Results: The analysis included approximately 1.6 million live births and 1749 stillbirths. In the unadjusted model, compared with two white parents, black/black and black/white couples had a significantly higher risk of fetal death. When all demographic, social, biological, genetic, congenital, and procedural risk factors except gestational age and birth weight were included, the odds ratios (OR) were all still significant.Black/black couples had the highest level of risk (OR 2.11, CI 1.77-2.51), followed by black mother/white father couples (OR 2.01, CI 1.16-3.48), and white mother/black father couples (OR 1.84, CI 1.33-2.54). Virtually all of the higher risk of fetal death was explainable by higher rates of low birth weight and prematurity.
Conclusions: Mixed race black and white couples face higher odds of prematurity and low birth weight, which appear to contribute to the substantially higher demonstrated risk for stillbirth. There are likely additional unmeasured factors that influence birth outcomes for mixed race couples.
53,293 deliveries occurred during the time period, of which 329 resulted in a stillbirth (6.2 per 1,000 births). Compared to White women, non-White ethnicity was associated with a doubling of the odds of stillbirth (aOR for Black women 2.15, 95% CI 1.56-2.97; and for South Asian women 2.33, 95% CI 1.42-3.83).
Obese women had a trend towards higher odds of stillbirth compared to women of recommended BMI (aOR 1.38, 95% CI 0.98-1.96), though this was not significant (p 0.07). < aka no excuse there
Both higher parity (≥2 compared to para 1) and hypertension were associated with a higher odds of stillbirth (parity ≥2 aOR 1.65, 95% CI 1.13-2.39; hypertension aOR 1.84, 95% CI 1.22-2.78) but there was no evidence that area deprivation or maternal age were independently associated with stillbirth in this population. < aka no excuse there either
There was some evidence of effect modification between ethnicity and obesity (p value for interaction 0.06), with obesity a particularly strong risk factor for stillbirth in South Asian women (aOR 4.64, 95% CI 1.84-11.70).
“There was a high prevalence of stillbirth in this multi-ethnic urban population. The increased risk of stillbirth observed in non-White women remains after adjusting for other factors.
Our finding of possible effect modification between ethnicity and obesity suggests that further research should be conducted in order to improve understanding of the interplay between ethnicity, obesity and stillbirth.”
but you just said obesity was non-sig?
And the only reason to do so has now gone.
“How is it that minority infants are still unhealthy if their parents are smoking less than White parents? This is probably due to genetics and environment as well. A Dutch study compared the various rates of smoking among different ethnicities/races, “Since maternal smoking during pregnancy is quite equally distributed among the ethnic populations, it does not contribute much to the observed differences in birthweight.”(34) That study compared native Dutch people and immigrants…..”
“….As one can see, the amount of stillbirths was higher for the interracial couples. This was especially true of White father/Black mother relationships, but the White mother/Black father rates were still far worse than the rates of stillbirth for White couples. The outcomes for mulatto infants was at the detriment of the surveyed White population.(10)
What are the specifics to this horrible process? For this, we have to understand that having mixed genetic ancestry through mtDNA and yDNA perpetuates this happening. Let’s look at one of the best studies that has been published on this subject: “Divergent Patterns of Mitochondrial and Nuclear Ancestry Are Associated with the Risk for Preterm Birth.” This study investigated the unusually high rates of preterm birth among African Americans: “Haplogroup-defining polymorphisms are not merely markers of ancestry, and have consequences for mitochondrial function, including the regulation of mitochondrial gene expression.”(20)
Some might be surprised to know that many African Americans have large amounts of European ancestry. The European ancestry that African Americans have was distributed from White males when slavery was still legal.(20) To put it another way, having ancestry from vastly different ancestors contributes to these genetic predispositions. It is necessary to demonstrate this with a principal component analysis. The turquoise dots below represent the African population. As one can see, the African American population is represented by a long cline between European American (CEU) and African (YRI) samples. (24)
The majority of mitochondrial haplogroups (mtDNA) that African Americans have are African, since these come from African women originally, “… individuals with L, U6, or U5b1 haplogroup mtDNA and primarily African nuclear ancestry were defined as having low levels of divergent ancestry, whereas individuals with non-African haplogroups and high degrees of African nuclear ancestry had high levels of divergent ancestry.”(20) These researchers looked at any potential confounding variables that would skew results for the African American population. They examined a number socioeconomic variables such as years of school taken, income, etc. According to the researchers, “There was no detected statistical association between divergent ancestry and any of these factors.” (21)”
“But White people aren’t marrying out as much as other ethnic groups. Therefore, in the rare instance that a White person has a non-White partner, it is unlikely that these interracial couples are being treated in a less-preferable way by their families:
“…Whites are somewhat less willing to marry and bear children interracially than to date interracially. These attitudes and behaviors are related to warmth toward racial outgroups, political conservatism, age, gender, education, and region. Third, White women are likely to approve of interracial relationships for others but not themselves, while White men express more willingness to engage in such relationships personally, particularly with Asians.
However, neither White men nor White women are very likely to actually engage in interracial relationships. Thus, positive global attitudes toward interracial relationships do not translate into high rates of actual interracial cohabitation or marriage despite the fact that most White Americans (especially White females) aren’t interested in being in a relationship with a non-White person.” (5)
In fact, different races/ethnicities do not experience the same amount of stress while they are in an interracial relationship. And this spans across different age groups, “The negative effects of interracial dating hold similarly for boys and girls and among White and Black youth. Interracial dating less negatively effects the depressive symptomatology of Hispanics, though, and actually appears to ‘protect’ Asian youth from depressive symptoms.”(9) Additionally, White women seem to get extremely stressed from being in a relationship between either Hispanic or Black males. Blacks in those relationships aren’t affected by stress in those relationships:”
“…Furthermore, non-Hispanic whites with non-Hispanic black spouses also fare worse than their interracially married peers with Hispanic spouses. In contrast, the self-reported health of married non-Hispanic blacks shows no significant difference between the interracially and the endogamously married.”(10)”
“Where this stress is coming from is another important aspect to consider: Even if there was a trend of neglect for interracial families/pregnancies, it would only be because of the natural inclination to find these relationships unacceptable, “Bias against interracial romance is correlated with self-reported feelings of disgust”. (12)This phenomenon can be observed via brain scans. There is an intense neural mechanism that is triggered when a person observes interracial couples,“Interracial couples elicit a neural disgust response among observers — as indicated by increased insula activation.”(12)In this case, this psychological disgust could be considered an extension of ethnic nepotism or association; if people are disgusted by these relationships it would imply that being involved with an individual of an out-group is not beneficial to the in-group. Because the effects of race mixing create unhealtheir children, this natural disgust is moral. And as I have shown above, the most distinctive evidence proves this.”
At a neurological level, they’re viewed as animals, as sub-human, and even babies perceive race and avoid the out-group.
“That indicates that viewing images of interracial couples evokes disgust at a neural level,” Skinner said.
Participants were quicker to associate interracial couples with non-human animals and same-race couples with humans. That suggests that interracial couples are more likely to be dehumanized than same-race couples, the researchers write, and previous studies have shown that people tend to exhibit more antisocial behavior and are more likely to use aggression and even violence toward dehumanized targets.”
Nobody is jealous. Are we also jealous of the dirty toilets used in the study? Anyone can trade down. It isn’t hard. Black women trading down to omega white men is still a trade down. Race-mixers are always omegas.
But we found that Asian American young women are at risk of high STDs. For instance, Asian American women had a higher prevalence of STDs than White women in both 1995 (10.4% vs. 7.7) and 2001 (13.5% vs. 8.3%). The incidence of STDs (not diagnosed with STDs in 1995, but developed STIs in 2001) among Asian American women was also higher than that of White women.”
The KA woman’s face did not fit the neoclassical facial canons. Compared with NAW women, 24 of the 26 facial measurements in KA women were significantly different. Only 9 of the 26 facial measurements were significantly different when the attractive KA women were compared with the NAW women. [surgery??] Nine of the 17 nonsignificant facial measurements were very similar to those of the NAW women; many of these facial features centered around the midface.
Although the average KA woman’s facial anthropometric measurements were very different from those of the NAW woman, attractive KA women reflected many of the facial features of NAW women. These findings support the need for ethnically sensitive facial canons and further research into transcultural aesthetics.
Facial beauty arises from symmetric, balanced, and harmonious proportions. Reestablishment of facial harmony requires restoration of proportional facial structures and elimination of disproportionate relationships. The optimal relationships between facial structures are used to assess the face during aesthetic and reconstructive consultations.
Although facial analysis and proportions are well discussed in whites1-3 and African Americans,4-7 only a limited number of studies exist for Asians,8-11 and none exist for Asian Americans. Aesthetic surgery on Asian American patients relying on white norms may result in dissonant facial proportions. Furthermore, many Asian Americans seek to maintain their ethnicity through cosmetic procedures. The challenge for surgeons is to maintain appropriate ethnic facial features and correct only the features that are disproportionate to the rest of the face.
