Brain size of males by race

https://thealternativehypothesis.org/index.php/2016/04/15/brain-size-race-and-iq/

includes this

The third piece of evidence is that the racial gaps have existed for a long time. In fact, based on the data from the tables above, the White/Black brain size gap doesn’t seem to have gotten any smaller during the 20th century.

Long before your 60s immigration acts.

The first thing to note is that brain size in the general population has a heritability of 87%. Thus, almost all the differences between individuals in brain size are caused by genes.

Secondly, we should note that racial brain size differences are present at, and even before, birth.

Keep blaming women though, that’ll help.

We need more direct studies like this. Gestation length and other variables must be accounted.

Menarche and other variables like WHR vary by race in the women too. More studies! MORE!

The manosphere is based on a myth of equalism, it was destined to fail.

Fourthly, racial brain size differences have been found all around the world. For instance, the Beals and Smith data set previously referred to features skulls from over 100 populations world wide. Many of the autopsy studies previously cited were done in East Asia, as were two of the MRI studies. This makes any gene independent cultural explanation less likely.

Fifthly, several studies have shown that mulattoes have an average brain size in between that of Blacks and Whites (Pearl, 1934Bean, 1906). This finding has been established on multiple occasions and is what a hereditarian hypothesis would predict since mulattoes are half White and half Black genetically speaking.

Do studies for other mixes.

Sixth, many traits which tend to co-evolve with larger brains also differ racially in a way that mirrors the body size adjusted brain size patternRushton and Rushton (2003) looked at 37 anatomical features which 3 textbooks on human evolution identified as tending to co-evolve in the hominid line with larger brains. For instance, larger pelvic size tends to co-evolve with brain size so that mothers can give birth to larger brained infants.  Rushton then utilized 5 forensic anthropology textbooks to look at racial differences in these traits. These traits followed the East Asian>White>Black pattern in 25 out of 31 cases. The probability of this happening at random is .000000001.

WHR is important.

Follow the tag, there are racial differences in studies.

And East-Asian isn’t a race, at best it’s a sub-group, do more studies. Properly.
Asians fail more often on IVF, also posted about that.

Similarly, Rushton (2004) showed that, across 234 mammalian species, brain size correlates with longevity, gestation time, birth weight, litter size, age of first mating, body weight, and body length. Various studies have shown that each of these variables also differ between the races in a way that, based on what we find across the animal kingdom, would predict the body size corrected brain size differences we observe (Rushton, 1995Templer 2006Rushton and Templer, 2009;).

Longevity – must look at averages e.g. not Danes for all whites nor Japs for all Asians.
Gestation – we know this varies.
Weight – control confounds.
Litter size – look at natural rate of twinning.
Virginity loss is r-select, do not want.
Body weight – vague.
Length – we already know racial height. Still, add to the rest, I guess.

Thus, we have six lines of evidence all of which would be predicted by a hereditarian view on racial differences in brain size. While each line of evidence on its own may not be compelling, the combination of all six seems to strongly imply that racial brain size gaps are partly heritable.

It is worth noting that the racial brain size gaps are probably not entirely attributable to genes. Some authors, including Richard Nisbett, have plausibly argued that nutrition also plays a role. However, there explanations are not mutually exclusive, no environmental variables has been shown to account for the majority of the gap, and,  as we have seen, there are many separate lines of evidence indicating that genes also play a role

A confound, in weight.

I suggest seafood consumption.

There is good evidence that the races evolved different brain sizes in response to climate. Specifically, various studies have found that a population’s brain size correlates with climate related variables. For instance, Pearce and Dunbar (2011) ‘s data set produces a correlation of .74 between a population’s brain size and its latitude. Similarly, Ash and Gallup (2007) found a correlation of .48 between the size of 109 fossilized human skulls and the latitude at which they were found. Further still, Bailey and Geary (2009) analyzed 175 skulls ranging in age from 10,000 years old to 1.9 million years old and found a correlation of -.41 between brain size and winter temperature and -.61 between size and latitude (larger brains were found in areas more distant from the equator).

