BMI in women, hormones and a little on race and Asian pregnancy

follow-up to

TLDR: Asians have higher BMI and body fat than White women. It’s a racial difference.

Logically, wouldn’t they have messed-up hormones and diabetes, then? Let’s see!

5,000 words -ish.

High BMI, high Testosterone

“cardiometabolic clinical correlates related to total testosterone (TT), free testosterone (fT), androstenedione (ASD), dehydroepiandrosterone-sulfate (DHEAS), estrone (E1), estradiol (E2), and sex hormone-binding globulin (SHBG).

Results: Waist circumference and BMI (β-coefficient: -0.03; 95% CI: -0.04; 0.03) were inversely related to SHBG, and BMI was positively related to TT (β-coefficient: 0.005; 95% CI: 0.001; 0.009), fT, E1, and E2. Smoking was positively related to TT (β-coefficient: 0.04; 95% CI: 0.01; 0.06), ASD, and fT. Systolic blood pressure (TT: β-coefficient: 0.002; 95% CI: 0.001; 0.003), hypertension (TT: β-coefficient: 0.05; 95% CI: 0.003; 0.11), low-density lipoprotein (LDL) cholesterol (TT: β-coefficient: 0.02; 95% CI: 0.01; 0.05), and total cholesterol (TT: β-coefficient: -0.03; 95% CI: 0.01; 0.05) were positively related to TT and ASD. Finally, type 2 diabetes mellitus (T2DM), and metabolic syndrome (MetS) were positively related to fT, but inversely related to SHBG.

Conclusions: Our population-based study, with sex hormone concentrations measured by liquid chromatography tandem mass spectrometry, revealed associations between clinical correlates including waist circumference, smoking, cohabitation, systolic blood pressure, cholesterol, and MetS with sex hormones. Thus, sex hormones and SHBG may play a role in the cardiovascular risk profile of women.”

I’ve posted about WHR before.

Both obesity and anxiety symptomatology were separately associated with the same sex hormone alteration in premenopausal women: higher total testosterone level (0.97 ± 0.50 in obese vs. 0.86 ± 0.49 nmol/L in normal-weight women, p = 0.026 and 1.04 ± 0.59 in women with vs. 0.88 ± 0.49 nmol/L in women without anxiety symptomatology, p = 0.023). However, women with anxiety symptomatology had non-significantly higher estradiol levels than women without anxiety symptomatology (548.0 ± 507.6 vs. 426.2 ± 474.0 pmol/L), whereas obesity was associated with lower estradiol levels compared with those in normal-weight group (332.7 ± 386.5 vs. 470.8 ± 616.0 pmol/L). Women with anxiety symptomatology had also significantly higher testosterone and estradiol composition (p = 0.006). No associations of sex hormone levels and BMI with anxiety symptomatology in postmenopausal women were found.

Conclusions: Although both obesity and anxiety symptomatology were separately associated with higher testosterone level, there was an opposite impact of anxiety and obesity on estradiol levels in premenopausal women. We did not find an evidence that the sex hormone alterations related to obesity are playing a significant role in anxiety symptomatology in premenopausal women. This could be the explanation why we did not find an association between obesity and anxiety. In postmenopausal women, other mechanisms seem to work than in the premenopausal group.

Regional fat distribution (RFD) has been associated with metabolic derangements in populations with obesity. For example, upper body fat patterning is associated with higher levels of free testosterone (FT) and lower levels of sex-hormone binding globulin (SHBG). We sought to determine the extent to which this relationship was true in a healthy (i.e., non-obese) female population and whether RFD influenced androgen responses to resistance exercise. This study examined the effects of RFD on total testosterone (TT), FT, and SHBG responses to an acute resistance exercise test (ARET) among 47 women (22+/-3 years; 165+/-6 cm; 62+/-8 kg; 25+/-5%BF; 23+/-3 BMI). RFD was characterized by 3 separate indices: waist-to-hip ratio (WHR), ratio of upper arm fat to mid-thigh fat assessed with magnetic resonance imaging (MRI ratio), and ratio of subscapular to triceps ratio (SB/TRi ratio). Skinfolds were measured for the triceps, chest, subscapular, mid-axillary, suprailaic, abdomen, and thigh regions. The ARET consisted of 6 sets of 10 RM squats separated by 2-min rest periods. Blood was obtained pre- and post- ARET. TT, FT, and SHBG concentrations were determined by radioimmunoassay. Subjects were divided into tertiles from the indices of RFD, and statistical analyses were performed by an ANOVA with repeated measures (RFD and exercise as main effects). Significant (p < or = .05) increases following the AHRET were observed for TT (approximately 25%), FT (approximately 25%), and SHBG (4%). With multiple regression analysis, anthropometric measures significantly predicted pre- concentrations of FT, post-concentrations of TT, and pre-concentrations of SHBG. The SB/TRi and MRI ratios but not the WHR, were discriminant for hormonal concentrations among the tertiles. In young, healthy women, resistance exercise can induce transient increases in testosterone, and anthropometric markers of adiposity correlate with testosterone concentrations.

So exercise will boost a woman’s natural T. If they already have high T….

If their BMI is higher for their size, they already have high T comparatively. If they already have it racially… probably not good.

Results: Breast cancer risk increased with increasing BMI (Ptrend = .002),

Not healthy.

Rare study looking at race directly. White v Black.


Compared to the decline in E2 concentrations, androgen concentrations declined minimally over the MT. T (β 9.180, p < 0.0001) and E1 (β 11.365, p < 0.0001) were higher in Whites than in AAs, while elevations in DHEAS (β 28.80, p = 0.061) and A4 (β 0.2556, p = 0.052) were borderline. Log-transformed E2 was similar between Whites and AAs (β 0.0764, p = 0.272). Body mass index (BMI) was not significantly associated with concentrations of androgens or E1 over time.

so black and white is off the hook


This report suggests that the declines in E2 during the 4 years before and after the FMP are accompanied by minimal changes in DHEAS, A4, T, and E1. There are modest differences between Whites and AAs and minimal differences by BMI.

Cancer and Testosterone link


During a median follow up of 6.3 years, 45 patients relapsed. Testosterone levels significantly increased across BMI categories (p = 0.001). Both circulating testosterone and BMI were positively associated with disease free survival (p = 0.005 and p = 0.021, respectively). A significant interaction was found between testosterone and BMI (p = 0.006). For normal-weight women, testosterone concentration around median (0.403 ng/mL) or third quartile (0.532 ng/mL) showed a high significant HR of relapse (5.52; 95% CI:1.65–18.49 and 4.55; 95% CI:1.09–18.98, respectively). Overweight patients showed increased HR at increasing testosterone levels, reaching a significant high HR (4.68; 95% CI:1.39–15.70) for testosterone values of 0.782 ng/mL (95th percentile). For obese patients HR decreased (not significantly) at increased testosterone concentrations, explaining the interaction between testosterone levels and BMI categories.


In ER-positive postmenopausal breast cancer patients, high testosterone levels are associated with worse prognosis in normal-weight and overweight women, whereas in obese seems to be associated with a better outcome. Although the results require further validation, they suggest that assessment of circulating testosterone and BMI could help to identify postmenopausal ER-positive patients at higher risk of relapse and potentially open new therapeutic strategies.

High T isn’t good, even in normal weight women. Water is wet.

“The findings of this study suggest high plasma levels of testosterone could play a role in the pathogenesis of type 2 diabetes among women,” Jon Jarløv Rasmussen, MD, PhD, a specialist registrar and postdoctoral researcher in the department of endocrinology at Rigshospitalet in Copenhagen, Denmark, told Healio. “The incidence of type 2 diabetes was rather low in the study, but the results implicate that screening for type 2 diabetes among women with higher plasma levels of testosterone may be beneficial, even among women who are young and without established comorbidities, such as polycystic ovary syndrome.”

In a retrospective study, Rasmussen and colleagues analyzed data from 8,876 healthy women (mean age, 38.5 years) who provided blood samples to measure plasma testosterone, dehydroepiandrosterone-sulfate (DHEAS), dihydrotestosterone (DHT) and sex hormone-binding globulin (SHBG) between January 2007 and December 2015. Researchers analyzed androgens using tandem liquid-chromatography mass spectrometry. Researchers used Poisson regression models to calculate incidence rate ratios for developing type 2 diabetes during a median follow-up of 8.1 years, stratified by androgen quartiles.

‘Normal weight’ women can get Type 2. Since Asians have higher T from higher BMI (against the white norm), they’ll be more likely to get it. This also explains the gestational diabetes common in Asian women, especially if the baby is mixed.

Nationwide, as many as 1 in 4 people who have diabetes don’t know they have it. But for Asian Americans, that number is much higher—1 in 2, the highest of all ethnic/racial groups. Why aren’t more getting diagnosed?

Weebs do not mention this. If your apparent rationale for avoiding fat white women is avoiding the Diabeetus genes, Asian is then categorically the worst racial group to mix with.

1 in 2, flip a coin, rice cooker.

I bet it’s higher in the women due to sweet tooth, so likely worse.

But people of Asian descent have less muscle and more fat than other groups and often develop diabetes at a younger age and lower weight. That extra body fat tends to be in the belly (visceral fat). This isn’t the “inch you can pinch,” the fat stored just under the skin. Visceral fat is out of sight, wrapped around organs deep in the body. You can’t tell how much visceral fat someone has by looking at them.

I didn’t call them skinny-fat to be mean, they really are!

Visceral fat is also sometimes known as “active” fat because it drives certain processes in the body that can increase the risk for heart disease, stroke, and other serious health conditions. Everybody has some visceral fat, but having too much is a major risk factor for developing type 2 diabetes.

….But BMI doesn’t catch Asian Americans in the normal weight range (18.5 to 24.9) who may very well have too much visceral fat and be at risk for type 2 diabetes. Researchers are now suggesting that people of Asian heritage get tested if their BMI is 23 or greater. Type 2 diabetes can be prevented or delayed, but only if people know they’re at risk and can take action!

They need a totally different (lower) testing standard, but they’re just like us, guys! Nay, SUPERIOR!

The same volume food in a smaller body, this isn’t hard to figure out. They’re not white women, eating like us makes them FAT.

re preggers diabetus-

Pregnant South Asian women carry a higher risk for developing gestational diabetes, a condition that’s dangerous for both mother and child. Between 2 and 10 percent of all pregnancies each year are complicated by gestational diabetes

2-10% in which demographic? Sounds like all? I bet it’s higher in certain ones, isn’t it?

Under risk factors is basically – be non-white

  • Being of Hispanic, Native American, African-American, Asian-American or Pacific Islander descent.

Women who have had gestational diabetes have a 20 to 50 percent chance of developing diabetes in the 5 to 10 years following pregnancy.

Mother Nature is a bitch.

Specific study on Asian women-

so healthy, much fitness

Our data indicate that although the historical or clinical risk factors for GDM are valid in Asians, using risk factors alone to select such patients for testing for GDM is inadequate. Many Asian women who develop GDM have no risk factors at all.

When Natural Selection hates you so much… maybe give it up?

r-types have higher numbers of issues like this, that would be fatal under natural law

They don’t ‘choose’ to stop at 1-2 kids, it isn’t ‘culture’, it’s fear (see below).

To avoid overlooking significant numbers of women with GDM, one may lower the specificity of the criteria, but this requires that the majority of patients be tested.

wow, that bad

Logistically, it is much simpler to conduct universal screening for all Asian women in Western countries, rather than to apply selective testing in order to spare a small percentage of women from being tested. Therefore, our findings strongly support recommendations for universal screening for GDM in pregnant women of Asian origin in Western countries. However, in places where the incidence of GDM is low, such as in some developing countries, the selection of patients for testing by the risk factors may be reasonable.

just like us, huh?

has a bloody paywall, nevermind, still linking

Introduction: Asian women have a higher prevalence of gestational diabetes mellitus than women of other races/ethnicities. We aimed to compare the prevalence of gestational diabetes among Asian American women to other racial/ethnic groups and explore whether the higher occurrence of the disorder among Asian women can be explained by acculturation.

