Female medieval English skeletons study

https://www.tandfonline.com/doi/full/10.1080/00766097.2015.1119392

“However, the period of ‘youth’ in medieval England, before the achievement of full social adulthood, may have extended well past physical adolescence, and the age of 25 years is often used as the cut-off point.14″

Louder for the pedos at the back.

“but for most medieval young women physical adulthood did not equate to social adulthood.16″

Obviously.

“Instead, puberty may have marked the beginning of the phase of ‘maidenhood’ rather than adulthood.17”

We now call it teenagehood but I prefer that name for women.

“Lifestyle changes for the teenager, in particular the onset of formal work, may have marked a further step away from childhood, particularly if this involved a move away from the parental home. That the 14th-century poll tax was levied on all those aged 14 years and above suggests that young women were expected to be earning their own money by this age.18

HA. Yeah, the guys who say women should sit at home all day twiddling their thumbs waiting to marry are 1. wrong and 2. have put too much stock in middle-class novelist Austen.

Like today;
“Although exact numbers are impossible to calculate, it is clear from the documentary evidence that a significant proportion of young women migrated to urban centres such as London and York to obtain employment, most commonly a service position.19″

Exactly like today:
“This move would have been a dramatic, and potentially a traumatic, change in lifestyle for young women. Although it may have brought greater freedom and responsibility, it does not seem to have conferred full adult status; there is evidence that young women in service were always viewed as ‘girls’ regardless of their age, just as young men were not viewed as full adults before the completion of an apprenticeship contract.20″

So they didn’t marry for money, they were already economically independent.

If you actually read history and here, forensics.

“in reality, marriage at such a young age was largely restricted to the nobility, with the average age at marriage in the general population estimated at 20–25 years,22
and perhaps even later following the Black Death.23
This would provide a very late age of achievement of ‘adulthood’ by modern standards. However, although marriage was very much the expected path a significant minority of women — perhaps around 15% — never married.24″

Who is dumb enough to have never looked this up?

I keep seeing Americans who make sweeping fictional statements about what ‘we’ Europeans did and it’s like… no. That’s never happened. Citation? Statistics? They are liars. Even in their revenge fantasies of ‘oppressing’ women from work (oh joy, welfare on the backs of random men? can’t win, can we?) then they assume all women would marry off (literally never happened in human history), all women are fertile and their children all magically survive (LOL) and that all men want to marry and got to choose who (LOL no). The economy also needs young workers, part of the immigrant problem is caused by not allowing teens to work.

They’re in bloody La La Land.

Extended maturation is K-selected, the men and women were tougher as a result.

Just realised my grandmothers might be in here.

Almost certainly. Yeah, don’t lie about my nana/s.

“Alongside these dramatic but infrequent events, most young medieval females would have experienced everyday hardships and hazards.”

” The average femoral diaphysis length recorded for the medieval 14-year-old females (354 mm) is closest to that recorded by Maresh for 20th-century 10-year-olds (348 mm). The average figures for medieval 15- and 16-year-old females (365 mm and 366 mm respectively) are still lower than for 20th-century 11-year-olds (367 mm). These data suggest that growth in medieval England fell well below modern standards, perhaps reflecting the lower standard of living medieval children would have experienced.”

If it was that hard on the girls, you don’t wanna go back to that, guys.

“It does not necessarily follow that medieval women were considerably shorter than their modern counterparts. When compared to dental formation, epiphyseal fusion in the female adolescent skeletons from our sample was delayed by two to three years in comparison to modern standards, allowing them to ‘catch-up’ their growth during the pubertal growth spurt.27 This pattern of extended growth appears to have been common in the medieval period;”

The English are tough.

” Only very slight differences in stature were noted between the women of Lincolnshire, London and Gloucester, although the London females had greater diversity in adult height.”

“This may suggest that girls who experienced poorer conditions for childhood and adolescent growth were more likely to die around or before the age of 25 years.”

K-selection. Stunted or shorter women likelier to die. Same with men.

“It has been suggested that female height may have suffered in comparison to male height in medieval Europe due to preferential feeding and care of male children,33causing greater sexual dimorphism in growth and final stature between the sexes. By comparison, the average stature of young men at our sites (156 individuals) was 169.5 cm (5 ft 7 in). This may simply be the result of sexual dimorphism as such comparisons are similar for modern western populations, and therefore does not support the hypothesis that girls experienced poorer nutrition and living standards than boys.”

K-types invest well in all offspring.

“According to these indicators, it appears that all of the individuals studied had entered the pubertal growth spurt by the age of 14 years. In the modern western world girls tend to begin puberty around the age of 10 years,37 and so this result would fit with modern expectations. “

Puberty begins then takes a few years, 14-18/19 matches what I read elsewhere about menarche (posted here).

The ‘modern’ data is skewed by non-whites, especially Asians and Africans, with much lower menarches.

The African is nine, measured in America, as I recall.

“More information can be gained from examining the epiphyseal fusion of the hand phalanges, a process known to occur during the deceleration phase of the pubertal growth spurt, and correlated with first menstruation in modern females. Although the age at which this event occurred varied in our sample just as among modern girls, fusion appears to have occurred most frequently between 15 and 17 years (Fig 2). At 14 years, only 36% of girls display fusing or fused hand phalangeal epiphyses, but by 17 years this figure has risen to 84%.”

Still not 100%, K-types have a later range of menarche.

“A second skeletal event known to be associated with first menstruation, the ossification of the iliac crest of the ilium, was also only found in girls aged at 15 years or over. Interestingly, this is roughly in line with the average age at menarche suggested by the few available documentary sources.38 An average age at menarche of between 15 and 16 years would be much later than the modern British average of just under 13 years.39In addition to their shorter stature, this finding adds weight to the argument that environmental factors such a deficient diet and disease were having a negative impact on medieval female growth and development. Interestingly, however, this average age at menarche is below the age of 17 years recorded for mid-19th century females,40indicating that urban conditions were not as detrimental as those experienced during rapid industrialisation.”

The female body takes YEARS to develop, periods often occur too early to carry a child to term. Hollywood lies, because it’s full of creeps.

Men shouldn’t be discussing a reproductive system they cannot understand.