How dare you use science.

The weebs might reee!

Lynn (2015) used Smith and Beals data set of 20,000 skulls from 122 populations to estimate that roughly 30% of the African-European IQ gap can be statistically accounted for by brain size differences. By contrast, brain size differences would actually predict an Asian-European IQ gap 35% larger than the one that actually exists. Thus, brain size is probably one of many factors, both biological and environmental, which account for racial intelligence differences.

Can ya rig the test?

https://hbdchick.wordpress.com/2013/07/02/intelligence-and-corruption/

Easy, who’s the most innovative?

Especially before colonialism spreading info. Right?

You can rig IQ tests and college transcripts, not more advanced farming equipment when the other group invented it centuries prior.

Estrogen and lifelong brain health, testosterone fraud

https://www.ncbi.nlm.nih.gov/pubmed/26109339

Estrogen facilitates higher cognitive functions by exerting effects on brain regions such as the prefrontal cortex and hippocampus. Estrogen induces spinogenesis and synaptogenesis in these two brain regions and also initiates a complex set of signal transduction pathways via estrogen receptors (ERs). Along with the classical genomic effects mediated by activation of ER α and ER β, there are membrane-bound ER α, ER β, and G protein-coupled estrogen receptor 1 (GPER1) that can mediate rapid nongenomic effects. All key ERs present throughout the body are also present in synapses of the hippocampus and prefrontal cortex. This review summarizes estrogen actions in the brain from the standpoint of their effects on synapse structure and function, noting also the synergistic role of progesterone. We first begin with a review of ER subtypes in the brain and how their abundance and distributions are altered with aging and estrogen loss (e.g., ovariectomy or menopause) in the rodent, monkey, and human brain. As there is much evidence that estrogen loss induced by menopause can exacerbate the effects of aging on cognitive functions, we then review the clinical trials of hormone replacement therapies and their effectiveness on cognitive symptoms experienced by women. Finally, we summarize studies carried out in nonhuman primate models of age- and menopause-related cognitive decline that are highly relevant for developing effective interventions for menopausal women. Together, we highlight a new understanding of how estrogen affects higher cognitive functions and synaptic health that go well beyond its effects on reproduction.

Men dosing testosterone are called meatheads for a reason, they would logically throw off their other hormones and functionally retard themselves. Because this ONE time, you can trust the pill people. No IQ studies in testosterone supplementing men, I guess there’s a good reason. They just forgot?

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4330791/

The results are, unfortunately, controversial and puzzling. Dosing, timing, even the application route seem to considerably affect the outcomes. 

You’re not trying to rig it at all, huh?

Reduction to dihydrotestosterone by 5-alpha reductase increases the androgen activity; conversion to estradiol by aromatase converts the androgen to estrogen activity.

Holy shit.

That’s hilarious.

Recently, the non-genomic effects of testosterone on behavior bypassing the nuclear receptors have attracted the interest of researchers. This review tries to summarize the current understanding of the complexity of the effects of testosterone on brain with special focus on their role in the known sex differences.

Not known.

a very important study in rhesus monkeys showed that pharmacological castration reduced and testosterone supplementation normalized anxiety levels (Suarez-Jimenez et al., ).

on the other hand, in both men and women, testosterone supplementation leads to improvement of depressive symptoms (Pope et al., ; Miller et al., ). However, not all interventional studies confirmed the anti-depressant effect of testosterone. At least in one published randomized controlled trial, the effects of testosterone were comparable to placebo effects (Seidman et al., ). Similarly, not all observational studies show a consistent picture. At least in one small study, depressive women had higher testosterone (Weber et al., ).

Another experiment on intact rats revealed that the effect of testosterone on depression is dose-dependent (Buddenberg et al., ).

Over the counter won’t work.

During the productive ages and even in early adulthood, men generally outperform women in spatial abilities (Linn and Petersen, )

Even for a few years you’re in college? Is that it?

