and their r-select offspring, too, if they have them. Quantity over quality doesn’t work in the long run, see, so don’t worry about immigrants that much. Low fitness cannot win, long-term. Mother Nature culls bachelors.
Slight repost for SEO. Nature, this year.
Impact of genetic risk score on the association between male childlessness and cardiovascular disease and mortality
Men need purpose of family or wither away. Believe no copes.
Childless men are reported to have a higher risk of cardiovascular disease (CVD) and mortality. Information on inherited genetic risk for CVD has improved the predictive models. Presuming that childlessness is a proxy of infertility we aimed to investigate if childless men inherit more often genetic traits for CVD and if combining genetic and parenthood information improves predictive models for CVD morbidity and mortality. Data was sourced from a large prospective population-based cohort where genetic risk score (GRS) was calculated using two sets of either 27 (GRS 27) or 50 (GRS 50) single nucleotide polymorphisms (SNPs) previously found to be associated with CVD. Part of the participants (n = 2572 men) were randomly assigned to a sub-cohort with focus on CVD which served as an exploratory cohort. The obtained statistically significant results were tested in the remaining (confirmatory) part of the cohort (n = 9548 men). GRS distribution did not differ between childless men and fathers (p-values for interaction between 0.29 and 0.76). However, when using fathers with low GRS as reference high GRS was a strong predictor for CVD mortality, the HR (95% CI) increasing from 1.92 (1.10–3.36) for GRS 50 and 1.54 (0.87–2.75) for GRS 27 in fathers to 3.12 (1.39–7.04) for GRS50 and 3.73 (1.75–7.99) for GRS27 in childless men. The confirmatory analysis showed similar trend. Algorithms including paternal information and GRS were more predictive for CVD mortality at 5 and 10 years follow-ups when compared to algorithms including GRS only (AUC 0.88 (95% CI 0.84–0.92) and 0.86 (95% CI 0.84–0.90), and, AUC 0.81 (95% CI 0.75–0.87) and 0.78 (95% CI 0.73–0.82), respectively). Combining information on parental status and GRS for CVD may improve the predictive power of risk algorithms in middle-aged men. Childless men and those with severe infertility problem may be an important target group for prevention of CVD.
The Wall literally kills men. It’s undeniable. The MGTOW types are an anti-natal death cult as much as SJWs, except spinsters live longer, actually. There is no accounting for this but genetics, as we see in the premature mortality of children if they do have them.
Men who wait until post–Wall to have kids, tend to have dead kids. It isn’t just paternal age, it’s poor genetic quality or fitness. Stop lying to men about this, they don’t have immortal sperm. No such thing. Don’t tell them to leave it too late while you hock them playa e-books. So the kids they do manage to have will die early, this is also seen among the mixed with rare genetic conditions.
What is known already: Semen quality is an established predictor of men’s somatic health. We can gain a better understanding of possible genetic or environmental determinants of the infertility phenotype by exploring familial aggregation of childhood mortality in relatives of men with poor semen quality.
Get rekt, you’re already dead. So are your r-type kids. This is why r-types evolved to have children as early as humanly possible, and as often. The high quality K strategy is genetically barred from them, no wonder they hate happy families, it’s like fat women hating thin ones. The kids don’t live long enough to act like a K, long-term. This is why fake Ks make me laugh and largely don’t concern me. You can’t cheat your own inferior genetic quality. Go ahead, have expensive medical bills for likely retarded or mentally ill kids, that will die before you get grandkids. See if I care.
The Lord works in mysterious ways. Or maybe you aren’t listening to Him. The sins of the father…..
“You say, ‘God stores away a man’s iniquity for his sons.’ Let God repay him so that he may know it.
“Prepare for his sons a place of slaughter Because of the iniquity of their fathers. They must not arise and take possession of the earth And fill the face of the world with cities.”
Sounds like early mortality to me!
There are many studies on weak sperm and premature mortality in men, I’m surprised really. Nature has the gold standard one because I know they’ll try to cherrypick around it. They never discuss real redpills like this, do they?
If something cut a childless woman’s lifespan in HALF, you’d be sure they’d talk about it. CONSTANTLY.
Men are, at best, dying of a broken heart in the mid-30s, early-40s because they have no family, and you hear crickets from the con artists who don’t really care about men, only wish to profiteer from them.
It directly contradicts their pure copium that ALL men have magical sexy singleton 30s (as K-type high fitness men might exclusively*), despite going outside, touching some grass and seeing how many let themselves go into grotesque swamp monsters. It isn’t just the women. Speaking to some Americans, I attributed a full decade or two onto their claimed age. I don’t think they were lying. The GMO carbs and booze age them like shit. The diabeetus fairy sprinkles them with powdered sugar.
*but Ks don’t sleep around, so it’s redundant to be hot. Rs want to be Ks so badly but slut genes lose.
CVD is the leading cause of morbidity and mortality in the US as well as in the European Union to a cost of hundreds of billion €, and responsible for 400,000 annual US deaths and 1.8 million EU deaths11,12,13. Therefore, improving CVD risk prediction and prevention is an important public health goal and is embedded in the Action Plan for the Prevention and Control of Non-Communicable Diseases in the WHO European Region for the time period 2016 to 202514.
This risk was most pronounced in childless men with high genetic risk scores having up to more than three times increased risk of CVD mortality, as compared to fathers with low GRS. Furthermore, we showed that adding information on paternal status to the GRS-based risk algorithm increases the predictive power for CVD mortality during 5- and 10-years follow-up.
Previously, by using data from a prospective cohort of 22,000 men with long follow-up from the same urban population, we were able to show that childlessness can be regarded as an independent risk marker for CVD along with other well-known risk factors2, an association previously reported by other authors4,26,27,28. In the current study we used a similar cohort from the same region of southern Sweden which provided genetic data and was specifically designed to study CVD risk. We were able to show the same effect of paternal status on CVD mortality estimates as previously published2. To the best of our knowledge this is the first study which evaluates the impact of established genetic risk scores for CVD on the association between parental status and the risk of CAD and CVD mortality.
Family size can be directly linked to the male fertility status29 and therefore male infertility is most likely overrepresented among childless men. Similarly, increased mortality and morbidity risk have been associated with impaired semen quality2 suggesting biological factors related to fertility to also play an important role for the risk of adverse health events in those men—association already established for women30.
The hypothesis of shared genetic traits for CVD and male infertility is based on the model proposed by Skakkebaek et al.18. It suggests a common mechanism, involving a combination of prenatal life exposures and adverse genetic factors to affect the future health of male fetuses making them more prone to develop subfertility as well as various diseases in adult life and to have shorter life span16,17,18,19. The mechanism suggests a primary testicular dysfunction including low testosterone—hypogonadism—as a possible mediator for the aforementioned risks31,32. Since up to 15% of the genome is directly involved in the physiology of reproduction16, disruption of non-reproductive, including metabolic, pathways likely impacts reproductive function and vice versa. However, the lack of interaction between parental status and inherited genetic risk for CVD reported by us suggests independent mechanisms when using childlessness as proxy for infertility.
Our study has several strengths but also some limitations. Comprehensive information from Swedish national registries allows for precise information on date and cause of death, emigration, disease diagnosis and represents men from all socioeconomic backgrounds. The meticulous data collection at baseline provides an opportunity to adjust for a large number of well-known risk factors for CVD. Furthermore, the genetic scores used in the analysis were previously verified as a risk factor using datafrom more than 55,000 individuals15, thus making it a reliable factor in risk estimation.
The robustness of our findings is underlined by confirmation of the findings based on MDC-CVC sub-cohort in the analysis of data from the remaining MDC subjects. In the latter analysis some additional GRS 27 subgroups showed statistically significantly increased CAD HRs, probably due to larger sample size.
therefore the risk of our cohort to reflect voluntary childlessness is low.
TLDR: Genetic r.
Solution? Simple. Compel hot people to breed like military service and ban the ugly.
Another time judging appearances turns out to be true.
Bad to the bone: facial structure predicts unethical behaviour
Researchers spanning many scientific domains, including primatology, evolutionary biology and psychology, have sought to establish an evolutionary basis for morality. While researchers have identified social and cognitive adaptations that support ethical behaviour, a consensus has emerged that genetically determined physical traits are not reliable signals of unethical intentions or actions. Challenging this view, we show that genetically determined physical traits can serve as reliable predictors of unethical behaviour if they are also associated with positive signals in intersex and intrasex selection. Specifically, we identify a key physical attribute, the facial width-to-height ratio, which predicts unethical behaviour in men. Across two studies, we demonstrate that men with wider faces (relative to facial height) are more likely to explicitly deceive their counterparts in a negotiation, and are more willing to cheat in order to increase their financial gain. Importantly, we provide evidence that the link between facial metrics and unethical behaviour is mediated by a psychological sense of power. Our results demonstrate that static physical attributes can indeed serve as reliable cues of immoral action, and provide additional support for the view that evolutionary forces shape ethical judgement and behaviour.
So Chad is a mindset?
Wait, is looksmaxing real? Like would surgery give you the mindset? Maybe?
Maybe this is why modern women prefer softer features?
The facial WHR is a sexually dimorphic trait (with men having larger ratios than women) that is independent of body size and is argued to have evolutionary origins . Consistent with the idea that evolutionary pressures account for this dimorphism, intrasex differences in facial WHRs have been linked to aggression in men, with greater facial WHRs associated with more aggressive behaviour [10,12]. For instance, men with greater facial WHRs are more likely to retaliate to perceived slights by others  and are more likely to act in their own self-interest, even if it means violating another’s trust .
Less likely to murder us.
I love when guys try to tell women what to fancy, it’s like telling them to stop liking tits. The buff look reads homo at best, dangerous at worst. Stop telling men it’s what women want. Not fat, not veiny. It isn’t hard.
“Conclusions The risk of miscarriage varies greatly with maternal age, shows a strong pattern of recurrence, and is also increased after some adverse pregnancy outcomes. Miscarriage and other pregnancy complications might share underlying causes, which could be biological conditions or unmeasured common risk factors.”
That’s important, write that down.
aka if you go Third World and force women to start breeding too early, they’ll be more likely to miscarry healthy children in future. Mother Nature hates r-types.
