Selective breeding is literally Darwin tho…

Am I gonna have to be the one to say this? Apparently. Ugh.

It’s no more novel than dog ‘breeds’ aka races. They also have inbreeding depression from the admixture (why it’s called mixing) and mixed race (aka mongrel) fertility issues, like ligers.

Anyone who denies this fact about selective breeding literature is literally anti-Darwin (or just plain ignorant) and against the evolutionary paradigm itself, in biology.
It’s not just in Charles Darwin’s most famous work, it’s cited in chapter ONE, you brainlets!


Teach the book in biology or none of the subject makes sense. It’s the paradigm of brain development, we can see it in scans! It’s definitely at least somewhat real when it dictates how a precious foetus develops.

Proof or it didn’t happen:

https://www.gutenberg.org/ebooks/2009
“The 6th Edition is often considered the definitive edition.” so STFU.

pre-contents: “In 1843-44 Professor Haldeman (“Boston Journal of Nat. Hist. U. States”, vol. iv, page 468) has ably given the arguments for and against the hypothesis of the development and modification of species: he seems to lean toward the side of change.”

“CHAPTER I. VARIATION UNDER DOMESTICATION.”

Literally the sodding chapter, and the first one!


Also, humans are a species, stop calling us a race. Homo Sapiens is a SPECIES.

It’s a quote gold mine!
and I don’t just mean the Sub-title:

“THE PRESERVATION OF FAVOURED RACES IN THE STRUGGLE FOR LIFE.”

Fitness is real, yo.

e.g.
“When we look to the hereditary varieties or races of our domestic animals and plants, and compare them with closely allied species, we generally perceive in each domestic race, as already remarked, less uniformity of character than in true species. Domestic races often have a somewhat monstrous character; by which I mean, that, although differing from each other and from other species of the same genus, in several trifling respects, they often differ in an extreme degree in some one part, both when compared one with another, and more especially when compared with the species under nature to which they are nearest allied. With these exceptions (and with that of the perfect fertility of varieties when crossed—a subject hereafter to be discussed), domestic races of the same species differ from each other in the same manner as do the closely allied species of the same genus in a state of nature, but the differences in most cases are less in degree. This must be admitted as true, for the domestic races of many animals and plants have been ranked by some competent judges as the descendants of aboriginally distinct species, and by other competent judges as mere varieties. If any well marked distinction existed between a domestic race and a species, this source of doubt would not so perpetually recur. It has often been stated that domestic races do not differ from each other in characters of generic value. It can be shown that this statement is not correct; but naturalists differ much in determining what characters are of generic value; all such valuations being at present empirical. When it is explained how genera originate under nature, it will be seen that we have no right to expect often to find a generic amount of difference in our domesticated races.”

For those who missed the obvious, mixed is not a race. They’re raceless. To argue otherwise is category error because a race is a mutually exclusive classification.

re de novo mutations:
“Some naturalists have maintained that all variations are connected with the act of sexual reproduction; but this is certainly an error; for I have given in another work a long list of “sporting plants;” as they are called by gardeners; that is, of plants which have suddenly produced a single bud with a new and sometimes widely different character from that of the other buds on the same plant. These bud variations, as they may be named, can be propagated by grafts, offsets, etc., and sometimes by seed. They occur rarely under nature, but are far from rare under culture. As a single bud out of many thousands produced year after year on the same tree under uniform conditions, has been known suddenly to assume a new character; and as buds on distinct trees, growing under different conditions, have sometimes yielded nearly the same variety—for instance, buds on peach-trees producing nectarines, and buds on common roses producing moss-roses—we clearly see that the nature of the conditions is of subordinate importance in comparison with the nature of the organism in determining each particular form of variation; perhaps of not more importance than the nature of the spark, by which a mass of combustible matter is ignited, has in determining the nature of the flames.”

This shit even applies to FLOWERS.