As illustrated by the work of artists and anatomists of the 17th to the 19th centuries, the concept of beauty and “normal” facial proportions has changed with time. [nope?] Furthermore, as the population becomes more heterogeneous, new facial proportions have emerged from interracial mixing. It is now apparent that what has been considered beautiful and acceptable as the norm for one culture may be different for another. Inherently, the notion of a single aesthetic standard and beauty is grossly inadequate and naïve. What is required is a new model of aesthetic standards and beauty that is unique to different ethnic groups to better fit their facial skeletal and skin profile and culture.
[diluting a standard is no standard? ego stroking won’t affect beauty!]
This study assesses the differences in facial proportions between Korean American (KA) women and North American white (NAW) women, and it also describes aesthetic facial features in the typical KA woman. The quantitative determinations will be useful in preoperative and postoperative facial assessment for cosmetic and reconstructive purposes.
[there it is]
Clearly I’m imagining this, right?
“There are inherent problems associated with applying outdated artists’ impressions of beauty to modern facial analysis and surgery. The subjects used in formulating the neoclassical paradigm were exclusively white.”
“Each student in the picture completed a Czech version of the Intelligence Structure Test that uses various types of tools to measure the different types of intelligence.” “Of the raters, 43 women and 42 men judged photos for intelligence, and 42 women and 33 men judged them for attractiveness using a scale of 1 to 7 (1 being the highest score, 7 being the lowest possible score).” “The findings revealed both men and women were able to accurately evaluate the intelligence of men by just viewing the facial photographs.” Lookism takes on a whole new level.
Good luck blaming muh Matriarchy (‘gynocentrism’) for your dumb face.
Pointy face and longer mid-face (nose area) preferred in women too, more model-like too e.g.
The modern short nose look is pure low IQ thot, since looks and IQ correlate.
White men prefer the smarter look more common to the white woman, naturally. Longer midface, smarter kids.
No IQ connection seen because eggs are expensive and they failed to distinguish r from k. Women also have a higher group average IQ compared to men (same pop) so the lower men assessing them is pretty funny. Otherwise, women couldn’t sexually select as a group.
“and these men in turn tend to prefer attractive women, the alleged covariance of attractiveness and intelligence should be of no surprise . However, such findings are controversial and should be approached cautiously since Kanazawa’s research methods and conclusions have attracted strong criticism –. As with physical attractiveness, intelligence is suggested to indicate good genes , . This notion is supported by the fact that during the fertile phase of their menstrual cycle, women display a higher preference for men who score highly in creative intelligence . Intelligence is also correlated with humour, which is suggested to have evolved in sexual selection as an intelligence-indicator . By modifying the good genes approach we find a bad genes hypothesis, which argues that even though unattractive faces signal poor genetic fitness, there is no difference in genetic fitness between faces of average and high attractiveness .”
r/k child outcomes though….
“Men were more accurately assessed for intelligence than women, while women were more accurate at assessing the intelligence of both men and women , .” and BOOM goes the dynamite
Men with longer faces are male models.
Longer midface and nose again.
Hollywood rhinoplasty must shorten the nose because it lacks structure to hold it up!!
“cardiometabolic clinical correlates related to total testosterone (TT), free testosterone (fT), androstenedione (ASD), dehydroepiandrosterone-sulfate (DHEAS), estrone (E1), estradiol (E2), and sex hormone-binding globulin (SHBG).
Results: Waist circumference and BMI (β-coefficient: -0.03; 95% CI: -0.04; 0.03) were inversely related to SHBG, and BMI was positively related to TT (β-coefficient: 0.005; 95% CI: 0.001; 0.009), fT, E1, and E2. Smoking was positively related to TT (β-coefficient: 0.04; 95% CI: 0.01; 0.06), ASD, and fT. Systolic blood pressure (TT: β-coefficient: 0.002; 95% CI: 0.001; 0.003), hypertension (TT: β-coefficient: 0.05; 95% CI: 0.003; 0.11), low-density lipoprotein (LDL) cholesterol (TT: β-coefficient: 0.02; 95% CI: 0.01; 0.05), and total cholesterol (TT: β-coefficient: -0.03; 95% CI: 0.01; 0.05) were positively related to TT and ASD. Finally, type 2 diabetes mellitus (T2DM), and metabolic syndrome (MetS) were positively related to fT, but inversely related to SHBG.
Conclusions: Our population-based study, with sex hormone concentrations measured by liquid chromatography tandem mass spectrometry, revealed associations between clinical correlates including waist circumference, smoking, cohabitation, systolic blood pressure, cholesterol, and MetS with sex hormones. Thus, sex hormones and SHBG may play a role in the cardiovascular risk profile of women.”
Both obesity and anxiety symptomatology were separately associated with the same sex hormone alteration in premenopausal women: higher total testosterone level (0.97 ± 0.50 in obese vs. 0.86 ± 0.49 nmol/L in normal-weight women, p = 0.026 and 1.04 ± 0.59 in women with vs. 0.88 ± 0.49 nmol/L in women without anxiety symptomatology, p = 0.023). However, women with anxiety symptomatology had non-significantly higher estradiol levels than women without anxiety symptomatology (548.0 ± 507.6 vs. 426.2 ± 474.0 pmol/L), whereas obesity was associated with lower estradiol levels compared with those in normal-weight group (332.7 ± 386.5 vs. 470.8 ± 616.0 pmol/L). Women with anxiety symptomatology had also significantly higher testosterone and estradiol composition (p = 0.006). No associations of sex hormone levels and BMI with anxiety symptomatology in postmenopausal women were found.
Conclusions: Although both obesity and anxiety symptomatology were separately associated with higher testosterone level, there was an opposite impact of anxiety and obesity on estradiol levels in premenopausal women. We did not find an evidence that the sex hormone alterations related to obesity are playing a significant role in anxiety symptomatology in premenopausal women. This could be the explanation why we did not find an association between obesity and anxiety. In postmenopausal women, other mechanisms seem to work than in the premenopausal group.
Regional fat distribution (RFD) has been associated with metabolic derangements in populations with obesity. For example, upper body fat patterning is associated with higher levels of free testosterone (FT) and lower levels of sex-hormone binding globulin (SHBG). We sought to determine the extent to which this relationship was true in a healthy (i.e., non-obese) female population and whether RFD influenced androgen responses to resistance exercise. This study examined the effects of RFD on total testosterone (TT), FT, and SHBG responses to an acute resistance exercise test (ARET) among 47 women (22+/-3 years; 165+/-6 cm; 62+/-8 kg; 25+/-5%BF; 23+/-3 BMI). RFD was characterized by 3 separate indices: waist-to-hip ratio (WHR), ratio of upper arm fat to mid-thigh fat assessed with magnetic resonance imaging (MRI ratio), and ratio of subscapular to triceps ratio (SB/TRi ratio). Skinfolds were measured for the triceps, chest, subscapular, mid-axillary, suprailaic, abdomen, and thigh regions. The ARET consisted of 6 sets of 10 RM squats separated by 2-min rest periods. Blood was obtained pre- and post- ARET. TT, FT, and SHBG concentrations were determined by radioimmunoassay. Subjects were divided into tertiles from the indices of RFD, and statistical analyses were performed by an ANOVA with repeated measures (RFD and exercise as main effects). Significant (p < or = .05) increases following the AHRET were observed for TT (approximately 25%), FT (approximately 25%), and SHBG (4%). With multiple regression analysis, anthropometric measures significantly predicted pre- concentrations of FT, post-concentrations of TT, and pre-concentrations of SHBG. The SB/TRi and MRI ratios but not the WHR, were discriminant for hormonal concentrations among the tertiles. In young, healthy women, resistance exercise can induce transient increases in testosterone, and anthropometric markers of adiposity correlate with testosterone concentrations.
So exercise will boost a woman’s natural T. If they already have high T….
If their BMI is higher for their size, they already have high T comparatively. If they already have it racially… probably not good.
Compared to the decline in E2 concentrations, androgen concentrations declined minimally over the MT. T (β 9.180, p < 0.0001) and E1 (β 11.365, p < 0.0001) were higher in Whites than in AAs, while elevations in DHEAS (β 28.80, p = 0.061) and A4 (β 0.2556, p = 0.052) were borderline. Log-transformed E2 was similar between Whites and AAs (β 0.0764, p = 0.272). Body mass index (BMI) was not significantly associated with concentrations of androgens or E1 over time.
so black and white is off the hook
This report suggests that the declines in E2 during the 4 years before and after the FMP are accompanied by minimal changes in DHEAS, A4, T, and E1. There are modest differences between Whites and AAs and minimal differences by BMI.
