Do trannies have copper toxicity?

(gammas too)

More estrogen (inc xenoestrogen) is stored in the body, along with a build-up of copper. These can produce psychiatric symptoms.
Maybe this is our version of Rome’s lead problem.
So glad I never touched those hormonal things.

Here is a short list of psychiatric symptoms and traits associated with copper overload:

Hyperactivity, academic underachievement, learning disabilities, ADHD, autism, skin sensitivity to tags in shirts or rough fabrics, intolerance to estrogen and birth control pills, onset during puberty, pregnancy or menopause, white spots on fingernails, skin intolerance to cheap metals, emotional meltdowns and frequent anger, ringing in ears, sensitivity to food dyes and shellfish, high anxiety, depression, poor immune function, sleep problems, poor concentration and focus, low dopamine activity, and elevated activity of norepinephrine and adrenaline.

“I have anxiety” sure Dave

Other medical conditions associated with copper overload include acne, allergies, Candida overgrowth, hypothyroidism, anemia, hair loss, chronic fatigue and fibromyalgia, migraines and male infertility.

Fatigue, hair loss, MALE infertility.

It can do this to a female body, imagine what it does to the male?

I’d sue, if I were them. The consent was uninformed.

The reason that copper is linked with such a long and varied list of conditions is that it is absolutely essential to the proper functioning of the immune system, the endocrine system, and the nervous system.

Copper is important for regulating the synthesis of neurotransmitters that mediate psychiatric symptoms. It is a co-factor in the chemical reaction that converts dopamine to norepinephrine. When copper levels are high, more norepinephrine and epinephrine (adrenaline) are synthesized from dopamine, which can causes feelings of agitation, anxiety and panic, overstimulation, racing thoughts, restlessness, and insomnia. In other words, it has an amphetamine-like effect, revving the nervous system into a state of overdrive. Consider that copper is often used in electrical wires because it conducts electricity well, and likewise increases nerve transmission, which is an electrical chemical process.

transduction, but ok

it’d be piss-easy to study, hair mineral analysis before meds, and after

Copper is also central to cellular energy production, and thus many patients with fibromyalgia and chronic fatigue conditions related to mitochondrial dysfunction have disorders of copper metabolism.

it all fits!

Copper overload is particularly common in women.

not anymore they just don’t check men

Estrogen can cause copper retention and accumulation, which can eventually result in toxicity. Hormonal events such as menarche, pregnancy or menopause can trigger it. These days when a patient tells me about a history of postpartum depression, severe PMS, dysmenorrhea or adverse effects related to the prescription of oral contraceptives, I immediately suspect copper overload.

Copper promotes the formation of blood vessels (angiogenesis) and when copper levels are elevated, it can predispose an individual to endometriosis and fibroid tumors, as well as increase the blood supply to other types of tumors. Excess copper can accumulate in the liver and impair its capacity for detoxification, which can result in chemical sensitivities.

I wonder if carnivores can get that?

I know it’s used in make-up a lot, as a colourant too.

Copper is carried in the blood by a specific protein called ceruloplasmin. Some patients have low levels of ceruloplasmin and thus have a large percentage of unbound copper in their blood. Unbound copper causes oxidative stress in the body. Oxidative stress is characterized by the presence of free radicals which interact with molecules in the body, damaging various cell components such as DNA, protein and lipids, and giving rise to various disease states, including autoimmune disorders, neurodegenerative disorders, and cancers, to name a few.

so it’s like poison to men, is what I’m getting

There is commonly an inverse relationship between zinc and copper in the body. Often when a patient has elevated copper, the zinc level is low. Zinc is another mineral essential to cellular function, regulation of the immune system, wound healing, and synthesis of neurotransmitters. An important ingredient in the treatment of copper overload is supplementation with zinc. This must be done very slowly and carefully, because zinc mobilizes copper stores. During this process, a person can initially feel even more anxious and symptomatic. Anti-oxidants are also used in the treatment of copper toxicity, as well as the elements molybdenum and manganese, and amino acids which promote metallothionein production. Metallothionein is another protein which binds heavy metals in the blood, and which is important for regulation of zinc and copper metabolism. It’s important to find a trained practitioner to help you with this process. A good resource is the Practitioner’s Page of the Walsh Research Institute website.