“The laws governing inheritance are for the most part unknown;

genetics

no one can say why the same peculiarity in different individuals of the same species, or in different species, is sometimes inherited and sometimes not so;

germline mutation or not e.g. parental age factor

why the child often reverts in certain characteristics to its grandfather or grandmother or more remote ancestor;

atavism

why a peculiarity is often transmitted from one sex to both sexes, or to one sex alone, more commonly but not exclusively to the like sex.

chromosomes and brain development

It is a fact of some importance to us, that peculiarities appearing in the males of our domestic breeds are often transmitted, either exclusively or in a much greater degree, to the males alone. A much more important rule, which I think may be trusted, is that, at whatever period of life a peculiarity first appears, it tends to reappear in the offspring at a corresponding age, though sometimes earlier. In many cases this could not be otherwise; thus the inherited peculiarities in the horns of cattle could appear only in the offspring when nearly mature; peculiarities in the silk-worm are known to appear at the corresponding caterpillar or cocoon stage. But hereditary diseases and some other facts make me believe that the rule has a wider extension, and that, when there is no apparent reason why a peculiarity should appear at any particular age, yet that it does tend to appear in the offspring at the same period at which it first appeared in the parent.

genetics are timed in expression, phenotype

he’s been proven correct ever since, it’s amazing

I believe this rule to be of the highest importance in explaining the laws of embryology. These remarks are of course confined to the first APPEARANCE of the peculiarity, and not to the primary cause which may have acted on the ovules or on the male element;

referring to gamete mutation, especially in sperm

predicting parental age factor over a century before it was mathematically confirmed

in nearly the same manner as the increased length of the horns in the offspring from a short-horned cow by a long-horned bull, though appearing late in life, is clearly due to the male element.

If Christians think humans are special as a species and foetuses while developing are too, they need evolutionary arguments for that. You’re dropping the ball by not using these facts.

Having alluded to the subject of reversion,

also regression to the mean, but Galton already covered that (legit polymath)

I may here refer to a statement often made by naturalists—namely, that our domestic varieties, when run wild, gradually but invariably revert in character to their aboriginal stocks. Hence it has been argued that no deductions can be drawn from domestic races to species in a state of nature.”

All quotes from early chapter 1.

When I use the word evolution, or related TECHNICAL words like race, my definition is correct because I read the literature which explains what these things mean.

Arm yourself with the truth.

Read the book.

Paper: Pneumonic plague as bioweapon, signs

https://www.nejm.org/doi/full/10.1056/NEJMra1409755

In this article, we review the clinical management of deliberate infection with several pathogens of greatest bioweapons concern. On the basis of historical incidents coupled with information on ease of dissemination, contagiousness, mortality rates, public health impact, ability to engender panic, and the need for special preparedness,1-3 the Centers for Disease Control and Prevention (CDC) stratifies pathogens and toxins into three risk categories — A, B, and C — with category A meriting the highest level of concern and preparedness.4,5 In this review, we consider diseases that are caused by category A agents for which there are high-quality clinical data in the unclassified literature (see the Supplementary Appendix, available with the full text of this article at NEJM.org). The category A viral hemorrhagic fever viruses are beyond the scope of this review.

Pneumonic Plague

Pneumonic plague is caused by infection with the fleaborne bacterium Yersinia pestis. This organism, found worldwide and responsible for the “Black Death,” can cause several forms of illness: bubonic (the most common) (Figure 1D), septicemic, and pneumonic plague.41 Because of the focus of this review, only pneumonic plague is discussed.

How fucking fascinating.

CARDINAL FEATURES OF PNEUMONIC PLAGUE

In a deliberate attack, primary pneumonic plague — rather than secondary spread from bubonic or septicemic forms — would occur 1 to 3 days after inhalation of the released bacterium or after droplet transmission from another infected person. The initial presentation of pneumonic plague is nonspecific and is difficult to differentiate from an ordinary pneumonia in its early stages. Hemoptysis, a unique feature, might be present, and rapid progression to respiratory failure and death would occur with greater frequency than in ordinary pneumonias.41

I imagine this would make the death count impossible to distinguish from regular pneumonia.