During a median follow up of 6.3 years, 45 patients relapsed. Testosterone levels significantly increased across BMI categories (p = 0.001). Both circulating testosterone and BMI were positively associated with disease free survival (p = 0.005 and p = 0.021, respectively). A significant interaction was found between testosterone and BMI (p = 0.006). For normal-weight women, testosterone concentration around median (0.403 ng/mL) or third quartile (0.532 ng/mL) showed a high significant HR of relapse (5.52; 95% CI:1.65–18.49 and 4.55; 95% CI:1.09–18.98, respectively). Overweight patients showed increased HR at increasing testosterone levels, reaching a significant high HR (4.68; 95% CI:1.39–15.70) for testosterone values of 0.782 ng/mL (95th percentile). For obese patients HR decreased (not significantly) at increased testosterone concentrations, explaining the interaction between testosterone levels and BMI categories.
In ER-positive postmenopausal breast cancer patients, high testosterone levels are associated with worse prognosis in normal-weight and overweight women, whereas in obese seems to be associated with a better outcome. Although the results require further validation, they suggest that assessment of circulating testosterone and BMI could help to identify postmenopausal ER-positive patients at higher risk of relapse and potentially open new therapeutic strategies.
High T isn’t good, even in normal weight women. Water is wet.
“The findings of this study suggest high plasma levels of testosterone could play a role in the pathogenesis of type 2 diabetes among women,” Jon Jarløv Rasmussen, MD, PhD, a specialist registrar and postdoctoral researcher in the department of endocrinology at Rigshospitalet in Copenhagen, Denmark, told Healio. “The incidence of type 2 diabetes was rather low in the study, but the results implicate that screening for type 2 diabetes among women with higher plasma levels of testosterone may be beneficial, even among women who are young and without established comorbidities, such as polycystic ovary syndrome.”
In a retrospective study, Rasmussen and colleagues analyzed data from 8,876 healthy women (mean age, 38.5 years) who provided blood samples to measure plasma testosterone, dehydroepiandrosterone-sulfate (DHEAS), dihydrotestosterone (DHT) and sex hormone-binding globulin (SHBG) between January 2007 and December 2015. Researchers analyzed androgens using tandem liquid-chromatography mass spectrometry. Researchers used Poisson regression models to calculate incidence rate ratios for developing type 2 diabetes during a median follow-up of 8.1 years, stratified by androgen quartiles.
‘Normal weight’ women can get Type 2. Since Asians have higher T from higher BMI (against the white norm), they’ll be more likely to get it. This also explains the gestational diabetes common in Asian women, especially if the baby is mixed.
Nationwide, as many as 1 in 4 people who have diabetes don’t know they have it. But for Asian Americans, that number is much higher—1 in 2, the highest of all ethnic/racial groups. Why aren’t more getting diagnosed?
Weebs do not mention this. If your apparent rationale for avoiding fat white women is avoiding the Diabeetus genes, Asian is then categorically the worst racial group to mix with.
1 in 2, flip a coin, rice cooker.
I bet it’s higher in the women due to sweet tooth, so likely worse.
But people of Asian descent have less muscle and more fat than other groups and often develop diabetes at a younger age and lower weight. That extra body fat tends to be in the belly (visceral fat). This isn’t the “inch you can pinch,” the fat stored just under the skin. Visceral fat is out of sight, wrapped around organs deep in the body. You can’t tell how much visceral fat someone has by looking at them.
I didn’t call them skinny-fat to be mean, they really are!
Visceral fat is also sometimes known as “active” fat because it drives certain processes in the body that can increase the risk for heart disease, stroke, and other serious health conditions. Everybody has some visceral fat, but having too much is a major risk factor for developing type 2 diabetes.
….But BMI doesn’t catch Asian Americans in the normal weight range (18.5 to 24.9) who may very well have too much visceral fat and be at risk for type 2 diabetes. Researchers are now suggesting that people of Asian heritage get tested if their BMI is 23 or greater. Type 2 diabetes can be prevented or delayed, but only if people know they’re at risk and can take action!
They need a totally different (lower) testing standard, but they’re just like us, guys! Nay, SUPERIOR!
The same volume food in a smaller body, this isn’t hard to figure out. They’re not white women, eating like us makes them FAT.
Pregnant South Asian women carry a higher risk for developing gestational diabetes, a condition that’s dangerous for both mother and child. Between 2 and 10 percent of all pregnancies each year are complicated by gestational diabetes
2-10% in which demographic? Sounds like all? I bet it’s higher in certain ones, isn’t it?
Under risk factors is basically – be non-white
Being of Hispanic, Native American, African-American, Asian-American or Pacific Islander descent.
Women who have had gestational diabetes have a 20 to 50 percent chance of developing diabetes in the 5 to 10 years following pregnancy.
Our data indicate that although the historical or clinical risk factors for GDM are valid in Asians, using risk factors alone to select such patients for testing for GDM is inadequate. Many Asian women who develop GDM have no risk factors at all.
When Natural Selection hates you so much… maybe give it up?
r-types have higher numbers of issues like this, that would be fatal under natural law
They don’t ‘choose’ to stop at 1-2 kids, it isn’t ‘culture’, it’s fear (see below).
To avoid overlooking significant numbers of women with GDM, one may lower the specificity of the criteria, but this requires that the majority of patients be tested.
wow, that bad
Logistically, it is much simpler to conduct universal screening for all Asian women in Western countries, rather than to apply selective testing in order to spare a small percentage of women from being tested. Therefore, our findings strongly support recommendations for universal screening for GDM in pregnant women of Asian origin in Western countries. However, in places where the incidence of GDM is low, such as in some developing countries, the selection of patients for testing by the risk factors may be reasonable.
Introduction:Asian women have a higher prevalence of gestational diabetes mellitus than women of other races/ethnicities. We aimed to compare the prevalence of gestational diabetes among Asian American women to other racial/ethnic groups and explore whether the higher occurrence of the disorder among Asian women can be explained by acculturation.
Clearly I am making this all up to feel better, right guys?
Among the 5,562 women studied, the weighted prevalence of gestational diabetes was 15.5% among Asian American women, followed by 9.0% among non-Hispanic black women, 10.7% among Hispanic women, and 7.9% among non-Hispanic white women.
15.5% v. 7.9%
Diabetes at DOUBLE the rate of whites!
but they’re just like us
2.44x the risk
and that’s controlled, independently
Compared with non-Hispanic white women, Asian women had 2.44 (95% confidence interval [CI], 1.81–3.29; P < .001) times the odds of having gestational diabetes, independent of maternal age, education, marital status, income, prenatal care adequacy, prepregnancy BMI, and physical activity. Acculturation was negatively associated with having gestational diabetes (odds ratio [OR] = 0.93; 95% CI, 0.86–0.99) and explained 15.9% (95% CI, 11.38%–25.08%; P < .001) of the association between Asian race and the condition.
About 85% genetic. Great odds.
We found that Asian race was an independent risk factor for gestational diabetes, and higher acculturation may play a protective role against it in Asian American women.
What is already known about this topic?
Asian women have a higher prevalence of gestational diabetes mellitus than women of other races. However, little data exist on why prevalence is highest among Asian women.
I sense genetics.
If they’re having unnatural babies (too large for their race, mixed) supported by modern medicine, they’d be more likely to die anyway, right? Medicine can only do so much. Weaker genes die a la Darwin.
The biggest r-select factor would be risk of death while breeding, that would be the surest thing. The genes trying to extinct themselves.
Does this data exist? Also for the neonates?
YOU BET IT DOES.
Let’s see the weebs explain away these studies. They’ll probably just ignore me… again.
Pregnancy related mortality can be defined as death of the mother during pregnancy, delivery, or within one year postpartum. While 700 pregnancy-related deaths occur each year, 2/3 of these deaths are considered to be preventable.
Modern medicine, dysgenic again.
Overall pregnancy related mortality in the United States occurs at an average rate of 17.2 deaths per 100,000 live births. However, that number jumps to 43.5/100,000 for non-Hispanic Black women and decreases to 12.7/100,000 for non-Hispanic white women and 11/100,000 for Hispanic women.
No data listed for Asian, odd?
For mothers of all backgrounds, leading causes of death include cardiovascular conditions, hemorrhage, and infection. However, for non-Hispanic Black women, leading causes of death include cardiovascular conditions in addition to cardiomyopathy, pre-eclampsia, and eclampsia (hypertensive disorders).
Non-Hispanic Black women are also significantly more likely to have a severe maternal morbidity (SMM) event at the time of delivery. For every maternal death there are 70 cases of SMM events that are considered “near misses.” These events can have long-term or short-term consequences to a woman’s health. Over the past 20 years, cases of SMM have increased by over 200%, while cases disproportionately affect Black women. One study found Black women experienced SMM at a rate 2.1 times greater than that of white women.
To better understand and address these disparities, researchers suggest providers increase screening for social determinants of health. Levels of stress, trauma, food insecurity, neighborhood violence, and access to prenatal care are all factors that may contribute to the disparities and warrant further investigation.
Although most maternal deaths result from cardiovascular and hypertensive disorders, researchers found Asian/Pacific Island women exhibit the highest prevalence of gestational diabetes, which can increase pregnancy complications, at 14.8%.
