“It might sound strange, but its true, this remedy has been passed around the feminist community since the 70’s, appearing in many grassroots publications, some of which are cited here. There are also numerous reports of women using it successfully from this era, I’ve heard many stories, but never saw any kind of documentation, which isn’t surprising in a time, where a woman’s right to choose an abortion and have access to safe legal abortion services was just being won.”
Great for ye olde days of gang rape though. Useful if the Red Army comes around town. Abortion does make sense where continuing would kill the mother so there is an ethical grey area e.g. ectopic. I acknowledge that. We also must know what kills a baby so all mothers know to AVOID it. This is why keeping women ignorant leaves them vulnerable to such evil. Parts of nature hate us. Wiccans are imbeciles.
This is why I don’t supplement liposomal Vitamin C, as I suggested for OLDER people.
“The scientists who conducted the research, Samborskaia and Ferdman came to the conclusion that high doses of Ascorbic Acid appeared to increase estrogen levels which contributed to the interruption of an otherwise normal pregnancy. 20 women who approached doctors requesting an abortion participated in the study. Research was conducted by ob/gyn L.I. Ivanyuta. The women ranged from 20 to 40 years of age. The article does not say if a positive pregnancy test was obtained from the participating women. We also don’t know how much ascorbic acid the women were given. They did however measure estrogen levels before and after treatment with ascorbic acid, finding that estrogen levels were higher after taking the ascorbic acid. Of the 20 women, 16 began menstrual type bleeding within 1 to 3 days from administration of ascorbic acid.”
It makes giving kids lemonade real sinister. Mountain Dew, Sunny D, the works.
“Vitamin C works to produce an unfavorable climate within the uterus so that the egg does not implant, or if implantation has already occurred, Vitamin C can weaken the fertilized ovum’s grip on the uterine wall. Possibly by stimulating estrogen, and interfering with progesterone. This also makes it useful as an emergency contraceptive, when taken before implantation occurs on the 6th day following ovulation. The hormone, progesterone is essential for pregnancy, its function is to prepare a nourishing bed for the fertilized egg, if there is not enough progesterone the uterus becomes less supportive to the egg. Which is desirable when the goal is to end pregnancy.”
Progesterone means pro-gestation. Anything that reduces that and/or increases oestrogen causes miscarriage, including xenoestrogens. BPA also causes genetic defects inc. Downs, and can cause abnormal egg development in a female fetus, which can go on to experience many miscarriages (modern rates?) and Downs children themselves.
Also NO parsley. Yes, it kill babies. Viva Italia some other time. Can be used to induce labour, ironically.
History will view the use of xenos as pure evil*. I think endometriosis is caused by it, like a poisoning. Explains the miscarriage common to it. Most common cause of infertility in women. Pure progesterone creams hard to come by, easier to patent a toxic variety close enough. Even pure creams can include preservatives that are oestrogenic! Vegan love of vit C may cause vegan menopause, imho. Xenos also cause premature puberty in girls as young as ONE, especially seen in high-estro skin products used by American blacks and not found in African ones. Xenos (including hops in beer**) also cause a small penis and breast development in boys/men. This shit should be BANNED forever in all skincare vehicles (10x more potent, bypassing liver filter). The amount required (parts per billion) is rarely tested for but maintains estrogenic effect at this level. Parabens were disused in some products due to this. Others like SLS and phthalates also. It isn’t hype, it’s killing men/women hormonally and babies silently. A silent killer in shampoo, lotion, food etc. No wonder American rates of miscarriage are so high. Test ALL skin products for endocrine disruption, especially those that break down into it (XENOS), in rats. Xenos can bio-accumulate for decades in the body (heard of DDT?) and stay for decades too. I share this hoping people won’t abuse the info.
*file under Molech
**how Anglos have gotten softer and softer and softer… literally and morally.
Synthetic perfume is also a xeno. Sorry. I’m sad about it too. They’re aiming this at teen girls and boys, who get fat. And in the case of girls, look sexual. The boys look twinkish. I’m sure the traffickers love that.
They blame kids for being fat when they’re hormonally drugged from seemingly everywhere. They cannot lose weight! The environment is too polluted!
Phyto-estrogen can bind protectively and reduce the capacity of xeno to attach. This is limited. It’s less potent but still oestrogenic and thus reduces progesterone. Can detox from the body in a matter of days since it’s natural.
“cardiometabolic clinical correlates related to total testosterone (TT), free testosterone (fT), androstenedione (ASD), dehydroepiandrosterone-sulfate (DHEAS), estrone (E1), estradiol (E2), and sex hormone-binding globulin (SHBG).
Results: Waist circumference and BMI (β-coefficient: -0.03; 95% CI: -0.04; 0.03) were inversely related to SHBG, and BMI was positively related to TT (β-coefficient: 0.005; 95% CI: 0.001; 0.009), fT, E1, and E2. Smoking was positively related to TT (β-coefficient: 0.04; 95% CI: 0.01; 0.06), ASD, and fT. Systolic blood pressure (TT: β-coefficient: 0.002; 95% CI: 0.001; 0.003), hypertension (TT: β-coefficient: 0.05; 95% CI: 0.003; 0.11), low-density lipoprotein (LDL) cholesterol (TT: β-coefficient: 0.02; 95% CI: 0.01; 0.05), and total cholesterol (TT: β-coefficient: -0.03; 95% CI: 0.01; 0.05) were positively related to TT and ASD. Finally, type 2 diabetes mellitus (T2DM), and metabolic syndrome (MetS) were positively related to fT, but inversely related to SHBG.
Conclusions: Our population-based study, with sex hormone concentrations measured by liquid chromatography tandem mass spectrometry, revealed associations between clinical correlates including waist circumference, smoking, cohabitation, systolic blood pressure, cholesterol, and MetS with sex hormones. Thus, sex hormones and SHBG may play a role in the cardiovascular risk profile of women.”
Both obesity and anxiety symptomatology were separately associated with the same sex hormone alteration in premenopausal women: higher total testosterone level (0.97 ± 0.50 in obese vs. 0.86 ± 0.49 nmol/L in normal-weight women, p = 0.026 and 1.04 ± 0.59 in women with vs. 0.88 ± 0.49 nmol/L in women without anxiety symptomatology, p = 0.023). However, women with anxiety symptomatology had non-significantly higher estradiol levels than women without anxiety symptomatology (548.0 ± 507.6 vs. 426.2 ± 474.0 pmol/L), whereas obesity was associated with lower estradiol levels compared with those in normal-weight group (332.7 ± 386.5 vs. 470.8 ± 616.0 pmol/L). Women with anxiety symptomatology had also significantly higher testosterone and estradiol composition (p = 0.006). No associations of sex hormone levels and BMI with anxiety symptomatology in postmenopausal women were found.
Conclusions: Although both obesity and anxiety symptomatology were separately associated with higher testosterone level, there was an opposite impact of anxiety and obesity on estradiol levels in premenopausal women. We did not find an evidence that the sex hormone alterations related to obesity are playing a significant role in anxiety symptomatology in premenopausal women. This could be the explanation why we did not find an association between obesity and anxiety. In postmenopausal women, other mechanisms seem to work than in the premenopausal group.