Spatial thing is probably due to error, thus would be discounted under a valid method.

Error rate as well as the reaction time negatively correlated with testosterone (Hooven et al., ).

In another study, actual testosterone was not associated with spatial abilities, but prenatal testosterone correlated positively with spatial abilities in women (Kempel et al., ).

Congratulations, you’re on the female level. Apply your non-toxic internet cream.

In line with these findings is the lack of an association between actual salivary testosterone levels and mental rotation in men and women (Puts et al., ).

Actual science, no replication issue.

However, in a large observational study analyzing spatial abilities in adult men from various age categories, low testosterone was associated with better spatial visualization (Yonker et al., ).

Actual science, no replication issue.

Plus multiple ages in ADULT men, important.

Good work.

In a very interesting study, it was found that in men, the pubertal concentrations of testosterone are negatively associated with mental rotation in the adulthood (Vuoksimaa et al., ). In the same paper, the comparison of twins is reported. The twin with higher testosterone scored worse in the mental rotation tests. The results are contradictory, but may depend on the test used for the assessment of spatial abilities.

Counting fluke correct answers and not errors to force a finding is scientism. Bad method.

When virtual Morris water maze was used, a positive correlation between testosterone and spatial navigation was found in women, but not in men (Burkitt et al., ). The size of the corpus callosum seems to add complexity in the relationship between spatial abilities and testosterone (Karadi et al., ). This might be one of the causes for negative findings in studies where some of the determinants are missing (Kubranska et al., ). Another cause is likely the selection of the tested population. In gifted children, a negative correlation between salivary testosterone and spatial abilities was found (Ostatnikova et al., ).

FFS.

Negative findings are real science.

And that’s important.

In Chinese men, the accuracy in mental rotation tests was comparable to Americans, but the reaction times were longer indicating that cultural differences could add to the variability of published results (Yang et al., ).

No they didn’t rush the test out of boredom. Lower error rate, I’d bet.

Last but not least, genetic factors likely modulate the effect of testosterone. We have previously shown that at least in gifted boys, genetic polymorphisms influencing testosterone metabolism affect also its relationship to mental rotation (Celec et al., ). Especially, the CAG short tandem repeat in the exon 1 of the androgen receptor gene seems to be important for the action of testosterone and its metabolites (Nowak et al., ). Despite all complexity, the current picture indicates that the association between testosterone and spatial abilities is curvilinear and sex-dependent.

aka more is NOT better and it’s genetic, morons

In women higher testosterone is associated with better mental rotation, in men lower testosterone is associated with better spatial abilities. This seems to be true both for actual testosterone (Moffat and Hampson, ) and for prenatal testosterone (Grimshaw et al., ). Supplementation of testosterone in older men results in improvement of spatial abilities, but it is accompanied with changes in estradiol metabolism and it is likely that this interferes with modifications of spatial abilities (Janowsky et al., ).

They only studied spatial, not global, I checked.

Even in rats, testosterone administration affects the strategy of the animals in spatial tasks (Spritzer et al., ). However, the interaction between testosterone and mental rotation tests is bidirectional. It has been shown that mental rotation testing affects testosterone, at least in women (Durdiakova et al., ).

Doing smart things causes the brain to…. adapt? Really?

Does our pulse increase when we run? Some Sherlock Holmes do a fucking study.

MRI

Moreover, participants with complete androgen insensitivity syndrome presented with female-like neural activation pattern in the parietal lobe, indicating that gonadal hormone exposure rather than genetic sex itself plays role in brain functions (Van Hemmen et al., ).

Supplementing won’t work, you’re worse than the trannies. They don’t claim brain benefits.

The menstrual cycle and thus the involvement of sex hormones, including testosterone, in spatial abilities was further confirmed by Pletzer et al. In their study, error rates linked with deactivation of inferior parietal lobes and prefrontal lobes were higher during luteal phase for verbal tasks, while in the follicular phase, spatial abilities in females were confirmed (Pletzer et al., ).

