The wages of sin – HPV in men and sperm infertility

https://pubmed.ncbi.nlm.nih.gov/32386620/

TLDR – NOT HARMLESS

Evaluation of human papilloma virus in semen as a risk factor for low sperm quality and poor in vitro fertilization outcomes: a systematic review and meta-analysis

A review of the literature regarding ART outcomes showed an association between HPV infection and decreased PR, and an even stronger association between HPV infection and increased MR.

-increased miscarriage rate, lower odds of conceiving

Conclusion: Our meta-analysis shows a negative effect of HPV on sperm concentration, motility, and morphology. Further subgroup and categorical analysis confirmed the clinical significance of impaired sperm motility in HPV-infected sperm, although the sperm count and morphology must be carefully analyzed. The studies reviewed reported lower PR and increased MR in couples with HPV-infected sperm. As most studies had a moderate risk of bias, these observations warrant further large, well-designed studies before introducing clinical management recommendations.

https://pubmed.ncbi.nlm.nih.gov/32279923/

Yes, this is a dealbreaker to sane women.

Human papilloma virus: to what degree does this sexually transmitted infection affect male fertility?

No abstract available

irony

MRAs: crickets

https://pubmed.ncbi.nlm.nih.gov/25992782/

Human papillomavirus infection and fertility alteration: a systematic review

Results: HPV infections are shown to be significantly associated to many adverse effects in the reproductive function. These adverse effects were reported in different levels from cells production to pregnancy and may be related to the infecting genotype.

Conclusions: It appears from this study that HPV detection and genotyping could be of great value in infertility diagnosis at least in idiopathic infertility cases. Like for the risk of carcinogenesis, another classification of HPV regarding the risk of fertility alteration may be considered after deep investigations.

https://pubmed.ncbi.nlm.nih.gov/30344281/

Human Papilloma Virus (HPV) and Fertilization: A Mini Review

Sorry but if something makes you less virile, you’re less of a man.

Human papilloma virus (HPV) is one of the most prevalent viral sexually transmitted diseases. The ability of HPV to induce malignancy in the anogenital tract and stomato-pharyngeal cavity is well documented. Moreover, HPV infection may also affect reproductive health and fertility. Although, the impact of HPV on female fertility has not been thoroughly studied it has been found also to have an impact on semen parameters. Relative information can be obtained from studies investigating the relationship between HPV and pregnancy success. Furthermore, there is an ongoing debate whether HPV alters the efficacy of assisted reproductive technologies. An association between HPV and assisted reproductive technologies (ART) programs has been reported. Nevertheless, due to conflicting data and the small number of existing studies further research is required. It remains to be clarified whether HPV detection and genotyping could be included in the diagnostic procedures in couples undergoing in vitro fertilization (IVF)/intrauterine insemination (IUI) treatments. Vaccination of both genders against HPV can reduce the prevalence of HPV infection and eliminate its implications on human fertility. The aim of the present mini-review is to reiterate the association between HPV and human fertility through a systematic literature review.

https://pubmed.ncbi.nlm.nih.gov/21666465/

The role of human papillomavirus on sperm function

I love how many yanks pull a Henry 8th and blame women for their own infertility, in this century.

Recent findings: HPVs are agents of the most common sexually transmitted disease and can lead to warts and cancers both in men and women. A high incidence of HPV infection has been demonstrated in sperm from sexually active men with and without risk factors for HPV and from infertile patients.

Semen infection is associated to an impairment of sperm parameters suggesting a possible role in male infertility. – really???

Interestingly, it has been demonstrated that when HPV is present in semen only a percentage of total cells are infected

-only? a? 100% is a percentage too…

and the virus can be localized in sperm or in exfoliated cells with different impact on sperm motility. Moreover, infected sperm are able to penetrate the oocyte, to deliver HPV genome in the oocyte and HPV genes can be actively transcribed by the fertilized oocyte.

-wouldn’t it be ironic if it made the kids or grandkids infertile instead? because they were conceived with it, a polluted germline

Recently an increased risk of pregnancy loss has been demonstrated in couples undergoing in-vitro fertilization and particularly when HPV DNA was present in semen samples of male partners.

– no blaming women this time, unless women haz sperm?

Summary: To date, no effective treatment, control strategy and prevention is provided for men despite the reported high incidence of HPV semen infection.

– no hurt their feefees? NAW

Because this infection in men is also a problem for partners, and because growing evidence suggests that semen infection may cause infertility and early miscarriage, more attention should be paid to male HPV infection. This study reviews the more recent literature about the role of HPV infection on sperm function and human reproduction.

– Manosphere fears this topic and all male degenerate accountability.

semen infection may cause infertility and early miscarriage

it’s the sins of the FATHER, you see…

https://pubmed.ncbi.nlm.nih.gov/30517657/

High-risk human papillomavirus in semen is associated with poor sperm progressive motility and a high sperm DNA fragmentation index in infertile men

Does the presence of human papillomavirus (HPV) in semen impact seminal parameters and sperm DNA quality in white European men seeking medical help for primary couple’s infertility?

>STD
>DNA quality
>in the germline of
>white men

Never talk about it, I’m sure it’ll be fine.

 HPV seminal infections involving high-risk (HR) genotypes are associated with impaired sperm progressive motility and sperm DNA fragmentation (SDF) values.

TLDR: yes.

HPV is commonly present in semen samples. 

No? F no it’s not. Stop sparing slutty blushes.

The overall rate of HPV positivity was 15.5%

so 1 in 7, uncommon at best. No normalizing pathology please.

And it varies majorly by race and sexuality. Not sex because it’s sexual, obviously.

 Sperm progressive motility was significantly lower (P = 0.01) while SDF values were higher (P = 0.005) in HPV+ men compared to those with no HPV. In particular, HR HPV+ men had lower sperm progressive motility (P = 0.007) and higher SDF values (P = 0.003) than those with a negative HPV test. Univariable analysis showed that HR HPV+ was associated with impaired sperm progressive motility (P = 0.002) and SDF values (P = 0.003). In the multivariable analysis, age, FSH levels and testicular volume were significantly associated with impaired sperm progressive motility (all P ≤ 0.04). Conversely BMI, CCI, smoking habits and HPV status were not. Only age (P = 0.02) and FSH (P = 0.01) were significantly associated with SDF, after accounting for BMI, CCI, testicular volume, smoking habits and HPV status.

It’s worse for the older men.

https://pubmed.ncbi.nlm.nih.gov/32381092/

Impact of human papillomavirus infection in semen on sperm progressive motility in infertile men: a systematic review and meta-analysis

Background: Human papillomavirus (HPV) has been considered as one of the most common sexually transmitted viruses that may be linked to unexplained infertility in men. The possible mechanisms underlying correlation between HPV infection and infertility could be related to the altered sperm parameters. Current studies have investigated the effect of HPV seminal infection on sperm quality in infertile men, but have shown inconsistent results.

Methods: We systematically searched PubMed, Embase, Web of Science and CNKI for studies that examined the association between HPV seminal infection and sperm progressive motility. Data were pooled using a random-effects model. Outcomes were the sperm progressive motility rate. Results are expressed as standardised mean difference (SMD) with 95% confidence interval (CI). Heterogeneity was evaluated by the I-square (I2) statistic.

Results: Ten studies were identified, including 616 infertile patients with HPV seminal infection and 2029 infertile controls without HPV seminal infection. Our meta-analysis results indicated that sperm progressive motility was significantly reduced in HPV-infected semen samples compared with non-infected groups [SMD:-0.88, 95% CI:-1.17 ~ – 0.59]. There existed statistical heterogeneity (I2 value: 86%) and the subgroup analysis suggested that study region might be the causes of heterogeneity.

Conclusions: HPV semen infection could significantly reduce sperm progressive motility in infertile individuals. There were some limitations in the study such as the differences in age, sample sizes and the number of HPV genotypes detected. Further evidences are needed to better elucidate the relationship between HPV seminal infection and sperm quality.

https://pubmed.ncbi.nlm.nih.gov/25659295/

Antisperm antibodies in infertile men and their effect on semen parameters: a systematic review and meta-analysis

what a mystery

The mechanism of ASA cause male infertility is not clear

does it look like HPV?

The present study illustrates that there was a significant negative effect of ASA on sperm concentration, sperm motility (a+b) and sperm liquefaction.

yes

https://pubmed.ncbi.nlm.nih.gov/26793663/

The prevalence of Human Papilloma Virus (HPV) infection in the oligospermic and azoospermic men

The current study shows that HPV infection can affect on sperm count and motility and decrease count of sperm cell and decrease motility capability of these cells.

duh?

Among 50 confirmed oligospermic male, 15 were HPV DNA positive (30%).

In azoospemic group we had 8 HPV DNA positive (40%) and in normal group just 3 of 20(15%) samples were positive.

-what r the odds?

we found statistical significant relationship for sperm count (p<0.05) and sperm motility (slow) (p<0.05) in oligospermic group positive samples compared with negative. In the present study, 13 HPV genotypes were detected among positive samples. HPV genotypes 16, 45 in the high risk group and 6,11,42 in the low risk group were more frequent than the others.

Medicine can’t spare you.

https://pubmed.ncbi.nlm.nih.gov/21536283/

Semen washing procedures do not eliminate human papilloma virus sperm infection in infertile patients

 Fifteen samples

-aka HALF

had HPV DNA on sperm and exfoliated cells. Sperm washing centrifugation showed no changes in the number of infected samples and in the percentage of infected cells. Ficoll and swim-up protocols induced a slight reduction in the number of infected samples (30 and 26, respectively).

no muh scientism and IVF cope

This study demonstrated that conventional sperm selection rarely eliminates HPV sperm infection. More attention should be paid to the reproductive health of infected patients because, not only can HPV be transmitted, but it may also have a negative effect on development of the fetus.

-may, LOL

a negative effect on development of the fetus

so even if they all married a virgin waifu, they’d infect her and have defective babies
comedy GOLD, 24K.

https://pubmed.ncbi.nlm.nih.gov/33763033/

Is HPV the Novel Target in Male Idiopathic Infertility? A Systematic Review of the Literature

Infertility is an important health problem that affects up to 16% of couples worldwide.

1 in 7, where have I heard THAT before….? [scroll up]

Male infertility is responsible for about 50% of the cases,

NAY, men are never responsible for their own in/fertility, have you been online recently?

and the various causes of male infertility may be classified in pre-testicular (for example hypothalamic diseases), testicular, and post-testicular (for example obstructive pathologies of seminal ducts) causes. Sexually transmitted infections (STI) are increasingly widely accepted by researchers and clinicians as etiological factors of male infertility. In particular, several recent reports have documented the presence of HPV in seminal fluid and observed that sperm infection can also be present in sexually active asymptomatic male and infertile patients.

In this review, we aimed to perform a systematic review of the whole body of literature exploring the impact of HPV infection in natural and assisted fertility outcomes, from both an experimental and a clinical point of view. Starting from in-vitro studies in animals up to in-vivo studies in humans, we aimed to study and evaluate the weight of this infection as a possible cause of idiopathic infertility in males with any known cause of conception failure.

https://pubmed.ncbi.nlm.nih.gov/30291691/

Significant Correlation between High-Risk HPV DNA in Semen and Impairment of Sperm Quality in Infertile Men

brace yourselves

guess the result

c’mon

go on
think

just guess

….

ready?

A total of 140 subjects participated in the current study. Among 70 confirmed infertile males, only 8 (11.43%) cases tested positive for high-risk HPV and all fertile men were HPV-negative. This data revealed a significant association between high-risk HPV and male infertility (P=0.03). The percentage of normal sperm morphology and sperm motility rate significantly declined in men infected with HPV (P<0.001).

and all fertile men were HPV-negative

oof and the sluts of both sexes are dying out, I am distraught.
The genetics of the future are fairing brighter than you’d think.

Conclusion: There was a significantly higher prevalence of high-risk HPV in infertile men than fertile men. HPV infection seemed to be a risk factor for male infertility. Additional, larger studies should be conducted to confirm the impact of HPV on male infertility.

Player burnout shall henceforth be dubbed HPV-driven infertility?

https://pubmed.ncbi.nlm.nih.gov/33666259/

2021

Association between human papillomavirus infection and sperm quality: A systematic review and a meta-analysis

Human papillomavirus (HPV) has a high incidence rate in both males and females.

-maybe where you live

HPV infection in women has been shown to affect fertility and lead to foetal death and pregnancy loss. However, research on HPV infection in men is limited.

-well the husbands are freshly infecting the wives so

-Ashley Madison wasn’t full of women stepping out, was it?

