Fake burger, real trans fats

Big Food has a lot to answer for.

DECODING LABELS: Beyond Meat Chicken

Higher consumption of unsaturated fats linked with lower mortality

Mmmmmm, bleeding vegetable oils.

Trans fats—on their way to being largely phased out of food—had the most significant adverse impact on health. Every 2% higher intake of trans fat was associated with a 16% higher chance of premature death during the study period.

That’s a big deal.


And cumulative.

Higher consumption of saturated fats was also linked with greater mortality risk. When compared with the same number of calories from carbohydrate, every 5% increase in saturated fat intake was associated with an 8% higher risk of overall mortality.

That’s practically insignificant proportionally, especially since the method painstakingly avoid citing animal source or non-animal sourced fats. Too vague.

Conversely, intake of high amounts of unsaturated fats—both polyunsaturated and monounsaturated—was associated with between 11% and 19% lower overall mortality compared with the same number of calories from carbohydrates.

You cannot compare to sick Americans as your control population. No.

Bad scientism.

They don’t want people learning how to be really healthy. It would show up on their data more. You need half a century of plant propaganda to convince people our declining health is good for us.

Among the polyunsaturated fats, both omega-6, found in most plant oils, and omega-3 fatty acids, found in fish and soy and canola oils, were associated with lower risk of premature death.

The Western diet is toxic purely because there’s too much omega 6, a deadly excess.

The lifespan of India also suggests avoiding animal products is…. ill-advised.

People who replaced saturated fats with carbohydrates had only slightly lower mortality risk.

eat your GMO grains, you’ll live longer

sounds legit

“Our study shows the importance of eliminating trans fat and replacing saturated fat with unsaturated fats, including both omega-6 and omega-3 polyunsaturated fatty acids. In practice, this can be achieved by replacing animal fats with a variety of liquid vegetable oils,” said senior author Frank Hu, professor of nutrition and epidemiology at Harvard Chan School and professor of medicine at Harvard Medical School.

An Asian at Chan school.

Looking as Asia’s meat consumption, you are lying.

Chinese meat imports to expand in 2019: https://apps.fas.usda.gov/psdonline/circulars/livestock_poultry.pdf

Despite global promises to reduce over the past few years.

Maybe I’ll do a whole post.

The only confirmed finding is lab grown shit is toxic.

This Is Your Brain On Trans Fats, Artificial Sweeteners And Alcohol

Trans Fats

As of June 18 of this year, trans fats have been officially banned from all manufactured foods.


Research in the 1990s found a strong link between trans fats, also known as “partially hydrogenated oil,” and heart disease.

You are what you eat.

In addition, more and more studies are finding that trans fats cause cellular destruction in the brain and can adversely impact hormone production, memory and increase inflammation by suppressing the production of Omega-3 fatty acids.

Actively suppresses omega 3.

Now, our brains do need fat to function. In fact, the brain relies on natural, healthy fats to maintain cell membranes, keep brain inflammation at bay and transport fat soluble vitamins such as vitamin A, D, E and K. Focus on getting your daily doses of fat from olive oil, coconut oil, nuts, seeds and avocado.

A study of almost 3000 people in 2017 reported a link between artificial sugar consumption and an increased risk of stroke and dementia. In the study, those participants that reported having just one diet beverage a day were three times more likely to develop dementia or suffer from a stroke. Although more research is needed, the findings are alarming.

Aspartame, one of the most commonly used artificial sweeteners in foods and beverages, has also been shown to cause neurodegeneration, mood imbalances and potentially cause brain cells to kill themselves. Instead of artificial sweeteners, use small amounts of natural sugar, such as raw honey, pure maple syrup or dates.

Yeah it also tastes like death.

A literal neurotoxin.

A study of 550 men and women over 30 years published in The BMJ found that even moderate drinking, which they defined as about 6-9 drinks a week, can damage the brain, including hippocampal atrophy.

Anyone that has had a few drinks knows that alcohol can impair judgement, balance, speech and even memory. Many studies have shown that long term heavy drinking can create serious and persistent changes in brain function that include mental confusion, eye nerve paralysis and difficulty with muscle coordination.

Drunkenness is just a form of brain damage that becomes more persistent with chronic alcoholism.

Until they never ‘sober up’ – clear the toxin, ever again.

Let the brain shrinkage vegans eat their fake flesh.