“The evidence for medieval England, however, shows a delay in the achievement of this milestone, which appears to have fallen between 17 and 18 years for most girls, based on 247 individuals with this bone surviving (Fig 3). Complete fusion of the iliac crest of the ilium, which signals the end of pelvic growth, was only seen in a minority of women aged below 20 years, based on the 277 individuals “

They’re K-types, it isn’t a delay, it’s NORMAL. Modern people are aberrant.

17-18 periods stabilize (this takes years, I have spoken to doctors about it).

The pelvis keeps growing to carry and support a child though, only when this is done (about 21, spinal plate fusion) is the woman actually sexually mature with a low risk of still birth, miscarriage or death.

Modern medicine is allowing a lot of non-white thots to survive a process Nature is telling them is fatal. Do not confuse that with Nature’s approval.

These data suggest that puberty was extended into the very late teens for young medieval women, pushing back the timing of achievement of full physical adulthood. This extended period of physical adolescence indicates that living standards for young medieval women, at least in the urban and small town environments, were considerably poorer than those of modern British adolescents. Some variation between the sites was noted, with pubertal development most advanced in the small town of Barton-upon-Humber, and most delayed in the urban hospital cemetery of St Mary Spital, London. This presumably reflects the harsher living conditions experienced by the girls living and working in London.”

Nah, hard work and low fat diet. Treating the women like men will delay them more.

“It is believed that the demographic changes caused by the Black Death may have led to increased opportunities for many women to migrate and work.43

Although less documentary evidence is available for women than for men, there is evidence for female servants much younger than 12 years in urban households,44and some migration may have occurred at a very young age. Although legislation was passed to regulate the minimum age for apprentices — 13 years in the early 14th century, rising to 16 years by the 15th century — apprenticeships were rarely available for girls, and no such legal minimum age existed for servants or casual workers. The available evidence suggests that girls started formal work away from home at a younger age than boys.45

This concept of female laziness is really American.

” a degree of personal freedom; the latter is perhaps most clearly indicated by the large number of migrant women recorded as making ‘merchet’ payments for the right to choose their own marriage partner.46 On the other hand, moving away from home, particularly to a town or city, could bring with it new challenges and hazards, such as sexual predation, mistreatment, injury and disease.”

Americans are so wrong it hurts.

” this result indicates that much greater numbers of women living and dying in London were actually suffering from tuberculosis.”

“Again, the numbers are too small for statistical analysis, but this may provide further evidence for girls having a more indoor lifestyle than boys in the medieval period.”

Forcing women to sit at home is literally bad for their health.

We aren’t mole people.

On the whole, the women actually had it harder than men.

“There can be little doubt that this extensive workload was exhausting for many women, but osteological study can provide further direct evidence for the impact that this had on young women’s bodies.

A wide range of trauma has been recorded on the skeletons of young medieval women, including fractures of the upper limb and finger bones, cranium and ribs, lower limbs and feet.57 However, the prevalence of fractures of each type is lower than among males, suggesting that girls were exposed to (or exposed themselves to) fewer risks of injury than boys.”

We hadn’t evolved for that labour, men did.

“It is notable that, of the 48 cases of trauma reported in the grey and published literature, cranial, rib and jaw injuries, suggestive of interpersonal violence, only started to appear in women aged 17–25 years, comprising 18.6% of the 43 fractures for this age group. This suggests that the risk of violence rose as girls turned into young women, perhaps reflecting domestic violence after marriage.58″

That would explain the death rate. Stress and fractures – no healthy baby.

There is one area of the skeleton where young women seem to have suffered virtually the same frequency of fractures as young men, the vertebral column. By far the highest prevalence rate for vertebral fractures (4.7%, n = 9) was found at St Mary Spital suggesting that female workers in the capital, or at least the poor workers buried in this hospital cemetery, were undertaking the activities most likely to cause spinal injury. The majority of these fractures were compression fractures, often caused by falls from a height, although avulsion and hyperflexion injuries were also present.59

The men sitting at a desk in an apprenticeship had it easy.

“Schmorl’s nodes are common, often asymptomatic, depressions caused by herniation of the nucleus pulposus on the superior and inferior surfaces of the vertebral bodies. Their aetiology is complex, although spinal trauma caused by vigorous activity and flexion and extension of the spine is most commonly associated with their formation.60 The age of their occurrence is not clear, but they generally appear before the age of 18 years.61Plomp et al argued that males are more susceptible to these lesions due to the size and shape of their vertebrae.62 In our study, medieval women had a higher prevalence of the lesions). Analysis of the location of Schmorl’s nodes on the vertebrae revealed that the lumbar vertebrae were affected far more often among women, and the central thoracic vertebrae among men. This mirrors vertebral fractures where in the women all of the fractures occurred in the lower thoracic and lumbar vertebrae, while in young men the central thoracic vertebrae were affected. This may suggest different activities; strain on the lumbar vertebrae, in particular, may be caused by bending and lifting.63″

aka back breaking labour, which could cause…

“Further evidence for stress being placed on the spines of young medieval women is provided by cases of spondylolysis. This describes the partial separation of the inferior facets on the neural arch from the vertebral body, usually between the ages of 10–12 years. The condition results from microtrauma in low grade stress on the lower back due to bending and lifting strains, or a fall from a height,64 but may have an underlying congenital cause. This injury was present in 4.4% of the female skeletons examined. This is higher than the prevalence of this condition found by the authors among young medieval males (2.9%), although the numbers involved were too small for statistical analysis. Again, the area involved is the lumbar region of the vertebral column. In addition, three young women, two aged at around 21 years and one at 22–25 years, display early degenerative joint disease of the vertebral column.”

Forcing women into labour like that kills them, reminder.

