CBD oil push is anti-white, anti-natal, harmful

I have to post this now because the product push is in full swing and it could literally save tiny lives.

I can’t wait until October, all the “influencers” are pushing this online NOW. Ethically, I must put my ego aside.

A lot of Pill failures are actually in the hippy chicks taking plant “extracts”. That’s why doctors now ask young women about supplements and herbal remedies immediately after the contraception question. It wouldn’t be ethical to study pill failures but they know there’s a link.

so in context:
How Smoking Marijuana Damages The Fetal Brain” is horrifying.
“Earlier studies have already found that children of marijuana-smoking mothers more frequently suffer from permanent cognitive deficits, concentration disorders, hyperactivity, and impaired social interactions than non-exposed children of the same age and social background.”

so you cannot blame poverty, Karen.

Remember, they’re now encouraging wine moms to drink while pregnant.
They used to claim nicotine was a “safe” plant extract because the addictive property was removed, supposedly.
“CBD, THC use during early pregnancy can disrupt fetal development”
Nicotine was justified as anti-inflammatory too, and it is:
Still addictive!

Weird how all these CBD products were pushed before safety studies like this, and now we have corona-chan distracting.
A new study published in Scientific Reports, a Nature Research journal, shows how a one-time exposure during early pregnancy to cannabinoids (CBs) – both synthetic and natural—can cause growth issues in a developing embryo. This is the first research to show such a connection in mammals.”
One-time. But not a drug, ya see. That makes sense.

It’s the first research to LOOK. Why was this cleared? And who calls CBD safe? Oh…. the WHO.


Let’s trust those guys!

“muh no public health risks”

Buckle up, I found a LOT.

“In this study, the brain and facial developmental effects caused by one-time exposure to CBs—CBD and THC (the primary ingredients of marijuana)—are very similar to what is seen in fetal alcohol syndrome (FAS). Parnell and colleagues also found that when CBs and alcohol were used together, the likelihood of these birth defects more than doubled. They went on to show that these drugs may be causing defects by interacting on a basic cellular level and disrupting signaling between molecules and cells that control growth and development.”
“The CBD amounts administered were within what is considered a therapeutic range for humans.”

trans. minimal.

“It is concerning how little we know about the use of marijuana, its CBs, and products like CBD oil during pregnancy,” Parnell said. “We know that there is no safe period to drink alcohol during a pregnancy, and I think this research shows the same is likely true of marijuana use.”

Drinking once doesn’t cause FAS, so this is objectively worse. Don’t expect the tradlarpers to talk about anything important re child IQ and birth rates. Nope! Let’s talk about some twit on Twitter again.
I don’t care if you like your CBD rollerball, Karen! You’re better off aborting it now it’s brain damaged!

If you want to de-stress take a poxy bubble bath like a non-druggy.

I expect it causes more miscarriage too, with that effect.

With the results of these one-time exposures, Parnell and Fish are planning to now test smaller, multiple exposures throughout a pregnancy that better mimics real-life usage in human pregnancy.”

That came out after this:

“That’s concerning because CBD may interact with commonly used anesthetics that might be needed during labor and delivery.”

That’s because it’s a drug. It is a drug.



“Many women report that they use CBD oil during pregnancy in order to reduce pregnancy-related nausea.

In general, using CBD while one is pregnant is thought to be safer than smoking cannabis itself or THC-rich products.” [DS: we now know this is wrong, see above]

The hippy Karens must be stopped.

I bet it damages sperm too. That’s all the soy boys need.

There is a lack of conclusive data to determine the effects of CBD hemp oil on a fetus. However, it is known that a growing fetus is equipped with an endocannabinoid system, even when the fetus is only composed of two cells. This system is in all humans and even some animals. The endocannabinoid system is a system composed of endocannabinoids, which are neurotransmitters that bind to cannabinoid receptors.

In a study conducted on mouse embryos, researchers found that the compound THC inhibited the development of the embryos which contained less than eight cells. Another natural cannabinoid found in the human body, anandamide, also stopped the embryos from developing. CBD can increase levels of anandamide, so there may be negative effects associated with CBD use during pregnancy. It is important to note that this was a study conducted on mice and the results may not be transferable to human subjects.

If you’re gonna be a Western baby birth rate cult, talk about things like this.

The advice to women must account for this.

It’s called dope for a reason.

“marijuana causes significant brain changes by slowing activity in the frontal and temporal lobes”

there’s a known connection to schizophrenia in men, especially those who start before 21, earlier in the teens the higher the risk

“Some argue that marijuana is not addictive, but as it demonstrates, it is a drug like any other.”

I knew this so I had to post.

Alcohol is a drug and it is addictive (alcoholism is an addiction to alcohol, not mere consumption, it’s a pattern of chronic consumption with withdrawal during cessation). As soon as they call their poison “not addictive”, you know it is. It’s like hearing “I can quit any time I want”. Cognitive dissonance, a month of cessation would be intolerable for them.


re above study:

after studying imaging of 1,000 cannabis users’ brains, there were signs of noticeable deficiencies of blood flow. The study, which included 25,168 non-cannabis users, and 100 healthy controls, shows a scary and obvious difference in blood flow levels for those that used cannabis. Additionally, those that used marijuana showed a significant lack of blood flow in the right hippocampus, the area of the brain that helps with memory formation. This part of the brain is severely affected with those that suffer from Alzheimer’s disease.”