The aim of this study was to study the effect of HPV infection in semen on sperm quality and present the findings of previous studies through a meta-analysis. Databases including PubMed, MEDLINE, EMBASE, Web of Science, Cochrane Library, WanFang data and China National Knowledge Infrastructure were searched for relevant studies. A systematic review and meta-analysis were performed, and 17 studies were included for analyses based on a set criterion. Meta-analyses indicated that HPV infection in semen significantly reduced sperm concentration (SMD = -0.12, 95% CI: -0.21 to -0.03, p = .009), sperm motility (SMD = -0.55, 95% CI: -0.780 to -0.33, p = .000), sperm viability (SMD = -0.55, 95% CI: -0.780 to -0.33, p = .000) and sperm morphology (SMD = -0.34, 95% CI: -0.61 to -0.07, p = .015). The high-risk HPV (HrHPV) infection could significantly reduce sperm count (SMD = -0.65, 95% CI: -1.11 to -0.18, p = .007) compared with high-risk HPV (LrHPV) infection.

In conclusion, HPV infection in semen significantly reduced sperm quality, and the HrHPV infection could significantly reduce sperm count compared with LrHPV.

b-b-but what does that matter? – bluepills

https://pubmed.ncbi.nlm.nih.gov/33725837/

tick tock goes your biological clock, nobody can wait as long as they want
NOBODY

Male sperm quality and risk of recurrent spontaneous abortion in Chinese couples: A systematic review and meta-analysis

Conclusions: The results of this analysis support an association of sperm density, sperm viability, sperm progressive motility rate, normal sperm morphology rate, sperm deformity rate, as well as sperm DFI with RSA. 

IF you conceived, magically, it would kill your baby. REPEATEDLY.

https://pubmed.ncbi.nlm.nih.gov/8671172/

Semen parameters and sperm morphology in men in unexplained recurrent spontaneous abortion, before and during a 3 year follow-up period

Baby death aborts the defective DNA, HPV fucks with your sperm’s DNA. Water is wet.

HPV makes you biologically unfit. According to the ultimate test, the womb.

To investigate the role of the ‘male factor’ in the pathogenesis of recurrent spontaneous abortion (RSA), especially sperm morphology abnormalities, 120 previously selected couples with unexplained RSA were studied for sperm parameters retrospectively and prospectively. The patients were subdivided into three subgroups, depending on their reproductive outcome during the 3 years of follow-up study: (i) 48 RSA couples who achieved a successful pregnancy; (ii) 39 RSA couples who experienced further abortions, and (iii) 33 RSA couples who experienced infertility during the follow-up period. A semen analysis was performed twice at the time of inclusion in this study, and twice again during the 3 year follow-up period. No significant differences in semen parameters were observed between RSA males and fertile controls. Instead, significant differences were observed between the group of RSA couples who experienced infertility during the follow-up and the other two groups (RSA couples who achieved successful pregnancy and RSA couples who experienced miscarriages and no live birth during the follow-up) for sperm concentration (P < 0.01 and P < 0.01 respectively), sperm motility (P < 0.01 and P < 0.01 respectively) and sperm morphology abnormalities (P < 0.01 and P < 0.01 respectively).

dat p-value

MORE STUDIES

https://pubmed.ncbi.nlm.nih.gov/23278374

Sperm DNA fragmentation in couples with unexplained recurrent spontaneous abortions

(((((“”unexplained“”)))))

The aim of the present study was to evaluate the degree of sperm DNA fragmentation in couples with idiopathic recurrent spontaneous abortion (RSA) and in those with no history of infertility or abortion. In this cohort study, 30 couples with RSA and 30 fertile couples as control group completed the demographic data questionnaires, and their semen samples were analysed according to World Health Organization (WHO) standards (September 2009-March 2010) for evaluation of sperm DNA fragmentation, using sperm chromatin dispersion (SCD) technique. The percentage of morphologically normal sperm was significantly lower in RSA patients compared with control group (51.50 ± 11.60 versus 58.00 ± 9.05, P = 0.019), but not in other parameters. Additionally, the level of abnormal DNA fragmentation in the RSA group was significantly higher than in the control group (43.3% versus 16.7%, P = 0.024). Our results indicated a negative correlation between the number of sperm with progressive motility and DNA fragmentation (r = -0.613; P < 0.001). The sperm from men with a history of RSA had a higher incidence of DNA fragmentation and poor motility than those of the control group, indicating a possible relationship between idiopathic RSA and DNA fragmentation.

– idiopathic? Are you shitting me?

(((idiopathic)))

sure it is

sure

https://pubmed.ncbi.nlm.nih.gov/23042403/

Correlation of recurrent pregnancy loss with sperm parameters and sperm DNA fragmentation

This study has indicated that sperm from men with a history of RPL have a higher incidence of DNA damage and poor motility compared with fertile males.

Water is wet. Miscarriage is meant to happen to dodgy DNA.

https://pubmed.ncbi.nlm.nih.gov/22519675/

Sperm chromatin integrity may predict future fertility for unexplained recurrent spontaneous abortion patients

“unexplained” – just assume the echo for comedic effect by now

The RSA group was further separated into three subgroups, depending on their reproductive outcome during the 12 months after they were enrolled in the study: the pregnancy subgroup consisted of 43 men whose partners achieved a successful pregnancy up to at least the 24th week of gestation; the abortion subgroup included 31 men whose partners experienced further abortions; and the infertile subgroup had 37 men whose partners did not have any positive pregnancy test after regular, unprotected intercourse. Significantly lower proportion of sperm with normal morphology was found in the abortion subgroup (14.7 ± 4.3%) than in the control group (17.5 ± 5.0%). Sperm concentrations were significantly lower in the infertile subgroup (55.7 ± 24.1%) than in the controls (68.6 ± 27.8%). The rates of abnormal sperm chromatin integrity were significantly higher in the abortion (16.7 ± 7.7%) and infertile (16.3 ± 6.6%) subgroups, compared to the control group (13.0 ± 4.4%). Logistic regression analysis showed that the subsequent reproductive outcome of the 111 RSA patients was negatively correlated to the rates of abnormal sperm chromatin integrity. In conclusion, sperm chromatin integrity, sperm morphology, and sperm concentration were associated with future reproductive outcome of RSA patients. The sperm chromatin integrity was a significant predictor for future abortion and infertility.

But men are never responsible for miscarriage, perish the THOUGHT.

I mean – where are the STUDIES?!

https://pubmed.ncbi.nlm.nih.gov/21806662/

Cytochemical evaluation of sperm chromatin and DNA integrity in couples with unexplained recurrent spontaneous abortions

unexplained….. sigh, ok.

Our study showed that in the cases of RSA, slow motility had a significant reduction in comparison with controls and also spermatozoa of men from RSA group had less chromatin condensation and poorer DNA integrity than spermatozoa that obtained from fertile men with no history of RSA.

https://www.sciencedirect.com/science/article/abs/pii/S000293780133898X

Known for 20 years.

Human sperm deoxyribonucleic acid fragmentation by specific types of papillomavirus

Conclusion: Human papillomavirus type 16 and 31 deoxyribonucleic acid caused deoxyribonucleic acid breakages characteristic of apoptotic but not necrotic sperm.

CAUSED

The data suggest that these human papillomavirus types may adversely affect subsequent embryonic development after fertilization. Sperm deoxyribonucleic acid appears to resist human papillomavirus types 18, 33, and 6/11 or repairing mechanisms occurred. Although enhanced motility was found in human papillomavirus–exposed sperm, important velocity parameters were decreased, suggesting impaired sperm function.

-swimming in circles isn’t motility, really

damages your baby DNA, kills babies =/= harmless!

it’s a viral abortion, really

https://www.mdpi.com/2077-0383/10/4/717

Negative Impact of Elevated DNA Fragmentation and Human Papillomavirus (HPV) Presence in Sperm on the Outcome of Intra-Uterine Insemination (IUI)

i.e. no, you won’t just get IVF

We wanted to determine the sperm DNA fragmentation index (DFI) cutoff for clinical pregnancies in women receiving intra-uterine insemination (IUI) with this sperm and to assess the contribution of Human Papillomavirus (HPV) infection on sperm DNA damage and its impact on clinical pregnancies. Prospective non-interventional multi-center study with 161 infertile couples going through 209 cycles of IUI in hospital fertility centers in Flanders, Belgium. Measurement of DFI and HPV DNA with type specific quantitative PCRs (HPV 6, 11, 16, 18, 31, 33, 35, 39, 45, 51, 52, 53, 56, 58, 59, 66 and 68) in sperm before its use in IUI. Clinical pregnancy (CP) rate was used as the outcome to analyze the impact on fertility outcome and to calculated the clinical cutoff value for DFI. A DFI criterion value of 26% was obtained by receiver operating characteristic (ROC) curve analysis. Couples with a male DFI > 26% had significantly less CPs than couples with DFI below 26% (OR 0.0326; 95% CI 0.0019 to 0.5400; p = 0.017). In sperm, HPV prevalence was 14.8%/IUI cycle. Sperm samples containing HPV had a significantly higher DFI compared to HPV negative sperm samples (29.8% vs. 20.9%; p = 0.011). When HPV-virions were present in sperm, no clinical pregnancies were observed. More than 1 in 5 of samples with normal semen parameters (17/78; 21.8%) had an elevated DFI or was HPV positive. Sperm DFI is a robust predictor of clinical pregnancies in women receiving IUI with this sperm. When DFI exceeds 26%, clinical pregnancies are less likely and in vitro fertilization techniques should be considered

When HPV-virions were present in sperm, no clinical pregnancies were observed.

but CLEARLY this is just my OPINION – misogynists reee-ing

https://www.sciencedirect.com/science/article/abs/pii/S0165037813000508

Sperm viral infection and male infertility: focus on HBV, HCV, HIV, HPV, HSV, HCMV, and AAV

Chronic viral infections can infect sperm and are considered a risk factor in male infertility. Recent studies have shown that the presence of HIV, HBV or HCV in semen impairs sperm parameters, DNA integrity, and in particular reduces forward motility. In contrast, very little is known about semen infection with human papillomaviruses (HPV), herpesviruses (HSV), cytomegalovirus (HCMV), and adeno-associated virus (AAV). At present, EU directives for the viral screening of couples undergoing assisted reproduction techniques require only the evaluation of HIV, HBV, and HCV.

-all trust the EU guys

However, growing evidence suggests that HPV, HSV, and HCMV might play a major role in male infertility and it has been demonstrated that HPV semen infection has a negative influence on sperm parameters, fertilization, and the abortion rate.

-somebody else look up herpes, I’m lazy

Besides the risk of horizontal or vertical transmission, the negative impact of any viral sperm infection on male reproductive function seems to be dramatic.

-Really, f-ing fascinating!

In addition, treatment with antiviral and antiretroviral therapies may further affect sperm parameters. In this review we attempted to focus on the interactions between defined sperm viral infections and their association with male fertility disorders. All viruses considered in this article have a potentially negative effect on male reproductive function and dangerous infections can be transmitted to partners and newborns. In light of this evidence, we suggest performing targeted sperm washing procedures for each sperm infection and to strongly consider screening male patients seeking fertility for HPV, HSV, and HCMV, both to avoid viral transmission and to improve assisted or even spontaneous fertility outcome

>male fertility disorders

k.

Oh, I’m not done yet.

https://www.cambridge.org/core/journals/epidemiology-and-infection/article/hpv-infection-in-semen-results-from-a-new-molecular-approach/B0B63D2A2760A03FCFF243F1DD5E9A7F

HPV infection in semen: results from a new molecular approach

Let’s get molecular.

Human papillomavirus (HPV) is the agent of the most common sexually transmitted diseases causing a variety of clinical manifestations ranging from warts to cancer. Oncogenic HPV infection is the major cause of cervical cancer and less frequently of penile cancers. Its presence in semen is widely known, but the effects on fertility are still controversial. – how? allergic to facts?

We developed a new approach to evaluate virus localisation in the different semen components. We analysed also the specific genotype localisation and viral DNA quantity by qPCR. Results show that HPV DNA can be identified in every fraction of semen: spermatozoa, somatic cells and seminal plasma. Different samples can contain the HPV DNA in different fractions and several HPV genotypes can be found in the same fraction. Additionally, different fractions may contain multiple HPV genotypes in different relative quantity. We analysed the wholeness of HPV DNA in sperm cells by qPCR. In one sample more than half of viral genomes were defective, suggesting a possible recombination event. The new method allows to easily distinguish different sperm infections and to observe the possible effects on semen. The data support the proposed role of HPV in decreased fertility and prompt new possible consequences of the infection in semen.

>HPV DNA can be identified in every fraction of semen: spermatozoa, somatic cells and seminal plasma

If you’re wondering why your nation is infertile, look in the mirror. Mutant sperm.

Your superpower is probably autism.