“Results There were 421 201 pregnancies during the study period. The risk of miscarriage was lowest in women aged 25-29 (10%), and rose rapidly after age 30, reaching 53% in women aged 45 and over. There was a strong recurrence risk of miscarriage, with age adjusted odds ratios of 1.54 (95% confidence interval 1.48 to 1.60) after one miscarriage, 2.21 (2.03 to 2.41) after two, and 3.97 (3.29 to 4.78) after three consecutive miscarriages. The risk of miscarriage was modestly increased if the previous birth ended in a preterm delivery (adjusted odds ratio 1.22, 95% confidence interval 1.12 to 1.29), stillbirth (1.30, 1.11 to 1.53), caesarean section (1.16, 1.12 to 1.21), or if the woman had gestational diabetes in the previous pregnancy (1.19, 1.05 to 1.36). The risk of miscarriage was slightly higher in women who themselves had been small for gestational age (1.08, 1.04 to 1.13).”
LOWEST of all ranges in the mid-late 20s, which, per The World We Have Lost, happens to be the age our wiser medieval ancestors commonly married and commenced reproduction. Almost like they didn’t want their wife to die?
You can’t expect modern medicine to bail you out of degeneracy.
And forcing a woman to start “too early” (really before the pelvic growth plates fuse at 21) makes it more likely your later heirs will be miscarried too. No blaming the woman for your own impatience.
All those described factors sound r-selected, especially the C-section, which doctors shouldn’t be forcing women into for convenience. These are your future kids they’re risking.
This study isn’t precise enough because they try to dodge the teen death issue but here
“Figure 3 shows the age related risk of spontaneous abortion stratified by parity status and number of previous spontaneous abortions. The association between spontaneous abortion and age was similar in all strata, although the level increased with increasing number of previous spontaneous abortions.”
Similar. It isn’t a huge difference by age alone like you falsely claim, stop being dumb. However….
if we look at marriage survival against IQ (linked to years ago) and cross-reference the J-curve beneath, delayed motherhood (sufficient time to educate) is healthiest for society in terms of infant survival and marital longevity. Divorce is lower in high IQ women, who tend to marry later, which we can lump into the No Shit category.
Fridge horror: The early marriage of the poor CAUSED a lot of their baby deaths! aka The Oven Ain’t Done Yet!
Pedos reee but nature hates them to breed. They’re extreme r.
“The incidence of spontaneous abortion varied according to a woman’s parity and number of spontaneous abortions in the preceding 10 years; among women aged 25-29 years spontaneous abortion occurred in 8.9% of nulliparous women and 9.3% of parous women without a history of spontaneous abortion, in 12.4% and 11.8% of those with a history of one spontaneous abortion, and in 22.7% and 17.7% of those with a history of two spontaneous abortions. After three or more spontaneous abortions, the proportion of pregnancies ending in spontaneous abortion increased to 44.6% in nulliparous women and 35.4% in parous women.”
Personal history and then family history are more important than age. Men need to get this through their thick skull. This is like the IQ and beauty versus popularity and personality divide. A man who praises his wife’s ‘nice’ personality is admitting her ugliness. She isn’t docile, she doesn’t respect you. If we plan to outlive a man, what does his opinion matter? ‘Nice’ is a quality of puppies, not a viable sexual partner. Your level is the best woman you can get – and keep. Men forget the second part. Cheating on a great wife to lose her is stupid.
Widows were hot commodities because they had proven fertility. Especially great if their husband was stoned to death for adultery, so she’ll be quite young.
Do you want to bet on the horse that has won races or never raced?
If marrying a woman at the proper time, with no personal fertility history, ask about the oldest aunt of theirs who had kids.
Ideally, you’d hear 40s for a firstborn. Those are top-tier genes, especially if the child was perfectly healthy. No genetic load. Miscarriages are common though (about 10% under ideal conditions) and hard to tell early on so it isn’t an exact science. It’s odds, it’s probability. So it isn’t so much age, it’s familial genetic load of mutations compounded by time, it only seems like age. The mutations already in their DNA (and higher in men because sperm constantly need to renew) simply become more of what they already are.
The IVF people do not want normie people to discover the simple ways to ensure better fertility health, they’d go out of business if we had a simple eugenic questionnaire prior to marriage e.g. period frequency. Also, miscarriage is actually good if very early because print error kids get expensive. That’s a sign the body is doing what it should, miscarriages aren’t all created equal, only most are bad.
In future we could probably devise a spiteful mutant test prior to marriage. Very Gattaca. On second thought, that might actually be what the test was. Ks approve.
Obviously with age the mutants (only one parent need be) become more apparent, and this also determines things like aging facial bone structure too, but it isn’t CAUSED by age, it’s their genome!
Age is not the true variable, the confound is mutation burden in your DNA (inc germline). Age can estimate on a population level but I implore you, on an individual one, speak to the family for same-sex history up to cousin level, there’s a reason doctors ask about it! It allows them to adjust their predictions without prejudice.
In general women have less abortions young because 1. it counts the healthiest time to breed, the twenties, which conceals the brief increase in the teens, 2. white women conceal the worse stats for non-white women while still a technical majority and 3. they’d have less time to experience anything, there’s been less time alive. This assumes they’re even having sex. Age is a poor metric. Ask about Aunt Meryl with the four kids after 30. You may strike gold and the woman has twins in the family.
Miscarriage is a J-curve by age, NOT linear. Younger is not automatically better, learn maths dudebros.
Then we isolate the J-curve with no history:
Gee, why don’t the socialists encouraging teen pregnancies tell you this in Sex Ed class?
For my next trick, because I’m that bitch, compare the teen miscarriage line to other young women? [young being prior to middle-age, for women approx 40s]
It’s data from 1,221,546 pregnancy outcomes in a white country.
The mid-30s miscarriage risk is the same for that woman as a teen with the same history.
It’s a deeper 20s scoop if both example women had a miscarriage history of one.
Data doesn’t care about your deviance, pedos.
Mother Nature hates you. So those data-ignorant “dusty egg” jokes of mothers in their 30s should logically be applied to ‘teen whore’ types too. If you were being logical, which we all know you aren’t. Teen mothers (and fathers) also tend to have lower IQ, which suggests spiteful mutant. The data lines up perfectly.
They don’t really ‘believe’ in starting prematurely, it’s their life history strategy talking.
They feel a need to breed immediately because they know they’d likely miscarry if they waited like a K-type. Suck it?
“In women with no history of spontaneous abortions we found a slightly lower overall risk of spontaneous abortion among nulliparous women than parous women (10.0% v 11.6%). This tendency was found in all strata of age except for women aged 40-44 years. “
Again, actual women’s middle age. You’d expect that. The system is shutting up shop.
It’s slightly better to have had NO abortions than ONE. Duh? I think women would agree. So if that one spontaneous abortion would be likelier in the teens, should a fertility-oriented high IQ society encourage teen pregnancy?
The answer is clearly no.
And the Middle Ages Western Europeans were smarter than current America.
And you wonder why the white birth rate is so, so low.
“Among women with a history of spontaneous abortion, the reverse tendency was observed; in general, nulliparous women had a higher age specific risk than did parous women (fig (fig33).”
Stop getting this wrong. We need to avoid spontaneous abortions (miscarriages) to increase the birth rate. You can’t throw conceptions at the wall to see what sticks.
That’s a male perspective on women’s bodies and it’s demonstrably, mathematically wrong.
Not to mention stressful on the longsuffering wife.
Teens (biological children) have a higher pregnancy risk than adult, mature mothers:
“Under the assumption that only 80% of women with abortions in recognised pregnancies were hospitalised the risk of spontaneous abortion would be: 12-19 years, 13.3%; 20-24, 11.1%; 25-29, 11.9%; 30-34, 15.0%; 35-39, 24.6%; 40-44, 51.0%; and 45 or more, 93.4%.” that’s :-
Minor: 13.3% natural abortions
20s: 11.5% natural abortions
30s: 19.8% natural abortions (average, more variation)
40s: basically at least half. You’d need top tier DNA to survive that.
So stop lying, pedos. Call yourself hebe all you like, a POS by any other name.
This doesn’t factor in the mental trauma of giving birth, PTSD is quite common, discounting obvious cases like episiotomies without cause and C-sections with no pain relief. It happens.
Obviously, traumatising your teenage girls will put them off breeding altogether.
Then what happens to your precious ego birth rate?
The teen ectopic pregnancy rate also peaks in the teens comparable to a near-thirty year old.
DAT J-shape curve.
You mad, pedos?
Wait, there’s more!
Now onto stillbirths:
The rate for minors (teens) peaks at the same level as women in their late 30s.
That’s gotta hurt.
Good luck with your scientism though.I’m sure 1M+ white births are lying.
DAT 20s dip:
and it’s fractions of a percent, hardly apocalyptic is it? They’re such special snowflakes with the bloody victim complex.
“The association between maternal age and stillbirth showed a J-shaped curve, but the effect of age was less than for spontaneous abortions and ectopic pregnancies (fig (fig5).5). When restricting the analysis to nulliparous women, we found an identical pattern, although the level was slightly higher. The proportion of stillbirths was substantially increased in teenage pregnancies and was at the same level as for the 35-39 year age group. The incidence of stillbirth was unchanged during the study period.”
I’d also like to see a subdivision of dead babies risk in teen/minor mothers by aged daddy. Maybe next time. I covered paternal age generally beforehand anyway.
It’s funny that the paper writers still try to make it about age though. Nice try. Miscarriage is the biggest factor in future fertility according to their actual data, age is more important for niche risk of ectopic and stillbirth, but less so. And most importantly, NONE OF THIS IS LINEAR. NONE OF IT. The curve is a J. Redpills read the data. I don’t care what the researchers claim to get gibs, read the data itself. It is a non sequitur to claim older = worse outcomes and also a non sequitur to claim younger = better outcomes when the data doesn’t show that, it blatantly shows the opposite, a kind of Goldilocks effect in the 20s.
To put this all on increasing age is false reasoning, as shown, it’s increasing mutant burden. Age is a vector of genetic load, not the cause. Like – Being in a car is a vector of drunk driving, it isn’t the alcohol!
But they wanna get cited so…. they’ll twist their own data. Or try? God forbid anything be genetic, even reproduction!
nb “The increase in risk of ectopic pregnancies in teenage women is most likely caused by pelvic inflammatory disease.”
Teenagers are not women but k. And that’s wrong. The female human reproductive system takes time to fully develop. r/K explains this. Inflammation takes years, it’s literally impossible to blame that or 20s would be still higher.
“The risk of stillbirth was found to be high among teenagers, as previously reported.24 This may be a result of unfavourable social and behavioural conditions among pregnant teenagers, although a biological explanation cannot be excluded. The risk of stillbirth among women aged more than 35 years was increased but to a lesser extent….”
Our study shows an important increase in the risk of spontaneous abortion and other types of fetal loss among women aged more than 40 years”
Middle-age, then? Duh? The body’s aborting print errors like it should?