“Our research has proven that marijuana users have lower cerebral blood flow than non-users.”

You also see sluggish cerebral flow in the low IQ. That’s why they call it dope. It has a dopifying effect.
Stupidity is bliss? It’s diagnostic.

We identified a strong inverse relationship between performance IQ and CBF (-1.5 points per 10 mL/100 g/min increase in CBF, P = .013).”

CBF = cerebral blood flow

I’m not making shit up. If a perma-bachelor over the age of 25 (mature brain) wants to be a druggy, I don’t care. But don’t kill and brain damage the babies!

This has a lot of studies linked: https://www.solcbd.com/blogs/news/cbd-oil-and-fertility-have-facts

In a 2010 Italian paper published online by BioMed Central, the authors argue that disrupting the normal activities of these ECS pathways by adding cannabinoids “can significantly alter many vital in utero processes, including angiogenesis, cellular replication, tissue differentiation, and [fetal] neural cognitive development.” [4] ….

While THC exposure increased sexual receptivity in female hamsters and rats, it also inhibited their release of luteinizing hormones. These hormones are responsible for ovulation and the development of the corpus luteum. The latter is a hormone-secreting structure in the ovary that plays a role during very early pregnancy. [3]

3= https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4034092/

They’re pushing CBD products as harmless and trendy onto MINORS.







Aimed at minors.

It inhibits development. Do the math. I couldn’t wait on this. I was going to post this last year but forgot.

In a 2016 paper published by BioMed Central, it is mentioned that in utero exposure to cannabis has been associated with early pregnancy failure, birth defects, and developmental delays in the fetus.




“The results of this study show that the use of cannabis in pregnancy is associated with increased risk of adverse birth outcomes. Prevention programs that address cannabis use during pregnancy are needed.”

I think this is the one: https://bmcpharmacoltoxicol.biomedcentral.com/track/pdf/10.1186/s40360-016-0085-6?site=bmcpharmacoltoxicol.biomedcentral.com

“With the increasing publicity of marijuana due to recent legislation, it is pertinent that the effects of fetal exposure
to the drug are assessed. While in utero cannabis exposure has been associated with early pregnancy failure, birth defects and developmental delay, the mechanisms of such outcomes are largely unexplained. Furthermore, the use of cannabinoids in cancer treatment via growth inhibition and apoptosis may indicate how cannabis exposure likely harms a growing fetus. Cannabinoid signaling is required for proper pre-implantation development, embryo transport to the uterus, and uterine receptivity during implantation. In post-implantation development, cannabinoid signaling functions in a multitude of pathways, including, but not limited to, folic acid, VEGF, PCNA, MAPK/ERK, and BDNF. Disrupting the normal activity of these pathways can significantly alter many vital in utero processes, including angiogenesis, cellular replication, tissue differentiation, and neural cognitive development. This paper aims to demonstrate the effects of cannabis exposure on a developing embryo in order to provide a molecular explanation for the adverse outcomes associated with cannabis use during pregnancy.”

The authors also argue that cannabis exposure could very likely harm a fetus due to phytocannabinoids’ effect on cells. This is because CBD and other cannabinoids are associated with cell growth inhibition and cell death, which may be good news for the treatment of cancer, but not for a growing fetus. [4]

4 =


These findings were based on petri dish research, but the same trend was observed in most cell cultures, not just in cancer cells. Obviously, this could mean that healthy cell growth can also be affected. [5]

5 =


This is speculative, however, and not an easy subject to study clinically, but it is clear that much more research is needed before any phytocannabinoid can be declared safe for use by pregnant women.


There appears to be no traceable data regarding CBD and male fertility. The available research centers mostly on marijuana and fertility in males, and it is mostly negative, except for one finding.

How are these products on the market? HOW?

It feels like a DDT style case study waiting to happen.

This research, conducted by Dr. Hans Hatt of Ruhr University in Bochum, Germany, has discovered a receptor in sperm cells that is part of the ECS.

Called the GPR18 receptor, it plays a big role in conception. Dr. Hatt explains its function very simply: “The endocannabinoid activates the spermatozoa for fertilization.” This means that it helps the sperm to fertilize the egg or the ova.

The doctor also pointed out that this receptor responds to THC and another cannabinoid, anandamide, with involvement in the sperm’s ability to penetrate the ova. This is encouraging news, but much more study is needed before phytocannabinoids can be considered a possible therapy for male infertility. [6]

This is also because other research points in the opposite direction for men.

Birth defects suggest it messes with the sperm’s genetics.

And it does.


