In the present Convention, genocide means any of the following acts committed with intent to destroy, in whole or in part, a national, ethnical, racial or religious group, as such:
- Killing members of the group;
- Causing serious bodily or mental harm to members of the group;
- Deliberately inflicting on the group conditions of life calculated to bring about its physical destruction in whole or in part;
- Imposing measures intended to prevent births within the group;
- Forcibly transferring children of the group to another group.
They know their own, in all cases of adoption, suspect pedo.
now Democratic activists are raising alarm about U.S. District Court Judge Amy Coney Barrett’s adoption of two children from Haiti.
Nowhere in the Bible does it support adoption of non-genetic children, it’s legalised child-snatching. The Bible says go forth and multiply if you want more kids. The CHINOs are an embarrassment.
Adoption of non-kin children is Satanic. It destroys the child’s legal right to their own heritage, culture and family. Celebrities could sponsor the child at home with the extended family but it’s all about pride. Looking at the child outcomes, like IQ and personality and such are even inherited from the real genetic parents. If you’re not blood, you are not their parent. Stop virtue signalling, children aren’t objects to be passed around like Pokemon cards to whoever has the most money.
The proportion of adopted kindergartners being raised by a mother of a different race or ethnic group rose by 50% between 1999 and 2011. The proportion of adoptees with Asian backgrounds nearly tripled over the same time period. Paradoxically, the fraction of adopted students who are African-American seems to have fallen. What has not changed is that a large majority of adoptive parents are white, older, well-educated, and relatively affluent.
I don’t think abuse of kids is justified if they’re another race, either. We must hold Christians to the correct standards. The Biblical one of if you want kids, make them.
It’s imperialistic. They treat the kid like a handbag, it’s sick.
How dare they call this Christian?
Their parents are generally well-educated and affluent. They receive more time and educational resources from those parents than the average child gets from theirs. Yet they get into more conflicts with their classmates at school, display relative little interest and enthusiasm about learning tasks, and register only middling academic performance. About whom are we talking? Adopted children. This is the paradox of adoption in America.
This is the first study of adopted children’s school behavior that is based on independent teacher reports and makes use of a representative national sample of students from adoptive families.
Yet my analysis shows that adoptees do not do as well in school as one would expect from their highly advantaged home environments. The results call into question the widely held assumption that larger investments of money and time in children can overcome the effects of early stress and deprivation and genetic risk factors.
Bad blood will out.
And the model minority thing is also propaganda, look at adolescent drinking/drug use/sexual promiscuity studies. There is no model minority, it’s just propaganda by the Boomers shaming the non-white kid into behaving. As you can see, when they’re not rigging the data by self-report, it doesn’t actually work.
Jayman used to blog about the non-existent parenting effect, even when they’re biologically yours.
At best they claim 2-5 IQ point different with within-race adoption white to white, which isn’t significant. It’s never the upper number.
Our analysis showed that, among the biological parents, each additional unit on the parental education scale was associated with 2.7 IQ points in the child, whereas among the adoptive parents, each additional unit of education was associated with 1.7 IQ points.
Can we stop coddling their ego please? I don’t care about adult feefees and ego over any child.
The residual difference between the IQs of the two groups of children was reduced from 4.4 to 3.4 when the difference between the biological and rearing parents’ education was included in the model.
Shared environment accounts for 0% of life outcome.
The high early shared environment influence shows that in youth, environmental factors can make a difference. These influences diminish and disappear with time, dashing hopes of lasting parental influence. Some voices – including preeminent behavioral geneticist Robert Plomin himself – often try to claim that the increasing heritability of IQ and other behavioral traits can be boiled down to “gene-environment correlations” (rGE). The idea being that people seek out environments to suit their genetic proclivities (which they do), and the influence of that environment leads to the final trait. This is a nice rosy idea, because it appears to leave the door open to environmental manipulation, if we could intervene in the “proper” ways. However, it is fantasy. We clearly saw in my earlier post that the “gene-environment co-variance” was often negative! One’s environment seemed to be “making” one the opposite of what one would expect. Our experiences don’t shape our political attitudes like we think they do. So is the case with IQ.