What emerges from the osteological evidence is that the workload of many young medieval women appears to have been literally backbreaking, and these early injuries may be expected to have led to significant back problems and pain in later life. It seems likely that these early spinal problems were caused primarily by carrying heavy loads at a time when the spine was still forming and vulnerable. Research from the grey and published literature reveals that rates of spinal injury were higher in urban than rural women65 and suggests that the workload of the young migrant women in service was harder than that of the young women who remained in the country or in small towns with their families. For example, the prevalence of vertebral fractures, spondylolysis and Schmorl’s nodes was lowest at Barton-upon-Humber, a wealthy small town.66″

Marriage, Sexual Activity and Childbirth

There is considerable evidence to suggest that marriage was a defining moment in the medieval female life course, marking the transition into true social adulthood.67 It is notable, however, that there was a significant gap between the legal age at marriage (12 years) and the average age at marriage (20–25 years even before the Black Death) in medieval England.68 The new analysis of pubertal development in medieval England discussed above suggests that the average age at menarche was 15–16 years. Full fertility, in terms of the likelihood of conception, carrying a healthy pregnancy to term and surviving childbirth, would only have followed several years after menarche with the completion of pelvic growth,69 which in our medieval sample appears to have been rare before the age of 19 years.

aka what I already typed, dammit

The fact that many young medieval women would not have been fertile before their 20s may be one reason for the relatively late average age of marriage during this period.70 It also suggests that marriage at the legal minimum age of 12 years would rarely have been fruitful, and any pregnancy that did ensue would have carried significant risks for the mother. We know of several medieval legal cases of the marriage of young girls where the ‘physical readiness for marriage’ of the girl in question was debated.71

This don’t go to college because you get periods thing from America is pig ignorant on female anatomy.

There is evidence to suggest, however, that the majority of cases of marriage before 15 years were confined to the nobility.72Today, girls of higher socio-economic status, with a considerably better standard of life, mature earlier than average. For example, high caste girls in 20th-century India have an average age at menarche over a year younger than low caste girls.73 The average age at menarche for noble girls in medieval England may therefore have been younger than the average age of 15–16 years described above.

more r-selected by men, explains eventual decadence and homosexuality rates, especially in the French

Even so, a pregnancy before the completion of pelvic development would have been dangerous; a famous example of this is provided by Margaret Beaufort, who appears to have been rendered sterile by a difficult first birth (of the future king Henry VII) at the age of just 13 years.74 An understanding of these risks is demonstrated by several contemporary authors,75 and was reflected in the Jewish rule that contraception (banned by Christian teaching) could be used to prevent pregnancy if the bride were too young to safely bear a child.767

The guys trying to force women to reproduce young would ironically render their own wife sterile via their stupidity. Good riddance. The Lord works in mysterious ways.

In theory, marriage coincided with sexual initiation for young women, and if the Church’s remonstrations to remain celibate until marriage were universally followed, it would indicate a relatively late age of sexual initiation. In reality, premarital sex among betrothed couples seems to have been common,77

that links to this study, no, they weren’t slutty

seems*

no

and sex with other partners, in not all cases consensual, was far from rare.

Are you really counting rape?

Evidence for this is provided by the erratic enforcement of ‘legerwite’ or ‘leyrwite’ fines on serf women who engaged in premarital sex.78

What about the men.

Premarital sex is thought to have been particularly common among young girls and women living away from home, for example in service roles, due to the greater freedom and availability of partners as well as the risk of sexual predation or pimping from employers.79 The sexual exploitation of girls in service appears to have been a frequent problem based on the legal record,80 and many young women must have lost their virginity in these circumstances. The extensive focus of many writers on admonishing young women to stay celibate until marriage may be taken as further evidence that premarital sex was seen (at least for women) as a significant societal problem.

Rape isn’t sleeping around, WTF.

Pedophiles raping virgins don’t really count as premarital sex, a choice, does it?

Two aspects of osteological analysis may shed light on sexual activity among young medieval women. The first is a sexually transmitted disease. Venereal syphilis, a treponemal disease, affects the skeleton in its tertiary stage, causing distinctive skeletal lesions.81 From the end of the 15th century, syphilis is believed to have been endemic in urban areas of England, although recent work has suggested that it may have been present at a much earlier date.82

Men spread that, sailors caught that. Your point?

If a virgin woman married a man with it, she’d get it. That can happen after marriage.

These female authors really want to present all women throughout history as sluts. Cui bono?

Among the 14–25 year old female individuals examined, four probable cases of treponemal disease were recorded, based on the presence of characteristic gummatous lesions in the cranium or long bones.83 Three of these were found in the young women from London (Fig 5), and one was found in York, at St Helen-on-the-Walls. One further case is known from Blackfriars, Gloucester;84 no cases were identified in the rural or small town sites consulted in the wider survey. The two youngest women to show signs of treponemal disease were aged at just 16 years. It is difficult to rule out congenital syphilis in these cases, as the presentation of the two conditions can be very similar, although none of these skeletons display the typical dental deformations of congenital syphilis.

So their fathers were sluts, so?

If the disease is the venereal form of treponemal disease, or syphilis, this would suggest the girls were very young when first infected. Syphilis generally takes several years to cause such destruction in the skeleton.85 Although the number of cases recorded is small, given that only 10–20% of individuals with tertiary syphilis experience skeletal involvement, and that skeletal lesions take several years to develop,86 it seems likely that much greater numbers of young women were affected by this disease.

To imply they wanted to be raped by syphilitic men is a bridge too far though.

The spread of sexually transmitted diseases such as syphilis was exacerbated by the problem of prostitution in medieval towns and cities. Karras argues that regulations of the Guilds limited women’s access to the normal labour market, forcing them to turn to prostitution out of necessity.87

Assuming that was a mistake.

There is little direct evidence that apprentices were procured as prostitutes, but one extant record from London City and Ecclesiastical Court (ad 1423) attests that one Alison Boston took apprentices who she hired out for the ‘horrible vice of lechery’.88 There are also accounts of men taking young girls (invenculae) to the London stews and selling them as prostitutes, suggesting the types of danger faced by young unskilled immigrant women. Goldberg89 cites the famous references from medieval York in ad 1482 that place prostitutes within the legal realm of ‘lepers’ and pigs in the hazards they caused for the local population.

Enslaved children.