Birth Control: Copper Toxicity & Estrogen Excess

Copper toxicity is a hidden epidemic. Birth control pills and copper IUD’s contribute to excess copper in the body. For thousands of years, copper has been known to be anti-microbial in nature. Copper IUD’s work by releasing small but significant amounts of copper into the uterus. Copper immobilizes sperm as it travels to the fallopian tubes. This copper can and will enter into the blood causing all sorts of problems.

Birth control pills also tend to raise copper levels in the body. High levels of copper destroys vitamin C in the body, can deplete zinc levels, can lower iron, can cause an unusual rise in Vitamin A, and can aggravate B-vitamin metabolism. Birth control pills contain estrogen and progestin, and these powerful hormones in birth control pills turn off a woman’s ovulatory cycle. Estrogen and copper are succinctly related. Copper tends to raise estrogen in the body, and estrogen tends to cause copper to rise. Both copper and estrogen tend to feed one another.

and they give these to MINORS

but smarties have too much sugar.

There are long term consequences of both copper toxicity and excess estrogen. High levels of copper can cause numerous symptoms ranging from migraines, to PMS, chronic fatigue and allergic reactions. Copper is used in the body to produce ceruloplasmin, the major copper carrying protein, which is involved in iron utilization and the formation of hemoglobin.

Excess levels of ceruloplasmin is found among those with OCD, Schizophrenia, Angina, Alzheimer’s, Rheumatoid Arthritis and Lymphoma.

Dangers Associated with Estrogen
Copper raises estrogen and vice versa. Estrogen is touted as a therapy for numerous medical conditions such as Osteoporosis, but this mass media hype fails to identify that estrogen is stress-promoting on bones and is age-promoting. Numerous studies have shown that estrogen can produce prolactin and that prolactin can cause osteoporosis.

In short, high estrogen and low progesterone increases bone loss. Osteoarthritis is associated with excess estrogen. It is true that estrogen can cause retention of calcium. But it is now known that high levels of calcium does very little to prevent or improve symptoms of osteoporosis. Osteoporosis is not bone loss, but rather the breakdown of the collagen matrix that holds bone together, a degenerative and catabolic condition which consists of deficiencies of minerals such as boron, magnesium and phosphorous. Only if the miracle estrogen or calcium was the answer to this and other degenerative diseases. They are promoted as such but all that calcium will just end up in the toilet, and all that estrogen will displace other hormones and nutrients, causing further complications.

soil fertility issue

Could also give them cancer

Iodine and IQ

aka most of the Thing behind the Flynn effect.

Vegans are deficient.

We assessed the association between maternal iodine status and child IQ at age 8 years and reading ability at age 9 years. We included 21 socioeconomic, parental, and child factors as confounders.

Our results show the importance of adequate iodine status during early gestation and emphasise the risk that iodine deficiency can pose to the developing infant, even in a country classified as only mildly iodine deficient. Iodine deficiency in pregnant women in the UK should be treated as an important public health issue that needs attention.

After adjustment for confounders, children of women with an iodine-to-creatinine ratio of less than 150 μg/g were more likely to have scores in the lowest quartile for verbal IQ (odds ratio 1·58, 95% CI 1·09–2·30; p=0·02), reading accuracy (1·69, 1·15–2·49; p=0·007), and reading comprehension (1·54, 1·06–2·23; p=0·02) than were those of mothers with ratios of 150 μg/g or more.


The present comments are restricted to the role of maternal thyroid hormone on early brain development, and are based mostly on information presently available for the human fetal brain. It emphasizes that maternal hypothyroxinemia – defined as thyroxine (T4) concentrations that are low for the stage of pregnancy – is potentially damaging for neurodevelopment of the fetus throughout pregnancy, but especially so before midgestation, as the mother is then the only source of T4 for the developing brain.

