1995 evidence of precedent for deceptive vector of transmission, official contamination reportage and hence, UN contravention of genocide law established in the 1940s, section (d) and (c):
“PIP: A priest, president of Human Life International (HLI) based in Maryland, has asked Congress to investigate reports of women in some developing countries unknowingly receiving a tetanus vaccine laced with the anti-fertility drug human chorionic gonadotropin (hCG). If it is true, he wants Congress to publicly condemn the mass vaccinations and to cut off funding to UN agencies and other involved organizations. The natural hormone hCG is needed to maintain pregnancy. The hormone would produce antibodies against hCG to prevent pregnancy. In the fall of 1994, the Pro Life Committee of Mexico was suspicious of the protocols for the tetanus toxoid campaign because they excludedallmalesandchildren and called for multipleinjections of the vaccine in onlywomenofreproductiveage. Yet, one injection provides protection for at least 10 years. The Committee had vials of the tetanus vaccine analyzed for hCG. It informed HLI about the tetanus toxoid vaccine. HLI then told its World Council members and HLI affiliates in more than 60 countries. Similar tetanus vaccines laced with hCG have been uncovered in the Philippines and in Nicaragua. In addition to the World Health Organization (WHO), other organizations involved in the development of an anti-fertility vaccine using hCG include the UNPopulationFund, the UN Development Programme, the World Bank, the PopulationCouncil, the RockefellerFoundation, the US National Institute of Child Health and Human Development, the All India Institute of Medical Sciences, and Uppsala, Helsinki, and Ohio State universities. The priest objects that, if indeed the purpose of the mass vaccinations is to prevent pregnancies, women are uninformed, unsuspecting, and unconsenting victims.”
“Vaccines are under development for the control of fertility in males and females. This review discusses developments in anti-fertility vaccines at the National Institute of Immunology, New Delhi, India. A single injection procedure for the sterilization or castration of male animals depending on the site at which the injection is given, has passed through fieldtesting and is expected to be on the market in the nearfuture. Vaccines inducing antibodiesagainstthehuman chorionic gonadotropin have gone through phase I trials with satisfactoryresults. A vaccine producing a consistentlybioeffectiveantibody response against gonadotropin-releasing hormone is ready for phase I/II clinical trials in patients of carcinoma of prostate after due experimentation in animals and toxicology studies. Research to identify sperm antigens for incorporation into second generation vaccines is in progress.”
Control, like farm animals. Single injection for castration of male animals possible.
Look forward to the “drop of testosterone levels”. At least you got to drink some bitch tits beer with the ‘boys’. Don’t come crying to me. You wanted to be ‘male animals’.
Evaluation of human papilloma virus in semen as a risk factor for low sperm quality and poor in vitro fertilization outcomes: a systematic review and meta-analysis
A review of the literature regarding ART outcomes showed an association between HPV infection and decreased PR, and an even stronger association between HPV infection and increased MR.
-increased miscarriage rate, lower odds of conceiving
Conclusion: Our meta-analysis shows a negative effect of HPV on sperm concentration, motility, and morphology. Further subgroup and categorical analysis confirmed the clinical significance of impaired sperm motility in HPV-infected sperm, although the sperm count and morphology must be carefully analyzed. The studies reviewed reported lower PR and increased MR in couples with HPV-infected sperm. As most studies had a moderate risk of bias, these observations warrant further large, well-designed studies before introducing clinical management recommendations.
Human papillomavirus infection and fertility alteration: a systematic review
Results: HPV infections are shown to be significantly associated to many adverse effects in the reproductive function. These adverse effects were reported in different levels from cells production to pregnancy and may be related to the infecting genotype.
Conclusions: It appears from this study that HPV detection and genotyping could be of great value in infertility diagnosis at least in idiopathic infertility cases. Like for the risk of carcinogenesis, another classification of HPV regarding the risk of fertility alteration may be considered after deep investigations.
Human Papilloma Virus (HPV) and Fertilization: A Mini Review
Sorry but if something makes you less virile, you’re less of a man.
Human papilloma virus (HPV) is one of the most prevalent viral sexually transmitted diseases. The ability of HPV to induce malignancy in the anogenital tract and stomato-pharyngeal cavity is well documented. Moreover, HPV infection may also affect reproductive health and fertility. Although, the impact of HPV on female fertility has not been thoroughly studied it has been found also to have an impact on semen parameters. Relative information can be obtained from studies investigating the relationship between HPV and pregnancy success. Furthermore, there is an ongoing debate whether HPV alters the efficacy of assisted reproductive technologies. An association between HPV and assisted reproductive technologies (ART) programs has been reported. Nevertheless, due to conflicting data and the small number of existing studies further research is required. It remains to be clarified whether HPV detection and genotyping could be included in the diagnostic procedures in couples undergoing in vitro fertilization (IVF)/intrauterine insemination (IUI) treatments. Vaccination of both genders against HPV can reduce the prevalence of HPV infection and eliminate its implications on human fertility. The aim of the present mini-review is to reiterate the association between HPV and human fertility through a systematic literature review.
The role of human papillomavirus on sperm function
I love how many yanks pull a Henry 8th and blame women for their own infertility, in this century.
Recent findings: HPVs are agents of the most common sexually transmitted disease and can lead to warts and cancers both in men and women. A high incidence of HPV infection has been demonstrated in sperm from sexually active men with and without risk factors for HPV and from infertile patients.
Semen infection is associated to an impairment of sperm parameters suggesting a possible role in male infertility. – really???
Interestingly, it has been demonstrated that when HPV is present in semen only a percentage of total cells are infected
-only? a? 100% is a percentage too…
and the virus can be localized in sperm or in exfoliated cells with different impact on sperm motility. Moreover, infected sperm are able to penetrate the oocyte, to deliver HPV genome in the oocyte and HPV genes can be actively transcribed by the fertilized oocyte.
-wouldn’t it be ironic if it made the kids or grandkids infertile instead? because they were conceived with it, a polluted germline
Recently an increased risk of pregnancy loss has been demonstrated in couples undergoing in-vitro fertilization and particularly when HPV DNA was present in semen samples of male partners.
– no blaming women this time, unless women haz sperm?
Summary: To date, no effective treatment, control strategy and prevention is provided for men despite the reported high incidence of HPV semen infection.
– no hurt their feefees? NAW
Because this infection in men is also a problem for partners, and because growing evidence suggests that semen infection may cause infertility and early miscarriage, more attention should be paid to male HPV infection. This study reviews the more recent literature about the role of HPV infection on sperm function and human reproduction.
– Manosphere fears this topic and all male degenerate accountability.
semen infection may cause infertility and early miscarriage
High-risk human papillomavirus in semen is associated with poor sperm progressive motility and a high sperm DNA fragmentation index in infertile men
Does the presence of human papillomavirus (HPV) in semen impact seminal parameters and sperm DNA quality in white European men seeking medical help for primary couple’s infertility?
>STD >DNA quality >in the germline of >white men
Never talk about it, I’m sure it’ll be fine.
HPV seminal infections involving high-risk (HR) genotypes are associated with impaired sperm progressive motility and sperm DNA fragmentation (SDF) values.
HPV is commonly present in semen samples.
No? F no it’s not. Stop sparing slutty blushes.
The overall rate of HPV positivity was 15.5%
so 1 in 7, uncommon at best. No normalizing pathology please.
And it varies majorly by race and sexuality. Not sex because it’s sexual, obviously.