Regional fat distribution (RFD) has been associated with metabolic derangements in populations with obesity. For example, upper body fat patterning is associated with higher levels of free testosterone (FT) and lower levels of sex-hormone binding globulin (SHBG). We sought to determine the extent to which this relationship was true in a healthy (i.e., non-obese) female population and whether RFD influenced androgen responses to resistance exercise. This study examined the effects of RFD on total testosterone (TT), FT, and SHBG responses to an acute resistance exercise test (ARET) among 47 women (22+/-3 years; 165+/-6 cm; 62+/-8 kg; 25+/-5%BF; 23+/-3 BMI). RFD was characterized by 3 separate indices: waist-to-hip ratio (WHR), ratio of upper arm fat to mid-thigh fat assessed with magnetic resonance imaging (MRI ratio), and ratio of subscapular to triceps ratio (SB/TRi ratio). Skinfolds were measured for the triceps, chest, subscapular, mid-axillary, suprailaic, abdomen, and thigh regions. The ARET consisted of 6 sets of 10 RM squats separated by 2-min rest periods. Blood was obtained pre- and post- ARET. TT, FT, and SHBG concentrations were determined by radioimmunoassay. Subjects were divided into tertiles from the indices of RFD, and statistical analyses were performed by an ANOVA with repeated measures (RFD and exercise as main effects). Significant (p < or = .05) increases following the AHRET were observed for TT (approximately 25%), FT (approximately 25%), and SHBG (4%). With multiple regression analysis, anthropometric measures significantly predicted pre- concentrations of FT, post-concentrations of TT, and pre-concentrations of SHBG. The SB/TRi and MRI ratios but not the WHR, were discriminant for hormonal concentrations among the tertiles. In young, healthy women, resistance exercise can induce transient increases in testosterone, and anthropometric markers of adiposity correlate with testosterone concentrations.
So exercise will boost a woman’s natural T. If they already have high T….
If their BMI is higher for their size, they already have high T comparatively. If they already have it racially… probably not good.
Compared to the decline in E2 concentrations, androgen concentrations declined minimally over the MT. T (β 9.180, p < 0.0001) and E1 (β 11.365, p < 0.0001) were higher in Whites than in AAs, while elevations in DHEAS (β 28.80, p = 0.061) and A4 (β 0.2556, p = 0.052) were borderline. Log-transformed E2 was similar between Whites and AAs (β 0.0764, p = 0.272). Body mass index (BMI) was not significantly associated with concentrations of androgens or E1 over time.
so black and white is off the hook
This report suggests that the declines in E2 during the 4 years before and after the FMP are accompanied by minimal changes in DHEAS, A4, T, and E1. There are modest differences between Whites and AAs and minimal differences by BMI.
During a median follow up of 6.3 years, 45 patients relapsed. Testosterone levels significantly increased across BMI categories (p = 0.001). Both circulating testosterone and BMI were positively associated with disease free survival (p = 0.005 and p = 0.021, respectively). A significant interaction was found between testosterone and BMI (p = 0.006). For normal-weight women, testosterone concentration around median (0.403 ng/mL) or third quartile (0.532 ng/mL) showed a high significant HR of relapse (5.52; 95% CI:1.65–18.49 and 4.55; 95% CI:1.09–18.98, respectively). Overweight patients showed increased HR at increasing testosterone levels, reaching a significant high HR (4.68; 95% CI:1.39–15.70) for testosterone values of 0.782 ng/mL (95th percentile). For obese patients HR decreased (not significantly) at increased testosterone concentrations, explaining the interaction between testosterone levels and BMI categories.
In ER-positive postmenopausal breast cancer patients, high testosterone levels are associated with worse prognosis in normal-weight and overweight women, whereas in obese seems to be associated with a better outcome. Although the results require further validation, they suggest that assessment of circulating testosterone and BMI could help to identify postmenopausal ER-positive patients at higher risk of relapse and potentially open new therapeutic strategies.
High T isn’t good, even in normal weight women. Water is wet.
“The findings of this study suggest high plasma levels of testosterone could play a role in the pathogenesis of type 2 diabetes among women,” Jon Jarløv Rasmussen, MD, PhD, a specialist registrar and postdoctoral researcher in the department of endocrinology at Rigshospitalet in Copenhagen, Denmark, told Healio. “The incidence of type 2 diabetes was rather low in the study, but the results implicate that screening for type 2 diabetes among women with higher plasma levels of testosterone may be beneficial, even among women who are young and without established comorbidities, such as polycystic ovary syndrome.”
In a retrospective study, Rasmussen and colleagues analyzed data from 8,876 healthy women (mean age, 38.5 years) who provided blood samples to measure plasma testosterone, dehydroepiandrosterone-sulfate (DHEAS), dihydrotestosterone (DHT) and sex hormone-binding globulin (SHBG) between January 2007 and December 2015. Researchers analyzed androgens using tandem liquid-chromatography mass spectrometry. Researchers used Poisson regression models to calculate incidence rate ratios for developing type 2 diabetes during a median follow-up of 8.1 years, stratified by androgen quartiles.
‘Normal weight’ women can get Type 2. Since Asians have higher T from higher BMI (against the white norm), they’ll be more likely to get it. This also explains the gestational diabetes common in Asian women, especially if the baby is mixed.
Nationwide, as many as 1 in 4 people who have diabetes don’t know they have it. But for Asian Americans, that number is much higher—1 in 2, the highest of all ethnic/racial groups. Why aren’t more getting diagnosed?
Weebs do not mention this. If your apparent rationale for avoiding fat white women is avoiding the Diabeetus genes, Asian is then categorically the worst racial group to mix with.
1 in 2, flip a coin, rice cooker.
I bet it’s higher in the women due to sweet tooth, so likely worse.
But people of Asian descent have less muscle and more fat than other groups and often develop diabetes at a younger age and lower weight. That extra body fat tends to be in the belly (visceral fat). This isn’t the “inch you can pinch,” the fat stored just under the skin. Visceral fat is out of sight, wrapped around organs deep in the body. You can’t tell how much visceral fat someone has by looking at them.
I didn’t call them skinny-fat to be mean, they really are!
Visceral fat is also sometimes known as “active” fat because it drives certain processes in the body that can increase the risk for heart disease, stroke, and other serious health conditions. Everybody has some visceral fat, but having too much is a major risk factor for developing type 2 diabetes.
….But BMI doesn’t catch Asian Americans in the normal weight range (18.5 to 24.9) who may very well have too much visceral fat and be at risk for type 2 diabetes. Researchers are now suggesting that people of Asian heritage get tested if their BMI is 23 or greater. Type 2 diabetes can be prevented or delayed, but only if people know they’re at risk and can take action!
They need a totally different (lower) testing standard, but they’re just like us, guys! Nay, SUPERIOR!
The same volume food in a smaller body, this isn’t hard to figure out. They’re not white women, eating like us makes them FAT.
Pregnant South Asian women carry a higher risk for developing gestational diabetes, a condition that’s dangerous for both mother and child. Between 2 and 10 percent of all pregnancies each year are complicated by gestational diabetes
2-10% in which demographic? Sounds like all? I bet it’s higher in certain ones, isn’t it?
Under risk factors is basically – be non-white
Being of Hispanic, Native American, African-American, Asian-American or Pacific Islander descent.
Women who have had gestational diabetes have a 20 to 50 percent chance of developing diabetes in the 5 to 10 years following pregnancy.
Our data indicate that although the historical or clinical risk factors for GDM are valid in Asians, using risk factors alone to select such patients for testing for GDM is inadequate. Many Asian women who develop GDM have no risk factors at all.
When Natural Selection hates you so much… maybe give it up?
r-types have higher numbers of issues like this, that would be fatal under natural law
They don’t ‘choose’ to stop at 1-2 kids, it isn’t ‘culture’, it’s fear (see below).
To avoid overlooking significant numbers of women with GDM, one may lower the specificity of the criteria, but this requires that the majority of patients be tested.
wow, that bad
Logistically, it is much simpler to conduct universal screening for all Asian women in Western countries, rather than to apply selective testing in order to spare a small percentage of women from being tested. Therefore, our findings strongly support recommendations for universal screening for GDM in pregnant women of Asian origin in Western countries. However, in places where the incidence of GDM is low, such as in some developing countries, the selection of patients for testing by the risk factors may be reasonable.
Introduction:Asian women have a higher prevalence of gestational diabetes mellitus than women of other races/ethnicities. We aimed to compare the prevalence of gestational diabetes among Asian American women to other racial/ethnic groups and explore whether the higher occurrence of the disorder among Asian women can be explained by acculturation.
Clearly I am making this all up to feel better, right guys?
Among the 5,562 women studied, the weighted prevalence of gestational diabetes was 15.5% among Asian American women, followed by 9.0% among non-Hispanic black women, 10.7% among Hispanic women, and 7.9% among non-Hispanic white women.