Despite a highly efficient uterine-placental ‘barrier’ to their transfer, very small amounts of T4 and triiodothyronine (T3) of maternal origin are present in the fetal compartment by 4 weeks after conception, with T4 increasing steadily thereafter. A major proportion of T4 in fetal fluids is not protein-bound: the ‘free’ T4 (FT4) available to fetal tissues is determined by the maternal serum T4, and reaches concentrations known to be of biological significance in adults. Despite very low T3 and ‘free’ T3 (FT3) in fetal fluids, the T3 generated locally from T4 in the cerebral cortex reaches adult concentrations by midgestation, and is partly bound to its nuclear receptor. Experimental results in the rat strongly support the conclusion that thyroid hormone is already required for normal corticogenesis very early in pregnancy. The first trimester surge of maternal FT4 is proposed as a biologically relevant event controlled by the conceptus to ensure its developing cerebral cortex is provided with the necessary amounts of substrate for the local generation of adequate amounts of T3 for binding to its nuclear receptor.

Don’t worry, the brain damage was genetic anyway.

Women unable to increase their production of T4 early in pregnancy would constitute a population at risk for neurological disabilities in their children. As mild-moderate iodine deficiency is still the most widespread cause of maternal hypothyroxinemia in Western societies, the birth of many children with learning disabilities may already be preventable by advising women to take iodine supplements as soon as pregnancy starts, or earlier if possible.


This editorial reviews the impact of iodine deficiency (1) on thyroid function in pregnant women and neonates and (2) on the neurointellectual development of infants and children.
All degrees of iodine deficiency (mild: iodine intake of 50-99 µg/day, moderate: 20-49 µg/day, and severe: <20 µg/day) affect thyroid function of the mother and the neonate as well as the mental development of the child. The damage increases with the degree of the deficiency, with overt endemic cretinism as the severest consequence. Maternal hypothyroxinaemia during early pregnancy is a key factor in the development of the neurological damage in the cretin. Selenium deficiency combined with iodine deficiency partly prevents the neurological damage but precipitates severe hypothyroidism in cretins.
Iodine deficiency results in a global loss of 10-15 IQ points at a population level and constitutes the world’s greatest single cause of preventable brain damage and mental retardation.

An entire Standard Deviation, no big deal!

Supplements won’t help, you’ll OD.

Iodine is a micronutrient that is essential for the production of thyroid hormones. The primary source of iodine is the diet via consumption of foods that have been fortified with iodine, including salt, dairy products and bread, or that are naturally abundant in the micronutrient, such as seafood. Recommended daily iodine intake is 150 μg in adults who are not pregnant or lactating. Ingestion of iodine or exposure above this threshold is generally well-tolerated. However, in certain susceptible individuals, including those with pre-existing thyroid disease, the elderly, fetuses and neonates, or patients with other risk factors, the risk of developing iodine-induced thyroid dysfunction might be increased. Hypothyroidism or hyperthyroidism as a result of supraphysiologic iodine exposure might be either subclinical or overt, and the source of the excess iodine might not be readily apparent.

The Great Iodine Debate


Iodine is critical to human health. It forms the basis of thyroid hormones and plays many other roles in human biochemistry. While the thyroid gland contains the body’s highest concentration of iodine, the salivary glands, brain, cerebrospinal fluid, gastric mucosea, breasts, ovaries and a part of the eye also concentrate iodine. In the brain, iodine is found in the choroid plexus, the area on the ventricles of the brain where cerebrospinal fluid (CSF) is produced, and in the substantia nigra, an area associated with Parkinson’s disease.

Iodine is essential to normal growth and development. Iodine deficiency in utero and during growth can result in cretinism, a condition of severely stunted physical and mental growth due to prolonged nutritional deficiency of iodine or from untreated congenital deficiency of thyroid hormones (hypothyroidism). The condition is characterized by short stature, delayed bone maturation and puberty, infertility, neurological impairment and cognitive impairment ranging from mild to severe. Iodine deficiency also causes goiter, the gradual enlargement of the thyroid gland. Both conditions have led to public health campaigns of iodine administration in many countries. The addition of iodine compounds to table salt or water represents the first attempt to provide nutrient supplementation via “fortification” of common foods.

At least they won’t have grandkids.

China paper here:

Click to access 32.pdf

also here

IQ so unreal it tracks brain damage.

The level of iodine nutrition plays a crucial role in the intellectual development of children. The intelligence damage of children exposed to severe ID was profound, demonstrated by 12.45 IQ points loss and they recovered 8.7 IQ points with iodine supplementation or IS before and during pregnancy. Iodine supplementation before and during pregnancy to women living in severe ID areas could prevent their children from intelligence deficit. This effect becomes evident in children born 3.5 years after the iodine supplementation program was introduced.