One of the major factors that might explain the differences between the results of various studies is the variability of the examined populations. As mentioned above, the cultural differences, sex and age have all been shown to impact the physiological effects of testosterone. 

Duh.

Standardization in this area would surely improve our understanding of the neuroendocrinology of testosterone. More systematic research using the whole spectrum of available tools and looking at the various physiological aspects is needed. However, to be able to publish such research, journals should accept manuscripts based on the design and not on the results. Otherwise, the publication bias that is obvious in the so far published literature will continue to be a big issue. Many researchers in this field complain about negative results that are very difficult to publish in the relevant journals. The number of such unpublished observations and experiments is unknown. But based on our humble experience, the negative results will probably be more common than the published positive ones. And if the contradictory published findings are added, the picture gets even more confusing. Large systematic research projects with more cooperation between the most productive research teams is definitely needed.

You can’t because the low IQ men will complain if you publish negative findings.

e.g.

https://ohsu.pure.elsevier.com/en/publications/testosterone-influences-spatial-cognition-in-older-men-2

Testosterone plays a role in the organization of behavior during development. The authors examined whether testosterone could play a maintenance role in behavior as well. In a double-blind manner, verbal and visual memory, spatial cognition, motor speed, cognitive flexibility, and mood in a group of healthy older men who were supplemented for 3 months with testosterone were assessed. The increase in testosterone levels to 150% of baseline levels resulted in a significant enhancement of spatial cognition, but no change in any other cognitive domain was found.

You’ll be slightly more able to parallel park. Like when you were young.

Louder for the slow:

no change in any other

cognitive domain was found

NO IMPROVEMENT IN MEMORY

NO IMPROVEMENT IN MOTOR SPEED

NO IMPROVEMENT IN COGNITIVE FLEXIBILITY

NO IMPROVEMENT IN MOOD.

Did I stutter?

Testosterone supplementation influenced the endogenous production of estradiol, and estradiol was found to have an inverse relationship to spatial cognitive performance. These results suggest that testosterone supplementation can modify spatial cognition in older men; however, it is likely that this occurs through testosterone’s influence on estrogen.

I’m sure the health effects will be golden.

Eat your aluminium, thots

That’s right, smear on a lipstick full of shimmery bullshit. Including lead (and wonder why girls are depressed). [Literally eating lead.]

The Victorians ate arsenic but this is worse. Arsenic temporarily improved the complexion.

Using the term ‘mica’ for what’s essentially powdered foil suggests women wouldn’t want it if they knew what it was.

It isn’t like there’s a connection between dissected and tested Alzheimer’s plaques and aluminium deposits, is there?

Why are the Boomers raised on cans of this stuff seeing an explosion in Alzheimer’s rates?

As always, at first, go to the ‘officials’. Find out the Party Line.

https://www.alzheimers.org.uk/about-dementia/risk-factors-and-prevention/metals-and-dementia
Although aluminium has been seen in amyloid plaques”

K.

“there is no solid evidence that aluminium is increased in the brains of people with Alzheimer’s disease.”
Whew, what cognitive dissonance.
It’s literally forming those plaques that ARE the disease but nbd?

Solid evidence – like a solid chemical analysis of solid plaques in the solid brains of solid dead people with Alzheimer’s?

Look at the hustle to move those goalposts.

https://www.sciencedaily.com/releases/2007/08/070813185007.htm
“Because some people with the disease had aluminum deposits in their brains, it was thought that there was a direct connection…”
Well… yeah.
That’s proof.
There’s no negative evidence possible for that to stop being proof.

It’s literally in their brains.

“However, after many years of study, no conclusive evidence links aluminum to neurodegenerative disease”
Bullshit, 2007.

I detest scientism. Look at these papers and tell me what’s ‘conclusive’.

(Apart from donations of mega-corps to Super PACs to prevent this mega-lawsuit).