Sperm progressive motility was significantly lower (P = 0.01) while SDF values were higher (P = 0.005) in HPV+ men compared to those with no HPV. In particular, HR HPV+ men had lower sperm progressive motility (P = 0.007) and higher SDF values (P = 0.003) than those with a negative HPV test. Univariable analysis showed that HR HPV+ was associated with impaired sperm progressive motility (P = 0.002) and SDF values (P = 0.003). In the multivariable analysis, age, FSH levels and testicular volume were significantly associated with impaired sperm progressive motility (all P ≤ 0.04). Conversely BMI, CCI, smoking habits and HPV status were not. Only age (P = 0.02) and FSH (P = 0.01) were significantly associated with SDF, after accounting for BMI, CCI, testicular volume, smoking habits and HPV status.
Impact of human papillomavirus infection in semen on sperm progressive motility in infertile men: a systematic review and meta-analysis
Background: Human papillomavirus (HPV) has been considered as one of the most common sexually transmitted viruses that may be linked to unexplained infertility in men. The possible mechanisms underlying correlation between HPV infection and infertility could be related to the altered sperm parameters. Current studies have investigated the effect of HPV seminal infection on sperm quality in infertile men, but have shown inconsistent results.
Methods: We systematically searched PubMed, Embase, Web of Science and CNKI for studies that examined the association between HPV seminal infection and sperm progressive motility. Data were pooled using a random-effects model. Outcomes were the sperm progressive motility rate. Results are expressed as standardised mean difference (SMD) with 95% confidence interval (CI). Heterogeneity was evaluated by the I-square (I2) statistic.
Results: Ten studies were identified, including 616 infertile patients with HPV seminal infection and 2029 infertile controls without HPV seminal infection. Our meta-analysis results indicated that sperm progressive motility was significantly reduced in HPV-infected semen samples compared with non-infected groups [SMD:-0.88, 95% CI:-1.17 ~ – 0.59]. There existed statistical heterogeneity (I2 value: 86%) and the subgroup analysis suggested that study region might be the causes of heterogeneity.
Conclusions: HPV semen infection could significantly reduce sperm progressive motility in infertile individuals. There were some limitations in the study such as the differences in age, sample sizes and the number of HPV genotypes detected. Further evidences are needed to better elucidate the relationship between HPV seminal infection and sperm quality.
The prevalence of Human Papilloma Virus (HPV) infection in the oligospermic and azoospermic men
The current study shows that HPV infection can affect on sperm count and motility and decrease count of sperm cell and decrease motility capability of these cells.
Among 50 confirmed oligospermic male, 15 were HPV DNA positive (30%).
In azoospemic group we had 8 HPV DNA positive (40%) and in normal group just 3 of 20(15%) samples were positive.
-what r the odds?
we found statistical significant relationship for sperm count (p<0.05) and sperm motility (slow) (p<0.05) in oligospermic group positive samples compared with negative. In the present study, 13 HPV genotypes were detected among positive samples. HPV genotypes 16, 45 in the high risk group and 6,11,42 in the low risk group were more frequent than the others.
Semen washing procedures do not eliminate human papilloma virus sperm infection in infertile patients
had HPV DNA on sperm and exfoliated cells. Sperm washing centrifugation showed no changes in the number of infected samples and in the percentage of infected cells. Ficoll and swim-up protocols induced a slight reduction in the number of infected samples (30 and 26, respectively).
no muh scientism and IVF cope
This study demonstrated that conventional sperm selection rarely eliminates HPV sperm infection. More attention should be paid to the reproductive health of infected patients because, not only can HPV be transmitted, but it may also have a negative effect on development of the fetus.
a negative effect on development of the fetus
so even if they all married a virgin waifu, they’d infect her and have defective babies comedy GOLD, 24K.
Is HPV the Novel Target in Male Idiopathic Infertility? A Systematic Review of the Literature
Infertility is an important health problem that affects up to 16% of couples worldwide.
1 in 7, where have I heard THAT before….? [scroll up]
Male infertility is responsible for about 50% of the cases,
–NAY, men are never responsible for their own in/fertility, have you been online recently?
and the various causes of male infertility may be classified in pre-testicular (for example hypothalamic diseases), testicular, and post-testicular (for example obstructive pathologies of seminal ducts) causes. Sexually transmitted infections (STI) are increasingly widely accepted by researchers and clinicians as etiological factors of male infertility. In particular, several recent reports have documented the presence of HPV in seminal fluid and observed that sperm infection can also be present in sexually active asymptomatic male and infertile patients.
In this review, we aimed to perform a systematic review of the whole body of literature exploring the impact of HPV infection in natural and assisted fertility outcomes, from both an experimental and a clinical point of view. Starting from in-vitro studies in animals up to in-vivo studies in humans, we aimed to study and evaluate the weight of this infection as a possible cause of idiopathic infertility in males with any known cause of conception failure.
Significant Correlation between High-Risk HPV DNA in Semen and Impairment of Sperm Quality in Infertile Men
guess the result
go on think
A total of 140 subjects participated in the current study. Among 70 confirmed infertile males, only 8 (11.43%) cases tested positive for high-risk HPV and all fertile men were HPV-negative. This data revealed a significant association between high-risk HPV and male infertility (P=0.03). The percentage of normal sperm morphology and sperm motility rate significantly declined in men infected with HPV (P<0.001).
and all fertile men were HPV-negative
oof and the sluts of both sexes are dying out, I am distraught. The genetics of the future are fairing brighter than you’d think.
Conclusion: There was a significantly higher prevalence of high-risk HPV in infertile men than fertile men. HPV infection seemed to be a risk factor for male infertility. Additional, larger studies should be conducted to confirm the impact of HPV on male infertility.
Player burnout shall henceforth be dubbed HPV-driven infertility?
Association between human papillomavirus infection and sperm quality: A systematic review and a meta-analysis
Human papillomavirus (HPV) has a high incidence rate in both males and females.
-maybe where you live
HPV infection in women has been shown to affect fertility and lead to foetal death and pregnancy loss. However, research on HPV infection in men is limited.
-well the husbands are freshly infecting the wives so–
-Ashley Madison wasn’t full of women stepping out, was it?
The aim of this study was to study the effect of HPV infection in semen on sperm quality and present the findings of previous studies through a meta-analysis. Databases including PubMed, MEDLINE, EMBASE, Web of Science, Cochrane Library, WanFang data and China National Knowledge Infrastructure were searched for relevant studies. A systematic review and meta-analysis were performed, and 17 studies were included for analyses based on a set criterion. Meta-analyses indicated that HPV infection in semen significantly reduced sperm concentration (SMD = -0.12, 95% CI: -0.21 to -0.03, p = .009), sperm motility (SMD = -0.55, 95% CI: -0.780 to -0.33, p = .000), sperm viability (SMD = -0.55, 95% CI: -0.780 to -0.33, p = .000) and sperm morphology (SMD = -0.34, 95% CI: -0.61 to -0.07, p = .015). The high-risk HPV (HrHPV) infection could significantly reduce sperm count (SMD = -0.65, 95% CI: -1.11 to -0.18, p = .007) compared with high-risk HPV (LrHPV) infection.
In conclusion, HPV infection in semen significantly reduced sperm quality, and the HrHPV infection could significantly reduce sperm count compared with LrHPV.
tick tock goes your biological clock, nobody can wait as long as they want NOBODY
Male sperm quality and risk of recurrent spontaneous abortion in Chinese couples: A systematic review and meta-analysis
Conclusions: The results of this analysis support an association of sperm density, sperm viability, sperm progressive motility rate, normal sperm morphology rate, sperm deformity rate, as well as sperm DFI with RSA.
IF you conceived, magically, it would kill your baby. REPEATEDLY.
Semen parameters and sperm morphology in men in unexplained recurrent spontaneous abortion, before and during a 3 year follow-up period
Baby death aborts the defective DNA, HPV fucks with your sperm’s DNA. Water is wet.