Clearly I am making this all up to feel better, right guys?

Why hide this one behind a paywall, hmm?

It is also here.


Among the 5,562 women studied, the weighted prevalence of gestational diabetes was 15.5% among Asian American women, followed by 9.0% among non-Hispanic black women, 10.7% among Hispanic women, and 7.9% among non-Hispanic white women.

15.5% v. 7.9%

Diabetes at DOUBLE the rate of whites!


but they’re just like us

2.44x the risk

and that’s controlled, independently

Compared with non-Hispanic white women, Asian women had 2.44 (95% confidence interval [CI], 1.81–3.29; P < .001) times the odds of having gestational diabetes, independent of maternal age, education, marital status, income, prenatal care adequacy, prepregnancy BMI, and physical activity. Acculturation was negatively associated with having gestational diabetes (odds ratio [OR] = 0.93; 95% CI, 0.86–0.99) and explained 15.9% (95% CI, 11.38%–25.08%; P < .001) of the association between Asian race and the condition.

About 85% genetic. Great odds.


We found that Asian race was an independent risk factor for gestational diabetes, and higher acculturation may play a protective role against it in Asian American women.

In Summary

What is already known about this topic?

Asian women have a higher prevalence of gestational diabetes mellitus than women of other races. However, little data exist on why prevalence is highest among Asian women.

I sense genetics.

If they’re having unnatural babies (too large for their race, mixed) supported by modern medicine, they’d be more likely to die anyway, right? Medicine can only do so much. Weaker genes die a la Darwin.

The biggest r-select factor would be risk of death while breeding, that would be the surest thing. The genes trying to extinct themselves.

Does this data exist? Also for the neonates?


Let’s see the weebs explain away these studies. They’ll probably just ignore me… again.

Go ahead. Ignore your baby and waifu’s graves?

Pregnancy related mortality can be defined as death of the mother during pregnancy, delivery, or within one year postpartum. While 700 pregnancy-related deaths occur each year, 2/3 of these deaths are considered to be preventable.

Modern medicine, dysgenic again.

Overall pregnancy related mortality in the United States occurs at an average rate of 17.2 deaths per 100,000 live births. However, that number jumps to 43.5/100,000 for non-Hispanic Black women and decreases to 12.7/100,000 for non-Hispanic white women and 11/100,000 for Hispanic women.

No data listed for Asian, odd?

For mothers of all backgrounds, leading causes of death include cardiovascular conditions, hemorrhage, and infection. However, for non-Hispanic Black women, leading causes of death include cardiovascular conditions in addition to cardiomyopathy, pre-eclampsia, and eclampsia (hypertensive disorders).

Non-Hispanic Black women are also significantly more likely to have a severe maternal morbidity (SMM) event at the time of delivery. For every maternal death there are 70 cases of SMM events that are considered “near misses.” These events can have long-term or short-term consequences to a woman’s health. Over the past 20 years, cases of SMM have increased by over 200%, while cases disproportionately affect Black women. One study found Black women experienced SMM at a rate 2.1 times greater than that of white women.

To better understand and address these disparities, researchers suggest providers increase screening for social determinants of health. Levels of stress, trauma, food insecurity, neighborhood violence, and access to prenatal care are all factors that may contribute to the disparities and warrant further investigation.

Although most maternal deaths result from cardiovascular and hypertensive disorders, researchers found Asian/Pacific Island women exhibit the highest prevalence of gestational diabetes, which can increase pregnancy complications, at 14.8%.

One study presented in the session focused on behavioral interventions and protective factors among women with gestational diabetes. A Kaiser Permanente analysis of women in northern California found Black women have a lower prevalence of gestational diabetes when compared with Asian Indian, Filipina, Southeast Asian and Chinese women. White women had the lowest rates of the disease overall.

Screening for postpartum diabetes is recommended to all women within 4 to 12 weeks postpartum. However, rates of screening vary among women with different racial and ethnic backgrounds, suggesting tailored strategies to reduce risk and improve healthcare behaviors may be effective.

Racial medicine, openly.

An additional study explored how racial and ethnic disparities impact severe neonatal morbidities, specifically among very preterm children (born <32 weeks of gestation). Preterm birth has been associated with several health conditions developing later in life, including diabetes.

Presenter Teresa Janevic, PhD, defined race as “linked to phenotype and /or ancestry that indexes one’s location on the US social hierarchy of socially constructed groupings (i.e., races) that has been based primarily on skin color.”

genes aren’t social

Africans in Africa also have the same ‘risk’ as one in America. No magic dirt.

In contrast, Janevic defined ethnicity as “tied to race and used both to distinguish diverse populations and to establish personal or group identity, usually based on shared culture or beliefs.”

Culture? Belief? Believe your way out of diabetes. I’ll wait.

In a population-based retrospective cohort analysis using hospital discharge data linked with vital statistics at birth and death records, researchers determined Black infants were at the highest risk of dying within less than 28 days after discharge, or suffering neonatal morbidities in the time between birth and discharge. Black infants were followed by Hispanic infants, while white and Asian infants had similar low risks.

We’ll see about that.

Of the 39 New York City hospitals included in the study, researchers found a 6-fold difference in risk of combined mortality and morbidity outcomes. “Black infants were at twice the risk of being at a hospital that has risk-adjusted high rates of combined mortality and morbidity,” Janevic noted, while Hispanic infants had a 1.5 increased risk to receive care from one of these hospitals. “Hospital quality where women of color deliver likely contributes to these disparities,” she concluded.

Like schools, it depends on the IQ of the people working there.

Another investigation detailed how environmental factors and population level exposures impact disparities in preterm birth and infant mortality. “Non-Hispanic Black infants compared with non-Hispanic white infants have twice the risk of death in the first year,” explained presenter Heather Burris, MD. “This is particularly striking because Black infants just make up 15% of all births in the United States but are counting for 29% of all deaths.”

no comment

Among causes of infant death, preterm birth and low birth weight related death, along with pregnancy complications, account for the highest racial and ethnic disparities between non-Hispanic Black and white infants. Black infants are also significantly more likely to be born preterm than white infants.

an r-factor unless twins

Researchers note genetics and education level have very little impact in accounting for disparities in preterm birth. Although women with higher education tend to have lower preterm birth rates, Black women who graduated from college have a higher risk of preterm birth than white women who dropped out of high school.

so, racial

I’m so glad white people already survived multiple genetic purges in our history.

Through analyzing delivery data and creating models based on air pollution severity in Philadelphia, Pennsylvania, investigators discovered air pollution is associated with spontaneous preterm birth. Data also show Black Americans experience consistently higher exposure to air pollutants, measured in fine particulate matter (PM)2.5.

An additional analysis between preterm birth and nationwide neighborhood deprivation index (encompassing income below the poverty level, vacant homes, education levels, among other factors) found that Black women experience neighborhood deprivation exposure at almost 2 standard deviations (SDs) higher than white women in Philadelphia.

Overall, Black women are 4 times more likely to live in a neighborhood with high violent crime and high air pollution than white women. “When we look at preterm birthweights, we can see that it is women living in these high-high neighborhoods that have the highest risk of preterm birth,” Burris said. However, these associations were consistent regardless of race.

so non-sig

They gestate for less time than whites, this is known. Africans in Africa do it.

Now we’ve established some things. An r-study in Asian women.

Increased Perinatal Morbidity and Mortality Among Asian American and Pacific Islander Women in the United States

Background: Asian American/Pacific Islanders (AAPIs) are the fastest-growing racial group in the United States.

America is now owned by Asia, demographically.

Despite a higher socioeconomic status, AAPI women experience higher rates of maternal morbidity and mortality.

can’t pay your way out of r-genes

if controlled for SES, aka $, their data would be even worse

Methods: Using the National Inpatient Sample, we performed a retrospective cohort analysis of women who were hospitalized for delivery from 2002 to 2013. The primary outcome variable was inpatient mortality rate, and the presence of severe maternal morbidities was estimated using the Bateman Comorbidity Index, a validated tool for predicting obstetric morbidity.

Results: AAPI women presenting for delivery between 2003 and 2012 were older, more likely to reside in a zip code in the top quartile of annual income, be privately insured than Caucasian women,

so oppressed

where’s Asian privilege?

and less likely to have a higher Bateman Comorbidity Index. However, AAPI women had a higher likelihood of postpartum hemorrhage (3.4% vs 2.7%, P < .001), uterine atony, severe perineal lacerations, and severe maternal morbidities. Procedures such as transfusion, hysterectomy,

So they could have one kid and die, have one kid and have that die, OR have one kid and then their organs all removed – so no more kids?

Yes clearly our biological superiors, right weebs? Totally not rationalising a fetish, are we?

I wonder why one child was law? They don’t have a culture of many kids because they’re too r-select to survive without modern medicine. Wake up. They pretend 1-2 is a choice and that’s why they mock and envy large white families (3+ standard) like the Amish. They envy us that ability. They would die.

and mechanical ventilation were also more common in AAPI women.

Calling it – Mother Nature is anti-Asian.

Furthermore, AAPI women had a higher mortality rate that persisted despite adjustment for an apparently higher income and comorbidities (odds ratio 1.72, 95% confidence interval: 1.14-2.59, P = .01).

Conclusions: Despite having a higher socioeconomic status, AAPI women had higher rates of maternal mortality during hospitalization for delivery. This increase persisted even after adjustment for factors known to affect peripartum outcomes. Further investigation is needed to better clarify the causes of racial differences in maternal morbidity and mortality.


Science says –

If you want to survive childbirth – be white.

Almost like we evolved to during the Ice Age.


Results: A total of 360,370 women with postpartum hemorrhage from 2012 to 2014 were included in this analysis. Risk for severe morbidity was significantly higher among non-Hispanic black women (26.6%) than non-Hispanic white, Hispanic, or Asian or Pacific Islander women (20.7%, 22.5%, and 21.4%, respectively, P < .01).

The white is 20%, Asian is 21%.

And these are the fattest white people, like, ever.

White and Asian bolded-

For non-Hispanic black compared with non-Hispanic white, Hispanic, and Asian or Pacific Islander women risk was higher for disseminated intravascular coagulation (8.4% vs 7.1%, 6.8%, and 6.8%, respectively, P < .01) and transfusion (19.4% vs 13.9%, 16.1%, and 15.8%, respectively, P < .01). Black women were also more likely than non-Hispanic white women to undergo hysterectomy (2.4% vs 1.9%, P < .01), although Asian or Pacific Islander women were at highest risk (2.9%). Adjusting for comorbidity, black women remained at higher risk for severe morbidity (P < .01). Risk for death for non-Hispanic black women was significantly higher than for nonblack women (121.8 per 100,000 deliveries, 95% confidence interval, 94.7-156.8 vs 24.1 per 100,000 deliveries, 95% confidence interval, 19.2-30.2, respectively, P < .01).

The weebs either did 1. no research (typical gammas) or 2. they’re delusional.

Almost double the risk of hysterectomy, roughly. An additional 52% risk over white women, minimum, in just this study.

What’s the point of being married to them, at that point? Their baby machine is broken.

Infant Mortality and Asian Americans

Yes, we’re going there.

  • Asian American infants are 40 percent more likely to die from maternal complications as compared to non-Hispanic white mothers.

They have boy hips, duh.

For such a small segment of the population, their numbers shouldn’t be so high.

It doesn’t discuss mixed kids, I wonder why?

  • Non-Hispanic black (black) and non-Hispanic American Indian/Alaska Native (AI/AN) women experienced higher PRMRs (40.8 and 29.7, respectively) than all other racial/ethnic populations (white PRMR was 12.7, Asian/ Pacific Islander PRMR was 13.5 and Hispanic PRMR was 11.5).  This was 3.2 and 2.3 times higher than the PRMR for white women – and the gap widened among older age groups.