“A multi-institutional team of researchers has defined for the first time how metal ions bind to amyloid fibrils in the brain in a way that appears toxic to neurons. Amyloid fibrils are linked to the development of neurodegenerative diseases such as Alzheimer’s, Parkinson’s and Creutzfeldt-Jakob. Although metal ions, most notably copper, can bind to amyloid in several specific ways, the researchers found that only one way appears toxic.”

But how, ‘critics’ argue, could this possibly occur? What’s the mechanism? Nobody has proven a mechanism….?

2011:

“Link between Aluminum and the Pathogenesis of Alzheimer’s Disease: The Integration of the Aluminum and Amyloid Cascade Hypotheses”

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3056430/

“In particular, the link between aluminum and Alzheimer’s disease has been the subject of scientific debate for several decades. However, the complex characteristics of aluminum bioavailability make it difficult to evaluate its toxicity and therefore, the relationship remains to be established.”

Uhuh. Isn’t that your job?

If it’s impossible, why are you getting paid?

“On the contrary, aluminum is a widely recognized neurotoxin that inhibits more than 200 biologically important functions and causes various adverse effects in plants, animals, and humans.”

True.

Mounting evidence has suggested that significance of oligomerization of β-amyloid protein and neurotoxicity in the molecular mechanism of AD pathogenesis. Aluminum may play crucial roles as a cross-linker in β-amyloid oligomerization.

Here, we review the detailed characteristics of aluminum neurotoxicity based on our own studies and the recent literatures. Our aim is to revisit the link between aluminum and AD and to integrate aluminum and amyloid cascade hypotheses in the context of β-amyloid oligomerization and the interactions with other metals.”

But HOW could this POSSIBLY operate? – intellectually dishonest douches.

Note the prestige of journal. International Journal of Alzheimer’s Disease, those hacks.

But on a MOLECULAR level…. – douchecanoes.

https://www.onlinelibrary.wiley.com/doi/10.1002/jms.739
“Quantification of the Aβ peptide in Alzheimer’s plaques by laser dissection microscopy combined with mass spectrometry”

It’s behind a paywall but it’s there. Check the academic pirate bay.

http://orthomolecular.org/library/jom/2000/articles/2000-v15n01-p021.shtml
It’s been known for a while.
2000: “Aluminum has been identified as a neurotoxin for over 100 years.”

So stop using it in food, cosmetics, deodorant, cookware and cans?

Their use actually came in well after it was a known neurotoxin. Because that isn’t dodgy.

Reference for that sentence:

Doelken P:Naunynschmiedeberger. Arch Exp Pathol Pharmakol 40: 58-120 cited by Crapper McLachlan DR., Lukiw WJ, Kruck TPA, Aluminum, altered transcription, and the pathogenesis of Alzheimer’s disease. Environ Geochem Health, 1990; 12(1-2): 103-114.

Link for that: https://link.springer.com/article/10.1007/BF01734059

Opening paragraph.

“The etiology of some, if not all, cases of Alzheimer’s disease is linked to a mutation in the proximal portion of the long arm of chromosome 21∶21q11.2 → 21q22.2. While the functional consequences of the mutation are unknown, we speculate that one consequence of the mutation is loss of the natural barriers and intracellular ligands for aluminum. As a result, aluminum gains access to several brain sites including the nuclear compartment in certain neurons of the central nervous system.”

I know both my shit and my bullshit.

1990. Who owns the aluminium? That’d be a fun tour of genocide.

https://www.onlinelibrary.wiley.com/doi/abs/10.1002/ana.410310310
1992 laser study, note prestige of journal.

What the fuck do they know, right, reddit?

“Selective accumulation of aluminum and iron in the neurofibrillary tangles of Alzheimer’s disease: A laser microprobe (LAMMA) study”

“In addition, probe sites directed to neurons identified in snapfrozen cryostat sections from 2 subjects with Alzheimer’s disease revealed similar spectra with prominent aluminum‐related peaks, confirming that our findings are not related to exogenous contamination through fixation, embedding, or other procedures prior to analysis. This study further confirms the association of aluminum and neurofibrillary tangle formation in Alzheimer’s disease.”
1992.