Trans fatty acids (TFA) are produced either by hydrogenation of unsaturated oils or by biohydrogenation in the stomach of ruminant animals. Vanaspati ghee and margarine have high contents of TFA. A number of studies have shown an association of TFA consumption and increased risk of cardiovascular disease (CVD). This increased risk is because TFA increase the ratio of LDL cholesterol to HDL cholesterol. Food and Agriculture Organization of the United Nations and World Health Organization have come up with the recommendation that the contents of TFA in human dietary fat should be reduced to less than 4%. There is high prevalence of CVD in Pakistan. High consumption of vanaspati ghee which contains 14.2-34.3% of TFA could be one of the factors for this increased burden of CVD in Pakistan. Consumption of dietary fat low in TFA would be helpful in reducing the risk of CVD in South Asia. Denmark by banning the sale of food items with TFA has brought down the number of deaths due to coronary heart disease by nearly 50% over a period of 20 years. Public awareness about the adverse effects of TFA on human health would be extremely important. Media can play a very effective role in educating the masses and advocating the policy for the sale of only low TFA food items.

It merits legal banning.


 A third pattern characterized by high trans fat was associated with less favorable cognitive function and less total cerebral brain volume.

Participants with higher plasma trans fat scores had worse cognitive function overall (memory, attention, language, processing speed, and global).

Each 1-SD increase in the trans fat score associated with a 0.30-SD decrease in global cognitive score.

You sure showed us!

They’re literally retarding themselves.

The high trans fat pattern was consistently associated with worse cognitive performance and less TCBV. Linolelaidic acid is predominantly found in bakery foods such as cookies, doughnuts, cakes, pastries, and pies.

I’d rather eat a donut than fake oozing flesh.

These foods are often prepared with hydrogenated vegetable oils to allow for a long shelf life. Higher trans fatty acid intake increases cardiovascular risk, systemic inflammation, and endothelial dysfunction, all of which may explain an association with cognition., Unfortunately, very few studies have assessed trans fat and risk for cognitive decline.

Funny that.

Big Food funding PACs?

Simple, scan vegan brains and see if they’re atrophied.

Trans fat may aggravate cognitive function independently and jointly through interaction with other dietary factors.e11 Trans fat may displace DHA in neuronal membranes, but apparently does not impact the neuropathologic Alzheimer hallmarks in mice.The consistency of the association of plasma trans fat with poorer cognitive function and more brain atrophy suggests neurologic consequences in humans, but these findings need to be confirmed.


5 Reasons Vegetable Oil is Worse Than Sugar

The European Food Safety Authority issued a report last year warning consumers about the high levels of potentially carcinogenic contaminants in palm oil (an ingredient used in Kit Kat Bars, Nutella, and Cheez-Its, among other treats) when refined at temperatures above 392°F.



With caveats inherent for ecologic, nutrient disappearance analyses, a healthy dietary allowance for n-3 LCFAs for current US diets was estimated at 3.5 g/d for a 2000-kcal diet. This allowance for n-3 LCFAs can likely be reduced to one-tenth of that amount by consuming fewer n-6 fats.


It’ll cost more in hospital bills.

Eat your aluminium, thots

That’s right, smear on a lipstick full of shimmery bullshit. Including lead (and wonder why girls are depressed). [Literally eating lead.]

The Victorians ate arsenic but this is worse. Arsenic temporarily improved the complexion.

Using the term ‘mica’ for what’s essentially powdered foil suggests women wouldn’t want it if they knew what it was.

It isn’t like there’s a connection between dissected and tested Alzheimer’s plaques and aluminium deposits, is there?

Why are the Boomers raised on cans of this stuff seeing an explosion in Alzheimer’s rates?

As always, at first, go to the ‘officials’. Find out the Party Line.

Although aluminium has been seen in amyloid plaques”


“there is no solid evidence that aluminium is increased in the brains of people with Alzheimer’s disease.”
Whew, what cognitive dissonance.
It’s literally forming those plaques that ARE the disease but nbd?

Solid evidence – like a solid chemical analysis of solid plaques in the solid brains of solid dead people with Alzheimer’s?

Look at the hustle to move those goalposts.

“Because some people with the disease had aluminum deposits in their brains, it was thought that there was a direct connection…”
Well… yeah.
That’s proof.
There’s no negative evidence possible for that to stop being proof.

It’s literally in their brains.