HPV makes you biologically unfit. According to the ultimate test, the womb.
To investigate the role of the ‘male factor’ in the pathogenesis of recurrent spontaneous abortion (RSA), especially sperm morphology abnormalities, 120 previously selected couples with unexplained RSA were studied for sperm parameters retrospectively and prospectively. The patients were subdivided into three subgroups, depending on their reproductive outcome during the 3 years of follow-up study: (i) 48 RSA couples who achieved a successful pregnancy; (ii) 39 RSA couples who experienced further abortions, and (iii) 33 RSA couples who experienced infertility during the follow-up period. A semen analysis was performed twice at the time of inclusion in this study, and twice again during the 3 year follow-up period. No significant differences in semen parameters were observed between RSA males and fertile controls. Instead, significant differences were observed between the group of RSA couples who experienced infertility during the follow-up and the other two groups (RSA couples who achieved successful pregnancy and RSA couples who experienced miscarriages and no live birth during the follow-up) for sperm concentration (P < 0.01 and P < 0.01 respectively), sperm motility (P < 0.01 and P < 0.01 respectively) and sperm morphology abnormalities (P < 0.01 and P < 0.01 respectively).
Sperm DNA fragmentation in couples with unexplained recurrent spontaneous abortions
The aim of the present study was to evaluate the degree of sperm DNA fragmentation in couples with idiopathic recurrent spontaneous abortion (RSA) and in those with no history of infertility or abortion. In this cohort study, 30 couples with RSA and 30 fertile couples as control group completed the demographic data questionnaires, and their semen samples were analysed according to World Health Organization (WHO) standards (September 2009-March 2010) for evaluation of sperm DNA fragmentation, using sperm chromatin dispersion (SCD) technique. The percentage of morphologically normal sperm was significantly lower in RSA patients compared with control group (51.50 ± 11.60 versus 58.00 ± 9.05, P = 0.019), but not in other parameters. Additionally, the level of abnormal DNA fragmentation in the RSA group was significantly higher than in the control group (43.3% versus 16.7%, P = 0.024). Our results indicated a negative correlation between the number of sperm with progressive motility and DNA fragmentation (r = -0.613; P < 0.001). The sperm from men with a history of RSA had a higher incidence of DNA fragmentation and poor motility than those of the control group, indicating a possible relationship between idiopathic RSA and DNA fragmentation.
Sperm chromatin integrity may predict future fertility for unexplained recurrent spontaneous abortion patients
“unexplained” – just assume the echo for comedic effect by now
The RSA group was further separated into three subgroups, depending on their reproductive outcome during the 12 months after they were enrolled in the study: the pregnancy subgroup consisted of 43 men whose partners achieved a successful pregnancy up to at least the 24th week of gestation; the abortion subgroup included 31 men whose partners experienced further abortions; and the infertile subgroup had 37 men whose partners did not have any positive pregnancy test after regular, unprotected intercourse. Significantly lower proportion of sperm with normal morphology was found in the abortion subgroup (14.7 ± 4.3%) than in the control group (17.5 ± 5.0%). Sperm concentrations were significantly lower in the infertile subgroup (55.7 ± 24.1%) than in the controls (68.6 ± 27.8%). The rates of abnormal sperm chromatin integrity were significantly higher in the abortion (16.7 ± 7.7%) and infertile (16.3 ± 6.6%) subgroups, compared to the control group (13.0 ± 4.4%). Logistic regression analysis showed that the subsequent reproductive outcome of the 111 RSA patients was negatively correlated to the rates of abnormal sperm chromatin integrity. In conclusion, sperm chromatin integrity, sperm morphology, and sperm concentration were associated with future reproductive outcome of RSA patients. The sperm chromatin integrity was a significant predictor for future abortion and infertility.
But men are never responsible for miscarriage, perish the THOUGHT.
Cytochemical evaluation of sperm chromatin and DNA integrity in couples with unexplained recurrent spontaneous abortions
unexplained….. sigh, ok.
Our study showed that in the cases of RSA, slow motility had a significant reduction in comparison with controls and also spermatozoa of men from RSA group had less chromatin condensation and poorer DNA integrity than spermatozoa that obtained from fertile men with no history of RSA.
Human sperm deoxyribonucleic acid fragmentation by specific types of papillomavirus
Conclusion: Human papillomavirus type 16 and 31 deoxyribonucleic acid caused deoxyribonucleic acid breakages characteristic of apoptotic but not necrotic sperm.
The data suggest that these human papillomavirus types may adversely affect subsequent embryonic development after fertilization. Sperm deoxyribonucleic acid appears to resist human papillomavirus types 18, 33, and 6/11 or repairing mechanisms occurred. Although enhanced motility was found in human papillomavirus–exposed sperm, important velocity parameters were decreased, suggesting impaired sperm function.
Negative Impact of Elevated DNA Fragmentation and Human Papillomavirus (HPV) Presence in Sperm on the Outcome of Intra-Uterine Insemination (IUI)
i.e. no, you won’t just get IVF
We wanted to determine the sperm DNA fragmentation index (DFI) cutoff for clinical pregnancies in women receiving intra-uterine insemination (IUI) with this sperm and to assess the contribution of Human Papillomavirus (HPV) infection on sperm DNA damage and its impact on clinical pregnancies. Prospective non-interventional multi-center study with 161 infertile couples going through 209 cycles of IUI in hospital fertility centers in Flanders, Belgium. Measurement of DFI and HPV DNA with type specific quantitative PCRs (HPV 6, 11, 16, 18, 31, 33, 35, 39, 45, 51, 52, 53, 56, 58, 59, 66 and 68) in sperm before its use in IUI. Clinical pregnancy (CP) rate was used as the outcome to analyze the impact on fertility outcome and to calculated the clinical cutoff value for DFI. A DFI criterion value of 26% was obtained by receiver operating characteristic (ROC) curve analysis. Couples with a male DFI > 26% had significantly less CPs than couples with DFI below 26% (OR 0.0326; 95% CI 0.0019 to 0.5400; p = 0.017). In sperm, HPV prevalence was 14.8%/IUI cycle. Sperm samples containing HPV had a significantly higher DFI compared to HPV negative sperm samples (29.8% vs. 20.9%; p = 0.011). When HPV-virions were present in sperm, no clinical pregnancies were observed. More than 1 in 5 of samples with normal semen parameters (17/78; 21.8%) had an elevated DFI or was HPV positive. Sperm DFI is a robust predictor of clinical pregnancies in women receiving IUI with this sperm. When DFI exceeds 26%, clinical pregnancies are less likely and in vitro fertilization techniques should be considered
When HPV-virions were present in sperm, no clinical pregnancies were observed.
Sperm viral infection and male infertility: focus on HBV, HCV, HIV, HPV, HSV, HCMV, and AAV
Chronic viral infections can infect sperm and are considered a risk factor in male infertility. Recent studies have shown that the presence of HIV, HBV or HCV in semen impairs sperm parameters, DNA integrity, and in particular reduces forward motility. In contrast, very little is known about semen infection with human papillomaviruses (HPV), herpesviruses (HSV), cytomegalovirus (HCMV), and adeno-associated virus (AAV). At present, EU directives for the viral screening of couples undergoing assisted reproduction techniques require only the evaluation of HIV, HBV, and HCV.
-all trust the EU guys
However, growing evidence suggests that HPV, HSV, and HCMV might play a major role in male infertility and it has been demonstrated that HPV semen infection has a negative influence on sperm parameters, fertilization, and the abortion rate.
-somebody else look up herpes, I’m lazy
Besides the risk of horizontal or vertical transmission, the negative impact of any viral sperm infection on male reproductive function seems to be dramatic.
-Really, f-ing fascinating!