She does not discuss the age at which women may have turned to prostitution, but suggests widows and daughters of labourers, known as ‘spinsters’ and ‘seamstresses’ (sempsters), needed to work several jobs to make ends meet, including petty theft, illegal ale retailing and prostitution. Goldberg argues that although full-time, ‘professional’ prostitutes were rare, many women were forced into occasional prostitution in hard times.90

Contradiction, Goldberg.

also why we have the welfare state

This would have been a particular risk for a migrant girl away from the safety of her family.91 Although it is impossible to state that any of the young medieval women examined were forced into this profession, this must be considered in the cases where possible syphilis is recorded.

No shit, nobody would choose that. The excuses these women make for rape are appalling.

A second consequence of sexual activity, pregnancy, may also in exceptional circumstances be visible in the archaeological record. In total, eight cases of young women buried with fetuses in utero have been recorded from medieval cemetery contexts. These burials represent ‘obstetric catastrophes’ with the death of both mother and child in late pregnancy or childbirth. Although there was a Christian injunction in place in medieval England for infants to be removed from their mother’s womb before burial,92 this does not appear to have been rigorously obeyed.

Yeah, who wouldn’t choose to die like that? I guess they were all just happy sluts, huh Mizz Feminist?

All of the individuals buried with a fetus in utero in medieval cemeteries have an estimated age at death of around 20 years or over, and thus none represent particularly young ‘teenage’ pregnancies.

Because they rarely got pregnant. Look at the evidence.

This may support the idea that in the medieval period teenage girls were not falling pregnant, as first pregnancies are often seen as the most hazardous.93

May? It’s anatomical.

It also fits with the known late pattern of marriage in this society. However, it is by no means certain that all of these women were married. The two examples from St Mary Spital may have represented extramarital pregnancies as the hospital was known to accept unmarried women in pregnancy or childbirth.94 It may be significant that neither of these women received an individual grave or any grave ornamentation. In contrast, the elaborate nature of one young mother’s burial at Barton-upon-Humber, in a coffin within the church and with a cloth of gold artefact,95 surely indicates that this woman was married and held a position of substantial social standing.

Clearly, their situation was a choice.

Given the high mortality rate of women in childbirth in the medieval period revealed by documentary sources,96s it is clear that these rare burials represent a dramatic under-estimation of the real levels of maternal mortality. In many cases, the churches prohibition on burying fetuses in utero may have been observed. In a large proportion of births, too, the child may have been saved, leaving little clue as to the cause of death of the mother.

But doctors (when sane) will elect to save the mother because she can have countless children later but an orphan baby is already financially a goner. Remember this, America.

Conclusion

The period of social adolescence for young medieval women seems to have been an important life stage, encompassing the growth to full physical adulthood and fertility, the adoption of adult working roles and, for most young women, the move from legal dependence on a father to legal dependence on a husband, with perhaps a few brief years of relative independence in between. The comparative absence of young women from documentary sources means that osteological information plays a vital role in our understanding of this group, and it can reveal a great deal about the way in which medieval girls grew into women, the living conditions they enjoyed or endured, the work they did and the health problems they faced.

Many of the conclusions drawn from osteological analysis of this group articulate with and illuminate the documentary evidence. The average age at which full fertility appears to have been achieved, around 20 years, is substantially later than in modern England, but ties in well with the known average age at marriage in this society. The greater susceptibility of young women to respiratory infections, from the relatively benign maxillary sinusitis to the deadly serious tuberculosis, chimes with the picture drawn from documentary sources of an indoor lifestyle for women, close to the smoky fire, and of the cramped living conditions that helped to spread disease. The backbreaking work clearly undertaken by many young women paints a clearer physical picture of their daily lives than that provided by documentary sources alone, and the development of signs of venereal disease in very young women hints at the problem of girls being driven to prostitution in England’s medieval cities.

Gang rape, we still have it. They are driven to it, slave-driven.

Problems with “Gladiator diet” rhetoric

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4198250/

  1. they were slaves, restricted diet
  2. they were poor, no choice in diet
  3. they were often diseased, multiple
  4. they’re studying the ones bad at the job, enough to die
  5. there’s no such thing as one gladiator, they weren’t even mostly male.

Gladiators in practice wanted to put on a little fat at least to protect the major organs from spears and knives. Do not carry modern Ho-wood assumptions to the ancient world.

The gladiator cemetery discovered in Ephesus (Turkey) in 1993 dates to the 2nd and 3rd century AD. The aim of this study is to reconstruct diverse diet, social stratification, and migration of the inhabitants of Roman Ephesus and the distinct group of gladiators. Stable carbon, nitrogen, and sulphur isotope analysis were applied, and inorganic bone elements (strontium, calcium) were determined. In total, 53 individuals, including 22 gladiators, were analysed.

aka most of those tested were NOT gladiators

yet included?

All individuals consumed C3 plants like wheat and barley as staple food. A few individuals show indication of consumption of C4 plants. The δ13C values of one female from the gladiator cemetery and one gladiator differ from all other individuals. Their δ34S values indicate that they probably migrated from another geographical region or consumed different foods. The δ15N values are relatively low in comparison to other sites from Roman times. A probable cause for the depletion of 15N in Ephesus could be the frequent consumption of legumes. The Sr/Ca-ratios of the gladiators were significantly higher than the values of the contemporary Roman inhabitants. Since the Sr/Ca-ratio reflects the main Ca-supplier in the diet, the elevated values of the gladiators might suggest a frequent use of a plant ash beverage, as mentioned in ancient texts.

Are you telling me they sucked down on almond butter? Legumes, really?

Beverages are not food.

And it might’ve been to stave off infection (use for ash).

Due to the preferred uptake of Ca compared to Sr and the resulting “biopurification” of Ca in the food chain, it was long believed that Sr functions as a dietary plant-to-meat ratio indicator . It was assumed that a high Sr amount in the bone reflected a high vegetarian intake, and a low Sr amount indicated a diet rich in meat. Therefore, studies of Sr/Ca-ratios for the reconstruction of diet were introduced in 1965 for extinct animals and in the 1970s and early 1980s for prehistoric humans . Since then, numerous studies have dealt with this subject. Thus, we refer to detailed review articles(e. g. by , and ). Two major issues with this concept of diet reconstruction have to be highlighted:

Assumed?