Racial medicine.

Notably, we found that, when aggregated, the top cause of death among Asian Americans is cancer. However, when disaggregated, there is wide variation in the leading cause of death. For instance, for Asian Indians, nearly twice as many men die of heart disease (31 percent), compared to cancer (18 percent). In contrast, for Koreans, the opposite is true — the death rate for cancer (34 percent) is much higher than the death rate for heart disease (19 percent).

Remember the breast cancer and Asian BMI/testosterone stuff?

Research led by the University of Birmingham has found that increased levels of hormones including testosterone could cause a brain condition that can lead to blindness in women.

We are all jealous of your waifu, yes.

Idiopathic Intracranial Hypertension—also known as IIH—is caused by  in the brain with consequences from blindness to incapacitating daily long-term headaches. IIH was originally identified over 100 years ago yet the cause of the condition has remained unknown although there has been much speculation about why more than 95 per cent of total incidence is in  with obesity.

And Asians, they’re 1/2 obese in America!

Lucky you.

 They then compared the results with the levels observed in women with obesity of the same age and body mass index (BMI), as well as a cohort of women with  (PCOS).

PCOS is far more common in Asians. Look it up.

Most notable were the high levels of the androgen ‘testosterone’ found in the blood in IIH women. Crucially, levels of androgens were uniquely increased in the brain fluid (CSF) of women with IIH. When the researchers, analysed human choroidal plexus tissue, which is the site in the brain where CSF is produced, they confirmed that androgens could increase the rate of CSF secretion, a potential driver for increased brain pressure.

Brain damage. How sexy.


On the other hand, South Asian women with PCOS, the region comprising the Indian subcontinent and surrounding countries, are known to have a higher degree of hirsutism…


Asian women with PCOS were more likely to have diabetes compared with Caucasian patients, suggesting they also have metabolic complications. 

Genetic components play important roles in the pathogenesis of PCOS,

Do their dodgy hormones make South Asians partially infertile?

Yes. Yes they do.

Results: We found that the South Asian women presented at a younger age for the management of sub-fertility. An extended stimulation phase and Caucasian ethnicity showed an inverse correlation with the number of oocytes retrieved in the PCOS subgroup. Caucasian ethnicity was associated with a higher fertilization rate however increase in body mass index (BMI) and the laboratory technique of IVF appeared to have a negative impact on fertilization rates in the PCOS subgroup. Commencing down regulation on day 1 of the cycles was negatively associated with fertilization rates in the tubal group. In terms of clinical pregnancy rates, the Caucasian PCOS had a 2.5 times (95% CI: 1.25-5) higher chance of an ongoing clinical pregnancy as compared with their Asian counterpart. Also, a unit increase in the basal FSH concentration reduced the odds of pregnancy by 18.6% (95% CI: 1.8-32.6%) in the PCOS group.

Conclusions: The Asian PCOS have a greater sensitivity to gonadotropin stimulation with lower fertilization and ongoing clinical pregnancy rates as compared with their Caucasian counterparts.

White women win again.

testosterone levels were higher in PCOS cases than in controls (P = 0.008 and 0.003, respectively).

But IVF, right? WRONG

The ethnicity of women undergoing fertility treatments like IVF can affect the rate of successful live births, according to new research. After adjusting for certain factors including age of patient at time of treatment, cause of female or male infertility, and type of treatment, the study found that White Irish, South Asian Indian, South Asian Bangladeshi, South Asian Pakistani, Black African, and Other Asian women had a significantly lower odds of a live birth than White British women.

White women, still winning. Thank God for the Ice Age.…-a0509015581

Overall, studies have shown higher testosterone levels in women and lower levels in men are related to incident diabetes. The major risk factors contributing to diabetes are biochemical, environmental, sedentary lifestyle, socioeconomic status and genetic factors. All of them together or independently are responsible for the development of the DM. [3] Besides, certain studies show Impaired Glucose Tolerance (IGT) is more common in females than males independent of age. [4]

We found a high prevalence of GDM among the Asian population. Asian women with common risk factors especially among those with history of previous GDM, congenital anomalies or macrosomia should receive additional attention from physician as high-risk cases for GDM in pregnancy.

 In the post-menopausal group, estrogens, testosterone and androstenedione increased with increasing BMI.

Let’s look at objective hotness!

Body mass index (BMI) was a very strong negative predictor of body attractiveness ratings, similar to previous findings. Zero-order associations between women’s mean hormone concentrations and mean attractiveness ratings were not significant; however, after controlling for BMI, attractiveness ratings were independently and positively associated with both estradiol and testosterone concentrations. Discussion focuses on the implications of these findings for whether attractiveness assessment mechanisms are specialized for the detection of cues of differential fecundity in young women’s bodies.

High T = ugly!

Did I mention water is wet? Can they seriously accuse of cherry picking? I’m not even looking hard.

Prior research has provided evidence that large breast size and low waist-to-hip ratio (WHR) are positively associated with women’s estrogen and progesterone concentrations,

Previously covered WHR, use search bar. Asians lose. Even black women do better.

Click to access ethnic-testosterone1.pdf

Asians have way more T as a race than Europeans, get over it. Historically, we considered them savages, less civilized, for that reason. How is this surprising? Do you think we colonised India for fun? It’s obvious in the broad manjaws, duh. Marquardt covered this. Anyone can do a replication study, but I suggest you include the women too, so it isn’t just a sexual effect but race.

From a blog “East Asians were found to have the highest average total plasma testosterone (5,673 ρg/mL) followed by Africans (5,442 ρg/mL) and then Europeans (4,992 ρg/mL). Given that the sample size for Africans is smaller (N < 10,000), their relative position may change with more data. Nonetheless, the claim that East Asians have the least testosterone is not supported by scientific data. “

Yeah, fake redpills who think T = manly, good thing. It’s just a hormone.

“There is no way of accurately determining free testosterone. Even if there was, this would also be irrelevant since bio-availability is prime. Since race realists use total serum testosterone, why is this an issue?”

true, it’s just applying the same standard

Culturally, gang rape is more normal in Asia than Africa. This is why. You don’t get African Taharrush, really. Asia has Eve Teasing and the like. Trust me, you don’t want this.

“Mass sexual assault is the collective sexual assault of women, and sometimes children, in public by groups of unrelated men. Typically acting under the protective cover of large gatherings, victims have reported being groped, stripped, beaten, bitten, penetrated and raped.”

As for the contention that there are no studies indicating a 10% difference between East Asians and Europeans, I did find one age controlled study where the Chinese sample had 8.8% more total T, 11.4% more bio-available T and 12% more free T than the European sample. The Japanese sample had 10.5%, 5.1% and 6.7% more than Europeans respectively [Wu et al. 1995]. Wonder if race realists discuss this study, or perhaps they are too busy in celebratory dance around the Korean/Swede campfire?

They’re not really redpill. I believe data even if I dislike it. Asians have high T as a race. Get over it.

High T can also dovetail with lower national IQs e.g. India, so…. why want this? Low IQ nations have more crime.

Additionally, this recent study shows HK Chinese having some 3% more bio-available T than US Europeans.

Lol, he’s right. But T isn’t a good thing. It’s just a hormone, in men or women.

Being a race realists seems to be a length engagement with delusion, fantasy and ‘scientific’ homo-erotica.

Not here, son. I believe the T-data. Penis size generally correlates to racial height (in white men), not really T. Forum bros are wrong again. Penis stuff is sexual selection, aka chosen by women.

There was a similar increase in the positive effect of penis size on attractiveness with a more masculine body shape (i.e., greater shoulder-to-hip ratio). Surprisingly, larger penis size and greater height had almost equivalent positive effects on male attractiveness. Our results support the hypothesis that female mate choice could have driven the evolution of larger penises in humans. More broadly, our results show that precopulatory sexual selection can play a role in the evolution of genital traits.

It’s even subracial (national male height).

I appreciate the skin tone joke in this graphic.

But if you go by nation, and percentage of height....

So according to this, hate the French!

Surprised Croatians rank so low.

It was concluded that all penile measurements are interrelated to each other; the height and weight also the other body measurements that are related to the penile measurements in less than 50%. It seems that the penile measurements are polygenic traits and are under multifactorial influences.

Not T.

There are racial differences in associations of hormone levels with age and BMI in late
reproductive age women. Further study is needed to replicate these findings and to determine the relationships of these hormonal associations with menopausal symptoms

Obesity is an important factor in hormone dynamics independent of age, race and smoking in mid-life women, although the mechanisms remain unclear.

From “A Study of the Correlation of Some Sex Hormone with Obesity in Women with Secondary Infertility” (google it)

Infertility is the inability to conceive a child for more than one year. The present study indicates
that the obesity associated with infertility. The aim of the study to determine follicle stimulating
hormone, luteinizing hormone, testosterone hormone and prolactin levels. and cholesterol and
triglyceride concentration in 2nd inferetid women. This study was carried out at kamal al-samaarai
hospital the data were collected from 95 secondary infertilial women were age between 16-45 years
old and grouped them in to obese (n = 46) and non obese(n = 49). There was no significant
difference between the two groups (p <0.05). Body mass index in Infertile obese women is slightly
higher than non obese Infertile women which is statistically significant (P<0.001). However LH,
TSH, cholesterol and triglyceride concentration in obese infertile women is significantly higher than
non obese infertile women (p >0.05).The BMI was correlated positively with triglyceride in obese
group while BMI was positive correlation highly significant with cholesterol in non obese group.
Regression analysis revealed obese to be strongly associated with observed infertility. The elevated
prolactin values in secondary infertile women clearly shows that there is a mechanism operating at
the anterior pituitary level which shows an abnormal distribution of FSH and LH which may further
explain the abnormal delay ovum maturation. This study also indicates obese associated with
infertile more than non obese women.

So, there’s a lot going on.

Blood type is racially determined, like fertility problems

There are four main blood types: O, A, B, and AB and two Th factors, positive or negative. Most people are either A positive or O positive and the fewest are AB negative. Because blood types are genetic, they are inherited from the parents,  blood types have different racial and ethnic differences. The majority of people in the world and across various ethnicities have Rh+ blood type. Subsaharan African populations have a 97-99% Rh+ factor. East Asian communities have 93-97% Rh+ bloodRh factor is a big determinant in both fertility and pregnancy. If you’re Rh-negative, you will need to take certain precautions during your pregnancy because an Rh positive fetus can conceivably be affected

wait so does Nature abort hybrid babies?

if the RH negative mother has previously been exposed to Rh positve blood and creates antibodies that cross the placenta and attack the fetus’ RH positive blood.

It isn’t our fault Asians need IVF more and more often fail at it, it’s literally in their blood. What are they implying otherwise, a curse?

Asian Women & Fertility Problems

Race is biological, see.

Unfortunately, many Asian couples face challenge trying to conceive naturally or using fertility treatment. The decline in natural fertility and the lower success of IUI and IVF in Asian women is documented in The US, UK, China, Japan, Korea and other Asian countries.

Fertility in Asian countries has declined to the population replacement rate 2.1 or lower. Many factors contribute to decline in natural fertility in Asian women;

Not our fault. Not our problem. Nature tends to curb the over-population of r-types by introducing more threats to thin the herd.

It happens to Asians who never lived in the West, stop blaming whites.

Repost on the Asian female infertility problem:

When compared to Caucasian women, Asian women undergoing IVF significantly produce less eggs at all Anti-Mullerian hormone (AMH) levels, even in women with high AMH. AMH is the most accurate marker for ovarian reserve.

Gynecologic and medical disorders that impairs fertility: PCOS, endometriosis and Systemic lupus (SLE) are more common in Asian women.

Vaginismus : may interfere with regular intercourse in some Asian women.

Environmental Factors: Asian women has more exposure to methyl Mercury and vitamin D deficiency.

Culture : surveys of Asian women and men indicate that they are less likely to consent to be contacted for fertility research, are fatalistic about failure to conceive, less informed about fertility issues, only 36 percent knew that chances of getting pregnant declined with age, and are less likely to suspect a male factor.