In addition, treatment with antiviral and antiretroviral therapies may further affect sperm parameters. In this review we attempted to focus on the interactions between defined sperm viral infections and their association with male fertility disorders. All viruses considered in this article have a potentially negative effect on male reproductive function and dangerous infections can be transmitted to partners and newborns. In light of this evidence, we suggest performing targeted sperm washing procedures for each sperm infection and to strongly consider screening male patients seeking fertility for HPV, HSV, and HCMV, both to avoid viral transmission and to improve assisted or even spontaneous fertility outcome
HPV infection in semen: results from a new molecular approach
Let’s get molecular.
Human papillomavirus (HPV) is the agent of the most common sexually transmitted diseases causing a variety of clinical manifestations ranging from warts to cancer. Oncogenic HPV infection is the major cause of cervical cancer and less frequently of penile cancers. Its presence in semen is widely known, but the effects on fertility are still controversial. – how? allergic to facts?
We developed a new approach to evaluate virus localisation in the different semen components. We analysed also the specific genotype localisation and viral DNA quantity by qPCR. Results show that HPV DNA can be identified in every fraction of semen: spermatozoa, somatic cells and seminal plasma. Different samples can contain the HPV DNA in different fractions and several HPV genotypes can be found in the same fraction. Additionally, different fractions may contain multiple HPV genotypes in different relative quantity. We analysed the wholeness of HPV DNA in sperm cells by qPCR. In one sample more than half of viral genomes were defective, suggesting a possible recombination event. The new method allows to easily distinguish different sperm infections and to observe the possible effects on semen. The data support the proposed role of HPV in decreased fertility and prompt new possible consequences of the infection in semen.
>HPV DNA can be identified in every fraction of semen: spermatozoa, somatic cells and seminal plasma
If you’re wondering why your nation is infertile, look in the mirror. Mutant sperm.
Women’s Preferences for Penis Size: A New Research Method Using Selection among 3D Models
Women’s preferences for penis size may affect men’s comfort with their own bodies and may have implications for sexual health. Studies of women’s penis size preferences typically have relied on their abstract ratings or selecting amongst 2D, flaccid images. This study used haptic stimuli to allow assessment of women’s size recall accuracy for the first time, as well as examine their preferences for erect penis sizes in different relationship contexts. Women (N = 75) selected amongst 33, 3D models. Women recalled model size accurately using this method, although they made more errors with respect to penis length than circumference. Women preferred a penis of slightly larger circumference and length for one-time (length = 6.4 inches/16.3 cm, circumference = 5.0 inches/12.7 cm) versus long-term (length = 6.3 inches/16.0 cm, circumference = 4.8 inches/12.2 cm) sexual partners. These first estimates of erect penis size preferences using 3D models suggest women accurately recall size and prefer penises only slightly larger than average.
but different races have sexually selected different averages, even by nation? So the mean IS the ideal.
The association between penis size and sexual health among men who have sex with men
Larger penis size has been equated with a symbol of power, stamina, masculinity, and social status. Yet, there has been little research among men who have sex with men assessing the association between penis size and social-sexual health. Survey data from a diverse sample of 1,065 men who have sex with men were used to explore the association between perceived penis size and a variety of psychosocial outcomes. Seven percent of men felt their penis was “below average,” 53.9% “average,” and 35.5% “above average.” Penis size was positively related to satisfaction with size and inversely related to lying about penis size (all ps < .01). Size was unrelated to condom use, frequency of sex partners, HIV status, or recent diagnoses of HBV, HCV, gonorrhea/Chlamydia/urinary tract infections, and syphilis. Men with above average penises were more likely to report HPV and HSV-2 (Fisher’s exact p <or= .05). Men with below average penises were significantly more likely to identify as “bottoms” (anal receptive) and men with above average penises were significantly more likely to identify as “tops” (anal insertive). Finally, men with below average penises fared significantly worse than other men on three measures of psychosocial adjustment. Though most men felt their penis size was average, many fell outside this “norm.” The disproportionate number of viral skin-to-skin STIs (HSV-2 and HPV) suggest size may play a role in condom slippage/breakage. Further, size played a significant role in sexual positioning and psychosocial adjustment. These data highlight the need to better understand the real individual-level consequences of living in a penis-centered society.
so men with big wangs, at least the ‘gay’ ones, are rendering themselves infertile or birth defect risks by sharing it around. Y NO str8 studies?
Female Orgasm and Overall Sexual Function and Habits: A Descriptive Study of a Cohort of U.S. Women
Results: FSFI scores, which were calculated for the 230 women who reported having had a steady male sex partner in the preceding 6 months, showed that 41% of the 230 women were at risk for female sexual dysfunction (a cutoff less than 26.55) and 21% were dissatisfied with their overall sexual life. Almost 90% of the overall cohort reported good emotional contact with their partner, that their partner was willing to have sex, satisfaction with the partner’s penis size (wherever applicable), and good erectile function and ejaculatory control of their partner (wherever applicable). 81% of the overall cohort claimed to be sexually active. Around 70% (70-72) did reach orgasm frequently, but around 10% never did so. Vaginal intercourse was reported by 62% of the overall cohort as the best trigger of orgasm, followed by external stimulation from the partner (48%) or themselves (37%). External stimulation was reported to be the fastest trigger to orgasm.
Clinical implications: The knowledge on how women reach orgasm and how it is related to the partners’ willingness to have sex and other factors can be incorporated in the clinical work.
Strengths & limitations: The use of a validated questionnaire and the relative large number of participants are strengths of the study. Limitations are the cross-sectional design, the lack of a sexual distress measure, and a possible selection bias.
Conclusion: Most women in the overall cohort were satisfied overall with their sexual life and partner-related factors, even though 41% (of those who cited a steady sex male partner) were at risk for female sexual dysfunction. Most women did reach orgasm through different kinds of stimulation. Correlation was good between preferred and performed sexual activities and positions. Shaeer O, Skakke D, Giraldi A, et al. Female Orgasm and Overall Sexual Function and Habits: A Descriptive Study of a Cohort of U.S. Women. J Sex Med 2020;17:1133-1143.
I bet the inorgasmic had ‘cut’ men, who produce fewer orgasms in all women in studies.
Impact of penile size on male sexual function and role of penile augmentation surgery
Penile augmentation is an ongoing debate. Emerging evidence proves a relationship between dissatisfaction with penile size and sexual dysfunction. Despite a widespread belief of the value of penile size, and the prevalent complaint of a “small penis,” penile augmentation still stands short of addressing the demand. This report highlights the studies added to medical literature on penile augmentation in 2011-2012, including data on normal penile dimensions and the expected dimensions for a normal person, determination of the prevalence of the dissatisfaction with penile size, its effect on erectile function, female partner’s opinion of the value of penile size, as well as further experience with augmentation techniques.
It’s mental, they need therapy. Also, I see the joke.
Why Are Men Satisfied or Dissatisfied with Penile Implants? A Mixed Method Study on Satisfaction with Penile Prosthesis Implantation
The issue is mental?
Content analysis revealed four main themes for men’s satisfaction with the PPI: (i) psychological factors were reported 54 times (n = 54) and included positive emotions, self-esteem, confidence, enhancement of male identity, major live change, and self-image;
(ii) improvement of sexual function was reported 54 times (n = 54) and referred to achievement of vaginal penetration, increase of sexual desire, sexual satisfaction, penis size, and improvement of erectile function;
– satisfaction is a female metric? ED is mental?
(iii) relationship factors were reported 11 times (n = 11) and referred to relationship improvement and the possibility of giving pleasure to the partner; and (iv) improvement in urinary function (n = 3).
