I wonder what these guys recently all had in common?
I WONDER. IF ONLY SOMEBODY HAD PREDICTED THIS.
No date on this:
What did I say? Depopulation shots, if I were to design them, would target overall fertility in women, and mutate the sperm in the men.
Nobody was checking the men, I’ve been blowing this whistle all year.
“All participants have never suffered from infertility and have not been treated for it. The semen samples were tested for parameters and immune factors.”
Did they have herp? That causes infertility in men. It’s hugely covered up though.
I’ve posted about it.
These guys were dead, so let’s consider this a win for natural selection. Maybe sub-fertility in general predicts premature mortality for men? (see below, checked) HOWEVER: If disease-ridden manwhores are most likely to die of coof, I’ll personally send a thank you letter to Pfizer.
THE WAGES OF SIN ARE STILL DEATH. Das the real redpill, and white.
Thus, we were able to observe the disruption of sperm production, which can be partially explained by the result of an increased immune response from the testes.
Inflammation? Like from modRNA?
In addition to this, autoimmune inflammation of the testicles occurred in some COVID-19 patients.
Specifically WHAT PERCENTAGE had received a modRNA injection of any variety?
NO data? Seriously? You believe that? If they had pure blood, they’d be screeching told ya so.
Is it the disease or symptoms of a reaction to the jabs?
The current study observed a signiﬁcant impact on male fertility and pathological changes in male gonads that might impact healthy reproductive well-being in male COVID-19 patients in the long-term. It is connected with impaired spermatogenesis with low sperm count, inflammatory responses in the testes and epididymis, and altered markers of seminal immunity indicating impaired immunity due to COVID-19 disease.
Why not tell us the percentages?
Research funded by China, this is a cover-up.
Impaired human spermatogenesis and delayed sperm maturation may be the result of an immune response in the testes and epididymis of COVID-19 patients, these factors are vital and determinant for healthy male fertility. It requires further care and support of males infected with SARS-CoV-2. It may indicate an increase in male infertility as a consequence.
Or immune response to the clot shots, since it impaired the tubes, which viruses don’t actually do?
“COVID-19 affects men more than women compared to its original form. SARS-CoV-2 can stay in human bodies longer than SARS-CoV. Its clinical characteristics are similar to SARS, but the male reproductive system is seriously affected by it. The male reproductive system is vulnerable to viral infections, and previous viral epidemics had documented varying impacts on male reproductive functions.
Depop by design.
There is substantial recorded evidence that the male reproductive system is vulnerable to viral infections like MUMPS, ZIKA, Hepatitis B virus (HBV), Hepatitis C Virus (HCV), Human Immunodeficiency Virus (HIV), Human papillomavirus (HPV), Herpes, EBOLA.“
It’s a bioweapon. Chinese ball bleach. But let them marry into your family, sure.
As such, the manosphere, as primary vectors of birth-rate killing disease, should be banned from any traditional society. Banishment is holy. God did it.
A lot of hebe/pedo guys you’d think are Chads, like Henry Cavill, are mostly infertile (sub-fertile) due to their HPV, herp, etc. Watch that scandal break, the wages of sin…. HC had a John Doe case in Canada for failing to inform a woman prior to sex that he had herp and passing it on, ruining her fertility. Due to the medical damage, she won because they are spreading infertility via their STDs, we didn’t lynch cads without just cause. Patriarchy was right. Soon it’ll be prosecuted as rape by fraud, like a tranny lying to a man. Here here.
The clot shot will target the epithelial cells in it.