Remember: “Hemoptysis, a unique feature, might be present…”

About that….

https://tribune.com.pk/story/2146154/1-coronavirus-scare-looms-karachi/

“Maintaining that the 2019-nCoV may cause mild to severe respiratory disease, initially clinically presented as fever, dry cough, myalgia (muscle pain), fatigue and gradually progressing to a more severe productive cough that produces phlegm, episodic headaches, hemoptysis (coughing up blood) and occasional diarrhoea.”

http://www.china.org.cn/china/Off_the_Wire/2020-01/26/content_75650590.htm

“About 55 percent of the patients developed dyspnoea. Less common symptoms were sputum production, headache, hemoptysis and diarrhea.”

https://www.msn.com/en-us/health/medical/is-it-a-cold-the-flu-or-coronavirus-how-to-tell-the-difference/ar-BBZzIsH

“Common coronavirus symptoms can include: — Fever — Dry cough — Shortness of breath — Aching muscles — Fatigue

Less typical coronavirus symptoms: — Phlegm buildup — Headache — Hemoptysis — Diarrhea ”

What PP is versus what we’re told CV is by the MSM.

“Hemoptysis, a unique feature…”

Why has nobody else done it this way? aka the empirical one

look at signs, look at symptoms

unique feature…

u n i q u e   f e a t u r e

I can’t be the only smartass.

But if I must.

DIAGNOSIS OF PNEUMONIC PLAGUE

Because the clinical features of pneumonic plague are nonspecific, diagnosis is largely based on the results of culture. Sputum, blood, or lymph-node aspirates could yield positive culture results. Chest radiography would reveal a severe pneumonic process. Serologic testing can also be useful but would not play much of a role during acute illness.41 Rapid antigen tests are available in regions in which plague is endemic, but none are FDA-approved.

Oh no!

So we’d see tests be useless early on…

and… a shortage of testing kits…

especially for America… who I imagine would call some state of emergency.

TREATMENT AND PREVENTION OF PNEUMONIC PLAGUE

The treatment of pneumonic plague involves a 10-day course of an aminoglycoside antibiotic agent, such as streptomycin or gentamicin. Doxycycline is considered a second-line treatment.41 However, a randomized, controlled trial of potential treatments for bubonic plague revealed equivalency between gentamicin and oral doxycycline; it is unclear whether these results can be extrapolated to pneumonic plague.42 There has been increased interest in the use of fluoroquinolones as primary treatment in mass-casualty settings.42 A 7-day course of doxycycline or ciprofloxacin would be used as postexposure prophylaxis.41 No vaccine against plague is available. Because pneumonic plague can be transmitted from person to person through respiratory droplets, droplet precautions must be implemented for all patients.41

Why do the Chicoms want disease-ridden people in hospitals that can supposedly do nothing for them?

https://www.medscape.com/viewarticle/500621_2

..supplemented with 10% fetal calf serum and penicillin/streptomycin. To make the quail QT6/ACE2 cell line, the gene encoding the receptor for SARS-CoV, human angiotensin-converting enzyme 2…

Rest behind a paywall. Great. It’s a 2005 paper about treating coronavirus.

https://www.nejm.org/doi/full/10.1056/NEJM199709043371004

Yersinia pestis is the causative agent of plague, a zoonotic disease transmitted to humans through flea bites and typically characterized by the appearance of a tender and swollen lymph node, the bubo. Human-to-human transmission can occur, through either the bite of fleas (bubonic plague) or respiratory droplets, causing an overwhelming infection called pneumonic plague.

Our history books missed out that part.

Suddenly those masks don’t look so stupid.

The last plague pandemic began in Hong Kong in 1894 and spread throughout the world, establishing many endemic foci. Antibiotics and enforcement of public health measures significantly decreased the morbidity and mortality associated with the disease but did not allow its eradication. In fact, plague is now considered a reemerging disease1 ….

We report high-level resistance to multiple antibiotics, including all the drugs recommended for plague prophylaxis and therapy, in a clinical isolate of Y. pestis. The resistance genes were carried by a plasmid that could conjugate to other Y. pestis isolates. This report should serve as a warning of the risk of the spread of resistance in Y. pestis, a species previously considered universally susceptible to antibiotics.