– not possible with implants, all surgeries produce numbness and see above study
A preliminary investigation of a novel method to manipulate penis length to measure female sexual satisfaction: a single case experimental design
Results: On average reducing the depth of penetration made a statistically significant 18% reduction of overall sexual pleasure with an average 15% reduction in length of the penis. The longer the erect penis, the less likely the rings had an impact on sexual pleasure. There was however a range of individual responses with a minority of women reporting that reducing the depth of penetration was more pleasurable on some occasions.
– the G spot?
Conclusions: Size may matter in women in a healthy stable relationship when there is penile shortening. Because of the small number of couples and men with an apparently long penises, our results are preliminary, and we welcome replication in a larger sample with a more diverse range of penile lengths. Our results should not be misinterpreted as meaning that increasing penile length will increase sexual pleasure in women.
shortening with age, and no, supplements don’t help
Non-pharmacological and non-surgical strategies to promote sexual recovery for men with erectile dysfunction
-quit the porn? sigh
the tobacco-lung cancer connection of this generation history will know you as a bunch of wankers
Erectile dysfunction (ED), the most commonly reported sexual problem for men,
in dick-mutilated, porn-addled America? also Israel?
reduces the quality of life for both patients and their partners. Even when physiologically effective, long-term adherence to ED treatments is poor.
– it’s behavioral…
We review here the implication of having patients’ partners involved in ED treatment, starting with treatment selection. We suggest that having partners engaged from the outset may promote an erotic association of the treatment with the partner, i.e., conceptually linking the aid to the sexual pleasure that the partner provides.
– quit ‘supernormal stimuli’ aka lechery, your dopamine is fried?
We hypothesize that this erotic association should enhance the sexual aid’s effectiveness and might potentially help improve long-term adherence. The primary focus of this review, though, is non-pharmacological and non-surgical options for maintaining sexual activity for men with ED. Though not ED treatments per se, anecdotal data suggest that these options may be effective for some patients and their partners in regaining a satisfying sex life. The aids discussed include external penile prostheses, penile sleeves, and penile support devices.
-easier just to give up the sexual visual crack, surely?
These devices can allow men to participate in penetrative sexual intercourse despite moderate to severe ED.
-aging happens, it’s natural selection
External penile prostheses can be personalized so they match in size and shape a man’s normal full erection.
– that is sad
Penile sleeves can similarly be customized with a lumen that fits best a patient’s penis for optimal tactile stimulation.
– so they’re fucking a toy? …. not the man…..
We review how multi-sensory integration can enhance sexual arousal for men who use such devices, allowing them to achieve orgasm despite intractable ED.
-it’s a silicon sock
Patients are not always advised within ED clinics about these options nor why and how they can facilitate non-erection dependent sexual recovery. Clinicians need to be aware of these devices and their positive attributes, so they can objectively counsel and encourage couples to explore their use as an alternative to more invasive treatments. The most commonly promoted non-medical ED aid offered to patients is the vacuum erection device. We discuss how erections achieved with the vacuum erection device have a “hinge effect”, that is an underappreciated barrier to the effectiveness of the erection. With a hinged erection, the penis points downward rather than upward. We show how the normal kinematics of the penis during coitus is not strictly linear (i.e., not uniaxial; not just in-and-out), and is impeded by hinging. Positional adjustment, such as the receptive partner being on top, may help overcome this problem for some couples. Lastly, we suggest that, in the case where ED can be anticipated from a pending medical treatment, such as a prostatectomy, pre-habilitative approaches may potentially improve adherence to sexual aid use in the long-term. In conclusion, non-pharmacological and non-surgical options for sexual recovery are available. Scientific studies on the effectiveness of these interventions in restoring satisfying sex are warranted.
Is Internet Pornography Causing Sexual Dysfunctions? A Review with Clinical Reports
Traditional factors that once explained men’s sexual difficulties appear insufficient to account for the sharp rise in erectile dysfunction, delayed ejaculation, decreased sexual satisfaction, and diminished libido during partnered sex in men under 40.
This review (1) considers data from multiple domains, e.g., clinical, biological (addiction/urology), psychological (sexual conditioning), sociological; and (2) presents a series of clinical reports, all with the aim of proposing a possible direction for future research of this phenomenon. Alterations to the brain’s motivational system are explored as a possible etiology underlying pornography-related sexual dysfunctions. This review also considers evidence that Internet pornography’s unique properties (limitless novelty, potential for easy escalation to more extreme material, video format, etc.) may be potent enough to condition sexual arousal to aspects of Internet pornography use that do not readily transition to real-life partners, such that sex with desired partners may not register as meeting expectations and arousal declines. Clinical reports suggest that terminating Internet pornography use is sometimes sufficient to reverse negative effects, underscoring the need for extensive investigation using methodologies that have subjects remove the variable of Internet pornography use. In the interim, a simple diagnostic protocol for assessing patients with porn-induced sexual dysfunction is put forth.
– it’s a supernormal stimulus designed to make you impotent, it’s a delusion that alters the brain
Based on the findings of historical studies cited earlier, older men would be expected to have far higher ED rates than the negligible rates of younger men [2,7]. However, in just a decade, things changed radically. The 2001–2002 rates for older men 40–80 were about 13% in Europe . By 2011, ED rates in young Europeans, 18–40, ranged from 14%–28% .
duh, porn addicts also look older, do that study
Finally, another 2015 study on men (mean age approximately 36), reported that ED accompanied by a low desire for partnered sex is now a common observation in clinical practice among men seeking help for their excessive sexual behavior, who frequently “use pornography and masturbate” .
aimed at Europeans, squarely
make them hypersexual, extreme r, and re-productively useless
The sole qualification for being a masculine man is having a functioning dick but they cuck for the industry ruining their lives.
The Potential Associations of Pornography Use with Sexual Dysfunctions: An Integrative Literature Review of Observational Studies
This paper reviews the associations between pornography use and sexual dysfunction based on evidence from observational studies. The existing data in this regard mostly derive from cross-sectional investigations and case reports. There is little if no evidence that pornography use may induce delayed ejaculation and erectile dysfunction, although longitudinal studies that control for confounding variables are required for a full assessment.
– decades later, we’ll be dead demographically…
The associations between pornography use and sexual desire may differ between women and men although the existing data is contradictory and causal relationships cannot be established. The strongest evidence is available for the relation of pornography use with decreased sexual satisfaction, although the results of prospective studies are inconsistent. The paper outlines future research prospects beneficial in understanding the nature of associations between pornography use and sexual dysfunctions in men and women.
Online Porn Addiction: What We Know and What We Don’t—A Systematic Review
In the last few years, there has been a wave of articles related to behavioral addictions; some of them have a focus on online pornography addiction. However, despite all efforts, we are still unable to profile when engaging in this behavior becomes pathological.
– publishing bias
Common problems include: sample bias, the search for diagnostic instrumentals, opposing approximations to the matter, and the fact that this entity may be encompassed inside a greater pathology (i.e., sex addiction) that may present itself with very diverse symptomatology.
= excuses so you don’t get later sued
Behavioral addictions form a largely unexplored field of study, and usually exhibit a problematic consumption model: loss of control, impairment, and risky use. Hypersexual disorder fits this model and may be composed of several sexual behaviors, like problematic use of online pornography (POPU). Online pornography use is on the rise, with a potential for addiction considering the “triple A” influence (accessibility, affordability, anonymity). This problematic use might have adverse effects in sexual development and sexual functioning, especially among the young population. We aim to gather existing knowledge on problematic online pornography use as a pathological entity. Here we try to summarize what we know about this entity and outline some areas worthy of further research.