A total of 1,215 young Danish men aged 18-28 years were recruited between 2008 and 2012 when they attended a compulsory medical examination to determine their fitness for military service. The participants delivered a semen sample, had a blood sample drawn, and underwent a physical examination. They responded to questionnaires including information on marijuana and recreational drug use during the past 3 months (no use, use once per week or less, or use more than once per week). A total of 45% had smoked marijuana within the last 3 months. Regular marijuana smoking more than once per week was associated with a 28% (95% confidence interval (CI): -48, -1) lower sperm concentration and a 29% (95% CI: -46, -1) lower total sperm count after adjustment for confounders. The combined use of marijuana more than once per week and other recreational drugs reduced the sperm concentration by 52% (95% CI: -68, -27) and total sperm count by 55% (95% CI: -71, -31). Marijuana smokers had higher levels of testosterone within the same range as cigarette smokers. Our findings are of public interest as marijuana use is common and may be contributing to recent reports of poor semen quality.

I think we found a cause of generational infertility in men, suck up that red pill.


IVF high in Denmark, wonder why?

Peak health by age and it still impairs their sperm….


The cause is STDs and drugs, obviously. Always has been.

Men are lied to and the SJWs love it.


From the sol link:

The inability to conceive affects both men and women equally. Among couples experiencing infertility, roughly 35 percent is due to problems in the male, while another 35 percent is due to issues in the female. Another 20 percent is a combination of issues in both the male and the female, and the remaining 10 percent is unknown.

Apart from CBD oil for fertility, a number of alternative or complementary therapies are on the rise, all with varying degrees of success.

They claim CBD is antidepressant despite cerebral flow connection to causing it. Gotta keep selling those SSRIs.

New Research Shows How Marijuana Drops Blood Flow to the Brain. Should You Be Concerned?

“demonstrates that marijuana can have significant negative effects on brain function. The media has given a general impression that marijuana is a safe recreational drug, this research directly challenges that notion.”

Beware anything the MSM pushes. What do druggies do? Sit around watching TV and consuming other ‘entertainment’ like Netflix.
It’s a known scandal that American farms of it are basically a monopoly.
If the effect is anti-stress, it’s a drug. If that’s the only purpose, drink tea. Put on a nicotine patch.
They never explain fully the biological mechanism that makes it work like a drug, without literally being a drug.
The dropper form of CBD is literally a drug, drops are used in pediatrics on babies. It’s sublingual absorption. If it’s ‘extracted’ from a drug, works like a drug and is delivered like a drug… it’s a drug.
Should adults be allowed to be druggies? Over 25, maybe. But why must I pay for stressed-out Karen’s “treatments” on the NHS? Must I pay for her alcohol too? Can she pay for my naps and chocolate?
Why not legalise morphine and laudanum and all the other naughties you used to buy over the counter before the nanny state?
Because people would grow their own poppies. That’s why. It’s too easy to extract.

The method was flawless, before AND after.

“Several studies of perfusion imaging in marijuana users have shown similar results compared to ours. A small O15 PET study in a sample of 12 marijuana users used a randomized clinical trial design to examine brain perfusion before and after marijuana use. The study results found frontal, temporal and occipital lobe hypo-perfusion – all findings concordant with our study.

Zoomers aren’t stupid, they’re polluted.

Although it is often portrayed as harmless, and sometimes even therapeutic, there has not been nearly enough studies done to prove this. In fact, marijuana is often prescribed for issues like anxiety, though studies cannot comprehensively show this to be true. Currently, the available information on the impact marijuana has on the neurophysiology of the brain show, predominantly, depressive effects.”

It’s pushed on women to harm their fetus. Skin cream lingers for hours and ends up in the blood.

Eat your aluminium, thots

That’s right, smear on a lipstick full of shimmery bullshit. Including lead (and wonder why girls are depressed). [Literally eating lead.]

The Victorians ate arsenic but this is worse. Arsenic temporarily improved the complexion.

Using the term ‘mica’ for what’s essentially powdered foil suggests women wouldn’t want it if they knew what it was.

It isn’t like there’s a connection between dissected and tested Alzheimer’s plaques and aluminium deposits, is there?

Why are the Boomers raised on cans of this stuff seeing an explosion in Alzheimer’s rates?

As always, at first, go to the ‘officials’. Find out the Party Line.

Although aluminium has been seen in amyloid plaques”


“there is no solid evidence that aluminium is increased in the brains of people with Alzheimer’s disease.”
Whew, what cognitive dissonance.
It’s literally forming those plaques that ARE the disease but nbd?

Solid evidence – like a solid chemical analysis of solid plaques in the solid brains of solid dead people with Alzheimer’s?

Look at the hustle to move those goalposts.

“Because some people with the disease had aluminum deposits in their brains, it was thought that there was a direct connection…”
Well… yeah.
That’s proof.
There’s no negative evidence possible for that to stop being proof.

It’s literally in their brains.

“However, after many years of study, no conclusive evidence links aluminum to neurodegenerative disease”
Bullshit, 2007.

I detest scientism. Look at these papers and tell me what’s ‘conclusive’.

(Apart from donations of mega-corps to Super PACs to prevent this mega-lawsuit).

“A multi-institutional team of researchers has defined for the first time how metal ions bind to amyloid fibrils in the brain in a way that appears toxic to neurons. Amyloid fibrils are linked to the development of neurodegenerative diseases such as Alzheimer’s, Parkinson’s and Creutzfeldt-Jakob. Although metal ions, most notably copper, can bind to amyloid in several specific ways, the researchers found that only one way appears toxic.”