One study presented in the session focused on behavioral interventions and protective factors among women with gestational diabetes. A Kaiser Permanente analysis of women in northern California found Black women have a lower prevalence of gestational diabetes when compared with Asian Indian, Filipina, Southeast Asian and Chinese women. White women had the lowest rates of the disease overall.
Screening for postpartum diabetes is recommended to all women within 4 to 12 weeks postpartum. However, rates of screening vary among women with different racial and ethnic backgrounds, suggesting tailored strategies to reduce risk and improve healthcare behaviors may be effective.
Racial medicine, openly.
An additional study explored how racial and ethnic disparities impact severe neonatal morbidities, specifically among very preterm children (born <32 weeks of gestation). Preterm birth has been associated with several health conditions developing later in life, including diabetes.
Presenter Teresa Janevic, PhD, defined race as “linked to phenotype and /or ancestry that indexes one’s location on the US social hierarchy of socially constructed groupings (i.e., races) that has been based primarily on skin color.”
genes aren’t social
Africans in Africa also have the same ‘risk’ as one in America. No magic dirt.
In contrast, Janevic defined ethnicity as “tied to race and used both to distinguish diverse populations and to establish personal or group identity, usually based on shared culture or beliefs.”
Culture? Belief? Believe your way out of diabetes. I’ll wait.
In a population-based retrospective cohort analysis using hospital discharge data linked with vital statistics at birth and death records, researchers determined Black infants were at the highest risk of dying within less than 28 days after discharge, or suffering neonatal morbidities in the time between birth and discharge. Black infants were followed by Hispanic infants, while white and Asian infants had similar low risks.
We’ll see about that.
Of the 39 New York City hospitals included in the study, researchers found a 6-fold difference in risk of combined mortality and morbidity outcomes. “Black infants were at twice the risk of being at a hospital that has risk-adjusted high rates of combined mortality and morbidity,” Janevic noted, while Hispanic infants had a 1.5 increased risk to receive care from one of these hospitals. “Hospital quality where women of color deliver likely contributes to these disparities,” she concluded.
Like schools, it depends on the IQ of the people working there.
Another investigation detailed how environmental factors and population level exposures impact disparities in preterm birth and infant mortality. “Non-Hispanic Black infants compared with non-Hispanic white infants have twice the risk of death in the first year,” explained presenter Heather Burris, MD. “This is particularly striking because Black infants just make up 15% of all births in the United States but are counting for 29% of all deaths.”
Among causes of infant death, preterm birth and low birth weight related death, along with pregnancy complications, account for the highest racial and ethnic disparities between non-Hispanic Black and white infants. Black infants are also significantly more likely to be born preterm than white infants.
an r-factor unless twins
Researchers note genetics and education level have very little impact in accounting for disparities in preterm birth. Although women with higher education tend to have lower preterm birth rates, Black women who graduated from college have a higher risk of preterm birth than white women who dropped out of high school.
I’m so glad white people already survived multiple genetic purges in our history.
Through analyzing delivery data and creating models based on air pollution severity in Philadelphia, Pennsylvania, investigators discovered air pollution is associated with spontaneous preterm birth. Data also show Black Americans experience consistently higher exposure to air pollutants, measured in fine particulate matter (PM)2.5.
An additional analysis between preterm birth and nationwide neighborhood deprivation index (encompassing income below the poverty level, vacant homes, education levels, among other factors) found that Black women experience neighborhood deprivation exposure at almost 2 standard deviations (SDs) higher than white women in Philadelphia.
Overall, Black women are 4 times more likely to live in a neighborhood with high violent crime and high air pollution than white women. “When we look at preterm birthweights, we can see that it is women living in these high-high neighborhoods that have the highest risk of preterm birth,” Burris said. However, these associations were consistent regardless of race.
They gestate for less time than whites, this is known. Africans in Africa do it.
Now we’ve established some things. An r-study in Asian women.
Increased Perinatal Morbidity and Mortality Among Asian American and Pacific Islander Women in the United States
Background: Asian American/Pacific Islanders (AAPIs) are the fastest-growing racial group in the United States.
America is now owned by Asia, demographically.
Despite a higher socioeconomic status, AAPI women experience higher rates of maternal morbidity and mortality.
can’t pay your way out of r-genes
if controlled for SES, aka $, their data would be even worse
Methods: Using the National Inpatient Sample, we performed a retrospective cohort analysis of women who were hospitalized for delivery from 2002 to 2013. The primary outcome variable was inpatient mortality rate, and the presence of severe maternal morbidities was estimated using the Bateman Comorbidity Index, a validated tool for predicting obstetric morbidity.
Results: AAPI women presenting for delivery between 2003 and 2012 were older, more likely to reside in a zip code in the top quartile of annual income, be privately insured than Caucasian women,
where’s Asian privilege?
and less likely to have a higher Bateman Comorbidity Index. However, AAPI women had a higher likelihood of postpartum hemorrhage (3.4% vs 2.7%, P < .001), uterine atony, severe perineal lacerations, and severe maternal morbidities. Procedures such as transfusion, hysterectomy,
So they could have one kid and die, have one kid and have that die, OR have one kid and then their organs all removed – so no more kids?
Yes clearly our biological superiors, right weebs? Totally not rationalising a fetish, are we?
I wonder why one child was law? They don’t have a culture of many kids because they’re too r-select to survive without modern medicine. Wake up. They pretend 1-2 is a choice and that’s why they mock and envy large white families (3+ standard) like the Amish. They envy us that ability. They would die.
and mechanical ventilation were also more common in AAPI women.
Calling it – Mother Nature is anti-Asian.
Furthermore, AAPI women had a higher mortality rate that persisted despite adjustment for an apparently higher income and comorbidities (odds ratio 1.72, 95% confidence interval: 1.14-2.59, P = .01).
Conclusions: Despite having a higher socioeconomic status, AAPI women had higher rates of maternal mortality during hospitalization for delivery. This increase persisted even after adjustment for factors known to affect peripartum outcomes. Further investigation is needed to better clarify the causes of racial differences in maternal morbidity and mortality.
Results: A total of 360,370 women with postpartum hemorrhage from 2012 to 2014 were included in this analysis. Risk for severe morbidity was significantly higher among non-Hispanic black women (26.6%) than non-Hispanic white, Hispanic, or Asian or Pacific Islander women (20.7%, 22.5%, and 21.4%, respectively, P < .01).
The white is 20%, Asian is 21%.
And these are the fattest white people, like, ever.
White and Asian bolded-
For non-Hispanic black compared with non-Hispanic white, Hispanic, and Asian or Pacific Islander women risk was higher for disseminated intravascular coagulation (8.4% vs 7.1%, 6.8%, and 6.8%, respectively, P < .01) and transfusion (19.4% vs 13.9%, 16.1%, and 15.8%, respectively, P < .01). Black women were also more likely than non-Hispanic white women to undergo hysterectomy (2.4% vs 1.9%, P < .01), although Asian or Pacific Islander women were at highest risk (2.9%). Adjusting for comorbidity, black women remained at higher risk for severe morbidity (P < .01). Risk for death for non-Hispanic black women was significantly higher than for nonblack women (121.8 per 100,000 deliveries, 95% confidence interval, 94.7-156.8 vs 24.1 per 100,000 deliveries, 95% confidence interval, 19.2-30.2, respectively, P < .01).
The weebs either did 1. no research (typical gammas) or 2. they’re delusional.
Almost double the risk of hysterectomy, roughly.An additional 52% risk over white women, minimum, in just this study.
What’s the point of being married to them, at that point? Their baby machine is broken.
Non-Hispanic black (black) and non-Hispanic American Indian/Alaska Native (AI/AN) women experienced higher PRMRs (40.8 and 29.7, respectively) than all other racial/ethnic populations (white PRMR was 12.7, Asian/ Pacific Islander PRMR was 13.5 and Hispanic PRMR was 11.5). This was 3.2 and 2.3 times higher than the PRMR for white women – and the gap widened among older age groups.
Notably, we found that, when aggregated, the top cause of death among Asian Americans is cancer. However, when disaggregated, there is wide variation in the leading cause of death. For instance, for Asian Indians, nearly twice as many men die of heart disease (31 percent), compared to cancer (18 percent). In contrast, for Koreans, the opposite is true — the death rate for cancer (34 percent) is much higher than the death rate for heart disease (19 percent).
Remember the breast cancer and Asian BMI/testosterone stuff?
Research led by the University of Birmingham has found that increased levels of hormones including testosterone could cause a brain condition that can lead to blindness in women.
We are all jealous of your waifu, yes.
Idiopathic Intracranial Hypertension—also known as IIH—is caused by high pressure in the brain with consequences from blindness to incapacitating daily long-term headaches. IIH was originally identified over 100 years ago yet the cause of the condition has remained unknown although there has been much speculation about why more than 95 per cent of total incidence is in women with obesity.
And Asians, they’re 1/2 obese in America!
They then compared the results with the levels observed in women with obesity of the same age and body mass index (BMI), as well as a cohort of women with polycystic ovary syndrome (PCOS).
