Covid spike protein damages heart paper

Water is also wet.

https://www.biorxiv.org/content/10.1101/2020.12.21.423721v1

The SARS-CoV-2 spike protein disrupts the cooperative function of human cardiac pericytes – endothelial cells through CD147 receptor-mediated signalling: a potential non-infective mechanism of COVID-19 microvascular disease

paper DECEMBER 2020

AKA they KNEW

(they knew and they pushed it on you – and your kids, soon!)

Findings We show, for the first time, that the recombinant S protein alone elicits functional alterations in cardiac PCs. This was documented as: (1) increased migration, (2) reduced ability to support EC network formation on Matrigel, (3) secretion of pro-inflammatory molecules typically involved in the cytokine storm, and (4) production of pro-apoptotic factors responsible for EC death. Furthermore, the S protein stimulates the phosphorylation/activation of the extracellular signal-regulated kinase 1/2 (ERK1/2) through the CD147 receptor, but not ACE2, in cardiac PCs.

Accordingly, the neutralization of CD147, using a blocking antibody, prevented the activation of ERK1/2 and partially rescued the PC function in the presence of the S protein.

SO do they permit this treatment to their loyal pets?

I think you’ll find the storms fully operational by the time flu season arrives. AKA Coof Coof Part Two. Satanist humour is killing everyone scared enough of death to get guinea pigged. Great way to sort wheat from chaff actually.

Interpretation Our findings suggest the new, intriguing hypothesis that the S protein may elicit vascular cell dysfunction, potentially amplifying, or perpetuating, the damage caused by the whole coronavirus. This mechanism may have clinical and therapeutic implication.

TECHNICALLY, EVERYONE DIES FROM HEART FAILURE.

TECHNICALLY.

which is the best kind of correct

allow me to un-memoryhole a meme

ANOTHER!

CBA, tbh
watch Black Books, I bid thee

Linoleic acid and heart issues

So well-researched I couldn’t do better myself.

Even used the red box chan method. Nice.

FI
https://timesofindia.indiatimes.com/home/science/Vegetarians-prone-to-strokes-Study/articleshow/3227413.cms

A study that tried to find positive (pro-veg) differences had arbitrary, strict age limits and time exclusions.
https://academic.oup.com/ajcn/article/70/3/516s/4714974
“There were no significant differences between vegetarians and nonvegetarians in mortality from cerebrovascular disease, stomach cancer, colorectal cancer, lung cancer, breast cancer, prostate cancer, or all other causes combined.”
Logically, if meat consumption caused colon cancer, that is impossible.

Pro-vegan paper
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4073139/
“Non-vegetarian diets were compared to vegetarian dietary patterns (i.e., vegan and lacto-ovo-vegetarian) on selected health outcomes.”
Why not compare vegetarian to vegan?
“Males experience greater health benefits than females. Limited prospective data is available on vegetarian diets and body weight change.”
Don’t bother studying that, though.
“Large randomized intervention trials on the effects of vegetarian diet patterns on neurological and cognitive functions, obesity, diabetes, and other cardiovascular outcomes are warranted to make meaningful recommendations.”
…useless.

https://www.tandfonline.com/doi/abs/10.1080/10408398.2016.1138447?journalCode=bfsn20
Vegetarian, vegan diets and multiple health outcomes: A systematic review with meta-analysis of observational studies
There appears to be a confound e.g. not drinking.
Claim:
“This comprehensive meta-analysis reports a significant protective effect of a vegetarian diet versus the incidence and/or mortality from ischemic heart disease (−25%) and incidence from total cancer (−8%). Vegan diet conferred a significant reduced risk (−15%) of incidence from total cancer.”

DATA: (they hope we won’t look at)
“With regard to prospective cohort studies, the analysis showed a significant reduced risk of incidence and/or mortality from ischemic heart disease (RR 0.75; 95% CI, 0.68 to 0.82) and incidence of total cancer (RR 0.92; 95% CI 0.87 to 0.98) but not of total cardiovascular and cerebrovascular diseases, all-cause mortality and mortality from cancer. No significant association was evidenced when specific types of cancer were analyzed.”
Clear as mud.

You’re literally more likely to DIE on this diet but they claim “protective”.

Vegans – putting the DIE in DIET.

“The analysis conducted among vegans reported significant association with the risk of incidence from total cancer (RR 0.85; 95% CI, 0.75 to 0.95), despite obtained only in a limited number of studies.

