Superstitious minds

Mini post. Kinda. Why is Benedict Cumberbatch so ugly?

No really. If we’re doing red pill observations, humour me.


I mentioned before about old world superstitions forgotten in recent years.
As recently as my parent’s generation, they considered ugly children the product of sin, that God was punishing their parents for their sin. You can still find this info around if you look but they rarely dive into it.

You could say it’s about STDs but back then people rarely travelled and slept around enough to frequently catch them. The modern microbiome of the slut is more taxed. So what?

Back to the school mocking. If a child had always married parents but became ugly in the teens, questions would be asked openly and they would get teased about whether one or both parents had ever cheated. This is where we get the term bastard. It isn’t actually about bastards, it’s about ugliness. The ugliness of parental deceit.

You can pretty much tell when there’s a birth defect in a baby, the eyes look dull if it’s mental. It’s a known indicator of fatal defects.

https://www.sciencedirect.com/science/article/pii/S1875957214001703

2015 Birth Defects in the Newborn Population: Race and Ethnicity

Overall birth defect prevalence was 29.2 per 1000 in a cohort of 1,048,252 live births, of which 51% were Caucasians.

Full white or mongrelised? Let’s assume pureblood despite America (mixed white, mostly). American whites are on average less attractive as white blended than single nation counterparts, even living in America. Models tend to come from homogeneous national areas, (i.e. subrace) a finding that is known to apply to white settlers in Brazil to this day, they send scouts. Specifically.

https://www.thecut.com/2010/06/model_scouts_find_more_than_ha.html

Compared with Caucasians, the risk of overall birth defects was lower in African–Americans (relative risk = 0.9, confidence interval 0.8–0.9) and Hispanics (relative risk = 0.9, confidence interval 0.8–0.9).

Failure to consider abortions for “no” reason or gender as defective. Selection bias. A lot of those already had abortions because they’re high abortion groups!

The risk of overall birth defects was similar in Caucasians and Asians. Relative to the Caucasians, African–Americans had a lower risk of cardiac, genitourinary, and craniofacial malformations but a higher risk of musculoskeletal malformations. Hispanics had a lower risk of genitourinary and gastrointestinal malformation. Asians had a higher risk of craniofacial and musculoskeletal malformations.

Didn’t control for proportion in the population, then non-whites are way ahead.

Craniofacial = ugly. 

Musculoskeletal = ugly. Well, dumpy.

Unless you’re going to argue a big is beautiful for literal birth defects?

And “similar” isn’t same. It isn’t statistical. This is like IVF success studies again (see below).

Why did some old world men witness the birth? All babies look like those reddish potatoes, it can’t be a resemblance. You can tell a resemblance to one parent over another by middle childhood to puberty.
We’re told that it’s about adultery and it might be true if you suspect a man with certain features e.g. skin colour, an extra finger.

Yet, what can you tell at birth? Ugliness.
Whether or not the man in question remembers that reason.

Cinderella effect also applies to genetic but ugly kids (lookism, it’s aka). The parents reject them, even if one genetically caused their fug.

Take Cumberbatch, product of a union involving adultery.
Fugly. Nice voice, but his father is the looker. Mother is a looker too. The issue cannot be genetic.

Some superstitions have a basis in fact.

Why did old ladies peer into a pram to judge the ugliness of the babe?

To see if you’re a SINNER!

[inc Thou shalt not adulterate]

Picking on an ugly white guy wouldn’t be totally kosher. I have other evidence.

We’re looking for spiteful mutants.

Now the post gets huge.

To more data, ever more data, smother the liars in data:

https://www.ons.gov.uk/aboutus/transparencyandgovernance/freedomofinformationfoi/informationregardingmixedraceparentage

“Please may I request the following information, records and documentation under the Freedom of Information Act:

Information in regard to people of mixed race parentage- often called ‘white and black Caribbean’, ‘white and black African’, ‘white and Asian’, ‘other mixed’- being at increased risk of being born with a birth defect, stillborn, or of suffering from fertility problems in their adult lives, which is related to their mixed race parentage

Information regarding NHS policy and practice on the advising of interracial couples, who are prospective parents, about the increased risk of their child being born with a birth defect, stillborn, or infertile in adult life, which would be connected to their, the child’s, mixed race parentage