Sexual studies dump

This should keep you all busy for a while.

https://pubmed.ncbi.nlm.nih.gov/26332467/

Women’s Preferences for Penis Size: A New Research Method Using Selection among 3D Models

Women’s preferences for penis size may affect men’s comfort with their own bodies and may have implications for sexual health. Studies of women’s penis size preferences typically have relied on their abstract ratings or selecting amongst 2D, flaccid images. This study used haptic stimuli to allow assessment of women’s size recall accuracy for the first time, as well as examine their preferences for erect penis sizes in different relationship contexts. Women (N = 75) selected amongst 33, 3D models. Women recalled model size accurately using this method, although they made more errors with respect to penis length than circumference. Women preferred a penis of slightly larger circumference and length for one-time (length = 6.4 inches/16.3 cm, circumference = 5.0 inches/12.7 cm) versus long-term (length = 6.3 inches/16.0 cm, circumference = 4.8 inches/12.2 cm) sexual partners. These first estimates of erect penis size preferences using 3D models suggest women accurately recall size and prefer penises only slightly larger than average.

but different races have sexually selected different averages, even by nation? So the mean IS the ideal.

https://pubmed.ncbi.nlm.nih.gov/19139986/

The association between penis size and sexual health among men who have sex with men

Larger penis size has been equated with a symbol of power, stamina, masculinity, and social status. Yet, there has been little research among men who have sex with men assessing the association between penis size and social-sexual health. Survey data from a diverse sample of 1,065 men who have sex with men were used to explore the association between perceived penis size and a variety of psychosocial outcomes. Seven percent of men felt their penis was “below average,” 53.9% “average,” and 35.5% “above average.” Penis size was positively related to satisfaction with size and inversely related to lying about penis size (all ps < .01). Size was unrelated to condom use, frequency of sex partners, HIV status, or recent diagnoses of HBV, HCV, gonorrhea/Chlamydia/urinary tract infections, and syphilis. Men with above average penises were more likely to report HPV and HSV-2 (Fisher’s exact p <or= .05). Men with below average penises were significantly more likely to identify as “bottoms” (anal receptive) and men with above average penises were significantly more likely to identify as “tops” (anal insertive). Finally, men with below average penises fared significantly worse than other men on three measures of psychosocial adjustment. Though most men felt their penis size was average, many fell outside this “norm.” The disproportionate number of viral skin-to-skin STIs (HSV-2 and HPV) suggest size may play a role in condom slippage/breakage. Further, size played a significant role in sexual positioning and psychosocial adjustment. These data highlight the need to better understand the real individual-level consequences of living in a penis-centered society.

so men with big wangs, at least the ‘gay’ ones, are rendering themselves infertile or birth defect risks by sharing it around. Y NO str8 studies?

https://pubmed.ncbi.nlm.nih.gov/32201145/

Female Orgasm and Overall Sexual Function and Habits: A Descriptive Study of a Cohort of U.S. Women

Results: FSFI scores, which were calculated for the 230 women who reported having had a steady male sex partner in the preceding 6 months, showed that 41% of the 230 women were at risk for female sexual dysfunction (a cutoff less than 26.55) and 21% were dissatisfied with their overall sexual life. Almost 90% of the overall cohort reported good emotional contact with their partner, that their partner was willing to have sex, satisfaction with the partner’s penis size (wherever applicable), and good erectile function and ejaculatory control of their partner (wherever applicable). 81% of the overall cohort claimed to be sexually active. Around 70% (70-72) did reach orgasm frequently, but around 10% never did so. Vaginal intercourse was reported by 62% of the overall cohort as the best trigger of orgasm, followed by external stimulation from the partner (48%) or themselves (37%). External stimulation was reported to be the fastest trigger to orgasm.

Clinical implications: The knowledge on how women reach orgasm and how it is related to the partners’ willingness to have sex and other factors can be incorporated in the clinical work.

Strengths & limitations: The use of a validated questionnaire and the relative large number of participants are strengths of the study. Limitations are the cross-sectional design, the lack of a sexual distress measure, and a possible selection bias.

Conclusion: Most women in the overall cohort were satisfied overall with their sexual life and partner-related factors, even though 41% (of those who cited a steady sex male partner) were at risk for female sexual dysfunction. Most women did reach orgasm through different kinds of stimulation. Correlation was good between preferred and performed sexual activities and positions. Shaeer O, Skakke D, Giraldi A, et al. Female Orgasm and Overall Sexual Function and Habits: A Descriptive Study of a Cohort of U.S. Women. J Sex Med 2020;17:1133-1143.

I bet the inorgasmic had ‘cut’ men, who produce fewer orgasms in all women in studies.

https://pubmed.ncbi.nlm.nih.gov/22729523/

Impact of penile size on male sexual function and role of penile augmentation surgery

Penile augmentation is an ongoing debate. Emerging evidence proves a relationship between dissatisfaction with penile size and sexual dysfunction. Despite a widespread belief of the value of penile size, and the prevalent complaint of a “small penis,” penile augmentation still stands short of addressing the demand. This report highlights the studies added to medical literature on penile augmentation in 2011-2012, including data on normal penile dimensions and the expected dimensions for a normal person, determination of the prevalence of the dissatisfaction with penile size, its effect on erectile function, female partner’s opinion of the value of penile size, as well as further experience with augmentation techniques.

It’s mental, they need therapy. Also, I see the joke.

https://pubmed.ncbi.nlm.nih.gov/26639576/

Why Are Men Satisfied or Dissatisfied with Penile Implants? A Mixed Method Study on Satisfaction with Penile Prosthesis Implantation

The issue is mental?

Content analysis revealed four main themes for men’s satisfaction with the PPI: (i) psychological factors were reported 54 times (n = 54) and included positive emotions, self-esteem, confidence, enhancement of male identity, major live change, and self-image;

– mental

(ii) improvement of sexual function was reported 54 times (n = 54) and referred to achievement of vaginal penetration, increase of sexual desire, sexual satisfaction, penis size, and improvement of erectile function;

– satisfaction is a female metric? ED is mental?

(iii) relationship factors were reported 11 times (n = 11) and referred to relationship improvement and the possibility of giving pleasure to the partner; and (iv) improvement in urinary function (n = 3).

– not possible with implants, all surgeries produce numbness and see above study

https://pubmed.ncbi.nlm.nih.gov/18649002/

Association between human papillomavirus in men and their sexual partners and uterine cervical intraepithelial neoplasia

“The sample size employed was not large enough to be able to determine any differences between both study groups.” aka useless study

Stop linking to bullshit. Slutty men are just as bad for society as the women. Blame shifting is blue pill.

https://pubmed.ncbi.nlm.nih.gov/33793040/

A preliminary investigation of a novel method to manipulate penis length to measure female sexual satisfaction: a single case experimental design

Results: On average reducing the depth of penetration made a statistically significant 18% reduction of overall sexual pleasure with an average 15% reduction in length of the penis. The longer the erect penis, the less likely the rings had an impact on sexual pleasure. There was however a range of individual responses with a minority of women reporting that reducing the depth of penetration was more pleasurable on some occasions.

– the G spot?

Conclusions: Size may matter in women in a healthy stable relationship when there is penile shortening. Because of the small number of couples and men with an apparently long penises, our results are preliminary, and we welcome replication in a larger sample with a more diverse range of penile lengths. Our results should not be misinterpreted as meaning that increasing penile length will increase sexual pleasure in women.

shortening with age, and no, supplements don’t help

unless you want earlier mortality (T-levels)

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5715194/

Non-pharmacological and non-surgical strategies to promote sexual recovery for men with erectile dysfunction

-quit the porn? sigh

the tobacco-lung cancer connection of this generation
history will know you as a bunch of wankers

Erectile dysfunction (ED), the most commonly reported sexual problem for men,

in dick-mutilated, porn-addled America? also Israel?

reduces the quality of life for both patients and their partners. Even when physiologically effective, long-term adherence to ED treatments is poor.

– it’s behavioral…

We review here the implication of having patients’ partners involved in ED treatment, starting with treatment selection. We suggest that having partners engaged from the outset may promote an erotic association of the treatment with the partner, i.e., conceptually linking the aid to the sexual pleasure that the partner provides.

– quit ‘supernormal stimuli’ aka lechery, your dopamine is fried?

We hypothesize that this erotic association should enhance the sexual aid’s effectiveness and might potentially help improve long-term adherence. The primary focus of this review, though, is non-pharmacological and non-surgical options for maintaining sexual activity for men with ED. Though not ED treatments per se, anecdotal data suggest that these options may be effective for some patients and their partners in regaining a satisfying sex life. The aids discussed include external penile prostheses, penile sleeves, and penile support devices.

-easier just to give up the sexual visual crack, surely?

These devices can allow men to participate in penetrative sexual intercourse despite moderate to severe ED.

-aging happens, it’s natural selection

External penile prostheses can be personalized so they match in size and shape a man’s normal full erection.

– that is sad

Penile sleeves can similarly be customized with a lumen that fits best a patient’s penis for optimal tactile stimulation.

– so they’re fucking a toy? …. not the man…..

We review how multi-sensory integration can enhance sexual arousal for men who use such devices, allowing them to achieve orgasm despite intractable ED.

-it’s a silicon sock

Patients are not always advised within ED clinics about these options nor why and how they can facilitate non-erection dependent sexual recovery. Clinicians need to be aware of these devices and their positive attributes, so they can objectively counsel and encourage couples to explore their use as an alternative to more invasive treatments. The most commonly promoted non-medical ED aid offered to patients is the vacuum erection device. We discuss how erections achieved with the vacuum erection device have a “hinge effect”, that is an underappreciated barrier to the effectiveness of the erection. With a hinged erection, the penis points downward rather than upward. We show how the normal kinematics of the penis during coitus is not strictly linear (i.e., not uniaxial; not just in-and-out), and is impeded by hinging. Positional adjustment, such as the receptive partner being on top, may help overcome this problem for some couples. Lastly, we suggest that, in the case where ED can be anticipated from a pending medical treatment, such as a prostatectomy, pre-habilitative approaches may potentially improve adherence to sexual aid use in the long-term. In conclusion, non-pharmacological and non-surgical options for sexual recovery are available. Scientific studies on the effectiveness of these interventions in restoring satisfying sex are warranted.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5039517/

Is Internet Pornography Causing Sexual Dysfunctions? A Review with Clinical Reports

Traditional factors that once explained men’s sexual difficulties appear insufficient to account for the sharp rise in erectile dysfunction, delayed ejaculation, decreased sexual satisfaction, and diminished libido during partnered sex in men under 40.

-no shit

This review (1) considers data from multiple domains, e.g., clinical, biological (addiction/urology), psychological (sexual conditioning), sociological; and (2) presents a series of clinical reports, all with the aim of proposing a possible direction for future research of this phenomenon. Alterations to the brain’s motivational system are explored as a possible etiology underlying pornography-related sexual dysfunctions. This review also considers evidence that Internet pornography’s unique properties (limitless novelty, potential for easy escalation to more extreme material, video format, etc.) may be potent enough to condition sexual arousal to aspects of Internet pornography use that do not readily transition to real-life partners, such that sex with desired partners may not register as meeting expectations and arousal declines. Clinical reports suggest that terminating Internet pornography use is sometimes sufficient to reverse negative effects, underscoring the need for extensive investigation using methodologies that have subjects remove the variable of Internet pornography use. In the interim, a simple diagnostic protocol for assessing patients with porn-induced sexual dysfunction is put forth.

– it’s a supernormal stimulus designed to make you impotent, it’s a delusion that alters the brain

Based on the findings of historical studies cited earlier, older men would be expected to have far higher ED rates than the negligible rates of younger men [2,7]. However, in just a decade, things changed radically. The 2001–2002 rates for older men 40–80 were about 13% in Europe [5]. By 2011, ED rates in young Europeans, 18–40, ranged from 14%–28% [6].

duh, porn addicts also look older, do that study

Finally, another 2015 study on men (mean age approximately 36), reported that ED accompanied by a low desire for partnered sex is now a common observation in clinical practice among men seeking help for their excessive sexual behavior, who frequently “use pornography and masturbate” [15].

aimed at Europeans, squarely

make them hypersexual, extreme r, and re-productively useless

The sole qualification for being a masculine man is having a functioning dick but they cuck for the industry ruining their lives.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6679165/

The Potential Associations of Pornography Use with Sexual Dysfunctions: An Integrative Literature Review of Observational Studies

This paper reviews the associations between pornography use and sexual dysfunction based on evidence from observational studies. The existing data in this regard mostly derive from cross-sectional investigations and case reports. There is little if no evidence that pornography use may induce delayed ejaculation and erectile dysfunction, although longitudinal studies that control for confounding variables are required for a full assessment.