…Strain 17/95 was resistant not only to all the antibiotics recommended for therapy (chloramphenicol, streptomycin, and tetracycline) and prophylaxis (sulfonamides and tetracycline) of plague4 but also to drugs that may represent alternatives to classic therapy, such as ampicillin, kanamycin, spectinomycin, and minocycline. The isolate remained susceptible to cephalosporins, other aminoglycosides, quinolones, and trimethoprim, and treatment with trimethoprim, despite its lack of synergism with sulfonamides, most likely led to the patient’s recovery….

https://www.nejm.org/doi/full/10.1056/NEJMoa2001316

The initial cases of novel coronavirus (2019-nCoV)–infected pneumonia (NCIP) occurred in Wuhan, Hubei Province, China, in December 2019 and January 2020. We analyzed data on the first 425 confirmed cases in Wuhan to determine the epidemiologic characteristics of NCIP.

LOL: https://www.theguardian.com/world/2019/nov/13/two-people-diagnosed-with-pneumonic-plague-in-china

identified by local hospitals using a surveillance mechanism for “pneumonia of unknown etiology” that was established in the wake of the 2003 severe acute respiratory syndrome (SARS) outbreak with the aim of allowing timely identification of novel pathogens such as 2019-nCoV.4 In recent days, infections have been identified in other Chinese cities and in more than a dozen countries around the world.5 Here, we provide an analysis of data on the first 425 laboratory-confirmed cases in Wuhan to describe the epidemiologic characteristics and transmission dynamics of NCIP.

Suspicious.

Furthermore, children might be less likely to become infected or, if infected, may show milder symptoms, and either of these situations would account for underrepresentation in the confirmed case count. Serosurveys after the first wave of the epidemic would clarify this question.

re Pneumonic plague above: “Serologic testing can also be useful but would not play much of a role during acute illness.”

If it looks like a duck, walks like a duck and quacks like a duck – sure, it could be a chicken in a duck suit for kinky reasons but I’m inclined on probability to call it a fucking duck.

delays to hospitalization were much longer, with 89% of patients not being hospitalized until at least day 5 of illness (Figure 2). This indicates the difficulty in identifying and isolating cases at an earlier stage of disease. 

re pneumonic plague, above: “The initial presentation of pneumonic plague is nonspecific and is difficult to differentiate from an ordinary pneumonia in its early stages.”

It’s the same hymn sheet. I can’t be the only one seeing this.

Our preliminary estimate of the incubation period distribution provides important evidence to support a 14-day medical observation period or quarantine for exposed persons.

re pneumonic plague, above: “In a deliberate attack, primary pneumonic plague — rather than secondary spread from bubonic or septicemic forms — would occur 1 to 3 days after inhalation of the released bacterium or after droplet transmission from another infected person.”
“The treatment of pneumonic plague involves a 10-day course of an aminoglycoside antibiotic agent, such as streptomycin or gentamicin. ”
“Because pneumonic plague can be transmitted from person to person through respiratory droplets, droplet precautions must be implemented for all patients.41

That takes about…. 14 days.

Our study suffers from the usual limitations of initial investigations of infections with an emerging novel pathogen, particularly during the earliest phase, when little is known about any aspect of the outbreak and there is a lack of diagnostic reagents.

re pneumonic plague: “The initial presentation of pneumonic plague is nonspecific and is difficult to differentiate from an ordinary pneumonia in its early stages.”

re pneumonic plague: “diagnosis is largely based on the results of culture”

Furthermore, the initial focus of case detection was on patients with pneumonia, but we now understand that some patients can present with gastrointestinal symptoms, and an asymptomatic infection in a child has also been reported.17

https://www.cdc.gov/plague/symptoms/index.html

“Pneumonic plague: Patients develop fever, headache, weakness, and a rapidly developing pneumonia with shortness of breath, chest pain, cough, and sometimes bloody or watery mucous. Pneumonic plague may develop from inhaling infectious droplets or may develop from untreated bubonic or septicemic plague after the bacteria spread to the lungs. The pneumonia may cause respiratory failure and shock. Pneumonic plague is the most serious form of the disease and is the only form of plague that can be spread from person to person (by infectious droplets).”