But how, ‘critics’ argue, could this possibly occur? What’s the mechanism? Nobody has proven a mechanism….?


“Link between Aluminum and the Pathogenesis of Alzheimer’s Disease: The Integration of the Aluminum and Amyloid Cascade Hypotheses”


“In particular, the link between aluminum and Alzheimer’s disease has been the subject of scientific debate for several decades. However, the complex characteristics of aluminum bioavailability make it difficult to evaluate its toxicity and therefore, the relationship remains to be established.”

Uhuh. Isn’t that your job?

If it’s impossible, why are you getting paid?

“On the contrary, aluminum is a widely recognized neurotoxin that inhibits more than 200 biologically important functions and causes various adverse effects in plants, animals, and humans.”


Mounting evidence has suggested that significance of oligomerization of β-amyloid protein and neurotoxicity in the molecular mechanism of AD pathogenesis. Aluminum may play crucial roles as a cross-linker in β-amyloid oligomerization.

Here, we review the detailed characteristics of aluminum neurotoxicity based on our own studies and the recent literatures. Our aim is to revisit the link between aluminum and AD and to integrate aluminum and amyloid cascade hypotheses in the context of β-amyloid oligomerization and the interactions with other metals.”

But HOW could this POSSIBLY operate? – intellectually dishonest douches.

Note the prestige of journal. International Journal of Alzheimer’s Disease, those hacks.

But on a MOLECULAR level…. – douchecanoes.

“Quantification of the Aβ peptide in Alzheimer’s plaques by laser dissection microscopy combined with mass spectrometry”

It’s behind a paywall but it’s there. Check the academic pirate bay.

It’s been known for a while.
2000: “Aluminum has been identified as a neurotoxin for over 100 years.”

So stop using it in food, cosmetics, deodorant, cookware and cans?

Their use actually came in well after it was a known neurotoxin. Because that isn’t dodgy.

Reference for that sentence:

Doelken P:Naunynschmiedeberger. Arch Exp Pathol Pharmakol 40: 58-120 cited by Crapper McLachlan DR., Lukiw WJ, Kruck TPA, Aluminum, altered transcription, and the pathogenesis of Alzheimer’s disease. Environ Geochem Health, 1990; 12(1-2): 103-114.

Link for that: https://link.springer.com/article/10.1007/BF01734059

Opening paragraph.

“The etiology of some, if not all, cases of Alzheimer’s disease is linked to a mutation in the proximal portion of the long arm of chromosome 21∶21q11.2 → 21q22.2. While the functional consequences of the mutation are unknown, we speculate that one consequence of the mutation is loss of the natural barriers and intracellular ligands for aluminum. As a result, aluminum gains access to several brain sites including the nuclear compartment in certain neurons of the central nervous system.”

I know both my shit and my bullshit.

1990. Who owns the aluminium? That’d be a fun tour of genocide.

1992 laser study, note prestige of journal.

What the fuck do they know, right, reddit?

“Selective accumulation of aluminum and iron in the neurofibrillary tangles of Alzheimer’s disease: A laser microprobe (LAMMA) study”

“In addition, probe sites directed to neurons identified in snapfrozen cryostat sections from 2 subjects with Alzheimer’s disease revealed similar spectra with prominent aluminum‐related peaks, confirming that our findings are not related to exogenous contamination through fixation, embedding, or other procedures prior to analysis. This study further confirms the association of aluminum and neurofibrillary tangle formation in Alzheimer’s disease.”

But I guess I’m just scared of non-stick pans, where’s the evidence?

Paper: Aluminium and Alzheimer’s


You could probably predict this one based on my last post.

Companies use plenty (metric fucktons) of aluminium machinery (watch any episode of How it’s Made online) because the odds of you proving the connection and the odds of tracking it back to their production and the odds of you suing is practically nil and they keep profit margins fat. Abstaining from “junk food” might make you feel better because you cut down on your foods processed using aluminium as a powder (e.g. for baking) or aluminium machines (most dangerous/leeching when handling acidic contents AKA most “junk” plus random popular foods like tomato sauce).

It would be so easy to force them to switch to (clean, no other pollutants) stainless steel but that’s more expensive and there’s no public pressure. I link to these to help that end.

Alzheimer’s disease (AD) is perhaps the principal example of cognitive failure in humans,

brain damage

and currently over 5.5 million Americans suffer from this incapacitating and progressive disorder of thought, reasoning and memory. Our laboratory has been evaluating the potential contribution of environmentally bioavailable neurotoxic metals to the onset, development and progression of AD for about 30 years

Filthy casuals.

(Lukiw et al., 1987). Largely because of its known multiple and potent neurotoxic effects, much of our research has focused on the potential contribution of aluminum to the AD process

Yeah, why do Americans get it so much, Aluminium Machine Manufacturing America????

And the generation who grew up with “safer” aluminium “tins”…

While I’m activating almonds.

Other elements might be iffy too, if you’re a man with titanium jewelry (skin contact), consider replacing it.


If it seems cheaper and better than (good thing), it’s probably a slow acting toxin.