But I guess I’m just scared of non-stick pans, where’s the evidence?

Marriage relieves stress, biologically

Don’t listen to Hollywood.

This Is the Secret Benefit of Marriage You Didn’t Even Realize

“A new study published in the journal PLOS One has provided evidence that having a spouse by your side can be a real stress reliever in a moment of crisis.”
“Interestingly enough, a 2018 study found that when romantic partners hold hands, their breathing, heart rate, and even brain wave patterns actually sync up, which enables them to relieve both emotional and physical pain. But this new BYU study is unique in that it used a more biological means of measuring stress, as opposed to relying on surveys.”

“The study also builds upon previous research that being married can help lower your blood pressure, body mass index, and cholesterol levels, reduce your risk of heart disease and dementia, and even boost your overall longevity. For more on this, find out why Science Says a Happy Spouse Means a Longer Life.”

happy wife, happy life
women don’t need to be told the reverse

If there were a supplement that provided all these benefits, the MGTOW lot would be jumping for it.

I think people would respect them more if they just admitted they haven’t found the right one, no shame in that.

Study Here: https://journals.plos.org/plosone/article?id=10.1371/journal.pone.0212703

“A new study that was published this week in the Proceedings of the National Academy of Sciences found that when romantic partners hold hands, their breathing, heart rate, and even brain wave patterns actually sync up. According to pain researchers, the more those brain waves synchronize, the more the pain that either of them feel subsides.”

Sweet hand-holding study: http://www.pnas.org/content/early/2018/02/16/1703643115

“The mechanisms that underlie social touch analgesia are largely unknown.* Here, we apply a hyperscanning approach with real-life interaction of dyads to examine the association between brain-to-brain coupling and pain relief. Our findings indicate that hand-holding during pain increases the brain-to-brain coupling network that correlates with the magnitude of the analgesia and the observer’s empathic accuracy. These findings make a unique contribution to our understanding of physiological mechanisms of touch-related analgesia.”

Look at the studies. Seems pretty biological to me. It had to pass Ethics.

The male pain is reduced more, it’s just difficult to tell since female results cluster.

Right end of the red line, compare to left legend. Male reduces to zero.

In real terms, the superior improvement of men should be reported.

*Unknown = pair bonding, idiots.

An important nb:

Why is all the religious stuff healthy?

Prayer, fasting, marriage, fidelity, community.

Activates the almonds.

Social evolution et al.

So it’s especially unhealthy that marriage rates have dropped among the working-class.

Why aren’t there rent controls on the cost of weddings? Just a basic wedding.

Not those poncy ice sculptures of a swan, screw those.

Pig brains revived hours post-mortem

But go ahead, be an organ donor!

https://www.sciencedaily.com/releases/2019/04/190417132805.htm

You won’t feel a thing!

“Circulation and cellular activity were restored in a pig’s brain four hours after its death, a finding that challenges long-held assumptions about the timing and irreversible nature of the cessation of some brain functions after death.”

Long-held?

These are the people you trust to know when you’re actually dead.

They might as well be informed by entrails.

Nerve damage in white males

Coincidental, just don’t look around.

https://www.sciencedirect.com/science/article/abs/pii/S0940299399800724

“The aim of this study was to investigate the effects of squalene on the central and peripheral nervous systems in rat at the ultrastructural level. Squalene was administered at a dose of 20 g/kg body weight, once daily for 4 days, and the animals were sacrificed 7 days and 30 days after the initiation of the experiment. After 7 days a mild swelling of mitochondria and dilation of the Golgi complex cisterns in few neurons in the cerebral cortex and hippocampus were observed. The swelling of astrocytes and their processes was also seen. Some myelin sheaths in the cerebral white matter were disintegrated. In the peripheral nervous system (the sciatic nerve), a damage of the Schwann cells, a destruction of the myelin sheaths, and lipid-like deposits between myelin lamellae causing a secondary compression of axons were present.”