Please may I also request statistical information and records which display the following:

The percentage of overall cases of babies born with a birth defect, which is attributable to each ethnic group

The percentage of overall cases of babies still born, which is attributable to each ethnic group

The percentage of overall cases of infertility, which is attributable to each ethnic group

The percentage of overall births, which is attributable to each ethnic group”

Reply:

“In Tables 8 and 10, mixed race is included in a single category of Mixed, Chinese and any other ethnic group. This is because the numbers in these groups are sufficiently low to risk being disclosive, and follows agreed statistical guidelines.
a) being born with a birth defect – this information is shown in Table 10.
b) being still born – this information is not published. However, you could request a special extract (further details of how to do this are explained below).
c) we do not hold any information on infertility, and are therefore not able to answer your question about adults suffering from fertility problems, connected to their mixed race parentage.”

Do not hold information my lily-white arse.

https://www.independent.co.uk/voices/infertility-ivf-nhs-race-lgbt-asian-black-women-a9216921.html

Table link: https://www.ons.gov.uk/peoplepopulationandcommunity/healthandsocialcare/causesofdeath/bulletins/gestationspecificinfantmortality/2014-10-15

“Page does not exist”.

It’s this paper.
https://www.ons.gov.uk/peoplepopulationandcommunity/healthandsocialcare/causesofdeath/bulletins/pregnancyandethnicfactorsinfluencingbirthsandinfantmortality/2014-10-15

“Some research suggests that Black and Asian women have shorter gestation than White European women, and that this may be due to earlier fetal maturation (Patel et al., 2004). The discrepancies in gestation by ethnicity may also be explained by socio-economic, behavioural and physiological differences among the different ethnic groups (Gray et al., 2009).”

In an ONS report. They know.

“Table 10 (184.5 Kb Excel sheet) shows that for four of the five combined ethnic groups analysed, the most common cause of infant death was immaturity related conditions

(Black, 54%;

Mixed, Chinese and any other group, 44%;

White, 43%;

For a majority, that’s incredibly low.

and those where ethnicity was

not stated, 49%).

For the Asian group, the most common cause was congenital anomalies (41%). A higher incidence of congenital anomalies in Asian populations is well-documented (Gray et al. 2009).”

http://www.ons.gov.uk/ons/rel/child-health/gestation-specific-infant-mortality-in-england-and-wales/2012/rft-table-1.xls

Low birthweight and prematurity are both measures of fetal development. Another measure is the baby’s size in relation to its gestational age. Babies whose birthweight lies below the tenth percentile for their gestational age are known as ‘small for gestational age’ (SGA).

Not all babies who are SGA have a pathological growth restriction; they may just be constitutionally small.

read: racially

This may explain why babies of Bangladeshi, Indian or Pakistani origin are more likely to be SGA than White British babies.”

Smaller brains too. Inbreeding depression but also group average by nation. Look at national IQ.

https://www.photius.com/rankings/national_iq_scores_country_ranks.html
Bangladesh 82
Over one whole standard deviation below. According to the likes of Peterson, useless to a Western economy. The average Bangladeshi.
India 82
Recall regression to the mean. Also, friendliness correlates more to low IQ. Do not be fooled.
Pakistan 84
Thailand 91
Philippines 86
Nigeria 84
Jamaica 71, where we’re picking up new NHS nurses.

Enjoy that decline.

Tables 8 and 10 mentioned in FOI request not listed, have to know it’s there.
Under Downloadable Tables:

“Table 8: Live births, neonatal and infant mortality by ethnic group and gestational age at birth, 2012 birth cohort, England and Wales

Table 10: Infant mortality by ONS cause groups and broad ethnic group, 2012 birth cohort, England and Wales”

For future reference, write your FOI requests as “concern for services provided to BAME women” and “progressive need for up-to-date medical guidance for mixed race couples and the biracial in family planning”.

You have to download the excel, click to tables 8 and 10, then read the footnote of superscript 1 to know to scroll right.