“However, after many years of study, no conclusive evidence links aluminum to neurodegenerative disease”
Bullshit, 2007.

I detest scientism. Look at these papers and tell me what’s ‘conclusive’.

(Apart from donations of mega-corps to Super PACs to prevent this mega-lawsuit).

“A multi-institutional team of researchers has defined for the first time how metal ions bind to amyloid fibrils in the brain in a way that appears toxic to neurons. Amyloid fibrils are linked to the development of neurodegenerative diseases such as Alzheimer’s, Parkinson’s and Creutzfeldt-Jakob. Although metal ions, most notably copper, can bind to amyloid in several specific ways, the researchers found that only one way appears toxic.”

But how, ‘critics’ argue, could this possibly occur? What’s the mechanism? Nobody has proven a mechanism….?


“Link between Aluminum and the Pathogenesis of Alzheimer’s Disease: The Integration of the Aluminum and Amyloid Cascade Hypotheses”


“In particular, the link between aluminum and Alzheimer’s disease has been the subject of scientific debate for several decades. However, the complex characteristics of aluminum bioavailability make it difficult to evaluate its toxicity and therefore, the relationship remains to be established.”

Uhuh. Isn’t that your job?

If it’s impossible, why are you getting paid?

“On the contrary, aluminum is a widely recognized neurotoxin that inhibits more than 200 biologically important functions and causes various adverse effects in plants, animals, and humans.”


Mounting evidence has suggested that significance of oligomerization of β-amyloid protein and neurotoxicity in the molecular mechanism of AD pathogenesis. Aluminum may play crucial roles as a cross-linker in β-amyloid oligomerization.

Here, we review the detailed characteristics of aluminum neurotoxicity based on our own studies and the recent literatures. Our aim is to revisit the link between aluminum and AD and to integrate aluminum and amyloid cascade hypotheses in the context of β-amyloid oligomerization and the interactions with other metals.”

But HOW could this POSSIBLY operate? – intellectually dishonest douches.

Note the prestige of journal. International Journal of Alzheimer’s Disease, those hacks.

But on a MOLECULAR level…. – douchecanoes.

“Quantification of the Aβ peptide in Alzheimer’s plaques by laser dissection microscopy combined with mass spectrometry”

It’s behind a paywall but it’s there. Check the academic pirate bay.

It’s been known for a while.
2000: “Aluminum has been identified as a neurotoxin for over 100 years.”

So stop using it in food, cosmetics, deodorant, cookware and cans?

Their use actually came in well after it was a known neurotoxin. Because that isn’t dodgy.

Reference for that sentence:

Doelken P:Naunynschmiedeberger. Arch Exp Pathol Pharmakol 40: 58-120 cited by Crapper McLachlan DR., Lukiw WJ, Kruck TPA, Aluminum, altered transcription, and the pathogenesis of Alzheimer’s disease. Environ Geochem Health, 1990; 12(1-2): 103-114.

Link for that: https://link.springer.com/article/10.1007/BF01734059

Opening paragraph.

“The etiology of some, if not all, cases of Alzheimer’s disease is linked to a mutation in the proximal portion of the long arm of chromosome 21∶21q11.2 → 21q22.2. While the functional consequences of the mutation are unknown, we speculate that one consequence of the mutation is loss of the natural barriers and intracellular ligands for aluminum. As a result, aluminum gains access to several brain sites including the nuclear compartment in certain neurons of the central nervous system.”

I know both my shit and my bullshit.

1990. Who owns the aluminium? That’d be a fun tour of genocide.

1992 laser study, note prestige of journal.

What the fuck do they know, right, reddit?

“Selective accumulation of aluminum and iron in the neurofibrillary tangles of Alzheimer’s disease: A laser microprobe (LAMMA) study”

“In addition, probe sites directed to neurons identified in snapfrozen cryostat sections from 2 subjects with Alzheimer’s disease revealed similar spectra with prominent aluminum‐related peaks, confirming that our findings are not related to exogenous contamination through fixation, embedding, or other procedures prior to analysis. This study further confirms the association of aluminum and neurofibrillary tangle formation in Alzheimer’s disease.”

But I guess I’m just scared of non-stick pans, where’s the evidence?

Nerve damage in white males

Coincidental, just don’t look around.