– decades later, we’ll be dead demographically…

The associations between pornography use and sexual desire may differ between women and men although the existing data is contradictory and causal relationships cannot be established. The strongest evidence is available for the relation of pornography use with decreased sexual satisfaction, although the results of prospective studies are inconsistent. The paper outlines future research prospects beneficial in understanding the nature of associations between pornography use and sexual dysfunctions in men and women.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6352245/

Online Porn Addiction: What We Know and What We Don’t—A Systematic Review

In the last few years, there has been a wave of articles related to behavioral addictions; some of them have a focus on online pornography addiction. However, despite all efforts, we are still unable to profile when engaging in this behavior becomes pathological.

– publishing bias

Common problems include: sample bias, the search for diagnostic instrumentals, opposing approximations to the matter, and the fact that this entity may be encompassed inside a greater pathology (i.e., sex addiction) that may present itself with very diverse symptomatology.

= excuses so you don’t get later sued

Behavioral addictions form a largely unexplored field of study, and usually exhibit a problematic consumption model: loss of control, impairment, and risky use. Hypersexual disorder fits this model and may be composed of several sexual behaviors, like problematic use of online pornography (POPU). Online pornography use is on the rise, with a potential for addiction considering the “triple A” influence (accessibility, affordability, anonymity). This problematic use might have adverse effects in sexual development and sexual functioning, especially among the young population. We aim to gather existing knowledge on problematic online pornography use as a pathological entity. Here we try to summarize what we know about this entity and outline some areas worthy of further research.

dupe

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2874247/

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4994844/

brain differences in the slutty

Risky sexual behaviors typically occur when a person is sexually motivated by potent, sexual reward cues. Yet, individual differences in sensitivity to sexual cues have not been examined with respect to sexual risk behaviors. A greater responsiveness to sexual cues might provide greater motivation for a person to act sexually; a lower responsiveness to sexual cues might lead a person to seek more intense, novel, possibly risky, sexual acts. In this study, event-related potentials were recorded in 64 men and women while they viewed a series of emotional, including explicit sexual, photographs. The motivational salience of the sexual cues was varied by including more and less explicit sexual images. Indeed, the more explicit sexual stimuli resulted in enhanced late positive potentials (LPP) relative to the less explicit sexual images. Participants with fewer sexual intercourse partners in the last year had reduced LPP amplitude to the less explicit sexual images than the more explicit sexual images, whereas participants with more partners responded similarly to the more and less explicit sexual images. This pattern of results is consistent with a greater responsivity model. Those who engage in more sexual behaviors consistent with risk are also more responsive to less explicit sexual cues.

– it’s r/K

trans. r-types seem to have dulled sexual perception (abnormal) so over-estimate opportunity in compensation

Reminder:

Mother Nature WILL have her pound of flesh.

men aren’t equal, either

also

No wicked man knows happiness, and least of all the seducer of others.

The Hollywood-worshiping whores are shameless, do not pity any of them. They chose to be alone, often in foreign lands.
The manosphere constantly fails (in its stated goals) because it also sells a delusion of equality, but exclusively to men. It denies men their manhood in masculine competition with one another. With nothing to prove or reach for, they are pulled down into the bucket by their own crabs of apathy and nihilism, with a smattering of genuine misogyny at times, to further cast off blame for the fruits of their manly agency (really shades of cowardice). They play themselves.

All in all, looking at photos like that, I understand my elder male relatives: we need a good war. Such decadence cannot endure long, degeneracy never lasts. It is the rule of history. “We’ve” had the greatest rise in history so I fear our collective downfall. As one party of many latter generations who shirked the poison chalice, this is frustrating. It doesn’t matter if you were AT the party if they were personally trashing your house (the West). The libertarian arguments of social harm (muh individualism) are sociopathic and atomistic, they don’t scale up to a village, let alone nation or race. They are anarchists in suits, moral relativists on the fence with a calculator. Fine, be free to do drugs as you please, and equally free to die from them, yes? No bailouts, no manmade exceptions to natural law. No synthetic sin-eating like antibiotics for slags. No exceptions at all for pity’s sake. And if men wish to leave the West, deserters in our time of demographic crisis, we must respect that r-select impulse by banning their return. A house divided and all, no man may serve two masters (or nations). No anchor babies, anchor wives or anchor dual financial lives.

The cucks must be shucked.

The thirst after fame is greater than that after virtue; for who embraces virtue if you take away its rewards? – Juvenal

The Hollywood siren song has been going for a long time. Everyone falls for it, whether or not we act upon it. It’s pre-critical (Piaget) childhood programming. So-called incels should attack Hollywood. Why is the Tony Stark playboy stripper pole stuff in a KID’s film? Why do twelve-year olds need to see a foursome? 18:41 Subtext nobody dare address: successful men must be a slut. Is that true to reality? No. Quite the opposite, it’s insulting and demeaning to agency, but they own Trojan stock and other corporations (read: penicillin shots) that would go bankrupt if the moral bankruptcy stopped. The anti-capitalists oddly never point this out.

How wrong would that one scene seem, if the sexes were reversed? Our natural reaction is dulled by repetition, and children viewed this.

in full;

Refrain from doing ill; for one all powerful reason, lest our children should copy our misdeeds; we are all too prone to imitate whatever is base and depraved.

Family units are made by avoiding family destroyers

Weird I’m shown this after watching ONE Better Bachelor video. Almost like these education videos make the r-types reeee. Hey, when anything gets big it becomes mainstream info and you can’t put the rattlesnake back in the bag. The narcissism signs channels also get pushed (go incognito to see)…. silly broflakes. Aren’t you ‘proud’ if you’re doing ‘nothing wrong’?

Much is said online of f-f gaslighting, like SJW propaganda that all men are scum but m-f gaslighting is more damaging to society and its future prospects, especially when deceptively aimed at sheltered K-women to impair bonding and exploit that damage. You get the rare female ‘player’ types like Amber Heard but all these antisocial tricks are sociopathic so it’s just ASPD prevalence in the population anyway. Sociopaths are scum, there are just more male ones. If I didn’t grow up in a redpill household I might’ve fallen for some of these myself, women are too trusting, it’s the oestrogen. It’s a genetic fallacy to assume the quality of men acting toward women is the same as the 1950s, let alone 1850s, and since men lead society, the bad example brings everyone down by standard. Moral decline is male-led.
However, society (America, cornerstone of The West) has become more psychopathic – hedonic, sexually promiscuous, violent crime/sadistic media, no future goals, parasitic lifestyle, compulsive (sweet) lies, takes no responsibility (hypoagency in adults). The Weinsteins have written the rulebooks. Hollywood romcoms suck ON PURPOSE. Happily married women don’t go to see them!
Men are natural liars, hence the glibness and superficial charm of psychopaths, who are almost exclusively male. Also, all great actors are men because men are natural born liars, character is overcoming that for integrity. That’s why female character doesn’t work the same and female liars are considered masculine (usually lesbian). If you have sisters, mothers, daughters, friends you care about, help the fellow Ks to avoid r-types in ALL forms. There is no Sisterhood or Bro Code, those are collectivist (multicultural*) r-select psyops. They are thought-terminating cliche to gaslight a group that translate as ‘injure your prospects to benefit me’. Toxic friendships are one thing, avoiding deadbeats saves LIVES.

*anti-patriotic

Shannon writes: “Tell yourself all the things you want to hear. “I’m beautiful, smart,etc…so you are not waiting for a man to tell you what you already know. Men will say and do anything to just use you. Know your worth!”

Good advice to avoid all glib predators. I grew up hearing nice things so when I heard them from users, I just shrugged and agreed because my parents loved me…. this pissed them off. This was instructive. Predators hate healthy self-esteem so they try to enable the insecurity of the young (parenting other people’s kids – creepy!) to make for easier pickings later or outright target the young (legally or illegally). Pedos are just the lazy sexual predators, all humans are objects to them, some on the lowest shelf to grab.
‘Game’ is not normal, it’s a term describing the gaslighting of sexual slavery. It normalises sexual predation, something patriarchy always condemned. It is actively anti-K by exploiting that gene pool and damaging its monogamous courtship model, it’s sexual strategy sabotage of Ks by r. We need to establish ways to raise good children who can navigate the low-trust psychopathic society. There’s no ability to mince words because well-raised children ARE naive.

Monet: “I always hear men say “Women don’t know what they want” heard that my whole life everywhere”
Only from the low IQ ones, projecting. They wait to see a woman they like and conform to her, for a while. PDs do this, especially BPD and HPD. Women seem to have life goals as teens: marriage yes, kids yes, where to live. Men are more freeform and the dumb ones have no concrete plans at all. Voting was based on property ownership to exclude the useless nomadic bachelor and Sparta actively taxed them for being parasites. Low IQ people project, it’s just that statistically, there are more low IQ men than women and society doesn’t notice the women due to normal socialisation skills.
Anytime you hear a sweeping statement complaint, with no data (or the contrary data) then it’s a projection. So much cope. If women were aimless and useless, the female suicide rate would dwarf the male and we don’t live in that world. The low IQ complain of a thing while also denying it in the same sentence because they assume everyone else is too stupid to notice too. Women have higher verbal IQs, that’s why women are turned off so many men. It’s our way of testing their IQ. So women want to TALK – in person, which shits up low IQ men. They panic without the digital crutch. Anyone can sound smart online (allo allo allo). They have to check-in with other men what to say because they’re too stupid to form a sentence (ghetto women too, both groups low IQ). The romance novel leads are always ELOQUENT. PUAs sell communication copy/paste hackjobs.
“Women don’t know what they want” – projection AND gaslighting. Women aren’t confused, they just disagree with you – probably because you’re stupid. These men who constantly bump into ‘crazy’ people never like to be asked, have you ever tried to see it through their eyes?

Low IQ has no insight, no empathy.

They need forums of other predators because their interpersonal retardation requires a collective hivemind, spoon-feeding ‘insights’ by crowd sourcing. They cannot think by themselves. They read every trendy self-improvement book going*, too. Trash brains.

Have you ever considered, you might be stupid? Smart people do. Stupid people don’t (fragile ego) and never ever improve, so all other people are ‘crazy’ or have an ‘attitude problem’. Maaaybe it’s you. Low IQ cannot mentalise somebody smarter than they are, so cognitive dissonance rationalises as ‘crazy’ or in cases of morality ’emotional’. Yes, serial killers view it like this too. They only killed three people omg.

Arm mothers and daughters with wisdom.

*Antisocial predators hate people discussing their ‘tactics’. Even tactics is aggressive language re intimacy. It’s not a war. The violence is implied by a predator/prey dynamic. A sneaky underhand coercive predation. Greene’s ‘seduction’ book (seduction is still illegal on American law books) calls the woman the ‘target’ – but all they want is fuzzy romance, right?

The Sexual MLM

The MLM aspect is trading on CENTURY-OLD hypnosis adverts.

H Y P N O S I S

no shit

accurate comment: “Guys like these are the reason so many women are single. It’s a bad circle: women avoid men because they act like this, and then guys say women don’t like nice men and act even more like douches because women avoiding men is all the women’s fault -_-“

Well, scientifically women are happier being single than men (as surveys of married women showed, when hubby wasn’t around to sway them) https://www.independent.co.uk/news/uk/home-news/women-happy-children-spouse-partner-relationship-unmarried-a8931816.html

The manosphere is not redpill because they ignore this fact and other data e.g. female lifespan when married drops, male increases. How is anybody faking their life expectancy, paranoid pansies?

What’s the difference between running around after one woman who loves you and bore your heirs versus running around more after lots of women who don’t actually respect you? They’re still wasting their lives on women, that’s sad. Crushes are not respect. They are reviled. Women don’t think like men. Respect is rare, women don’t respect 99.9% of men they ever met, not truly. Where’s the study? Someone tell Woodley. I’ve respected two men I’ve known IRL the entire time I’ve been alive. Women don’t respect money, women respect morally. The concept of honour needs study.

There are too many super-mum duties and pressures to make it worthwhile nowadays, modern marriage isn’t worth it for women, he’ll still cheat with porn. Why bother? women question, I hear them of all political stripe. We can’t work two full-time jobs (the money job and mothering) plus house cleaner plus chef plus the energy for depraved sexuality. What does the man even do anymore in postmodern marriages except sperm donor? No protection, no patriotism, no religion (conversions don’t count after the party is over), no breadwinning exclusively, so what? Why? What is the gender role of men getting married now, their duty, since they expect the woman to be wife, mother and breadwinning husband, therapist and his Mommy like he can’t do his own laundry after all that. I’m not American so I can see it a mile off. That’s why your birth rate will continue to plummet.