That’s why hide it.

https://www.mayoclinic.org/diseases-conditions/plague/symptoms-causes/syc-20351291

“Pneumonic plague affects the lungs. It’s the least common variety of plague but the most dangerous, because it can be spread from person to person via cough droplets. Signs and symptoms can begin within a few hours after infection, and may include:

  • Cough, with bloody mucus (sputum)
  • Difficulty breathing
  • Nausea and vomiting
  • High fever
  • Headache
  • Weakness
  • Chest pain

Pneumonic plague progresses rapidly and may cause respiratory failure and shock within two days of infection. Pneumonic plague needs to be treated with antibiotics within a day after signs and symptoms first appear, or the infection is likely to be fatal.”

repeating coronavirus paper:

Although delays between the onset of illness and seeking medical attention were generally short, with 27% of patients seeking attention within 2 days after onset.

Complete coincidence.

Back to CV paper generally

Early infections with atypical presentations may have been missed, and it is likely that infections of mild clinical severity have been under-ascertained among the confirmed cases.18 We did not have detailed information on disease severity for inclusion in this analysis.

In conclusion, we found that cases of NCIP have been doubling in size approximately every 7.4 days in Wuhan at this stage. Human-to-human transmission among close contacts has occurred since the middle of December and spread out gradually within a month after that. Urgent next steps include identifying the most effective control measures to reduce transmission in the community. The working case definitions may need to be refined as more is learned about the epidemiologic characteristics and outbreak dynamics. The characteristics of cases should continue to be monitored to identify any changes in epidemiology — for example, increases in infections among persons in younger age groups or health care workers. Future studies could include forecasts of the epidemic dynamics and special studies of person-to-person transmission in households or other locations, and serosurveys to determine the incidence of the subclinical infections would be valuable.14

re Pneumonic plague above: “Serologic testing can also be useful but would not play much of a role during acute illness.”

These initial inferences have been made on a “line list” that includes detailed individual information on each confirmed case, but there may soon be too many cases to sustain this approach to surveillance, and other approaches may be required.19

Study allowed by Chinese Government, who arrested 8 doctors for trying to release some piece of information, can’t imagine what.

If they’d been told to give people antibiotics for a virus though, I imagine they had some pointed questions.

Especially if the Chicoms wanted to buy time to buy up global supply and produce more.

https://en.wikipedia.org/wiki/Pneumonic_plague

“Pneumonic plague is a severe lung infection caused by the bacterium Yersinia pestis. Symptoms include fever, headache, shortness of breath, chest pain, and cough. They typically start about three to seven days after exposure.”

Long lag time.

PP can cause meningitis, which might explain the headache.

from CV article above: “”Common coronavirus symptoms can include: — Fever — Dry cough — Shortness of breath — Aching muscles — Fatigue”

https://www.cdc.gov/coronavirus/about/symptoms.html

“For confirmed 2019-nCoV infections, reported illnesses have ranged from people with little to no symptoms to people being severely ill and dying. Symptoms can include:

  • Fever
  • Cough
  • Shortness of breath”

All three just so happen to be also the symptoms of pneumonic plague.

“CDC believes at this time that symptoms of 2019-nCoV may appear in as few as 2 days or as long as 14 after exposure”

So three, three days on the low end. How familiar.

About dat headache symptom…

Click to access 23064.pdf

Coronaviruses as Encephalitis

-Inducing Infectious Agents

so why is headache not described as a symptom?

also

“In acute encephalitis, viral replication occurs in the brain tissue itself, possibly causing destructive lesions of the
gray matter, as was described after herpes simplex virus (HSV), rabies, or some arbovirus infections. ”

Yes, herpes can reach the brain. Again, I must remind you. Yes, it can.

This concludes why I’m banned from appearing on tv (pretty much).

Corona-chan hates Asian men

h/t Vox Day

Pandemics are racist but race isn’t biological, maybe?

https://www.biorxiv.org/content/10.1101/2020.01.26.919985v1.full

A comparison between eight individual samples demonstrated that the Asian male one has an extremely large number of ACE2-expressing cells in the lung. This study provides a biological background for the epidemic investigation of the 2019-nCov infection disease, and could be informative for future anti-ACE2 therapeutic strategy development.