“The aim of this study was to investigate the effects of squalene on the central and peripheral nervous systems in rat at the ultrastructural level. Squalene was administered at a dose of 20 g/kg body weight, once daily for 4 days, and the animals were sacrificed 7 days and 30 days after the initiation of the experiment. After 7 days a mild swelling of mitochondria and dilation of the Golgi complex cisterns in few neurons in the cerebral cortex and hippocampus were observed. The swelling of astrocytes and their processes was also seen. Some myelin sheaths in the cerebral white matter were disintegrated. In the peripheral nervous system (the sciatic nerve), a damage of the Schwann cells, a destruction of the myelin sheaths, and lipid-like deposits between myelin lamellae causing a secondary compression of axons were present.”

Mercury also destroys myelin sheaths. However, it is eventually cleared from the body by chelating factors in the diet, with the exception of repeat doses e.g. from amalgam.
Why are modern boys slow in school, weirdly dulled and heavy addicts of social media, porn etc.?

Squalene administration caused a stimulation of fibroblast to synthesize collagen and an activation of macrophages in the perineurium.”

The collagen bit makes it fine topically for vain women (including moi).

“After 30 days, the lipid-like material was present in some neurons as well as in the myelin sheaths in the central nervous system. Endothelial cells were hypertrophic and a few demonstrated features of apoptosis.”

I find it odd a famous billionaire magically had a son who “grew out of” autism…

Italians supposedly tested a vaccine in December and found it …didn’t contain a vaccine. At all.
So technically, the things labelled and given as ‘vaccines’ cause issues.

What’s being called vaccinegate is this mystery mix death potion sold and forced on children as “vaccines”.

“Vaccinegate: Initial results on Infanrix Hexa chemical composition”

Click to access ob_d9ff7d_corelva-rapport-composition-vaccin-in.pdf

Almost like genetic targeting, but you’d need enough dumb ones submitting free samples for “research” in the T&Cs to make that a bioweapon.
You’d also need to own the labs and pharmaceutical companies. Do they use squalene?
https://www.cdc.gov/flu/protect/vaccine/adjuvant.htm See end.
Male genes are easier to target than female but the rumour is that the three-part tetanus (no such thing, it’s one-shot) is intended to prevent pregnancy.

In Infanrix Hexa we found
● chemical contamination from the manufacturing process or cross-contamination with other manufacturing lines;
● chemical toxins;
● bacterial peptide toxins;
● insoluble and indigestible macromolecule that reacts to the protein assay, but cannot be recognized by any protein databases.

We have not found:
● Protein antigens of diphtheria toxoids, tetanus, pertussis, hepatitis B, haemophylus influenzae B, Poliomyelitis 1-2-3;
● Formaldehyde and glutaraldehyde, phenoxyethanol, antibiotic residues indicated in the composition;

Autism symptoms is not autism, polio symptoms is not polio.

Note the wording.



I may be George Soros in a wig. Long odds but…. maybe.

Would there happen to be perineurium damage?


I fucking bet they are. Bet you squalene antibodies in their bloodstream, easily resolved with another injection for the immune system to attack said antibodies.

They won’t pay out the Social Security bennies.


FLUAD is a standard-dose, three-component (trivalent) inactivated flu vaccine, manufactured by Seqirus that contains an adjuvant. FLUAD is designed specifically for people 65 years and older.

Uhuh. Which adjuvant is this?

MF59 is an oil-in-water emulsion of squalene oil. Squalene, a naturally occurring substance found in humans, animals and plants, is highly purified for the vaccine manufacturing process. FLUAD is approved for use among people 65 years and older, who often have a lower protective immune response after flu vaccination compared to younger, healthier people.

I hate being right.

I hate it, I hate it, I hate it.

The Aluminium Age and Alzheimer’s

Where’s the science?


“The results demonstrated that Al(III) induced the transformation of the initial random coil structure to the β-sheet configuration in the Aβ40 peptides. These structural changes facilitated the aggregation of Aβ40.”

Meanwhile, denial train. Choo choo!

“Here’s the evidence behind the presence of metals such as copper, zinc, iron and aluminium.”
The first three are needed by the body and not neurotoxins.
“But the evidence doesn’t yet show whether this relationship actually causes Alzheimer’s disease.”
Have you tried checking?
“It is also unclear whether reducing metals in the brain via drugs or reducing our exposure would have any effect.”
It is unclear whether Caesar was stabbed but there is a lot of blood and all these knives might’ve had some effect.
“These metals are essential to the healthy function of our brain,”
100% LIE.
Aluminium is not necessary for any bodily function.
But it makes food manufacturing dirt cheap!