15.5% v. 7.9%
Diabetes at DOUBLE the rate of whites!
but they’re just like us
2.44x the risk
and that’s controlled, independently
Compared with non-Hispanic white women, Asian women had 2.44 (95% confidence interval [CI], 1.81–3.29; P < .001) times the odds of having gestational diabetes, independent of maternal age, education, marital status, income, prenatal care adequacy, prepregnancy BMI, and physical activity. Acculturation was negatively associated with having gestational diabetes (odds ratio [OR] = 0.93; 95% CI, 0.86–0.99) and explained 15.9% (95% CI, 11.38%–25.08%; P < .001) of the association between Asian race and the condition.
About 85% genetic. Great odds.
We found that Asian race was an independent risk factor for gestational diabetes, and higher acculturation may play a protective role against it in Asian American women.
What is already known about this topic?
Asian women have a higher prevalence of gestational diabetes mellitus than women of other races. However, little data exist on why prevalence is highest among Asian women.
I sense genetics.
If they’re having unnatural babies (too large for their race, mixed) supported by modern medicine, they’d be more likely to die anyway, right? Medicine can only do so much. Weaker genes die a la Darwin.
The biggest r-select factor would be risk of death while breeding, that would be the surest thing. The genes trying to extinct themselves.
Does this data exist? Also for the neonates?
YOU BET IT DOES.
Let’s see the weebs explain away these studies. They’ll probably just ignore me… again.
Pregnancy related mortality can be defined as death of the mother during pregnancy, delivery, or within one year postpartum. While 700 pregnancy-related deaths occur each year, 2/3 of these deaths are considered to be preventable.
Modern medicine, dysgenic again.
Overall pregnancy related mortality in the United States occurs at an average rate of 17.2 deaths per 100,000 live births. However, that number jumps to 43.5/100,000 for non-Hispanic Black women and decreases to 12.7/100,000 for non-Hispanic white women and 11/100,000 for Hispanic women.
No data listed for Asian, odd?
For mothers of all backgrounds, leading causes of death include cardiovascular conditions, hemorrhage, and infection. However, for non-Hispanic Black women, leading causes of death include cardiovascular conditions in addition to cardiomyopathy, pre-eclampsia, and eclampsia (hypertensive disorders).
Non-Hispanic Black women are also significantly more likely to have a severe maternal morbidity (SMM) event at the time of delivery. For every maternal death there are 70 cases of SMM events that are considered “near misses.” These events can have long-term or short-term consequences to a woman’s health. Over the past 20 years, cases of SMM have increased by over 200%, while cases disproportionately affect Black women. One study found Black women experienced SMM at a rate 2.1 times greater than that of white women.
To better understand and address these disparities, researchers suggest providers increase screening for social determinants of health. Levels of stress, trauma, food insecurity, neighborhood violence, and access to prenatal care are all factors that may contribute to the disparities and warrant further investigation.
Although most maternal deaths result from cardiovascular and hypertensive disorders, researchers found Asian/Pacific Island women exhibit the highest prevalence of gestational diabetes, which can increase pregnancy complications, at 14.8%.
One study presented in the session focused on behavioral interventions and protective factors among women with gestational diabetes. A Kaiser Permanente analysis of women in northern California found Black women have a lower prevalence of gestational diabetes when compared with Asian Indian, Filipina, Southeast Asian and Chinese women. White women had the lowest rates of the disease overall.
Screening for postpartum diabetes is recommended to all women within 4 to 12 weeks postpartum. However, rates of screening vary among women with different racial and ethnic backgrounds, suggesting tailored strategies to reduce risk and improve healthcare behaviors may be effective.
Racial medicine, openly.
An additional study explored how racial and ethnic disparities impact severe neonatal morbidities, specifically among very preterm children (born <32 weeks of gestation). Preterm birth has been associated with several health conditions developing later in life, including diabetes.
Presenter Teresa Janevic, PhD, defined race as “linked to phenotype and /or ancestry that indexes one’s location on the US social hierarchy of socially constructed groupings (i.e., races) that has been based primarily on skin color.”
genes aren’t social
Africans in Africa also have the same ‘risk’ as one in America. No magic dirt.
In contrast, Janevic defined ethnicity as “tied to race and used both to distinguish diverse populations and to establish personal or group identity, usually based on shared culture or beliefs.”
Culture? Belief? Believe your way out of diabetes. I’ll wait.
In a population-based retrospective cohort analysis using hospital discharge data linked with vital statistics at birth and death records, researchers determined Black infants were at the highest risk of dying within less than 28 days after discharge, or suffering neonatal morbidities in the time between birth and discharge. Black infants were followed by Hispanic infants, while white and Asian infants had similar low risks.
We’ll see about that.
Of the 39 New York City hospitals included in the study, researchers found a 6-fold difference in risk of combined mortality and morbidity outcomes. “Black infants were at twice the risk of being at a hospital that has risk-adjusted high rates of combined mortality and morbidity,” Janevic noted, while Hispanic infants had a 1.5 increased risk to receive care from one of these hospitals. “Hospital quality where women of color deliver likely contributes to these disparities,” she concluded.
Like schools, it depends on the IQ of the people working there.
Another investigation detailed how environmental factors and population level exposures impact disparities in preterm birth and infant mortality. “Non-Hispanic Black infants compared with non-Hispanic white infants have twice the risk of death in the first year,” explained presenter Heather Burris, MD. “This is particularly striking because Black infants just make up 15% of all births in the United States but are counting for 29% of all deaths.”
Among causes of infant death, preterm birth and low birth weight related death, along with pregnancy complications, account for the highest racial and ethnic disparities between non-Hispanic Black and white infants. Black infants are also significantly more likely to be born preterm than white infants.
an r-factor unless twins
Researchers note genetics and education level have very little impact in accounting for disparities in preterm birth. Although women with higher education tend to have lower preterm birth rates, Black women who graduated from college have a higher risk of preterm birth than white women who dropped out of high school.
I’m so glad white people already survived multiple genetic purges in our history.
Through analyzing delivery data and creating models based on air pollution severity in Philadelphia, Pennsylvania, investigators discovered air pollution is associated with spontaneous preterm birth. Data also show Black Americans experience consistently higher exposure to air pollutants, measured in fine particulate matter (PM)2.5.
An additional analysis between preterm birth and nationwide neighborhood deprivation index (encompassing income below the poverty level, vacant homes, education levels, among other factors) found that Black women experience neighborhood deprivation exposure at almost 2 standard deviations (SDs) higher than white women in Philadelphia.
Overall, Black women are 4 times more likely to live in a neighborhood with high violent crime and high air pollution than white women. “When we look at preterm birthweights, we can see that it is women living in these high-high neighborhoods that have the highest risk of preterm birth,” Burris said. However, these associations were consistent regardless of race.
They gestate for less time than whites, this is known. Africans in Africa do it.
Now we’ve established some things. An r-study in Asian women.
Increased Perinatal Morbidity and Mortality Among Asian American and Pacific Islander Women in the United States
Background: Asian American/Pacific Islanders (AAPIs) are the fastest-growing racial group in the United States.
America is now owned by Asia, demographically.
Despite a higher socioeconomic status, AAPI women experience higher rates of maternal morbidity and mortality.
can’t pay your way out of r-genes
if controlled for SES, aka $, their data would be even worse
Methods: Using the National Inpatient Sample, we performed a retrospective cohort analysis of women who were hospitalized for delivery from 2002 to 2013. The primary outcome variable was inpatient mortality rate, and the presence of severe maternal morbidities was estimated using the Bateman Comorbidity Index, a validated tool for predicting obstetric morbidity.
Results: AAPI women presenting for delivery between 2003 and 2012 were older, more likely to reside in a zip code in the top quartile of annual income, be privately insured than Caucasian women,
where’s Asian privilege?
and less likely to have a higher Bateman Comorbidity Index. However, AAPI women had a higher likelihood of postpartum hemorrhage (3.4% vs 2.7%, P < .001), uterine atony, severe perineal lacerations, and severe maternal morbidities. Procedures such as transfusion, hysterectomy,
So they could have one kid and die, have one kid and have that die, OR have one kid and then their organs all removed – so no more kids?