PCOS is far more common in Asians. Look it up.
Most notable were the high levels of the androgen ‘testosterone’ found in the blood in IIH women. Crucially, levels of androgens were uniquely increased in the brain fluid (CSF) of women with IIH. When the researchers, analysed human choroidal plexus tissue, which is the site in the brain where CSF is produced, they confirmed that androgens could increase the rate of CSF secretion, a potential driver for increased brain pressure.
Results: We found that the South Asian women presented at a younger age for the management of sub-fertility. An extended stimulation phase and Caucasian ethnicity showed an inverse correlation with the number of oocytes retrieved in the PCOS subgroup. Caucasian ethnicity was associated with a higher fertilization rate however increase in body mass index (BMI) and the laboratory technique of IVF appeared to have a negative impact on fertilization rates in the PCOS subgroup. Commencing down regulation on day 1 of the cycles was negatively associated with fertilization rates in the tubal group. In terms of clinical pregnancy rates, the Caucasian PCOS had a 2.5 times (95% CI: 1.25-5) higher chance of an ongoing clinical pregnancy as compared with their Asian counterpart. Also, a unit increase in the basal FSH concentration reduced the odds of pregnancy by 18.6% (95% CI: 1.8-32.6%) in the PCOS group.
Conclusions: The Asian PCOS have a greater sensitivity to gonadotropin stimulation with lower fertilization and ongoing clinical pregnancy rates as compared with their Caucasian counterparts.
The ethnicity of women undergoing fertility treatments like IVF can affect the rate of successful live births, according to new research. After adjusting for certain factors including age of patient at time of treatment, cause of female or male infertility, and type of treatment, the study found that White Irish, South Asian Indian, South Asian Bangladeshi, South Asian Pakistani, Black African, and Other Asian women had a significantly lower odds of a live birth than White British women.
White women, still winning. Thank God for the Ice Age.
Overall, studies have shown higher testosterone levels in women and lower levels in men are related to incident diabetes. The major risk factors contributing to diabetes are biochemical, environmental, sedentary lifestyle, socioeconomic status and genetic factors. All of them together or independently are responsible for the development of the DM.  Besides, certain studies show Impaired Glucose Tolerance (IGT) is more common in females than males independent of age. 
We found a high prevalence of GDM among the Asian population. Asian women with common risk factors especially among those with history of previous GDM, congenital anomalies or macrosomia should receive additional attention from physician as high-risk cases for GDM in pregnancy.
Body mass index (BMI) was a very strong negative predictor of body attractiveness ratings, similar to previous findings. Zero-order associations between women’s mean hormone concentrations and mean attractiveness ratings were not significant; however, after controlling for BMI, attractiveness ratings were independently and positively associated with both estradiol and testosterone concentrations. Discussion focuses on the implications of these findings for whether attractiveness assessment mechanisms are specialized for the detection of cues of differential fecundity in young women’s bodies.
High T = ugly!
Did I mention water is wet? Can they seriously accuse of cherry picking? I’m not even looking hard.
Previously covered WHR, use search bar. Asians lose. Even black women do better.
Asians have way more T as a race than Europeans, get over it. Historically, we considered them savages, less civilized, for that reason. How is this surprising? Do you think we colonised India for fun? It’s obvious in the broad manjaws, duh. Marquardt covered this. Anyone can do a replication study, but I suggest you include the women too, so it isn’t just a sexual effect but race.
From a blog “East Asians were found to have the highest average total plasma testosterone (5,673 ρg/mL) followed by Africans (5,442 ρg/mL) and then Europeans (4,992 ρg/mL). Given that the sample size for Africans is smaller (N < 10,000), their relative position may change with more data. Nonetheless, the claim that East Asians have the least testosterone is not supported by scientific data. “
Yeah, fake redpills who think T = manly, good thing. It’s just a hormone.
“There is no way of accurately determining free testosterone. Even if there was, this would also be irrelevant since bio-availability is prime. Since race realists use total serum testosterone, why is this an issue?”
true, it’s just applying the same standard
Culturally, gang rape is more normal in Asia than Africa. This is why. You don’t get African Taharrush, really. Asia has Eve Teasing and the like. Trust me, you don’t want this.
“Mass sexual assault is the collective sexual assault of women, and sometimes children, in public by groups of unrelated men. Typically acting under the protective cover of large gatherings, victims have reported being groped, stripped, beaten, bitten, penetrated and raped.”
As for the contention that there are no studies indicating a 10% difference between East Asians and Europeans, I did find one age controlled study where the Chinese sample had 8.8% more total T, 11.4% more bio-available T and 12% more free T than the European sample. The Japanese sample had 10.5%, 5.1% and 6.7% more than Europeans respectively [Wu et al. 1995]. Wonder if race realists discuss this study, or perhaps they are too busy in celebratory dance around the Korean/Swede campfire?
They’re not really redpill. I believe data even if I dislike it. Asians have high T as a race. Get over it.
High T can also dovetail with lower national IQs e.g. India, so…. why want this? Low IQ nations have more crime.
Additionally, this recent study shows HK Chinese having some 3% more bio-available T than US Europeans.
Lol, he’s right. But T isn’t a good thing. It’s just a hormone, in men or women.
Being a race realists seems to be a length engagement with delusion, fantasy and ‘scientific’ homo-erotica.
Not here, son. I believe the T-data. Penis size generally correlates to racial height (in white men), not really T. Forum bros are wrong again. Penis stuff is sexual selection, aka chosen by women.
There was a similar increase in the positive effect of penis size on attractiveness with a more masculine body shape (i.e., greater shoulder-to-hip ratio). Surprisingly, larger penis size and greater height had almost equivalent positive effects on male attractiveness. Our results support the hypothesis that female mate choice could have driven the evolution of larger penises in humans. More broadly, our results show that precopulatory sexual selection can play a role in the evolution of genital traits.
It was concluded that all penile measurements are interrelated to each other; the height and weight also the other body measurements that are related to the penile measurements in less than 50%. It seems that the penile measurements are polygenic traits and are under multifactorial influences.
There are racial differences in associations of hormone levels with age and BMI in late reproductive age women. Further study is needed to replicate these findings and to determine the relationships of these hormonal associations with menopausal symptoms
Obesity is an important factor in hormone dynamics independent of age, race and smoking in mid-life women, although the mechanisms remain unclear.
From “A Study of the Correlation of Some Sex Hormone with Obesity in Women with Secondary Infertility” (google it)
Infertility is the inability to conceive a child for more than one year. The present study indicates
that the obesity associated with infertility. The aim of the study to determine follicle stimulating
hormone, luteinizing hormone, testosterone hormone and prolactin levels. and cholesterol and
triglyceride concentration in 2nd inferetid women. This study was carried out at kamal al-samaarai
hospital the data were collected from 95 secondary infertilial women were age between 16-45 years old and grouped them in to obese (n = 46) and non obese(n = 49). There was no significant difference between the two groups (p <0.05).Body mass index in Infertile obese women is slightly higher than non obese Infertile women which is statistically significant (P<0.001). However LH,
TSH, cholesterol and triglyceride concentration in obese infertile women is significantly higher than
non obese infertile women (p >0.05).The BMI was correlated positively with triglyceride in obese
group while BMI was positive correlation highly significant with cholesterol in non obese group.
Regression analysis revealed obese to be strongly associated with observed infertility. The elevated
prolactin values in secondary infertile women clearly shows that there is a mechanism operating at
the anterior pituitary level which shows an abnormal distribution of FSH and LH which may further
explain the abnormal delay ovum maturation. This study also indicates obese associated with
infertile more than non obese women.
TLDR Asians carry more fat than whites (in America, at least).
Well yeah, the junk food and spices diet is hell on the hormones too. They evolved for multiple famines (especially the Chinese and Indians) so they tend to weight-gain like that, too. Fat deposition isn’t the same between race (black thighs/’ass’) so why would accumulation patterns and proclivity to gain work like that either?
Results: A convenient sample of 1626 adults with BMIs ≤35 was evaluated. Independent percentage body fat predictor variables in multiple regression models included 1/BMI, sex, age, and ethnic group (R values from 0.74 to 0.92 and SEEs from 2.8 to 5.4% fat). The prediction formulas were then used to prepare provisional healthy percentage body fat ranges based on published BMI limits for underweight (<18.5), overweight (≥25), and obesity (≥30).
Conclusion: This proposed approach and initial findings provide the groundwork and stimulus for establishing international healthy body fat ranges.
But they’re not the same. Bio-logically.
The Asian ‘gym girls’ also trying to pass off the manjaw as ‘gains’ by matching on the stomach and thighs, I’ve seen white lawyer types do the same. Do NOT fall for it, men. If the jaw is square and broad as a child and pre-teen, RUN.
The small, squat (squarish) skeleton and pedomorphic features of the Asian race (historically, accurately called Mongoloid if you can search for it in anthropology) already pre-dispose their young people (teens) to lower BMI anyway, so the ‘losing weight’ excuse is bullshit, they’re building muscle to match the jaw and trap some sucker with their high T and bad temper. This lower BMI is not a consequence of health (dietary or genetic) so compared to the unhealthiest white people EVER (sorry America) they still have higher BMIs. HOW.