Yeah because like I said about the r/K system starting up, it also takes years to wind down?
Why aren’t you getting this?
“increase is already considerable among those in their 30s.”
no it isn’t data varies too much in that decade so you cannot accurately comment
“This increase is observed irrespective of a woman’s reproductive history.”
but that’s the bigger effect size? it’s the objectively more important factor?
Can’t hurt feels or lose those IVF shekels, huh?
The effect is still there but that’s a curious omission of scale.
“For society, such findings would indicate that tendencies to postpone pregnancy increase the overall incidence of fetal loss and possibly the costs of health care.”
ooooh they’re pushing teen pregnancies
“overall” POPULATION is not filial risk (personal risk)
filial risk is genetic, kin based
socialists shouldn’t be allowed to science
postponing in a K-select manner is MATURING
higher actual birth rate, higher maternal safety, higher child survival
healthier children! higher IQs!
WHAT IS THE DOWNSIDE
= fewer r-types, I weep!
“these factors are highly correlated” = NOT CAUSATION
for the reproductive equation, you must include the age of BOTH parents at conception
That’s the genetic equation of causation. Single parents are not up for discussion here, they didn’t impregnate themselves?!!
12-19 (minor/teen) pregnancies, not aborted: 51,132.
That’s a huge dataset of adverse pregnancy outcomes. How will the hebes recover?
“It might sound strange, but its true, this remedy has been passed around the feminist community since the 70’s, appearing in many grassroots publications, some of which are cited here. There are also numerous reports of women using it successfully from this era, I’ve heard many stories, but never saw any kind of documentation, which isn’t surprising in a time, where a woman’s right to choose an abortion and have access to safe legal abortion services was just being won.”
Great for ye olde days of gang rape though. Useful if the Red Army comes around town. Abortion does make sense where continuing would kill the mother so there is an ethical grey area e.g. ectopic. I acknowledge that. We also must know what kills a baby so all mothers know to AVOID it. This is why keeping women ignorant leaves them vulnerable to such evil. Parts of nature hate us. Wiccans are imbeciles.
This is why I don’t supplement liposomal Vitamin C, as I suggested for OLDER people.
“The scientists who conducted the research, Samborskaia and Ferdman came to the conclusion that high doses of Ascorbic Acid appeared to increase estrogen levels which contributed to the interruption of an otherwise normal pregnancy. 20 women who approached doctors requesting an abortion participated in the study. Research was conducted by ob/gyn L.I. Ivanyuta. The women ranged from 20 to 40 years of age. The article does not say if a positive pregnancy test was obtained from the participating women. We also don’t know how much ascorbic acid the women were given. They did however measure estrogen levels before and after treatment with ascorbic acid, finding that estrogen levels were higher after taking the ascorbic acid. Of the 20 women, 16 began menstrual type bleeding within 1 to 3 days from administration of ascorbic acid.”
It makes giving kids lemonade real sinister. Mountain Dew, Sunny D, the works.
“Vitamin C works to produce an unfavorable climate within the uterus so that the egg does not implant, or if implantation has already occurred, Vitamin C can weaken the fertilized ovum’s grip on the uterine wall. Possibly by stimulating estrogen, and interfering with progesterone. This also makes it useful as an emergency contraceptive, when taken before implantation occurs on the 6th day following ovulation. The hormone, progesterone is essential for pregnancy, its function is to prepare a nourishing bed for the fertilized egg, if there is not enough progesterone the uterus becomes less supportive to the egg. Which is desirable when the goal is to end pregnancy.”
Progesterone means pro-gestation. Anything that reduces that and/or increases oestrogen causes miscarriage, including xenoestrogens. BPA also causes genetic defects inc. Downs, and can cause abnormal egg development in a female fetus, which can go on to experience many miscarriages (modern rates?) and Downs children themselves.
Also NO parsley. Yes, it kill babies. Viva Italia some other time. Can be used to induce labour, ironically.
History will view the use of xenos as pure evil*. I think endometriosis is caused by it, like a poisoning. Explains the miscarriage common to it. Most common cause of infertility in women. Pure progesterone creams hard to come by, easier to patent a toxic variety close enough. Even pure creams can include preservatives that are oestrogenic! Vegan love of vit C may cause vegan menopause, imho. Xenos also cause premature puberty in girls as young as ONE, especially seen in high-estro skin products used by American blacks and not found in African ones. Xenos (including hops in beer**) also cause a small penis and breast development in boys/men. This shit should be BANNED forever in all skincare vehicles (10x more potent, bypassing liver filter). The amount required (parts per billion) is rarely tested for but maintains estrogenic effect at this level. Parabens were disused in some products due to this. Others like SLS and phthalates also. It isn’t hype, it’s killing men/women hormonally and babies silently. A silent killer in shampoo, lotion, food etc. No wonder American rates of miscarriage are so high. Test ALL skin products for endocrine disruption, especially those that break down into it (XENOS), in rats. Xenos can bio-accumulate for decades in the body (heard of DDT?) and stay for decades too. I share this hoping people won’t abuse the info.
*file under Molech
**how Anglos have gotten softer and softer and softer… literally and morally.
Synthetic perfume is also a xeno. Sorry. I’m sad about it too. They’re aiming this at teen girls and boys, who get fat. And in the case of girls, look sexual. The boys look twinkish. I’m sure the traffickers love that.
They blame kids for being fat when they’re hormonally drugged from seemingly everywhere. They cannot lose weight! The environment is too polluted!
Phyto-estrogen can bind protectively and reduce the capacity of xeno to attach. This is limited. It’s less potent but still oestrogenic and thus reduces progesterone. Can detox from the body in a matter of days since it’s natural.
The UK is trying to push a chemical abortifacient over the counter. No minimum age limit, paedo paradise. How many rape gangs or child groomers will feed this to their prey, coercive or secretly? The capacity of even parents for reproductive abuse, secretly giving it to their children like a harmless preventative (when they might already be taking it themselves) is abhorrent. There are no proportionate laws criminalizing giving this to children. Mull over that.
““Pharmacists have the expertise to advise women on whether desogestrel is an appropriate and safe oral contraceptive pill for them to use and to give women the information they need, to make informed choices,” she said.” It isn’t only women who will buy it, fathers raping their daughters, rape gangs attacking girls en masse, controlling mothers drugging their daughters ‘just in case’…. pharmacists are not doctors and cannot order appropriate tests to maintain healthy hormone levels. You cannot make informed choices as a minor by definition.
“The MHRA’s decision to reclassify the desogestrel products follows a safety review by the Commission on Human Medicines (CHM) and a public consultation taking in views from patients, pharmacists and doctors.” Patients are idiots and so are pharmacists. They’ll opine when they’re damaged, but why didn’t you warn us?
“Edward Morris, the president of the Royal College of Obstetricians and Gynaecologists, said he was delighted that some contraceptive pills would be available in local pharmacies after the college’s years of campaigning against “unnecessary barriers” for women and girls.” Women and girls. No minimum age noted.
He said: “Even before the pandemic, too many women and girls were struggling to access basic women’s health services. The consequences of this include an increase in the number of unplanned pregnancies, which can result in poorer outcomes for women and their babies.” These people are child rape enablers. Apparently the TRUE evil (sarc) is holding child rapists accountable for the fruit of their loins. This is rape culture if the term ever applied.
Saving money on actual medical advice again, cost cutting: “She said: “The fragmented sexual and reproductive healthcare system is notoriously difficult for women to navigate, and successive cuts to public health budgets have made it harder for women to get the contraception they need. Reclassification may also reduce unnecessary pressures on GPs, who will not need to see patients for repeat prescriptions.” They need to monitor anything hormonal. When are steriods OTC? No? They have noted health benefits.
The Pill’s push to the unmarried will go down in history as true evil. Yes, it counts as abortion. They continue to lie about its basic mechanism. They already pushed the morning after form OTC. Long term use of any medication requires doctor supervision to ensure the organs aren’t being poisoned. Oestrogen dominance is rising amid well, everyone, and causes severe damage including obesity. Progesterone can mess with all other hormone levels. The idea of drugging just women/girls with this shit is misogyny. It’s purely misogynistic. You must hate them to endorse this. Are we selling testosterone and other supplements OTC at drug concentrations to anyone, whatever the age? This is tacitly enabling child rapists and pretending children can consent to be raped. It places blame squarely on the child. It enables the continual abuse of children under the guise of ‘responsibility’. There will now also be social pressure to conform among girls. It isn’t harmless. If you have to ban sugar lollipops at schools for being ‘bad for health’, this is nothing less than evil to push. A gyno on GB News, claiming to be a patriotic TV station here, had her go on about ‘painful periods’ – which is no excuse to drug the reproductive organs of a developing child. You also know the BPD parents will be forcing this onto their sons like tranny abuse of the drug class for ‘therapy’. Even if a girl has painful periods, painkillers already relieve that, as I well know. There are no longitudinal studies on the damage of chronic Pill use. I repeat, no long term studies. In adult starters, let alone minors. The Guardian even sub-links to an article about blood clots caused by the Pill.
Tracking Pill use is vital to monitor whether a woman is being abused, and whether SHE actually wants to take a medication, rather than being drugged. She must attend her own appointment and talk to a doctor, bearing in mind her medical history. This system of protection prevents coercion. They tried pushing this shit on me for no medical reason as a minor and virgin (obviously) so I know they do it. Thankfully I understood what longitudinal studies were and told them like three or four times on separate occasions, no I don’t want their drug pushing (I had a normal ovarian cyst*, the first time – making it completely random and insulting to suggest) and no amount of fear mongering or ‘you’ll be sorrys’ will make me anything less than really really sure. The longest I had to repeat this was about twenty to twenty five minutes. They don’t take no for an answer. They don’t even care if your family has an extensive history of damage reactions to hormones. They will still look you dead in the eye and gaslight you that it’s ‘really harmless’. Based upon??? They are actively PUSHING this shit onto women/girls. Painful periods require giving up sugar half a week before it starts, not this bullshit. But they want to save money and face on child abortions. It’s difficult to call a girl a liar if she’s literally pregnant. This is all about enabling child abusers and disempowering victims, who have no legal recourse against food adulteration and coercion when it’s available like a breath mint. No proof of crime, no time.
*one or two cysts happen naturally when you ovulate but they’re so accustomed to seeing women and girls drugged up to the eyeballs that they treated this like I needed meds. More like they wanted kickbacks.
