexplained recurrent spontaneous abortions
The aim of the present study was to evaluate the degree of sperm DNA fragmentation in couples with idiopathic recurrent spontaneous abortion (RSA) and in those with no history of infertility or abortion. In this cohort study, 30 couples with RSA and 30 fertile couples as control group completed the demographic data questionnaires, and their semen samples were analysed according to World Health Organization (WHO) standards (September 2009-March 2010) for evaluation of sperm DNA fragmentation, using sperm chromatin dispersion (SCD) technique. The percentage of morphologically normal sperm was significantly lower in RSA patients compared with control group (51.50 ± 11.60 versus 58.00 ± 9.05, P = 0.019), but not in other parameters. Additionally, the level of abnormal DNA fragmentation in the RSA group was significantly higher than in the control group (43.3% versus 16.7%, P = 0.024). Our results indicated a negative correlation between the number of sperm with progressive motility and DNA fragmentation (r = -0.613; P < 0.001). The sperm from men with a history of RSA had a higher incidence of DNA fragmentation and poor motility than those of the control group, indicating a possible relationship between idiopathic RSA and DNA fragmentation.
Sperm chromatin integrity may predict future fertility for unexplained recurrent spontaneous abortion patients
“unexplained” – just assume the echo for comedic effect by now
The RSA group was further separated into three subgroups, depending on their reproductive outcome during the 12 months after they were enrolled in the study: the pregnancy subgroup consisted of 43 men whose partners achieved a successful pregnancy up to at least the 24th week of gestation; the abortion subgroup included 31 men whose partners experienced further abortions; and the infertile subgroup had 37 men whose partners did not have any positive pregnancy test after regular, unprotected intercourse. Significantly lower proportion of sperm with normal morphology was found in the abortion subgroup (14.7 ± 4.3%) than in the control group (17.5 ± 5.0%). Sperm concentrations were significantly lower in the infertile subgroup (55.7 ± 24.1%) than in the controls (68.6 ± 27.8%). The rates of abnormal sperm chromatin integrity were significantly higher in the abortion (16.7 ± 7.7%) and infertile (16.3 ± 6.6%) subgroups, compared to the control group (13.0 ± 4.4%). Logistic regression analysis showed that the subsequent reproductive outcome of the 111 RSA patients was negatively correlated to the rates of abnormal sperm chromatin integrity. In conclusion, sperm chromatin integrity, sperm morphology, and sperm concentration were associated with future reproductive outcome of RSA patients. The sperm chromatin integrity was a significant predictor for future abortion and infertility.
But men are never responsible for miscarriage, perish the THOUGHT.
Cytochemical evaluation of sperm chromatin and DNA integrity in couples with unexplained recurrent spontaneous abortions
unexplained….. sigh, ok.
Our study showed that in the cases of RSA, slow motility had a significant reduction in comparison with controls and also spermatozoa of men from RSA group had less chromatin condensation and poorer DNA integrity than spermatozoa that obtained from fertile men with no history of RSA.
Human sperm deoxyribonucleic acid fragmentation by specific types of papillomavirus
Conclusion: Human papillomavirus type 16 and 31 deoxyribonucleic acid caused deoxyribonucleic acid breakages characteristic of apoptotic but not necrotic sperm.
The data suggest that these human papillomavirus types may adversely affect subsequent embryonic development after fertilization. Sperm deoxyribonucleic acid appears to resist human papillomavirus types 18, 33, and 6/11 or repairing mechanisms occurred. Although enhanced motility was found in human papillomavirus–exposed sperm, important velocity parameters were decreased, suggesting impaired sperm function.