First, the dietary plant-to-meat ratio approach experienced a major correction by Burton and Wright . They showed that Sr/Ca ratios in bone reflect the Sr/Ca ratio of the strongest Ca supplier rather than the whole plant-to-meat ratio of a multicomponent diet. This limits future Sr/Ca studies to identifying the major Ca source of the diet.

A second major drawback for the investigation of trace elements arose from the fact that bones buried in soil undergo diagenetic alterations . Many attempts were made to identify and quantify diagenetic changes to Sr/Ca-ratio with the target to reconstruct original Sr/Ca-ratios (summarized in ). Because of the complexity and variability of soil processes, this seems impossible up to now. Therefore, the scientific focus has shifted from trace elements to stable isotopes for diet reconstruction in recent years. Only few combined studies on trace elements and stable isotopes are available. Though we believe that trace element and stable isotope investigations complement each other and have the potential to draw a more comprehensive picture of ancient diets.

it gets worse

run while you still can

The bone sample preparation followed a protocol developed for spectroscopic trace element analysis in bone or teeth . First, the bones were superficially cleaned with tap water and dried at room temperature.

Archaeologists are idiots with spades, exhibit A.

TAP WATER.

ELEMENT ANALYSIS CLEANING.

THEY USE FUCKING TAP WATER.

on ancient samples, my stars

leave it out on the counter near students, it’ll be fiiiiiine

All individuals consumed C3 plants like wheat and barley as staple food.

i.e. Not fucking kale.

Pliny the Elder reported in his Naturalis historia that barley (hordearii), a C3 plant, was a main component of the gladiators nutrition which matches our findings . However, most NG individuals apparently also had C3 plants as a staple food since no statistical difference is detectable.

apparently? no ‘statistical’ difference? detectable?

whose job dis?

Me marking papers, the flashbacks.

A few individuals show more positive δ13C values. The most probable cause for this is an increased consumption of C4 plants such as millet.

probable how

no

how about no

keep filling in those gaps with BS, I’ll wait

Millet was an important nutrition crop in Eurasia and its consumption has already been verified in several ancient societies by stable isotope investigations , especially since the Middle Bronze Age in Italy  published similar δ13C data for imperial Rome Isola Sacra (−18.8‰ versus −18.9‰ here). They mention ancient literature in which millet is described as an animal food and as less desirable for human consumption under “normal” circumstances.

So animal feed. Not suggested for modern humans. K.

In their study, the δ13C values of the faunal remains are more negative than those of the humans, so it was assumed that the terrestrial herbivores basically fed on C3 plants. Although animal bones have not been investigated yet for reference in our study, we agree with .

wut

Cunts with spades, I swear.

Yeah just throw some tap water on it then wonder about the calcium. It’s fine!

If the δ13C values were caused by the consumption of millet-fed animals, then higher δ15N values would be expected for the Ephesus population.

If. I read a scientific analysis of elements for a fucking IF.

It’s like grading a philosophy paper. IF.

If I were Batman.

One of the individuals with a rather strong signal for C4 plants, a δ13C value of −17.8, belongs to the gladiator group (EPH-DAM 187/93 rFEM 6) (Fig. 3).

One person?

One?

Freud had larger sample sizes!

Another gladiator (EPH-DAM 248/93 rFEM 6) and one female (EPH-DAM 72/93 rFEM-1) show at least hints for a mixed signal of C3 and C4 plants.

Kill me now.

show – ok

at least – numbers?

hints – no

Fuck off and die. NO.

mixed signal – a pox on your family line

This individual is also extraordinary as she was the only female to be found in the gladiator cemetery.

No.

This leads to two hypotheses: 1) these individuals had a different diet than the others

no shit Sherlock

or 2) they came from other geographical regions with a more C4 plant-based diet and migrated to Ephesus.

and died before leaving, let’s tell everyone to eat like slaves who died young!

The second one is more probable because the extensive cultivation of millet is only evidenced since the Early Byzantine Period, ca. AD450, for this region . Comparable δ13C data from Roman North Africa (Tunisia) with a mean of −17.7‰ were published by . However, their combined δ13C and δ15N values suggest a significant amount of dietary protein intake from marine resources.

You slipped that one in there, didn’t you?

Last sentence of section. Thought I wouldn’t notice, but I did.

An ancient cookbook written by Apicius which contains a collection of Roman recipes, suggests that seafood was probably consumed in Ephesus. Fish was most likely eaten as fish sauce (garum), but also cooked and salted.

Yeah another thing Whites invented.

 In contrast to this, the two individuals with the highest values are from the gladiator group, too (EPH-DAM 146/93 rFEM 1 and EPH-DAM 76/93 rFEM 2). That indicates a regular consumption of animal proteins and a lower intake of legumes. Within the gladiator group, the isotope data extend over more than one trophic level for δ15N but also for δ13C. This leads to the conclusion that the individuals from the gladiator cemetery were a very heterogeneous group who consumed different kind of foods.

Too pissed, you read the rest.

The meat values simply don’t show up because their methodological test is biased to conceal it with legume intake.

Fake burger, real trans fats

Big Food has a lot to answer for.

DECODING LABELS: Beyond Meat Chicken

Higher consumption of unsaturated fats linked with lower mortality

Mmmmmm, bleeding vegetable oils.

Trans fats—on their way to being largely phased out of food—had the most significant adverse impact on health. Every 2% higher intake of trans fat was associated with a 16% higher chance of premature death during the study period.

That’s a big deal.

Eightfold.

And cumulative.

Higher consumption of saturated fats was also linked with greater mortality risk. When compared with the same number of calories from carbohydrate, every 5% increase in saturated fat intake was associated with an 8% higher risk of overall mortality.

That’s practically insignificant proportionally, especially since the method painstakingly avoid citing animal source or non-animal sourced fats. Too vague.

Conversely, intake of high amounts of unsaturated fats—both polyunsaturated and monounsaturated—was associated with between 11% and 19% lower overall mortality compared with the same number of calories from carbohydrates.

You cannot compare to sick Americans as your control population. No.

Bad scientism.

They don’t want people learning how to be really healthy. It would show up on their data more. You need half a century of plant propaganda to convince people our declining health is good for us.