Why don’t you get Trump to announce companies have to label which metals (elements) are in their products, especially important for say, kitchenware compounds (e.g. stainless steel)? You already have the lead law although that’s in dire need of a re-write because they now label everything as a lawsuit proofing measure.

You need to know what you’re buying, literally.

Simple consumer liberty. Why aren’t we funding this?

I couldn’t make it up

The wages of sin are death.


“Free love”

How’s that sexual revolution?

Still doubling down huh?

Yeah, your parents couldn’t have a point, they were just squares.

I don’t think it is the herpes, although that damage is real.

I think it’s the aluminium still.

The Aluminium Age and Alzheimer’s

Where’s the science?


“The results demonstrated that Al(III) induced the transformation of the initial random coil structure to the β-sheet configuration in the Aβ40 peptides. These structural changes facilitated the aggregation of Aβ40.”

Meanwhile, denial train. Choo choo!

“Here’s the evidence behind the presence of metals such as copper, zinc, iron and aluminium.”
The first three are needed by the body and not neurotoxins.
“But the evidence doesn’t yet show whether this relationship actually causes Alzheimer’s disease.”
Have you tried checking?
“It is also unclear whether reducing metals in the brain via drugs or reducing our exposure would have any effect.”
It is unclear whether Caesar was stabbed but there is a lot of blood and all these knives might’ve had some effect.
“These metals are essential to the healthy function of our brain,”
100% LIE.
Aluminium is not necessary for any bodily function.
But it makes food manufacturing dirt cheap!

“The body is able to tolerate these metals in small amounts by clearing through the kidneys. ”

Well, clearly not.

These include aluminium and lead, for example it has been shown that if they are not taken out by the kidneys through organ failure or by exposure to extremely high doses these metals are able to deposit in the brain.
These metals are known to cause negative effects in the brain and have been implicated in several neurological conditions.”
Safe as lead, guys!
And no that’s outdated, the body doesn’t really clear it. See end.
“In 1965, researchers found that rabbits injected with an extremely high dose of aluminium developed toxic tau tangles in their brains. This led to speculation that aluminium from cans, cookware, processed foods and even the water supply could be causing dementia.”
But the can manufacturers were slipped a bribe because they recently had to switch from toxic tin.
That generation’s children now seem to be presenting with unprecedented Alzheimer’s…. coincidence?
“Importantly, these results were only seen with extremely high exposures that far exceed the levels that can enter the body through food or potentially through contact with aluminium cookware.”
That limit does not exist and assumes perfect health – no alcoholism, polluted air, toxic water, stress, chronic diseases.
“As yet no study or group of studies has been able to confirm that aluminium is involved in the development of Alzheimer’s disease.”
Ethics and finding the funding.
Also a half-lie, there are many studies. As you’ll see.
“Aluminium is seen in the normal, healthy brain.”


No, it isn’t.
Not ever. Not at all.
It shouldn’t get past the barrier.
Normal for the modern world is not healthy.

Opening line here: “Aluminium is neurotoxic.”

Back to the liars:

“Although aluminium has been seen in amyloid plaques there is no solid evidence that aluminium is increased in the brains of people with Alzheimer’s disease.”
Yes there was, you’re ignoring it.

They dissected Alzheimer’s patients and found it in there.
Blatant lie.

No convincing relationship between amount of exposure or aluminium in the body and the development of Alzheimer’s disease has been established.”
Wait before it didn’t exist and in the very next sentence, it isn’t convincing?
“Aluminium in food and drink is in a form that is not easily absorbed in to the body. Hence the amount taken up is less than 1% of the amount present in food and drink.”
Bullshit. It’s the most processed form it goes through the whole digestive tract and then into the bloodstream like food nutrients.
Less than 1% – per meal or drink. PER meal or drink.
“Most of the aluminium taken into the body is cleaned out by the kidneys.”
And wouldn’t it be the liver?
“failed to find a convincing causal association between aluminium exposure in humans and Alzheimer’s disease.”
Give us the data instead of finding excuses to hide it.
1991, before the cash cow was in full milking rotation
“exposure to aluminium has been implicated by epidemiological studies and the finding of aluminium in the cerebral plaques and tangles.”
3 decades later? Still covering it up?
While they blame copper:
It’s neuroprotective.
“a high brain tissue ratio of copper to aluminium protects against neurotoxicity associated with the deposition of amyloid-b and the amyloid cascade hypothesis.”
“The study on 60 aged human brains identified a number of relationships between the degree of severity of amyloid-b neuropathology and the metal content of tissue from the donor brains. The latter was recently published for aluminium, copper and iron (House et al. (2012) Metallomics 4, 56-65).

Relationships, PLURAL and severity.

“Specifically, the extent and severity of amyloid-b deposition was inversely related to the copper content of brain tissue. Lower copper resulted in more severe and more extensive deposition of amyloid-b in the donor brain.”
But obviously you’re crazy to read about it.
“The research suggested that for those individuals with moderate to severe amyloid pathology, a copper to aluminium ratio of less than 20 predicted dementia.”
“Professor Exley said: “The hypothesis requires further testing but if proven correct it could explain why some individuals with senile plaques do not suffer from dementia. The implication being that a high brain tissue ratio of copper to aluminium protects against neurotoxicity associated with the deposition of amyloid-b and the amyloid cascade hypothesis.””