Risky sexual behaviors typically occur when a person is sexually motivated by potent, sexual reward cues. Yet, individual differences in sensitivity to sexual cues have not been examined with respect to sexual risk behaviors. A greater responsiveness to sexual cues might provide greater motivation for a person to act sexually; a lower responsiveness to sexual cues might lead a person to seek more intense, novel, possibly risky, sexual acts. In this study, event-related potentials were recorded in 64 men and women while they viewed a series of emotional, including explicit sexual, photographs. The motivational salience of the sexual cues was varied by including more and less explicit sexual images. Indeed, the more explicit sexual stimuli resulted in enhanced late positive potentials (LPP) relative to the less explicit sexual images. Participants with fewer sexual intercourse partners in the last year had reduced LPP amplitude to the less explicit sexual images than the more explicit sexual images, whereas participants with more partners responded similarly to the more and less explicit sexual images. This pattern of results is consistent with a greater responsivity model. Those who engage in more sexual behaviors consistent with risk are also more responsive to less explicit sexual cues.
– it’s r/K
trans. r-types seem to have dulled sexual perception (abnormal) so over-estimate opportunity in compensation
Studies are in their description, duplicated below.
Reminder: ED is the PC term for impotence – and a common side effect of porn addiction. How about studying penis size between promiscuous men and chaste ones? They’d never publish it. I’d read it. Why are we not funding this?
The role of exposure to phthalates in variations of anogenital distance: A systematic review and meta-analysis
Environmental chemicals such as phthalate esters may have adverse effects on anogenital distance (AGD), but the evidence in both genders has not been reviewed systematically. The objective of the present study is to conduct a systematic review and meta-analysis of studies that analyzed the relationship between exposure to phthalates and AGD. English papers published up to March 2018 were searched in PubMed, Scopus, Clarivate-Web of Science, and Google scholar. We applied fixed-effects models to calculate pooled beta coefficient [β]. In the case of heterogeneity, random-effects models were used. Using the comprehensive search strategies, 313 papers were identified and after screening, 10 of them were included in this study. In primary analyses, we found that exposure to phthalates was not associated with short AGD (β = -0.11; 95% CI, -0.27, 0.06; I2 = 0%). However, results of subgroup analyses indicated that in boys, the sum of di-2-ethylhexyl phthalate (∑DEHP) metabolites had significant association with the risk of shortened anopenile distance (AGDAP) (β = -0.915, 95% CI: 1.629, -0.2) and anoscrotal distance (AGDAS) (β = -0.857, 95% CI: 1.455, -0.26). In addition, urinary monobutyl phthalate (MBP), monoethyl phthalate (MEP), and monoisobutyl phthalate (MiBP) were associated with short AGDAP. We also observed significant association between monobenzylphthalate (MBzP) and anofourchette distance (AGDAF) in girls. Our study provided findings on significant association of exposure to ∑DEHP metabolites, MBP, MEP, and MiBP with shortened AGDAP in boys. The mechanisms of phthalates effect on AGD may involve receptors and enzymes involved in steroidgenesis, negative influence on Leydig cells, cell proliferation, gonocyte cell numbers, and testosterone production.
I have studies and other things I could post, but haven’t yet. It’s a red herring, presently. I see a raft of new comments. I’m not checking or approving them, I miss many important ones anyway (somebody deletes them). If people won’t listen for years, talking now is quite futile.
I’ve mentally checked out of their endgames long ago, truth be told, nobody knows how this story ends. I used to care, I remember the impulse to interfere well. The do-gooder instinct, now I abhor it with equal fervor. It struck me how ill-advised this impulse is, when dealing with certain types of ruthless, dangerous people. It was pointed out to me that I was too soft and it could, could be the death of me. It took years but… I corrected this deficit. That’s why I’m still here, unlike many before me, and doing as I please. QED. I’m self-righteous, not stupid. Many rigid principled people end up dead and I’d rather not be among them. There is no honour dying nameless in a gutter.
I’m not important anyway. I think people know if they’re important. I’m not. I’m not your messiah or your prophet, I put puzzle pieces together quite well, like everyone else. If I knew too much I’d be six foot under, frankly, because I’m not related to enough VIPs. I think of myself as probable back-up flesh, back-up organs to some …. well… persons. Let’s leave it at that. Rare blood type.
I’m much more useful to the world alive and talking, in my own way. I do know key things, sometimes. There is no binary choice to be a good person or not, I was wrong. Too many Disney films, maybe. Idealism is a …problem. There are ways to be good and some are strategically wiser than others. Game theory. Try to help All and die, vainly, or try to help some and live, well. Simple when laid out. To suffer needlessly is a vanity.
The cardinal rule is never be the most ignorant party, keep your ear to the ground and listen to any whispers, however slight. Better to have subtle forewarning than none at all. If you don’t know, anyone can take advantage of that ignorance, however pure your intent. As a mentor told me years ago “You’re good, but you’re no good to anyone dead”. It shocked me. It was necessary. I am a believer in choice and liberty, which is to say agency. If the People want Hell on earth, let them have it with both barrels, I say. If they walk to the slaughter willingly, let them die. God’s mind is the same, let them take the Mark, if they so, loudly, wish. Millennia of warning foretold. A suicidal populace is already done for. Mouse utopia narcissism, at the cost of fertility. It takes years to show. Is society so far gone? Is the average mind a reprobate, fully deserving of their longsuffering? I haven’t the foggiest. I hope not. I have faith in God, if not men. Do not put faith in men.
We live in desperate times, full of desperate minds. Too much noise. What’s Sodom got on present America? Really. List the sins. I tried. Hell isn’t necessary, deep down evil people know what they deserve and seek out oblivion in their self-destruction. Let them be. Sometimes natural selection is self-selecting. If you conform to evil, I have seen evil conform to thee. Jesus himself wouldn’t spare the unrepenting. I don’t know better, that’s for sure. Do I know more? Nah. I opt for humility over the popular, worldly conceit. I don’t need people to agree with me, they’ll see.
I’m not cutting off my nose, hoping to spit another’s face. Reality bites eventually.
It’s domestic. They should be up in the Hague. Nuremberg 2.0. Armbands when? Subhuman rights.
No food shopping, /wallstreetbets should demolish their stock (Tesco etc.). No getting a job, going to hospital (ER too!), going to Gov buildings (e.g. to pay your bills) and no skipping the country when they round you up as a ‘refuser’ or ‘denier’. Online shopping will rocket, until that is twisted to somehow need it too, how can preventing people from buying food be made legal? Holodomor 2.0? The pubs and shops are ‘only following orders’…. like that works.
PAPERS PLEASE will be SCAN YOUR ID HERE. Never scan yourself, this isn’t a farm.
Phone problems > ID implant. Slippery slope is really coercion, a vitiation of any consent obtained under false pretenses like misinfo or disinfo.
Normies: Do you think this will end with a one-off ‘experimental’ (no sue) ‘jab’? No, 3-4 per year, ‘unforeseen complications in young people’ (infertility, NS paralysis/neurodegeneration, mortality) and multiple other ‘jabs’ you’ll be legally forced to get with this precedent. The MPs do NOT own my nor your bodies. I TOLD YOU ALL SO. FOR YEARS NOW.
There are lots of stories about what this vaccine will do to people a log time down the road, from sterilisation to cancer, its all too scary knowing what we know
Immunologist: Pfizer, Moderna Vaccines Could Cause Long-Term Chronic Illness
February 15, 2021
In new research published in Microbiology & Infectious Diseases, immunologist J. Bart Classen warns the mRNA technology used in the Pfizer and Moderna COVID vaccines could create “new potential mechanisms” of adverse events that may take years to come to light.