Negative Impact of Elevated DNA Fragmentation and Human Papillomavirus (HPV) Presence in Sperm on the Outcome of Intra-Uterine Insemination (IUI)
i.e. no, you won’t just get IVF
We wanted to determine the sperm DNA fragmentation index (DFI) cutoff for clinical pregnancies in women receiving intra-uterine insemination (IUI) with this sperm and to assess the contribution of Human Papillomavirus (HPV) infection on sperm DNA damage and its impact on clinical pregnancies. Prospective non-interventional multi-center study with 161 infertile couples going through 209 cycles of IUI in hospital fertility centers in Flanders, Belgium. Measurement of DFI and HPV DNA with type specific quantitative PCRs (HPV 6, 11, 16, 18, 31, 33, 35, 39, 45, 51, 52, 53, 56, 58, 59, 66 and 68) in sperm before its use in IUI. Clinical pregnancy (CP) rate was used as the outcome to analyze the impact on fertility outcome and to calculated the clinical cutoff value for DFI. A DFI criterion value of 26% was obtained by receiver operating characteristic (ROC) curve analysis. Couples with a male DFI > 26% had significantly less CPs than couples with DFI below 26% (OR 0.0326; 95% CI 0.0019 to 0.5400; p = 0.017). In sperm, HPV prevalence was 14.8%/IUI cycle. Sperm samples containing HPV had a significantly higher DFI compared to HPV negative sperm samples (29.8% vs. 20.9%; p = 0.011). When HPV-virions were present in sperm, no clinical pregnancies were observed. More than 1 in 5 of samples with normal semen parameters (17/78; 21.8%) had an elevated DFI or was HPV positive. Sperm DFI is a robust predictor of clinical pregnancies in women receiving IUI with this sperm. When DFI exceeds 26%, clinical pregnancies are less likely and in vitro fertilization techniques should be considered
When HPV-virions were present in sperm, no clinical pregnancies were observed.
Sperm viral infection and male infertility: focus on HBV, HCV, HIV, HPV, HSV, HCMV, and AAV
Chronic viral infections can infect sperm and are considered a risk factor in male infertility. Recent studies have shown that the presence of HIV, HBV or HCV in semen impairs sperm parameters, DNA integrity, and in particular reduces forward motility. In contrast, very little is known about semen infection with human papillomaviruses (HPV), herpesviruses (HSV), cytomegalovirus (HCMV), and adeno-associated virus (AAV). At present, EU directives for the viral screening of couples undergoing assisted reproduction techniques require only the evaluation of HIV, HBV, and HCV.
-all trust the EU guys
However, growing evidence suggests that HPV, HSV, and HCMV might play a major role in male infertility and it has been demonstrated that HPV semen infection has a negative influence on sperm parameters, fertilization, and the abortion rate.
-somebody else look up herpes, I’m lazy
Besides the risk of horizontal or vertical transmission, the negative impact of any viral sperm infection on male reproductive function seems to be dramatic.
-Really, f-ing fascinating!
In addition, treatment with antiviral and antiretroviral therapies may further affect sperm parameters. In this review we attempted to focus on the interactions between defined sperm viral infections and their association with male fertility disorders. All viruses considered in this article have a potentially negative effect on male reproductive function and dangerous infections can be transmitted to partners and newborns. In light of this evidence, we suggest performing targeted sperm washing procedures for each sperm infection and to strongly consider screening male patients seeking fertility for HPV, HSV, and HCMV, both to avoid viral transmission and to improve assisted or even spontaneous fertility outcome
HPV infection in semen: results from a new molecular approach
Let’s get molecular.
Human papillomavirus (HPV) is the agent of the most common sexually transmitted diseases causing a variety of clinical manifestations ranging from warts to cancer. Oncogenic HPV infection is the major cause of cervical cancer and less frequently of penile cancers. Its presence in semen is widely known, but the effects on fertility are still controversial. – how? allergic to facts?
We developed a new approach to evaluate virus localisation in the different semen components. We analysed also the specific genotype localisation and viral DNA quantity by qPCR. Results show that HPV DNA can be identified in every fraction of semen: spermatozoa, somatic cells and seminal plasma. Different samples can contain the HPV DNA in different fractions and several HPV genotypes can be found in the same fraction. Additionally, different fractions may contain multiple HPV genotypes in different relative quantity. We analysed the wholeness of HPV DNA in sperm cells by qPCR. In one sample more than half of viral genomes were defective, suggesting a possible recombination event. The new method allows to easily distinguish different sperm infections and to observe the possible effects on semen. The data support the proposed role of HPV in decreased fertility and prompt new possible consequences of the infection in semen.