Among the polyunsaturated fats, both omega-6, found in most plant oils, and omega-3 fatty acids, found in fish and soy and canola oils, were associated with lower risk of premature death.

The Western diet is toxic purely because there’s too much omega 6, a deadly excess.

The lifespan of India also suggests avoiding animal products is…. ill-advised.

People who replaced saturated fats with carbohydrates had only slightly lower mortality risk.

eat your GMO grains, you’ll live longer

sounds legit

“Our study shows the importance of eliminating trans fat and replacing saturated fat with unsaturated fats, including both omega-6 and omega-3 polyunsaturated fatty acids. In practice, this can be achieved by replacing animal fats with a variety of liquid vegetable oils,” said senior author Frank Hu, professor of nutrition and epidemiology at Harvard Chan School and professor of medicine at Harvard Medical School.

An Asian at Chan school.

Looking as Asia’s meat consumption, you are lying.

Chinese meat imports to expand in 2019: https://apps.fas.usda.gov/psdonline/circulars/livestock_poultry.pdf

Despite global promises to reduce over the past few years.

Maybe I’ll do a whole post.

The only confirmed finding is lab grown shit is toxic.

This Is Your Brain On Trans Fats, Artificial Sweeteners And Alcohol

Trans Fats

As of June 18 of this year, trans fats have been officially banned from all manufactured foods.

Nope.

Research in the 1990s found a strong link between trans fats, also known as “partially hydrogenated oil,” and heart disease.

You are what you eat.

In addition, more and more studies are finding that trans fats cause cellular destruction in the brain and can adversely impact hormone production, memory and increase inflammation by suppressing the production of Omega-3 fatty acids.

Actively suppresses omega 3.

Now, our brains do need fat to function. In fact, the brain relies on natural, healthy fats to maintain cell membranes, keep brain inflammation at bay and transport fat soluble vitamins such as vitamin A, D, E and K. Focus on getting your daily doses of fat from olive oil, coconut oil, nuts, seeds and avocado.

A study of almost 3000 people in 2017 reported a link between artificial sugar consumption and an increased risk of stroke and dementia. In the study, those participants that reported having just one diet beverage a day were three times more likely to develop dementia or suffer from a stroke. Although more research is needed, the findings are alarming.

Aspartame, one of the most commonly used artificial sweeteners in foods and beverages, has also been shown to cause neurodegeneration, mood imbalances and potentially cause brain cells to kill themselves. Instead of artificial sweeteners, use small amounts of natural sugar, such as raw honey, pure maple syrup or dates.

Yeah it also tastes like death.

A literal neurotoxin.

A study of 550 men and women over 30 years published in The BMJ found that even moderate drinking, which they defined as about 6-9 drinks a week, can damage the brain, including hippocampal atrophy.

Anyone that has had a few drinks knows that alcohol can impair judgement, balance, speech and even memory. Many studies have shown that long term heavy drinking can create serious and persistent changes in brain function that include mental confusion, eye nerve paralysis and difficulty with muscle coordination.

Drunkenness is just a form of brain damage that becomes more persistent with chronic alcoholism.

Until they never ‘sober up’ – clear the toxin, ever again.

Let the brain shrinkage vegans eat their fake flesh.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3955571/

Trans fatty acids (TFA) are produced either by hydrogenation of unsaturated oils or by biohydrogenation in the stomach of ruminant animals. Vanaspati ghee and margarine have high contents of TFA. A number of studies have shown an association of TFA consumption and increased risk of cardiovascular disease (CVD). This increased risk is because TFA increase the ratio of LDL cholesterol to HDL cholesterol. Food and Agriculture Organization of the United Nations and World Health Organization have come up with the recommendation that the contents of TFA in human dietary fat should be reduced to less than 4%. There is high prevalence of CVD in Pakistan. High consumption of vanaspati ghee which contains 14.2-34.3% of TFA could be one of the factors for this increased burden of CVD in Pakistan. Consumption of dietary fat low in TFA would be helpful in reducing the risk of CVD in South Asia. Denmark by banning the sale of food items with TFA has brought down the number of deaths due to coronary heart disease by nearly 50% over a period of 20 years. Public awareness about the adverse effects of TFA on human health would be extremely important. Media can play a very effective role in educating the masses and advocating the policy for the sale of only low TFA food items.

It merits legal banning.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3280054/

 A third pattern characterized by high trans fat was associated with less favorable cognitive function and less total cerebral brain volume.

Participants with higher plasma trans fat scores had worse cognitive function overall (memory, attention, language, processing speed, and global).

Each 1-SD increase in the trans fat score associated with a 0.30-SD decrease in global cognitive score.

You sure showed us!

They’re literally retarding themselves.

The high trans fat pattern was consistently associated with worse cognitive performance and less TCBV. Linolelaidic acid is predominantly found in bakery foods such as cookies, doughnuts, cakes, pastries, and pies.

I’d rather eat a donut than fake oozing flesh.

These foods are often prepared with hydrogenated vegetable oils to allow for a long shelf life. Higher trans fatty acid intake increases cardiovascular risk, systemic inflammation, and endothelial dysfunction, all of which may explain an association with cognition., Unfortunately, very few studies have assessed trans fat and risk for cognitive decline.

Funny that.

Big Food funding PACs?

Simple, scan vegan brains and see if they’re atrophied.

Trans fat may aggravate cognitive function independently and jointly through interaction with other dietary factors.e11 Trans fat may displace DHA in neuronal membranes, but apparently does not impact the neuropathologic Alzheimer hallmarks in mice.The consistency of the association of plasma trans fat with poorer cognitive function and more brain atrophy suggests neurologic consequences in humans, but these findings need to be confirmed.

Well?

5 Reasons Vegetable Oil is Worse Than Sugar

The European Food Safety Authority issued a report last year warning consumers about the high levels of potentially carcinogenic contaminants in palm oil (an ingredient used in Kit Kat Bars, Nutella, and Cheez-Its, among other treats) when refined at temperatures above 392°F.

AMERICA

https://www.ncbi.nlm.nih.gov/pubmed/16841858

With caveats inherent for ecologic, nutrient disappearance analyses, a healthy dietary allowance for n-3 LCFAs for current US diets was estimated at 3.5 g/d for a 2000-kcal diet. This allowance for n-3 LCFAs can likely be reduced to one-tenth of that amount by consuming fewer n-6 fats.