And now… we wait.

Food babies

Insight into vegan menopause.
“Drinking whole fat milk and eating ice cream appears to be better for women trying to become pregnant than a diet consisting of low-fat dairy products such as skim milk and yogurt, according to new research published in Human Reproduction journal. Researchers in the United States have found a link between a low-fat dairy diet and increased risk of anovulatory infertility.”
You don’t have to tell me twice to eat ice cream, mate.
“Further, lactose (the main carbohydrate in milk and dairy products) may not affect fertility within the usual range of intake levels in humans.”
Good to know.
“Consumption of iron supplements and nonheme iron from other sources may decrease the risk of ovulatory infertility.”
Yes, my favourite iron supplement is called steak. Recommend.
Especially with three times “too much” garlic butter.
“Folic acid appeared to explain part of the association between multivitamin supplement use and risk of ovulatory infertility.”
Sorry but I don’t trust Crunchy Nut cornflakes with the fate of my future children’s health.
“Dairy consumption was not significantly correlated with PCOS. However, after adjustment for confounders, there was an direct relationship between milk consumption and risk of PCOS.”

“the results showed that the intake amounts of Ca, Mg, D vitamin, dairy, fruits and nuts and seeds were remarkably low among the women with PCOS.”

What are they living on, Oreos?

“some evidence on the decreased amount of adiponectine, calcium, D vitamin in the patients suffering from PCOS with having a higher thyroglobulin”


“Numerous studies have demonstrated the association between the diet and its components and risk factors developing various diseases.[] However, previous studies did not address the relationship between nutrition choices and type of diet chosen by the patients.[]”
“According to existing data, women trying to achieve pregnancy are encouraged to increase consumption of whole grains, omega-3 fatty acids, fish, and soy and to reduce consumption of trans fats and red meat. In addition, a daily multivitamin that contains folic acid before and during pregnancy may not only prevent birth defects, but also improve the chance of achieving and maintaining a pregnancy. In contrast, there is limited evidence supporting an association betweenvitamin D and human fecundity outcomes despite promising evidence from nonhuman studies.
Yeah because that’s relevant.
Questions for future research included the roles of other types of fat (especially omega-6 and monounsaturated fats) and protein (especially white meat and seafood) on female fertility; particular attention should also be paid to exposure to environmental contaminants in foods. Although much work remains, this review accrued best available evidence to provide practical dietary recommendations for women trying to conceive.”
Bolded valuable parts.
“One approach to minimize the impact of anovulation on fertility is supplementation with progesterone during recruitment, selection and final stages of development of the preovulatory follicle. It is suggested that a minimum of 2.0 ng/mL of progesterone is needed during growth of the preovulatory follicle to achieve P/AI similar to that of cows growing the preovulatory follicle during diestrus.”
But one of the Pills is that….
Does taking that Pill increase odds of pregnancy?
Big if true.
The literature on the relationship between diet and human fertility has greatly expanded over the last decade, resulting in the identification of a few clear patterns. Intake of supplemental folic acid, particularly at doses higher than those recommended for the prevention of neural tube defects, has been consistently related to lower frequency of infertility, lower risk of pregnancy loss, and greater success in infertility treatment. On the other hand and despite promising evidence from animal models, vitamin D does not appear to exert an important role in human fertility in the absence of deficiency.
But most people are deficient….
That’s like saying you can stand outside normally -except when there’s a hurricane.
And there’s currently a hurricane.
Antioxidant supplementation does not appear to offer any benefits to women undergoing infertility treatment, but it appears to be beneficial when it is the male partner who is supplemented.
Reduce genetic load.
However, the available evidence does not allow discerning which specific antioxidants, or at which doses, are responsible for this benefit. Long-chain omega-3 fatty acids appear to improve female fertility, although it remains unclear to what extent contamination of shared food sources, such as fish with high levels of environmental toxicants, can dampen this benefit.
Eat this mercury (sushi fad) it’s good for your baby!
Lastly, adherence to healthy diets favoring seafood, poultry, whole grainsfruits, and vegetables are related to better fertility in women and better semen quality in men.
Could you be a little more specific?
You just named, like, most food.
They’re paid with our taxes, daylight robbery.
The cumulative evidence has also piled against popular hypotheses.
I doubt that.
Sounds like someone wants a juicy research grant.
Dairy and soy, once proposed as reproductive toxicants, have not been consistently related to poor fertility.
Because you refuse to test them separately.
If only a group ate one and not t’uther.
In fact, soy and soy supplements appear to exert a beneficial effect among women undergoing infertility treatment.
Many have low estrogen, next!
Giving people with scurvy vitamin C helps! Next!
Similarly, because data from large, high-quality studies continue to accumulate, the evidence of a potentially deleterious effect of moderate alcohol and caffeine intake on the ability to become pregnant seems less solid than it once did.
But potentially, if you can’t put down the coffee or wine, Barbara, maybe you shouldn’t be having kids? Same goes for men without the energy. Now. Without kids.
Why do narcs consider children a human right?
Sort your energy level out BEFORE kids, at least?
While a complete picture of the role of nutrition on fertility is far from complete, much progress has been made. The most salient gaps in the current evidence include jointly considering female and male diets and testing the most consistent findings in randomized trials.
Fuck, men influence pregnancy? What witchcraft is this?
Their only biological contribution is determined by things that occurred in the years before conception?
Say it isn’t so.
What term could possibly exist to describe this Darwinian fitness between men?
Hold men responsible for their bad habits affecting their innocent child’s health outcomes and literal lifespan and I might be impressed.
Posting will be patchy because of my feelings.