>HPV DNA can be identified in every fraction of semen: spermatozoa, somatic cells and seminal plasma
If you’re wondering why your nation is infertile, look in the mirror. Mutant sperm.
Women’s Preferences for Penis Size: A New Research Method Using Selection among 3D Models
Women’s preferences for penis size may affect men’s comfort with their own bodies and may have implications for sexual health. Studies of women’s penis size preferences typically have relied on their abstract ratings or selecting amongst 2D, flaccid images. This study used haptic stimuli to allow assessment of women’s size recall accuracy for the first time, as well as examine their preferences for erect penis sizes in different relationship contexts. Women (N = 75) selected amongst 33, 3D models. Women recalled model size accurately using this method, although they made more errors with respect to penis length than circumference. Women preferred a penis of slightly larger circumference and length for one-time (length = 6.4 inches/16.3 cm, circumference = 5.0 inches/12.7 cm) versus long-term (length = 6.3 inches/16.0 cm, circumference = 4.8 inches/12.2 cm) sexual partners. These first estimates of erect penis size preferences using 3D models suggest women accurately recall size and prefer penises only slightly larger than average.
but different races have sexually selected different averages, even by nation? So the mean IS the ideal.
The association between penis size and sexual health among men who have sex with men
Larger penis size has been equated with a symbol of power, stamina, masculinity, and social status. Yet, there has been little research among men who have sex with men assessing the association between penis size and social-sexual health. Survey data from a diverse sample of 1,065 men who have sex with men were used to explore the association between perceived penis size and a variety of psychosocial outcomes. Seven percent of men felt their penis was “below average,” 53.9% “average,” and 35.5% “above average.” Penis size was positively related to satisfaction with size and inversely related to lying about penis size (all ps < .01). Size was unrelated to condom use, frequency of sex partners, HIV status, or recent diagnoses of HBV, HCV, gonorrhea/Chlamydia/urinary tract infections, and syphilis. Men with above average penises were more likely to report HPV and HSV-2 (Fisher’s exact p <or= .05). Men with below average penises were significantly more likely to identify as “bottoms” (anal receptive) and men with above average penises were significantly more likely to identify as “tops” (anal insertive). Finally, men with below average penises fared significantly worse than other men on three measures of psychosocial adjustment. Though most men felt their penis size was average, many fell outside this “norm.” The disproportionate number of viral skin-to-skin STIs (HSV-2 and HPV) suggest size may play a role in condom slippage/breakage. Further, size played a significant role in sexual positioning and psychosocial adjustment. These data highlight the need to better understand the real individual-level consequences of living in a penis-centered society.
so men with big wangs, at least the ‘gay’ ones, are rendering themselves infertile or birth defect risks by sharing it around. Y NO str8 studies?
Female Orgasm and Overall Sexual Function and Habits: A Descriptive Study of a Cohort of U.S. Women
Results: FSFI scores, which were calculated for the 230 women who reported having had a steady male sex partner in the preceding 6 months, showed that 41% of the 230 women were at risk for female sexual dysfunction (a cutoff less than 26.55) and 21% were dissatisfied with their overall sexual life. Almost 90% of the overall cohort reported good emotional contact with their partner, that their partner was willing to have sex, satisfaction with the partner’s penis size (wherever applicable), and good erectile function and ejaculatory control of their partner (wherever applicable). 81% of the overall cohort claimed to be sexually active. Around 70% (70-72) did reach orgasm frequently, but around 10% never did so. Vaginal intercourse was reported by 62% of the overall cohort as the best trigger of orgasm, followed by external stimulation from the partner (48%) or themselves (37%). External stimulation was reported to be the fastest trigger to orgasm.
Clinical implications: The knowledge on how women reach orgasm and how it is related to the partners’ willingness to have sex and other factors can be incorporated in the clinical work.