The mammalian epididymis is a highly specialized region of the male reproductive tract that is lined with a continuous layer of epithelial cells that display a remarkable level of regionalized secretory and absorptive activity. The luminal environment created by this combined secretory and absorptive activity is directly responsible for promoting the functional maturation of spermatozoa and their maintenance in a quiescent and viable state prior to ejaculation. This study was designed to identify the complement of microRNAs (miRNAs) that are expressed within the mouse epididymal epithelial cells and the maturing populations of spermatozoa. Through the use of Next Generation Sequencing technology we have demonstrated that both epididymal epithelial cells and spermatozoa harbour a complex repertoire of miRNAs that have substantially different expression profiles along the length of the tract. These data, deposited in the Gene Expression Omnibus (GEO) with the accession numbers GSE70197 and GSE70198, afford valuable insight into the post–transcriptional control of gene expression within the epididymis and provide the first evidence for the dynamic transformation of the miRNA content of maturing sperm cells. Ultimately such information promises to inform our understanding of the aetiology of male infertility. Herein we provide a detailed description of the methodology used to generate these important data.
It applies to sperm in other mammals too.
Mutant sperm causes miscarriages.
So the side effect of women miscarrying may not have been THEIR clot shot.
But some research over the past decade suggests that this might not always be the case. Some cases of recurrent miscarriages seem to involve the father having a high incidence of abnormal chromosomes in his sperm.
There aren’t any real estimates for how frequently the sperm is a factor in recurrent miscarriages, and chromosome problems in sperm aren’t believed to be a major cause of repeat losses. It does seem to be a possibility—especially in men whose sperm showed abnormal morphology or other markers of low fertility.
Sperm cannot repair cell damage like other cells in the body and this is a leading cause of male infertility. The damage also affects the DNA structure within the sperm and if it fertilizes an egg, this can potentially lead to miscarriage.
One study compared couples who experienced multiple miscarriages to couples with infertile men and fertile men. The result is that the sperm involved in the miscarriages was more likely to have signs of fragmentation at nearly the same rate as that of the infertile men.
TLDR: Stop blaming the women.
These sorts of results suggest that the correlation between SDF and fertility—both the inability to conceive and miscarriages—can be a factor. However, researchers are cautious to note that SDF alone cannot predict a couple’s risk of miscarriage.
Miscarriage is the woman’s body rejecting him because her mind was too dumb to. Like female weebs.
Unhealthy choices can lead to the many factors that decrease the chances of a successful pregnancy. This includes decreased sperm mobility and vitality, lower sperm counts, and abnormal morphology (the size and shape of sperm). It can also lead to physical damage. Any damage to sperm can cause fertility problems and, if an egg is fertilized, it may also lead to a miscarriage.
Test men early. No reason not to.
Don’t wait until miscarriage number 3.
Some physicians may recommend that men undergo tests for sperm quality when no other cause for recurrent miscarriages is found. The standard test is a sperm analysis, which looks at the shape, mobility, and sperm count in the sample.
Or test them FIRST prior to the trauma that’s 100% preventable by them jizzing in a cup?
The trauma that puts women off trying more?
Childless men more likely to die, as I suspected.
Manosphere get rekt.
It’s likely the sperm mutation is also affecting the brain by similar disease vectors like herp, causing childlessness but also early death. Like the rats attracted to cats.
They sense their low quality and hence, don’t bother to invest. Self-selection of genetic suicide. This is why the Spartans had a bachelor tax, it was a tax on the genetic dregs. All healthy men have a paternal instinct.
Men who wait until post-Wall to have kids, tend to have dead kids.
Risk of childhood mortality in family members of men with poor semen quality
So the kids they do have will die early, this is also seen among the mixed with rare genetic conditions.
What is known already: Semen quality is an established predictor of men’s somatic health. We can gain a better understanding of possible genetic or environmental determinants of the infertility phenotype by exploring familial aggregation of childhood mortality in relatives of men with poor semen quality.
Wider implications of the findings: We are not the first study to show a relationship between fertility and CMs. Children conceived through ART may be at higher risk of birth defects, however it is not known if the relationship is causal or if there is some underlying factor linking infertility and birth outcomes. This study provides further evidence that the increased risk of congenital birth defects may not be due to the ART, but rather genetic or environmental factors that link the two outcomes. We encourage further research in order to confirm a relationship between semen quality and increased risk for CM.
R-type children, like the fathers, age like shit.