White women have a naturally high BMI (taller, curves) so it’s actually worse than it looks. The effect is maintained in every age group, how can one fuck up age 20? Any 20yo complaining about their metabolism needs a slap. The fat Asian hypothesis also holds true within the race, compared the Asian men to women matching age.
IF ONLY THE MANOSPHERE ACTUALLY READ THE DATA.
Every other claim about Asians it’s like  yet when I go to look…. they lied.
I expected the one claim the weebs consistently made about Asians in America (being thin) must be true but … no.
18.5 is a terrible BMI cut-off because it includes anorexics, smokers, druggies and petite women all in one. Women look wildly different at low and too-low body fats, which also varies by race. At the same BMI, Asians look more husky because the skeleton is squared-off. They’re also likelier to lay down fat than muscle.
The Asians are larger than white women pre-menopause, what EXCUSE is there?
The only time whites actually have slightly higher BMIs (ever so slightly) is in the elderly range (60-79, at which point yeah you’re excused, you can go).
I’m seeing through the model minority propaganda and there’s nothing to it. Rule #1 of moving to America – DON’T GET FAT. YOU HAD ONE JOB.
“In contrast, models for Asians predicted a different percentage body fat from that predicted for African Americans and whites. “
That’s code for ‘they evolved so much for famine that we had to invent new maths to count it’.
“For example, Deurenberg et al (35) found that American blacks had a 1.3-unit lower and Polynesians a 4.5-unit higher BMI than whites with the same body fatness (35). Even within the white cohort, the investigators observed small differences between Americans and Europeans.”
Asian placement is denser. Squat Mongoloid skeleton, anthropologically. If you disagree, it’s cope.
Americans are mixed white, generally, that’s why. German/French hybrids and other nonsense. You lose adaptive advantage from your subrace too.
They studied one of the thinnest types of Asians (japs) so I guarantee you others would be worse.
“The underlying causes of ethnic variation in relations between BMI and percentage body fat are likely due to small between-center body fat measurement differences and biological between-group differences (35).”
“The evaluation of Asians was confined to Japan and that of African Americans to the United States. Therefore, the underlying causes of observed ethnic differences in terms of measurement, environmental, and genetic factors are difficult to ascertain. Nevertheless, it appears evident that a single set of universal percentage body fat ranges cannot be easily developed without considerable additional analysis of this problem. Our equations and associated tables provide several ethnic-specific ranges as working guidelines. Because African Americans and whites differed only slightly in percentage body fat (by 1–2%) after BMI was first controlled for, we presented a combined equation (Equation 5) and table (Table 4) based on 4C percentage body fat for these 2 groups.”
That’s right, our fat deposition is more alike blacks than Asians. That’s how different the races are.
Background: Asians who have a healthy body mass index (BMI) range have been observed to have higher levels of obesity and risk of cardiovascular disease than whites, which suggests that the relation between BMI and adiposity may be different for Asians.
Objective: The primary aim of this study was to investigate the influence of childhood and adolescent exposure to a westernized environment on the relation between BMI and percentage body fat in young Asian American women.
So no, this isn’t their age. No cope here son.
Design: Secondary data from 129 Asian women, aged 20–25 y, with variable lengths of residence in the United States and 327 white women of comparable ages who had participated in the Latina and Asian Bone Health Study (1999–2000) and the Berkeley Bone Health Study (1998–2000), respectively, were analyzed by using multiple linear regression with percentage body fat as the outcome variable and place of birth, ethnicity, length of US residency, and BMI as predictor variables.
Results: Asians who lived in the United States <12 y showed the same relation between BMI and percentage body fat as did whites. In contrast, Asians who had lived in the United States ≥12 y had higher percentage body fat than did whites for BMIs (in kg/m2) <20.5 and lower percentage body fat for BMIs in the overweight and obese range.
Responding to the same stimuli wildly differently > genetic!
They’re more likely skinny-fat, aka fatal fat.
Whites evolved to eat Roman grains like processed wheat.
Conclusions: Our findings suggest that childhood environments may influence the relation between BMI and adiposity. Research is warranted on the role that childhood environments play in the accumulation and distribution of body fat and hence metabolic disease risk later in life.
There’s still a huge difference in the same environment.
Asians with more years in the United States had higher mean percentage body fat than did whites at low BMIs and lower mean percentage body fat at high BMIs with the regression lines intersecting at BMI ≈20.5 and body fat of 26.5%.
Again, Magic Dirt isn’t real. Enjoy your BBW Ting Tong for your golden years.
We are supremely jealous.
Asian-immigrant adults who were living in North America were observed to have higher adiposity levels
at the same BMI level than did whites (6). However, most of the Asian-immigrant adults studied were raised outside of North America. To our knowledge, our study was among the first studies to observe a difference in how BMI relates to percentage body fat between Asian Americans who had spent more years of childhood in the United States compared with Asian Americans who were minimally exposed to the United States during childhood and adolescence.
Lesson for weebs – do NOT move the Thai bride to America.
Stay in Asia or do NOT bother. She WILL get fat. You have been warned.
American-born Asians had a higher percentage of mothers with college degrees (59% of mothers compared with 37% of mothers in foreign-born Asians and 32% of mothers in whites).
Fat SJWs, great catch!
in our study, which suggested that obesity rates in our sample of Asians were likely lower than in the general population.
They admit it.
Is the general observation that, at the same BMI level, Asians have higher total body fat and visceral fat and higher risk of cardiovascular disease risk than do whites applicable to Asians in general,
or is this observation applicable only to Asians living in Asian societies? If the latter, does the environment during early life influence how body fat accumulates and distributes and, therefore, affects the relation between BMI and body fat? How strong a predictor of child growth and body composition is socioeconomic background and, in particular, the mother’s education? Are our findings also applicable to men?
We concluded that the early life environment may influence the relation between BMI and adiposity in later life. In particular, we speculated that Asian women substantially exposed to the American environment early in life exhibited different patterns of accumulation of body fat than did Asian women raised in Asia;
non-sig, see above
these differences may be partially attributable to environmental and lifestyle factors that affected diet and physical activity. For example, compared with Asian children raised in the United States, children who live in Asia tend to spend more time studying, whether in school or at home, and less time doing sports and engaging in recreational activities (25). In the past, diets of Asians raised in Asia also tended to be different, with a lower consumption of breakfast cereals, dairy products, and processed meats, than diets of Asians raised in the United States (26).
Yes, they do try to eat white. Silly Asians, didn’t evolve for dairy.
So your grandkids will definitely be fat, even if she isn’t by some fluke? Great, very likely to pass on your genome.
What a waste of time and resources.
However, with the rapid changes that have been taking place in Asian economies and the globalization of the food supply, we expect to see further changes in the diets of children in Asia (27) and, consequently, in body composition.
The understanding of how childhood environments influence the relation between BMI and percentage body fat has implications for the use of BMI as a screening indicator for obesity and obesity-related conditions. In addition, research to understand the influence of environmental factors on the accumulation and distribution of total body fat and metabolically active visceral fat during critical life stages will add insight into the use of clinically relevant screening tools for chronic disease risk.
Plan on growing old together, spending a loooong time with fatass.
South Asians are susceptible to insulin resistance even without obesity. We examined the characteristics of body fat content, distribution and function in South Asian men and their relationships to insulin resistance compared to Caucasians.
so no, you can’t tell by ‘looking at her’, your norms are informed by white women
Research Design and Methods
Twenty-nine South Asian and 18 Caucasian non-diabetic men (age 27±3 and 27±3 years, respectively) underwent euglycemic-hyperinsulinemic clamp for insulin sensitivity, underwater weighing for total body fat, MRI of entire abdomen for intraperitoneal (IP) and subcutaneous abdominal (SA) fat and biopsy of SA fat for adipocyte size.
Compared to Caucasians, in spite of similar BMI, South Asians had higher total body fat (22±6 and 15±4% of body weight; p-value<0.0001), higher SA fat (3.5±1.9 and 2.2±1.3 kg, respectively; p-value = 0.004), but no differences in IP fat (1.0±0.5 and 1.0±0.7 kg, respectively; p-value = 0.4). SA adipocyte cell size was significantly higher in South Asians (3491±1393 and 1648±864 µm2; p-value = 0.0001) and was inversely correlated with both glucose disposal rate (r-value = −0.57; p-value = 0.0008) and plasma adiponectin concentrations (r-value = −0.71; p-value<0.0001). Adipocyte size differences persisted even when SA was matched between South Asians and Caucasians.
Marry those genes, go ahead.