Indeed, a meta-analysis of longitudinal twin and adoption studies attempted to test this idea. It sought to determine whether the increasing heritability of IQ could be explained by on-going environmental influence or genetic “amplification”; that is, the compounding of genetic effects over time. This is likely because the effect of each additional gene becomes more and more relevant as children grow up. Indeed, amplification is what they found:
Proponents of the efficacy of nurture – especially parenting – often repeat a few erroneous arguments. Here I will address them. One of them is the idea that parenting, while ineffective for most, may make a difference for individuals with certain temperaments. For example, perhaps the low IQ/shiftless/delinquent/criminal or otherwise poorly dispositioned might benefit from more authoritative parenting, say. It’s a nice idea to think about, but it doesn’t happen. This is essentially “Stolen Generations” wisdom. As we’ve seen in my earlier post, a massive review of twin and adoption studies found no significant shared environment effect on criminality in adults (well, modeling found a shared environment contribution of 0.09, which can generally regard to be non-significant given the enormous measurement error expected). Even an effect that operated on some children but not others would contribute to the overall average shared environment, which was negligible.
Edit, 6/5/14: [I wanted to expand on the above mentioned review of criminality (by Rhee & Waldman, R&W), particularly the appearance of a small but nonzero (though non-significant) shared environment finding. As we saw, the age the subjects are assessed seems to make a difference. As well, as discussed in my analysis on adolescent psychopathology below, the particular measure used – such who is doing the ratings – affect the values found. For example, self-ratings or ratings by parents tend to attenuate the heritability estimate, and both appear to inflate the shared environment estimate, at least in youth. The Rhee & Waldman meta-analysis is no exception. Here are the ADCE (A, or a2 = additive genetic variance; D, or d2 = non-additive genetic variance; C, or c2 = shared environment; E, or e2 = remaining variance) components as computed based on information given by different raters:
||Total no. of pairs in category
|Other report (usually parents)
The total, or broad-sense heritability, H2 , is the sum of the additive (the narrow-sense heritability) and the non-additive genetic components. As we can see, when actual criminal records (a semi-objective metric) are used, as we’ve seen, the heritability shoots up to the usual range, at 0.75, and the shared environment estimate vanishes. The criminal record analysis also captures the largest number of subjects, bolstering its reliability. Parent reports, as seen below, inflate the shared environment measure. The self-report gives a negligible shared environment estimate, but reports a lower heritability estimate – which is not surprising, given that we can expect self-reported criminal behavior to be poorly reliable. It is unfortunate that R&W don’t separate out parents from peers and other non-relative raters in “other report.” Additionally, the adoption studies found a negligible shared environment impact of 0.05 between adoptive parents and adoptees. It is also too bad that R&W don’t cross tabulate the results by rating and age. But, as discussed below, adolescent shared environment effects maybe an artifact of unreliable raters anyway.
(For the record, the countries spanned by the studies in the meta-analysis include the U.S., Canada, the U.K., Australia, Denmark, and Sweden.)
The bottom line, it’s clear that when it comes to anti-social behavior, the 75-0-25 rule holds perfectly firm. Parents and parenting do nothing to create upstanding citizens, and heredity is considerably important. ***End Edit***]
But the eugenicists were wrong about everything, ignore the historic era of prosperity exactly one generation after their American sterilizations in the 20s… which they predicted.
Indeed, also supporting this is another massive meta-analysis of behavioral genetic influences on adolescent psychopathology (personality disorders). These captures various types of child misbehavior and dysfunction, including convenient diagnoses such as “oppositional-defiant disorder.” A look and the breakdown of their results is far more interesting than their main reported results. Typically, shared environment effects are seen in children (<18 years old). The main study reported this, but fortunately, they decomposed the type of measurements used. In addition to self-report and parental report, they also had teacher report, peer reports, and clinical diagnoses. The self and parental reports showed lower heritabilities (0.3-0.5) and significant (though small) shared environment components. However, when teacher or peer reports were used, they found much higher heritabilities, in the 0.65-0.8 range. As well, the shared environment impact vanished. Using clinical diagnosis also produced a zero shared environment impact. Considering the sheer size of this review, it’s clear that parental behavior dosen’t contribute to this malaise, even at these ages.
Adoptive parents can lie? Why do that?
The problem of somewhat unreliable measurements (noise), especially coming from self-report, was illustrated in my earlier posts. Averaged peer ratings serve to adjust for this problem to an extent both by providing more proper social context by which to make accurate comparative ratings and by cancelling out fluke readings. Indeed, one behavioral genetic study, which attempted to investigate the idea of a “general factor of personality” (GFP), akin to g for cognitive ability, found that when using the combined scores of self and peer ratings, the heritabilities of the Big Five personality traits shot through the roof, with the additive heritable component being:
- Extraversion: 0.86
- Openness: 0.92
- Neuroticism: 0.59
- Agreeableness: 0.85
- Conscientiousness: 0.81
This demonstrates that more accurate measurements consistently push up the heritability estimate (even pushing them towards 100%), giving us the basis of the 75-0-25 or something rule.