Table 8: All others^1
7.1% under 37wks
9.2% SGA

Black SGA: 9.2 and 12.3%.
Bangladeshi, Indian, Pakistani only SGA: 17%, 16.3%, 14.2%.
White SGA: 7.2%, 6.2%.
Unknown 8.2%.
ALL SGA average: 8.2%.

Something’s off.

Pre-term neonatal deaths
Total: 869
B,I,P: 9, 30, 47
Black: 39, 13
White: 549, 63
Unknown, not stated: 32
All others^1: 87
For such a vanishingly small percentage of the population, how is it 87?
10% of pre-term deaths were “1 Chinese, Other Asian, Other black, Other and all Mixed groups.”

Do you see what I see?

For non-statistically minded people:

Infant death, pre-term
Total: 1232
B 21
I 41
P 66
Black African: 62
Black Caribbean: 20
W native 750
W other 86
Not stated 48
All others^1: 138

See it yet? If you controlled for population ratio, it’d be more dramatic by far.

This is why they hide it and I have to make my own charts.

Term infant deaths
Total: 895
All others^1: 102.
That’s 11.4% from a tiny group of mixed.

Table 10 screen-capped, do your own charts.

Related studies, I do have a point about measurement error.

https://iussp2009.princeton.edu/papers/93139
2009 Fertility by ethnic and religious groups in the UK, trends in a multi-cultural context

Asian tsunami in USA too
https://www.statista.com/statistics/226292/us-fertility-rates-by-race-and-ethnicity/

https://www.statista.com/statistics/281416/birth-rate-in-the-united-kingdom-uk/

From one of the links, can’t find which. Calm down. Either they’re abstaining from having kids once here, infertile, the neonate dies or it’s retarded. Being here is actually a curse since they’re held to the standards and economy of a higher IQ nation. They’re voter birds here for a season or tax chattel and they’ll leave when it’s convenient to.

Ethnicity and IVF

“How a patient’s ethnic background affects her chance of pregnancy, especially with IVF, is a fascinating yet poorly studied area of research. According to a 1995 national survey of family growth, non-Caucasian married women were more likely to experience infertility than Caucasian married women, yet these same non-Caucasian women were less likely to receive any type of infertility treatment—especially treatment with assisted reproductive technologies.

There is very little data in the literature examining ethnicity and its affect upon pregnancy rates with in vitro fertilization (IVF). Ethnic minorities compose a small percentage of patients in the nation’s IVF programs, making it relatively difficult to examine how they respond to various infertility treatments. In the few studies that have examined the affect of ethnicity on IVF pregnancy rates, differing outcomes have been found.

There have been only a few studies specifically comparing IVF success rates between African Americans and Caucasians. The results of two of these studies contradict each other, with one showing that African Americans had decreased pregnancy rates with IVF as compared to Caucasians, and the other finding no difference in pregnancy outcomes with IVF between these two ethnic groups.

Likewise, there are only a few studies directly comparing IVF pregnancy outcomes between Indians and Caucasians. One shows a trend towards decreased pregnancy rates in Indian women and finds that Indian women were significantly more likely to have their cycle cancelled as compared to Caucasian women. In comparison, another study found no significant difference in IVF pregnancy rates between Indians and Caucasians. A more recent study has shown that Asian ethnicity was an independent predictor of poor outcome with IVF. There have been no studies examining IVF pregnancy outcomes in Hispanics in comparison to any other ethnic groups.

We’ll see why.

When I was in training, I published the first study comparing IVF outcomes among multiple ethnic groups. It was a retrospective study utilizing a data set that was the result of the collaboration between three IVF centers in the Boston area: Boston IVF, Brigham and Women’s Hospital IVF Center, and Reproductive Science Center.
We retrospectively reviewed the cycles of 1,135 women undergoing IVF between 1994 and 1998. Only the first IVF cycle for each couple was reviewed. Ethnicity was self-reported. Women who categorized themselves as having a mixed ethnic background were excluded.

Seriously. Measurement bias much?

….In order to better understand how ethnicity affects IVF outcome, it will be necessary to study a larger number of minority patients. In these studies, it is important that all ethnicities be included. If racial differences do exist, IVF treatment protocols could be adjusted to improve the success rates for patients of all ethnic backgrounds. Therefore, further exploration in this area is necessary and very important.”