We may dub it The Elliot gene.

…..We also noticed that the only Asian donor (male) has a much higher ACE2-expressing cell ratio than white and African American donors (2.50% vs. 0.47% of all cells). This might explain the observation that the new Coronavirus pandemic and previous SARS-Cov pandemic are concentrated in the Asian area….

Almost like the Chinese wanted to wipe out most of their own race IF there were a war.

So it’s like weaponised Asian bug spray.

http://voxday.blogspot.com/2020/01/is-coronavirus-weaponized.html

To kill off proximate enemy soldiers, if you thought like that. Didn’t the fake Jews pivot away from China? Wouldn’t they have motive to burn bridges to their former allies?

You know, it’s possible there are two parallel, similar looking conditions, right?

https://www.theguardian.com/world/2019/nov/13/two-people-diagnosed-with-pneumonic-plague-in-china

Video: Non-GPS tracking

One purpose of 5G. It’s like a prison ankle bracelet.

“Oh!” you say, “I’m fine because my phone is 2G/3G/4G!”

Except they’re decommissioning those towers, turning prepper emergency phones into bricks.

(Whether it’s legal to decomm emergency phones? Nobody seems to care).

But sure, Boris, let the Chicoms know where almost every UK citizen is at all times.

U smart Etonian bro.

I’m sure this won’t be used to put political opponents in prison.

They also want to force you cashless or put chips into the cash.

This makes old “hoarded”* cash worthless and they can inflate away your PP while making you pay any additional “taxes” they made up that morning based on whatever you buy, instantly.

The i in iPhone stands for idiot.

Two things I don’t understand the libertarians wanna keep: 1. the religious concept of a “sin tax” and 2. state-funded RE classes (really Multicultural Studies 101).

So they can see where you are, what you’re doing, control what you’re buying and monitor what you think online by viewing stats, what you say through the microphone (no battery release option in newer phones) and say online.

Yeah, China itself is SO bad, right?

We lost a war.

*Under Communism, any ownership is hoarding.

Paper: Dismantling the virus theory

Click to access Dismantling-the-Virus-Theory.pdf

Scientists must question everything and especially
what they love the most, i.e. their own discoveries
and ideas. This basic rule of scientific research helps
avoid erroneous developments and reveals the ones
that already exist. Also, we must all be allowed to
question the status quo, otherwise we would live in a
dictatorship.

but muh muh scientism! – redditfags

Moreover, science cannot be limited to a
selected number of institutions and experts. Science
can and must be conducted by anyone who has the
necessary knowledge and the appropriate methods.

Science can be considered science only if its claims are verifiable, reproducible and if they allow predictions. Science also needs external control, because, as we will see, a part of the medical sciences has lost touch with reality for quite some time.

They believe in invisible leprechaun atoms floating in nothing, popping out of existence. Also tiny strings. They can’t tell you what a field is. They’re mad, mad as hatters. But they make up equation models that can’t be verified and their real world studies make no sense. The world does make sense, they’re just wrong. It’s human error.

Square peg, they are wrong.

Anyone who has knowledge of biology and the genesis of life, of the development and functions of the tissue, of the body and of the brain, will automatically question the assumptions about viruses.

In the reality of the body and of its mechanisms, there is no place for hypothetical malignant processes.

You must be possessed. Tiny demons have besieged your body.

All biological processes, including those that can end in suffering, pain and death, are originally meant to be useful. A different approach to the virus phenomenon is possible and necessary: any layman with some background knowledge reading scientific papers about pathogenic viruses can realize that such viruses do not exist and what is being described are only typical components and characteristics of cells. This background knowledge will be provided in this article.

Remember, ebola is a virus. And they happened to have a vaccine, ready to go!

What ARE the odds?

It’s incredible, if not impossible.

Forget the delay of at least two years before you’re set for human trials, why can’t they do that process with every damn thing?

Biotech should be criminally accountable for with-holding cures, that’s all I’ll say. Currently, they are not.

They can also use corporate espionage and political contacts to with-hold funding from small competition who want a cure.