Yes clearly our biological superiors, right weebs? Totally not rationalising a fetish, are we?
I wonder why one child was law? They don’t have a culture of many kids because they’re too r-select to survive without modern medicine. Wake up. They pretend 1-2 is a choice and that’s why they mock and envy large white families (3+ standard) like the Amish. They envy us that ability. They would die.
and mechanical ventilation were also more common in AAPI women.
Calling it – Mother Nature is anti-Asian.
Furthermore, AAPI women had a higher mortality rate that persisted despite adjustment for an apparently higher income and comorbidities (odds ratio 1.72, 95% confidence interval: 1.14-2.59, P = .01).
Conclusions: Despite having a higher socioeconomic status, AAPI women had higher rates of maternal mortality during hospitalization for delivery. This increase persisted even after adjustment for factors known to affect peripartum outcomes. Further investigation is needed to better clarify the causes of racial differences in maternal morbidity and mortality.
Results: A total of 360,370 women with postpartum hemorrhage from 2012 to 2014 were included in this analysis. Risk for severe morbidity was significantly higher among non-Hispanic black women (26.6%) than non-Hispanic white, Hispanic, or Asian or Pacific Islander women (20.7%, 22.5%, and 21.4%, respectively, P < .01).
The white is 20%, Asian is 21%.
And these are the fattest white people, like, ever.
White and Asian bolded-
For non-Hispanic black compared with non-Hispanic white, Hispanic, and Asian or Pacific Islander women risk was higher for disseminated intravascular coagulation (8.4% vs 7.1%, 6.8%, and 6.8%, respectively, P < .01) and transfusion (19.4% vs 13.9%, 16.1%, and 15.8%, respectively, P < .01). Black women were also more likely than non-Hispanic white women to undergo hysterectomy (2.4% vs 1.9%, P < .01), although Asian or Pacific Islander women were at highest risk (2.9%). Adjusting for comorbidity, black women remained at higher risk for severe morbidity (P < .01). Risk for death for non-Hispanic black women was significantly higher than for nonblack women (121.8 per 100,000 deliveries, 95% confidence interval, 94.7-156.8 vs 24.1 per 100,000 deliveries, 95% confidence interval, 19.2-30.2, respectively, P < .01).
The weebs either did 1. no research (typical gammas) or 2. they’re delusional.
Almost double the risk of hysterectomy, roughly.An additional 52% risk over white women, minimum, in just this study.
What’s the point of being married to them, at that point? Their baby machine is broken.
Non-Hispanic black (black) and non-Hispanic American Indian/Alaska Native (AI/AN) women experienced higher PRMRs (40.8 and 29.7, respectively) than all other racial/ethnic populations (white PRMR was 12.7, Asian/ Pacific Islander PRMR was 13.5 and Hispanic PRMR was 11.5). This was 3.2 and 2.3 times higher than the PRMR for white women – and the gap widened among older age groups.
Notably, we found that, when aggregated, the top cause of death among Asian Americans is cancer. However, when disaggregated, there is wide variation in the leading cause of death. For instance, for Asian Indians, nearly twice as many men die of heart disease (31 percent), compared to cancer (18 percent). In contrast, for Koreans, the opposite is true — the death rate for cancer (34 percent) is much higher than the death rate for heart disease (19 percent).
Remember the breast cancer and Asian BMI/testosterone stuff?
Research led by the University of Birmingham has found that increased levels of hormones including testosterone could cause a brain condition that can lead to blindness in women.
We are all jealous of your waifu, yes.
Idiopathic Intracranial Hypertension—also known as IIH—is caused by high pressure in the brain with consequences from blindness to incapacitating daily long-term headaches. IIH was originally identified over 100 years ago yet the cause of the condition has remained unknown although there has been much speculation about why more than 95 per cent of total incidence is in women with obesity.
And Asians, they’re 1/2 obese in America!
They then compared the results with the levels observed in women with obesity of the same age and body mass index (BMI), as well as a cohort of women with polycystic ovary syndrome (PCOS).
PCOS is far more common in Asians. Look it up.
Most notable were the high levels of the androgen ‘testosterone’ found in the blood in IIH women. Crucially, levels of androgens were uniquely increased in the brain fluid (CSF) of women with IIH. When the researchers, analysed human choroidal plexus tissue, which is the site in the brain where CSF is produced, they confirmed that androgens could increase the rate of CSF secretion, a potential driver for increased brain pressure.
Results: We found that the South Asian women presented at a younger age for the management of sub-fertility. An extended stimulation phase and Caucasian ethnicity showed an inverse correlation with the number of oocytes retrieved in the PCOS subgroup. Caucasian ethnicity was associated with a higher fertilization rate however increase in body mass index (BMI) and the laboratory technique of IVF appeared to have a negative impact on fertilization rates in the PCOS subgroup. Commencing down regulation on day 1 of the cycles was negatively associated with fertilization rates in the tubal group. In terms of clinical pregnancy rates, the Caucasian PCOS had a 2.5 times (95% CI: 1.25-5) higher chance of an ongoing clinical pregnancy as compared with their Asian counterpart. Also, a unit increase in the basal FSH concentration reduced the odds of pregnancy by 18.6% (95% CI: 1.8-32.6%) in the PCOS group.
Conclusions: The Asian PCOS have a greater sensitivity to gonadotropin stimulation with lower fertilization and ongoing clinical pregnancy rates as compared with their Caucasian counterparts.
The ethnicity of women undergoing fertility treatments like IVF can affect the rate of successful live births, according to new research. After adjusting for certain factors including age of patient at time of treatment, cause of female or male infertility, and type of treatment, the study found that White Irish, South Asian Indian, South Asian Bangladeshi, South Asian Pakistani, Black African, and Other Asian women had a significantly lower odds of a live birth than White British women.
White women, still winning. Thank God for the Ice Age.
Overall, studies have shown higher testosterone levels in women and lower levels in men are related to incident diabetes. The major risk factors contributing to diabetes are biochemical, environmental, sedentary lifestyle, socioeconomic status and genetic factors. All of them together or independently are responsible for the development of the DM.  Besides, certain studies show Impaired Glucose Tolerance (IGT) is more common in females than males independent of age. 
We found a high prevalence of GDM among the Asian population. Asian women with common risk factors especially among those with history of previous GDM, congenital anomalies or macrosomia should receive additional attention from physician as high-risk cases for GDM in pregnancy.
Body mass index (BMI) was a very strong negative predictor of body attractiveness ratings, similar to previous findings. Zero-order associations between women’s mean hormone concentrations and mean attractiveness ratings were not significant; however, after controlling for BMI, attractiveness ratings were independently and positively associated with both estradiol and testosterone concentrations. Discussion focuses on the implications of these findings for whether attractiveness assessment mechanisms are specialized for the detection of cues of differential fecundity in young women’s bodies.
High T = ugly!
Did I mention water is wet? Can they seriously accuse of cherry picking? I’m not even looking hard.
Previously covered WHR, use search bar. Asians lose. Even black women do better.
Asians have way more T as a race than Europeans, get over it. Historically, we considered them savages, less civilized, for that reason. How is this surprising? Do you think we colonised India for fun? It’s obvious in the broad manjaws, duh. Marquardt covered this. Anyone can do a replication study, but I suggest you include the women too, so it isn’t just a sexual effect but race.
From a blog “East Asians were found to have the highest average total plasma testosterone (5,673 ρg/mL) followed by Africans (5,442 ρg/mL) and then Europeans (4,992 ρg/mL). Given that the sample size for Africans is smaller (N < 10,000), their relative position may change with more data. Nonetheless, the claim that East Asians have the least testosterone is not supported by scientific data. “
Yeah, fake redpills who think T = manly, good thing. It’s just a hormone.