Aluminium-specific chelators reduce symptoms. Coincidence?
Effect is seen with low levels too:
By this means, the body burden of aluminum in humans has increased. Epidemiological and experimental findings indicate that aluminum is not as harmless as was previously thought, and that aluminum may contribute to the inception and advancement of Alzheimer’s disease. Epidemiological data is reinforced by indications that aluminum exposure can result in excess inflammatory activity within the brain.

“Evidence is presented that reinforces the likelihood that aluminum is a factor speeding the rate of brain aging. Such acceleration would inevitably enlarge the incidence of age-related neurological diseases.”

Maybe most Baby Boomers would be okay if their brains weren’t poisoned?

“However, disruption of these mechanisms, or absorption of detrimental metals with no known biological function, alter the ionic balance and can result in a disease state, including several neurodegenerative disorders such as Alzheimer’s disease. Understanding the complex structural and functional interactions of metal ions with the various intracellular and extracellular components of the central nervous system, under normal conditions and during neurodegeneration, is essential for the development of effective therapies.”

I hope you plan on someone in Big Pharma coming to kill you for developing therapies.

Opening line: “Aluminum has been identified as a neurotoxin for over 100 years.”
As safe as lead or mercury.

Click to access Aluminum2.pdf

You know where the society said no studies?
“A 2008 systematic review of studies that included aluminum exposure through drinking water, diet, and occupation found 23 studies demonstrated an increased risk for Alzheimer’s disease with elevated aluminum exposure, 3 found that there was no connection…”
Cover-up? Where’s the science?

I can’t seem to find it, guys!

We screened 4784 studies and included 60 in the review. Risk factors were considered in six categories: air quality, toxic heavy metals, other metals, other trace elements, occupational-related exposures, and miscellaneous environmental factors. Few studies took a life course approach. There is at least moderate evidence implicating the following risk factors: air pollution; aluminium; silicon; selenium; pesticides; vitamin D deficiency; and electric and magnetic fields.”
Life course is ideal.

There is a threshold.
“Our article examines the question of whether environmental and therapeutic aluminum exposure increases the risk of disease. To this end, Alzheimer’s disease and breast cancer are taken as critical endpoints. Aluminum’s neurotoxic effects in humans and its embryotoxic effects in animal models have been proven (4).”

Click to access f004582d987d2142ba418d26149d258d64e024e9.pdf

“An inevitable consequence of humans living in
the Aluminium Age is the presence of aluminium in the
brain. This non-essential, neurotoxic metal gains entry to
the brain throughout all stages of human development,
from the foetus through to old age. Human exposure to
myriad forms of this ubiquitous and omnipresent metal
makes its presence in the brain inevitable, while the
structure and physiology of the brain makes it particularly
susceptible to the accumulation of aluminium with age. In
spite of aluminium’s complete lack of biological essentiality,
it actually participates avidly in brain biochemistry
and substitutes for essential metals in critical biochemical
processes. The degree to which such substitutions are disruptive
and are manifested as biological effects will depend
upon the biological availability of aluminium in any particular
physical or chemical compartment, and will under
all circumstances be exerting an energy load on the brain.
In short, the brain must expend energy in its ‘unconscious’
response to an exposure to biologically available aluminium.
There are many examples where ‘biological effect’
has resulted in aluminium-induced neurotoxicity and most
potently in conditions that have resulted in an aluminiumassociated
encephalopathy. However, since aluminium is
non-essential and not required by the brain, its biological
availability will only rarely achieve such levels of acuity,
and it is more pertinent to consider and investigate the
brain’s response to much lower though sustained levels of
biologically reactive aluminium. This is the level of
exposure that defines the putative role of aluminium in
chronic neurodegenerative disease and, though thoroughly
investigated in numerous animal models, the chronic toxicity
of aluminium has yet to be addressed experimentally
in humans. A feasible test of the ‘aluminium hypothesis’,
whereby aluminium in the human brain is implicated in
chronic neurodegenerative disease, would be to reduce the
brain’s aluminium load to the lowest possible level by noninvasive
means. The simplest way that this aim can be
fulfilled in a significant and relevant population is by
facilitating the urinary excretion of aluminium through the
regular drinking of a silicic acid-rich mineral water over an
extended time period. This will lower the body and
brain burden of aluminium, and by doing so will test
whether brain aluminium contributes significantly to
chronic neurodegenerative diseases such as Alzheimer’s
and Parkinson’s.”

Of course, A Drink is cheaper than all the care of the sick patients and you can’t steal their house legally by telling the council they lack competence.

“Certainly aluminium either directly as a
particulate or indirectly following the dissolution of
nanoparticulates could induce an inflammatory action in
the human brain, and this has been demonstrated in animal
models [64]. The immunopotency of aluminiumbased
adjuvants outside their role as adjuvants in vaccine
and allergy therapies seems to have been largely ignored
as a potential mechanism of aluminium toxicity
throughout the body [65] and especially in the nervous
system [66]. The consistent observation of significant
accumulations of aluminium in the brain should at least
be a warning of the potential for such to participate in
neuroinflammatory toxicity.”