The lower IQ men who buy sexual MLM material are generally too stupid to get the message of collective female retreat: man-up. Gentleman. Marriage advice from bachelors is like exercise routines from the obese. Imagine if this guy married and had a daughter, what kind of father would he be? This is how women think and FGTOW is older than MGTOW. Why should a woman (any woman*) shorten her lifespan by marrying him? In the age of no-fault divorce because of Ashley Madison adultery? Men are not equal so not equally viable as husband, rejects always existed pre-internet, women just joined the convent. Men want the genetic legacy, women don’t care if they take care of their children or other family. That’s why patriarchy forced them culturally to marry. SJWs and CHINO types both baby other women’s kids in schools and church groups. Wake up. They get all the dopamine with NONE of the maternal mortality.

*Sexual selection belongs to women, the sexual MLM is a ponzi that promises to reverse literal evolutionary forces in the species. Women would rather have no kids than those of PUA types. Get it through your skull. Otherwise lonely Boomer men committing suicide is their destiny.

Psychopaths are also narcissists and online their MO is known to include a ‘harem garage’ so that psycho isn’t even original. He’s ripping off clinical narcissism discussions directly.

comment “That metaphor is so creepy and terrible because it’s a term used in human trafficking. He knew what he was doing, and he’s pretending it’s “just a metaphor””

It’s dehumanization.

comment “As a horse trainer I was appalled by that mare/stable “metaphor” 🤮🤮 this guy is so gross. Like can we stop using animals to make sexual analogues please it’s disgusting Also mares are the ones who run the herd so if his analogy of coming in and out of the stable was real it would be wrong. The mares (specifically the lead mare) they would be the ones deciding who comes in and out. 💀💀 the “stallion” would not decide if the “thoroughbred” stays or not.” LOL

It’s sexual AA for non-whites (most PUAs, if you look into it) combined with an MLM – take this course to sell this course… but English majors becoming English teachers are a Ponzi? Really? Do you really need a programme telling you how to be a gaslighting r-selected creep? It’s like teaching fish how to swim. The course Ponzi is the new one after e-books failed to account for Americans refusing to fucking read (try the Bible, y’all need Jesus) so figured they’d pay for Youtube videos like a sexual version of skillshare’s hipster welfare ponzi full of people you’d never heard of with results saying ‘trust me’ ’cause they’re not married but they COULD BE. If they WANTED, get off my back MOM I don’t wanna give you GRANDKIDS you’re oppressing ME. Breeding is for SISSIES. Girls have COOTIES, my bros on the forums UNDERSTAND ME. This is not a cult of anti-natal creeps, this is WHO I AM. They call me ALPHA. They never lie, they’re all millionaires banging babes daily they just also post on forums constantly. They’re bitter because they’re TOO successful with women. I can’t find a good wife and it’s NOT my fault. It’s society. I have transcended EGO, obviously.

Calhoun’s mutant rats didn’t breed and neither shall we. We’re independent thinkers with no kids who believe in Patriarchy like a rich guy wants a wealth tax.

Then there’s the ‘satire’ argument of endorsing national rape of white women… as if Rotherham rapes in private weren’t bad enough, you now have to ban freedom of association for the literal majority of white people according to totally not rapey non-white males.

They all have ‘player burnout’ being alpha widows to that one hot chick they scored from pity or drugging and refuse to admit their pair bonding is ruined by porn addiction and promiscuity frying their dopamine circuits because it applies to sluts but only women can be, right? #cope Not like dopamine circuits all work the same in the whole species, is it?

The funniest is the naturalistic fallacy that sterile sex equates to something evolutionary, nope.

It’s overt a con as ‘retire early by paying me’ by that guy who married a fug Asian despite the higher divorce rate (I got suspended for pointing this out, literal divorce rates only). The MSM endorsement is coincidence.

Corruption was illegal in ancient white society for a reason. Psychopaths are glib and try to normalise their parasitism (Marxism) and clinical, pathological degeneracy. Not to mention, all the coercion and rape law, which they deny even though I checked CPS guidelines and the rape law in America before forming that legally accurate opinion. Incitement to rape more white women is the last thing we need.. it enables new citizens to crime. They’re not going to go after black women statistically, are they?

Psychopaths have average IQs. Sexual criminals have the lower end of that scale. There is no freedom to incite violence against a free Western citizen and rape is right up there. Concepts like ‘building kino’ (assault, grabbing women randomly like we don’t already have laws….) and LMR – when she says no but you ‘bang’ (note violent term re white women only) anyway (das called rape, son).

When America was great, the seduction law on legal books still was enforced by Patriarchy.

Pretending this material is aimed at white men is a psyop.

They always show young solid 5 white men prior to middle age (mid-30s) for their point, what happens to the old men put out to pasture, age 40, 50, 60, 70, 80?

This is not about ‘dating’. It’s incitement of white rape.

Alcohol is the most common date rape drug, statistically. See, they’re showing they can get away with it. That’s incitement. Do we take them seriously (true stories) or not (satire, joking, metaphor)? Also, unconscious and weak people cannot consent. That’s how men and minors get raped too.

BMI in women, hormones and a little on race and Asian pregnancy

follow-up to https://disenchantedscholar.wordpress.com/2020/09/14/explains-the-asian-fitness-influencer-rise-asian-body-fat/

TLDR: Asians have higher BMI and body fat than White women. It’s a racial difference.

Logically, wouldn’t they have messed-up hormones and diabetes, then? Let’s see!

5,000 words -ish.

High BMI, high Testosterone
https://pubmed.ncbi.nlm.nih.gov/27506736/

“cardiometabolic clinical correlates related to total testosterone (TT), free testosterone (fT), androstenedione (ASD), dehydroepiandrosterone-sulfate (DHEAS), estrone (E1), estradiol (E2), and sex hormone-binding globulin (SHBG).

Results: Waist circumference and BMI (β-coefficient: -0.03; 95% CI: -0.04; 0.03) were inversely related to SHBG, and BMI was positively related to TT (β-coefficient: 0.005; 95% CI: 0.001; 0.009), fT, E1, and E2. Smoking was positively related to TT (β-coefficient: 0.04; 95% CI: 0.01; 0.06), ASD, and fT. Systolic blood pressure (TT: β-coefficient: 0.002; 95% CI: 0.001; 0.003), hypertension (TT: β-coefficient: 0.05; 95% CI: 0.003; 0.11), low-density lipoprotein (LDL) cholesterol (TT: β-coefficient: 0.02; 95% CI: 0.01; 0.05), and total cholesterol (TT: β-coefficient: -0.03; 95% CI: 0.01; 0.05) were positively related to TT and ASD. Finally, type 2 diabetes mellitus (T2DM), and metabolic syndrome (MetS) were positively related to fT, but inversely related to SHBG.

Conclusions: Our population-based study, with sex hormone concentrations measured by liquid chromatography tandem mass spectrometry, revealed associations between clinical correlates including waist circumference, smoking, cohabitation, systolic blood pressure, cholesterol, and MetS with sex hormones. Thus, sex hormones and SHBG may play a role in the cardiovascular risk profile of women.”

I’ve posted about WHR before.

https://www.frontiersin.org/articles/10.3389/fpsyt.2019.00479/full

Both obesity and anxiety symptomatology were separately associated with the same sex hormone alteration in premenopausal women: higher total testosterone level (0.97 ± 0.50 in obese vs. 0.86 ± 0.49 nmol/L in normal-weight women, p = 0.026 and 1.04 ± 0.59 in women with vs. 0.88 ± 0.49 nmol/L in women without anxiety symptomatology, p = 0.023). However, women with anxiety symptomatology had non-significantly higher estradiol levels than women without anxiety symptomatology (548.0 ± 507.6 vs. 426.2 ± 474.0 pmol/L), whereas obesity was associated with lower estradiol levels compared with those in normal-weight group (332.7 ± 386.5 vs. 470.8 ± 616.0 pmol/L). Women with anxiety symptomatology had also significantly higher testosterone and estradiol composition (p = 0.006). No associations of sex hormone levels and BMI with anxiety symptomatology in postmenopausal women were found.

Conclusions: Although both obesity and anxiety symptomatology were separately associated with higher testosterone level, there was an opposite impact of anxiety and obesity on estradiol levels in premenopausal women. We did not find an evidence that the sex hormone alterations related to obesity are playing a significant role in anxiety symptomatology in premenopausal women. This could be the explanation why we did not find an association between obesity and anxiety. In postmenopausal women, other mechanisms seem to work than in the premenopausal group.

https://pubmed.ncbi.nlm.nih.gov/11915780/

Regional fat distribution (RFD) has been associated with metabolic derangements in populations with obesity. For example, upper body fat patterning is associated with higher levels of free testosterone (FT) and lower levels of sex-hormone binding globulin (SHBG). We sought to determine the extent to which this relationship was true in a healthy (i.e., non-obese) female population and whether RFD influenced androgen responses to resistance exercise. This study examined the effects of RFD on total testosterone (TT), FT, and SHBG responses to an acute resistance exercise test (ARET) among 47 women (22+/-3 years; 165+/-6 cm; 62+/-8 kg; 25+/-5%BF; 23+/-3 BMI). RFD was characterized by 3 separate indices: waist-to-hip ratio (WHR), ratio of upper arm fat to mid-thigh fat assessed with magnetic resonance imaging (MRI ratio), and ratio of subscapular to triceps ratio (SB/TRi ratio). Skinfolds were measured for the triceps, chest, subscapular, mid-axillary, suprailaic, abdomen, and thigh regions. The ARET consisted of 6 sets of 10 RM squats separated by 2-min rest periods. Blood was obtained pre- and post- ARET. TT, FT, and SHBG concentrations were determined by radioimmunoassay. Subjects were divided into tertiles from the indices of RFD, and statistical analyses were performed by an ANOVA with repeated measures (RFD and exercise as main effects). Significant (p < or = .05) increases following the AHRET were observed for TT (approximately 25%), FT (approximately 25%), and SHBG (4%). With multiple regression analysis, anthropometric measures significantly predicted pre- concentrations of FT, post-concentrations of TT, and pre-concentrations of SHBG. The SB/TRi and MRI ratios but not the WHR, were discriminant for hormonal concentrations among the tertiles. In young, healthy women, resistance exercise can induce transient increases in testosterone, and anthropometric markers of adiposity correlate with testosterone concentrations.

So exercise will boost a woman’s natural T. If they already have high T….

If their BMI is higher for their size, they already have high T comparatively. If they already have it racially… probably not good.

https://academic.oup.com/jnci/article/95/16/1218/2520391

Results: Breast cancer risk increased with increasing BMI (Ptrend = .002),

Not healthy.

Rare study looking at race directly. White v Black.

https://womensmidlifehealthjournal.biomedcentral.com/articles/10.1186/s40695-017-0028-4

Results

Compared to the decline in E2 concentrations, androgen concentrations declined minimally over the MT. T (β 9.180, p < 0.0001) and E1 (β 11.365, p < 0.0001) were higher in Whites than in AAs, while elevations in DHEAS (β 28.80, p = 0.061) and A4 (β 0.2556, p = 0.052) were borderline. Log-transformed E2 was similar between Whites and AAs (β 0.0764, p = 0.272). Body mass index (BMI) was not significantly associated with concentrations of androgens or E1 over time.

so black and white is off the hook

Conclusion

This report suggests that the declines in E2 during the 4 years before and after the FMP are accompanied by minimal changes in DHEAS, A4, T, and E1. There are modest differences between Whites and AAs and minimal differences by BMI.

https://bmccancer.biomedcentral.com/articles/10.1186/s12885-018-4558-4

Cancer and Testosterone link

Results

During a median follow up of 6.3 years, 45 patients relapsed. Testosterone levels significantly increased across BMI categories (p = 0.001). Both circulating testosterone and BMI were positively associated with disease free survival (p = 0.005 and p = 0.021, respectively). A significant interaction was found between testosterone and BMI (p = 0.006). For normal-weight women, testosterone concentration around median (0.403 ng/mL) or third quartile (0.532 ng/mL) showed a high significant HR of relapse (5.52; 95% CI:1.65–18.49 and 4.55; 95% CI:1.09–18.98, respectively). Overweight patients showed increased HR at increasing testosterone levels, reaching a significant high HR (4.68; 95% CI:1.39–15.70) for testosterone values of 0.782 ng/mL (95th percentile). For obese patients HR decreased (not significantly) at increased testosterone concentrations, explaining the interaction between testosterone levels and BMI categories.

Conclusions

In ER-positive postmenopausal breast cancer patients, high testosterone levels are associated with worse prognosis in normal-weight and overweight women, whereas in obese seems to be associated with a better outcome. Although the results require further validation, they suggest that assessment of circulating testosterone and BMI could help to identify postmenopausal ER-positive patients at higher risk of relapse and potentially open new therapeutic strategies.