We did that.

https://www.rcog.org.uk/en/news/bjog-release/

“After adjusting for certain factors including the age of the patient at time of treatment, cause of female or male infertility, and type of treatment (ICSI vs IVF), the study found that White Irish, South Asian Indian, South Asian Bangladeshi, South Asian Pakistani, Black African, and Other Asian women had a significantly lower odds of a live birth than White British women. For example, the live birth rate for White British women was 26.4% compared to 17.2% for White Irish women and 17.4% for Black African women.

The study also found that some groups of women including South Asian Bangladeshi, Black African, Middle Eastern, have a significantly lower number of eggs collected than White British women.

Moreover, South Asian Indian, South Asian Bangladeshi, South Asian Pakistani, Black British, Black African, Black Caribbean and Middle Eastern women were at a higher risk of not reaching the embryo transfer stage.

The paper explores the possible reasons behind the variation and states that while genetic background could be a potential determinant of egg and sperm quality, variation in environmental exposures relating to lifestyle, dietary factors, socio-economic and cultural factors could be influencing egg and sperm quality, accessibility of fertility treatment and behaviour towards seeking medical care and consequently reproductive outcomes.

No, they were living in the same place. Muh Magic Dirt.

Genetics is the ONLY difference now.

You have NOTHING.

DNA causes germline DNA, really? Maybe?

Furthermore, the increased prevalence of polycystic ovary syndrome (PCOS) in South Asian women may have an impact on egg quality and lower implantation rates.

Shit tier WHR tipped us off on that one, see end.

Dr Kanna Jayaprakasan, Consultant subspecialist in Reproductive Medicine, Derby Fertility Unit, Royal Derby Hospital; Honorary Associate Professor in Gynaecology, University of Nottingham and senior author of the paper, said:

“The data suggests that ethnicity is a major independent factor determining the chances of IVF or ICSI treatment success.

“While the reason for this association is difficult to explain, the potential factors could be the observed differences in cause of infertility, ovarian response, fertilisation rates and implantation rates, which are all independent predictors of IVF success.

“The main strengths of the study are the use of the UK HFEA national database which includes a large number of women treated in all UK units. However, the numbers in some of the sub-ethnic minorities, such as Bangladeshi women, were low in the study.”

Professor Adam Balen, spokesperson for the Royal College of Obstetricians and Gynaecologists (RCOG) and Chair of the British Fertility Society (BFS) said:

“Infertility affects 10-15% of the population and more people are seeking fertility treatment.

“This interesting study looking at maternal ethnicity provides useful data based on a large number of women undergoing fertility treatment. The reasons behind the variation need to be looked at in more detail but in the future could potentially help improve success rates amongst all groups of women.”

Nope!

https://www.sciencedirect.com/science/article/abs/pii/S1472648315002564

“Black and South Asian women were found to have lower live birth rates compared with White women”
“Black and South Asian women seem to have the poorest outcome, which is not explained by the commonly known confounders. Future research needs to investigate the possible explanations for this difference and improve IVF outcome for all women.”

Almost like Anglo women evolved to breed in the Anglo climate?

The Ice Age killed the boyish ones.

MORE:

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2636517/

“Variation in risk factors and outcomes was found in infants of White mothers by paternal race/ethnicity.”

I wonder which way.
Inbreeding or outbreeding depression?

Guess.

“Status exchange hypothesizes that in a marriage market framework, minority men marry less-desired White women (e.g., of lower education) in exchange for higher social status. The second hypothesis, in-group preference, simply suggests that people prefer members from their own group, and thus, intermarriage is the less desirable scenario.”

Dudebros like “where’s da studies?”

I’m like “Have you even looked?”

“Together they found that mixed-race couples differed significantly with respect to their sociodemographic characteristics from the endogamous couples. After control for those variables, biracial infants were found to have worse birth outcomes than infants with 2 White parents but better than infants with 2 Black parents.6,8–12 (Henceforth, infant’s race/ethnicity will be referred to by the notation “maternal race/ethnicity–paternal race/ethnicity” [e.g., White–Black].)”

DING DING DING DING DING

TIL Wombs iz white supremacist.