Asian women are commonly late at seeking care for infertility and overestimate the chance for getting pregnant.

Look at the national IQs, hardly surprising.

Genetics : Many genes are likely involved. FMR1 is a gene on X chromosome responsible for Fragile X syndrome and its variants. High repeats at this gene may reduce ovarian reserve.

It’s literally genetic. R-selection doesn’t keep those repeats low in the populace.

Did pesky white women interfere with their genes?

Yeah we tinkered with the Ts, the Gs, the As, all of it!

Fertility Treatment Outcomes in Asian Couples

  1. Pregnancy and delivery rates are lower in Asian women following ovarian stimulation and IUI compared to white women
  2. IVF: when compared to white women in the US,  31 per cent of the Asian women gave birth successfully compared to 48 per cent of the white women. Asian women were also less likely to become pregnant; 43 percent against 59 per cent even after control for many fertility factors. Endometrial lining was thinner in Asian women compared to Caucasian women.

Shit, is white supremacy real but only for women? We did need to weather the Ice Age.

I think endometrial lining has a connection to T-levels, it gets thinner with higher T, if memory serves.

I didn’t find enough conclusive data on white men v Asian when I linked sperm quality studies. A little but not as clear. There are fewer studies like that on men in general.

Asian women should be aware that fertility treatment may be less successful and seek care of a reproductive endocrinologist and fertility specialist as early as possible.

In addition there are other factors that require attention in Asian women during fertility treatment especially the higher prevalence of chronic hepatitis B infection.


After conception, asian women at are a higher risk for gestational diabetes.

Only 18% of people in the U.S. have a negative blood type. Yet, when someone with a negative blood type needs blood, only another person with a negative type can save his or her life.

The blood type correlations to medical conditions like mental problems really trigger SJWs.

Are rh negatives more intelligent?

I may sound conceited but yes, yes we are.

That’s why anglos, who have a lot of Rh-neg, have so many historic geniuses.

From obi

Blood type by race/ethnicity:

O-positive is the most common blood type. Blood types vary by ethnic group. More Hispanic people, for example, have O blood type, while Asian people are more likely to be type B. In 2014, Oklahoma Blood Institute saw this ethnic diversity and blood types in its donors.

The O stats for whites are shockingly different.

Limited by who donated, wish I could find broader data.

Some patients require a closer blood match than that provided by ABO positive/negative blood typing. For example, the risk of a reaction to transfused blood can sometimes be reduced if a patient receives blood that is from a donor with the same ethnicity. That’s why African-American donors may be the best hope for patients with sickle cell disease, 98 percent of whom are of African-American descent.

Why mention this now?

“Why do ‘BAME’ get it more?” Well damn I dunno, maybe look at their blood type?

If they don’t wanna catch it here they can always …. go somewhere else?

Short and tall women are slutty
h/t Dutton

I wish they’d look racially, but this explains the r-selected LBFMs.
Tall women I have noted, like Tilda Swinton seem to be left-wing in an openly aggressive, manly fashion.
Short women tend to be left-wing in a subversive fashion, i.e. infantilising rape gangs and cheating on their simp of a husband as a “poly”, the type to know they can’t play the tall woman’s game of leftism so fakes femininity to extract resources (from State, Church, simp). Assuming a short woman is more feminine is statistically wrong. She’ll be more likely to cheat from insecurity.

re Keller, M. (2013). The Genetic Correlation Between Height and IQ. PLOS ONE.
I already knew taller men are smarter and kinder to women, they’re protective. That’s why women like them, not the height per se.
Short men view women as social competition so are more likely to bully, gaslight, verbally abuse and hit them. They think domination makes up for lack of dominance. It’s moral cowardice, “pick on someone your own size” as they used to say.
Nature made taller men healthier because they’re generally better people.
Misogyny is a known factor of inferiority and most of them (with vitriol aimed in-race) are shorter than the women or average man, so subconsciously hate their mother.
Height in a man is broadly like a human’s peacock tail. It develops as a signal of genetic quality which cannot be faked and demonstrates low mutation load. There’s plenty of time before the growth plates fuse to get adequate nutrition and exercise so either their parents hate them (and parents hate more ugly children or products of other unions) or they are burdened with so many mutations it’s a small wonder they weren’t miscarried (and probably would have been without modern medicine).
One big reason modern humans are uglier is IVF, the other being maternal care for parents who simply don’t deserve it.
Read into that what you will.
Maybe the solution is more abortion, but more eugenic abortion.

Average height women have “more reproductive success” (Nettle, 2002) – cited top paper.
This also applies to the third world (they cite). Perhaps the fetish for pedomorphic women (short women, short limbs) is an r-select feature?
Men always prefer women a few inches shorter than them as a norm (so probably same class, assortative) but “markedly” short (as the paper puts it) is abnormal and suggests r-type breeding preferences (young and done, no investment).
Tall women are less symmetrical, although the way they write that sentence up is vague to spare blushes.
Medium height women have “highest mate value” – more studies needed on this. What does that actually mean? Just fertility? Beauty? Personality? IQ? What? Good family?
Jealous women were “taller or shorter than average” – damn, dare you to do a cross-racial study, that would be funny. 
So if you don’t want a harpy guys, select an average height woman (for her own race, presumably, also your own?).

If you wanna be cucked, women, marry a short man,

-or men, marry a short or tall woman.

Short is clearly a non sequitur to feminine, as it states short women are more jealous (along with tall women) of “feminine” beauty.

Average women are more repulsed by masculine women but… isn’t everyone? Can you guess my height by that?

I actually thought that was just everyone.

A few screencaps:

  • Humans are K-selected as a species, believe nobody who says otherwise. No, men are not meant to “sow oats”, it’s degenerate. The quality men don’t do that. Reputation evolved to explain this commentary on your genetic quality.
  • K-types are MORE reproductively successful in the long run, as you’d expect. Evolution is about the long run and who actually reproduces, not just “screws” in completely sterile fashion.
  • Married isn’t always better for the children if the parents (or one) are unstable, in loyalty or sanity.
  • Problem children come from problematic parents.
  • If your child is a slut, it’s your fault.
  • Sex is a physical distraction from growing up mentally, usually escaping from childhood trauma.
  • r/K isn’t really a choice, it’s mostly genetics. Posing as the other type won’t work.
  • 65% genetically heritable. Your kids will be just as slutty as you are, whoever you marry.
  • Hot women can hold out for marriage. Not – cannot.
  • Women are more than morally offended when you treat them as promiscuous (when they’re not) because you’re also calling them ugly.
  • If you want a spouse who can pair bond neurologically and raise your children as a good role model, don’t marry a former slut.
  • Better spouses are genetically fitter spouses (average women, tall men).

Pretty fertile

A woman’s face is the true indicator of fertility, it isn’t really nearly as amenable to later changes at the gym, at the surgeon or by diet. It’s a true signal, trust it over the body if there is a conflict. If a woman has an average face for her race and a “great” body, the body is fake. It must be, because they’re supposed to have developed at the same time, with the same nutrients and genes and hormones. A highly dimorphic body would also produce a highly dimorphic face by the same conditions.

slight repost for SEO

“Attractive facial features in women are assumed to signal fertility, but whether facial attractiveness predicts reproductive success in women is still a matter of debate. We investigated the association between facial attractiveness at young adulthood and reproductive life history-number of children and pregnancies-in women of a rural community. For the analysis of reproductive success, we divided the sample into women who used contraceptives and women who did not. Introducing two-dimensional geometric morphometric methodology, we analysed which specific characteristics in facial shape drive the assessment of attractiveness and covary with lifetime reproductive success. A set of 93 (semi)landmarks was digitized as two-dimensional coordinates in postmenopausal faces. We calculated the degree of fluctuating asymmetry and regressed facial shape on facial attractiveness at youth and reproductive success. Among women who never used hormonal contraceptives, we found attractive women to have more biological offspring than less attractive women. These findings are not affected by sociodemographic variables. Postmenopausal faces corresponding to high reproductive success show more feminine features facial characteristics previously assumed to be honest cues to fertility. Our findings support the notion that facial attractiveness at the age of mate choice predicts reproductive success and that facial attractiveness is based on facial characteristics, which seem to remain stable until postmenopausal age.”

This is how men traditionally found a good wife in a time of petticoats. The face, neck, shoulders and arms show you the rest of her body. Their books emphasize these as important* and their fashions allowed a plunging neckline to better judge fitness. It’s also why they hated make-up, not for the flush but the drawing-on of superior features. This still happens, largely with the midface and eye area. If a girl draws on her nose, just say no.

*They were incredibly bitchy about scrawny, ugly necks and shoulders up until the Edwardian Era. Kiera Knightley’s man bod would be the epitome of ugly. They wanted tapered full shoulders and neck.

That came back in the 1950s, for similar post-war fertility need reasons.

Note the thighs match the neck, so no thin necks either. Lithe arms, nice legs. It’s a rule. They considered the neck and wrists feminine and seriously, what woman nowadays goes to the surgeon about those? It’s a clear signal, still.

Breadth of hips imitated in elbows, palms and knee joints. It’s the same genetic input. A woman with large hips and tiny knee joints has had surgery. The knees develop before puberty to accommodate broad hips. At puberty, they can get even bigger as the legs get longer, so there’s no woman with long legs, regardless of height, and tiny knee joints. It’s impossible, biologically. Otherwise, they’d have biomechanical issues with walking. There’s an angle I can’t be bothered to look up produced by broader hips down to the knee joint and when it exceeds a certain level or gradient the woman can’t actually walk, literal mobility issues. The shoulders aren’t broad in bones (that’s masculine) nor are the feet in women. Long feet are a direct metric of height to stay upright, it’s a hinge but slender feet are the feminine marker and foot-binding’s purpose was narrowness. A woman with broad shoulders and narrow, bound feet has trouble walking. Men also have broader feet to match their shoulders. They throw a lot of weight forward.

A gamine type body, with one or two pleasing features (e.g. just a small waist and long legs) would also produce deficits in maternal instinct and capacity. Caveat emptor. This is important for men selecting a wife, as opposed to a quick shag (what society tells you). If you have a choice of two women, pick the nubile one.

Curvy women may sag, yes, but they don’t come to resemble a man, especially after menopause (see study above).

I’d like to see a study of husband’s desire for his wife by body type. Imagine the outrage. Apples/Naturals would be the worst. Just avoid women with broader shoulder bones than hips. You can tell at a distance.

Healthy shoulders and such are ignored nowadays with dire sub-fertility consequences. Babies eat that fat.

Note, a daintier wrist because the elbow join is broader.

The entire body is a signal, T&A is a ((distraction.)) Padding will do those.

Note the emphasis. That was behind Marilyn’s charm. Victorian body in 50s Americana clothing.

Nowadays we have knee length skirts as normal but very low necklines as odd or even offensive. It’s possible to derive fitness better from the legs, like a deer, really, or a horse, but most men have lost the skill or drive, fetishising tights and heels instead. Upper class men still judge by and fetishize the legs and were behind the rationing shift in fashions to display them. Middle class men fixate on backsides but not hips, which would be a superior indicator as breadth. Lower orders fixate on breasts, high time preference indicated. The woman herself may already be pregnant.

HIV cure was a lie


They wanted money?


In other news:

They want to control your curiosity. Part of the mentacide protocol.

Dya have a license to search for the holodomor?

But curiosity has also been associated with characteristics that reflect risk taking, stress tolerance and thrill seeking. This is how curiosity got its bad wrap as a mortal danger to felines.

They’re trying to replicate white characteristics so they can replace white people.

Doesn’t work – the genome is one whole thing, even junk DNA affects the rest.

We all know some people tend to be more curious than others. Supporting this, research shows some individuals experience curiosity more frequently or intensely than others. But is curiosity as a  just a level of degree—more versus less?

They know it’s connected to IQ.