Socialism kills. All Marxism is anti-natal, it’s the only thing it has in common. Hence, national socialism is fucking retarded, sorry. It doesn’t work. You can’t have a half-evil system. It makes people lazy, as we see:
A number of population scholars have asserted that social security programs such as old-age programs lead to decreased fertility levels because parents need not rely on children for “security” in old age. There is, however, a paucity of empirical data on the above. This paper analyzes 67 countries and shows that social security programs have a measurable negative effect on subsequent levels of fertility.
This might go down in history as the thing to doom the Boomers, demographically.
In fact, the social security programs appear to have as much of an independent impact on fertility as do the traditional correlates of fertility (infant mortality, education and per capita income).
Explains the sheer drop in fertility in places like Poland, and why places like Russia and China are currently bricking it.
Then you have fake Catholics like the French, fucking around and never marrying because there’s no stigma any longer. Socialism preserves a husk of civilization while hollowing out the core.
It’s popular for insecure morons to say higher education should be banned but it replaces ballrooms for the high IQ to meet one another. Education is K-select, that’s why Marxists have tried to destroy it. Would you rather they screw the local waitress and have dipshit kids? On your tax bill? Assortative mating must be enabled for a healthy, functioning society. Ban foreign students and there’s more room by far. Then you don’t have national security risks like the Chinese stealing missile data AMERICA.
College educated parents (yes, both) have higher IQ children. The right wing gets triggered over this and other facts where the universe doesn’t revolve around men (or women, but they don’t care there). As covered previously, most child IQ is inherited maternally. So we know it’s men to blame for fucking the dumb broads, who exhibit sexual selection like a leech being discerning in a blood bank. Stop fucking stupid women. At least, don’t be surprised when the kids are entitled douchebags. If your kids are stupid, and you think it isn’t you – it’s your choice of woman.
Evaluation of human papilloma virus in semen as a risk factor for low sperm quality and poor in vitro fertilization outcomes: a systematic review and meta-analysis
A review of the literature regarding ART outcomes showed an association between HPV infection and decreased PR, and an even stronger association between HPV infection and increased MR.
-increased miscarriage rate, lower odds of conceiving
Conclusion: Our meta-analysis shows a negative effect of HPV on sperm concentration, motility, and morphology. Further subgroup and categorical analysis confirmed the clinical significance of impaired sperm motility in HPV-infected sperm, although the sperm count and morphology must be carefully analyzed. The studies reviewed reported lower PR and increased MR in couples with HPV-infected sperm. As most studies had a moderate risk of bias, these observations warrant further large, well-designed studies before introducing clinical management recommendations.
Human papillomavirus infection and fertility alteration: a systematic review
Results: HPV infections are shown to be significantly associated to many adverse effects in the reproductive function. These adverse effects were reported in different levels from cells production to pregnancy and may be related to the infecting genotype.
Conclusions: It appears from this study that HPV detection and genotyping could be of great value in infertility diagnosis at least in idiopathic infertility cases. Like for the risk of carcinogenesis, another classification of HPV regarding the risk of fertility alteration may be considered after deep investigations.
Human Papilloma Virus (HPV) and Fertilization: A Mini Review
Sorry but if something makes you less virile, you’re less of a man.
Human papilloma virus (HPV) is one of the most prevalent viral sexually transmitted diseases. The ability of HPV to induce malignancy in the anogenital tract and stomato-pharyngeal cavity is well documented. Moreover, HPV infection may also affect reproductive health and fertility. Although, the impact of HPV on female fertility has not been thoroughly studied it has been found also to have an impact on semen parameters. Relative information can be obtained from studies investigating the relationship between HPV and pregnancy success. Furthermore, there is an ongoing debate whether HPV alters the efficacy of assisted reproductive technologies. An association between HPV and assisted reproductive technologies (ART) programs has been reported. Nevertheless, due to conflicting data and the small number of existing studies further research is required. It remains to be clarified whether HPV detection and genotyping could be included in the diagnostic procedures in couples undergoing in vitro fertilization (IVF)/intrauterine insemination (IUI) treatments. Vaccination of both genders against HPV can reduce the prevalence of HPV infection and eliminate its implications on human fertility. The aim of the present mini-review is to reiterate the association between HPV and human fertility through a systematic literature review.
The role of human papillomavirus on sperm function
I love how many yanks pull a Henry 8th and blame women for their own infertility, in this century.
Recent findings: HPVs are agents of the most common sexually transmitted disease and can lead to warts and cancers both in men and women. A high incidence of HPV infection has been demonstrated in sperm from sexually active men with and without risk factors for HPV and from infertile patients.
Semen infection is associated to an impairment of sperm parameters suggesting a possible role in male infertility. – really???
Interestingly, it has been demonstrated that when HPV is present in semen only a percentage of total cells are infected
-only? a? 100% is a percentage too…
and the virus can be localized in sperm or in exfoliated cells with different impact on sperm motility. Moreover, infected sperm are able to penetrate the oocyte, to deliver HPV genome in the oocyte and HPV genes can be actively transcribed by the fertilized oocyte.
-wouldn’t it be ironic if it made the kids or grandkids infertile instead? because they were conceived with it, a polluted germline
Recently an increased risk of pregnancy loss has been demonstrated in couples undergoing in-vitro fertilization and particularly when HPV DNA was present in semen samples of male partners.
– no blaming women this time, unless women haz sperm?
Summary: To date, no effective treatment, control strategy and prevention is provided for men despite the reported high incidence of HPV semen infection.
– no hurt their feefees? NAW
Because this infection in men is also a problem for partners, and because growing evidence suggests that semen infection may cause infertility and early miscarriage, more attention should be paid to male HPV infection. This study reviews the more recent literature about the role of HPV infection on sperm function and human reproduction.
– Manosphere fears this topic and all male degenerate accountability.
semen infection may cause infertility and early miscarriage
High-risk human papillomavirus in semen is associated with poor sperm progressive motility and a high sperm DNA fragmentation index in infertile men
Does the presence of human papillomavirus (HPV) in semen impact seminal parameters and sperm DNA quality in white European men seeking medical help for primary couple’s infertility?
>STD >DNA quality >in the germline of >white men
Never talk about it, I’m sure it’ll be fine.
HPV seminal infections involving high-risk (HR) genotypes are associated with impaired sperm progressive motility and sperm DNA fragmentation (SDF) values.
HPV is commonly present in semen samples.
No? F no it’s not. Stop sparing slutty blushes.
The overall rate of HPV positivity was 15.5%
so 1 in 7, uncommon at best. No normalizing pathology please.
And it varies majorly by race and sexuality. Not sex because it’s sexual, obviously.
Sperm progressive motility was significantly lower (P = 0.01) while SDF values were higher (P = 0.005) in HPV+ men compared to those with no HPV. In particular, HR HPV+ men had lower sperm progressive motility (P = 0.007) and higher SDF values (P = 0.003) than those with a negative HPV test. Univariable analysis showed that HR HPV+ was associated with impaired sperm progressive motility (P = 0.002) and SDF values (P = 0.003). In the multivariable analysis, age, FSH levels and testicular volume were significantly associated with impaired sperm progressive motility (all P ≤ 0.04). Conversely BMI, CCI, smoking habits and HPV status were not. Only age (P = 0.02) and FSH (P = 0.01) were significantly associated with SDF, after accounting for BMI, CCI, testicular volume, smoking habits and HPV status.
Impact of human papillomavirus infection in semen on sperm progressive motility in infertile men: a systematic review and meta-analysis
Background: Human papillomavirus (HPV) has been considered as one of the most common sexually transmitted viruses that may be linked to unexplained infertility in men. The possible mechanisms underlying correlation between HPV infection and infertility could be related to the altered sperm parameters. Current studies have investigated the effect of HPV seminal infection on sperm quality in infertile men, but have shown inconsistent results.
Methods: We systematically searched PubMed, Embase, Web of Science and CNKI for studies that examined the association between HPV seminal infection and sperm progressive motility. Data were pooled using a random-effects model. Outcomes were the sperm progressive motility rate. Results are expressed as standardised mean difference (SMD) with 95% confidence interval (CI). Heterogeneity was evaluated by the I-square (I2) statistic.
Results: Ten studies were identified, including 616 infertile patients with HPV seminal infection and 2029 infertile controls without HPV seminal infection. Our meta-analysis results indicated that sperm progressive motility was significantly reduced in HPV-infected semen samples compared with non-infected groups [SMD:-0.88, 95% CI:-1.17 ~ – 0.59]. There existed statistical heterogeneity (I2 value: 86%) and the subgroup analysis suggested that study region might be the causes of heterogeneity.
Conclusions: HPV semen infection could significantly reduce sperm progressive motility in infertile individuals. There were some limitations in the study such as the differences in age, sample sizes and the number of HPV genotypes detected. Further evidences are needed to better elucidate the relationship between HPV seminal infection and sperm quality.
The prevalence of Human Papilloma Virus (HPV) infection in the oligospermic and azoospermic men
The current study shows that HPV infection can affect on sperm count and motility and decrease count of sperm cell and decrease motility capability of these cells.
Among 50 confirmed oligospermic male, 15 were HPV DNA positive (30%).
In azoospemic group we had 8 HPV DNA positive (40%) and in normal group just 3 of 20(15%) samples were positive.
-what r the odds?
we found statistical significant relationship for sperm count (p<0.05) and sperm motility (slow) (p<0.05) in oligospermic group positive samples compared with negative. In the present study, 13 HPV genotypes were detected among positive samples. HPV genotypes 16, 45 in the high risk group and 6,11,42 in the low risk group were more frequent than the others.
Semen washing procedures do not eliminate human papilloma virus sperm infection in infertile patients
had HPV DNA on sperm and exfoliated cells. Sperm washing centrifugation showed no changes in the number of infected samples and in the percentage of infected cells. Ficoll and swim-up protocols induced a slight reduction in the number of infected samples (30 and 26, respectively).
no muh scientism and IVF cope
This study demonstrated that conventional sperm selection rarely eliminates HPV sperm infection. More attention should be paid to the reproductive health of infected patients because, not only can HPV be transmitted, but it may also have a negative effect on development of the fetus.
a negative effect on development of the fetus
so even if they all married a virgin waifu, they’d infect her and have defective babies comedy GOLD, 24K.
Is HPV the Novel Target in Male Idiopathic Infertility? A Systematic Review of the Literature
Infertility is an important health problem that affects up to 16% of couples worldwide.