“The body is able to tolerate these metals in small amounts by clearing through the kidneys. ”

Well, clearly not.

These include aluminium and lead, for example it has been shown that if they are not taken out by the kidneys through organ failure or by exposure to extremely high doses these metals are able to deposit in the brain.
These metals are known to cause negative effects in the brain and have been implicated in several neurological conditions.”
Safe as lead, guys!
And no that’s outdated, the body doesn’t really clear it. See end.
“In 1965, researchers found that rabbits injected with an extremely high dose of aluminium developed toxic tau tangles in their brains. This led to speculation that aluminium from cans, cookware, processed foods and even the water supply could be causing dementia.”
But the can manufacturers were slipped a bribe because they recently had to switch from toxic tin.
That generation’s children now seem to be presenting with unprecedented Alzheimer’s…. coincidence?
“Importantly, these results were only seen with extremely high exposures that far exceed the levels that can enter the body through food or potentially through contact with aluminium cookware.”
That limit does not exist and assumes perfect health – no alcoholism, polluted air, toxic water, stress, chronic diseases.
“As yet no study or group of studies has been able to confirm that aluminium is involved in the development of Alzheimer’s disease.”
Ethics and finding the funding.
Also a half-lie, there are many studies. As you’ll see.
“Aluminium is seen in the normal, healthy brain.”


No, it isn’t.
Not ever. Not at all.
It shouldn’t get past the barrier.
Normal for the modern world is not healthy.

Opening line here: “Aluminium is neurotoxic.”

Back to the liars:

“Although aluminium has been seen in amyloid plaques there is no solid evidence that aluminium is increased in the brains of people with Alzheimer’s disease.”
Yes there was, you’re ignoring it.

They dissected Alzheimer’s patients and found it in there.
Blatant lie.

No convincing relationship between amount of exposure or aluminium in the body and the development of Alzheimer’s disease has been established.”
Wait before it didn’t exist and in the very next sentence, it isn’t convincing?
“Aluminium in food and drink is in a form that is not easily absorbed in to the body. Hence the amount taken up is less than 1% of the amount present in food and drink.”
Bullshit. It’s the most processed form it goes through the whole digestive tract and then into the bloodstream like food nutrients.
Less than 1% – per meal or drink. PER meal or drink.
“Most of the aluminium taken into the body is cleaned out by the kidneys.”
And wouldn’t it be the liver?
“failed to find a convincing causal association between aluminium exposure in humans and Alzheimer’s disease.”
Give us the data instead of finding excuses to hide it.
1991, before the cash cow was in full milking rotation
“exposure to aluminium has been implicated by epidemiological studies and the finding of aluminium in the cerebral plaques and tangles.”
3 decades later? Still covering it up?
While they blame copper:
It’s neuroprotective.
“a high brain tissue ratio of copper to aluminium protects against neurotoxicity associated with the deposition of amyloid-b and the amyloid cascade hypothesis.”
“The study on 60 aged human brains identified a number of relationships between the degree of severity of amyloid-b neuropathology and the metal content of tissue from the donor brains. The latter was recently published for aluminium, copper and iron (House et al. (2012) Metallomics 4, 56-65).

Relationships, PLURAL and severity.

“Specifically, the extent and severity of amyloid-b deposition was inversely related to the copper content of brain tissue. Lower copper resulted in more severe and more extensive deposition of amyloid-b in the donor brain.”
But obviously you’re crazy to read about it.
“The research suggested that for those individuals with moderate to severe amyloid pathology, a copper to aluminium ratio of less than 20 predicted dementia.”
“Professor Exley said: “The hypothesis requires further testing but if proven correct it could explain why some individuals with senile plaques do not suffer from dementia. The implication being that a high brain tissue ratio of copper to aluminium protects against neurotoxicity associated with the deposition of amyloid-b and the amyloid cascade hypothesis.””

Aluminium-specific chelators reduce symptoms. Coincidence?
Effect is seen with low levels too:
By this means, the body burden of aluminum in humans has increased. Epidemiological and experimental findings indicate that aluminum is not as harmless as was previously thought, and that aluminum may contribute to the inception and advancement of Alzheimer’s disease. Epidemiological data is reinforced by indications that aluminum exposure can result in excess inflammatory activity within the brain.