Strengths & limitations: The use of a validated questionnaire and the relative large number of participants are strengths of the study. Limitations are the cross-sectional design, the lack of a sexual distress measure, and a possible selection bias.
Conclusion: Most women in the overall cohort were satisfied overall with their sexual life and partner-related factors, even though 41% (of those who cited a steady sex male partner) were at risk for female sexual dysfunction. Most women did reach orgasm through different kinds of stimulation. Correlation was good between preferred and performed sexual activities and positions. Shaeer O, Skakke D, Giraldi A, et al. Female Orgasm and Overall Sexual Function and Habits: A Descriptive Study of a Cohort of U.S. Women. J Sex Med 2020;17:1133-1143.
I bet the inorgasmic had ‘cut’ men, who produce fewer orgasms in all women in studies.
Impact of penile size on male sexual function and role of penile augmentation surgery
Penile augmentation is an ongoing debate. Emerging evidence proves a relationship between dissatisfaction with penile size and sexual dysfunction. Despite a widespread belief of the value of penile size, and the prevalent complaint of a “small penis,” penile augmentation still stands short of addressing the demand. This report highlights the studies added to medical literature on penile augmentation in 2011-2012, including data on normal penile dimensions and the expected dimensions for a normal person, determination of the prevalence of the dissatisfaction with penile size, its effect on erectile function, female partner’s opinion of the value of penile size, as well as further experience with augmentation techniques.
It’s mental, they need therapy. Also, I see the joke.
Why Are Men Satisfied or Dissatisfied with Penile Implants? A Mixed Method Study on Satisfaction with Penile Prosthesis Implantation
The issue is mental?
Content analysis revealed four main themes for men’s satisfaction with the PPI: (i) psychological factors were reported 54 times (n = 54) and included positive emotions, self-esteem, confidence, enhancement of male identity, major live change, and self-image;
(ii) improvement of sexual function was reported 54 times (n = 54) and referred to achievement of vaginal penetration, increase of sexual desire, sexual satisfaction, penis size, and improvement of erectile function;
– satisfaction is a female metric? ED is mental?
(iii) relationship factors were reported 11 times (n = 11) and referred to relationship improvement and the possibility of giving pleasure to the partner; and (iv) improvement in urinary function (n = 3).
– not possible with implants, all surgeries produce numbness and see above study
A preliminary investigation of a novel method to manipulate penis length to measure female sexual satisfaction: a single case experimental design
Results: On average reducing the depth of penetration made a statistically significant 18% reduction of overall sexual pleasure with an average 15% reduction in length of the penis. The longer the erect penis, the less likely the rings had an impact on sexual pleasure. There was however a range of individual responses with a minority of women reporting that reducing the depth of penetration was more pleasurable on some occasions.
– the G spot?
Conclusions: Size may matter in women in a healthy stable relationship when there is penile shortening. Because of the small number of couples and men with an apparently long penises, our results are preliminary, and we welcome replication in a larger sample with a more diverse range of penile lengths. Our results should not be misinterpreted as meaning that increasing penile length will increase sexual pleasure in women.
shortening with age, and no, supplements don’t help
Non-pharmacological and non-surgical strategies to promote sexual recovery for men with erectile dysfunction
-quit the porn? sigh
the tobacco-lung cancer connection of this generation history will know you as a bunch of wankers
Erectile dysfunction (ED), the most commonly reported sexual problem for men,
in dick-mutilated, porn-addled America? also Israel?
reduces the quality of life for both patients and their partners. Even when physiologically effective, long-term adherence to ED treatments is poor.
– it’s behavioral…
We review here the implication of having patients’ partners involved in ED treatment, starting with treatment selection. We suggest that having partners engaged from the outset may promote an erotic association of the treatment with the partner, i.e., conceptually linking the aid to the sexual pleasure that the partner provides.
– quit ‘supernormal stimuli’ aka lechery, your dopamine is fried?