Inc. gems like:

The search for these pathogenic poisons remains to date fruitless, however, when bacteria were discovered, it was assumed that they were producing the pathogenic poisons. This supposition, called
“the germ theory”, was immediately accepted and remains very successful up to the present time.
This theory is so successful that the majority of the people are still not aware of the fact that the
so-called bacterial toxins are actually normal enzymes, which either cannot appear in a human
being, or, if they do, they never appear in such an amount as to make them dangerous.

….

Before it could be established that the “bacterial viruses” cannot kill natural bacteria, but they
are instead helping them to live and that bacteria themselves emerge from such structures, these
“phages” were already used as models for the alleged human and animal viruses. It was assumed
that the human and animal viruses looked like the “phages”, were allegedly killing cells and thereby
causing diseases, while at the same time producing new disease poisons and in this way transmitting
the diseases. To date, many new or apparently new diseases have been attributed to viruses if their origin is unknown or not acknowledged.

cough military cough

This reflex found an apparent confirmation in the discovery of the “bacterial viruses.

Don’t take the vaccine, unless you voted Hillary in which case go ahead sweetie. Take two.

The Wall beckons for everyone

It’s strange that some Americans are so youth-obsessed and narcissistic they are actually deluding themselves about their mortal body.

https://thingshealth.com/what-nobody-tells-you-about-aging-until-its-too-late/

Best part:

Now, men, don’t be alarmed. As you get older, you might also experience shrinking of the penis and testicles

That is an amazing fact I am now horrifying you with, too.

I’d heard about old man balls (they sag, like breasts) but their wives didn’t share the shrinkage thing.

Maybe the old guys were commissioning statues to feel better.

Genetic load and no, you’re not entitled to breed.

The low IQ Americans: MUH ANCESTORS
-died. Mostly died. STFU with the snowflaking outrage.

Maths below.

https://www.researchgate.net/publication/297657116_Mutation_and_Human_Exceptionalism_Our_Future_Genetic_Load

Although the human germline mutation rate is higher than that in any other well-studied species, the rate is not exceptional once the effective genome size and effective population size are taken into consideration. Human somatic mutation rates are substantially elevated above those in the germline, but this is also seen in other species.

What is exceptional about humans is the recent detachment from the challenges of the natural environment and the ability to modify phenotypic traits in ways that mitigate the fitness effects of mutations, e.g., precision and personalized medicine. This results in a relaxation of selection against mildly deleterious mutations, including those magnifying the mutation rate itself.

Actually, it’s anti-selection aka dysgenics. There is always a pressure in some direction, read Darwin?

You can’t have dystopia without dysgenics. That’s all a dystopia is.

The long-term consequence of such effects is an expected genetic deterioration in the baseline human condition,

Non-uniform.

By race and subrace.

potentially measurable on the timescale of a few generations in westernized societies,

Which metrics?

Technically you only need one truly fuck-up generation (say Boomers) to install those social policies up to 3 (living memory). This is without external group effects i.e. invasion on a genetic level, rape. So it isn’t fair to say immigration caused this, it compounds it severely. The Boomers and their outsized ingroup-gene infanticide will go down in history as mass murderers, if there’s anyone left.

and because the brain is a particularly large mutational target, this is of particular concern. Ultimately, the price will have to be covered by further investment in various forms of medical intervention.

Medicine isn’t magic. It cannot do that. We already cannot afford the current population with the present and dwindling useful tax base, let alone Japan levels of old coots living to infinity and China levels of population size.

You can’t fuck your way out of this, r-types. You can’t immigrate it either, those new entries have a lower IQ, higher overall group fertility and represent a smaller usable tax base. Debt doesn’t exist to cover this medical cost, even digital money typing. You can’t even type your way out of it. Hyperinflation would occur first, long before actually. Try running the numbers, see if you’re as smart as me. The cost of quality food is the anchor point. Of all living expenses, that one actually keeps you alive?

Don’t become a doctor, kids. Medicine bubble, heard it here first.

Hell, NHS GPs are already quitting now. Retention will only get worse. The ones who stay have lower IQ and can’t find gainful employment anywhere else. This is how socialism degrades infrastructure, the first generation the NHS seemed fine but the second, it attracted parasites to become GPs for the money and by the third, the original talented ones (by private sector standards) had retired and died, leaving training downhill from there.