“Evidence is presented that reinforces the likelihood that aluminum is a factor speeding the rate of brain aging. Such acceleration would inevitably enlarge the incidence of age-related neurological diseases.”

Maybe most Baby Boomers would be okay if their brains weren’t poisoned?

“However, disruption of these mechanisms, or absorption of detrimental metals with no known biological function, alter the ionic balance and can result in a disease state, including several neurodegenerative disorders such as Alzheimer’s disease. Understanding the complex structural and functional interactions of metal ions with the various intracellular and extracellular components of the central nervous system, under normal conditions and during neurodegeneration, is essential for the development of effective therapies.”

I hope you plan on someone in Big Pharma coming to kill you for developing therapies.

Opening line: “Aluminum has been identified as a neurotoxin for over 100 years.”
As safe as lead or mercury.

Click to access Aluminum2.pdf

You know where the society said no studies?
“A 2008 systematic review of studies that included aluminum exposure through drinking water, diet, and occupation found 23 studies demonstrated an increased risk for Alzheimer’s disease with elevated aluminum exposure, 3 found that there was no connection…”
Cover-up? Where’s the science?

I can’t seem to find it, guys!

We screened 4784 studies and included 60 in the review. Risk factors were considered in six categories: air quality, toxic heavy metals, other metals, other trace elements, occupational-related exposures, and miscellaneous environmental factors. Few studies took a life course approach. There is at least moderate evidence implicating the following risk factors: air pollution; aluminium; silicon; selenium; pesticides; vitamin D deficiency; and electric and magnetic fields.”
Life course is ideal.

There is a threshold.
“Our article examines the question of whether environmental and therapeutic aluminum exposure increases the risk of disease. To this end, Alzheimer’s disease and breast cancer are taken as critical endpoints. Aluminum’s neurotoxic effects in humans and its embryotoxic effects in animal models have been proven (4).”

Click to access f004582d987d2142ba418d26149d258d64e024e9.pdf

“An inevitable consequence of humans living in
the Aluminium Age is the presence of aluminium in the
brain. This non-essential, neurotoxic metal gains entry to
the brain throughout all stages of human development,
from the foetus through to old age. Human exposure to
myriad forms of this ubiquitous and omnipresent metal
makes its presence in the brain inevitable, while the
structure and physiology of the brain makes it particularly
susceptible to the accumulation of aluminium with age. In
spite of aluminium’s complete lack of biological essentiality,
it actually participates avidly in brain biochemistry
and substitutes for essential metals in critical biochemical
processes. The degree to which such substitutions are disruptive
and are manifested as biological effects will depend
upon the biological availability of aluminium in any particular
physical or chemical compartment, and will under
all circumstances be exerting an energy load on the brain.
In short, the brain must expend energy in its ‘unconscious’
response to an exposure to biologically available aluminium.
There are many examples where ‘biological effect’
has resulted in aluminium-induced neurotoxicity and most
potently in conditions that have resulted in an aluminiumassociated
encephalopathy. However, since aluminium is
non-essential and not required by the brain, its biological
availability will only rarely achieve such levels of acuity,
and it is more pertinent to consider and investigate the
brain’s response to much lower though sustained levels of
biologically reactive aluminium. This is the level of
exposure that defines the putative role of aluminium in
chronic neurodegenerative disease and, though thoroughly
investigated in numerous animal models, the chronic toxicity
of aluminium has yet to be addressed experimentally
in humans. A feasible test of the ‘aluminium hypothesis’,
whereby aluminium in the human brain is implicated in
chronic neurodegenerative disease, would be to reduce the
brain’s aluminium load to the lowest possible level by noninvasive
means. The simplest way that this aim can be
fulfilled in a significant and relevant population is by
facilitating the urinary excretion of aluminium through the
regular drinking of a silicic acid-rich mineral water over an
extended time period. This will lower the body and
brain burden of aluminium, and by doing so will test
whether brain aluminium contributes significantly to
chronic neurodegenerative diseases such as Alzheimer’s
and Parkinson’s.”

Of course, A Drink is cheaper than all the care of the sick patients and you can’t steal their house legally by telling the council they lack competence.