They KNOW.

Everyone knows at this point.

Epistemic curiosity has been widely researched. This describes a person’s desire to acquire new information—such as facts, concepts or ideas—and bridge any gaps in their knowledge.

God forbid people seek out their own information and fact-check you asshats instead of blinding swallowing your shit.

Hey, didn’t you a-holes say “educate yourselves”?

I know it was a master-suppression technique but Ys and Zoomers actually listened.

People who show perceptual curiosity, on the other hand, try to maximise the sensory information they take in—like your friend who can’t stop looking around at anything and everything.

That’s called bad parenting. It’s connected to low IQ. That is perverting the definition of curiosity.

Curiosity = need for cognition.

This research can help us to understand how we can better harness curiosity in the real world, such as in work and educational settings.


Imagine your boss dialing up your arousal at work like a fucking thermostat.

Gaining knowledge in this way would be very different from just delivering a set teaching material.

Trying to cover IQ range differences in school, eh?

Maybe another century of pretending that works, might work?

If the teachers are so lazy, replace them with robots.

Current research has shown that the effects of curiosity on learning are even stronger for children from families with a low socioeconomic status.

Idiots are less likely to zone out if you jangle keys, can confirm.

“The fornix is part of the limbic system.”

So it’s an r/K thing.

Click to access CLUFF.Edwards.UNDERGRAD.pdf

Need for Cognition, Intelligence, and Aging 

This study examined the constructs of need for cognition and intelligence (using the constituent
crystallized and fluid abilities that comprise overall intelligence) in relation to one another and over aging. Fifty young-old adults (54-69 years old) and 55 old-old adults (70-92 years old) were tested on a variety of measures, of which need for cognition, digit symbol (a measure of fluid intelligence), and vocabulary (a test of crystallized intelligence) were identified as outcome variables. The results suggest that need for cognition is significantly correlated to crystallized intelligence, need for cognition remains stable over aging, and fluid intelligence is best predicted by age.

They want to brainwash your kids out of all instincts for self-preservation.

“The study, led by Professor Elinor McKone, examined the “other-race effect,” a phenomenon

they made up

noticing things means you brain is WORKING

in which people have difficulty telling apart individuals of a different race to their own.”

This is why you don’t go on Third World foreign holidays, btw.

Unless you want them going off age 18 and turning up dead in Thailand with their false sense of security finally burst.

Kids need more bonding time with BOTH parents (no deadbeat dads), the oxytocin will offset the propaganda.

“The other-race effect can have serious real-world consequences,” Dr. Dawel said.

“For example, inaccurate cross-race eyewitness testimony has contributed to wrongful criminal convictions, passport misidentifications and even magazines mistakenly illustrating stories with a picture of the wrong person.”

That isn’t serious so they’re lying.

The team is now working on developing new training methods to reduce the other-race effect in adulthood.

That doesn’t sound ominous at all.

They’re still only studying the women. Like women self-impregnate.

Can’t have the guys knowing all the DNA damage of soy, wheatgrass, HGH, etc.

Look at diet and bone marrow, duh.

Female medieval English skeletons study

“However, the period of ‘youth’ in medieval England, before the achievement of full social adulthood, may have extended well past physical adolescence, and the age of 25 years is often used as the cut-off point.14″

Louder for the pedos at the back.

“but for most medieval young women physical adulthood did not equate to social adulthood.16″


“Instead, puberty may have marked the beginning of the phase of ‘maidenhood’ rather than adulthood.17”

We now call it teenagehood but I prefer that name for women.

“Lifestyle changes for the teenager, in particular the onset of formal work, may have marked a further step away from childhood, particularly if this involved a move away from the parental home. That the 14th-century poll tax was levied on all those aged 14 years and above suggests that young women were expected to be earning their own money by this age.18

HA. Yeah, the guys who say women should sit at home all day twiddling their thumbs waiting to marry are 1. wrong and 2. have put too much stock in middle-class novelist Austen.

Like today;
“Although exact numbers are impossible to calculate, it is clear from the documentary evidence that a significant proportion of young women migrated to urban centres such as London and York to obtain employment, most commonly a service position.19″

Exactly like today:
“This move would have been a dramatic, and potentially a traumatic, change in lifestyle for young women. Although it may have brought greater freedom and responsibility, it does not seem to have conferred full adult status; there is evidence that young women in service were always viewed as ‘girls’ regardless of their age, just as young men were not viewed as full adults before the completion of an apprenticeship contract.20″

So they didn’t marry for money, they were already economically independent.

If you actually read history and here, forensics.

“in reality, marriage at such a young age was largely restricted to the nobility, with the average age at marriage in the general population estimated at 20–25 years,22
and perhaps even later following the Black Death.23
This would provide a very late age of achievement of ‘adulthood’ by modern standards. However, although marriage was very much the expected path a significant minority of women — perhaps around 15% — never married.24″

Who is dumb enough to have never looked this up?

I keep seeing Americans who make sweeping fictional statements about what ‘we’ Europeans did and it’s like… no. That’s never happened. Citation? Statistics? They are liars. Even in their revenge fantasies of ‘oppressing’ women from work (oh joy, welfare on the backs of random men? can’t win, can we?) then they assume all women would marry off (literally never happened in human history), all women are fertile and their children all magically survive (LOL) and that all men want to marry and got to choose who (LOL no). The economy also needs young workers, part of the immigrant problem is caused by not allowing teens to work.

They’re in bloody La La Land.

Extended maturation is K-selected, the men and women were tougher as a result.

Just realised my grandmothers might be in here.

Almost certainly. Yeah, don’t lie about my nana/s.

“Alongside these dramatic but infrequent events, most young medieval females would have experienced everyday hardships and hazards.”

” The average femoral diaphysis length recorded for the medieval 14-year-old females (354 mm) is closest to that recorded by Maresh for 20th-century 10-year-olds (348 mm). The average figures for medieval 15- and 16-year-old females (365 mm and 366 mm respectively) are still lower than for 20th-century 11-year-olds (367 mm). These data suggest that growth in medieval England fell well below modern standards, perhaps reflecting the lower standard of living medieval children would have experienced.”

If it was that hard on the girls, you don’t wanna go back to that, guys.

“It does not necessarily follow that medieval women were considerably shorter than their modern counterparts. When compared to dental formation, epiphyseal fusion in the female adolescent skeletons from our sample was delayed by two to three years in comparison to modern standards, allowing them to ‘catch-up’ their growth during the pubertal growth spurt.27 This pattern of extended growth appears to have been common in the medieval period;”

The English are tough.

” Only very slight differences in stature were noted between the women of Lincolnshire, London and Gloucester, although the London females had greater diversity in adult height.”

“This may suggest that girls who experienced poorer conditions for childhood and adolescent growth were more likely to die around or before the age of 25 years.”

K-selection. Stunted or shorter women likelier to die. Same with men.

“It has been suggested that female height may have suffered in comparison to male height in medieval Europe due to preferential feeding and care of male children,33causing greater sexual dimorphism in growth and final stature between the sexes. By comparison, the average stature of young men at our sites (156 individuals) was 169.5 cm (5 ft 7 in). This may simply be the result of sexual dimorphism as such comparisons are similar for modern western populations, and therefore does not support the hypothesis that girls experienced poorer nutrition and living standards than boys.”

K-types invest well in all offspring.

“According to these indicators, it appears that all of the individuals studied had entered the pubertal growth spurt by the age of 14 years. In the modern western world girls tend to begin puberty around the age of 10 years,37 and so this result would fit with modern expectations. “

Puberty begins then takes a few years, 14-18/19 matches what I read elsewhere about menarche (posted here).

The ‘modern’ data is skewed by non-whites, especially Asians and Africans, with much lower menarches.

The African is nine, measured in America, as I recall.

“More information can be gained from examining the epiphyseal fusion of the hand phalanges, a process known to occur during the deceleration phase of the pubertal growth spurt, and correlated with first menstruation in modern females. Although the age at which this event occurred varied in our sample just as among modern girls, fusion appears to have occurred most frequently between 15 and 17 years (Fig 2). At 14 years, only 36% of girls display fusing or fused hand phalangeal epiphyses, but by 17 years this figure has risen to 84%.”

Still not 100%, K-types have a later range of menarche.

“A second skeletal event known to be associated with first menstruation, the ossification of the iliac crest of the ilium, was also only found in girls aged at 15 years or over. Interestingly, this is roughly in line with the average age at menarche suggested by the few available documentary sources.38 An average age at menarche of between 15 and 16 years would be much later than the modern British average of just under 13 years.39In addition to their shorter stature, this finding adds weight to the argument that environmental factors such a deficient diet and disease were having a negative impact on medieval female growth and development. Interestingly, however, this average age at menarche is below the age of 17 years recorded for mid-19th century females,40indicating that urban conditions were not as detrimental as those experienced during rapid industrialisation.”

The female body takes YEARS to develop, periods often occur too early to carry a child to term. Hollywood lies, because it’s full of creeps.

Men shouldn’t be discussing a reproductive system they cannot understand.

“The evidence for medieval England, however, shows a delay in the achievement of this milestone, which appears to have fallen between 17 and 18 years for most girls, based on 247 individuals with this bone surviving (Fig 3). Complete fusion of the iliac crest of the ilium, which signals the end of pelvic growth, was only seen in a minority of women aged below 20 years, based on the 277 individuals “

They’re K-types, it isn’t a delay, it’s NORMAL. Modern people are aberrant.

17-18 periods stabilize (this takes years, I have spoken to doctors about it).

The pelvis keeps growing to carry and support a child though, only when this is done (about 21, spinal plate fusion) is the woman actually sexually mature with a low risk of still birth, miscarriage or death.

Modern medicine is allowing a lot of non-white thots to survive a process Nature is telling them is fatal. Do not confuse that with Nature’s approval.

These data suggest that puberty was extended into the very late teens for young medieval women, pushing back the timing of achievement of full physical adulthood. This extended period of physical adolescence indicates that living standards for young medieval women, at least in the urban and small town environments, were considerably poorer than those of modern British adolescents. Some variation between the sites was noted, with pubertal development most advanced in the small town of Barton-upon-Humber, and most delayed in the urban hospital cemetery of St Mary Spital, London. This presumably reflects the harsher living conditions experienced by the girls living and working in London.”

Nah, hard work and low fat diet. Treating the women like men will delay them more.

“It is believed that the demographic changes caused by the Black Death may have led to increased opportunities for many women to migrate and work.43

Although less documentary evidence is available for women than for men, there is evidence for female servants much younger than 12 years in urban households,44and some migration may have occurred at a very young age. Although legislation was passed to regulate the minimum age for apprentices — 13 years in the early 14th century, rising to 16 years by the 15th century — apprenticeships were rarely available for girls, and no such legal minimum age existed for servants or casual workers. The available evidence suggests that girls started formal work away from home at a younger age than boys.45

This concept of female laziness is really American.

” a degree of personal freedom; the latter is perhaps most clearly indicated by the large number of migrant women recorded as making ‘merchet’ payments for the right to choose their own marriage partner.46 On the other hand, moving away from home, particularly to a town or city, could bring with it new challenges and hazards, such as sexual predation, mistreatment, injury and disease.”

Americans are so wrong it hurts.

” this result indicates that much greater numbers of women living and dying in London were actually suffering from tuberculosis.”

“Again, the numbers are too small for statistical analysis, but this may provide further evidence for girls having a more indoor lifestyle than boys in the medieval period.”

Forcing women to sit at home is literally bad for their health.

We aren’t mole people.

On the whole, the women actually had it harder than men.

“There can be little doubt that this extensive workload was exhausting for many women, but osteological study can provide further direct evidence for the impact that this had on young women’s bodies.

A wide range of trauma has been recorded on the skeletons of young medieval women, including fractures of the upper limb and finger bones, cranium and ribs, lower limbs and feet.57 However, the prevalence of fractures of each type is lower than among males, suggesting that girls were exposed to (or exposed themselves to) fewer risks of injury than boys.”