Back in 1999, leading U.S. Food and Drug Administration (FDA) official Dr. Peter Patriarca contended that modern advances in vaccine technology were rapidly “outpacing researchers’ ability to predict potential vaccine-related adverse events.” Patriarca mused that this could lead to “a situation of unforeseen and unpredictable vaccine outcomes.”
In a new research article published in Microbiology & Infectious Diseases, veteran immunologist J. Bart Classen expresses similar concerns and writes that “RNA-based COVID vaccines have the potential to cause more disease than the epidemic of COVID-19.”
For decades, Classen has published papers exploring how vaccination can give rise to chronic conditions such as Type 1 and Type 2 diabetes — not right away, but three or four years down the road.
In this latest paper, Classen warns that the RNA-based vaccine technology could create “new potential mechanisms” of vaccine adverse events that may take years to come to light.
Classen’s study establishes the potential for the messenger RNA (mRNA) vaccines developed by Pfizer and Moderna to activate human proteins to take on “pathologic configurations” — configurations associated with chronic degenerative neurological diseases. Although his specific interest is in prion diseases (conditions associated with misfolded versions of normal proteins), Classen also outlines a handful of other mechanisms whereby RNA-based vaccines could give rise to “multiple other potential fatal adverse events.”
Ensuring that patients clearly understand risks — including known risks as well as potential unknown risks — is an important component of the informed consent process. This is all the more true when the intervention is experimental and lacks long-term safety data, as is the case with the Pfizer and Moderna vaccines against COVID-19. The FDA authorized the two vaccines for widespread emergency use based on just two months of clinical trial data.
Unfortunately, it is not unusual for researchers’ communication of risks to be perfunctory. In October, researchers at New York University and Tulane reported that the information communicated to participants in the coronavirus clinical trials about a worrisome problem known as pathogenic priming was “sufficiently obscured” as to make “adequate patient comprehension” of risks “unlikely.”
It would be interesting to know what those researchers would say about Classen’s blunt conclusion that “Approving a vaccine, utilizing novel RNA technology without extensive testing is extremely dangerous.”
Those contemplating COVID injections may be ignoring potential risks at their own peril.
Microbiology & Infectious Diseases, COVID-19 RNA Based Vaccines and the Risk of Prion Disease, J. Bart Classen, MD.
Shouldn’t you be in a kitchen, not talking? Also, is nobody else able to tell just by the face? Why do you think YT pushes her videos? Elizabeth comments “I used to get ads for her channel all the time and I literally blocked her channel and they still didn’t go away” I wonder who’s behind that? They all have the same pinched snippy solid 5 look because they’re all so inbred.
Bold for a woman with a hunky manjaw, narrow eyes and a tranny double dip chin to comment on white women being unnatural?
I agree with the initial premise that they’re arguing in bad faith, but if you have to strawman SJWs, your arguments must be pretty shit. There’s a self-righteous Pharisee vibe with those women, either some codependent disorder (they always cheat, eventually) or NPD, where a man/woman gets incredibly …. SMUG, all caps, after marriage, like /notlikeothergirls like getting married is a personal accomplishment or testament to the quality of your vagina. The Bible says none of that, Jesus never married? Are you more Christian than Jesus? Bloody Hell. They wear thot makeup, slutty perfumes, attention-seek in videos and show their shoulders and legs, while ‘judging’ other women for doing the same in a style they dislike. That sounds like envy to me. Proto-Karen larping as Christian. Jesus warned us about the Pharisees, fixated on self-righteous indignation and I want nothing to do with that arrogance. Most left-wingers are mild and leave me alone so I’m fine with them just existing, they’re not all Marxist bastards trying to stab me with my own crucifix nor smother me with a burqa – but I feel like Abby would, for a distinctly Talmudic interpretation of modesty and fucking through a hole in a bedsheet. These glorified CHINO sugar babies are trying to act like they’re all oppressed when they’re not.
WHO is oppressing you?
woe is me I married a rich man – okay Ivanka?
Oh, somebody DARE to disagree with your lifestyle? So? Grow up. They’re not trying to legally block you nor impose theirs. Disagreement is not HATE. I repeat: DISAGREEMENT DOESN’T MEAN THEY HATE YOU. Then they wonder why other women swerve them. You’re not high IQ, you’re Becky. You’re so bland you’re claiming oppression from a nice house paid for by hubby, after moving out of a nice house paid by Daddy. You are not an adult. You’re a womanchild. Married or not. Getting married doesn’t mean you ever grew up, or needed to. I know a womanchild in her seventies, it is horrifying. Everyone avoids her. She sounds like a teenager. People are ignoring them like they do Big Red – because they’re making up bullshit to complain about. Woe is me pity parties are sad from grown men and women. At some point as an adult you become 100% responsible for your own life. Without Christianity, they’d have no life and no personality and that isn’t healthy. Without being married, what would they have to talk about? Nobody forced you, don’t be a Boomer. Muh cultural norms r oppressing me! Please. I know we like to bash lefties here but there’s a growing number of fake K-types, paper Ks with the same toxic patterns and I am not turning a blind eye to it. Such people make awful parents, we need to smoke out their entitled bullshit. Like they’ll criticise women for ‘not making the effort’ but turn a blind eye to the fact women may not be safe walking around in a dress and heels in most multicultural areas of the West. Most white people don’t have the money to live in gated communities.They also need to leave the house sometimes! The peasants buy their own food! They are just as out of touch as Huffington Post writers. Women don’t always have men around to keep them safe like ‘Daddy’, or self-defence weapons legally, and rape or acid attacks or murders happen all the time. The victim blame is disgustingly anti-white, that’s what Marxists do! One happened the other week by a police officer so we won’t go running to them either. It’s like talking to a five year old. Meanwhile they have this pseudo-mature judgey intellectualism fallback like ‘oh I read War & Peace’. And? This isn’t school. You cannot social commentary the real world when you’ve never lived in the real world. I blame the simps, whom they attention-whore for. It’s like political onlyfans. They never say anything critical of the men (who are not their husband!) paying them; not about deadbeats, adultery or manwhores. God forbid you have Christian standards, so they are PC, just conservathot PC. They are worldly. These people represent nobody but their own ego. Christian white women aren’t a hivemind like SJWs, sorry? We don’t do cult of personality. It’s idolatry.
comment, Francois writes “The natural way for humans to live is in nomadic hunter-gatherer tribes of around 20 to 30 people, with a life expectancy of about 35, infant mortality of around 50% and being on the menu of most large predators living alongside us. I doubt very much Abby would be ok with returning to that level of “natural”.”
These women are making people like me look stupid and I’m pissed about it. I had a hard life and earned everything I have and these vapid cunts swan in complaining that ‘feminists’ (they mean prettier white women) are stopping them from living in some of the best economic conditions in all of human history, in some of the richest empires/countries the world has ever seen and they DARE blame being a Christian.
Bitches, we used to get gang-raped by Vikings and burned alive by Romans! You are NOT oppressed.
YOU WOULD KNOW. Where may I file a complaint? They’re hardly selling their lifestyle since they STILL complain!
Kristen writes “Appealing to nature has to be the weakest argument to tell someone to live or not live a certain way.”