1 in 7, where have I heard THAT before….? [scroll up]
Male infertility is responsible for about 50% of the cases,
–NAY, men are never responsible for their own in/fertility, have you been online recently?
and the various causes of male infertility may be classified in pre-testicular (for example hypothalamic diseases), testicular, and post-testicular (for example obstructive pathologies of seminal ducts) causes. Sexually transmitted infections (STI) are increasingly widely accepted by researchers and clinicians as etiological factors of male infertility. In particular, several recent reports have documented the presence of HPV in seminal fluid and observed that sperm infection can also be present in sexually active asymptomatic male and infertile patients.
In this review, we aimed to perform a systematic review of the whole body of literature exploring the impact of HPV infection in natural and assisted fertility outcomes, from both an experimental and a clinical point of view. Starting from in-vitro studies in animals up to in-vivo studies in humans, we aimed to study and evaluate the weight of this infection as a possible cause of idiopathic infertility in males with any known cause of conception failure.
Significant Correlation between High-Risk HPV DNA in Semen and Impairment of Sperm Quality in Infertile Men
guess the result
go on think
A total of 140 subjects participated in the current study. Among 70 confirmed infertile males, only 8 (11.43%) cases tested positive for high-risk HPV and all fertile men were HPV-negative. This data revealed a significant association between high-risk HPV and male infertility (P=0.03). The percentage of normal sperm morphology and sperm motility rate significantly declined in men infected with HPV (P<0.001).
and all fertile men were HPV-negative
oof and the sluts of both sexes are dying out, I am distraught. The genetics of the future are fairing brighter than you’d think.
Conclusion: There was a significantly higher prevalence of high-risk HPV in infertile men than fertile men. HPV infection seemed to be a risk factor for male infertility. Additional, larger studies should be conducted to confirm the impact of HPV on male infertility.
Player burnout shall henceforth be dubbed HPV-driven infertility?
Association between human papillomavirus infection and sperm quality: A systematic review and a meta-analysis
Human papillomavirus (HPV) has a high incidence rate in both males and females.
-maybe where you live
HPV infection in women has been shown to affect fertility and lead to foetal death and pregnancy loss. However, research on HPV infection in men is limited.
-well the husbands are freshly infecting the wives so–
-Ashley Madison wasn’t full of women stepping out, was it?
The aim of this study was to study the effect of HPV infection in semen on sperm quality and present the findings of previous studies through a meta-analysis. Databases including PubMed, MEDLINE, EMBASE, Web of Science, Cochrane Library, WanFang data and China National Knowledge Infrastructure were searched for relevant studies. A systematic review and meta-analysis were performed, and 17 studies were included for analyses based on a set criterion. Meta-analyses indicated that HPV infection in semen significantly reduced sperm concentration (SMD = -0.12, 95% CI: -0.21 to -0.03, p = .009), sperm motility (SMD = -0.55, 95% CI: -0.780 to -0.33, p = .000), sperm viability (SMD = -0.55, 95% CI: -0.780 to -0.33, p = .000) and sperm morphology (SMD = -0.34, 95% CI: -0.61 to -0.07, p = .015). The high-risk HPV (HrHPV) infection could significantly reduce sperm count (SMD = -0.65, 95% CI: -1.11 to -0.18, p = .007) compared with high-risk HPV (LrHPV) infection.
In conclusion, HPV infection in semen significantly reduced sperm quality, and the HrHPV infection could significantly reduce sperm count compared with LrHPV.
tick tock goes your biological clock, nobody can wait as long as they want NOBODY
Male sperm quality and risk of recurrent spontaneous abortion in Chinese couples: A systematic review and meta-analysis
Conclusions: The results of this analysis support an association of sperm density, sperm viability, sperm progressive motility rate, normal sperm morphology rate, sperm deformity rate, as well as sperm DFI with RSA.
IF you conceived, magically, it would kill your baby. REPEATEDLY.
Semen parameters and sperm morphology in men in unexplained recurrent spontaneous abortion, before and during a 3 year follow-up period
Baby death aborts the defective DNA, HPV fucks with your sperm’s DNA. Water is wet.
HPV makes you biologically unfit. According to the ultimate test, the womb.
To investigate the role of the ‘male factor’ in the pathogenesis of recurrent spontaneous abortion (RSA), especially sperm morphology abnormalities, 120 previously selected couples with unexplained RSA were studied for sperm parameters retrospectively and prospectively. The patients were subdivided into three subgroups, depending on their reproductive outcome during the 3 years of follow-up study: (i) 48 RSA couples who achieved a successful pregnancy; (ii) 39 RSA couples who experienced further abortions, and (iii) 33 RSA couples who experienced infertility during the follow-up period. A semen analysis was performed twice at the time of inclusion in this study, and twice again during the 3 year follow-up period. No significant differences in semen parameters were observed between RSA males and fertile controls. Instead, significant differences were observed between the group of RSA couples who experienced infertility during the follow-up and the other two groups (RSA couples who achieved successful pregnancy and RSA couples who experienced miscarriages and no live birth during the follow-up) for sperm concentration (P < 0.01 and P < 0.01 respectively), sperm motility (P < 0.01 and P < 0.01 respectively) and sperm morphology abnormalities (P < 0.01 and P < 0.01 respectively).
Sperm DNA fragmentation in couples with unexplained recurrent spontaneous abortions
The aim of the present study was to evaluate the degree of sperm DNA fragmentation in couples with idiopathic recurrent spontaneous abortion (RSA) and in those with no history of infertility or abortion. In this cohort study, 30 couples with RSA and 30 fertile couples as control group completed the demographic data questionnaires, and their semen samples were analysed according to World Health Organization (WHO) standards (September 2009-March 2010) for evaluation of sperm DNA fragmentation, using sperm chromatin dispersion (SCD) technique. The percentage of morphologically normal sperm was significantly lower in RSA patients compared with control group (51.50 ± 11.60 versus 58.00 ± 9.05, P = 0.019), but not in other parameters. Additionally, the level of abnormal DNA fragmentation in the RSA group was significantly higher than in the control group (43.3% versus 16.7%, P = 0.024). Our results indicated a negative correlation between the number of sperm with progressive motility and DNA fragmentation (r = -0.613; P < 0.001). The sperm from men with a history of RSA had a higher incidence of DNA fragmentation and poor motility than those of the control group, indicating a possible relationship between idiopathic RSA and DNA fragmentation.
Sperm chromatin integrity may predict future fertility for unexplained recurrent spontaneous abortion patients
“unexplained” – just assume the echo for comedic effect by now
The RSA group was further separated into three subgroups, depending on their reproductive outcome during the 12 months after they were enrolled in the study: the pregnancy subgroup consisted of 43 men whose partners achieved a successful pregnancy up to at least the 24th week of gestation; the abortion subgroup included 31 men whose partners experienced further abortions; and the infertile subgroup had 37 men whose partners did not have any positive pregnancy test after regular, unprotected intercourse. Significantly lower proportion of sperm with normal morphology was found in the abortion subgroup (14.7 ± 4.3%) than in the control group (17.5 ± 5.0%). Sperm concentrations were significantly lower in the infertile subgroup (55.7 ± 24.1%) than in the controls (68.6 ± 27.8%). The rates of abnormal sperm chromatin integrity were significantly higher in the abortion (16.7 ± 7.7%) and infertile (16.3 ± 6.6%) subgroups, compared to the control group (13.0 ± 4.4%). Logistic regression analysis showed that the subsequent reproductive outcome of the 111 RSA patients was negatively correlated to the rates of abnormal sperm chromatin integrity. In conclusion, sperm chromatin integrity, sperm morphology, and sperm concentration were associated with future reproductive outcome of RSA patients. The sperm chromatin integrity was a significant predictor for future abortion and infertility.
But men are never responsible for miscarriage, perish the THOUGHT.
Cytochemical evaluation of sperm chromatin and DNA integrity in couples with unexplained recurrent spontaneous abortions
unexplained….. sigh, ok.
Our study showed that in the cases of RSA, slow motility had a significant reduction in comparison with controls and also spermatozoa of men from RSA group had less chromatin condensation and poorer DNA integrity than spermatozoa that obtained from fertile men with no history of RSA.
Human sperm deoxyribonucleic acid fragmentation by specific types of papillomavirus
Conclusion: Human papillomavirus type 16 and 31 deoxyribonucleic acid caused deoxyribonucleic acid breakages characteristic of apoptotic but not necrotic sperm.
The data suggest that these human papillomavirus types may adversely affect subsequent embryonic development after fertilization. Sperm deoxyribonucleic acid appears to resist human papillomavirus types 18, 33, and 6/11 or repairing mechanisms occurred. Although enhanced motility was found in human papillomavirus–exposed sperm, important velocity parameters were decreased, suggesting impaired sperm function.
Negative Impact of Elevated DNA Fragmentation and Human Papillomavirus (HPV) Presence in Sperm on the Outcome of Intra-Uterine Insemination (IUI)
i.e. no, you won’t just get IVF
We wanted to determine the sperm DNA fragmentation index (DFI) cutoff for clinical pregnancies in women receiving intra-uterine insemination (IUI) with this sperm and to assess the contribution of Human Papillomavirus (HPV) infection on sperm DNA damage and its impact on clinical pregnancies. Prospective non-interventional multi-center study with 161 infertile couples going through 209 cycles of IUI in hospital fertility centers in Flanders, Belgium. Measurement of DFI and HPV DNA with type specific quantitative PCRs (HPV 6, 11, 16, 18, 31, 33, 35, 39, 45, 51, 52, 53, 56, 58, 59, 66 and 68) in sperm before its use in IUI. Clinical pregnancy (CP) rate was used as the outcome to analyze the impact on fertility outcome and to calculated the clinical cutoff value for DFI. A DFI criterion value of 26% was obtained by receiver operating characteristic (ROC) curve analysis. Couples with a male DFI > 26% had significantly less CPs than couples with DFI below 26% (OR 0.0326; 95% CI 0.0019 to 0.5400; p = 0.017). In sperm, HPV prevalence was 14.8%/IUI cycle. Sperm samples containing HPV had a significantly higher DFI compared to HPV negative sperm samples (29.8% vs. 20.9%; p = 0.011). When HPV-virions were present in sperm, no clinical pregnancies were observed. More than 1 in 5 of samples with normal semen parameters (17/78; 21.8%) had an elevated DFI or was HPV positive. Sperm DFI is a robust predictor of clinical pregnancies in women receiving IUI with this sperm. When DFI exceeds 26%, clinical pregnancies are less likely and in vitro fertilization techniques should be considered
When HPV-virions were present in sperm, no clinical pregnancies were observed.