We hypothesize that this erotic association should enhance the sexual aid’s effectiveness and might potentially help improve long-term adherence. The primary focus of this review, though, is non-pharmacological and non-surgical options for maintaining sexual activity for men with ED. Though not ED treatments per se, anecdotal data suggest that these options may be effective for some patients and their partners in regaining a satisfying sex life. The aids discussed include external penile prostheses, penile sleeves, and penile support devices.
-easier just to give up the sexual visual crack, surely?
These devices can allow men to participate in penetrative sexual intercourse despite moderate to severe ED.
-aging happens, it’s natural selection
External penile prostheses can be personalized so they match in size and shape a man’s normal full erection.
– that is sad
Penile sleeves can similarly be customized with a lumen that fits best a patient’s penis for optimal tactile stimulation.
– so they’re fucking a toy? …. not the man…..
We review how multi-sensory integration can enhance sexual arousal for men who use such devices, allowing them to achieve orgasm despite intractable ED.
-it’s a silicon sock
Patients are not always advised within ED clinics about these options nor why and how they can facilitate non-erection dependent sexual recovery. Clinicians need to be aware of these devices and their positive attributes, so they can objectively counsel and encourage couples to explore their use as an alternative to more invasive treatments. The most commonly promoted non-medical ED aid offered to patients is the vacuum erection device. We discuss how erections achieved with the vacuum erection device have a “hinge effect”, that is an underappreciated barrier to the effectiveness of the erection. With a hinged erection, the penis points downward rather than upward. We show how the normal kinematics of the penis during coitus is not strictly linear (i.e., not uniaxial; not just in-and-out), and is impeded by hinging. Positional adjustment, such as the receptive partner being on top, may help overcome this problem for some couples. Lastly, we suggest that, in the case where ED can be anticipated from a pending medical treatment, such as a prostatectomy, pre-habilitative approaches may potentially improve adherence to sexual aid use in the long-term. In conclusion, non-pharmacological and non-surgical options for sexual recovery are available. Scientific studies on the effectiveness of these interventions in restoring satisfying sex are warranted.
Is Internet Pornography Causing Sexual Dysfunctions? A Review with Clinical Reports
Traditional factors that once explained men’s sexual difficulties appear insufficient to account for the sharp rise in erectile dysfunction, delayed ejaculation, decreased sexual satisfaction, and diminished libido during partnered sex in men under 40.
This review (1) considers data from multiple domains, e.g., clinical, biological (addiction/urology), psychological (sexual conditioning), sociological; and (2) presents a series of clinical reports, all with the aim of proposing a possible direction for future research of this phenomenon. Alterations to the brain’s motivational system are explored as a possible etiology underlying pornography-related sexual dysfunctions. This review also considers evidence that Internet pornography’s unique properties (limitless novelty, potential for easy escalation to more extreme material, video format, etc.) may be potent enough to condition sexual arousal to aspects of Internet pornography use that do not readily transition to real-life partners, such that sex with desired partners may not register as meeting expectations and arousal declines. Clinical reports suggest that terminating Internet pornography use is sometimes sufficient to reverse negative effects, underscoring the need for extensive investigation using methodologies that have subjects remove the variable of Internet pornography use. In the interim, a simple diagnostic protocol for assessing patients with porn-induced sexual dysfunction is put forth.
– it’s a supernormal stimulus designed to make you impotent, it’s a delusion that alters the brain
Based on the findings of historical studies cited earlier, older men would be expected to have far higher ED rates than the negligible rates of younger men [2,7]. However, in just a decade, things changed radically. The 2001–2002 rates for older men 40–80 were about 13% in Europe . By 2011, ED rates in young Europeans, 18–40, ranged from 14%–28% .
duh, porn addicts also look older, do that study
Finally, another 2015 study on men (mean age approximately 36), reported that ED accompanied by a low desire for partnered sex is now a common observation in clinical practice among men seeking help for their excessive sexual behavior, who frequently “use pornography and masturbate” .
aimed at Europeans, squarely
make them hypersexual, extreme r, and re-productively useless
The sole qualification for being a masculine man is having a functioning dick but they cuck for the industry ruining their lives.