You’d think.

The brain is an obvious target for aluminium toxicity.
Neurotoxicity is evident under acute conditions such as
encephalopathies, and it is predicted but not necessarily
recognised as such under chronic or everyday exposures to
environmental aluminium. The mechanisms of neurotoxicity
are potentially myriad, while their manifestations as
biochemical changes are probably quite subtle for all but
the most vulnerable groups.”

Headaches, stomach aches, etc. “Brain fog” might be entirely toxin based.

“While the latter must include
the foetus and neonate, there are few indications as to the
identities of others who are susceptible to the neurotoxicity
of aluminium. Since the advent of the Hall-He´roult process
(and thereafter Bayer process) towards the end of the
nineteenth century and our ability to extract aluminium
from its inert ores on an industrial scale, we have all been
living in the Aluminium Age [67]. Now, in the twenty-first
century, we can no longer completely avoid environmental
exposure to aluminium. Since there is as yet no proven
requirement for aluminium in any living organism, never
mind in humans, it would be prudent to reduce our
everyday exposure to avoid aluminium entering the body
and persisting in the human brain [68].”

It’s used to process junk food, watch How It’s Made.
When people feel better after giving up junk, it might be aluminium exposure reduction.

It would be easy to study: people who eat a lot of junk and people who don’t, Al urine amounts.

“We have begun to
show that this can be achieved by using nature’s own way
of avoiding biologically available aluminium. We have
shown that regular consumption of silicon-rich mineral
waters both reduce our gastrointestinal uptake of aluminium
and, importantly, facilitate our urinary excretion of
systemic aluminium [48]. Life on Earth evolved in spite of
a crust of aluminosilicate [1]. However, the Hall-He´roult
process and the subsequent arrival of an Aluminium Age
have let the aluminium genie out of the bottle. Our final
wish should be that the unique inorganic chemistry of
aluminium and silicic acid will help to put the genie back
where it can be used effectively but, most importantly,

Trans. We evolved to take silicon into our brain but aluminium is taking its place.
It lied on its CV, it needs to be fired.

“This article summarizes the various ways in which Al induces oxidative stress, eventually leading to cell death. It also gives a brief account of manifold epidemiological studies that relate the abundant occurrence of Al in soil and water and the prevalence of AD. Al carriers, their role in AD, Al in neurofibrillary tangles, biochemical reactions altered by Al influx in mitochondria have been briefly discussed.”
One of the key words is apoptosis…

“If aluminium contributes to the development of sporadic AD, it must do so indirectly, perhaps via effects on the synthesis or metabolism of APP, or by contributing generally to the age-related attrition of neurons and thus reducing the threshold for deficits produced by more specific disease-related processes.”

Final study describes a mechanism.

Click to access 81923975.pdf

Back-up PDFs are always useful.



Active effect of aluminium on the brain’s self-cleaning.

“Aluminum, as an extremely high charge density cation (Z2/r = 18), has the remarkable capability to both (1) aggregate and compact Aβ42 peptide monomers into higher order, more neurotoxic oligomeric, and fibrillar structures, and (2) impair, at the molecular-genetic* level, the cellular machinery responsible for Aβ42 peptide monomer phagocytosis and clearance from the cell (4–13).”

*Hints at genetic damage.

Apparently the levels we ingest are “physiologically realistic.” As in, it can affect you.

They need chelation therapy.

The neurotoxin aluminium

Paper dump for SEO, a solid starting point.

Anecdotal but observational science: Baby Boomers have record Alzheimer’s rates. Boomers are also the first generation raised with aluminium, as “safer” than tin.
…It’s also cheaper.


A lot of restaurants don’t want to get sued (cosmetics companies neither).
The claims of papers like these, widely/falsely cited as ‘debunking’, don’t hold up to a basic level of biological knowledge. They are critical of a hypothesis for theoretical reasons they do not actively study, so it is not the all-clear being claimed by the intellectually dishonest, which would require actually testing them in a longitudinal experimental study (medical field standard). This is particularly important as any method design with heavy metals, which build up in the body (forming compounds together, in a cocktail effect) over decades. Rat studies go one-by-one, an element at a time, which lacks external validity/ real-world relevance.

1. the blood-brain barrier thins with age. This is a fact.
2. Aluminium can combine with other elements e.g. fluoride, to form a compound which might pass the barrier where healthy and young.
If you look at what’s literally IN the brain tissue of dead patients?

Respectfully, to the critics:

How the hell did it get there?

There’s your smoking gun, your bloodied Macbeth hands.

That is rock solid, irrefutable proof. As it stands, here is more proof.
Evidence for point 2: http://www.tandfonline.com/doi/abs/10.1080/10611860400015936
Known since at least 1998: http://www.actionpa.org/fluoride/aluminum.html
EU drinking/cooking water may contain arsenic, which explains a lot:

This is like the talc-cancer thing that recently came out in all the lawsuits.
I don’t have to explain that to you, do I?

Of course the brain is part of the immune system, we’ve been trying to tell you for years


I want to draw your attention to something.