“There is no way of accurately determining free testosterone. Even if there was, this would also be irrelevant since bio-availability is prime. Since race realists use total serum testosterone, why is this an issue?”
true, it’s just applying the same standard
Culturally, gang rape is more normal in Asia than Africa. This is why. You don’t get African Taharrush, really. Asia has Eve Teasing and the like. Trust me, you don’t want this.
“Mass sexual assault is the collective sexual assault of women, and sometimes children, in public by groups of unrelated men. Typically acting under the protective cover of large gatherings, victims have reported being groped, stripped, beaten, bitten, penetrated and raped.”
As for the contention that there are no studies indicating a 10% difference between East Asians and Europeans, I did find one age controlled study where the Chinese sample had 8.8% more total T, 11.4% more bio-available T and 12% more free T than the European sample. The Japanese sample had 10.5%, 5.1% and 6.7% more than Europeans respectively [Wu et al. 1995]. Wonder if race realists discuss this study, or perhaps they are too busy in celebratory dance around the Korean/Swede campfire?
They’re not really redpill. I believe data even if I dislike it. Asians have high T as a race. Get over it.
High T can also dovetail with lower national IQs e.g. India, so…. why want this? Low IQ nations have more crime.
Additionally, this recent study shows HK Chinese having some 3% more bio-available T than US Europeans.
Lol, he’s right. But T isn’t a good thing. It’s just a hormone, in men or women.
Being a race realists seems to be a length engagement with delusion, fantasy and ‘scientific’ homo-erotica.
Not here, son. I believe the T-data. Penis size generally correlates to racial height (in white men), not really T. Forum bros are wrong again. Penis stuff is sexual selection, aka chosen by women.
There was a similar increase in the positive effect of penis size on attractiveness with a more masculine body shape (i.e., greater shoulder-to-hip ratio). Surprisingly, larger penis size and greater height had almost equivalent positive effects on male attractiveness. Our results support the hypothesis that female mate choice could have driven the evolution of larger penises in humans. More broadly, our results show that precopulatory sexual selection can play a role in the evolution of genital traits.
It was concluded that all penile measurements are interrelated to each other; the height and weight also the other body measurements that are related to the penile measurements in less than 50%. It seems that the penile measurements are polygenic traits and are under multifactorial influences.
There are racial differences in associations of hormone levels with age and BMI in late reproductive age women. Further study is needed to replicate these findings and to determine the relationships of these hormonal associations with menopausal symptoms
Obesity is an important factor in hormone dynamics independent of age, race and smoking in mid-life women, although the mechanisms remain unclear.
From “A Study of the Correlation of Some Sex Hormone with Obesity in Women with Secondary Infertility” (google it)
Infertility is the inability to conceive a child for more than one year. The present study indicates
that the obesity associated with infertility. The aim of the study to determine follicle stimulating
hormone, luteinizing hormone, testosterone hormone and prolactin levels. and cholesterol and
triglyceride concentration in 2nd inferetid women. This study was carried out at kamal al-samaarai
hospital the data were collected from 95 secondary infertilial women were age between 16-45 years old and grouped them in to obese (n = 46) and non obese(n = 49). There was no significant difference between the two groups (p <0.05).Body mass index in Infertile obese women is slightly higher than non obese Infertile women which is statistically significant (P<0.001). However LH,
TSH, cholesterol and triglyceride concentration in obese infertile women is significantly higher than
non obese infertile women (p >0.05).The BMI was correlated positively with triglyceride in obese
group while BMI was positive correlation highly significant with cholesterol in non obese group.
Regression analysis revealed obese to be strongly associated with observed infertility. The elevated
prolactin values in secondary infertile women clearly shows that there is a mechanism operating at
the anterior pituitary level which shows an abnormal distribution of FSH and LH which may further
explain the abnormal delay ovum maturation. This study also indicates obese associated with
infertile more than non obese women.
TLDR Asians carry more fat than whites (in America, at least).
Well yeah, the junk food and spices diet is hell on the hormones too. They evolved for multiple famines (especially the Chinese and Indians) so they tend to weight-gain like that, too. Fat deposition isn’t the same between race (black thighs/’ass’) so why would accumulation patterns and proclivity to gain work like that either?
Results: A convenient sample of 1626 adults with BMIs ≤35 was evaluated. Independent percentage body fat predictor variables in multiple regression models included 1/BMI, sex, age, and ethnic group (R values from 0.74 to 0.92 and SEEs from 2.8 to 5.4% fat). The prediction formulas were then used to prepare provisional healthy percentage body fat ranges based on published BMI limits for underweight (<18.5), overweight (≥25), and obesity (≥30).
Conclusion: This proposed approach and initial findings provide the groundwork and stimulus for establishing international healthy body fat ranges.
But they’re not the same. Bio-logically.
The Asian ‘gym girls’ also trying to pass off the manjaw as ‘gains’ by matching on the stomach and thighs, I’ve seen white lawyer types do the same. Do NOT fall for it, men. If the jaw is square and broad as a child and pre-teen, RUN.
The small, squat (squarish) skeleton and pedomorphic features of the Asian race (historically, accurately called Mongoloid if you can search for it in anthropology) already pre-dispose their young people (teens) to lower BMI anyway, so the ‘losing weight’ excuse is bullshit, they’re building muscle to match the jaw and trap some sucker with their high T and bad temper. This lower BMI is not a consequence of health (dietary or genetic) so compared to the unhealthiest white people EVER (sorry America) they still have higher BMIs. HOW.
White women have a naturally high BMI (taller, curves) so it’s actually worse than it looks. The effect is maintained in every age group, how can one fuck up age 20? Any 20yo complaining about their metabolism needs a slap. The fat Asian hypothesis also holds true within the race, compared the Asian men to women matching age.
IF ONLY THE MANOSPHERE ACTUALLY READ THE DATA.
Every other claim about Asians it’s like  yet when I go to look…. they lied.
I expected the one claim the weebs consistently made about Asians in America (being thin) must be true but … no.
18.5 is a terrible BMI cut-off because it includes anorexics, smokers, druggies and petite women all in one. Women look wildly different at low and too-low body fats, which also varies by race. At the same BMI, Asians look more husky because the skeleton is squared-off. They’re also likelier to lay down fat than muscle.
The Asians are larger than white women pre-menopause, what EXCUSE is there?
The only time whites actually have slightly higher BMIs (ever so slightly) is in the elderly range (60-79, at which point yeah you’re excused, you can go).
I’m seeing through the model minority propaganda and there’s nothing to it. Rule #1 of moving to America – DON’T GET FAT. YOU HAD ONE JOB.
“In contrast, models for Asians predicted a different percentage body fat from that predicted for African Americans and whites. “
That’s code for ‘they evolved so much for famine that we had to invent new maths to count it’.
“For example, Deurenberg et al (35) found that American blacks had a 1.3-unit lower and Polynesians a 4.5-unit higher BMI than whites with the same body fatness (35). Even within the white cohort, the investigators observed small differences between Americans and Europeans.”
Asian placement is denser. Squat Mongoloid skeleton, anthropologically. If you disagree, it’s cope.
Americans are mixed white, generally, that’s why. German/French hybrids and other nonsense. You lose adaptive advantage from your subrace too.
They studied one of the thinnest types of Asians (japs) so I guarantee you others would be worse.
“The underlying causes of ethnic variation in relations between BMI and percentage body fat are likely due to small between-center body fat measurement differences and biological between-group differences (35).”
“The evaluation of Asians was confined to Japan and that of African Americans to the United States. Therefore, the underlying causes of observed ethnic differences in terms of measurement, environmental, and genetic factors are difficult to ascertain. Nevertheless, it appears evident that a single set of universal percentage body fat ranges cannot be easily developed without considerable additional analysis of this problem. Our equations and associated tables provide several ethnic-specific ranges as working guidelines. Because African Americans and whites differed only slightly in percentage body fat (by 1–2%) after BMI was first controlled for, we presented a combined equation (Equation 5) and table (Table 4) based on 4C percentage body fat for these 2 groups.”
That’s right, our fat deposition is more alike blacks than Asians. That’s how different the races are.