The Potential Associations of Pornography Use with Sexual Dysfunctions: An Integrative Literature Review of Observational Studies
This paper reviews the associations between pornography use and sexual dysfunction based on evidence from observational studies. The existing data in this regard mostly derive from cross-sectional investigations and case reports. There is little if no evidence that pornography use may induce delayed ejaculation and erectile dysfunction, although longitudinal studies that control for confounding variables are required for a full assessment.
– decades later, we’ll be dead demographically…
The associations between pornography use and sexual desire may differ between women and men although the existing data is contradictory and causal relationships cannot be established. The strongest evidence is available for the relation of pornography use with decreased sexual satisfaction, although the results of prospective studies are inconsistent. The paper outlines future research prospects beneficial in understanding the nature of associations between pornography use and sexual dysfunctions in men and women.
Online Porn Addiction: What We Know and What We Don’t—A Systematic Review
In the last few years, there has been a wave of articles related to behavioral addictions; some of them have a focus on online pornography addiction. However, despite all efforts, we are still unable to profile when engaging in this behavior becomes pathological.
– publishing bias
Common problems include: sample bias, the search for diagnostic instrumentals, opposing approximations to the matter, and the fact that this entity may be encompassed inside a greater pathology (i.e., sex addiction) that may present itself with very diverse symptomatology.
= excuses so you don’t get later sued
Behavioral addictions form a largely unexplored field of study, and usually exhibit a problematic consumption model: loss of control, impairment, and risky use. Hypersexual disorder fits this model and may be composed of several sexual behaviors, like problematic use of online pornography (POPU). Online pornography use is on the rise, with a potential for addiction considering the “triple A” influence (accessibility, affordability, anonymity). This problematic use might have adverse effects in sexual development and sexual functioning, especially among the young population. We aim to gather existing knowledge on problematic online pornography use as a pathological entity. Here we try to summarize what we know about this entity and outline some areas worthy of further research.
Risky sexual behaviors typically occur when a person is sexually motivated by potent, sexual reward cues. Yet, individual differences in sensitivity to sexual cues have not been examined with respect to sexual risk behaviors. A greater responsiveness to sexual cues might provide greater motivation for a person to act sexually; a lower responsiveness to sexual cues might lead a person to seek more intense, novel, possibly risky, sexual acts. In this study, event-related potentials were recorded in 64 men and women while they viewed a series of emotional, including explicit sexual, photographs. The motivational salience of the sexual cues was varied by including more and less explicit sexual images. Indeed, the more explicit sexual stimuli resulted in enhanced late positive potentials (LPP) relative to the less explicit sexual images. Participants with fewer sexual intercourse partners in the last year had reduced LPP amplitude to the less explicit sexual images than the more explicit sexual images, whereas participants with more partners responded similarly to the more and less explicit sexual images. This pattern of results is consistent with a greater responsivity model. Those who engage in more sexual behaviors consistent with risk are also more responsive to less explicit sexual cues.
– it’s r/K
trans. r-types seem to have dulled sexual perception (abnormal) so over-estimate opportunity in compensation
Studies are in their description, duplicated below.
Reminder: ED is the PC term for impotence – and a common side effect of porn addiction. How about studying penis size between promiscuous men and chaste ones? They’d never publish it. I’d read it. Why are we not funding this?