Other people have explained that before. That one isn’t me.

Resolving the uncertainties of the magnitude and timescale of these effects will require the establishment of stable, standardized, multigenerational measurement procedures for various human traits.

Measurement? We’re lower IQ than ingroup Victorian ancestors by reaction time.

No action?

No correction?

No control?

No standards?

No relevant barriers to entry? Say, for breeding? At least on state funding?

Shows what they think of the producers, dunnit?

Leave the leech alone! The parasites are fine!

Yeah wait a few generations, maybe a century and hope the metrics are correctly chosen to matter!

Long after the researchers are dead so you can’t kill them for being wrong.

This is Idiocracy, even academia is full of nitwits.

We used to have a breeding license, it’s called a marriage certificate.

Below a certain IQ, you can’t actually consent to get married or breed. Maybe study that first?

No, that would be both logical and responsible.

See, I don’t just sit here bitching. I have solutions but nobody listens.

https://www.researchgate.net/publication/291734415_Mean_household_size_in_mid-Tudor_Englandclackclose_hundred_Norfolk

cites
https://www.researchgate.net/publication/313794802_Mutation_Accumulation_Theory

nb Historians and real scientists say European, liars typically say Caucasian.

For example, among European populations in the year 1600 AD the average individual had around a 25-40 % chance of dying in infancy, a 50 % chance of dying during childhood (Volk and Atkinson 2008), and only around a 40 % chance of fully participating in reproduction (Rühli and Henneberg 2013). The average family size was close to five in 1600s England (Arkell & Whiteman, 1998) -given the high rates of pre- term, infant, and child mortality, the numbers ever conceived would likely have been considerably higher. These historical Western infant and child mortality statistics are similar to those observed in contemporary hunter-gatherer populations (Volk and Atkinson 2008)

I’ll list the maths since there’s always that one idiot who “disagrees”.

Of those born, low ball:

100 – 25% = 75
75 – 50% = 37.5
37.5 – 40% = 15
15 of 100 births eventually reproduced, at best.

Your ancestors in 1600 weren’t entitled to breed either. STFU, stupid sections of America.
Natural selection is important.

RITES OF PASSAGE. TOUGH ONES.

Assuming you aren’t tradlarping?

Bear in mind, that wasn’t sex-specific and those estimates are the population i.e. they have to breed with one another.*

Less conservative estimate:

100 – 40% = 60
60 – 50% = 30
30 – 40% = 12
12 of 100 births eventually reproduced, by academic estimate. The more realistic one.

Again, stop being so entitled. Considering the odds, five kids average is actually pretty low.
The entitled brats, appealing to a tradition that’s totally ignorant and imaginary, are the spiteful mutants. In any other time period, you’d probably be dead by now. Male infant mortality is higher than female overall for humans, which hasn’t been factored in. 

And WWs 1 and 2 culled the bravest genes of that millennia selection by machine gun and sniper.

At least the bankers made mo- wait, they’ve already “run out” of fake money. Less than a century later.

What was it all for?

so

7.5%

or 7.5/100 births eventually reproduced as a couple TOPS

down to, more reasonably

6% of MEN* (or women, maybe**) compared to the grandfather’s generation.

[Father 50% reproduction as male, Grandfather 100% comparison, since all grandfathers would have bred logically.]

or 6/100 births from the total population, coupled.

Assuming 50/50 male/female birth split and flat survival, which doesn’t exist.**

Since breeding requires TWO people, America.

3 generations tops, with a 6% male survival in 1600 Europe.

BE CAREFUL WHAT YOU WISH FOR.

6% by sex.

This doesn’t further subdivide by health, wealth, religion or attractiveness.

If one surviving guy in that 100 births total was infertile or refused marriage, you can kinda see why it was a big deal.

This is why inheritance was always conditional on religion, approved choice of spouse and vitally, children.

If the Boomers wanna do some good, write into your will your kids get nothing unless actively Christian, married, with at least one child with a spouse you approve of. They won’t do it. They’ll complain about no grandkids though. That never gets old.