As for the sixth dimension of personality, “honesty-humility”, the H component of the six factor HEXACO, evidence of its high heritability is also established, as we saw previously. Indeed, a recent post by Peter Frost (Evo and Proud: Compliance with moral norms: a partly heritable trait?) discussed a twin study from Sweden that looked at various forms of dishonesty, such as fraudulently claiming sick benefits or evading taxes. And sure enough, these particular behaviors showed considerable heritability. There is a desperate need for cross cultural behavioral genetic analyses. Many dimensions of personality systems like the HEXACO (as imperfect as they are) are likely to systematically vary from culture to culture.
Adoption is dyscivic. It’s AA for bad parents.
The usefulness of behavioral genetics – indeed, the single most powerful and solid area of all social science – is highly evident. But behavioral genetic methods can be used to address several long-standing questions. Here we see it’s clear that parents don’t leave much of an impact on our behavioral traits. But what about people who aren’t parents? Here I will look at two sets of important people, spouses and peers.
It is no secret that spouses correlate on behavioral traits. This, assortative mating, is a powerful force, as we’ve seen previously. There are two aspects where spouses are highly correlated – the things you don’t talk about in a bar: politics and religion. Some have assumed that a good bit of this is because spouses grow more similar with time. But is this the case?
This is where the “extended twin” design comes in handy. One large study (N > 20,000) in particular looked at precisely that. By including twins, their spouses, and parents, etc, they were able to directly measure assortative mating. What did they find? Spouses were correlated for several traits. But the traits they were most correlated in were political orientation and religiosity. Social “homogamy” (having the same background as your spouse) couldn’t explain this, as the correlation between MZ twins and their co-twin’s spouse were consistently higher than that of DZ twins, and so on. As well, spouses weren’t influencing each other, as the correlation between spouses was not affected by length of the marriage (even when only couples married <2 years were examined).
The neocons marrying lefties are kidding themselves.
And leagues clearly exist, assortative mating is genetic.
The study was also able to lay to rest another persistent myth. We’ve heard that we choose spouses like our opposite sex parent (like our mothers for men and like our fathers for women). Anyone who’s remotely genetically informed should be able to see that this could just be due to choosing mates like ourselves. And so is the case. As the authors put it:
there was no evidence for the sexual imprinting hypothesis. Twins’ partners were not significantly more similar in any trait to the twin’s opposite-sex parent than to the twin’s same-sex parent or a DZ co-twin of either sex, nor was there even a trend in this direction
These results were also consistent with the Peter Hatemi et al extended twin study on political attitudes featured previously.
The similarity between spouses has nothing to do with mutual influence, but assortment. At least this bit is common sense. I suspect few long married individuals will believe that they changed their spouse.
On that note, a key theory put forward by the woman who first elucidated the non effect of parents, Judith Rich Harris, was that the unique environment “influence” might be boiled down to peer influence. Staffan did a nice recap of Harris’s theory (see The Nurture Enigma – How Does the Environment Influence Human Nature? | Staffan’s Personality Blog). We all have heard of peer pressure. And indeed, peers seem to be an important force when it comes to language and behaviors like smoking initiation. But do peers really have this great influence, as Harris posits? Well, as I posted over at the Lion of the Blogosphere:
Most research into peer effects is confounded by the same thing that standard parenting studies are: inability to control for the effect of heredity.
A behavioral genetic study (on the Add Health data) that looked specifically at GPA and found that 72% of the similarity between U.S. high school students and their peers could be explained by genetic factors. In other words, school performance and the apparent peer “influence” is really just kids choosing to associate with kids of similar intelligence and motivation:
A behavior genetic analysis of the tendency for youth to associate according to GPA
Peers seem like a fine avenue to get excited about, because it seemed like a vehicle through which parents could assert some influence. But, when you really consider it, peers can’t really be all that important in the long run, because if there were systematic effects of peers on adult outcomes, it’d turn up in the shared environment, which it doesn’t. One could posit that the effect of peers is completely random, but if that were true (aside from the major violation of Occam’s Razor that presents), why worry about it?
The “75-0-25 or something” rule is robust and reliable. This instructs that should we find some major deviation from this, it can be taken to be a sign something is seriously amiss. We saw that with male homosexuality (see Greg Cochran’s “Gay Germ” Hypothesis – An Exercise in the Power of Germs). Now I will discuss two curious exceptions to this pattern.