Mercury also destroys myelin sheaths. However, it is eventually cleared from the body by chelating factors in the diet, with the exception of repeat doses e.g. from amalgam.
Why are modern boys slow in school, weirdly dulled and heavy addicts of social media, porn etc.?

Squalene administration caused a stimulation of fibroblast to synthesize collagen and an activation of macrophages in the perineurium.”

The collagen bit makes it fine topically for vain women (including moi).

“After 30 days, the lipid-like material was present in some neurons as well as in the myelin sheaths in the central nervous system. Endothelial cells were hypertrophic and a few demonstrated features of apoptosis.”

I find it odd a famous billionaire magically had a son who “grew out of” autism…

Italians supposedly tested a vaccine in December and found it …didn’t contain a vaccine. At all.
So technically, the things labelled and given as ‘vaccines’ cause issues.

What’s being called vaccinegate is this mystery mix death potion sold and forced on children as “vaccines”.

“Vaccinegate: Initial results on Infanrix Hexa chemical composition”
http://data.over-blog-kiwi.com/1/47/73/60/20190101/ob_d9ff7d_corelva-rapport-composition-vaccin-in.pdf

Almost like genetic targeting, but you’d need enough dumb ones submitting free samples for “research” in the T&Cs to make that a bioweapon.
You’d also need to own the labs and pharmaceutical companies. Do they use squalene?
https://www.cdc.gov/flu/protect/vaccine/adjuvant.htm See end.
Male genes are easier to target than female but the rumour is that the three-part tetanus (no such thing, it’s one-shot) is intended to prevent pregnancy.

In Infanrix Hexa we found
● chemical contamination from the manufacturing process or cross-contamination with other manufacturing lines;
● chemical toxins;
● bacterial peptide toxins;
● insoluble and indigestible macromolecule that reacts to the protein assay, but cannot be recognized by any protein databases.

We have not found:
● Protein antigens of diphtheria toxoids, tetanus, pertussis, hepatitis B, haemophylus influenzae B, Poliomyelitis 1-2-3;
● Formaldehyde and glutaraldehyde, phenoxyethanol, antibiotic residues indicated in the composition;

Autism symptoms is not autism, polio symptoms is not polio.

Note the wording.

https://www.iflscience.com/health-and-medicine/us-outbreak-paralysis-may-have-been-caused-newly-identified-virus/

“May”

I may be George Soros in a wig. Long odds but…. maybe.

Would there happen to be perineurium damage?

https://www.nbcnews.com/health/health-news/cdc-still-stumped-cause-mystery-paralysis-kids-n750276

I fucking bet they are. Bet you squalene antibodies in their bloodstream, easily resolved with another injection for the immune system to attack said antibodies.

They won’t pay out the Social Security bennies.

CDC:

FLUAD is a standard-dose, three-component (trivalent) inactivated flu vaccine, manufactured by Seqirus that contains an adjuvant. FLUAD is designed specifically for people 65 years and older.

Uhuh. Which adjuvant is this?

MF59 is an oil-in-water emulsion of squalene oil. Squalene, a naturally occurring substance found in humans, animals and plants, is highly purified for the vaccine manufacturing process. FLUAD is approved for use among people 65 years and older, who often have a lower protective immune response after flu vaccination compared to younger, healthier people.

I hate being right.

I hate it, I hate it, I hate it.

Feminine brain, youthful brain

https://www.smithsonianmag.com/smart-news/womens-metabolic-brain-age-around-three-years-younger-mens-180971425/

Predictable.

“New research suggests that women’s brains stay energized well into old age, making their brains appear about three years younger than men’s of the same chronological age.”

Given

https://disenchantedscholar.wordpress.com/2015/11/07/estrogen-boosts-brainpower-actually/

Linked in first:

https://www.pnas.org/content/early/2019/01/29/1815917116

Men didn’t evolve to live long, so it isn’t so much remarkable that women live longer with healthier brains but men,  to be competitive with other men in youth and war, age terribly simply because so few would survive there’s no evolutionary pressure.