We hadn’t evolved for that labour, men did.

“It is notable that, of the 48 cases of trauma reported in the grey and published literature, cranial, rib and jaw injuries, suggestive of interpersonal violence, only started to appear in women aged 17–25 years, comprising 18.6% of the 43 fractures for this age group. This suggests that the risk of violence rose as girls turned into young women, perhaps reflecting domestic violence after marriage.58″

That would explain the death rate. Stress and fractures – no healthy baby.

There is one area of the skeleton where young women seem to have suffered virtually the same frequency of fractures as young men, the vertebral column. By far the highest prevalence rate for vertebral fractures (4.7%, n = 9) was found at St Mary Spital suggesting that female workers in the capital, or at least the poor workers buried in this hospital cemetery, were undertaking the activities most likely to cause spinal injury. The majority of these fractures were compression fractures, often caused by falls from a height, although avulsion and hyperflexion injuries were also present.59

The men sitting at a desk in an apprenticeship had it easy.

“Schmorl’s nodes are common, often asymptomatic, depressions caused by herniation of the nucleus pulposus on the superior and inferior surfaces of the vertebral bodies. Their aetiology is complex, although spinal trauma caused by vigorous activity and flexion and extension of the spine is most commonly associated with their formation.60 The age of their occurrence is not clear, but they generally appear before the age of 18 years.61Plomp et al argued that males are more susceptible to these lesions due to the size and shape of their vertebrae.62 In our study, medieval women had a higher prevalence of the lesions). Analysis of the location of Schmorl’s nodes on the vertebrae revealed that the lumbar vertebrae were affected far more often among women, and the central thoracic vertebrae among men. This mirrors vertebral fractures where in the women all of the fractures occurred in the lower thoracic and lumbar vertebrae, while in young men the central thoracic vertebrae were affected. This may suggest different activities; strain on the lumbar vertebrae, in particular, may be caused by bending and lifting.63″

aka back breaking labour, which could cause…

“Further evidence for stress being placed on the spines of young medieval women is provided by cases of spondylolysis. This describes the partial separation of the inferior facets on the neural arch from the vertebral body, usually between the ages of 10–12 years. The condition results from microtrauma in low grade stress on the lower back due to bending and lifting strains, or a fall from a height,64 but may have an underlying congenital cause. This injury was present in 4.4% of the female skeletons examined. This is higher than the prevalence of this condition found by the authors among young medieval males (2.9%), although the numbers involved were too small for statistical analysis. Again, the area involved is the lumbar region of the vertebral column. In addition, three young women, two aged at around 21 years and one at 22–25 years, display early degenerative joint disease of the vertebral column.”

Forcing women into labour like that kills them, reminder.

What emerges from the osteological evidence is that the workload of many young medieval women appears to have been literally backbreaking, and these early injuries may be expected to have led to significant back problems and pain in later life. It seems likely that these early spinal problems were caused primarily by carrying heavy loads at a time when the spine was still forming and vulnerable. Research from the grey and published literature reveals that rates of spinal injury were higher in urban than rural women65 and suggests that the workload of the young migrant women in service was harder than that of the young women who remained in the country or in small towns with their families. For example, the prevalence of vertebral fractures, spondylolysis and Schmorl’s nodes was lowest at Barton-upon-Humber, a wealthy small town.66″

Marriage, Sexual Activity and Childbirth

There is considerable evidence to suggest that marriage was a defining moment in the medieval female life course, marking the transition into true social adulthood.67 It is notable, however, that there was a significant gap between the legal age at marriage (12 years) and the average age at marriage (20–25 years even before the Black Death) in medieval England.68 The new analysis of pubertal development in medieval England discussed above suggests that the average age at menarche was 15–16 years. Full fertility, in terms of the likelihood of conception, carrying a healthy pregnancy to term and surviving childbirth, would only have followed several years after menarche with the completion of pelvic growth,69 which in our medieval sample appears to have been rare before the age of 19 years.

aka what I already typed, dammit

The fact that many young medieval women would not have been fertile before their 20s may be one reason for the relatively late average age of marriage during this period.70 It also suggests that marriage at the legal minimum age of 12 years would rarely have been fruitful, and any pregnancy that did ensue would have carried significant risks for the mother. We know of several medieval legal cases of the marriage of young girls where the ‘physical readiness for marriage’ of the girl in question was debated.71

This don’t go to college because you get periods thing from America is pig ignorant on female anatomy.

There is evidence to suggest, however, that the majority of cases of marriage before 15 years were confined to the nobility.72Today, girls of higher socio-economic status, with a considerably better standard of life, mature earlier than average. For example, high caste girls in 20th-century India have an average age at menarche over a year younger than low caste girls.73 The average age at menarche for noble girls in medieval England may therefore have been younger than the average age of 15–16 years described above.

more r-selected by men, explains eventual decadence and homosexuality rates, especially in the French

Even so, a pregnancy before the completion of pelvic development would have been dangerous; a famous example of this is provided by Margaret Beaufort, who appears to have been rendered sterile by a difficult first birth (of the future king Henry VII) at the age of just 13 years.74 An understanding of these risks is demonstrated by several contemporary authors,75 and was reflected in the Jewish rule that contraception (banned by Christian teaching) could be used to prevent pregnancy if the bride were too young to safely bear a child.767

The guys trying to force women to reproduce young would ironically render their own wife sterile via their stupidity. Good riddance. The Lord works in mysterious ways.

In theory, marriage coincided with sexual initiation for young women, and if the Church’s remonstrations to remain celibate until marriage were universally followed, it would indicate a relatively late age of sexual initiation. In reality, premarital sex among betrothed couples seems to have been common,77

that links to this study, no, they weren’t slutty



and sex with other partners, in not all cases consensual, was far from rare.

Are you really counting rape?

Evidence for this is provided by the erratic enforcement of ‘legerwite’ or ‘leyrwite’ fines on serf women who engaged in premarital sex.78

What about the men.

Premarital sex is thought to have been particularly common among young girls and women living away from home, for example in service roles, due to the greater freedom and availability of partners as well as the risk of sexual predation or pimping from employers.79 The sexual exploitation of girls in service appears to have been a frequent problem based on the legal record,80 and many young women must have lost their virginity in these circumstances. The extensive focus of many writers on admonishing young women to stay celibate until marriage may be taken as further evidence that premarital sex was seen (at least for women) as a significant societal problem.

Rape isn’t sleeping around, WTF.

Pedophiles raping virgins don’t really count as premarital sex, a choice, does it?

Two aspects of osteological analysis may shed light on sexual activity among young medieval women. The first is a sexually transmitted disease. Venereal syphilis, a treponemal disease, affects the skeleton in its tertiary stage, causing distinctive skeletal lesions.81 From the end of the 15th century, syphilis is believed to have been endemic in urban areas of England, although recent work has suggested that it may have been present at a much earlier date.82

Men spread that, sailors caught that. Your point?

If a virgin woman married a man with it, she’d get it. That can happen after marriage.

These female authors really want to present all women throughout history as sluts. Cui bono?

Among the 14–25 year old female individuals examined, four probable cases of treponemal disease were recorded, based on the presence of characteristic gummatous lesions in the cranium or long bones.83 Three of these were found in the young women from London (Fig 5), and one was found in York, at St Helen-on-the-Walls. One further case is known from Blackfriars, Gloucester;84 no cases were identified in the rural or small town sites consulted in the wider survey. The two youngest women to show signs of treponemal disease were aged at just 16 years. It is difficult to rule out congenital syphilis in these cases, as the presentation of the two conditions can be very similar, although none of these skeletons display the typical dental deformations of congenital syphilis.

So their fathers were sluts, so?

If the disease is the venereal form of treponemal disease, or syphilis, this would suggest the girls were very young when first infected. Syphilis generally takes several years to cause such destruction in the skeleton.85 Although the number of cases recorded is small, given that only 10–20% of individuals with tertiary syphilis experience skeletal involvement, and that skeletal lesions take several years to develop,86 it seems likely that much greater numbers of young women were affected by this disease.

To imply they wanted to be raped by syphilitic men is a bridge too far though.

The spread of sexually transmitted diseases such as syphilis was exacerbated by the problem of prostitution in medieval towns and cities. Karras argues that regulations of the Guilds limited women’s access to the normal labour market, forcing them to turn to prostitution out of necessity.87

Assuming that was a mistake.

There is little direct evidence that apprentices were procured as prostitutes, but one extant record from London City and Ecclesiastical Court (ad 1423) attests that one Alison Boston took apprentices who she hired out for the ‘horrible vice of lechery’.88 There are also accounts of men taking young girls (invenculae) to the London stews and selling them as prostitutes, suggesting the types of danger faced by young unskilled immigrant women. Goldberg89 cites the famous references from medieval York in ad 1482 that place prostitutes within the legal realm of ‘lepers’ and pigs in the hazards they caused for the local population.

Enslaved children.

She does not discuss the age at which women may have turned to prostitution, but suggests widows and daughters of labourers, known as ‘spinsters’ and ‘seamstresses’ (sempsters), needed to work several jobs to make ends meet, including petty theft, illegal ale retailing and prostitution. Goldberg argues that although full-time, ‘professional’ prostitutes were rare, many women were forced into occasional prostitution in hard times.90

Contradiction, Goldberg.

also why we have the welfare state

This would have been a particular risk for a migrant girl away from the safety of her family.91 Although it is impossible to state that any of the young medieval women examined were forced into this profession, this must be considered in the cases where possible syphilis is recorded.

No shit, nobody would choose that. The excuses these women make for rape are appalling.

A second consequence of sexual activity, pregnancy, may also in exceptional circumstances be visible in the archaeological record. In total, eight cases of young women buried with fetuses in utero have been recorded from medieval cemetery contexts. These burials represent ‘obstetric catastrophes’ with the death of both mother and child in late pregnancy or childbirth. Although there was a Christian injunction in place in medieval England for infants to be removed from their mother’s womb before burial,92 this does not appear to have been rigorously obeyed.

Yeah, who wouldn’t choose to die like that? I guess they were all just happy sluts, huh Mizz Feminist?

All of the individuals buried with a fetus in utero in medieval cemeteries have an estimated age at death of around 20 years or over, and thus none represent particularly young ‘teenage’ pregnancies.

Because they rarely got pregnant. Look at the evidence.

This may support the idea that in the medieval period teenage girls were not falling pregnant, as first pregnancies are often seen as the most hazardous.93

May? It’s anatomical.

It also fits with the known late pattern of marriage in this society. However, it is by no means certain that all of these women were married. The two examples from St Mary Spital may have represented extramarital pregnancies as the hospital was known to accept unmarried women in pregnancy or childbirth.94 It may be significant that neither of these women received an individual grave or any grave ornamentation. In contrast, the elaborate nature of one young mother’s burial at Barton-upon-Humber, in a coffin within the church and with a cloth of gold artefact,95 surely indicates that this woman was married and held a position of substantial social standing.

Clearly, their situation was a choice.

Given the high mortality rate of women in childbirth in the medieval period revealed by documentary sources,96s it is clear that these rare burials represent a dramatic under-estimation of the real levels of maternal mortality. In many cases, the churches prohibition on burying fetuses in utero may have been observed. In a large proportion of births, too, the child may have been saved, leaving little clue as to the cause of death of the mother.

But doctors (when sane) will elect to save the mother because she can have countless children later but an orphan baby is already financially a goner. Remember this, America.


The period of social adolescence for young medieval women seems to have been an important life stage, encompassing the growth to full physical adulthood and fertility, the adoption of adult working roles and, for most young women, the move from legal dependence on a father to legal dependence on a husband, with perhaps a few brief years of relative independence in between. The comparative absence of young women from documentary sources means that osteological information plays a vital role in our understanding of this group, and it can reveal a great deal about the way in which medieval girls grew into women, the living conditions they enjoyed or endured, the work they did and the health problems they faced.