Sperm viral infection and male infertility: focus on HBV, HCV, HIV, HPV, HSV, HCMV, and AAV
Chronic viral infections can infect sperm and are considered a risk factor in male infertility. Recent studies have shown that the presence of HIV, HBV or HCV in semen impairs sperm parameters, DNA integrity, and in particular reduces forward motility. In contrast, very little is known about semen infection with human papillomaviruses (HPV), herpesviruses (HSV), cytomegalovirus (HCMV), and adeno-associated virus (AAV). At present, EU directives for the viral screening of couples undergoing assisted reproduction techniques require only the evaluation of HIV, HBV, and HCV.
-all trust the EU guys
However, growing evidence suggests that HPV, HSV, and HCMV might play a major role in male infertility and it has been demonstrated that HPV semen infection has a negative influence on sperm parameters, fertilization, and the abortion rate.
-somebody else look up herpes, I’m lazy
Besides the risk of horizontal or vertical transmission, the negative impact of any viral sperm infection on male reproductive function seems to be dramatic.
-Really, f-ing fascinating!
In addition, treatment with antiviral and antiretroviral therapies may further affect sperm parameters. In this review we attempted to focus on the interactions between defined sperm viral infections and their association with male fertility disorders. All viruses considered in this article have a potentially negative effect on male reproductive function and dangerous infections can be transmitted to partners and newborns. In light of this evidence, we suggest performing targeted sperm washing procedures for each sperm infection and to strongly consider screening male patients seeking fertility for HPV, HSV, and HCMV, both to avoid viral transmission and to improve assisted or even spontaneous fertility outcome
HPV infection in semen: results from a new molecular approach
Let’s get molecular.
Human papillomavirus (HPV) is the agent of the most common sexually transmitted diseases causing a variety of clinical manifestations ranging from warts to cancer. Oncogenic HPV infection is the major cause of cervical cancer and less frequently of penile cancers. Its presence in semen is widely known, but the effects on fertility are still controversial. – how? allergic to facts?
We developed a new approach to evaluate virus localisation in the different semen components. We analysed also the specific genotype localisation and viral DNA quantity by qPCR. Results show that HPV DNA can be identified in every fraction of semen: spermatozoa, somatic cells and seminal plasma. Different samples can contain the HPV DNA in different fractions and several HPV genotypes can be found in the same fraction. Additionally, different fractions may contain multiple HPV genotypes in different relative quantity. We analysed the wholeness of HPV DNA in sperm cells by qPCR. In one sample more than half of viral genomes were defective, suggesting a possible recombination event. The new method allows to easily distinguish different sperm infections and to observe the possible effects on semen. The data support the proposed role of HPV in decreased fertility and prompt new possible consequences of the infection in semen.
>HPV DNA can be identified in every fraction of semen: spermatozoa, somatic cells and seminal plasma
If you’re wondering why your nation is infertile, look in the mirror. Mutant sperm.
Women’s Preferences for Penis Size: A New Research Method Using Selection among 3D Models
Women’s preferences for penis size may affect men’s comfort with their own bodies and may have implications for sexual health. Studies of women’s penis size preferences typically have relied on their abstract ratings or selecting amongst 2D, flaccid images. This study used haptic stimuli to allow assessment of women’s size recall accuracy for the first time, as well as examine their preferences for erect penis sizes in different relationship contexts. Women (N = 75) selected amongst 33, 3D models. Women recalled model size accurately using this method, although they made more errors with respect to penis length than circumference. Women preferred a penis of slightly larger circumference and length for one-time (length = 6.4 inches/16.3 cm, circumference = 5.0 inches/12.7 cm) versus long-term (length = 6.3 inches/16.0 cm, circumference = 4.8 inches/12.2 cm) sexual partners. These first estimates of erect penis size preferences using 3D models suggest women accurately recall size and prefer penises only slightly larger than average.
but different races have sexually selected different averages, even by nation? So the mean IS the ideal.
The association between penis size and sexual health among men who have sex with men
Larger penis size has been equated with a symbol of power, stamina, masculinity, and social status. Yet, there has been little research among men who have sex with men assessing the association between penis size and social-sexual health. Survey data from a diverse sample of 1,065 men who have sex with men were used to explore the association between perceived penis size and a variety of psychosocial outcomes. Seven percent of men felt their penis was “below average,” 53.9% “average,” and 35.5% “above average.” Penis size was positively related to satisfaction with size and inversely related to lying about penis size (all ps < .01). Size was unrelated to condom use, frequency of sex partners, HIV status, or recent diagnoses of HBV, HCV, gonorrhea/Chlamydia/urinary tract infections, and syphilis. Men with above average penises were more likely to report HPV and HSV-2 (Fisher’s exact p <or= .05). Men with below average penises were significantly more likely to identify as “bottoms” (anal receptive) and men with above average penises were significantly more likely to identify as “tops” (anal insertive). Finally, men with below average penises fared significantly worse than other men on three measures of psychosocial adjustment. Though most men felt their penis size was average, many fell outside this “norm.” The disproportionate number of viral skin-to-skin STIs (HSV-2 and HPV) suggest size may play a role in condom slippage/breakage. Further, size played a significant role in sexual positioning and psychosocial adjustment. These data highlight the need to better understand the real individual-level consequences of living in a penis-centered society.
so men with big wangs, at least the ‘gay’ ones, are rendering themselves infertile or birth defect risks by sharing it around. Y NO str8 studies?
Female Orgasm and Overall Sexual Function and Habits: A Descriptive Study of a Cohort of U.S. Women
Results: FSFI scores, which were calculated for the 230 women who reported having had a steady male sex partner in the preceding 6 months, showed that 41% of the 230 women were at risk for female sexual dysfunction (a cutoff less than 26.55) and 21% were dissatisfied with their overall sexual life. Almost 90% of the overall cohort reported good emotional contact with their partner, that their partner was willing to have sex, satisfaction with the partner’s penis size (wherever applicable), and good erectile function and ejaculatory control of their partner (wherever applicable). 81% of the overall cohort claimed to be sexually active. Around 70% (70-72) did reach orgasm frequently, but around 10% never did so. Vaginal intercourse was reported by 62% of the overall cohort as the best trigger of orgasm, followed by external stimulation from the partner (48%) or themselves (37%). External stimulation was reported to be the fastest trigger to orgasm.
Clinical implications: The knowledge on how women reach orgasm and how it is related to the partners’ willingness to have sex and other factors can be incorporated in the clinical work.
Strengths & limitations: The use of a validated questionnaire and the relative large number of participants are strengths of the study. Limitations are the cross-sectional design, the lack of a sexual distress measure, and a possible selection bias.
Conclusion: Most women in the overall cohort were satisfied overall with their sexual life and partner-related factors, even though 41% (of those who cited a steady sex male partner) were at risk for female sexual dysfunction. Most women did reach orgasm through different kinds of stimulation. Correlation was good between preferred and performed sexual activities and positions. Shaeer O, Skakke D, Giraldi A, et al. Female Orgasm and Overall Sexual Function and Habits: A Descriptive Study of a Cohort of U.S. Women. J Sex Med 2020;17:1133-1143.
I bet the inorgasmic had ‘cut’ men, who produce fewer orgasms in all women in studies.
Impact of penile size on male sexual function and role of penile augmentation surgery
Penile augmentation is an ongoing debate. Emerging evidence proves a relationship between dissatisfaction with penile size and sexual dysfunction. Despite a widespread belief of the value of penile size, and the prevalent complaint of a “small penis,” penile augmentation still stands short of addressing the demand. This report highlights the studies added to medical literature on penile augmentation in 2011-2012, including data on normal penile dimensions and the expected dimensions for a normal person, determination of the prevalence of the dissatisfaction with penile size, its effect on erectile function, female partner’s opinion of the value of penile size, as well as further experience with augmentation techniques.
It’s mental, they need therapy. Also, I see the joke.
Why Are Men Satisfied or Dissatisfied with Penile Implants? A Mixed Method Study on Satisfaction with Penile Prosthesis Implantation
The issue is mental?
Content analysis revealed four main themes for men’s satisfaction with the PPI: (i) psychological factors were reported 54 times (n = 54) and included positive emotions, self-esteem, confidence, enhancement of male identity, major live change, and self-image;
(ii) improvement of sexual function was reported 54 times (n = 54) and referred to achievement of vaginal penetration, increase of sexual desire, sexual satisfaction, penis size, and improvement of erectile function;
– satisfaction is a female metric? ED is mental?
(iii) relationship factors were reported 11 times (n = 11) and referred to relationship improvement and the possibility of giving pleasure to the partner; and (iv) improvement in urinary function (n = 3).
– not possible with implants, all surgeries produce numbness and see above study
A preliminary investigation of a novel method to manipulate penis length to measure female sexual satisfaction: a single case experimental design
Results: On average reducing the depth of penetration made a statistically significant 18% reduction of overall sexual pleasure with an average 15% reduction in length of the penis. The longer the erect penis, the less likely the rings had an impact on sexual pleasure. There was however a range of individual responses with a minority of women reporting that reducing the depth of penetration was more pleasurable on some occasions.
– the G spot?
Conclusions: Size may matter in women in a healthy stable relationship when there is penile shortening. Because of the small number of couples and men with an apparently long penises, our results are preliminary, and we welcome replication in a larger sample with a more diverse range of penile lengths. Our results should not be misinterpreted as meaning that increasing penile length will increase sexual pleasure in women.
shortening with age, and no, supplements don’t help
Non-pharmacological and non-surgical strategies to promote sexual recovery for men with erectile dysfunction
-quit the porn? sigh
the tobacco-lung cancer connection of this generation history will know you as a bunch of wankers
Erectile dysfunction (ED), the most commonly reported sexual problem for men,
in dick-mutilated, porn-addled America? also Israel?
reduces the quality of life for both patients and their partners. Even when physiologically effective, long-term adherence to ED treatments is poor.
– it’s behavioral…
We review here the implication of having patients’ partners involved in ED treatment, starting with treatment selection. We suggest that having partners engaged from the outset may promote an erotic association of the treatment with the partner, i.e., conceptually linking the aid to the sexual pleasure that the partner provides.
– quit ‘supernormal stimuli’ aka lechery, your dopamine is fried?
We hypothesize that this erotic association should enhance the sexual aid’s effectiveness and might potentially help improve long-term adherence. The primary focus of this review, though, is non-pharmacological and non-surgical options for maintaining sexual activity for men with ED. Though not ED treatments per se, anecdotal data suggest that these options may be effective for some patients and their partners in regaining a satisfying sex life. The aids discussed include external penile prostheses, penile sleeves, and penile support devices.
-easier just to give up the sexual visual crack, surely?
These devices can allow men to participate in penetrative sexual intercourse despite moderate to severe ED.
