Diabetes caused by… make-up??!

While the thots are eating their aluminium like good little morons, there’s a great causative agent of diabetes lurking in all those shitty “highlighters” that do the same thing anyway (compare Dior ingredients to Topshop).

It’s in things everyone consumes like toothpaste.

https://www.sciencedaily.com/releases/2018/06/180620125907.htm
And candy, and food, and paper….

Is somebody trying to poison us?

Obviously.

In the mid-20th century, titanium dioxide pigment replaced highly toxic lead-based pigments. It became the most commonly used white pigment in paints and in foods, medications, toothpaste, cosmetics, plastics and paper. As a result, annual production of titanium dioxide has increased by 4 million tons since the 1960s.

Who bought it beforehand?

Who bought all the titanium before bringing it to market as ‘safer’?

According to the World Health Organization, the number of people with diabetes has quadrupled during the past four decades, affecting approximately 425 million people, with T2D comprising the majority of recorded cases. Although obesity and an aging population are still considered major factors leading to a rise in T2D cases worldwide, Heller’s study suggests that increased use of titanium dioxide may also be linked to the rapid rise in the number of people suffering from the disease.

Huh.

“The increased use of titanium dioxide over the last five decades could be a factor in the Type 2 diabetes epidemic,” Heller said. “The dominant T2D-associated pancreatic particles consist of TiO2 crystals, which are used as a colorant in foods, medications and indoor wall paint, and they are transported to the pancreas in the bloodstream. The study raises the possibility that humanity’s increasing use of TiO2 pigment accounts for part of the global increase in the incidence of T2D.”

Why are Government buildings the same damn white colour?

We know we can breathe that shit in.

Given the wide-reaching implications of his findings, Heller is keen to repeat the study, but this time using a larger sample. “We have already begun a broader study,” he said. “Our work isn’t over yet.””

You heard it here first.

Diet effects vary by race

What happened when scientists put African Americans on an African diet and Africans on an American diet

That’s right.

The lefties know.

This is why you keep contacts. Quid pro quo.

Study here: http://www.nature.com/ncomms/2015/150428/ncomms7342/abs/ncomms7342.html

Carnivore/Anglo diet kills Africans.

We should deport them from the West, for their own sake!

Teachers’ unions HATE THEM!

Need a piece of paper? You sure?

Moneyball your education.

http://www.openculture.com/free_certificate_courses

Delicious progress.

“You can’t put a price on a good education.”

You know, you were right….

Teachers will, by and large, go the way of horse-shoe makers and coachmen.

Protip: I know a lot of people who do these. Take 2 fun courses (they can be zany) for every 4 serious ones.

We’ve had at least 10 years warning for cooling

When the food shortages (like now) and eventual famines happen, place the blame where it is deserved.

Not the honest scientists but the lying journalists who didn’t cover it, illustrated.

I made dis but feel free to nick it.

http://iceagefarmer.com/docs/Science-GrandSolarMinimum/WilsonSpinCoupling.pdf

Final page, 2008 paper, Does a Spin–Orbit Coupling Between the Sun and the Jovian
Planets Govern the Solar Cycle?:

4.3 Important Consequences of the Resonance Model

Interestingly, the Sun’s solar cycle has been in the phase locked mode for the last 105 yr (1900–2005) and the indications are that it is about to suffer another phase catastrophe in the later part of cycle 24 (i.e. the solar cycle that will peak in ∼2011–2012).

If this is the case, then we should expect that in the two decades following the phase catastrophe, the world’s mean temperature should be noticeably cooler i.e. the cooling should start in the late 2010s. This claim is based on the precedent that there were noticeable decreases in the world’s mean temperature following the last two phase catastrophes.

The cool period know as the Dalton Minimum (1800–1820) that followed the phase catastrophe in the early 1790s and a similar cool period called the Victorian Minimum (1880–1900) that followed the phase catastrophe in the late 1870s.

Food supply issues incoming, based on the pure physics of THE SUN.

I’ve been following this for a while, as you can see. [unrelated data there but interesting]

http://iceagefarmer.com/docs/Science-GrandSolarMinimum/ValentinaZharkova2015.pdf

Re-post: PREDICTION OF SOLAR ACTIVITY FROM SOLAR BACKGROUND MAGNETIC FIELD
VARIATIONS IN CYCLES 21–23

I think it’s a re-post. I post so many studies it’s impossible to tell. I’d rather re-post than forget something important you may wish to read (even to disagree).

Listed, 2017: Food security

https://disenchantedscholar.wordpress.com/2017/11/23/where-should-white-people-move/

along with:

  • “The weather isn’t trying to kill ya.”
  • “More concerned with their own survival than the fucking pandas.”

The people who wanna live on an island in a hurricane region like the Pacific are morons. People used to live on the tiny islands. They died. That’s why it’s so cheap. THEY ALL DIED.

Eat your aluminium, thots

That’s right, smear on a lipstick full of shimmery bullshit. Including lead (and wonder why girls are depressed). [Literally eating lead.]

The Victorians ate arsenic but this is worse. Arsenic temporarily improved the complexion.

Using the term ‘mica’ for what’s essentially powdered foil suggests women wouldn’t want it if they knew what it was.

It isn’t like there’s a connection between dissected and tested Alzheimer’s plaques and aluminium deposits, is there?

Why are the Boomers raised on cans of this stuff seeing an explosion in Alzheimer’s rates?

As always, at first, go to the ‘officials’. Find out the Party Line.

https://www.alzheimers.org.uk/about-dementia/risk-factors-and-prevention/metals-and-dementia
Although aluminium has been seen in amyloid plaques”

K.