Many of the conclusions drawn from osteological analysis of this group articulate with and illuminate the documentary evidence. The average age at which full fertility appears to have been achieved, around 20 years, is substantially later than in modern England, but ties in well with the known average age at marriage in this society. The greater susceptibility of young women to respiratory infections, from the relatively benign maxillary sinusitis to the deadly serious tuberculosis, chimes with the picture drawn from documentary sources of an indoor lifestyle for women, close to the smoky fire, and of the cramped living conditions that helped to spread disease. The backbreaking work clearly undertaken by many young women paints a clearer physical picture of their daily lives than that provided by documentary sources alone, and the development of signs of venereal disease in very young women hints at the problem of girls being driven to prostitution in England’s medieval cities.

Gang rape, we still have it. They are driven to it, slave-driven.

Food babies

Insight into vegan menopause.
“Drinking whole fat milk and eating ice cream appears to be better for women trying to become pregnant than a diet consisting of low-fat dairy products such as skim milk and yogurt, according to new research published in Human Reproduction journal. Researchers in the United States have found a link between a low-fat dairy diet and increased risk of anovulatory infertility.”
You don’t have to tell me twice to eat ice cream, mate.
“Further, lactose (the main carbohydrate in milk and dairy products) may not affect fertility within the usual range of intake levels in humans.”
Good to know.
“Consumption of iron supplements and nonheme iron from other sources may decrease the risk of ovulatory infertility.”
Yes, my favourite iron supplement is called steak. Recommend.
Especially with three times “too much” garlic butter.
“Folic acid appeared to explain part of the association between multivitamin supplement use and risk of ovulatory infertility.”
Sorry but I don’t trust Crunchy Nut cornflakes with the fate of my future children’s health.
“Dairy consumption was not significantly correlated with PCOS. However, after adjustment for confounders, there was an direct relationship between milk consumption and risk of PCOS.”

“the results showed that the intake amounts of Ca, Mg, D vitamin, dairy, fruits and nuts and seeds were remarkably low among the women with PCOS.”

What are they living on, Oreos?

“some evidence on the decreased amount of adiponectine, calcium, D vitamin in the patients suffering from PCOS with having a higher thyroglobulin”


“Numerous studies have demonstrated the association between the diet and its components and risk factors developing various diseases.[] However, previous studies did not address the relationship between nutrition choices and type of diet chosen by the patients.[]”
“According to existing data, women trying to achieve pregnancy are encouraged to increase consumption of whole grains, omega-3 fatty acids, fish, and soy and to reduce consumption of trans fats and red meat. In addition, a daily multivitamin that contains folic acid before and during pregnancy may not only prevent birth defects, but also improve the chance of achieving and maintaining a pregnancy. In contrast, there is limited evidence supporting an association betweenvitamin D and human fecundity outcomes despite promising evidence from nonhuman studies.
Yeah because that’s relevant.
Questions for future research included the roles of other types of fat (especially omega-6 and monounsaturated fats) and protein (especially white meat and seafood) on female fertility; particular attention should also be paid to exposure to environmental contaminants in foods. Although much work remains, this review accrued best available evidence to provide practical dietary recommendations for women trying to conceive.”
Bolded valuable parts.
“One approach to minimize the impact of anovulation on fertility is supplementation with progesterone during recruitment, selection and final stages of development of the preovulatory follicle. It is suggested that a minimum of 2.0 ng/mL of progesterone is needed during growth of the preovulatory follicle to achieve P/AI similar to that of cows growing the preovulatory follicle during diestrus.”
But one of the Pills is that….
Does taking that Pill increase odds of pregnancy?
Big if true.
The literature on the relationship between diet and human fertility has greatly expanded over the last decade, resulting in the identification of a few clear patterns. Intake of supplemental folic acid, particularly at doses higher than those recommended for the prevention of neural tube defects, has been consistently related to lower frequency of infertility, lower risk of pregnancy loss, and greater success in infertility treatment. On the other hand and despite promising evidence from animal models, vitamin D does not appear to exert an important role in human fertility in the absence of deficiency.
But most people are deficient….
That’s like saying you can stand outside normally -except when there’s a hurricane.
And there’s currently a hurricane.
Antioxidant supplementation does not appear to offer any benefits to women undergoing infertility treatment, but it appears to be beneficial when it is the male partner who is supplemented.
Reduce genetic load.
However, the available evidence does not allow discerning which specific antioxidants, or at which doses, are responsible for this benefit. Long-chain omega-3 fatty acids appear to improve female fertility, although it remains unclear to what extent contamination of shared food sources, such as fish with high levels of environmental toxicants, can dampen this benefit.
Eat this mercury (sushi fad) it’s good for your baby!
Lastly, adherence to healthy diets favoring seafood, poultry, whole grainsfruits, and vegetables are related to better fertility in women and better semen quality in men.
Could you be a little more specific?
You just named, like, most food.
They’re paid with our taxes, daylight robbery.
The cumulative evidence has also piled against popular hypotheses.
I doubt that.
Sounds like someone wants a juicy research grant.
Dairy and soy, once proposed as reproductive toxicants, have not been consistently related to poor fertility.
Because you refuse to test them separately.
If only a group ate one and not t’uther.
In fact, soy and soy supplements appear to exert a beneficial effect among women undergoing infertility treatment.
Many have low estrogen, next!
Giving people with scurvy vitamin C helps! Next!
Similarly, because data from large, high-quality studies continue to accumulate, the evidence of a potentially deleterious effect of moderate alcohol and caffeine intake on the ability to become pregnant seems less solid than it once did.
But potentially, if you can’t put down the coffee or wine, Barbara, maybe you shouldn’t be having kids? Same goes for men without the energy. Now. Without kids.
Why do narcs consider children a human right?
Sort your energy level out BEFORE kids, at least?
While a complete picture of the role of nutrition on fertility is far from complete, much progress has been made. The most salient gaps in the current evidence include jointly considering female and male diets and testing the most consistent findings in randomized trials.
Fuck, men influence pregnancy? What witchcraft is this?
Their only biological contribution is determined by things that occurred in the years before conception?
Say it isn’t so.
What term could possibly exist to describe this Darwinian fitness between men?
Hold men responsible for their bad habits affecting their innocent child’s health outcomes and literal lifespan and I might be impressed.
Posting will be patchy because of my feelings.

Waist-Hip Ratio and female beauty

The sexual dimorphism for this metric is obviously lowest on Asians.

Evidence is presented showing that body fat distribution as measured by waist-to-hip ratio (WHR) is correlated with youthfulness, reproductive endocrinologic status, and long-term health risk in women. Three studies show that men judge women with low WHR as attractive. Study 1 documents that minor changes in WHRs of Miss America winners and Playboy playmates have occurred over the past 30-60 years. Study 2 shows that college-age men find female figures with low WHR more attractive, healthier, and of greater reproductive value than figures with a higher WHR. In Study 3, 25- to 85-year-old men were found to prefer female figures with lower WHR and assign them higher ratings of attractiveness and reproductive potential. It is suggested that WHR represents an important bodily feature associated with physical attractiveness as well as with health and reproductive potential. A hypothesis is proposed to explain how WHR influences female attractiveness and its role in mate selection.

Hello sexual selection, tied intimately to natural selection.

PDF here:

also connected to “desire and capability for having childrenp7 or 299.

so K-type women may have better WHR.

Normal weight women have the most positive attributes associated.

Overweight category was universally unattractive.
It’d be nice to see a male study on this. I think Western women would want more children if fewer men were obese.

Why Asians are considered youthful but not sexy (they’d usually fall in the underweight group):

The variables of attractiveness, sexiness, and good health were located close to each other, suggesting that subjects perceived them to be closely related.

Attributes of desire and capability for having children were located close to each other in the solution space but farther from attractiveness, sexiness, and good health, implying that subjects did not perceive a great similarity between these two sets of attributes.

Finally, the attribute of youthfulness was located alone and away from both sets of other attributes. Thus, subjects apparently did not perceive youthfulness to be related to any other measured attributes of good health, sexiness, attractiveness, and desire and capability for having children.

So there’s that. Nobody’s jealous.

Figure N7 was located closer to attractiveness, sexiness, and good health as well as desire and capability for having children than any other Figure.

Normal weight for frame (and race) + most nubile WHR would make sense.
More of those genes survived.

Figure N9 was located closest to desire and capability for having children, whereas Figure N8 was located between Figure N7 and Figure N9. The figure N10 was grouped along with overweight figures, which were not perceived to be closely associated with any of the attributes under investigation. Underweight female figures, U7 and U8, were associated only with youthfulness. However, underweight figures with high WHR (U9 and U10) were perceived as neither youthful nor healthy, in spite of the fact that their depicted body weight was quite similar to figures with lower WHR.

Women with an atrocious WHR (boy hips, no waist) and under or overweight for their skeleton are objectively unattractive from an evolutionary standpoint. This would apply whether it’s a Jap, a Ruskie or an American.

Stop calling sexy science ‘racist’ because it doesn’t share your fetish.

This chart drags you harder than I ever could.

Your Asian girlfriend with the boy hips is approximately as attractive to the world as the average WHR white fat chick. That’s your level, accept it.

It’s also a fact we cannot accurately perceive attractiveness of the racial outgroup as well as our own, so an awareness of ingroup flaws changes nothing.

Most modern women straight up don’t look healthy, whether they’re American, European or, yes, Asian.

Stop trying to make boy hips = sexy happen. It’s not going to happen.

Look at the damn gradient on that underweight thing. The solution to fat women isn’t anorexia. That also suggests bad genes. In fact, at least the fat percentage on slightly overweight 0.7 WHR women suggests femininity and fertility.

“Overall, it seems that subjects inferred reproductive capability from body fat”

What does a foetus feed from?

“Thus, it seems that although WHR is related to health and attractiveness, body weight is perceived to be related to reproductive capability”


“As a group, underweight figures were assigned the lowest reproductive capability, followed by overweight figures and then normal weight figures.”

Suck on that, soyboys.

You actually tend to downgrade. That’s why the Democrat-voting soyboys all want an Asian girlfriend and expressly don’t want kids with it.

“Overall, it appears that both fatness and thinness are perceived as unattractive, and such figures are not perceived as having especially high reproductive potential. “

Not womanly. Remember that word? This:

Not girly, not sexy, not cute, not hot. Womanly.

You can’t discuss women in a reproductive, evolutionary context without it.

Thus, consistent with the present findings, men did not find thin or underweight figures attractive.

If you only care for other male opinions.

There is some evidence that suggests that being extremely underweight or overweight can have adverse effects on female reproductive functions.

Ya don’t say?

A critical body mass has been shown to be significantly related to the onset of menstrual cycle and its maintenance (Frisch & McCarthur, 1974), although recent evidence (DeRidder et al., 1990) suggests that it is the body fat distribution, rather than body fat mass or body weight, that is related to early pubertal development.

Distribution varies by race.

Africans are the most pronounced in women then Europeans shapely but delicate then Asians last – no shape, very yang flesh (broad but flat or full in the middle like cortisol fat) and almost nothing to distribute.

Am I imagining all of this?

Underweight females (15% below ideal body weight) have been reported to have a higher incidence of oligomenorrhea (menses 35 days or more apart) and amenorrhea and to have a higher prevalence of ovu-latory infertility than normal weight females (Green, Weiss, & Daling, 1986).

Underweight women also give birth to infants who are small and growth delayed, and such infants often have permanently impaired intellectual and physical development (Supy, Steer, McCusker, Steele, & Jacobs, 1988).

Menstrual dysfunction and ovulatory infertility also occur more often in females who are 20% above ideal body weight (Green et al., 1986). Morbid obesity in females with high WHR has been shown to increase the degree of androgenicity (increased percentage of free testosterone) and associated menstrual and ovulatory problems (Kirschner & Samojilik, 1991). Thus, the reproductive success of a woman may be low in spite of a high level of fat deposits if the regional distribution of fat is not appropriate, that is, gynoid.