Background: Asians who have a healthy body mass index (BMI) range have been observed to have higher levels of obesity and risk of cardiovascular disease than whites, which suggests that the relation between BMI and adiposity may be different for Asians.
Objective: The primary aim of this study was to investigate the influence of childhood and adolescent exposure to a westernized environment on the relation between BMI and percentage body fat in young Asian American women.
So no, this isn’t their age. No cope here son.
Design: Secondary data from 129 Asian women, aged 20–25 y, with variable lengths of residence in the United States and 327 white women of comparable ages who had participated in the Latina and Asian Bone Health Study (1999–2000) and the Berkeley Bone Health Study (1998–2000), respectively, were analyzed by using multiple linear regression with percentage body fat as the outcome variable and place of birth, ethnicity, length of US residency, and BMI as predictor variables.
Results: Asians who lived in the United States <12 y showed the same relation between BMI and percentage body fat as did whites. In contrast, Asians who had lived in the United States ≥12 y had higher percentage body fat than did whites for BMIs (in kg/m2) <20.5 and lower percentage body fat for BMIs in the overweight and obese range.
Responding to the same stimuli wildly differently > genetic!
They’re more likely skinny-fat, aka fatal fat.
Whites evolved to eat Roman grains like processed wheat.
Conclusions: Our findings suggest that childhood environments may influence the relation between BMI and adiposity. Research is warranted on the role that childhood environments play in the accumulation and distribution of body fat and hence metabolic disease risk later in life.
There’s still a huge difference in the same environment.
Asians with more years in the United States had higher mean percentage body fat than did whites at low BMIs and lower mean percentage body fat at high BMIs with the regression lines intersecting at BMI ≈20.5 and body fat of 26.5%.
Again, Magic Dirt isn’t real. Enjoy your BBW Ting Tong for your golden years.
We are supremely jealous.
Asian-immigrant adults who were living in North America were observed to have higher adiposity levels
at the same BMI level than did whites (6). However, most of the Asian-immigrant adults studied were raised outside of North America. To our knowledge, our study was among the first studies to observe a difference in how BMI relates to percentage body fat between Asian Americans who had spent more years of childhood in the United States compared with Asian Americans who were minimally exposed to the United States during childhood and adolescence.
Lesson for weebs – do NOT move the Thai bride to America.
Stay in Asia or do NOT bother. She WILL get fat. You have been warned.
American-born Asians had a higher percentage of mothers with college degrees (59% of mothers compared with 37% of mothers in foreign-born Asians and 32% of mothers in whites).
Fat SJWs, great catch!
in our study, which suggested that obesity rates in our sample of Asians were likely lower than in the general population.
They admit it.
Is the general observation that, at the same BMI level, Asians have higher total body fat and visceral fat and higher risk of cardiovascular disease risk than do whites applicable to Asians in general,
or is this observation applicable only to Asians living in Asian societies? If the latter, does the environment during early life influence how body fat accumulates and distributes and, therefore, affects the relation between BMI and body fat? How strong a predictor of child growth and body composition is socioeconomic background and, in particular, the mother’s education? Are our findings also applicable to men?
We concluded that the early life environment may influence the relation between BMI and adiposity in later life. In particular, we speculated that Asian women substantially exposed to the American environment early in life exhibited different patterns of accumulation of body fat than did Asian women raised in Asia;
non-sig, see above
these differences may be partially attributable to environmental and lifestyle factors that affected diet and physical activity. For example, compared with Asian children raised in the United States, children who live in Asia tend to spend more time studying, whether in school or at home, and less time doing sports and engaging in recreational activities (25). In the past, diets of Asians raised in Asia also tended to be different, with a lower consumption of breakfast cereals, dairy products, and processed meats, than diets of Asians raised in the United States (26).
Yes, they do try to eat white. Silly Asians, didn’t evolve for dairy.
So your grandkids will definitely be fat, even if she isn’t by some fluke? Great, very likely to pass on your genome.
What a waste of time and resources.
However, with the rapid changes that have been taking place in Asian economies and the globalization of the food supply, we expect to see further changes in the diets of children in Asia (27) and, consequently, in body composition.
The understanding of how childhood environments influence the relation between BMI and percentage body fat has implications for the use of BMI as a screening indicator for obesity and obesity-related conditions. In addition, research to understand the influence of environmental factors on the accumulation and distribution of total body fat and metabolically active visceral fat during critical life stages will add insight into the use of clinically relevant screening tools for chronic disease risk.
Plan on growing old together, spending a loooong time with fatass.
South Asians are susceptible to insulin resistance even without obesity. We examined the characteristics of body fat content, distribution and function in South Asian men and their relationships to insulin resistance compared to Caucasians.
so no, you can’t tell by ‘looking at her’, your norms are informed by white women
Research Design and Methods
Twenty-nine South Asian and 18 Caucasian non-diabetic men (age 27±3 and 27±3 years, respectively) underwent euglycemic-hyperinsulinemic clamp for insulin sensitivity, underwater weighing for total body fat, MRI of entire abdomen for intraperitoneal (IP) and subcutaneous abdominal (SA) fat and biopsy of SA fat for adipocyte size.
Compared to Caucasians, in spite of similar BMI, South Asians had higher total body fat (22±6 and 15±4% of body weight; p-value<0.0001), higher SA fat (3.5±1.9 and 2.2±1.3 kg, respectively; p-value = 0.004), but no differences in IP fat (1.0±0.5 and 1.0±0.7 kg, respectively; p-value = 0.4). SA adipocyte cell size was significantly higher in South Asians (3491±1393 and 1648±864 µm2; p-value = 0.0001) and was inversely correlated with both glucose disposal rate (r-value = −0.57; p-value = 0.0008) and plasma adiponectin concentrations (r-value = −0.71; p-value<0.0001). Adipocyte size differences persisted even when SA was matched between South Asians and Caucasians.
Marry those genes, go ahead.
Insulin resistance in young South Asian men can be observed even without increase in IP fat mass and is related to large SA adipocytes size. Hence ethnic excess in insulin resistance in South Asians appears to be related more to excess truncal fat and dysfunctional adipose tissue than to excess visceral fat.
in the men
harsh to call a racial adaptation to famine ‘dysfunctional’
stop acting like estrogen is a poison when it actually protects your body and makes your brain stress-resistant
Although the prevalence of obesity is higher among women than men, they are somewhat protected from the associated cardiometabolic consequences.
It’s easier to get a higher % when you already have a higher %. There are some of the hottest women alive under the carb loading fatties, sadly. Their natural curves predispose them to obesity.
Bring back keto!
The increase in cardiovascular disease risk seen after the menopause suggests a role for estrogens. There is also growing evidence for the importance of estrogen on body fat and metabolism in males. We hypothesized that that estrogen administration would ameliorate the adverse effects of obesity on metabolic parameters in males.
Having high T, lower E in a man can make fat more stubborn, the E cannot signal properly to clear it.
Thus, DIO induces sex-specific changes in glucose–insulin homeostasis, which are ameliorated in males treated with estrogen, highlighting the importance of sex steroids in metabolism. Given that altered peripheral glucocorticoid metabolism has been observed in rodent and human obesity, our results also suggest that sexually dimorphic expression and activity of glucocorticoid metabolizing enzymes may have a role in the differential metabolic responses to obesity in males and females.
So fatter women are healthier than fatter men. This makes sense because of baby weight. Women can lose it breastfeeding, that’s why we get it.
Plus we are naturally fatter. To grow the baby in the first place.
Estrogens play a fundamental role in the physiology of the reproductive, cardiovascular, skeletal, and central nervous systems. In this report, we review the literature in both rodents and humans on the role of estrogens and their receptors in the control of energy homeostasis and glucose metabolism in health and metabolic diseases. Estrogen actions in hypothalamic nuclei differentially control food intake, energy expenditure, and white adipose tissue distribution.
brain stuff = bitch tits
fat boys could be ruined for life, the developmental windows have closed
they may respond like girls medically, forever
Estrogen actions in skeletal muscle, liver, adipose tissue, and immune cells are involved in insulin sensitivity as well as prevention of lipid accumulation and inflammation. Estrogen actions in pancreatic islet β-cells also regulate insulin secretion, nutrient homeostasis, and survival. Estrogen deficiency promotes metabolic dysfunction predisposing to obesity, the metabolic syndrome, and type 2 diabetes. We also discuss the effect of selective estrogen receptor modulators on metabolic disorders.