One rather astonishing example was the heritability of social trust. A behavioral genetic study out of the Netherlands found that the heritability of trust in others, as measured by:
The trust-in-others and trust-in-self scales were designed to include three items that were central in existing scales … thereby capturing items with positive valence (“I completely trust most other people”) and negative valence (“When push comes to shove, I do not trust most other people”), both of which explicitly used the word “trust”, and an item that captured the broad behavioral implication of the trust: the intention to accept vulnerability, as explicated in one of the most widely-accepted definitions of trust … (“I dare to put my fate in the hands of most other people”)
…found no significant heritable influence on these. The extent that people trusted, at least as captured by these measures, was virtually entirely a function of the unique environment.
homogeneous environment > high trust
This was a puzzling result. The clear pattern of the high heritability of all behavioral traits was established, as I’ve discussed. So how could a propensity to trust not also be influenced by genetic factors? One explanation touted around was that trust is contingent on experience; if we found people trustworthy, we would trust. If we didn’t, we would not. While that might sound convincing, the trouble is that the same could be said for many other behavioral traits. Is general trust less socially contingent than say bigoted feelings against some groups, like homophobia (which is at least 54% heritable)? That seems rather unreasonable.
One key question: how do they assess “trust”? Just how good was their measurement? Measurements in social science need to meet three basic criteria: they need to be reliable (that is multiple testing instances of the same individual should give roughly the same results), they need to be “valid” (that is, be predictive of some real-world outcome), and they should be heritable. This trust measure clearly fails on the third criterion. However, the study authors claim the test-retest correlation was good, so it is reliable. But what about the second? Does this trust measure actually predict anything?
To find out, I looked at a study that sought to answer that very question. This study, done in Germany, looked in detail at the reliability and the validity of their measurement of trust, a measurement very similar to the Dutch study. The noted a key point, one HBD Chick will appreciate. That is, trust is multi-faceted. There is trust in institutions, which is distinct from trust in known others, which is distinct from trust in strangers (I’d imagine HBD Chick would break it down one more, and separate “known others” into family and non-family). But more importantly, to question of validity, they assessed this by the correlation between trust in strangers and trusting behavior in the “dictator game.” They found a correlation, but only with trust in strangers.
But their correlation was very small (Spearman’s = 0.17) – and this is with a game which itself has questionable relation to trust behavior in the real world. I suspect that their instrument is not predictive of any trusting behavior in the real world. It’s worth mentioning another (fairly small) study of the heritability of trust from Australia found a non-insignificant heritability, though a smallish one (0.14-0.31).
The situation with trust is unclear. But this brings me to another example of a feature for which the heritability estimate appears to be trivial. That is the female G-spot. A study on about 1,800 female twins from Britain found that the heritability of the reported presence of a G-spot wasn’t significant. The result was virtually entirely unshared environment. Debate has raged on as to whether or not the female G spot exists at all, but that is to be expected, since research into human sexual behavior is among the most difficult to conduct properly. But, the result from this study indicating that the G spot isn’t heritable is puzzling. If the G spot was a real anatomical feature, and one that wasn’t universal, then one would expect a rather significant heritable impact. The finding that it’s not heritable points to one of two conclusions. One, perhaps the G-spot is in fact universal, but only some women have “discovered” it. That seems rather implausible, given the rather significant variation in heritable morphological features of sex organs in women. The second possibility is that the G-spot in fact doesn’t exist at all, and women who claim to have one are mistaken. That seems more likely, but I wouldn’t want to completely dismiss the claims of women who state they have such a feature. The mystery remains.
The findings of behavioral genetics, particularly the highly significant impact of heredity and the absence of shared environment effects, in addition to the complete failure to find reliable environmental sources that contribute to the “unique environment” component of the variance, calls into question virtually every pet environmental theory that has been put forward. It guides one to be suspicious of most “environmental” explanations of behavior. Now, let me be clear, I am not saying that these environmental influences don’t exist. I am not saying that if they do exist, we won’t be able to ever find them. I am also not saying that development doesn’t require a complex interplay between genes and environment. Try going without food, water, air, or speaking to another person if you don’t believe me. I am also not saying that the secular changes in human traits that are brought about by gross environmental changes don’t happen. The increase in average height over the past century disproves that. But what I am saying is that you should be doubtful of most pet theories of how the environment influences us, especially those that promise we can control, or sometimes even predict it. For as we see, that’s far from an easy task.