High T isn’t good, even in normal weight women. Water is wet.

https://www.healio.com/news/endocrinology/20200515/high-endogenous-testosterone-levels-contribute-to-type-2-diabetes-risk-among-young-healthy-women

“The findings of this study suggest high plasma levels of testosterone could play a role in the pathogenesis of type 2 diabetes among women,” Jon Jarløv Rasmussen, MD, PhD, a specialist registrar and postdoctoral researcher in the department of endocrinology at Rigshospitalet in Copenhagen, Denmark, told Healio. “The incidence of type 2 diabetes was rather low in the study, but the results implicate that screening for type 2 diabetes among women with higher plasma levels of testosterone may be beneficial, even among women who are young and without established comorbidities, such as polycystic ovary syndrome.”

In a retrospective study, Rasmussen and colleagues analyzed data from 8,876 healthy women (mean age, 38.5 years) who provided blood samples to measure plasma testosterone, dehydroepiandrosterone-sulfate (DHEAS), dihydrotestosterone (DHT) and sex hormone-binding globulin (SHBG) between January 2007 and December 2015. Researchers analyzed androgens using tandem liquid-chromatography mass spectrometry. Researchers used Poisson regression models to calculate incidence rate ratios for developing type 2 diabetes during a median follow-up of 8.1 years, stratified by androgen quartiles.

‘Normal weight’ women can get Type 2. Since Asians have higher T from higher BMI (against the white norm), they’ll be more likely to get it. This also explains the gestational diabetes common in Asian women, especially if the baby is mixed.

https://www.cdc.gov/diabetes/library/spotlights/diabetes-asian-americans.html

Nationwide, as many as 1 in 4 people who have diabetes don’t know they have it. But for Asian Americans, that number is much higher—1 in 2, the highest of all ethnic/racial groups. Why aren’t more getting diagnosed?

Weebs do not mention this. If your apparent rationale for avoiding fat white women is avoiding the Diabeetus genes, Asian is then categorically the worst racial group to mix with.

1 in 2, flip a coin, rice cooker.

I bet it’s higher in the women due to sweet tooth, so likely worse.

But people of Asian descent have less muscle and more fat than other groups and often develop diabetes at a younger age and lower weight. That extra body fat tends to be in the belly (visceral fat). This isn’t the “inch you can pinch,” the fat stored just under the skin. Visceral fat is out of sight, wrapped around organs deep in the body. You can’t tell how much visceral fat someone has by looking at them.

I didn’t call them skinny-fat to be mean, they really are!

Visceral fat is also sometimes known as “active” fat because it drives certain processes in the body that can increase the risk for heart disease, stroke, and other serious health conditions. Everybody has some visceral fat, but having too much is a major risk factor for developing type 2 diabetes.

….But BMI doesn’t catch Asian Americans in the normal weight range (18.5 to 24.9) who may very well have too much visceral fat and be at risk for type 2 diabetes. Researchers are now suggesting that people of Asian heritage get tested if their BMI is 23 or greater. Type 2 diabetes can be prevented or delayed, but only if people know they’re at risk and can take action!

They need a totally different (lower) testing standard, but they’re just like us, guys! Nay, SUPERIOR!

The same volume food in a smaller body, this isn’t hard to figure out. They’re not white women, eating like us makes them FAT.

re preggers diabetus-

https://www.sutterhealth.org/health/south-asian/womens/gestational-diabetes

Pregnant South Asian women carry a higher risk for developing gestational diabetes, a condition that’s dangerous for both mother and child. Between 2 and 10 percent of all pregnancies each year are complicated by gestational diabetes

2-10% in which demographic? Sounds like all? I bet it’s higher in certain ones, isn’t it?

Under risk factors is basically – be non-white

  • Being of Hispanic, Native American, African-American, Asian-American or Pacific Islander descent.

Women who have had gestational diabetes have a 20 to 50 percent chance of developing diabetes in the 5 to 10 years following pregnancy.

Mother Nature is a bitch.

Specific study on Asian women-

https://care.diabetesjournals.org/content/24/5/955

so healthy, much fitness

Our data indicate that although the historical or clinical risk factors for GDM are valid in Asians, using risk factors alone to select such patients for testing for GDM is inadequate. Many Asian women who develop GDM have no risk factors at all.

When Natural Selection hates you so much… maybe give it up?

r-types have higher numbers of issues like this, that would be fatal under natural law

They don’t ‘choose’ to stop at 1-2 kids, it isn’t ‘culture’, it’s fear (see below).

To avoid overlooking significant numbers of women with GDM, one may lower the specificity of the criteria, but this requires that the majority of patients be tested.

wow, that bad

Logistically, it is much simpler to conduct universal screening for all Asian women in Western countries, rather than to apply selective testing in order to spare a small percentage of women from being tested. Therefore, our findings strongly support recommendations for universal screening for GDM in pregnant women of Asian origin in Western countries. However, in places where the incidence of GDM is low, such as in some developing countries, the selection of patients for testing by the risk factors may be reasonable.

just like us, huh?

https://www.medscape.com/viewarticle/923661

has a bloody paywall, nevermind, still linking

Introduction: Asian women have a higher prevalence of gestational diabetes mellitus than women of other races/ethnicities. We aimed to compare the prevalence of gestational diabetes among Asian American women to other racial/ethnic groups and explore whether the higher occurrence of the disorder among Asian women can be explained by acculturation.

Clearly I am making this all up to feel better, right guys?

Why hide this one behind a paywall, hmm?

https://www.cdc.gov/pcd/issues/2019/19_0212.htm

It is also here.

Results

Among the 5,562 women studied, the weighted prevalence of gestational diabetes was 15.5% among Asian American women, followed by 9.0% among non-Hispanic black women, 10.7% among Hispanic women, and 7.9% among non-Hispanic white women.

15.5% v. 7.9%

Diabetes at DOUBLE the rate of whites!

DOUBLE!

but they’re just like us

2.44x the risk

and that’s controlled, independently

Compared with non-Hispanic white women, Asian women had 2.44 (95% confidence interval [CI], 1.81–3.29; P < .001) times the odds of having gestational diabetes, independent of maternal age, education, marital status, income, prenatal care adequacy, prepregnancy BMI, and physical activity. Acculturation was negatively associated with having gestational diabetes (odds ratio [OR] = 0.93; 95% CI, 0.86–0.99) and explained 15.9% (95% CI, 11.38%–25.08%; P < .001) of the association between Asian race and the condition.

About 85% genetic. Great odds.

Conclusion

We found that Asian race was an independent risk factor for gestational diabetes, and higher acculturation may play a protective role against it in Asian American women.

In Summary

What is already known about this topic?

Asian women have a higher prevalence of gestational diabetes mellitus than women of other races. However, little data exist on why prevalence is highest among Asian women.

I sense genetics.

If they’re having unnatural babies (too large for their race, mixed) supported by modern medicine, they’d be more likely to die anyway, right? Medicine can only do so much. Weaker genes die a la Darwin.

The biggest r-select factor would be risk of death while breeding, that would be the surest thing. The genes trying to extinct themselves.

Does this data exist? Also for the neonates?

YOU BET IT DOES.

Let’s see the weebs explain away these studies. They’ll probably just ignore me… again.

Go ahead. Ignore your baby and waifu’s graves?

https://www.ajmc.com/view/racial-disparities-persist-in-maternal-morbidity-mortality-and-infant-health

Pregnancy related mortality can be defined as death of the mother during pregnancy, delivery, or within one year postpartum. While 700 pregnancy-related deaths occur each year, 2/3 of these deaths are considered to be preventable.

Modern medicine, dysgenic again.

Overall pregnancy related mortality in the United States occurs at an average rate of 17.2 deaths per 100,000 live births. However, that number jumps to 43.5/100,000 for non-Hispanic Black women and decreases to 12.7/100,000 for non-Hispanic white women and 11/100,000 for Hispanic women.

No data listed for Asian, odd?

For mothers of all backgrounds, leading causes of death include cardiovascular conditions, hemorrhage, and infection. However, for non-Hispanic Black women, leading causes of death include cardiovascular conditions in addition to cardiomyopathy, pre-eclampsia, and eclampsia (hypertensive disorders).

Non-Hispanic Black women are also significantly more likely to have a severe maternal morbidity (SMM) event at the time of delivery. For every maternal death there are 70 cases of SMM events that are considered “near misses.” These events can have long-term or short-term consequences to a woman’s health. Over the past 20 years, cases of SMM have increased by over 200%, while cases disproportionately affect Black women. One study found Black women experienced SMM at a rate 2.1 times greater than that of white women.

To better understand and address these disparities, researchers suggest providers increase screening for social determinants of health. Levels of stress, trauma, food insecurity, neighborhood violence, and access to prenatal care are all factors that may contribute to the disparities and warrant further investigation.

Although most maternal deaths result from cardiovascular and hypertensive disorders, researchers found Asian/Pacific Island women exhibit the highest prevalence of gestational diabetes, which can increase pregnancy complications, at 14.8%.

One study presented in the session focused on behavioral interventions and protective factors among women with gestational diabetes. A Kaiser Permanente analysis of women in northern California found Black women have a lower prevalence of gestational diabetes when compared with Asian Indian, Filipina, Southeast Asian and Chinese women. White women had the lowest rates of the disease overall.

Screening for postpartum diabetes is recommended to all women within 4 to 12 weeks postpartum. However, rates of screening vary among women with different racial and ethnic backgrounds, suggesting tailored strategies to reduce risk and improve healthcare behaviors may be effective.

Racial medicine, openly.

An additional study explored how racial and ethnic disparities impact severe neonatal morbidities, specifically among very preterm children (born <32 weeks of gestation). Preterm birth has been associated with several health conditions developing later in life, including diabetes.

Presenter Teresa Janevic, PhD, defined race as “linked to phenotype and /or ancestry that indexes one’s location on the US social hierarchy of socially constructed groupings (i.e., races) that has been based primarily on skin color.”

genes aren’t social

Africans in Africa also have the same ‘risk’ as one in America. No magic dirt.

In contrast, Janevic defined ethnicity as “tied to race and used both to distinguish diverse populations and to establish personal or group identity, usually based on shared culture or beliefs.”

Culture? Belief? Believe your way out of diabetes. I’ll wait.

In a population-based retrospective cohort analysis using hospital discharge data linked with vital statistics at birth and death records, researchers determined Black infants were at the highest risk of dying within less than 28 days after discharge, or suffering neonatal morbidities in the time between birth and discharge. Black infants were followed by Hispanic infants, while white and Asian infants had similar low risks.

We’ll see about that.

Of the 39 New York City hospitals included in the study, researchers found a 6-fold difference in risk of combined mortality and morbidity outcomes. “Black infants were at twice the risk of being at a hospital that has risk-adjusted high rates of combined mortality and morbidity,” Janevic noted, while Hispanic infants had a 1.5 increased risk to receive care from one of these hospitals. “Hospital quality where women of color deliver likely contributes to these disparities,” she concluded.

Like schools, it depends on the IQ of the people working there.

Another investigation detailed how environmental factors and population level exposures impact disparities in preterm birth and infant mortality. “Non-Hispanic Black infants compared with non-Hispanic white infants have twice the risk of death in the first year,” explained presenter Heather Burris, MD. “This is particularly striking because Black infants just make up 15% of all births in the United States but are counting for 29% of all deaths.”

no comment

Among causes of infant death, preterm birth and low birth weight related death, along with pregnancy complications, account for the highest racial and ethnic disparities between non-Hispanic Black and white infants. Black infants are also significantly more likely to be born preterm than white infants.

an r-factor unless twins

Researchers note genetics and education level have very little impact in accounting for disparities in preterm birth. Although women with higher education tend to have lower preterm birth rates, Black women who graduated from college have a higher risk of preterm birth than white women who dropped out of high school.

so, racial

I’m so glad white people already survived multiple genetic purges in our history.

Through analyzing delivery data and creating models based on air pollution severity in Philadelphia, Pennsylvania, investigators discovered air pollution is associated with spontaneous preterm birth. Data also show Black Americans experience consistently higher exposure to air pollutants, measured in fine particulate matter (PM)2.5.

An additional analysis between preterm birth and nationwide neighborhood deprivation index (encompassing income below the poverty level, vacant homes, education levels, among other factors) found that Black women experience neighborhood deprivation exposure at almost 2 standard deviations (SDs) higher than white women in Philadelphia.

Overall, Black women are 4 times more likely to live in a neighborhood with high violent crime and high air pollution than white women. “When we look at preterm birthweights, we can see that it is women living in these high-high neighborhoods that have the highest risk of preterm birth,” Burris said. However, these associations were consistent regardless of race.

so non-sig

They gestate for less time than whites, this is known. Africans in Africa do it.