Insulin resistance in young South Asian men can be observed even without increase in IP fat mass and is related to large SA adipocytes size. Hence ethnic excess in insulin resistance in South Asians appears to be related more to excess truncal fat and dysfunctional adipose tissue than to excess visceral fat.
in the men
harsh to call a racial adaptation to famine ‘dysfunctional’
So-called meditation is a religious practice, a form of prayer. It is communication with spiritual entities beyond one’s body. It has no place in schools. Schools want to impose it because it forms a kind of repression in the developing brain, it is cliche Victorian emotional repression (the stiff upper lip). I happen to think the state and agents of the state should have no ‘right’ to call a citizen’s emotions invalid or wrong. Children are not hysterics to be corrected into quiet submission. These mindful or mindless devotionals are used to control children, by adults, a great evil. The meditation as they would practise it is pagan in origin and cannot be made secular. One’s Higher Self is godly, the Creator. It is the thing we rejoin once we die. Clearly, this is horrendously inappropriate to ‘teach’ children in a school setting, under the guise of keeping them quiet (stiff upper lip) and teaching ‘focus’. All prayer teaches focus by repetition of words or concepts with the express purpose of drowning out personal emotion. Schools should not be emptying the minds of the students. It is a toxic concept, and you can point to all the scientism studies you like and it won’t change the fact children should not be lectured on their very thought process and censored therein, by the State! This is how docile populaces are made, and the brain changes are permanent. We call this clipping of their wings brain damage, if we’re being more accurate. There is nothing inherently wrong with their natural, evolving brain development! Being a healthy, developing child is not a sin and should not be condemned with appropriated, shallow religious practices.
Piaget believed that what he referred to as “hypothetical-deductive reasoning” was essential at this stage of intellectual development. At this point, teens become capable of thinking about abstract and hypothetical ideas.
Capable! Prior to this, critical rejection is impossible.
But religious material is taught as RE, prior to this…..
The State cannot preach contentment and faith in devotional practices like a church. It is curious that such a ‘process’ does not work without faith and this failure would present in older children more often, so the schools push the prayer earlier. In the same way children are not physically capable of performing all the same activities, it is a violation of human rights to foist any censorship onto their emotional mind. The right to feel is human and sacrosanct, legally. Blotting out ‘undesirable’ emotions by the state agents, like anger, is cruel and unusual punishment. Not to mention, impossible. The military doesn’t go so far as to call possibly righteous emotions like anger ‘wrong’. A negative emotion is not innately ‘bad’ or wrong. If the teachers have failed to earn the respect (and obedience) of the students, however many drugs are pushed into that population, then perhaps the fault is with the adults and not the children? If we look at the league tables and life outcomes, a child’s disrespect of their teachers (and ‘misbehaviour’, aka questioning authority) is entirely valid.
“brain training” – it’s brainwashing
It’s creepy to watch those kids do qi or psi practices (those hand gestures) and think this is scientific.
Pushing Buddhism, they literally have a guy right there.
“their brains are being reshaped” – by the State
They’re encouraging ego death in white kids. See the fleeting reference to privilege, later? It forces a kind of depersonalization and derealization. Let me guess, this only ‘needs’ to be done on white kids in white schools?
If it were scientific, it’d work 100% of the time. This is based on faith.
When you drink water, you’re hydrated. There’s cause. With this, the ‘stress’ placebo only works if you believe it will. Slow breathing will literally do the same thing, but they want an empty mind.
Marcus Aurelius taught about ‘controlling the mind’ too but he was white and didn’t promote ego death for Cultural Marxism, so you won’t hear about it.
“delusions such as anger or attachment” ATTACHMENT. Like to your family, country, attachment!
“ignorance, jealousy, so when the mind is out of control” They’re gaslighting children as hysterics.
Lobotomies have such a bad rep.
“They can’t control what they say or what they do”
I think they can they’re human beings, this is dehumanisation. Worshipping Buddha, a man, is idolatry. It isn’t actually a religion, there is no God.
“everything we do relates to our intention”
the road to Hell actually the West judges by character and behaviour
I’m sure there are some really nice, lonely pedophiles, they’re still evil.
“be mindful of the correct things” WHAT correct things? oh DO tell us!
(he does not)
“everyone needs to meditate” – some weeb larper
“clarity and peace within our mind” – you mean around authorities like school?
We had those things in the West for millennia.
Since you can’t do anything about political correctness, why not just make the best of it?
Why not exploit the situation instead of moaning about it?
Do what is expedient – why not?
Why not make a successful career out of PC – like so many others?
Why not surrender your private mind to PC, in the same way as you have already surrendered your public behaviour?
By having any reservations at all, you are making yourself miserable – why not simply cast-aside those reservations?
Just say an inner yes to what you will, anyway, be forced to do…
Since you necessarily inhabit the thought prison that is political correctness – then why not, at least, become one of the ‘trustys’ among the inmates – to assist with the smooth running of the gaol, and get yourself a few privileges.
Why not, indeed, strive to become one of the guards? Somebody has to do the job? Maybe you could temper the severity of the regime?
And herein lies the particular temptation for the intellectual elite – a temptation few resist.
That (literally) soul-destroying pragmatism by which (for eminently sensible reasons) we quietly, by gradual degrees, change sides in the spiritual battle of the world: that unseen warfare between The Good and that which opposes The Good.
Well why not?
There is no earthly reason why not.
In a world of pervasive and powerful PC, there is really only one compelling reason for holding back and resisting in any way, shape or form – which is that embracing political correctness will shrink your soul.
If you do not believe in the soul, this reason will carry no force at all: so by your own calculations you are stupid to resist PC.
the reprobate mind
Or, if you believe the soul is inviolable, and that nothing you think or do can affect the soul: then also, by your own calculations, you are stupid to resist PC.
If you do not believe in Natural Law (innate knowledge of The Good), and that breaking Natural Law harms the soul: then logically you should learn to love PC.
If you do not believe in the reality of transcendental good – then you might as well go with the flow, allow yourself to be re-programmed: to learn, by regular practice, to re-label lies as truth, ugliness as beauty, evil as virtue; until PC has entered into your heart and soul, as well as pouring into your ears and out-from your mouth.
But political correctness is nihilism; therefore it is not merely political: it is also existential.
To fight against political correctness is therefore ultimately an existential act: a battle to preserve the eternal soul.
But if you do not believe that political correctness will harm your eternal soul: then you would be well-advised to suck it up.
“as a scientist, categorically”
Mengele was a scientist, wouldn’t trust him with kids. Scientists are not a superior race, free of motive and emotionality.
The nose in the air is creepy.
It’s a placebo action, there is no actual control because the kids know when they’re doing it.
Controlled breathing would be a control. But they won’t do that- no effect!
“the stories that we tell about ourselves, about other people, aren’t necessarily true” paging Scientology How is this the business of the State? How is self-image and identity their concern?
and “about other people” – such AS? “so we can enquire into that”
violation of human rights, right to privacy this is experimentation violating the Nuremberg Code
Can the State psychoanalyze you? Where is the consent?
“a healthy skepticism about our thought process” well calling it healthy makes it healthy pass the deep-fried Mars bar this is science
can I be skeptical of your skepticism?
if it’s real, why can’t I question it?
If you guessed I was the one replying, you’d be right. Also measurement error.
the ‘get em young’ thing is a huge red flag
they go from showing teens, discussing teens to…
“8 and 9 year-olds”
and the Buddhist guy wants to be left alone around them, in a hypnotic state.
I had a feeling about this…
Can Mindfulness Help Reduce Racism?
Can we override hidden prejudice? A new study says, yes, it can be done—and the key might be mindfulness meditation.
In their study, 72 white college students were measured on their levels of implicit bias of blacks and the elderly using the IAT.
Was it a KKK convention? How else to only include white kids? What are the odds?
Another anti-white methodology, ignore the coincidence.
Some participants then listened to a 10-minute mindfulness meditation in which they were instructed to “become aware of bodily sensations (heartbeat and breath) and fully accept these sensations and any thoughts without restriction, resistance, or judgment”;
That’s one way pedos groom kids. So it’s beyond just steady breathing, you see. It’s self-hypnosis.
They don’t assign it as homework because then they couldn’t implant various notions while the kid is suggestible.
other participants listened to a recording about natural history, voiced by the same narrator.
Why not steady breathing? That isn’t a real control.
Afterwards, the two groups of students were evaluated on their levels of mindfulness and then reassessed on their levels of implicit bias using the IAT.
IAT is BS.
Results showed that people who listened to the 10-minute mindfulness recording demonstrated less implicit bias against blacks and old people on the race and age IATs than individuals who listened to the other 10-minute recording. In other words, the mindfulness intervention decreased students’ automatic biases against blacks and older adults.
The mind is numb, they’d be less likely to associate rabbits with carrots, too! It retards the thought process.
For example, mindfulness training has been shown to help overeaters decrease the automatic attractiveness of fatty foods, allowing them to resist eating those foods when the foods are presented to them.
It literally over-rides survival, yeah.
This is like the hen-chalk thing with people, it suppresses nervous function (fear appraisal). A fox could walk right in front of them and they wouldn’t move. Non-response is not good.
They’re doing this to your kids.