“there is no solid evidence that aluminium is increased in the brains of people with Alzheimer’s disease.”
Whew, what cognitive dissonance.
It’s literally forming those plaques that ARE the disease but nbd?

Solid evidence – like a solid chemical analysis of solid plaques in the solid brains of solid dead people with Alzheimer’s?

Look at the hustle to move those goalposts.

https://www.sciencedaily.com/releases/2007/08/070813185007.htm
“Because some people with the disease had aluminum deposits in their brains, it was thought that there was a direct connection…”
Well… yeah.
That’s proof.
There’s no negative evidence possible for that to stop being proof.

It’s literally in their brains.

“However, after many years of study, no conclusive evidence links aluminum to neurodegenerative disease”
Bullshit, 2007.

I detest scientism. Look at these papers and tell me what’s ‘conclusive’.

(Apart from donations of mega-corps to Super PACs to prevent this mega-lawsuit).

“A multi-institutional team of researchers has defined for the first time how metal ions bind to amyloid fibrils in the brain in a way that appears toxic to neurons. Amyloid fibrils are linked to the development of neurodegenerative diseases such as Alzheimer’s, Parkinson’s and Creutzfeldt-Jakob. Although metal ions, most notably copper, can bind to amyloid in several specific ways, the researchers found that only one way appears toxic.”

But how, ‘critics’ argue, could this possibly occur? What’s the mechanism? Nobody has proven a mechanism….?

2011:

“Link between Aluminum and the Pathogenesis of Alzheimer’s Disease: The Integration of the Aluminum and Amyloid Cascade Hypotheses”

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3056430/

“In particular, the link between aluminum and Alzheimer’s disease has been the subject of scientific debate for several decades. However, the complex characteristics of aluminum bioavailability make it difficult to evaluate its toxicity and therefore, the relationship remains to be established.”

Uhuh. Isn’t that your job?

If it’s impossible, why are you getting paid?

“On the contrary, aluminum is a widely recognized neurotoxin that inhibits more than 200 biologically important functions and causes various adverse effects in plants, animals, and humans.”

True.

Mounting evidence has suggested that significance of oligomerization of β-amyloid protein and neurotoxicity in the molecular mechanism of AD pathogenesis. Aluminum may play crucial roles as a cross-linker in β-amyloid oligomerization.

Here, we review the detailed characteristics of aluminum neurotoxicity based on our own studies and the recent literatures. Our aim is to revisit the link between aluminum and AD and to integrate aluminum and amyloid cascade hypotheses in the context of β-amyloid oligomerization and the interactions with other metals.”

But HOW could this POSSIBLY operate? – intellectually dishonest douches.

Note the prestige of journal. International Journal of Alzheimer’s Disease, those hacks.

But on a MOLECULAR level…. – douchecanoes.

https://www.onlinelibrary.wiley.com/doi/10.1002/jms.739
“Quantification of the Aβ peptide in Alzheimer’s plaques by laser dissection microscopy combined with mass spectrometry”

It’s behind a paywall but it’s there. Check the academic pirate bay.

http://orthomolecular.org/library/jom/2000/articles/2000-v15n01-p021.shtml
It’s been known for a while.
2000: “Aluminum has been identified as a neurotoxin for over 100 years.”

So stop using it in food, cosmetics, deodorant, cookware and cans?

Their use actually came in well after it was a known neurotoxin. Because that isn’t dodgy.

Reference for that sentence:

Doelken P:Naunynschmiedeberger. Arch Exp Pathol Pharmakol 40: 58-120 cited by Crapper McLachlan DR., Lukiw WJ, Kruck TPA, Aluminum, altered transcription, and the pathogenesis of Alzheimer’s disease. Environ Geochem Health, 1990; 12(1-2): 103-114.

Link for that: https://link.springer.com/article/10.1007/BF01734059

Opening paragraph.

“The etiology of some, if not all, cases of Alzheimer’s disease is linked to a mutation in the proximal portion of the long arm of chromosome 21∶21q11.2 → 21q22.2. While the functional consequences of the mutation are unknown, we speculate that one consequence of the mutation is loss of the natural barriers and intracellular ligands for aluminum. As a result, aluminum gains access to several brain sites including the nuclear compartment in certain neurons of the central nervous system.”

I know both my shit and my bullshit.

1990. Who owns the aluminium? That’d be a fun tour of genocide.

https://www.onlinelibrary.wiley.com/doi/abs/10.1002/ana.410310310
1992 laser study, note prestige of journal.

What the fuck do they know, right, reddit?

“Selective accumulation of aluminum and iron in the neurofibrillary tangles of Alzheimer’s disease: A laser microprobe (LAMMA) study”

“In addition, probe sites directed to neurons identified in snapfrozen cryostat sections from 2 subjects with Alzheimer’s disease revealed similar spectra with prominent aluminum‐related peaks, confirming that our findings are not related to exogenous contamination through fixation, embedding, or other procedures prior to analysis. This study further confirms the association of aluminum and neurofibrillary tangle formation in Alzheimer’s disease.”
1992.

But I guess I’m just scared of non-stick pans, where’s the evidence?

Pig brains revived hours post-mortem

But go ahead, be an organ donor!

https://www.sciencedaily.com/releases/2019/04/190417132805.htm

You won’t feel a thing!

“Circulation and cellular activity were restored in a pig’s brain four hours after its death, a finding that challenges long-held assumptions about the timing and irreversible nature of the cessation of some brain functions after death.”

Long-held?

These are the people you trust to know when you’re actually dead.

They might as well be informed by entrails.