“Certainly aluminium either directly as a
particulate or indirectly following the dissolution of
nanoparticulates could induce an inflammatory action in
the human brain, and this has been demonstrated in animal
models [64]. The immunopotency of aluminiumbased
adjuvants outside their role as adjuvants in vaccine
and allergy therapies seems to have been largely ignored
as a potential mechanism of aluminium toxicity
throughout the body [65] and especially in the nervous
system [66]. The consistent observation of significant
accumulations of aluminium in the brain should at least
be a warning of the potential for such to participate in
neuroinflammatory toxicity.”

You’d think.

The brain is an obvious target for aluminium toxicity.
Neurotoxicity is evident under acute conditions such as
encephalopathies, and it is predicted but not necessarily
recognised as such under chronic or everyday exposures to
environmental aluminium. The mechanisms of neurotoxicity
are potentially myriad, while their manifestations as
biochemical changes are probably quite subtle for all but
the most vulnerable groups.”

Headaches, stomach aches, etc. “Brain fog” might be entirely toxin based.

“While the latter must include
the foetus and neonate, there are few indications as to the
identities of others who are susceptible to the neurotoxicity
of aluminium. Since the advent of the Hall-He´roult process
(and thereafter Bayer process) towards the end of the
nineteenth century and our ability to extract aluminium
from its inert ores on an industrial scale, we have all been
living in the Aluminium Age [67]. Now, in the twenty-first
century, we can no longer completely avoid environmental
exposure to aluminium. Since there is as yet no proven
requirement for aluminium in any living organism, never
mind in humans, it would be prudent to reduce our
everyday exposure to avoid aluminium entering the body
and persisting in the human brain [68].”

It’s used to process junk food, watch How It’s Made.
When people feel better after giving up junk, it might be aluminium exposure reduction.

It would be easy to study: people who eat a lot of junk and people who don’t, Al urine amounts.

“We have begun to
show that this can be achieved by using nature’s own way
of avoiding biologically available aluminium. We have
shown that regular consumption of silicon-rich mineral
waters both reduce our gastrointestinal uptake of aluminium
and, importantly, facilitate our urinary excretion of
systemic aluminium [48]. Life on Earth evolved in spite of
a crust of aluminosilicate [1]. However, the Hall-He´roult
process and the subsequent arrival of an Aluminium Age
have let the aluminium genie out of the bottle. Our final
wish should be that the unique inorganic chemistry of
aluminium and silicic acid will help to put the genie back
where it can be used effectively but, most importantly,

Trans. We evolved to take silicon into our brain but aluminium is taking its place.
It lied on its CV, it needs to be fired.

“This article summarizes the various ways in which Al induces oxidative stress, eventually leading to cell death. It also gives a brief account of manifold epidemiological studies that relate the abundant occurrence of Al in soil and water and the prevalence of AD. Al carriers, their role in AD, Al in neurofibrillary tangles, biochemical reactions altered by Al influx in mitochondria have been briefly discussed.”
One of the key words is apoptosis…

“If aluminium contributes to the development of sporadic AD, it must do so indirectly, perhaps via effects on the synthesis or metabolism of APP, or by contributing generally to the age-related attrition of neurons and thus reducing the threshold for deficits produced by more specific disease-related processes.”

Final study describes a mechanism.

Click to access 81923975.pdf

Back-up PDFs are always useful.



Active effect of aluminium on the brain’s self-cleaning.

“Aluminum, as an extremely high charge density cation (Z2/r = 18), has the remarkable capability to both (1) aggregate and compact Aβ42 peptide monomers into higher order, more neurotoxic oligomeric, and fibrillar structures, and (2) impair, at the molecular-genetic* level, the cellular machinery responsible for Aβ42 peptide monomer phagocytosis and clearance from the cell (4–13).”

*Hints at genetic damage.

Apparently the levels we ingest are “physiologically realistic.” As in, it can affect you.

They need chelation therapy.

Are Paleo and ketone diets effective because of their effect on mitochondria?


To explain the phenomenon, Ames came up with a theory rooted in our hunter-gatherer past, when micronutrient shortages must have been recurrent: To make sure the species was perpetuated, natural selection imposed a “strategic rationing response,” shunting the vital nutrients toward functions essential for short-term survival and reproduction, and away from longevity systems affected by dysfunctional mitochondria.

Personal theory of mine. Lots of overlap to be a coincidence. Plus the umami hit of high-protein foods, broken down by aging and roasting.