Click to access e24d13bacf38f3ddc6178b07312d566a4d8d.pdf


excellent example of propaganda

Insulin > Heart attack connection

Click to access Article_with_review_WMC00851.pdf

https://health.harvard.edu/heart-health/takotsubo-cardiomyopathy-broken-heart-syndrome

Years of gender-based research have shown that in matters of the heart, sex differences abound. One striking example is the temporary heart condition known as takotsubo cardiomyopathy, first described in 1990 in Japan. More than 90% of reported cases are in women ages 58 to 75. Research suggests that up to 5% of women evaluated for a heart attack actually have this disorder, which has only recently been reported in the United States and may go largely unrecognized. Fortunately, most people recover rapidly with no long-term heart damage.”

“Takotsubo cardiomyopathy is a weakening of the left ventricle, the heart’s main pumping chamber, usually as the result of severe emotional or physical stress, such as a sudden illness, the loss of a loved one, a serious accident, or a natural disaster such as an earthquake.”

What if you induce it in a man?

And who isn’t “stressed”? Gangstalking rationale?

And that’d look nice and natural on a death certificate.

The precise cause isn’t known, but experts think that surging stress hormones (for example, adrenaline) essentially “stun” the heart, triggering changes in heart muscle cells or coronary blood vessels (or both) that prevent the left ventricle from contracting effectively. Researchers suspect that older women are more vulnerable because of reduced levels of estrogen after menopause. In studies with rats whose ovaries had been removed, the ones given estrogen while under stress had less left-ventricle dysfunction and higher levels of certain heart-protective substances.

That might be the single reason women live longer.

Takotsubo symptoms are indistinguishable from those of a heart attack. And an electrocardiogram (ECG) may show abnormalities similar to those found in some heart attacks — in particular, changes known as ST-segment elevation. Consequently, imaging studies and other measures are needed to rule out a heart attack.

Stress proteins and heart disease

https://www.researchgate.net/publication/21348724_Stress_proteins_and_cardiovascular_disease

Heart problems are basically the indicator of male mortality, mind.

Type A men

stop getting your anime panties in a bunch?

Wrath kills.

It explains why married men live longer. We tell you to slow your fucking roll.

In addition, members of this family have essential roles in protein processing and assembly of macromolecular complexes, and in regulation of gene expression, even in unstressed cells. 

Because a day of rest is for weaklings, right?

BTW yes this is the epigenetic trigger for stressed population models.
The out-of-touch politicians eat like Kings. Champagne socialists?
What happened to the French again?

Don’t comfort eat either because you tell yourself you’re stressed and deserve it.

Drop the starvation pattern, guys, you are not competing on ANTM.

Stress is biochemical!

https://www.researchgate.net/publication/258213031_Food_restriction_by_intermittent_fasting_induces_diabetes_and_obesity_and_aggravates_spontaneous_atherosclerosis_development_in_hypercholesterolaemic_mice

Contrary to our expectation, epididymal and carcass fat depots and adipocyte size were significantly enlarged by 15, 72 and 68 %, respectively, in the IF mice compared with the ad libitum-fed mice. Accordingly, plasma levels of leptin were 50 % higher in the IF mice than in the ad libitum-fed mice. In addition, the IF mice showed increased plasma levels of total cholesterol (37 %), VLDL-cholesterol (195 %) and LDL-cholesterol (50 %). 

Science.

Ask any rich woman about cortisol fat, this is not news to us. Higher testosterone makes this problem worse. Have you seen the way manly women expand once they hit menopause?
Manly – by build and bone structure. Not height or race. Does she have a waist or is she just skinny? Does she have bone structure or a manjaw? Prime example – Jolie. What does she do? Starve herself to stay thin, stressing out her body more. It’s a cycle.

Have you looked up leptin?

Obese people have more leptin, it suppresses hunger. The fact you don’t feel hungry starving is a red flag. Your body’s signals are wrong. Women feel full sooner, because we’re meant to have a higher body fat percentage and therefore more leptin is produced.

Soy is a chick food

http://www.sciencedaily.com/releases/2014/07/140730043439.htm

Need I mention phytoestrogens?

Lifelong soy consumption, similar to the diet of women in Asia, produces the least atherosclerosis. Switching to a Western diet after menopause, similar to Asian migrants to North America, leads to just as much atherosclerosis as a lifelong Western diet, and switching to soy from a Western diet after menopause helps only if there isn’t much atherosclerosis already.