Video: Earth’s magnetic field

This is kinda more important than whatever is going on on the internet.

I don’t think strip-mining the magnetic ore in the earth really helps.

As the planet is approaching solar minimum, the global cooling (look at the reduction in sunspots and numerous crop failures, set to increase) mean that currently inhospitable dry lands might grow food.

This is why China is buying useless hot land in Africa.

Cooling causes Empire die-offs. The planet’s population will drop suddenly, over a few cycle epidemics. My money is on mutated flu. No medicine could stop it. Fruit is going to get more expensive, why do you think there’s an effort to grow it indoors under fake lights? Why do that experiment in a world that’s warming?

A lot of electronic malfunctions will mean this Victorian/Industrial wealth is fast coming to an end, since the fossil fuel problem is mitigated by technology.

There will be a standard living “crunch”, for everyone, it’s inevitable.

Subsistence living will make a comeback this century.

ADD, ADHD, ABCD – potassium deficiency?

I wouldn’t be surprised if that were the case.

ADD/ADHD/whatever they’re calling it now isn’t a real disorder. Attention doesn’t operate like other variables because everybody has it. You cannot have a deficit, unless you’re in a coma. If you put the average ADD kid in a forest, it’d disappear. Like magic. [yes I have tested that theory] Why? School is boring as shit [dumb teachers, unchallenged gifted trope] and bad parenting that would rather drug the poor kid up to the eyeballs than face a tough conversation once in a while about what a terrible job they’re doing and how to better support the kids’ needs. Notice the parents never require drugs with a long list of side effects, even though they say it’s genetic to feel less like the total scum they know deep down they are?

A woman who experienced this sensory overstimulation found that it subsided about 20 minutes after taking a potassium supplement. A neurologist found that her symptoms were the same as in “hypokalemic periodic paralysis,” where ion channels in the muscles become overactive when potassium levels are low.

K is a vital element in the body, especially the brain. Look up the sodium-potassium pump. [wordy explanation] [mathey explanation]
What caught my eye was this;

eating a high-carb diet and eating salty foods triggered her sensory overstimulation

Yes, that makes sense. Most salt is pure sodium, no trace minerals (the reason we crave it aside from simple electrolyte balance). There must be a BALANCE of Na AND K for the brain ‘pump’ messaging signals to operate effectively.
Practically speaking, anyone who eats too much salt and too little potassium is giving themselves brain damage. For those who didn’t read the “vital” link [tut tut]: “If the pump does not function properly in brain cells, the result is severe neurological conditions such as migraine with aura, muscle spasms or unilateral paralysis (hemiplegia).”

yup damon ian somerhalder vampire diaries hot duh yes uhuh
And you thought I was exaggerating.

n.b. To the nootropic crowd, upping K doses is a thing (esp. to cover dietary high-salt intake). You’re welcome.