I hope the fat acceptance lot don’t find this.
But not all fat is bad, especially evolved fat on women.
It makes WOMEN healthier, but never men. Classic sex differences. Women have curves.
Adipose tissue is an organ with active endocrine function involved in the regulation of energy balance and glucose homeostasis via multiple metabolic signaling pathways targeting the brain, liver, skeletal muscle, pancreas, and other organs. There is increasing evidence demonstrating that the female sex hormone, estrogen, regulates adipose development and improves systemic glucose homeostasis in both males and females. The underlying mechanism linking estrogenic regulation in adipose tissue and systemic glucose metabolism has not been fully elucidated, but is thought to include interactions of estrogen receptor signaling events involving lipolytic and/or lipogenic enzyme activity, free fatty acid metabolism, and adipocytokine production. Thus, understanding the effects of estrogen replacement on adipose tissue biology and metabolism is important in determining the risk of developing obesity-related metabolic disorders in patients undergoing treatment for sex hormone deficiency. In this report, we review literature regarding the role of estrogens and their corresponding receptors in the control of adipose metabolism and glucose homeostasis in both rodents and humans. We also discuss the effects of selective estrogen receptor modulators on glucose metabolism.
Fat is a more active organ in women.
Men taking T can have heart risks and metabolic issues, regardless of weight.
The data suggest that estrogen use in American Indian postmenopausal women may relate to deterioration of glucose tolerance. Longer duration of estrogen use among current users may relate to an increased risk of type 2 diabetes.
wouldn’t the pill do the same? is that why young women are obese with diabetes now?
The normal range of estrogen varies depending upon the patient’s age. Typically a women aged 20 to 29 will have an average level of 149 pg/ml (pictograms per milliliter). A female aged 30 to 39 will average a level of 210 pg/ml. And those over 40 but not in menopause will have an average level of 152 pg/ml. These average levels can vary day to day depending on each female’s menstrual cycle.
Yes, female estrogen peaks in the 30s.
The pedophiles like to try and bury that blood test fact by claiming nonsense about teens.
Stress also can contribute to a high estrogen level.
.According to a 2016 study in the Macedonian Journal of Medical Science, HRT could help treat insulin resistance due to low estrogen levels, although more research is needed before HRT can be said to effectively treat insulin resistance in women with low estrogen levels 13⭐
.are they suggesting fat people need more estrogen?
Signs of Insulin Resistance
According to Diabetes.co.uk, the signs of insulin resistance can include excessive fatigue, hunger, difficulty concentrating and weight gain
. Low estrogen levels may be associated with insulin resistance, which is also more likely to occur if you have gained excess weight or tend to carry fat in the belly area .
Diabetes.co.uk also states that insulin resistance can be improved by doing things like eating less carbohydrates, reducing calories, getting more exercise, reducing stress or even having weight loss surgery 13⭐
Really at this point it’s a controlled late abortion than a birth, isn’t it?
Globally, preterm birth and low birth weight (LBW) cause more than a third of the 2.9 million neonatal deaths each year, and prevention of these events is an important component of reducing the mortality rate among children younger than 5 years of age (1, 2). The causes of preterm birth, however, are complex, and few interventions have been successful in preventing it (3).
Eat some fucking cheese. So complex.
tfw Mother Nature hates you.
However, in a recent meta-analysis, Haider and Bhutta (10) concluded that multiple-micronutrient supplementation may reduce the risk of LBW and the number of stillbirths but not the risk of preterm birth or neonatal mortality. Thus, a more targeted micronutrient supplementation practice may be warranted.
You can’t supplement stupidity.
If they’d rather the baby die than get over themselves, they don’t deserve kids.
I love the ineffectual response of “supplements won’t work, but our solution is more of them”.
Your intellectual betters, behold!
Importantly, B12 deficiency may be a proxy for inadequate nutritional status, and it is possible that some of our findings are related to nutritional status and not specifically to B12. A predominantly plant-based diet is low in B12 but also other nutrients, such as vitamin D and zinc, that to some degree may be associated with preterm birth (42–44). We did not have information on dietary intake or blood levels of these nutrients. Nutritional status could explain the present finding of an association between B12 and birth weight in low- and middle-income countries but not high-income countries. However, lower vitamin B12 levels were associated with higher risk of preterm birth irrespective of country income. It seems less likely that nutritional status can fully explain this finding.
Vegetarianism: the Third World baby death diet.
Indian women generally have lower dietary intakes of B12 because of their mainly vegetarian diet, making them susceptible to B12 deficiency (46). Additionally, Indian newborns are among the smallest in the world (45). Our findings suggest that pregnancies already at the greatest risk of resulting in small newborns were the ones that were most vulnerable to low levels of B12. The association between B12 and the risk of preterm birth was consistent across studies in both high-income and low- and middle-income countries, and generalization to countries not studied may be feasible.
Stop eating vegan junk food.
In line with our findings, maternal obesity has been associated with B12 deficiency in several populations (47, 48). It has been hypothesized that this association is due to altered fat distribution and metabolism in overweight women compared with normal-weight women (47). Maternal weight is positively correlated with newborn weight (49), and failure to adjust for maternal weight may underestimate a positive association between B12 and birth weight.
For the many obese vegans: https://www.sciencedirect.com/science/article/pii/S1472648310616008
“In young women being overweight appears to be one of the major and still neglected causes of subfertility. Not only the excessive amount but also the distribution of body fat is clearly related to loss of fertility.”
We already knew it causes subfertility in men.
“These high concentrations of androgen and insulin in turn are important factors in the preferential abnormal localization of body fat.” aka Unhealthy women have boy hips and no waist.
(Actually, I’d pay good money to see a subfertility study in Asians, especially considering all the GMO soybean products).
“in addition, have a higher rate of miscarriages”
forcing pregnancy means sickly babies, a miscarriage where not stress-based is just rejecting the genome that would be an infant mortality statistic anyway
If they aren’t responsible for their body, why are they allowed a baby?
“Drugs increasing insulin sensitivity also improve spontaneous ovulation and fertility in obese women but still need to be tested in larger controlled trials.”
Why don’t you listen to Mother Nature and stop trying to force babies?
At least do a follow-up study on their own health risks to show the practice is safe.
Fertility is not an entitlement.
“had more positive feelings toward their dietary in-group (higher private regard)”
so vegans are racist?
“evaluated out-group dieters more negatively (lower out-group regard)”
yep, most meat-eaters in the world aren’t white…
Veganism was invented in 1944 in England, so clearly it’s racist.
“A retrospective review indicated that vegans should ensure adequate intake of a few nutrients.”
Supplements are either animal derived or tested on animals.
“”We found that some of these nutrients, which can have implications in neurologic disorders, anemia, bone strength and other health concerns, can be deficient in poorly planned vegan diets,”
“The study points out that some vegans rely heavily on processed foods and may not eat a sufficient variety of fruits, vegetables and whole grains.”
They also tend to consume non-organic coffee that’s been sprayed with fungicides, herbicides and pesticides.
Vegans dosing b12 probably don’t get it https://academic.oup.com/ajcn/article/108/3/525/5042715
“Approximately 85% of participants categorized as having low vitamin B-12 were taking vitamin B-12 supplements at doses in excess of the Recommended Dietary Allowance, which suggests that more research is needed to determine breast-milk adequacy values.”
again, women suffer more from stupid fad dieting, as the sex with a more complex body that’s intended to support another body in the form of a fetus
it suggests since their body isn’t adsorbing it that they’re still suffering brain shrinkage and also logically so too would the baby
You’d think that would be an important study to do.