-aging happens, it’s natural selection
External penile prostheses can be personalized so they match in size and shape a man’s normal full erection.
– that is sad
Penile sleeves can similarly be customized with a lumen that fits best a patient’s penis for optimal tactile stimulation.
– so they’re fucking a toy? …. not the man…..
We review how multi-sensory integration can enhance sexual arousal for men who use such devices, allowing them to achieve orgasm despite intractable ED.
-it’s a silicon sock
Patients are not always advised within ED clinics about these options nor why and how they can facilitate non-erection dependent sexual recovery. Clinicians need to be aware of these devices and their positive attributes, so they can objectively counsel and encourage couples to explore their use as an alternative to more invasive treatments. The most commonly promoted non-medical ED aid offered to patients is the vacuum erection device. We discuss how erections achieved with the vacuum erection device have a “hinge effect”, that is an underappreciated barrier to the effectiveness of the erection. With a hinged erection, the penis points downward rather than upward. We show how the normal kinematics of the penis during coitus is not strictly linear (i.e., not uniaxial; not just in-and-out), and is impeded by hinging. Positional adjustment, such as the receptive partner being on top, may help overcome this problem for some couples. Lastly, we suggest that, in the case where ED can be anticipated from a pending medical treatment, such as a prostatectomy, pre-habilitative approaches may potentially improve adherence to sexual aid use in the long-term. In conclusion, non-pharmacological and non-surgical options for sexual recovery are available. Scientific studies on the effectiveness of these interventions in restoring satisfying sex are warranted.
Is Internet Pornography Causing Sexual Dysfunctions? A Review with Clinical Reports
Traditional factors that once explained men’s sexual difficulties appear insufficient to account for the sharp rise in erectile dysfunction, delayed ejaculation, decreased sexual satisfaction, and diminished libido during partnered sex in men under 40.
This review (1) considers data from multiple domains, e.g., clinical, biological (addiction/urology), psychological (sexual conditioning), sociological; and (2) presents a series of clinical reports, all with the aim of proposing a possible direction for future research of this phenomenon. Alterations to the brain’s motivational system are explored as a possible etiology underlying pornography-related sexual dysfunctions. This review also considers evidence that Internet pornography’s unique properties (limitless novelty, potential for easy escalation to more extreme material, video format, etc.) may be potent enough to condition sexual arousal to aspects of Internet pornography use that do not readily transition to real-life partners, such that sex with desired partners may not register as meeting expectations and arousal declines. Clinical reports suggest that terminating Internet pornography use is sometimes sufficient to reverse negative effects, underscoring the need for extensive investigation using methodologies that have subjects remove the variable of Internet pornography use. In the interim, a simple diagnostic protocol for assessing patients with porn-induced sexual dysfunction is put forth.
– it’s a supernormal stimulus designed to make you impotent, it’s a delusion that alters the brain
Based on the findings of historical studies cited earlier, older men would be expected to have far higher ED rates than the negligible rates of younger men [2,7]. However, in just a decade, things changed radically. The 2001–2002 rates for older men 40–80 were about 13% in Europe . By 2011, ED rates in young Europeans, 18–40, ranged from 14%–28% .
duh, porn addicts also look older, do that study
Finally, another 2015 study on men (mean age approximately 36), reported that ED accompanied by a low desire for partnered sex is now a common observation in clinical practice among men seeking help for their excessive sexual behavior, who frequently “use pornography and masturbate” .
aimed at Europeans, squarely
make them hypersexual, extreme r, and re-productively useless
The sole qualification for being a masculine man is having a functioning dick but they cuck for the industry ruining their lives.
The Potential Associations of Pornography Use with Sexual Dysfunctions: An Integrative Literature Review of Observational Studies
This paper reviews the associations between pornography use and sexual dysfunction based on evidence from observational studies. The existing data in this regard mostly derive from cross-sectional investigations and case reports. There is little if no evidence that pornography use may induce delayed ejaculation and erectile dysfunction, although longitudinal studies that control for confounding variables are required for a full assessment.
– decades later, we’ll be dead demographically…
The associations between pornography use and sexual desire may differ between women and men although the existing data is contradictory and causal relationships cannot be established. The strongest evidence is available for the relation of pornography use with decreased sexual satisfaction, although the results of prospective studies are inconsistent. The paper outlines future research prospects beneficial in understanding the nature of associations between pornography use and sexual dysfunctions in men and women.
Online Porn Addiction: What We Know and What We Don’t—A Systematic Review
In the last few years, there has been a wave of articles related to behavioral addictions; some of them have a focus on online pornography addiction. However, despite all efforts, we are still unable to profile when engaging in this behavior becomes pathological.
– publishing bias
Common problems include: sample bias, the search for diagnostic instrumentals, opposing approximations to the matter, and the fact that this entity may be encompassed inside a greater pathology (i.e., sex addiction) that may present itself with very diverse symptomatology.
= excuses so you don’t get later sued
Behavioral addictions form a largely unexplored field of study, and usually exhibit a problematic consumption model: loss of control, impairment, and risky use. Hypersexual disorder fits this model and may be composed of several sexual behaviors, like problematic use of online pornography (POPU). Online pornography use is on the rise, with a potential for addiction considering the “triple A” influence (accessibility, affordability, anonymity). This problematic use might have adverse effects in sexual development and sexual functioning, especially among the young population. We aim to gather existing knowledge on problematic online pornography use as a pathological entity. Here we try to summarize what we know about this entity and outline some areas worthy of further research.
Risky sexual behaviors typically occur when a person is sexually motivated by potent, sexual reward cues. Yet, individual differences in sensitivity to sexual cues have not been examined with respect to sexual risk behaviors. A greater responsiveness to sexual cues might provide greater motivation for a person to act sexually; a lower responsiveness to sexual cues might lead a person to seek more intense, novel, possibly risky, sexual acts. In this study, event-related potentials were recorded in 64 men and women while they viewed a series of emotional, including explicit sexual, photographs. The motivational salience of the sexual cues was varied by including more and less explicit sexual images. Indeed, the more explicit sexual stimuli resulted in enhanced late positive potentials (LPP) relative to the less explicit sexual images. Participants with fewer sexual intercourse partners in the last year had reduced LPP amplitude to the less explicit sexual images than the more explicit sexual images, whereas participants with more partners responded similarly to the more and less explicit sexual images. This pattern of results is consistent with a greater responsivity model. Those who engage in more sexual behaviors consistent with risk are also more responsive to less explicit sexual cues.
– it’s r/K
trans. r-types seem to have dulled sexual perception (abnormal) so over-estimate opportunity in compensation
Studies are in their description, duplicated below.
Reminder: ED is the PC term for impotence – and a common side effect of porn addiction. How about studying penis size between promiscuous men and chaste ones? They’d never publish it. I’d read it. Why are we not funding this?
The role of exposure to phthalates in variations of anogenital distance: A systematic review and meta-analysis
Environmental chemicals such as phthalate esters may have adverse effects on anogenital distance (AGD), but the evidence in both genders has not been reviewed systematically. The objective of the present study is to conduct a systematic review and meta-analysis of studies that analyzed the relationship between exposure to phthalates and AGD. English papers published up to March 2018 were searched in PubMed, Scopus, Clarivate-Web of Science, and Google scholar. We applied fixed-effects models to calculate pooled beta coefficient [β]. In the case of heterogeneity, random-effects models were used. Using the comprehensive search strategies, 313 papers were identified and after screening, 10 of them were included in this study. In primary analyses, we found that exposure to phthalates was not associated with short AGD (β = -0.11; 95% CI, -0.27, 0.06; I2 = 0%). However, results of subgroup analyses indicated that in boys, the sum of di-2-ethylhexyl phthalate (∑DEHP) metabolites had significant association with the risk of shortened anopenile distance (AGDAP) (β = -0.915, 95% CI: 1.629, -0.2) and anoscrotal distance (AGDAS) (β = -0.857, 95% CI: 1.455, -0.26). In addition, urinary monobutyl phthalate (MBP), monoethyl phthalate (MEP), and monoisobutyl phthalate (MiBP) were associated with short AGDAP. We also observed significant association between monobenzylphthalate (MBzP) and anofourchette distance (AGDAF) in girls. Our study provided findings on significant association of exposure to ∑DEHP metabolites, MBP, MEP, and MiBP with shortened AGDAP in boys. The mechanisms of phthalates effect on AGD may involve receptors and enzymes involved in steroidgenesis, negative influence on Leydig cells, cell proliferation, gonocyte cell numbers, and testosterone production.
Men have sex for the pleasure, women for the ego trip.
Stay with me, it’s worth it. I’m giving away Woman Inc. trade secrets here. Don’t use them for evil.
It’s erroneously claimed women fantasise about rape. Hell no. No woman has ever done that. It’s a fear worse than death for women, just ask us. Although rape can be a cause of death in women, like the stadium girl during Katrina, who was gang-raped to death (heart attack). It isn’t that… thing, by definition, as one cannot fantasise about a thing we do not want (paradox) and we see the life outcomes on actual rape victims, as bad in adulthood as if it happened as a child. Women do not fantasise about the act or the supposed sex, then. There’s something symbolic in being wanted, taken is a visual proof. We desire to BE irresistible. Look at Aphrodite, goddess of love and pleasure but not her pleasure, that of men. Men gain pleasure (now called male gaze, it’s real) even by looking at her (imagine the howling if bikinis were banned and reconcile the popularity of porn voyeurism versus imagination) and yet the Bible condemns this as the act of lechery, further, as a crime of adultery. …Why? …. Anyone?
Re Venus/Aphrodite and why cults of Satan worship her/Divine Feminine and worship with orgies as their ritual (nb. any fornication counts):
She is not an object, she is a deified subject who draws pleasure to her like a whirlpool of power. She is called vain, for being honest about her allure, she loves to be irresistible. That is what women want, not men. All witches are also enchantresses of men. Men are the source of their power, even Biblical kings. Men are made weak only by desire. the Bible warns men to forswear fornication (to retain spiritual power) and avoid seductive women entirely. Sexual desire flowed to women empowers the earth and its creatures (women) as well as its ruler (Satan). Women are not servants of the devil, though, since it’s men, who are serving their energy up, gladly. They are told they need to, they have to, that it’s good for them physically and socially. Father of Lies is whom? Look at all the lust over Hollywood celebrities and porn, two faces of the same location. Films intended for minors now have sex scenes and it’s considered normal. Men are the weak point in the species because earthliness (worldliness) is not a part of their natural energy, the lure is greater. Women already have a connection to nature via her menstrual cycles. So all major Satanist figures are men. Women have no seed to sacrifice (Onan’s sin) and no energy (active) to give.