JUST STOP EATING PROCESSED CRAP.

It’ll cost more in hospital bills.

Fat soil makes you healthier

Nature is so weird.

https://www.sciencedaily.com/releases/2019/05/190529094003.htm

The discovery, published Monday in the journal Psychopharmacology, may at least partly explain how the bacterium, Mycobacterium vaccae, quells stress-related disorders. It also brings the researchers one step closer to developing a microbe-based “stress vaccine.”

Why get a vaccine when you can literally stick your hand in the ground?

British scientist David Strachan first proposed the controversial “hygiene hypothesis” in 1989, suggesting that in our modern, sterile world, lack of exposure to microorganisms in childhood was leading to impaired immune systems and higher rates of allergies and asthma. Researchers have since refined that theory, suggesting that it is not lack of exposure to disease-causing germs at play, but rather to “old friends” — beneficial microbes in soil and the environment that we have long lived alongside — and that mental health is also impacted.

Blood and soil. Foreign invaders haven’t adapted to our land. Useful if you know a pandemic is coming. Nationalism is biological, it’s about adaptation through generations to one ecosystem and even overall evolution e.g. nasal shape. Birthright is a term for it legally. Your success and potential to flourish depends on remaining in that land. I know eventually our boat people will leave and part of it will be a sense they don’t fit here, climate events or not, people move to where their genome evolved to, over a long timeline.

Of course, cutting off the electric would hasten the process.

Also why you eat local, at least a little. And keep away from most spices, they inhibit adaptation (absorption) to local valuable microbes (priceless in SHTF). At least don’t eat them at the same time. Those Middle Eastern spices came about because their local microbes are deadly.

I know I mentioned this finding before but still.

They also found that when cells were pre-treated with the lipid they were more resistant to inflammation when stimulated.

Fat isn’t the enemy. Lab fat is.

And if further studies show the novel fat alone has therapeutic effects, that molecule could become a target for drug development, he said.

It’s literally in dirt, fuck you I’ll buy a shovel.

Can we make shekels off this free natural product?

Overall, the study offers further proof that our “old friends” have a lot to offer.

I wonder if a study of immigrants to an area would show the same benefits. Probably reduced if any.

They didn’t co-evolve.

“This is just one strain of one species of one type of bacterium that is found in the soil but there are millions of other strains in soils,” Lowry said. “We are just beginning to see the tip of the iceberg in terms of identifying the mechanisms through which they have evolved to keep us healthy. It should inspire awe in all of us.”

So glad I was ordered to play in dirt as a child.

Iodine and IQ

aka most of the Thing behind the Flynn effect.

Vegans are deficient.

https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(13)60436-5/fulltext

We assessed the association between maternal iodine status and child IQ at age 8 years and reading ability at age 9 years. We included 21 socioeconomic, parental, and child factors as confounders.

Our results show the importance of adequate iodine status during early gestation and emphasise the risk that iodine deficiency can pose to the developing infant, even in a country classified as only mildly iodine deficient. Iodine deficiency in pregnant women in the UK should be treated as an important public health issue that needs attention.

After adjustment for confounders, children of women with an iodine-to-creatinine ratio of less than 150 μg/g were more likely to have scores in the lowest quartile for verbal IQ (odds ratio 1·58, 95% CI 1·09–2·30; p=0·02), reading accuracy (1·69, 1·15–2·49; p=0·007), and reading comprehension (1·54, 1·06–2·23; p=0·02) than were those of mothers with ratios of 150 μg/g or more.

How?

https://www.ncbi.nlm.nih.gov/pubmed/15554884

The present comments are restricted to the role of maternal thyroid hormone on early brain development, and are based mostly on information presently available for the human fetal brain. It emphasizes that maternal hypothyroxinemia – defined as thyroxine (T4) concentrations that are low for the stage of pregnancy – is potentially damaging for neurodevelopment of the fetus throughout pregnancy, but especially so before midgestation, as the mother is then the only source of T4 for the developing brain.

Despite a highly efficient uterine-placental ‘barrier’ to their transfer, very small amounts of T4 and triiodothyronine (T3) of maternal origin are present in the fetal compartment by 4 weeks after conception, with T4 increasing steadily thereafter. A major proportion of T4 in fetal fluids is not protein-bound: the ‘free’ T4 (FT4) available to fetal tissues is determined by the maternal serum T4, and reaches concentrations known to be of biological significance in adults. Despite very low T3 and ‘free’ T3 (FT3) in fetal fluids, the T3 generated locally from T4 in the cerebral cortex reaches adult concentrations by midgestation, and is partly bound to its nuclear receptor. Experimental results in the rat strongly support the conclusion that thyroid hormone is already required for normal corticogenesis very early in pregnancy. The first trimester surge of maternal FT4 is proposed as a biologically relevant event controlled by the conceptus to ensure its developing cerebral cortex is provided with the necessary amounts of substrate for the local generation of adequate amounts of T3 for binding to its nuclear receptor.

Don’t worry, the brain damage was genetic anyway.

Women unable to increase their production of T4 early in pregnancy would constitute a population at risk for neurological disabilities in their children. As mild-moderate iodine deficiency is still the most widespread cause of maternal hypothyroxinemia in Western societies, the birth of many children with learning disabilities may already be preventable by advising women to take iodine supplements as soon as pregnancy starts, or earlier if possible.

More!

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1741987/

This editorial reviews the impact of iodine deficiency (1) on thyroid function in pregnant women and neonates and (2) on the neurointellectual development of infants and children.
All degrees of iodine deficiency (mild: iodine intake of 50-99 µg/day, moderate: 20-49 µg/day, and severe: <20 µg/day) affect thyroid function of the mother and the neonate as well as the mental development of the child. The damage increases with the degree of the deficiency, with overt endemic cretinism as the severest consequence. Maternal hypothyroxinaemia during early pregnancy is a key factor in the development of the neurological damage in the cretin. Selenium deficiency combined with iodine deficiency partly prevents the neurological damage but precipitates severe hypothyroidism in cretins.
Iodine deficiency results in a global loss of 10-15 IQ points at a population level and constitutes the world’s greatest single cause of preventable brain damage and mental retardation.