One interesting consideration is that in the present study the mindfulness training was very brief and non-specific—in other words, the authors did not specifically try to train the participants in bias-reduction.
Retardation, same thing. Stop noticing studies.
With no warning of the true purpose, the subject cannot resist – just like that chicken.
This leads the authors to suggest that brief mindfulness training may be a good substitute for—or may augment—more traditional anti-bias training.
Even if we try to act adopt a colorblind view in the world, it doesn’t work because our brains don’t actually work that way.
They know what they’re doing.
In my own work, I identify, develop and examine the efficacy of a set of practices that intentionally link inner and outer work to raise awareness about race and racial experience in our lives, with a focus on personal, interpersonal, and systemic or structural levels.
The resulting “ColorInsight Practices ” combine mindfulness-based practices with teaching and learning about race and color to increase awareness of how race and color impact us all, and give rise to insight and greater understanding. They pave the way to new experiences that help us loosen our attachments to narratives and other forms of suffering that give rise to biases along the way…..
Attachments are GOOD, a human RIGHT.
The Nazis wanted to end suffering, if we’re going in that direction.
Why is nobody talking about this? Controlled ops. Plenty of proof.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3862075/ Discrimination hurts, but mindfulness may help: Trait mindfulness moderates the relationship between perceived discrimination and depressive symptoms
Discriminatory experiences are not only momentarily distressing, but can also increase risk for lasting physical and psychological problems. Specifically, significantly higher rates of depression and depressive symptoms are reported among people who are frequently the target of prejudice. Given the gravity of this problem, this research focuses on an individual difference, trait mindfulness, as a protective factor in the association between discrimination and depressive symptoms. In a community sample of 605 individuals, trait mindfulness dampens the relationship between perceived discrimination and depressive symptoms. Additionally, mindfulness provides benefits above and beyond those of positive emotions. Trait mindfulness may thus operate as a protective individual difference for targets of discrimination.
Discrimination is critical thought, it’s vital for cognition.
Brief Mindfulness Meditation Reduces Discrimination.
Recent research has demonstrated that mindfulness meditation reduces implicit race and age bias by weakening the associations of the target group with negative constructs. The current research examined the potential for mindfulness to also affect discriminatory behavior. Participants listened to either a 10-min mindfulness audio or a control audio before playing a game in which they interacted with partners of different races in a simulation and decided how much they trusted them with their money. Results indicated that the mindfulness condition exhibited significantly less discrimination in the Trust Game than did either of the 2 control conditions. The implications and importance of mindfulness meditation in alleviating bias are discussed.
As thought control, it’s more effective than regular placebo!
Can direct change in state of consciousness through specific mental techniques extend human life and reverse age-related declines? To address this question, 73 residents of 8 homes for the elderly (mean age = 81 years) were randomly assigned among no treatment and 3 treatments highly similar in external structure and expectations: the Transcendental Meditation (TM) program, mindfulness training (MF) in active distinction making, or a relaxation (low mindfulness) program. A planned comparison indicated that the “restful alert” TM group improved most, followed by MF, in contrast to relaxation and no-treatment groups, on paired associate learning; 2 measures of cognitive flexibility; mental health; systolic blood pressure; and ratings of behavioral flexibility, aging, and treatment efficacy. The MF group improved most, followed by TM, on perceived control and word fluency. After 3 years, survival rate was 100% for TM and 87.5% for MF in contrast to lower rates for other groups.
Notice that this is one of only a handful of meditation studies that carefully tries to control for expectation — the placebo effect — so it is a more robust study-design than most people are used to seeing and the findings should therefore be that much more reliable.
I cannot find a straight placebo study.
Almost like they’re the same, at differing intensity.
Why not do a study with a control that just counts their breath to 100 again and again?
When we walk slower in parks, our breathing unconsciously adjusts down to our walking pace, it isn’t woo woo magical thinking bullshit.
In a pre-industrial West, we had no problem relaxing.
An association between multiculturalism and psychological distress
Amidst increasing focus on rising rates of substance abuse and suicide among white Americans and extending prior research on intergroup attitudes and health, this study examines a novel factor associated with psychological distress: disagreement with multiculturalism. Using the Portraits of American Life Study (N = 2,292), logistic regressions indicate that for Whites and Hispanics, increased likelihood of psychological distress (depression, hopelessness and worthlessness) is associated with stronger disagreement with multiculturalism, measured as “If we want to create a society where people get along, we must recognize that each ethnic group has the right to maintain its own unique traditions.” For Blacks, however, attitudes toward multiculturalism are not associated with psychological distress. Future research might determine if these results can be replicated, and if so, identify the causal mechanism(s) at work
Really it should be mentacide but that’s just English.*
That’s what my books say. I guess either works.
The concept of “menticide” indicates an organized system of judicial perversion and psychological intervention, in which a powerful tyrant transfers his own thoughts and words into the minds and mouths of the victims he plans to destroy or to use for his own propaganda. Modern psychiatry may deliver him several tools for this perversion. Our psychiatric standpoint toward this challenge has to be formulated. Examples of menticide are described and ways of protection indicated.
The energy equivalence was actually Tesla, as a concept, who discussed in an interview telling Einstein about it.
He also tried for an equation for the Swami. Years prior. When Einstein was a dumb little baby.
A Frenchwoman almost discovered e=mc^2 by experiment in the 18th century (she was Voltaire’s lover) but her scales were slightly off. Hollywood is a propaganda machine for America to corrupt the world with lies.
Non-nutritive amino acids are key, in case the video is shoahed.
Why isn’t there an allergy panel option, say on the skin for various plant anti-nutrients?
Autoimmune causes brain fog compliance.
Phytochemistry, another key word.
Canavanine – In every legume studied so far (100’s not studied yet), alfalfa sprouts Looks like L-arginine, tricks tRNA into putting Canavanine into protein structure in place of L-arginine. This Misincorporation makes the Protein looks like non-self…
Azetidine-2-carboxylic Acid – found in both sugar beets & Garden Beets/Beet Greens, chives, garlic, leek, onion, and shallots. Looks like proline, tricks tRNA into putting Aze in place of proline. This Misincorporation makes the protein look like non-you, therefore…
All the ‘Alt Right’, “Manosphere” types will pretend to have gotten here first.
Last week, the EMP Task Force on National and Homeland Security issued a scary report on China’s ability to conduct an Electromagnetic Pulse attack on the United States. The key takeaway, according to Dr. Peter Pry, executive director of the task force, is that China now has super-EMP weapons, knows how to protect itself against an EMP attack, and has developed protocols to conduct a first-strike attack, even as they deny they would ever do so.
imagine my shock
the pearls clutched
the tea unsipped
cold, desolate, left on a side table in a darkened hallway
the English horror
According to the Center for Strategic International Studies, China has the most active ballistic missile development program in the world, so this is doubly troubling. China used stolen U.S. technology to develop at least three types of high-tech weapons to attack the electric grid and key technologies that could cause a surprise “Pearl Harbor” attack that could produce a deadly blackout to the entire country.
Dr. Pry outlines how China has built a network of satellites, high-speed missiles, and super-electromagnetic pulse weapons that could melt down our electric grid, fry critical communications, and even takeout the ability of our aircraft carrier groups to respond.
I told ya so I told ya so
I told ya so
I am so tired of the doubters. Some people with info are actually trying to help you.
How many times need I be right?
[laughs in higher IQ than you]
Ask me how I know.
hint hint bloodlines hint
Imma do a barrel role of adjacent Told Ya Sos for the new decade. A handful.
I quote myself literally all the time, you just don’t see how.
“Multiple Asians have stolen defense information from American universities and you still let them in, rocket information, only reason they have rockets (and gave some to Korea).
All of this is fact if you only look into it. They’re not smart enough to develop any of this alone.”
“As we noted here, China has created over three times as much money supply as the US since 2008.
Yeah but they’re gonna take over the world, right?”
“Tiny houses – below the legal international levels required for sanity and happiness. Prisons you pay for. And you’re proud of this? Buy a caravan FFS. Car + bed = win on two huge life expenses.” “Stocks go up because national debt goes up. Wake up, this is Communism.”
this, in 2019
I’m kind of a big deal. Actually.
Given this timeline, I’m starting to run out of major drops. Been here years.
“At around 90%, every economy declines. That is a rule of history and economics. That is the tipping point. There will be no recovery.
Choose self-reliance, panic a socialist today.
The promises of socialism are communism in a silk glove. Yes, it’s soft – now.”
“They want us crawling over one another like worms, begging them to enslave us.”
WHERE IS THE LIE
“I don’t need to advertise. Why do I want this on public record, bitch?” source
America needs infrastructure, like the whole West, it’s crumbling and almost gone. It took the best part of a century, but we’re pretty much there. You cannot get the funding or public will for it without solid currency. The Immigration Act and hullabaloo over the Gold Standard happened strangely proximate to one another, didn’t they?…..
Say, don’t you want to arrange your money on this ID?
Let’s ban cash.
Conflict: not wanting a war but wanting to see the weebs drafted.