Masculine women have sex for the pleasure, effeminate men for the ego trip. This is why slutty men and women have a cultural stigma as inferior quality in personal character, or whorish (or the rake, the cad etc), women as animal (base material nature) and men as trying to prove something about themselves by ‘notch counting’, objectifying the women out of personal insecurity (weakness, from degenerating the originally pure Jesus-like soul of the man, making soul ties that bind and curse the man’s life/witch women ‘cursing’ their prey, the Witch seducing the pure boy in the Wardrobe film, objectifying effete/weak men as animals to serve their pleasure – Circe to Pinocchio). Worldliness destroys men and nothing less. In Pinocchio, they were turned into donkeys/asses, related to horses. What does the Bible, Song of Solomon for example, say about such things? Other parts about anatomy? … I’ll let you draw your own conclusions, how they were using those boys once they became worldly… what were centaurs known for? https://www.boundless.org/relationships/solomons-line-on-premarital-sex/
That is the modern era, where male virginity is shamed. Male purity and power over any women mocked.
Meanwhile, the alpha woman is desired irresistibly (read: LOYALLY) by the alpha male, and the alpha man like a king desires only his alpha woman like a queen. His possession technically (so he has more duties) but both powerful, each body belonging to the other as the Bible decrees. Aragorn and Arwen. K-selection. Prosperous order, God’s path. Two energies made one flesh in union. The two made whole, or HOLY. Good tree producing good fruit. All myths and scriptures describe the same things.
The mistake of degenerate ancient pagans was to make the fertility goddess a man, because they liked men. Man are not fertile. Women are fertile. Mother Earth is fertile. Material things are fertile physically e.g. Lady Luck for cash gambling. The goddess of the mint, where gold coins were made. Men are immaterial, the cerebral sex. A male fertility goddess is not worshipped for his sex (or in Zeus’ case, rape) but his virility. As mentioned before ‘the embrace of a god is always fertile’. But look what happened to the Greeks and Romans, compared to the Empires of Christian societies, where the man is pure (free of sin, free of corruption, clear-THINKING) before marriage like Jesus then devoted to the family by clear cerebral choice and oath. Patriarchies are made by controlled masculine energy (self-control > self-mastery > The Master – of the house/womb) and from this, lineages are produced, great houses and legacies, by working with the woman’s inherent materalism (taming the dragon of Medusa-like rage) or I guess woman’s prima materia, her womb. The womb is an oven in alchemy, called furnace, pregnancy is a ‘bun in the oven’. The woman (womb of man) has generative power as wet (yes – lunar cycles prove it) and earthbound by the male’s seed of heat (primal fire, desire) and air (his cerebral, immaterial nature from God, Lord of the Sky/Heavens). Earth+Water+Fire+Air = Life, through the vesica pisces portal of the vulva. …I guess I also just explained the reality of alchemy. This would be referenced in weddings, right? It’s in the Bible like ‘my cup runneth over’ as material prosperity, blessed by the grace of God, especially when one is kind to women (like Jesus!) as we see in stories* but what about the words spoken? What was chosen? Why?
SO what are the vows? Let’s test this. To husband: honour and OBEY. To wife: love and to CHERISH. If either party neglects these gender roles, the marriage and its union is doomed. The man agrees to desire her and be loyal to her. What is the most common cause of divorce? Adultery. Most common cheat? Husband. Not ‘having and HOLDING’ was he? Opposite of cherishing: rejection and humiliation. Why is Medusa angry? Rejection, broadly. Rage caused by men (here rape) kills men. It all fits. Hera, also angry. Why? Only when he cheats. Again, it all fits. The woman agrees to respect him, honour his place above her (equal yoke of protection) because that in turn protects their fruit (children) in devoted sanctity and to provide this fully, she must obey his wishes because her vantage point is material, lower -but not inferior- than his pure (before marriage, chaste, Christ-like) spiritual origin in energy terms. Woman made of rib (material) and man made of God’s ‘dust’, (air) of pure spiritual will. I AM – is God’s name, God is his Will. His Will be done. Yada yada. Aside: It’s impossible for a woman to be corrupted by the physical nor fully understand the spiritual (no female disciples). Made complementary in energies, as a couple, to produce both sexes of progeny. The success or failure of any marriage thus falls squarely on the husband’s shoulders, as it is his duty and responsibility to lead the union. The vows aren’t just words, but an oath.A commitment of the very soul of man itself. No time for careerism or time with the boys, family comes first. Prov 31, study the husband, his qualities.
And if God had a sex himself, as the creator and birther of the birthers, God would technically be a woman, by his own choice (will) of role allocation. The linguistic has caused confusion since Babel.
God concept = male if in Heavens. Creating souls. God physically = female if on Earth, creating bodies (so sent a son as progeny, a child not conceived by the materialism of sex, spiritually pure masculinity, a lamb). If Christianity’s God has you worship a total virgin, what would Satan? Satanism tries to turn Earth into Revelation’s Whore of Babylon, stealing power from God.
So if Satan had a body, Satan would also be a woman (watch Ninth Gate). Anything materialised is feminine. Naturally, God’s place is in Heaven, ‘He’ would never materialise to become a She, so it’s purely theoretical. But the Anti-Christ would likely claim to be, and hence be female.
Looks like Amber Heard. The sheer irony. Somebody tell Johnny.
Husbands are the maintaining energy between two planes of creation, with religion.
Two planes intersecting, forming a cross….
Religion gives the cross meaning.
Don’t take my word:
“What shall I say, O my son?
What, O son of my womb?“
from Prov 31, included that wording in the Bible, not really a metaphor as often claimed, it’s God speaking to you as the maker of your maker (mother, Holy ‘Father’ to, Lording over-), also Prov 31:
“Do not spend your strength on women
or your vigor on those who ruin kings.”
spend their spiritual energy materially, thereby also old slang for ejaculation much later, we had ‘spend a penny’, to urinate – the cost of public toilets. pre-dec.
“The heart of her husband trusts in her,
and he lacks nothing of value.”
AND he gains AND
A husband’s devotion to his wife is his material expression of his love of God.
It’s all right there. You don’t see epic love stories of a woman’s devotion, do you?
re holy dynamic
“Many daughters have done noble things,
but you surpass them all!”
Charm is deceptive and beauty is fleeting,
but a woman who fears the LORD is to be praised.” God’s creative power being a higher thing.
Main cause of marital arguments? Money. Section of life? Career. Careerism of either party kills marriages and their sanctity but especially male careerism, it’s abandonment of the primary spiritual commitment. Now you know why the 20th century pushed it on men as desirable to abuse one’s wife and children. How so? Why the Mad Men ideal (and why make that show?)… well, neglect is a form of abuse. The children get neglected. No paternal role model, marriage has no head. A headless body flailing about. Mindless, aimless, but bodily – easily tempted and tainted by worldly things. No wits to resist, no guidance or moral authority. Household has no direction, home empties. No active, masculine energy. Employer rapes it, so Fight Club references the distinct impression that modern bosses rape their male employees of their direction, purpose and life’s energy. Wife is abandoned. Feels undesirable. Her righteous anger at this incorrectly makes him think more retreat will fix it. Man is now effete seeming, passive energy. Emasculating himself. Children sense it, act out, no moral authority will punish them effectively. Children go astray morally. Viscious cycle. Marital failure spirals. Man may leave entirely, the deadbeat or remain absent spiritually as guiding marital force. In either case, union dissolves without his steady stream of energy placed on it. Man’s energy must go somewhere, hence secretary cliche. The reason behind argument, a vow made to place energy. Woman placed in home, energy present. Void of husband? Unloved, uncherished. Nothing to give to children without personal loss (as women = passive). Feels helpless without her man, turns to spiritual intoxicants to blot out misery through the physical, her realm (alcohol, food, sex, smoking, sloth). Life outcomes of absentee parent, kids: not good. The spiritual and the material are not separate. You’re just not looking. Husband/father corrupts his wife and their union’s fruit, children, by omission – neglect and spiritual abandonment. His resentment is self-directed but acted-out in a passive way – like a woman, flees. His leading duties are neglected, so the union erodes. Like Medea, women are rageful out of revenge for being wronged. Cannot be active, becomes more passive out of spite. May be parasitic. Crazed/feared woman always made that way by men in every story e.g. bunny boiler, former mistress or wife.
It applies to a great deal of pop culture. Most people don’t see it.
ONE MORE TEST
Example? Name another cliche trope re women. Love triangle. Symbol: irresistible plus masculine, evolved competition. She gets the best mate, the one who desires her most and, here’s the purpose, would sacrifice the most for his family with her (i.e. God’s spiritual purpose of devotion and loyalty in men). So loyalty in men is praised, cowardice disgraced but there’s no female equivalent of cowardice. If you look at Hera’s rage in myth at a body-changer admitting women enjoy sex more, that’s why fertility gods are correctly women – never heard of a man have multiple orgasms. Meanwhile, a man’s pleasure in life and with wife is in the CHILDREN, his lineage and legacy. Male depression and suicide dovetails nicely with the lowest marriage (and reproduction) rates ever. BOOM.
I do wonder how many male suicides have no (surviving) children. Women simply caretake pre-existing family.
*Fact: Men unkind to women suffer materially their whole lives, self-cursed by the material and their rejection of its God-designed representatives. The material is not wrong, sick or evil per se – only perceived so by the weakness of effeminate men who cannot control/influence it and thusly, feel impotent. If you get the dragon, you did something to deserve it. If Lady Luck hates you, start being a gentleman to woo her. Civilized cultures raise men as gents and the ladies arrange themselves, reflecting like the moon to their sun, the quality and calibre of their society’s men. That’s all a society is. The quality of its procreating men, the fathers and husbands. THEREFORE, EVERY SOCIETY IS A PATRIARCHY. A strong (healthy) or weak (dysfunctional) one. Their control of that role. Is it too much, too little? Is there proper energy exchange? Chivalry isn’t historic, it describes a sophisticated power dynamic of holy energies. It was a Christian series of metaphors. Only the West nailed this, for a while. Look what we achieved with it. Then we let it slip, for domineering over the feminine to ‘prove something’ (egocentric stupidity, selfish, too desperate, ultimately impotent) or degeneracy (self-weakening of men via desires rather than morally weakening women, the soft watery mirrors of solar, fiery light). Weakened men (clue: mutilated manhood, less pleasing to women in studies) prefer to push degeneracy to entice their fellow man competition into destroying themselves.