Finally, the finding that underweight figures were assigned high rankings for youthfulness but not for attractiveness (or other attributes related to reproductive potential) is difficult to reconcile with some evolutionarily based mate selection hypotheses.

Normal men aren’t pedos.

Youthfulness and health have been proprosed as absolute criteria for female attractiveness (Symons, 1987).

Stick with health.

Health has good or bad, you have no negative way to assess youth e.g. immature.

Features of physical appearance associated with youth supposedly provide the strongest and most reliable cues for female reproductive potential. The present finding illustrates that the relationship of youthfulness and attractiveness is quite complex.

Not really.

A woman who is judged to be attractive is also found to be youthful; however, youthfulness alone does not make a woman attractive. Apparently, youthfulness is a necessary, but not a sufficient condition, for determination of female physical attractiveness.

crazed pointing-

also, don’t try to chalk this up to taste:

“Furthermore, if the ideal of female attractiveness is arbitrary and ever changing, no evidence of transgenerational stability in the meaning of WHR should be found, as older men are more likely to be exposed to different ideals of attractiveness than are younger men.”


“Older men did not associate health with underweight figures, including those with lower WHR.”

TLDR: Pedos are wrong. Underweight, waistless wonders are not attractive.

Study 2, rubbing salt in that fact.

Optimal Waist-to-Hip Ratios in Women Activate Neural Reward Centers in Men

Secondary sexual characteristics convey information about reproductive potential. In the same way that facial symmetry and masculinity, and shoulder-to-hip ratio convey information about reproductive/genetic quality in males, waist-to-hip-ratio (WHR) is a phenotypic cue to fertility, fecundity, neurodevelopmental resources in offspring, and overall health, and is indicative of “good genes” in women. Here, using fMRI, we found that males show activation in brain reward centers in response to naked female bodies when surgically altered to express an optimal (∼0.7) WHR with redistributed body fat, but relatively unaffected body mass index (BMI). Relative to presurgical bodies, brain activation to postsurgical bodies was observed in bilateral orbital frontal cortex. While changes in BMI only revealed activation in visual brain substrates, changes in WHR revealed activation in the anterior cingulate cortex, an area associated with reward processing and decision-making. When regressing ratings of attractiveness on brain activation, we observed activation in forebrain substrates, notably the nucleus accumbens, a forebrain nucleus highly involved in reward processes.

These findings suggest that an hourglass figure (i.e., an optimal WHR) activates brain centers that drive appetitive sociality/attention toward females that represent the highest-quality reproductive partners. This is the first description of a neural correlate implicating WHR as a putative honest biological signal of female reproductive viability and its effects on men’s neurological processing.


Study 3

Men report stronger attraction to femininity in women’s faces when their testosterone levels are high

Many studies have shown that women’s judgments of men’s attractiveness are affected by changes in levels of sex hormones. However, no studies have tested for associations between changes in levels of sex hormones and men’s judgments of women’s attractiveness. To investigate this issue, we compared men’s attractiveness judgments of feminized and masculinized women’s and men’s faces in test sessions where salivary testosterone was high and test sessions where salivary testosterone was relatively low.

This is why we need studies on men too.

Men reported stronger attraction to femininity in women’s faces in test sessions where salivary testosterone was high than in test sessions where salivary testosterone was low. This effect was found to be specific to judgments of opposite-sex faces. The strength of men’s reported attraction to femininity in men’s faces did not differ between high and low testosterone test sessions, suggesting that the effect of testosterone that we observed for judgments of women’s faces was not due to a general response bias. Collectively, these findings suggest that changes in testosterone levels contribute to the strength of men’s reported attraction to femininity in women’s faces and complement previous findings showing that testosterone modulates men’s interest in sexual stimuli.

Study 4

Beauty is in the eye of the plastic surgeon: Waist–hip ratio (WHR) and women’s attractiveness

Attractiveness conveys reliable information about a woman’s age, health, and fertility. Body fat distribution, as measured by waist-to-hip ratio (WHR), is a reliable cue to a woman’s age, health, and fertility, and affects judgment of women’s attractiveness. WHR is positively correlated with overall body weight or body mass index (BMI). Some researchers have argued that BMI, rather than WHR, affects judgments of female attractiveness. To evaluate the role of WHR, independent of BMI, we secured photographs of pre- and post-operative women who have undergone micro-fat grafting surgery. In this surgery, surgeons harvest fat tissue from the waist region and implant it on the buttocks. Post-operatively, all women have a lower WHR but some gain weight whereas others lose body weight. Results indicate that participants judge post-operative photographs as more attractive than pre-operative photographs, independent of post-operative changes in body weight or BMI. These results indicate that WHR is a key feature of women’s attractiveness.


Let’s look historically. Study 5

Trends in waist-to-hip ratio and its determinants in adults in Finland from 1987 to 1997

Background: Although abdominal obesity has been shown to be an important risk factor for cardiovascular disease and a variety of other diseases, secular changes in fat distribution in populations have rarely been documented.

Objective: Our objective was to assess trends in waist-to-hip ratio (WHR) in the Finnish population during a 10-y period. In addition, we investigated the associations of WHR with body mass index (BMI), age, education, and lifestyle factors.

Design: Three independent cross-sectional surveys were carried out at 5-y intervals between 1987 and 1997. Altogether, 15096 randomly selected men and women aged 25–64 y participated in these surveys.

Results: The WHR increased in both men and women during the 10-y period (P< 0.0001). In men, the strongest upward trend took place in the first 5-y period and then seemed to plateau; in women, the WHR continued to increase into the 1990s. In both sexes, the most prominent increase was observed in subjects aged ≥45 y. The WHR increased in all education-level groups, the lowest WHR being among those with the highest education. Age (18% in men, 12% in women) and BMI (33% in men, 25% in women) accounted for most of the variation in WHR, whereas only 3% was explained by education and lifestyle factors.

Conclusions: Abdominal obesity is a growing problem in Finland, especially in persons aged ≥45 y. These adverse changes in body shape continued to take place, particularly in women, in the 1990s.

Something in the food?

More history, prehistoric. Study 6

Preferred Women’s Waist-to-Hip Ratio Variation over the Last 2,500 Years

The ratio between the body circumference at the waist and the hips (or WHR) is a secondary sexual trait that is unique to humans and is well known to influence men’s mate preferences. Because a woman’s WHR also provides information about her age, health and fertility, men’s preference concerning this physical feature may possibly be a cognitive adaptation selected in the human lineage. However, it is unclear whether the preferred WHR in western countries reflects a universal ideal, as geographic variation in non-western areas has been found, and discordances about its temporal consistency remain in the literature. We analyzed the WHR of women considered as ideally beautiful who were depicted in western artworks from 500 BCE to the present. These vestiges of the past feminine ideal were then compared to more recent symbols of beauty: Playboy models and winners of several Miss pageants from 1920 to 2014. We found that the ideal WHR has changed over time in western societies: it was constant during almost a millennium in antiquity (from 500 BCE to 400 CE) and has decreased from the 15th century to the present. Then, based on Playboy models and Miss pageants winners, this decrease appears to slow down or even reverse during the second half of the 20th century. The universality of an ideal WHR is thus challenged, and historical changes in western societies could have caused these variations in men’s preferences. The potential adaptive explanations for these results are discussed.

Should’ve controlled for race.

Why not look at male WHR? Plus sperm health? Found:

  • The volume of ejaculate decreases in a linear fashion with increasing BMI (suggesting an inverse relationship).
  • The sperm quality and viability declines with increasing waist circumference.
  • Investigators also discovered that quality of semen decreases (such as sperm viability, motility, semen volume) with increasing body size; however, no relationship was observed between sperm DNA fragmentation index and physical activity or obesity.

Latter requires time.

Various research and clinical studies suggests that subfertility in men is multifactorial i.e. several factors can impact the quality of reproductive health.

  • Abnormal sperm production: Study conducted by Jensen and associates (2) suggested that abnormal BMI is very strongly linked to impaired sperm production. One of the many reasons is, abnormal metabolism of testosterone (which plays a key role in the production of healthy and viable sperms).
  • Abdominal obesity and risk of metabolic disorders: According to a new study reported in the Human Reproduction (3), investigators provided statistical evidence that abnormal BMI and abdominal obesity is very strongly linked to a number of health issues (such as cardiovascular dysfunction, atherosclerosis, type 2 diabetes, hypertension and others). Needless to say that these health issues have a deleterious effect on the sexual health regardless of the body-mass index (or BMI).
  • Obesity, physical activity and testosterone: Testosterone levels tends to decline in males who have a sedentary lifestyle. Various research and clinical studies indicates that aerobic activity or exercise can improve testosterone metabolism in males significantly.

1. Eisenberg, M. L., Kim, S., Chen, Z., Sundaram, R., Schisterman, E. F., & Louis, G. M. B. (2014). The relationship between male BMI and waist circumference on semen quality: data from the LIFE study. Human Reproduction, 29(2), 193-200.

2. Jensen, T. K., Andersson, A. M., Jørgensen, N., Andersen, A. G., Carlsen, E., & Skakkebæk, N. E. (2004). Body mass index in relation to semen quality and reproductive hormones among 1,558 Danish men. Fertility and sterility, 82(4), 863-870.

3. Hammiche, F., Laven, J. S., Twigt, J. M., Boellaard, W. P., Steegers, E. A., & Steegers-Theunissen, R. P. (2012). Body mass index and central adiposity are associated with sperm quality in men of subfertile couples. Human reproduction, 27(8), 2365-2372.

Yet they don’t tell men this information.

Back to women

Cross-cultural variation in men’s preference for sexual dimorphism in women’s faces.

Both attractiveness judgements and mate preferences vary considerably cross-culturally.


We investigated whether men’s preference for femininity in women’s faces varies between 28 countries with diverse health conditions by analysing responses of 1972 heterosexual participants. Although men in all countries preferred feminized over masculinized female faces, we found substantial differences between countries in the magnitude of men’s preferences. Using an average femininity preference for each country, we found men’s facial femininity preferences correlated positively with the health of the nation, which explained 50.4% of the variation among countries. The weakest preferences for femininity were found in Nepal and strongest in Japan. As high femininity in women is associated with lower success in competition for resources and lower dominance, it is possible that in harsher environments, men prefer cues to resource holding potential over high fecundity.

Asia is weird for dimorphism studies.

Hence the focus on health.

While the economy is bad, it isn’t surprising men prefer manly looking women.

It’s temporary. There’ll be a flood of divorces as the economy improves. Men will suddenly see how mannish the wife has been and be repulsed. Menopause also makes women look more mannish, including higher WHR. So much for a youth argument there.

Click to access nihms827194.pdf

Factors Underlying the Temporal Increase in Maternal Mortality in the United States

They don’t say more non-white mothers or more mixed race babies, so it’s wrong. They guess.

Female fertility doesn’t encourage cheating

Moving on to correct an old, old error. I’m sick of seeing it. I went out of my way to find the citation to do this.

There’s one study still going round, being cited, that is absolute bull.

No, women aren’t likelier to cheat when fertile. What sort of Lilith shit is this?

Quite the opposite.

Click to access EHB.pdf

It’s been debunked, for many years.

If you can’t read/understand beyond the abstract.

“Women’s desire for their own partners did not differ significantly between high and low fertility sessions”.

This happened when they actually tested what was only suggested (to get published) in the previous study, that has never been replicated.

If anything high fertility makes her more interested in her mate, which matches literally all the evobio on the topic, including Trivers and the fact that overwhelmingly most fathers are the biological fathers of their children. The exception is a forced match (arranged marriage) to an inferior male, see last post on ugly men. Sexual selection will occur in women, before or after nuptials, get used to it. Naturally, men denying that looks have anything to do with sexual attraction (LOL) will continue to ignore the most important variable in Darwinian theories – your genes.

Where you can go back to Peterson’s bollocks and choke on all his little blue pills.