Now we’ve established some things. An r-study in Asian women.

https://pubmed.ncbi.nlm.nih.gov/28099290/

Increased Perinatal Morbidity and Mortality Among Asian American and Pacific Islander Women in the United States

Background: Asian American/Pacific Islanders (AAPIs) are the fastest-growing racial group in the United States.

America is now owned by Asia, demographically.

Despite a higher socioeconomic status, AAPI women experience higher rates of maternal morbidity and mortality.

can’t pay your way out of r-genes

if controlled for SES, aka $, their data would be even worse

Methods: Using the National Inpatient Sample, we performed a retrospective cohort analysis of women who were hospitalized for delivery from 2002 to 2013. The primary outcome variable was inpatient mortality rate, and the presence of severe maternal morbidities was estimated using the Bateman Comorbidity Index, a validated tool for predicting obstetric morbidity.

Results: AAPI women presenting for delivery between 2003 and 2012 were older, more likely to reside in a zip code in the top quartile of annual income, be privately insured than Caucasian women,

so oppressed

where’s Asian privilege?

and less likely to have a higher Bateman Comorbidity Index. However, AAPI women had a higher likelihood of postpartum hemorrhage (3.4% vs 2.7%, P < .001), uterine atony, severe perineal lacerations, and severe maternal morbidities. Procedures such as transfusion, hysterectomy,

So they could have one kid and die, have one kid and have that die, OR have one kid and then their organs all removed – so no more kids?

Yes clearly our biological superiors, right weebs? Totally not rationalising a fetish, are we?

I wonder why one child was law? They don’t have a culture of many kids because they’re too r-select to survive without modern medicine. Wake up. They pretend 1-2 is a choice and that’s why they mock and envy large white families (3+ standard) like the Amish. They envy us that ability. They would die.

and mechanical ventilation were also more common in AAPI women.

Calling it – Mother Nature is anti-Asian.

Furthermore, AAPI women had a higher mortality rate that persisted despite adjustment for an apparently higher income and comorbidities (odds ratio 1.72, 95% confidence interval: 1.14-2.59, P = .01).

Conclusions: Despite having a higher socioeconomic status, AAPI women had higher rates of maternal mortality during hospitalization for delivery. This increase persisted even after adjustment for factors known to affect peripartum outcomes. Further investigation is needed to better clarify the causes of racial differences in maternal morbidity and mortality.

D.N.A.

Science says –

If you want to survive childbirth – be white.

Almost like we evolved to during the Ice Age.

Almost….

https://pubmed.ncbi.nlm.nih.gov/29752934/

Results: A total of 360,370 women with postpartum hemorrhage from 2012 to 2014 were included in this analysis. Risk for severe morbidity was significantly higher among non-Hispanic black women (26.6%) than non-Hispanic white, Hispanic, or Asian or Pacific Islander women (20.7%, 22.5%, and 21.4%, respectively, P < .01).

The white is 20%, Asian is 21%.

And these are the fattest white people, like, ever.

White and Asian bolded-

For non-Hispanic black compared with non-Hispanic white, Hispanic, and Asian or Pacific Islander women risk was higher for disseminated intravascular coagulation (8.4% vs 7.1%, 6.8%, and 6.8%, respectively, P < .01) and transfusion (19.4% vs 13.9%, 16.1%, and 15.8%, respectively, P < .01). Black women were also more likely than non-Hispanic white women to undergo hysterectomy (2.4% vs 1.9%, P < .01), although Asian or Pacific Islander women were at highest risk (2.9%). Adjusting for comorbidity, black women remained at higher risk for severe morbidity (P < .01). Risk for death for non-Hispanic black women was significantly higher than for nonblack women (121.8 per 100,000 deliveries, 95% confidence interval, 94.7-156.8 vs 24.1 per 100,000 deliveries, 95% confidence interval, 19.2-30.2, respectively, P < .01).

The weebs either did 1. no research (typical gammas) or 2. they’re delusional.

Almost double the risk of hysterectomy, roughly. An additional 52% risk over white women, minimum, in just this study.

What’s the point of being married to them, at that point? Their baby machine is broken.

https://www.minorityhealth.hhs.gov/omh/browse.aspx?lvl=4&lvlid=53

Infant Mortality and Asian Americans

Yes, we’re going there.

  • Asian American infants are 40 percent more likely to die from maternal complications as compared to non-Hispanic white mothers.

They have boy hips, duh.

For such a small segment of the population, their numbers shouldn’t be so high.

It doesn’t discuss mixed kids, I wonder why?

https://www.cdc.gov/media/releases/2019/p0905-racial-ethnic-disparities-pregnancy-deaths.html

  • Non-Hispanic black (black) and non-Hispanic American Indian/Alaska Native (AI/AN) women experienced higher PRMRs (40.8 and 29.7, respectively) than all other racial/ethnic populations (white PRMR was 12.7, Asian/ Pacific Islander PRMR was 13.5 and Hispanic PRMR was 11.5).  This was 3.2 and 2.3 times higher than the PRMR for white women – and the gap widened among older age groups.

https://www.nimhd.nih.gov/news-events/features/community-health/causes-asian-american-mortality.html

Racial medicine.

Notably, we found that, when aggregated, the top cause of death among Asian Americans is cancer. However, when disaggregated, there is wide variation in the leading cause of death. For instance, for Asian Indians, nearly twice as many men die of heart disease (31 percent), compared to cancer (18 percent). In contrast, for Koreans, the opposite is true — the death rate for cancer (34 percent) is much higher than the death rate for heart disease (19 percent).

Remember the breast cancer and Asian BMI/testosterone stuff?

https://medicalxpress.com/news/2019-03-excess-hormones-condition-women.html

Research led by the University of Birmingham has found that increased levels of hormones including testosterone could cause a brain condition that can lead to blindness in women.

We are all jealous of your waifu, yes.

Idiopathic Intracranial Hypertension—also known as IIH—is caused by  in the brain with consequences from blindness to incapacitating daily long-term headaches. IIH was originally identified over 100 years ago yet the cause of the condition has remained unknown although there has been much speculation about why more than 95 per cent of total incidence is in  with obesity.

And Asians, they’re 1/2 obese in America!

Lucky you.

 They then compared the results with the levels observed in women with obesity of the same age and body mass index (BMI), as well as a cohort of women with  (PCOS).

PCOS is far more common in Asians. Look it up.

Most notable were the high levels of the androgen ‘testosterone’ found in the blood in IIH women. Crucially, levels of androgens were uniquely increased in the brain fluid (CSF) of women with IIH. When the researchers, analysed human choroidal plexus tissue, which is the site in the brain where CSF is produced, they confirmed that androgens could increase the rate of CSF secretion, a potential driver for increased brain pressure.

Brain damage. How sexy.

re PCOS

https://obgyn.onlinelibrary.wiley.com/doi/full/10.1111/jog.14132

On the other hand, South Asian women with PCOS, the region comprising the Indian subcontinent and surrounding countries, are known to have a higher degree of hirsutism…

sexy

Asian women with PCOS were more likely to have diabetes compared with Caucasian patients, suggesting they also have metabolic complications. 

Genetic components play important roles in the pathogenesis of PCOS,

Do their dodgy hormones make South Asians partially infertile?

https://pubmed.ncbi.nlm.nih.gov/17367914/

Yes. Yes they do.

Results: We found that the South Asian women presented at a younger age for the management of sub-fertility. An extended stimulation phase and Caucasian ethnicity showed an inverse correlation with the number of oocytes retrieved in the PCOS subgroup. Caucasian ethnicity was associated with a higher fertilization rate however increase in body mass index (BMI) and the laboratory technique of IVF appeared to have a negative impact on fertilization rates in the PCOS subgroup. Commencing down regulation on day 1 of the cycles was negatively associated with fertilization rates in the tubal group. In terms of clinical pregnancy rates, the Caucasian PCOS had a 2.5 times (95% CI: 1.25-5) higher chance of an ongoing clinical pregnancy as compared with their Asian counterpart. Also, a unit increase in the basal FSH concentration reduced the odds of pregnancy by 18.6% (95% CI: 1.8-32.6%) in the PCOS group.

Conclusions: The Asian PCOS have a greater sensitivity to gonadotropin stimulation with lower fertilization and ongoing clinical pregnancy rates as compared with their Caucasian counterparts.

White women win again.

https://europepmc.org/article/PMC/3893977

testosterone levels were higher in PCOS cases than in controls (P = 0.008 and 0.003, respectively).

But IVF, right? WRONG

https://www.sciencedaily.com/releases/2016/08/160818212907.htm

The ethnicity of women undergoing fertility treatments like IVF can affect the rate of successful live births, according to new research. After adjusting for certain factors including age of patient at time of treatment, cause of female or male infertility, and type of treatment, the study found that White Irish, South Asian Indian, South Asian Bangladeshi, South Asian Pakistani, Black African, and Other Asian women had a significantly lower odds of a live birth than White British women.

White women, still winning. Thank God for the Ice Age.

https://www.thefreelibrary.com/A+study+of+association+of+sex+hormones+with+insulin+resistance+and…-a0509015581

Overall, studies have shown higher testosterone levels in women and lower levels in men are related to incident diabetes. The major risk factors contributing to diabetes are biochemical, environmental, sedentary lifestyle, socioeconomic status and genetic factors. All of them together or independently are responsible for the development of the DM. [3] Besides, certain studies show Impaired Glucose Tolerance (IGT) is more common in females than males independent of age. [4]

https://bmcpregnancychildbirth.biomedcentral.com/articles/10.1186/s12884-018-2131-4

We found a high prevalence of GDM among the Asian population. Asian women with common risk factors especially among those with history of previous GDM, congenital anomalies or macrosomia should receive additional attention from physician as high-risk cases for GDM in pregnancy.

https://pubmed.ncbi.nlm.nih.gov/14763914/

 In the post-menopausal group, estrogens, testosterone and androstenedione increased with increasing BMI. 

https://www.sciencedirect.com/science/article/abs/pii/S1090513814000026

Let’s look at objective hotness!

Body mass index (BMI) was a very strong negative predictor of body attractiveness ratings, similar to previous findings. Zero-order associations between women’s mean hormone concentrations and mean attractiveness ratings were not significant; however, after controlling for BMI, attractiveness ratings were independently and positively associated with both estradiol and testosterone concentrations. Discussion focuses on the implications of these findings for whether attractiveness assessment mechanisms are specialized for the detection of cues of differential fecundity in young women’s bodies.

High T = ugly!

Did I mention water is wet? Can they seriously accuse of cherry picking? I’m not even looking hard.

Prior research has provided evidence that large breast size and low waist-to-hip ratio (WHR) are positively associated with women’s estrogen and progesterone concentrations,

Previously covered WHR, use search bar. Asians lose. Even black women do better.

Click to access ethnic-testosterone1.pdf

Asians have way more T as a race than Europeans, get over it. Historically, we considered them savages, less civilized, for that reason. How is this surprising? Do you think we colonised India for fun? It’s obvious in the broad manjaws, duh. Marquardt covered this. Anyone can do a replication study, but I suggest you include the women too, so it isn’t just a sexual effect but race.

From a blog “East Asians were found to have the highest average total plasma testosterone (5,673 ρg/mL) followed by Africans (5,442 ρg/mL) and then Europeans (4,992 ρg/mL). Given that the sample size for Africans is smaller (N < 10,000), their relative position may change with more data. Nonetheless, the claim that East Asians have the least testosterone is not supported by scientific data. “

Yeah, fake redpills who think T = manly, good thing. It’s just a hormone.

“There is no way of accurately determining free testosterone. Even if there was, this would also be irrelevant since bio-availability is prime. Since race realists use total serum testosterone, why is this an issue?”

true, it’s just applying the same standard

Culturally, gang rape is more normal in Asia than Africa. This is why. You don’t get African Taharrush, really. Asia has Eve Teasing and the like. Trust me, you don’t want this.

“Mass sexual assault is the collective sexual assault of women, and sometimes children, in public by groups of unrelated men. Typically acting under the protective cover of large gatherings, victims have reported being groped, stripped, beaten, bitten, penetrated and raped.”

As for the contention that there are no studies indicating a 10% difference between East Asians and Europeans, I did find one age controlled study where the Chinese sample had 8.8% more total T, 11.4% more bio-available T and 12% more free T than the European sample. The Japanese sample had 10.5%, 5.1% and 6.7% more than Europeans respectively [Wu et al. 1995]. Wonder if race realists discuss this study, or perhaps they are too busy in celebratory dance around the Korean/Swede campfire?

They’re not really redpill. I believe data even if I dislike it. Asians have high T as a race. Get over it.

High T can also dovetail with lower national IQs e.g. India, so…. why want this? Low IQ nations have more crime.

Additionally, this recent study shows HK Chinese having some 3% more bio-available T than US Europeans.

Lol, he’s right. But T isn’t a good thing. It’s just a hormone, in men or women.

Being a race realists seems to be a length engagement with delusion, fantasy and ‘scientific’ homo-erotica.

Not here, son. I believe the T-data. Penis size generally correlates to racial height (in white men), not really T. Forum bros are wrong again. Penis stuff is sexual selection, aka chosen by women.

https://www.pnas.org/content/110/17/6925

There was a similar increase in the positive effect of penis size on attractiveness with a more masculine body shape (i.e., greater shoulder-to-hip ratio). Surprisingly, larger penis size and greater height had almost equivalent positive effects on male attractiveness. Our results support the hypothesis that female mate choice could have driven the evolution of larger penises in humans. More broadly, our results show that precopulatory sexual selection can play a role in the evolution of genital traits.

It’s even subracial (national male height).

https://www.penissizes.org/average-penis-size-ethnicity-race-and-country

I appreciate the skin tone joke in this graphic.

But if you go by nation, and percentage of height....

https://www.worlddata.info/average-penissize.php

So according to this, hate the French!

Surprised Croatians rank so low.

https://journals.lww.com/humanandrology/Fulltext/2011/08000/Relationship_between_penile_size_and_body.4.aspx

It was concluded that all penile measurements are interrelated to each other; the height and weight also the other body measurements that are related to the penile measurements in less than 50%. It seems that the penile measurements are polygenic traits and are under multifactorial influences.

Not T.

https://www.fertstert.org/article/S0015-0282(00)01723-4/pdf

There are racial differences in associations of hormone levels with age and BMI in late
reproductive age women. Further study is needed to replicate these findings and to determine the relationships of these hormonal associations with menopausal symptoms

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2888623/

Obesity is an important factor in hormone dynamics independent of age, race and smoking in mid-life women, although the mechanisms remain unclear.

From “A Study of the Correlation of Some Sex Hormone with Obesity in Women with Secondary Infertility” (google it)

Infertility is the inability to conceive a child for more than one year. The present study indicates
that the obesity associated with infertility. The aim of the study to determine follicle stimulating
hormone, luteinizing hormone, testosterone hormone and prolactin levels. and cholesterol and
triglyceride concentration in 2nd inferetid women. This study was carried out at kamal al-samaarai
hospital the data were collected from 95 secondary infertilial women were age between 16-45 years
old and grouped them in to obese (n = 46) and non obese(n = 49). There was no significant
difference between the two groups (p <0.05). Body mass index in Infertile obese women is slightly
higher than non obese Infertile women which is statistically significant (P<0.001). However LH,
TSH, cholesterol and triglyceride concentration in obese infertile women is significantly higher than
non obese infertile women (p >0.05).The BMI was correlated positively with triglyceride in obese
group while BMI was positive correlation highly significant with cholesterol in non obese group.
Regression analysis revealed obese to be strongly associated with observed infertility. The elevated
prolactin values in secondary infertile women clearly shows that there is a mechanism operating at
the anterior pituitary level which shows an abnormal distribution of FSH and LH which may further
explain the abnormal delay ovum maturation. This study also indicates obese associated with
infertile more than non obese women.

So, there’s a lot going on.

K-times change r-minds

Hard times produce baby bust.

As do low record marriage rates….. the MGTOW don’t get to complain about the birth rate.

https://www.bustle.com/p/covid-19-made-women-change-their-minds-about-having-kids-27639772

Despite early reports of an anticipated coronavirus baby boom, demographers say that’s probably less than likely.

The Pill exists and it’s given to minors.

With so many Americans unemployed or furloughed, fewer people are likely to feel financially stable enough to start a family.

Student loans are anti-natal.

As a result of COVID-19, more than 40% of American women changed their plans to have children or how many children to have, according to the Guttmacher Institute’s new analysis of data from more than 2,000 women. More than one-third of cisgender women said they would delay getting pregnant or wanted fewer children than they’d wanted before; that number is even higher for Black, Hispanic, queer, and low-income women.

It’s all about incentives. Suddenly motherhood doesn’t look easy and glamorous.

A smart right-wing would push on this button.

Nearly one-fifth of respondents in the Guttmacher study said COVID-19 has made them want to have a child sooner, or to have more children.

Time out of corporations does that. Those are K-types. They know they can survive from home.

I might not be able to have one if I wait

Early 30s is considered young here so lol, teen moms has brainwashed Americans about the safety of r-strategy. Teen mothers have higher stillbirth and miscarriage rates than 20s women.

Delayed motherhood produces higher IQ children because higher IQ women delay until stability (usually marriage).

Should we really have a kid right now, when we don’t even know where we’ll be living in a year? With everything up in the air, it puts off our timeline until we feel more comfortable.

Responsibility, exactly. K-select couple.

The problem is delaying marriage, in that case. Opening to Idiocracy scene.

 I’ve been on birth control since I was 16, and now I’m worried I’ll never be able to have a baby.

Can a minor make medical decisions?

I have no sympathy with the Pill takers, but if it took years for your husband to come along, then there’s obviously a logistical problem.

I had always wanted kids. Then five years ago, my husband and I got a dog, and I was like, “Oh God, this is a lot of responsibility.” We like to travel; we want the flexibility to do what we want, when we want.

That’s pure r-type. “Liberal fertility” (as Jayman has noted) is a good thing.

Like I said, if you want a baby boom, ban pet ownership.

At the start of WW2 all pets were put down, THAT caused a baby boom.

Literally a week before the shutdown, my husband got his dream job he’s been waiting his whole life to get.

Can we ban that phrase? Dream job?

What about dream house? Dream spouse?

Dream job is Plato’s natural slave. Careerism in men is a patriarchy replacement.

They sell it like “eventually you’ll spend time with your kids, only have less of them and they won’t be kids anymore”.

Even before the pandemic, we were worried about climate change and the impacts of the current presidential administration on the world. It feels like if you have a pro-con list for whether or not to have a kid, the con list is just growing.

With the pandemic, it sucks to feel lucky to not have kids. I briefly thought I was pregnant last summer. If I had been, I would have been due in the spring. I kept thinking, “Thank God I’m not pregnant.” It feels irresponsible to ignore the things I thought the world could just figure out.

The baby cult needs to account for the political imbeciles.

I cannot wait to be a mother. People always ask me if my abortion work — I’m an abortion storyteller and on the board of an abortion fund — conflicts with me wanting kids. I’m like, “I do want kids, but on my own terms.” If anything, the pandemic made me realize how important families are to me. I have a mother and brother and sisters, but queer folks, we often have to create our own families.

These articles are a guilty pleasure. The hypocrisy, like “pro-family” libertarians who want abortion clinics AND parental responsibility. Well…. men can be parents? Take responsibility? Men support abortion more than women. It’s a Get out your Gender Role free card.

Spot the r-type.

You can tell the child outcomes before they’re even conceived. They’ll call the kid Moose and it’ll be a Marxist before it’s five.

Nothing about kids is on one’s own terms, the Right really need to contradict this selfish model of parenthood. Both parents must sacrifice for the child.

Despite all of that, it’s wonderful to know there are people working to make sure we have a society where someone like me — someone who is queer and Latinx and lives in Texas — can raise a family.

Exh A of spiteful mutant.

“I can’t have kids so you shouldn’t be able to!”

they reduce the fertility of the in-group (like MGTOW, a psyop aimed at evil white men to stop producing more oppressors). It’s never aimed in the Darwinian way, at the out-group. All hail Bill Gates.

Darwin Awards global finale

me watching the weebs trapped in Asia and the stock bros during circuit breakers

Nobody’s laughing at the preppers anymore and they’re fast reckoning that it’s an actual skillset taking years to develop instead of buying 500 toilet rolls like a hoarding normie.

Nobody’s calling me racist for righteously hating China and Communism, which is nice.

All in all, I’m pleased as punch. I hope it gets worse so we skip a repeat (pandemics repeat if the first case is mild). It’s the first thing to wake up the middle-class cucks that open borders can and shall hurt them. No holiday to Italy now, Karen!

Pandemics always hit the sluts hardest.

repost link; https://www.medrxiv.org/content/10.1101/2020.02.12.20022418v1.full.pdf

“From the paper: “The protein and mRNA expression of ACE2 in the testes is almost the highest in the body. Moreover, both cells inseminiferous ducts and Leydig cells showed high ACE2 expression level. These results indicate that testicular cells are the potential targets of 2019-nCoV.

The bioweapon’s coming for yar balls.

We believe in you, Corona-Chan! Sterilise the Commies! Sterilise them all!

https://www.ncbi.nlm.nih.gov/pubmed/20449780

Orchitis and male infertility

Infections and inflammations of the genital tract are considered the most frequent causes of reduced male fertility, but conclusive epidemiological data are not available. In view of the exposure of germ cells to pathogenic components as well as the cells and mediators involved in the inflammatory processes, irreversible damage to spermatogenesis and corresponding decline of ejaculate quality are to be expected, particularly in cases of chronic orchitis. While the consequences of orchitis and epididymo-orchitis that exhibit clinical symptoms due to systemic or local infections are well known, including testicular atrophy and complete loss of fertility, those cases of inflammatory reactions of the testicles that manifest an asymptomatic or subclinical course, or are not even due to an infection, have received little attention until now. However, systematic histopathological analyses have shown a high prevalence of asymptomatic inflammatory reactions in testicular biopsies from infertile men. The mostly focal lymphocytic infiltrates correlate with the degree of damage to spermatogenesis and corresponding clinical and endocrinological parameters of testicular function. Noninvasive diagnostic techniques are not yet available so that chronic asymptomatic inflammations of the testicles as the primary cause or cofactor of male fertility disorders are underestimated. Except for administration of pathogen-specific antibiotics, treatment recommendations are to a large extent still lacking.

Slutty men make themselves infertile with repeat infections. Even with antibiotics, it inflames the area and eventually it just… shuts down. The male reproductive set is external because it’s fragile. God hates sluts. ‘Sowing your oats’ is Satan rhetoric, dear. Scientism won’t save you.

Some thots are still going shopping in person in London.

It’s called the internet, bitch!

Spoiled rich pricks are complaining they were corona-cancelled.

I don’t think anyone’s told them no before. GOOD.

BE OFFENDED. Pandemics are marvelously impersonal.

Most of all, I was 100%, totally completely right.

and now I can be a cunt reminding everyone for the next ten years.

I wonder if tranny girls will stop taking T because high T makes viruses worse?

Well well well

“brown power” who’d come up with a concept like that?

The people who call them brown are being polite, shitskin is the impolite term.

Sargon: “If I didn’t know better, I’d say this was being created by Nazis who’d infiltrated the news organisation like ‘right, we’re gonna piss off as many white people as possible'”.

Why? Why would it anger them? That’s what immigration is, racial replacement, since the time of Rome.

The non-white useful idiots of the (cultural) marxists are slow and dense. Their masters with moneybags have a prescribed slow drip drop plan to extract max shekels. If you crank it up, to hit beat after beat in culturally offended hyperspeed, that triggers critical thinking in the dullest of normies. Thankfully, low IQ nationals are notoriously muh gibs impatient. Just tell them they’re entitled to everything every white person has ever stolen from them. It doesn’t have to make sense. They’re entitled and stupid. That’s why their countries are shit. High Time Preference is a political weakness in the r-type.

You have higher IQ sociopath ones and the majority (90%) low.

Their masters cannot rein them in nor control them. They don’t know that yet.

And they can’t object or roll-back their own plan in advance without undoing their work. All r-types even above average IQ are incredibly loss averse. Some say greedy.

Let them lock themselves in with an increasingly hungry crocodile, it’ll be fine!

For us, as long as nobody cucks and tries to “rescue” them. This is the anti-rescue, anti-cucking initiative. Mask drop the whole sodding lot of them.

They haven’t actually read the history of white people and how dangerous we are to one another.

They’ll remember.

Tick tock until they openly call for white genocide, Ash is already doing the pre-emptive dehumanisation section of the process.

The pro-immigrant whites are inherently white supremacist, thinking White 1st World countries are “better” and give a “better life” to poor browns and they should be grateful for it and become culturally white.

The Guardian whites are literal white supremacists – they want the world culturally white.

The virtue signalling and missionary work is insulting.

and do you, Sargon? Do you “know better”?

I don’t think so.

Chill, supposedly mixed race bro, it’s all a game. We’re winning.

Because unlike the fakecons, we’re actually trying?

People are waking up.

And we’re everywhere. 

In every industry.

Even showbiz.

In the places you’d least think.

We just wanted to be left alone but nooooooooooooooooooooo.

It’s the old Anonymous model, no central group for entryism and no obvious leader to pick out and imprison.

Good luck stopping that, SJW scum.