An entire Standard Deviation, no big deal!

Supplements won’t help, you’ll OD.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3976240/

Iodine is a micronutrient that is essential for the production of thyroid hormones. The primary source of iodine is the diet via consumption of foods that have been fortified with iodine, including salt, dairy products and bread, or that are naturally abundant in the micronutrient, such as seafood. Recommended daily iodine intake is 150 μg in adults who are not pregnant or lactating. Ingestion of iodine or exposure above this threshold is generally well-tolerated. However, in certain susceptible individuals, including those with pre-existing thyroid disease, the elderly, fetuses and neonates, or patients with other risk factors, the risk of developing iodine-induced thyroid dysfunction might be increased. Hypothyroidism or hyperthyroidism as a result of supraphysiologic iodine exposure might be either subclinical or overt, and the source of the excess iodine might not be readily apparent.

The Great Iodine Debate

FYI

Iodine is critical to human health. It forms the basis of thyroid hormones and plays many other roles in human biochemistry. While the thyroid gland contains the body’s highest concentration of iodine, the salivary glands, brain, cerebrospinal fluid, gastric mucosea, breasts, ovaries and a part of the eye also concentrate iodine. In the brain, iodine is found in the choroid plexus, the area on the ventricles of the brain where cerebrospinal fluid (CSF) is produced, and in the substantia nigra, an area associated with Parkinson’s disease.

Iodine is essential to normal growth and development. Iodine deficiency in utero and during growth can result in cretinism, a condition of severely stunted physical and mental growth due to prolonged nutritional deficiency of iodine or from untreated congenital deficiency of thyroid hormones (hypothyroidism). The condition is characterized by short stature, delayed bone maturation and puberty, infertility, neurological impairment and cognitive impairment ranging from mild to severe. Iodine deficiency also causes goiter, the gradual enlargement of the thyroid gland. Both conditions have led to public health campaigns of iodine administration in many countries. The addition of iodine compounds to table salt or water represents the first attempt to provide nutrient supplementation via “fortification” of common foods.

At least they won’t have grandkids.

https://www.drbrownstein.com/iodine-the-essential-nutrient-for-every-cell/

https://articles.mercola.com/sites/articles/archive/2013/06/29/iodine-deficiency-risk.aspx

China paper here:

http://apjcn.nhri.org.tw/server/APJCN/14/1/32.pdf

also here https://www.ncbi.nlm.nih.gov/pubmed/15734706

IQ so unreal it tracks brain damage.

The level of iodine nutrition plays a crucial role in the intellectual development of children. The intelligence damage of children exposed to severe ID was profound, demonstrated by 12.45 IQ points loss and they recovered 8.7 IQ points with iodine supplementation or IS before and during pregnancy. Iodine supplementation before and during pregnancy to women living in severe ID areas could prevent their children from intelligence deficit. This effect becomes evident in children born 3.5 years after the iodine supplementation program was introduced.

And now… we wait.

Vegans stupid while pregnant again

B12 deficiency causes pre-term birth (higher risk of disease, death) and lower birth weight (yeah, like smoking).

https://academic.oup.com/aje/article/185/3/212/2918733

Really at this point it’s a controlled late abortion than a birth, isn’t it?

Globally, preterm birth and low birth weight (LBW) cause more than a third of the 2.9 million neonatal deaths each year, and prevention of these events is an important component of reducing the mortality rate among children younger than 5 years of age (1, 2). The causes of preterm birth, however, are complex, and few interventions have been successful in preventing it (3).

Really.

Eat some fucking cheese. So complex.

tfw Mother Nature hates you.

However, in a recent meta-analysis, Haider and Bhutta (10) concluded that multiple-micronutrient supplementation may reduce the risk of LBW and the number of stillbirths but not the risk of preterm birth or neonatal mortality. Thus, a more targeted micronutrient supplementation practice may be warranted.

Won’t work.

You can’t supplement stupidity.

If they’d rather the baby die than get over themselves, they don’t deserve kids.

I love the ineffectual response of “supplements won’t work, but our solution is more of them”.

Your intellectual betters, behold!

Importantly, B12 deficiency may be a proxy for inadequate nutritional status, and it is possible that some of our findings are related to nutritional status and not specifically to B12. A predominantly plant-based diet is low in B12 but also other nutrients, such as vitamin D and zinc, that to some degree may be associated with preterm birth (42–44). We did not have information on dietary intake or blood levels of these nutrients. Nutritional status could explain the present finding of an association between B12 and birth weight in low- and middle-income countries but not high-income countries. However, lower vitamin B12 levels were associated with higher risk of preterm birth irrespective of country income. It seems less likely that nutritional status can fully explain this finding.

Vegetarianism: the Third World baby death diet.

Indian women generally have lower dietary intakes of B12 because of their mainly vegetarian diet, making them susceptible to B12 deficiency (46). Additionally, Indian newborns are among the smallest in the world (45). Our findings suggest that pregnancies already at the greatest risk of resulting in small newborns were the ones that were most vulnerable to low levels of B12. The association between B12 and the risk of preterm birth was consistent across studies in both high-income and low- and middle-income countries, and generalization to countries not studied may be feasible.

Stop eating vegan junk food.

In line with our findings, maternal obesity has been associated with B12 deficiency in several populations (47, 48). It has been hypothesized that this association is due to altered fat distribution and metabolism in overweight women compared with normal-weight women (47). Maternal weight is positively correlated with newborn weight (49), and failure to adjust for maternal weight may underestimate a positive association between B12 and birth weight.

Clearly Mother Nature is fatphobic.

paper 43
Zinc supplementation for improving pregnancy and infant outcome
https://www.ncbi.nlm.nih.gov/pubmed/25927101
44
Vitamin D supplementation for women during pregnancy
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3747784/
https://www.cochrane.org/CD008873/PREG_vitamin-d-supplementation-women-during-pregnancy

Put down the leafy greens.
“However, it appears that when vitamin D and calcium are combined, the risk of preterm birth is increased.”