The role of exposure to phthalates in variations of anogenital distance: A systematic review and meta-analysis
Environmental chemicals such as phthalate esters may have adverse effects on anogenital distance (AGD), but the evidence in both genders has not been reviewed systematically. The objective of the present study is to conduct a systematic review and meta-analysis of studies that analyzed the relationship between exposure to phthalates and AGD. English papers published up to March 2018 were searched in PubMed, Scopus, Clarivate-Web of Science, and Google scholar. We applied fixed-effects models to calculate pooled beta coefficient [β]. In the case of heterogeneity, random-effects models were used. Using the comprehensive search strategies, 313 papers were identified and after screening, 10 of them were included in this study. In primary analyses, we found that exposure to phthalates was not associated with short AGD (β = -0.11; 95% CI, -0.27, 0.06; I2 = 0%). However, results of subgroup analyses indicated that in boys, the sum of di-2-ethylhexyl phthalate (∑DEHP) metabolites had significant association with the risk of shortened anopenile distance (AGDAP) (β = -0.915, 95% CI: 1.629, -0.2) and anoscrotal distance (AGDAS) (β = -0.857, 95% CI: 1.455, -0.26). In addition, urinary monobutyl phthalate (MBP), monoethyl phthalate (MEP), and monoisobutyl phthalate (MiBP) were associated with short AGDAP. We also observed significant association between monobenzylphthalate (MBzP) and anofourchette distance (AGDAF) in girls. Our study provided findings on significant association of exposure to ∑DEHP metabolites, MBP, MEP, and MiBP with shortened AGDAP in boys. The mechanisms of phthalates effect on AGD may involve receptors and enzymes involved in steroidgenesis, negative influence on Leydig cells, cell proliferation, gonocyte cell numbers, and testosterone production.
I have studies and other things I could post, but haven’t yet. It’s a red herring, presently. I see a raft of new comments. I’m not checking or approving them, I miss many important ones anyway (somebody deletes them). If people won’t listen for years, talking now is quite futile.
I’ve mentally checked out of their endgames long ago, truth be told, nobody knows how this story ends. I used to care, I remember the impulse to interfere well. The do-gooder instinct, now I abhor it with equal fervor. It struck me how ill-advised this impulse is, when dealing with certain types of ruthless, dangerous people. It was pointed out to me that I was too soft and it could, could be the death of me. It took years but… I corrected this deficit. That’s why I’m still here, unlike many before me, and doing as I please. QED. I’m self-righteous, not stupid. Many rigid principled people end up dead and I’d rather not be among them. There is no honour dying nameless in a gutter.
I’m not important anyway. I think people know if they’re important. I’m not. I’m not your messiah or your prophet, I put puzzle pieces together quite well, like everyone else. If I knew too much I’d be six foot under, frankly, because I’m not related to enough VIPs. I think of myself as probable back-up flesh, back-up organs to some …. well… persons. Let’s leave it at that. Rare blood type.
I’m much more useful to the world alive and talking, in my own way. I do know key things, sometimes. There is no binary choice to be a good person or not, I was wrong. Too many Disney films, maybe. Idealism is a …problem. There are ways to be good and some are strategically wiser than others. Game theory. Try to help All and die, vainly, or try to help some and live, well. Simple when laid out. To suffer needlessly is a vanity.
The cardinal rule is never be the most ignorant party, keep your ear to the ground and listen to any whispers, however slight. Better to have subtle forewarning than none at all. If you don’t know, anyone can take advantage of that ignorance, however pure your intent. As a mentor told me years ago “You’re good, but you’re no good to anyone dead”. It shocked me. It was necessary. I am a believer in choice and liberty, which is to say agency. If the People want Hell on earth, let them have it with both barrels, I say. If they walk to the slaughter willingly, let them die. God’s mind is the same, let them take the Mark, if they so, loudly, wish. Millennia of warning foretold. A suicidal populace is already done for. Mouse utopia narcissism, at the cost of fertility. It takes years to show. Is society so far gone? Is the average mind a reprobate, fully deserving of their longsuffering? I haven’t the foggiest. I hope not. I have faith in God, if not men. Do not put faith in men.
We live in desperate times, full of desperate minds. Too much noise. What’s Sodom got on present America? Really. List the sins. I tried. Hell isn’t necessary, deep down evil people know what they deserve and seek out oblivion in their self-destruction. Let them be. Sometimes natural selection is self-selecting. If you conform to evil, I have seen evil conform to thee. Jesus himself wouldn’t spare the unrepenting. I don’t know better, that’s for sure. Do I know more? Nah. I opt for humility over the popular, worldly conceit. I don’t need people to agree with me, they’ll see.
I’m not cutting off my nose, hoping to spit another’s face. Reality bites eventually.