Something small. The lesson is crucial.

“but the true significance of the discovery lies in the effects it could have on the study and treatment of neurological diseases ranging from autism to Alzheimer’s disease to multiple sclerosis.”

Called it on Alzheimer’s.

Called it on autism.

I feel like fucking Cassandra. I don’t say this from a point of vanity, it enrages me, the arrogance of these people to not look in the places pointed out to them for years (autoimmune response from vaccines) has caused how many millions to suffer?

And now they’re laughing it off, like, LOL, textbooks need to be changed $$$$$ for us.

I, among many others, have tried in vain to point this out, only to be told the same things: “there is no uncharted part of anatomy hence no point in even looking” and “that’s for real Doctors, we just need to give them drugs”.

Remember how this finding is being treated with flippancy when you automatically accord respect to researchers and scientists thanks to their credentials or fancy title. This is why I don’t use my real credentials, (see About) either the facts and truth are there or they aren’t – regardless of whom puts them forth. They don’t care about the truth, most of them, some care about their ego and others profiteering from ailments they likely caused when they pushed for things they didn’t understand and – worst of all, didn’t want to.

fury anger hades

Aluminium in Autism (and Alzheimers, Parkinsons etc).


good links

….“Aluminum is an experimentally demonstrated neurotoxin and the most commonly used vaccine adjuvant. Despite almost 90 years of widespread use of aluminum adjuvants, medical science’s medical understanding of their mechanisms of action is still remarkably poor. There is also a concerning scarcity of data on toxicology and pharmacokinetics of these compounds. In spite of this, the notion that aluminum in vaccines is safe appears to be widely accepted.” (5) – Dr. Chris Shaw, with the UBC Departments of Ophthalmology and Visual Sciences and Experimental Medicine and the Graduate Program in Neuroscience


Professor Christopher Shaw and Dr. Lucija Tomljenovic of UBC (in a recent study) also show that the more children receive vaccines with aluminum adjuvants, the greater their chance is of developing autism, autoimmune diseases and neurological problems later in life.  A demonstrated neurotoxin, Aluminum is the only approved adjuvant in the US.   Its use presents the risk of brain inflammation, autoimmunity and other adverse health consequences.(6)

Just as a side note, it is known that aluminum accumulates in the brain and that this accumulation is associated with Alzheimer’s and Parkinson’s diseases and with Gulf War Syndrome. (7)(8)

And dementia in tandem with flouride: http://www.psychologytoday.com/blog/iage/201407/is-dementia-caused-aluminum-through-fluoridation
FYI: sodium fluoride is an industrial waste product companies pay councils to dump in local water supplies.

As noted above, Dr. Seneff commonly makes the case that neurological brain diseases are a result of an insufficient supply of sulfate to the brain. She argues that systematic sulfate deficiency “may be the most important factor in many of the health issues facing us today.”

“One of the consequences of insufficient sulfate in the brain is that it impairs the brain’s ability to eliminate heavy metals and other toxins. To make matters worse, those same toxic metals also interfere with sulfate synthesis. The net result can be an accumulation of cellular debris.” – (source) Claire I. Viadro, MPH, PhD

This is supported by sulphur studies that show people don’t get enough in their diet: http://www.nutritionandmetabolism.com/content/4/1/24

It seems a paleo diet increases sulphur in one’s diet anyway: http://www.livestrong.com/article/289250-list-of-foods-high-in-sulfur/

“In addition, melatonin not only transports sulfate but also is an outstanding antioxidant and binds toxic metals to help dispose of them. It may come as no surprise, then, that melatonin impairment has been implicated in autism.” – (source) Claire I. Viadro, MPH, PhD

“Scientists are taking note of the fact that we live in an “age of aluminum,” with aluminum exposure occurring through vaccines as well as multiple other channels. Moreover, although many experts would have us believe that the question of thimerosal and vaccine safety went away after federal agencies issued lukewarm recommendations to reduce its use as a vaccine preservative in the early 2000s, Dr. Seneff noted that thimerosal is still very much relevant.” (source) Claire I. Viadro, MPH, PhD

In summary, Dr. Seneff is pointing to the fact that many neurological diseases of the brain have a common origin, which includes an insufficient supply of sulfate to the brain. She is concluding that enhanced toxic metal exposure (aluminum) impairs the brains ability to detox itself and eliminate them. As a result, these toxic metals interfere with sulfate synthesis, create a heparan sulfate deficency which in turn leads to autism (as mentioned and pointed out with the studies cited above.) (12)

Wait, are the preventative medicine people ODing on antioxidants correct?

I’d also like to mention that autism is comprised of a very large spectrum. Some of the ailments associated with diagnosis might not be ailments at all, but gifts that are in no way associated with vaccines. On the other hand, I do believe in some cases, characteristics that are seen in some autistic children are the cause of various toxins from pesticides to vaccines, especially children who have a genetic make up that makes them more sensitive to these chemicals. In some cases (I believe) it’s a result of these various toxins, and again, in other cases I believe it is a gift, and possibly an evolutionary step.

On an unrelated note, I’m throwing away my aluminium cookware. All of it.

fear scared woman retro