Off-putting classism aside (NOT EVERYONE IS RICH), why are they still working? Why are they putting off having kids? Married Catholics can’t use contraception, especially chemical abortifacients – and you know they are. I won’t name names.
comment “Abby should’ve just delete her channel because obviously she’s gaining more than her husband now. Cus no “classy woman” should do the man’s job.” ^ She’s acting like a preacher, yes. I follow no Christian channels for this reason, I just go to the Bible. They don’t actually listen to women. They talk over us. Stop gaslighting women on what they want from life. We’re not all the same, some want kids, others don’t. You don’t own her womb. She might literally die. Jesus at no point told off the women following him for ‘wasting their fertile years’ because that’s a Darwinist argument. In theology, the world will end anyway so demographics don’t matter? What genetic survival is possible, remotely, after the End Times? How are they this stupid and still alive? Then dishonesty it must be. Emma writes “The economy and high cost of living is why I can’t afford to pop out three kids by age 27. Not feminism.” Caused by globalisation, since women always worked – even in factories. There is photo and video. The problem is international hiring suppressing domestic wages since the hippies voted it all in – Immigration act in the 60s, diverse hiring etc. Nay, never discuss it! Women always worked outside the home, long before there was a home. Did they think cavewomen were sweeping a hearth? Utter midwits, eloquent but vapid. Women worked in the fields into the Victorian era. Not ‘family values’ enough? Kids worked too but ignore the ones down the mines, they were the wrong colour to care. Replacement is caused by Third world nations over-populating, nothing we do can stop it. I have covered the basic primary school mathematics.
They seem like spoiled brats imho. “It’s natural for women to stay home” So Abby would clearly be ok with the free the nipple mindset right? Like bras aren’t natural and if she’s all about being natural there’s no problem! But wait, she’s not a fan of that? Then maybe this isn’t about nature….” Exactly you can’t pick and choose cultural norms. Initially only prostitutes liked wearing underwear as a layer to show off their assets. In Jesus’ time women wore zero underwear. A cause I support. And what of male modesty? They’re so two-faced, clearly using any simp in a seventy mile radius to extract funds. Is that not prostitution?
Some people never want kids – and that’s okay. Or they can’t have them and other life purposes are fine. This is so toxic and bigoted. HBD suggests it’s better if left-wingers don’t have kids, since partisanship is genetic. The Lord works in mysterious ways and the oddly controlling nature of such women suggests they’re miserable. Just go be happy being married, surely? Sod off.
comment “I went to a high school in a wealthy neighborhood and a lot of my friends were really rich. These kids living in houses costing like 3 million would be like “But it’s not my money it’s my parents money” and I, who had grown up just above the poverty line, was like yeah, it’s your parents money, that they’re using to pay for you to go to Harvard. Your parents money that they’re paying your car insurance with. Your parents money that’s going to buy you an apartment in New York City. You can’t discount your class privilege just because you’re not getting a cut of your parents’ salary. That’s the issue that a lot of people like Abby have, they don’t see the fact that not everyone’s families can afford to take care of them or let them live at home. I honestly doubt Abby could’ve supported herself on her career alone, given the fact that she’s in an arts career and she’s probably used to a certain kind of lifestyle, so a lot of her words here feel so empty because she has the luxury of working simply because she loves her job, and not because she has to. So much ignorance had to have gone into thinking that everyone has that option.”
Anna comments on the arrogance, she is a big-nose alright: “Can’t people literally just mind their own business? The thought of carrying on the way she does, sticking my nose into other people’s perfectly happy lives makes me cringe so violently. It’s unthinkable to me. Unless someone asks me to involve myself or needs my help. That would be the only exception. It makes me so angry, the actual audacity of people like Abby. How dare they? What I do or don’t do with my uterus or my life is nobody’s business but mine. I’m sorry, rant over 😂🤣 They just infuriate me.” They must hate their life and regret their life choices, it does beg that question. Meddlesome people always do. They never discuss studies or listen to anyone outside their echo chamber, like, maternal mortality in America increased? Why would sane women with a choice there have kids now? And complaining about women not having kids when men are at record low levels of proposal and marriage? Sheer hypoagency blame-shifting. This is Gender War psyops. They are inciting hatred onto real Christians by doing this. Judge not lest ye be judged? She’s trying to put off white women from starting a family by heaping on all sorts of additional non-rules based on her controlling nature. This is a spiteful mutant, folks. False dichotomies abound.
Eva writes “I feel like conservatives are like “FEMINISM MEANS THEY WANT MEN TO BE FEMININE AND WOMEN TO BE MASCULINE” because they can’t fathom a belief system that doesn’t force anybody into categories and limitations” Yep, strawmanning. The economic arguments are easy to check, and the marriage rates… unless they want women to be single mothers? Judgy proto-Karens.
I feel like even the middle-class now doesn’t see how rich they are. Telling working class or under-class people to ‘just work harder’ doesn’t mathematically work, they didn’t have rich parents putting them years ahead in the race at every turn. Are women supposed to live at home, magically meet the right man and pray he marries them? When do they learn adult responsibilities like keeping a home? Paying bills? Never, according to these people. Sick.
American Christianity is insane Puritanism, no other Christians like them, we hate them. They’re not real Christians mostly, they’re weird on a global scale and weirdly pedantic. Like I heard they tell women not to masturbate (not men though….? Onan, anyone?) when erm, women do that in their sleep? Anatomically they need to*, to keep the muscles toned for childbirth? Did they not know, how? HOW? Did they just lie and think it was only them? Likely. We all know for certain during red week, don’t lie, and you know how showers work. To keep clean down there as a pre-teen you know what it is** and it isn’t the ‘devil’s doorbell’, God put it there for a biological reason or become a Muslim and get parts of it cut off? No? Thought not. Liars and hypocrites. Minus masturbation, men get nocturnal emissions. Do they deny that biology too? Nowhere in the Bible does it speak against masturbation prior to marriage. It speaks about other things multiple times. Nowhere that. Yanks are being weird Puritans again. Do they think this sleep effect only happens to men, are you fucking serious? You should have to pass a sexual biology class before being allowed to lawfully marry, sorry. Do you also think periods are about blood, rather than clots or uterine chunks? Midwits make us all look bad. They are educated dunces who think batting their eyelashes at losers is an argument. *Vaginal prolapse happens when you don’t, in the long run, as does vaginal atrophy in the very short-term (months). Even nuns masturbate, you dumb cunts. I read this in a Victorian gynecology textbook in a blase (yes, needs must) tone so yes our ancestors definitely knew and if you doubt that, nun’s dildos still exist in museums and such. Some were made of rubber and shot out milk. Not everyone has a dry vagina, ironically touching yourself more often would help with that and probably solve your marital woes. I’ve asked many doctors socially and they all agree, any woman who wants kids has to keep things toned down there. It is a MUSCLE. Wait, is that why so many American women are dying? Poor muscle tone > baby gets stuck > baby gets brain damage from lack of oxygen in the vagina > heart attack from strain > mother dies? I can see why ‘hysteria’ was such an issue, unique to America. Never take female health advice from a man – unless he’s literally a gynecologist. SMH.
**women have to clean their prepuce, just like men? I am worried you lack knowledge of basic hygiene over there. How else do you get the menses out of the area? Women have folds, you know? …Or do you? Like I’m genuinely fearful now.
Zelda writes “Ugh. I stayed home years ago ( I’m a grandmother now). It was a mistake. Sure, I enjoyed my child, but in the long run it wasn’t the right choice for me.” Paternal and maternal feelings vary in both sexes, why does this type push misery at home as normal, unless they are? How about preventing suffering, since the Bible advised not to wed, there’s NO Christian duty unless you ‘burn in sin’, and prevent child abuse while we’re at it? What you do to the least of these, you do to me? I hate CHINOs. Abby Shapiro isn’t even white, are we sure this Jewess isn’t larping in Marxist disguise to make us look dumb and out of touch? Are we sure? Sus. Lady bits aren’t a horror movie, they’re not inherently scary, the self-loathing for the way God made you is repulsive. If you have so much Jewish guilt, you shouldn’t have murdered Jesus. Talking down to the white working class people who make your brattish lifestyle possible is deviant. They always play the victim, every – single – time. We’re sick of being told we’re doing Christianity wrong by the people who murdered our messiah. F off forever. Every passing year, I understand the 30s better.