“Consistent with Table 1, infants in the White–unreported group had the worst birth outcomes in each category.”

Trans. mixed. Likely Asian since S. America and Black are already covered.

Learn to read, weebs.

“In general, I found substantial variation in birth outcomes within the group of infants with White mothers and fathers of different racial/ethnic groups. This is interesting because it shows that the common practice of using maternal race/ethnicity to refer to the infant’s race/ethnicity, regardless of father’s race/ethnicity, can be problematic.

aka nice way of calling out deception

For example, it is not uncommon for a study to refer to infants of White mothers as “White infants,” even though “White infants” may imply that the fathers are White. In this study, I demonstrated that infants of a White mother and a White father, the real “White infants,” have the better birth outcomes than do those infants of a White mother and a non-White father. Therefore, the practice of using “White mother” to refer to White infants will yield lower estimation of the birth outcomes because there are infants of non-White fathers in the sample.”

They know. It’s a cover-up.

Category errors galore.

“The infants in the White–White group had the most-advantaged birth outcomes, followed by infants in the 3 Hispanic-father groups. Infants in the White–Black group had the second-most-disadvantaged birth outcomes; the differences in birth outcomes between White–Black and White–White infants were statistically significant: White–White infants had a 2% (70 g) higher average birthweight, 26% lower LBW rate (4.64% vs 6.26%), and 39% lower infant mortality rate (0.43% vs 0.71%) than did White–Black infants. Infants in the White–unknown group had the most-disadvantaged outcomes in each category. These heterogeneities within White mothers show that the common practice of using maternal race/ethnicity to refer to the race/ethnicity of the infant is problematic: White–White infants had the best birth outcomes among the groups studied, so any other paternal race/ethnicity pulls down the averages for all White mothers. That is, the birth outcomes of White–White infants are actually underestimated by researchers who use mothers’ race/ethnicity to refer to infants’ race/ethnicity, and thus, the racial/ethnic disparities between White and any other race/ethnicity may be underestimated accordingly as well.”

Relevant!

“…Clearly, the unreported father is a proxy for more-noteworthy factors, because if unreported fathers were merely missing from certificates, their infants’ outcomes should not be so much worse.”

What DO these studies have in common? [Asians]

Could also be child of rape as a confound.

You’ll see.

2012 Biracial couples and adverse birth outcomes: a systematic review and meta-analyses.
https://www.ncbi.nlm.nih.gov/pubmed/22776059

“Biracial status of parents was associated with higher risk for adverse pregnancy outcomes than both White parents but lower than both Black parents, with maternal race having a greater influence than paternal race on pregnancy outcomes.”

Evolution is racist or instincts evolved for reasons? Pick ONE.

Your Third World surrogate plan may need retouching.

If it fails or dies or gets retarded, you still gotta pay up! What are the odds?

Why is it so hard to find studies about the most populous race on the planet?
https://www.ncbi.nlm.nih.gov/pubmed/31238617

https://www.ncbi.nlm.nih.gov/pubmed/30564431
2018
What is associated with IQ and other development issues? Pre-term birth.

“Maternal age, education level, race and ethnicity, smoking during pregnancy, and parity were significant risk factors associated with PTB.”

It’s mentioned along with smoking.

“…The analysis of interactions between maternal characteristics and perinatal health behaviors showed that Asian women have the highest prevalence of PTB in the youngest age group (< 20 years; AOR, 1.40; 95% confidence interval (CI), 1.28-1.54).”

I want more studies about them. I’m not scared of reality.

That suggests a genetic predisposition to be present so young. I’d compare PTB to WHR, personally.

“Pacific Islander, American Indian, and African American women ≥40 years of age had a greater than two-fold increase in the prevalence of PTB compared with women in the 20-24 year age group.”

Their own women.

Pre-term study and IQ:

https://pediatrics.aappublications.org/content/136/3/415
“RESULTS: Across all assessments, VP/VLBW individuals had significantly lower IQ scores than term-born controls, even when individuals with severe cognitive impairment (n = 69) were excluded. IQ scores were found to be more stable over time for VP/VLBW than term-born individuals, yet differences in stability disappeared when individuals with cognitive impairment were excluded. Adult IQ could be predicted with fair certainty (r > 0.50) from age 20 months onward for the whole VP/VLBW sample (n = 260) and from 6 years onward for term-born individuals (n = 229).