Racial confound but okay https://academic.oup.com/ajcn/article/107/6/909/5032650
“4.5% lower body fat in vegan women”
thinner isn’t better, it isn’t healthier, especially for women
women need a higher body fat %
doesn’t control for source of meat
“Patterns of differences by diet group were similar in white men.”
Thinner men is also bad.
“In the Indian population, compared with meat eaters, vegetarian women were shorter (−1.1 cm) and had lower lean mass (−0.5 kg), and both vegetarian women and men had lower grip strength (−1.3 and −1.4 kg, respectively).” Malnutrition 101
This suggests an Asian subfertility study must be done.
They also had less lean mass… hmm….
“Differences in anthropometric and physiologic characteristics were observed across diet groups in white participants, but fewer differences were observed in British Indian participants.”
Diet is more important for white people who evolved to eat more meat during the Ice Age.
Water is wet.
And the word you use from the passport doesn’t count medically. They are not British, they are their bodies – Indian.
British Indian makes it sound like they belong to the Empire, technically they do. They can’t medically blend with their passport, they’ll never be British. Ever. They missed the local evolutionary events as we missed theirs.
We ain’t related, mate. You aren’t entitled to any claim on this place culturally. You will never belong here. A passport doesn’t make you a Martian either. It reminds me of Doctor Who psychic paper, just stop.
Someone tell them a passport is actually a license to draft. Including stupid dual passport holders thinking that places like Singapore don’t have any legal hold over them.
It isn’t a golden ticket. You’d have to have a permanently slow group IQ to think that.
Actually, diet x IQ study? Why no?
You have to scroll for the real findings.
“In contrast, both lacto-ovo-vegetarian and vegan females do not seem to have a lower risk of mortality from cardiovascular diseases. Not so clear patterns are observed for cancer outcomes. While lacto-ovo-vegetarians have lower risk of cancer of the gastrointestinal tract, vegans experience a higher risk for cancer of the urinary tract. For other-cancer sites, the risk is slightly but not significantly lower for both lacto-ovo-vegetarians and vegans compared to non-vegetarians. Subsequent reports with longer follow-up time and more cancer cases will help clarify the role of specific vegetarian diets with cancer outcomes.”
Little to no difference, especially for women.
Some cancer rates higher.
Any benefits are largely not being obese. If properly controlled for, that suggests cancer may be higher risk.
“In the EPIC-Oxford study , vegans had 30% higher fracture rates than meat-eaters. ”
“Vitamin B12 deficiency may increase CVD risk factors , and is associated for a wide range of neurological disorders . In addition, for those following a vegan diet, optimizing intakes of n-3 fatty acids is highly recommended ”
reconcile absorption study above
the lower cardio risk is an assumption based on supplementation, not the dietary fact per se
“large randomized dietary intervention trials on vegan and lacto-ovo-vegetarian patterns are warranted to duplicate the findings and further investigate the health effects of these diets.”
They’re mainly studying religious people, not the typical atheist vegan.
“The research on the health effects of vegan diets and chronic diseases have mostly derived from observational studies of Adventist and Oxford vegetarians cohorts”
That isn’t generalisable. “In general, the protective effects of vegetarian diets are stronger in men than in women.”
Paleo makes no sense considering all the diseases men are at risk of most, are made worse by eating tons of red meat. If you won’t listen to science, you’ll have to talk to Mr Prostate Cancer.
Where’s the sex realism in dietary advice? No, men can’t get away with eating like women either. Almost like there are chemicals varying between the two.
And body builders have atrocious health. I’d like to see a study on how unhealthy gyms are. Exercising in those are the worst places you can exercise. I heard there’s HPV on the machines.
Contrary to our expectation, epididymal and carcass fat depots and adipocyte size were significantly enlarged by 15, 72 and 68 %, respectively, in the IF mice compared with the ad libitum-fed mice. Accordingly, plasma levels of leptin were 50 % higher in the IF mice than in the ad libitum-fed mice. In addition, the IF mice showed increased plasma levels of total cholesterol (37 %), VLDL-cholesterol (195 %) and LDL-cholesterol (50 %).
Ask any rich woman about cortisol fat, this is not news to us. Higher testosterone makes this problem worse. Have you seen the way manly women expand once they hit menopause?
Manly – by build and bone structure. Not height or race. Does she have a waist or is she just skinny? Does she have bone structure or a manjaw? Prime example – Jolie. What does she do? Starve herself to stay thin, stressing out her body more. It’s a cycle.
Have you looked up leptin?
Obese people have more leptin, it suppresses hunger. The fact you don’t feel hungry starving is a red flag. Your body’s signals are wrong. Women feel full sooner, because we’re meant to have a higher body fat percentage and therefore more leptin is produced.
fine, I won’t buy those bars, neither will anyone else
adults have the money and won’t buy supposed child portions
communists can’t do market forces
nothing to do with currency devaluation, to pay for the newcomers, I’m sure
war on chocolate, never a war on alcohol, which contains the most natural sugar and empty calories without any balancing ingredients name and shame companies providing what is promised? trading standards anyone?
ooh some cuck penpusher is mad at you, better run and hide those profits because they can smell money and it makes them angry
irony is working class children used to gorge thousands of calories in chocolate during the industrial revolution, to have the energy from the cheap chocolate to work; it’s actually antibiotics, GFS and carbs like bread making them fat
Use it as marketing, proud to rebel against our insane and FAT civil servants.
coffee and tea are worse for you Fuck, why not go back to rations?
wait that would include champagne you socialist scum
If it’s about the sugar, ban it in restaurants, especially fast food – but noooooo they won’t do that. Ban butter too, processed bread and so on.
That would affect them.
The shock will be when their healthcare becomes expensive because people like them pushed up the prices (parts of the NHS here now charge) and finally, their kids won’t look after them because they were raised on Independence Dogma and Don’t Trust Anyone Over 30 and not Honour thy parents, which is outdated. One of those abusive care homes pour vous, I see in your future.
Health officials say the “sandwich generation” of people caring for children and ageing parents do not take enough time to look after themselves.
Boomer karma is a-coming.
You fucked the system. The system is fucked and fucking you.
And the cycle is complete.
You wanted to change society. No safety net for you. That’s a conservative thing to do.
You wanted it young? You get it while old too.
This is why you don’t meddle with the OS code for society. It used to work, now it will fail in the strangest ways and most unexpected (if you’re dumb) places.
No country for conservative values. It’s like the childless people I keep pointing out who expect a pension. From whose work, exactly? Why should somebody else’s children, somebody else’s investment of time and a LOT of money, work cuck for you? Is that not slavery?
That’s if the state slave pension isn’t totally abolished, that looks increasingly likely as new recruits with massive aged families claim and leech.
Common Filth is right, they don’t have a lifestyle. They have a deathstyle.
What you sow, you shall reap.
You don’t sow anything? Guess you starve.
There’s a 22-stone guy who supposedly didn’t know he was fat.
These people also wouldn’t see the beauty of epigenetics e.g. you snub the chub BEFORE you reproduce, or it carries through in the germline. How do the gluttons NOT see it causes their obesity?
It’s a biological fact. It’s a direct causal connection.
He says: “You can become very complacent when you are in your forties. You kind of think you’ve done everything and so you can relax and eat pizzas and Chinese in the week.”
No, that’s the lifestyle of a student. A teenager. When they can’t cook.
After cutting down on alcohol, Ms Henderson said she had more time for things, was less stressed, coped with work better and, that family life was more pleasant.
Shouldn’t they have done this two decades ago?
It’s like the stupidity of a deathbed conversion. The damage is still done and present, it doesn’t count. There is no sin eater in a calorie counter.
Overestimation of body weight among normal-weight adolescents is relatively uncommon; potentially a cause for celebration. However, almost half of boys and a third of girls with a BMI placing them in the overweight or obese BMI range perceived themselves to be about the right weight. Lack of awareness of excess weight among overweight and obese adolescents could be cause for concern.
It mentions ‘size acceptance’ once as a contradictory factor from healthy lifestyle programmes, chickening out in the Introduction, and keeps bringing up eating disorders unnecessarily, but it’s there, and fat boys are far more likely to think they’re normal.