Some tough choices ahead for the cooties crowd.

Stick to imaginary phantasms of 20th century masculine superiority solely intended to run up credit debt or actually be healthier.

Here comes the academese:

“Researchers remain uncertain why such metabolic differences exist”

EVOLUTION.

“It’s not that women’s brains seem to age slower than men’s,”

Literally is.
Watch the twist.

“Rather, it seems that women’s brains start off at a younger age when they reach adulthood, and they keep that throughout the remainder of their adulthood, basically buying them a few extra years.”

Not how it works. At all.

It’s like saying you start a race at the finish line, words have meanings.

If they’ve reached adulthood, they’ve reached adulthood. You can’t change the meaning to be quantum. Women mature faster (partially from earlier puberty and slower, longer sum development time because of that) and go on to metabolically age better because testosterone is known to burn out the system.

Gradual puberty + lower T = good for long-term brain health.

aka Not women’s fault. Help your fellow men. Maybe that explains why celibates like Tesla (who, let’s face it, probably didn’t masturbate), who kept their T-levels low as a result, lived longer and stayed lucid. Maybe that’s why monks live longer too, staying sharp. Male chastity has observable benefits.

The autobiographies of genius seem to evidence longer average puberty, too. The sensitive kid who’s a later bloomer?

The Victorians called masturbation self-abuse because the compulsion/addiction would tend to be co-morbid with other mental problems, including depression. How many porn addicts are depressed?

And what do they do to self-soothe because (someone) tells them it’s unanimously ‘good for you’?

Even nuns tend to live longer which does point to lower testosterone being a pre-requisite for a long life, neatly explaining why male levels gradually lower. It isn’t like evolution is picking on men. Aggressive animals have shorter lifespans across species. This is the price of being the protector/provider sex.

https://www.healthline.com/health-news/men-too-much-testosterone-linked-to-shorter-lifespan-112213

“Overall, men with circulating testosterone levels between 9.8 to 15.8 nmol/L range tended to live the longest.”

Men are entitled to know what their healthy levels should be, study them!

Men age faster cognitively, he’s like a magician trying to switch your attention onto women when there’s a medical problem with men.
Assuming they want to live long and prosper.

Like, sorry, if that’s a sexist assumption.

“Previous studies have found that aging women often exhibit stronger reasoning, memory and problem-solving skills than males of the same age. But it’s unclear whether this trend is related specifically to metabolism or to a different aspect of brain function.”

Hormonal interaction.

“This might mean women are a little bit more resilient to certain aspects of brain aging in general, but it could also introduce certain vulnerabilities,”

Lying liar. Not even one example? How can you try to spin a youthful brain as a bad thing?

Women aren’t so much MORE resilient (although oestrogen helps) but men are MORE fragile.

HELP THEM.

“Having a younger brain for longer could make the brain more vulnerable to certain things as well.”

Still no mention of men? This isn’t a woman’s issue, there’s nothing new here as stated, women already perform better as a known overall finding*, he totally ignores what men need because it involves admitting they aren’t the best at something….

These arrogant men are sex traitors. Their ego won’t let them admit men need help. So have fun with higher neurodegeneration risk because one guy won’t admit he’s not #1 at everything.

Men are not the control group, it’s the mean between both groups.

Basic category error and erroneous maths.

Hubris is a deadly sin. Treating male ill-health like it’s normal will literally kill men.

OT but relevant:

If we took men as the control, the shorter male lifespan is not only normal but hinted at being desirable, you don’t want to change the norm, with women being unusually longer-lived (greater distance from norm). Actually, the average is between the groups, again. The difference from the norm is smaller, easier to broach and bring those below it up to speed.

*Because unlike IQ studies, they can’t conveniently ‘exclude’ the left-side of the male bell curve to skew their findings dishonestly. This happens after they sample from middle-class academia pupils largely from suburban populations, a huge bias/filter itself.

Sure, throw other men under the bus and blame women for biology. Sexism both ways!

Reminder: