Human genetic load dragging us down

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4788123/
The long-term consequence of such effects is an expected genetic deterioration in the baseline human condition, potentially measurable on the timescale of a few generations in westernized societies, and because the brain is a particularly large mutational target, this is of particular concern.” The word is dysgenic for $200.
“Ultimately, the price will have to be covered by further investment in various forms of medical intervention.”


Good luck with dropping global IQ, if anyone dared measure such a thing. Sum IQ/global population is the equation.

Who will pay for this global NHS? Our NHS is already running down with a slight immigrant pressure.

It is slight compared to The World.

As JF has mentioned, “clusters” as seen in racial categorization, pertain to findings of genetic distance.

http://journals.plos.org/plosgenetics/article/file?id=10.1371/journal.pgen.0010070&type=printable

Examination of the relationship between genetic and geographic distance supports a view in which the clusters arise not as an artifact of the sampling scheme, but from small discontinuous jumps in genetic distance for most population pairs on opposite sides of geographic barriers, in comparison with genetic distance for pairs on the same side.

Called it.
https://disenchantedscholar.wordpress.com/2018/01/23/chromosomes-make-culture-including-language/

Speaking of germline mutations ruining society.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5597845/

Older fathers’ children have lower evolutionary fitness across four centuries and in four populations

Is that a large enough sample for the manosphere? They don’t like the answer so obviously not.

As if I don’t post topics strategically… tut tut.
https://disenchantedscholar.wordpress.com/2018/01/17/old-men-dead-babies/

“Higher paternal age at offspring conception increases de novo genetic mutations. Based on evolutionary genetic theory we predicted older fathers’ children, all else equal, would be less likely to survive and reproduce, i.e. have lower fitness.”
The sins of the father…
Old dads, no grandkids?
“showed negative paternal age effects on infant survival and offspring reproductive success.”
Sexual selection goes for young men, with better hormone profiles and less obesity.*
Darwin says aged paternal societies are bad for it. Nobody needs your dusty DNA. That’s a sign of corruption – keeping the young men down, not breeding. *another direct link to children again
“Effects survived tests for key competing explanations, including maternal age and parental loss,”
Again, you cannot blame the women.

“We can use our findings to aid in predicting the effect increasingly older parents in today’s society will have on their children’s survival and reproductive success. To the extent that we succeeded in isolating a mutation-driven effect of paternal age, our results can be understood to show that de novo mutations reduce offspring fitness across populations and time periods.”

As close as science gets to settled.

Old fathers are a sign of a dying society. Right up there with strippers and crossdressers. You passed the time period where you were viable with decadence but managed to breed anyway, that society’s social norms are seriously damaged. Applies to women too but paternal age is a bigger factor so I have to spotlight. In a Patriarchy, you would expect fathers are held to the highest standards possible. Their total authority relies on the moral authority consequent of that fact.

Chromosomes make culture, including language

Language is genetic, and it’s racial.

https://www.mpg.de/19395/Language_genetics

https://www.technologyreview.com/s/409215/the-genetics-of-language/
FOXP2 mutations exist in humans and we still count them as human despite the devolution (atavism) being common to chimps. For the same reason you can look like a long-dead relative, humans can have pre-human mutations. This is likely the (un-PC) explanation behind retardation and certain other disability in the nervous system. You’ve heard people can be maladapted for the modern world (low IQ) well, how about maladapted in any environment? Full stop. In every generation, those mutations re-emerge but less so over time since they don’t procreate.

“They connected FOXP2 to more than 200 genes that control the development of neurons, the release of neurotransmitters that send messages between nerves, and the changes in synapses that underlie learning and memory. Some of these genes will very likely turn out to be involved in speech and language.”
When people act like one gene between two groups isn’t a big deal, you can tell they’re thinking in ratios and not in biology. Your heuristic of enough is wrong.
“Geschwind has been captivated by this asymmetry, and by its relationship to handedness. Roughly 90 percent of us are right-handed, and nearly all righties depend on that left “perisylvian” region for speech and language. (About 40 percent of lefties instead rely on the right peri­sylvian region or use both hemispheres.) “There’s some kind of benefit to the kind of processing that’s going on in language–which is extremely rapid processing–to keep everything in one circuit in one hemisphere,” he concludes.”
Handedness being genetic and based on the nervous system. The brain is also part of the nervous system.
That would be the logical next step, to use the full brain. There are no racial studies on handedness.
“Francks and his colleagues could not corroborate that suggestion, but they did find a region of chromosome 2 that seemed linked to left-handedness. They then examined the DNA of pairs of healthy left-handed brothers: the same linkage to chromosome 2 surfaced, evidence that a gene or genes in that region might influence handedness.”
Again, one chromosome and a huge effect.
“They found four DNA differences that distinguished the schizophrenics from the mentally healthy lefties; the location of these variations led them to a gene called LRRTM1. Geschwind collaborated in the work that helped identify where in the human brain LRRTM1 was turned on, or expressed: it probably helps shape forebrain structures and influences how neurons connect.”
Imagine if we applied these methods to other concepts.
Neurogenetics.
“Moving from correlations between genes and disorders to knowledge of the neural circuitry that allows a human but not a chimp to ask, “To be, or not to be?” requires researchers to find connections between seemingly disparate findings.”
Like all races have a ton of Shakespeare level art. Where?
“Linguists and psychologists who have studied “talking apes,” including researchers who have taught them to communicate, stress that the animals rarely combine even two words into a semantic whole and never utter the type of complex “recursive” sentence–like this one–that embeds one thought in another.”
Possible to answer what is white by what is chimp?
These questions are old as Darwin. Genus>Species>Race. Undeniable. I can no less be non-white than a non-mammal.
“In the hope of beginning to explain this discrepancy, Geschwind investigated which genes are turned on in the brains of humans and in those of chimpanzees, our closest genetic relatives. He found hundreds of differences but had no way to determine which ones mattered–which were most significant in driving evolution and determining brain function. Overwhelmed, he turned to a mathe­matician friend at UCLA, Steve Horvath.
With Horvath’s guidance, Geschwind and his grad student Michael Oldham arrived at a new way to approach the problem. Rather than looking at differences between individual genes, they analyzed differences between networks of genes expressed at the same time.”
“It became startlingly clear not only which genes are uniquely human, but also which of those are most important.”
Race-mixing is risky because mutations are usually very bad for the fitness of the organism.
“Biologists believe that if proteins undergo little alteration over an evolutionary span of tens of millions of years, they must perform such essential functions that they simply cannot tolerate change.”
They aren’t as tailored to the old environment, or its pressures but the temporary social one.

Have you noticed African-Americans, left to their own devices, switch to speech patterns more common to Africa, compared with European Americans, who switch to European modes? It’s the little things. Bear in mind, this is after Africans were mixed for centuries.

Fake scientists are already trying to conflate language with racial classification, despite admitting you can learn if you start early enough, a language you wouldn’t naturally conform to.
https://www.nature.com/articles/s41598-017-01837-7
“the vast majority (97.3%) of individuals have mixed ancestry” then the term is meaningless, you’re conflating the species with the sub-species (race) and who are the exception?
“The data indicate that continents, ethno-linguistic groups, races, ethnicities, and individuals all show substantial ancestral heterogeneity. We estimated correlation coefficients ranging from 0.522 to 0.962 between ancestries and language families or branches.”
If language and race are so tied, if language is real…
“Initial efforts to characterize the movement of early humans in relation to ancestry grouped populations according to five geographical regions: Sub-Saharan Africa, Europe/the Middle East/Central Asia/South Asia, East Asia, Oceania, and the Americas. Subsequent analyses allowed for refinement of the genetic history of global ancestries, revealing regional structure through the identification of 7, 14, and 19 ancestries.”
And you don’t believe in race?
If you don’t believe in gravity, jump off a cliff. You’ll float.
The language/race correlation has clear genetic evidence elsewhere of causation and those same genes are also racial markers because races by geography differ in language structure.

Wait, so wouldn’t it work the other way? you ask, shitposting me.

Yes.

Yes, it would.

https://news.stanford.edu/2015/01/30/language-genes-compare-012915/

Multiregional?
Impossible…
“Now, scientists at Stanford and other universities have combined large databases of globally distributed linguistic and genetic data, revealing in greater detail how languages might change in parallel with genes.”
“Through an advanced statistical analysis, the authors found that geographic distance was linked to both genetic and phonemic distance. On average, the closer together two languages or two genetic samples were to one another, the more similar they were, even when the languages compared were not in the same language family.”
If genetic distance seems familiar, that’s the gap between races.
It doesn’t exist between sexes because a white woman can give birth to a white male, a white man can father a white female. Good luck trying that with race, if it isn’t genetic.

http://www.pnas.org/content/89/12/5620.full.pdf
http://journals.plos.org/plosgenetics/article?id=10.1371/journal.pgen.1003460
“Numerous studies of human populations in Europe and Asia have revealed a concordance between their extant genetic structure and the prevailing regional pattern of geography and language.”
Different people are different. Groundbreaking.
You can’t talk immigrants out of their genetic neural structures.
They won’t be uplifted being surrounded by white people, they’ll sink lower and lower from inbreeding until their envy provokes a civil war. You can’t bus in new brains.

In other news, Lombroso’s concept of criminal atavism has largely been proven, with the genetics of criminology and behavioural genetics coming to the fore.

Update: I should’ve given you search terms.

Look up left realism, right realism, Instrumental Marxism and Marxist criminology. All of these are real. Why is crime so bad? Well… there are a lot of liars after that human rights money. Ignore biocriminology at your peril, but they care about all the new rape victims. At least, their bank manager cares a lot.

Asian-European fertility issue

Partial re-post from: https://disenchantedscholar.wordpress.com/2017/03/30/racial-realities-mixed-race-fertility-and-neanderthals/
I wanted to expand a little because it’s ridiculous that I’m the top search result and I want to encourage public, detailed research on this topic.

http://blogs.discovermagazine.com/gnxp/2008/10/the-prenatal-wages-of-interracial-relationships/

Speciation is an ongoing process, it’s part of evolution, also an ongoing force. As members of a sub-species, better known as race, continue to diverge over time, the characteristic event will be infertility, fertility issues, birth defects and miscarriage. Once it is born, a failure to thrive and reproduce itself would also count as an adverse selection pressure.

My simple question: do we see this?

Oh, boy. Grab a drink, tall one.

The mixed-race dating pool is limited, to the other mixed-race, for example.
This lowers the potential fitness of the organism, compared to its parents’ baseline.

I’ll take a biomedical approach, from the limited information available.

“From EurekaAlert, Asian-white couples face distinct pregnancy risks…:”
http://www.eurekalert.org/pub_releases/2008-10/sumc-acf092508.php

Asians have a lower median birth weight, a racial difference as real as shorter African gestation periods compared to Whites.

“Although past studies have looked at ethnic differences in perinatal outcomes, the majority of research has focused on white- African-American couples. Few studies have focused specifically on Asian-white couples, said El-Sayed, who is also associate chief of maternal-fetal medicine.

More specifically, the researchers found that white mother/Asian father couples had the lowest rate (23 percent) of caesarean delivery, while Asian mother/white father couples had the highest rate (33.2 percent). Because birth weights between these two groups were similar, the researchers say the findings suggest that the average Asian woman’s pelvis may be smaller than the average white woman’s and less able to accommodate babies of a certain size.”

Nature is trying to tell you something there.

There is a clear natural selection pressure exerting itself.
Also, C-section birth puts the baby at a distinct disadvantage, those children have a weakened immune system, poorer health and fare worse in pair bonding.
https://www.newscientist.com/article/dn14662-c-sections-may-weaken-bonding-with-baby/
http://journals.plos.org/plosmedicine/article?id=10.1371/journal.pmed.1001973
Learning disability is on the tag list. Look for yourself.

https://jamanetwork.com/journals/jama/fullarticle/2473493

It doesn’t decrease infant mortality and can actually kill the mother over time.
https://www.theguardian.com/lifeandstyle/2017/oct/04/one-in-three-us-births-happen-by-c-section-caesarean-births
It’s serious surgery.
“It becomes routine but it is still a major surgery. That carries a long-term effect on maternal health.”
“Compared with women having a vaginal birth, those having a C-section for the first time have… a 5.7 times greater risk of an unplanned hysterectomy”

Nature is telling you something there.

“El-Sayed and his colleagues also found that the incidence of gestational diabetes was lowest among white couples at 1.61 percent and highest among Asian couples at 5.73 percent – and just under 4 percent for Asian-white couples. These findings weren’t altogether surprising: past studies have shown an increased risk of diabetes among Asian couples, which researchers attribute to an underlying genetic predisposition. But the interesting finding, El-Sayed said, was that the risk for interracial couples was about the same regardless of which parent was Asian.”

Dominant genes? No!

“Because of the results on Caesarean section rates they adduce that there is a pelvic size difference between Asian women and white women. Objective male observer acquaintances of mine have generally tended to back up this phenotypic difference between the populations.”

They’re shaped like pre-pubescent boys. Why else get surgery?
You should study it formally though. Asians have the lowest sexual dimorphism and it’s important to know the numbers.

http://med.stanford.edu/news/all-news/2008/10/asian-white-couples-face-distinct-pregnancy-risks-stanfordpackard-study-finds.html

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2867623/

“Although births of multiracial and multiethnic infants are becoming more common in the United States, little is known about birth outcomes and risks for adverse events. We evaluated risk of fetal death for mixed race couples compared with same race couples and examined the role of prematurity and low birth weight as potential mediating risk factors.”

Miscegenation doesn’t work, even with modern medicine.
This applies to black-white pairings too.

It is a disgrace adults are marrying without knowledge of the biology involved.

“Mixed race black and white couples face higher odds of prematurity and low birth weight, which appear to contribute to the substantially higher demonstrated risk for stillbirth. There are likely additional unmeasured factors that influence birth outcomes for mixed race couples.”

I cannot find a stillbirth study for Asian-White pairings, I’m sorry. Is it so common they need not study it?

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3807602/
I am looking, nobody is studying it.
I’m sorry, I am looking. It would be nicer if fewer babies were dying.

We have anecdotes?
https://www.temptasian.com/fyooz/after-3-miscarriages-the-zuckerbergs-are-finally-expecting-a-girl/
“Most people don’t discuss miscarriages because you worry your problems will distance you or reflect upon you — as if you’re defective or did something to cause this.” Mate choice is something you did. The baby didn’t choose to be conceived by you two. Part of your biology must be defective because miscarriage is an outcome of defective conception and/or pregnancy (there are many possible reasons, some environmental, a few random plus ‘stress’). It sounds cruel but yes, medically, something is wrong.

OT: Jews have a non-White miscarriage rate.
Jews invented/funded IVF because they needed it.
http://www.jewishvirtuallibrary.org/assisted-reproduction-and-judaism
https://www.haaretz.com/israel-news/.premium-1.529569
Israel is a eugenic ethnostate.
https://en.wikipedia.org/wiki/Simon_Fishel#Early_life_and_education
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1182091/
“The issue of the rate of recurrent miscarriages in high-risk Jewish women is unresolved.”
I am biting my tongue.

When trying really hard, the only evidence for hybrid vigour in White Americans vs. mulattos, which they sought to prove (scientism) is “relatively small.” …Is it present or not?
https://www.nature.com/articles/nature14618
“this study provides evidence [DS: the evidence isn’t proof?] that increased stature and cognitive function have been positively selected in human evolution, whereas many important risk factors for late-onset complex diseases may not have been.”
That’s bullshit, everyone is getting taller and getting better grades.
May not have been? In Nature?
Listen to the twisting in this: http://www.medicaldaily.com/g00/interracial-couples-may-make-taller-smarter-children-due-greater-genetic-diversity-341348
“Meanwhile, human evolution is more focused on the ability to create healthy offspring and have them survive infancy to continue raising them.”
Yes.

…Yes, it is.

“Whether you come from a genetically diverse background or not, in the end even the most common medical ailments that affect society will affect everyone, with genetic diversity having little to no impact.”
No, genes. The most common fatal medical ailments aren’t a cold, they’re genetic-based, it’s established fact. And if it had no impact, why push it?
“It combines the parents’ genetic material, resulting in offspring that possess a unique set of genetic blueprints that increase their chances of surviving and thriving compared to a population with limited genetic variability.”
No such thing. Limited genetic variability? No such thing. Where is this thing?
They’re just talking absolute crap to cover how their study was a non-result. Every genome is unique, between twins even. Thriving and surviving varies by individual genome, that should be studied by the natal people. You know this. You hide the scant data that is there with delusions. This is propaganda. It continues:
“This encapsulates Charles Darwin’s theory of natural selection,”
No, he wrote a whole book. Look at the subtitle to The Origin of the Species.
Natural selection is about death and mortality, which you have not studied. Disease is not death.
“where individuals with characteristics that increase their probability of survival”
how? like being able to give birth?
“will have more opportunities to reproduce,”
in a limited dating pool
“according to the University of California, Berkeley’s Understanding Evolution.”
If California understood evolution, it would be Alaska.
“As a result, their offspring will benefit from the variants,”
no, not if they’re the more common disadvantageous mutations or if the combination is novel and fatal
“which will spread throughout the population.”
No, you’re assuming they breed. Infertility exists, and it exists on a spectrum.

http://theconversation.com/being-south-asian-is-as-great-a-risk-factor-for-stillbirth-as-smoking-80074
There would be signs.

“This is an increased risk equivalent to smoking, advanced maternal age or obesity.”

“While other research has found the mother’s ethnicity places a role in the risk of a stillbirth, this has largely been put down to factors related to migration and social disadvantage. What our research shows is women born in South Asia and giving birth in Australia are at increased risk even when other factors are taken into account.”

D.N.A.

“There is growing evidence to suggest a mother’s ethnicity influences how fast her placenta ages as her pregnancy progresses.”

Asian placenta is old, got it.

“For some women, they can go into spontaneous labour sooner. In our study, we found South Asian-born women went into labour a median one week earlier than Australian- or New Zealand-born women.”

Racial differences in gestation duration, again.

“However, for others, an ageing placenta cannot meet the fetus’ increasing metabolic needs at term and beyond. And this increases the risk of stillbirth.”

Infertility, insufficient maternal resources for the fetus. That’s a kind of infertility. Considering how skinny they are and how those female curves are supposed to feed a baby, historically, this is not surprising.
Nature is aborting babies that would starve. Before it kills the mother too.

“And the length of telomeres in placentas from pregnancies ending in stillbirth are two times shorter than those from live births. In other words, the placental cells had aged faster.”

Superior Asian genetics people might wanna cover their innocent eyes.

“Some researchers have also studied ethnic differences in placental telomere length.
In an American study, placental telomeres from pregnancies in black women were significantly shorter than from pregnancies in white women (the ethnic backgrounds of the women were not further defined in the study).”

Superior European placentas. As you’d expect for the one race hit hard by an Ice Age. Perhaps this is an unknown r/K variable.

“Whether telomeres are shorter in placentas from pregnancies in South Asian-born women is unknown.”

Oh, I think I can guess.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4272534/

“There was a high prevalence of stillbirth in this multi-ethnic urban population. The increased risk of stillbirth observed in non-White women remains after adjusting for other factors.”

Whites are different? Biologically? Shudder-gasp!

Let’s see if BMI matters.
https://jamanetwork.com/journals/jama/fullarticle/1860462
Yes. Of course it does. They only studied high BMI though.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4392932/

“However, BMI does not take into account the relative proportions of fat and lean tissue and cannot distinguish the location of fat distribution”

“However, these are based on information derived from the general population, based on risk of mortality, without consideration for racial or ethnic specificity and were not determined to specifically identify those at risk for diabetes. Recently, the U.S. Centers for Disease Control and Prevention presented initial findings from an oversampling of Asian Americans in the 2011–2012 National Health and Nutrition Examination Survey. These data, utilizing general population criteria for obesity, showed the prevalence of obesity in Asian Americans was only 10.8% compared with 34.9% in all U.S. adults (13). Paradoxically, many studies from Asia, as well as research conducted in several Asian American populations, have shown that diabetes risk has increased remarkably in populations of Asian origin, although in general these populations have a mean BMI significantly lower than defined at-risk BMI levels (14,15). Moreover, U.S. clinicians who care for Asian patients have noticed that many with diabetes do not meet the published criteria for obesity or even overweight.”

So we’d need to look at WHR, instead of BMI.

New Evidence on Waist-Hip Ratio Reveals Surprising Relationship to Fertility, Urges Revision of Attractiveness Theories

“In women, the connection between WHR and health measures appears to be hormonal. It is known that ratios of estrogen, progesterone, and prolactin affect all of these features. The “right” balance promotes both health and low WHR. One version of the “attractiveness theory” posits that our attraction to this body shape developed as an indicator of overall health.”

“Another crucial part of the attractiveness theory of wait-hip-ratio (WHR) is that this body shape has to be indicative of something related to fertility, or else it wouldn’t have any evolutionary value.

The key feature in a potential mate is biological fitness, that is, the potential to give birth to many healthy and successful offspring.

Desirable females, in the evolutionary sense, are those that are likely to be healthy, fertile, and robust.

Robust = pelvis, btw.
Venus was never a narrow-hipped vixen.
The body acceptance people should really focus on the hips.

A low WHR, it is thought, must correlate with fertility (ability to have children) and/or fecundity (tendency to have large numbers of children).”

There is such a thing as too low. Boyish figures have less fat, fewer curves and narrower hips.
They’re confusing women who have obesity and babies for State money with natural attractiveness, fecundity in the state of nature and blurring BMI with WHR. Nobody said unhealthy (low) WHR is wealthy, for fecundity. That’s a strawman. The hormones and other details, medical details, are better profiled in the most nubile WHR range. It is a range. Don’t line graph me, study.

It doesn’t mention race although many women in the world do not have a figure. Unless you count a figure of 1.

Hormones and junk: http://www.independent.co.uk/life-style/health-and-families/health-news/health-what-a-man-cant-resist-the-perfect-waist-hip-ratio-forget-about-breasts-says-jerome-burne-its-1440859.html

“The waist is one of the distinguishing human features, such as speech, making tools and a sense of humour,’ says Professor Singh. ‘No other primate has one. We developed it as a result of another unique feature – standing upright. We needed bigger buttock muscles for walking on two legs.”

If the waist makes the human, a lot of women are fucked.

The ideal ratio in healthy pre- menopausal women ranges between 0.67 and 0.8. In terms of the tape measure, this is produced by waists between 24in and 28in with 36in hips, and waists between 27in and 31in with 40in hips.”

A range.
…How many Asian women have a 36″ hip?
The fat ones I’ve seen were pufferfish.

“come puberty, the sex hormones start directing it differently.”

sexual dimorphism

“Oestrogen, the hormone of female sexual characteristics, concentrates it on the buttocks and hips while the masculinising hormone testosterone encourages fat to form around the waist.’ At the same time testosterone encourages fat to be burnt off the buttocks while oestrogen takes it off the abdomen.
These characteristically feminine fat stores are used in the last months of pregnancy and during breast-feeding. This is another reason why women who are seriously underweight often stop menstruating – they would not have the resources to support a pregnancy or a baby.”

Confetti time.

“Women with a low ratio, Professor Singh says, tend to start ovulating younger, and those with a high ratio find it more difficult to become pregnant and tend to have children later. [not by choice]
Although a high waist-hip ratio most commonly goes with being overweight, it can also be found in women of normal weight who have high testosterone levels – a condition that is also associated with being hairy, infertile and having a ‘male’ body shape.”

Manly body, fertility problems. Study it. Avert tragedy.

“In a survey of 106 men aged 18 to 22, the favourite was a female of average weight with the classic hour-glass figure. Not only were such women rated as young, sexy and healthy, they were also seen as ideal for childbearing.”

Again, sexy is different from beautiful.
Porn is a lie.

“The young men regarded the underweight women – defined as women of 5ft 5in weighing less than 90lb – as ‘youthful’ but not particularly attractive, especially for childbearing.”

To prefer the obese over the mannish figured for motherhood is huge.
Youthful is code for making them feel like a pedophile.

“In Professor Singh’s other surveys, men of all ages agreed with these findings – thus bearing out her theory of the waist-hip ratio.”

Women dropped the corset to signal they weren’t just baby-making machines.
It’s hard to test low-WHR women in a world of obesity.

https://www.newscientist.com/article/dn4953-barbie-shaped-women-more-fertile/

“Women who were extremely underweight or overweight were not included.”
Study them separately?
Porn is making you drawn to infertile women, with boy hips. Conditioning.

WHRxfertility
http://www.sciencedirect.com/science/article/pii/S0191886904003617
“Figures of average weight and a WHR of 0.7 were rated as most attractive and healthy.”
It is important.

I want to see a study that looks at racial WHR against pregnancy issues.
Is that so hard to ask?

http://www.sciencedirect.com/science/article/pii/S109051381200044X

“These data indicate that BF% appears to be a strong cue for attractiveness and that the impact of WHR and BMI on attractiveness is dependent, in part, on BF%. The appearance of body fat may provide disruption in the visual cues of both shape and size of the female body, potentially impacting behavior.”

Speciation is determined by biological compatibility in sum. This includes many factors. On none I have seen do Asian-White hybrids succeed over their parental groups’ averages; even IQ gains, if true, would be worse for the individual’s own fertility rate.

The only other thing I could think of is a study on STD rates between couples.

https://www.ncbi.nlm.nih.gov/pubmed/2117964
http://sti.bmj.com/content/87/Suppl_2/ii14
http://www.expat.or.id/medical/stds.html
“The association between travel and STDs has been known for centuries”
What’s the Asian version of burn the coal? Pick the chopstick, get ripped?
http://global-disease-burden.healthgrove.com/l/24974/Syphilis-in-Southeast-Asia
Prevalance: “fairly common.”
The wages of sin. You can’t blame the white man.
https://link.springer.com/article/10.1007/BF02438113
Syphilis present in Asian archaeological samples.
http://www.scmp.com/news/asia/east-asia/article/2060294/young-women-among-sufferers-japan-records-huge-spike-syphilis
“Endemic syphilis”
https://emedicine.medscape.com/article/1952297-overview
http://sti.bmj.com/content/76/6/415
‘referred to as “the intraracial network effect,”’
Oh, that’s why they don’t study it.
“suggest that assortative mixing prevents the spread of STI to other subpopulations.”
“A number of studies in the literature, many of which did not measure biomedical markers of STI, suggest that mixing across subpopulations may contribute to spread of STI in the population, particularly across subpopulations.”
If you increase the microbe’s exposure to different parts of the human genome, it will evolve faster. Simple?
Age groups can be a larger factor, since the older immune system is weak and better for the microbe.
“In a recent study conducted in Seattle we found that most of the disease burden for gonococcal and chlamydial infections in both high prevalence and low prevalence subpopulations was attributable to mixing within the subpopulations”
I think we’ve found the reason white women mix out the least. Same reason we don’t like to eat meat raw – to avoid disease.
‘the proportion of infection attributable to indirect mixing, or so called “bridge populations,”
So it is attributable and naturally must inform sexual behaviour.
“While we found that sexual mixing between particular racial ethnic subpopulations increased the risk of STI significantly, the proportion of the population engaging in sexual mixing, and the numbers of sex partners reported by individuals engaging in sexual mixing across racial-ethnic subpopulations were too low for this increased risk to play a major part in disease burden.”
Hybrid vigour, guys!
The risk isn’t the major part, it’s fine! Water’s fine!
“The literature on racial-ethnic differentials in STI rates and the role of racial ethnic mixing on the spread of STI is emergent; many questions still remain unanswered.”

If miscegenation were unhealthy, we’d know, right?

Taxonomic approaches to race

There is no race, only the human race.”

Literary terms [1] are anti-science.

http://www.toqonline.com/archives/v8n3/TOQv8n3Hamilton.pdf

Purely to wind up Sargon of Cuckad.

On that last line, boy do I have an interesting post scheduled.

Links or it didn’t poem.

http://www.bartleby.com/205/25.html

Oh! that the Desert were my dwelling-place,
  With one fair Spirit for my minister,        
  That I might all forget the human race,
  And, hating no one, love but only her!

“What is white?”

http://genome.cshlp.org/content/14/9/1679.full

I knew I had the reference somewhere.
Racial allele gradients needn’t be precise to count for categorization, Sargon.
The absence of so-called discrete clusters would be expected WITHIN a species.
Race = sub-species.
The weasel word in the abstract is “major”.
The PDF is free.
Discontinuity is itself an arbitrary concept, depending how you crunch the numbers.

Typical Asian IQ lower than Whites

I thought this was obvious but I hadn’t cited why before.

Flynn is one of the greatest writers on IQ. He wrote a book on it.
https://www.amazon.co.uk/Asian-Americans-Achievement-Beyond-Iq/dp/0805811109

Historically, this has been the case, there isn’t something magical and innate about it. Mathematics, while teachable, is only one element of IQ. Then there’s the widespread cheating, which would drop the score average a few points, bringing us to approximate parity in spite of their educational investments and parental abuse.

Many modern studies make the sampling error of only studying brain-drain immigrants, which confounds by class (the biggest factor) and education (affluence to be in the first world). They also isolate specific populations of Asia instead of testing the entire race.
Flynn did the leg-work. If you want to know about Asian IQ, this is the seminal book on the topic.
Accept nothing less.
He distinguishes between IQ and achievement because no, they are not the same. The variables are discrete.
“In this case, the author shows that Chinese Americans’ occupational achievements are generally far beyond their IQ — as if they had a mean IQ 21 points higher than they actually do.”
You can’t blame nepotism on all of it.
We can learn from them to boost our own. Not to copy everything, but some of it.

Here’s a taster he wrote on the topic if you’re unsure about his writing.

http://www.eyeonsociety.co.uk/resources/UAIChapter18.pdf
Abstract:

“The problem addressed in this paper is first to thoroughly document,
and then to explain, the impressive scholastic, occupational, and income
achievements of Asians in America. In the past, some psychologists
have cited apparently impressive evidence of a superiority in general
cognitive ability. It turns out that that this evidence is seriously flawed
– and not merely because of failure to allow for the intergenerational
increase in scores but also as a result of seemingly endlessly compounded
sampling deficits and corrections and adjustments introduced into the
norming studies. Be that as it may, with IQ held constant, the Asian’s
achievements exceed those of Whites by a huge amount. Once an IQ based
explanation has been discredited, attention focuses on issues rarely
discussed by psychologists – such as other psychological characteristics
and multiple cultural supports. These are contrasted with those operating
in other cultural groups, some of which perform far below what might
otherwise be expected.”

In pure IQ terms, Asian supremacy is a myth. Do not conflate the variables.

This book debunks in full why that is and I can add nothing to it.

IQ isn’t everything, as the “model minority” show.

Here, Asian is a PC term used to refer to certain types of religious rape gangs. It isn’t a paragon in our minds, they represent something closer to the Asian average (look at the rape rates and compare to Africa), and yes, regression to the racial and lower-class mean does apply in these cases, over time (as they’d be breeding in those populations and living in those conditions).

Genographic Southeast Asia

https://voices.nationalgeographic.org/2015/04/21/genographic-southeast-asia/

Oh look, more migrations they try to shoehorn into Out of Africa but actually fits Multiregional better.

I am shocked at the continual efforts to bury lies with genetic truth.

Over half of SE Asian males? At least the Yellow Fever acolytes have new reading material.

For exactly what type of cuck they are, on a precise genetic level.