CONCLUSIONS: VP/VLBW individuals more often suffer from cognitive problems across childhood into adulthood and these problems are relatively stable from early childhood onward. VP/VLBW children’s risk for cognitive problems can be reliably diagnosed at the age of 20 months. These findings provide strong support for the timing of cognitive follow-up at age 2 years to plan special support services for children with cognitive problems.”

So it doesn’t cause but it is associated. Humans evolved long gestation for the brain.

Clear defect evidence in the genes- study it!
https://www.ncbi.nlm.nih.gov/pubmed/29903290

But surely, you say, genetic issues would be also hormonal (hormones regulate genes as well) and apply to men?
Well…
https://www.ncbi.nlm.nih.gov/pubmed/31348744
Yes. Yes it would.

“A total of 9079 patients were reviewed, of which 3956 patients had complete data. Of these, 839 (21.2%) were azoospermic. After adjusting for age, African-Canadians (odds ratio [OR] 1.70; 95% confidence interval [CI] 1.28-2.25) and Asians (1.34; 95% CI 1.11-1.62) were more likely to be azoospermic compared to Caucasians.”

Some of us form opinions AFTER reading.
White men are literally more fertile and most fertile with white women.

“Similarly, African Canadians (OR 1.75; 95% CI 1.33-2.29) were more likely to be oligospermic and Asians (OR 0.82; 95% CI 0.70-0.97) less likely to be oligospermic. Low volume was found in African-Canadian (OR 1.42; 95% CI 1.05-1.91), Asians (OR 1.23; 95% CI 1.01-1.51), and Indo-Canadians (OR 1.47; 95% CI 1.01-2.13). Furthermore, Asians (OR 0.73; 95% CI 0.57-0.93) and Hispanics (OR 0.58; 95% CI 034-0.99) were less likely to have asthenospermia. Asians (OR 0.73; 95% CI 0.57-0.94) and Indo-Canadians (OR 0.58; 95% CI 0.35-0.99) were less likely to have teratozospermia. No differences were seen for vitality. No differences were seen for FSH levels, however, Asians (p<0.01) and Indo-Canadians (p<0.01) were more likely to have lower testosterone.”

It’s always the damn Asians.
Magic Dirt won’t fix your shitty sperm.

Maybe if we spend more on the NHS! The evolution fairy may visit!

The lower sexual dimorphism of Asians makes them functionally partially infertile. This is why they marry so young (it isn’t traditionalism) and despite this, have a low birth count per person, and are the most populous race on Earth. They’re actually the most r-selected, Mother Nature holds them back from fertilization with mutations. Along with r-selection, more total fertility issues in the male/offspring (azoospermia, infant death), lower volume AND lower testosterone, it all fits!

Is that my fault? No. Stop blaming me for reading. I’m not, in fact, God.

Hey, we have our own group with shitty sperm. Theirs is just bigger and more characteristic of the whole.

from https://www.ncbi.nlm.nih.gov/pubmed/26962784

“AR-CAG repeat length was longer in infertile men in Asian, Caucasian, and mixed races (SMD = 0.25, 95% CI: 0.08-0.43, P <0.01; SMD = 0.13, 95% CI: 0.02-0.25, P <0.05; SMD = 0.39, 95% CI: 0.15-0.63, P <0.01).

Notice p-value difference is so loose for white it doesn’t meet the medical standard? 0.05 is too high. Absurdly.

The overall study shows that increased AR-CAG repeat length was associated with male infertility. The subgroup study on races shows that increased AR-CAG repeat length was associated with male infertility in Asian, Caucasian, and mixed races. Increased AR-CAG repeat length was also associated with azoospermia. This meta-analysis supports that increased androgen receptor CAG length is capable of causing male infertility susceptibility.”

In the interest of intellectual honesty.

WHR

We literally have the studies. e.g. It’s metabolic.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4306982/

“Sixty-four PCOS patients and 40 women served as the control group were studied. The two groups were subdivided according to the body mass index (BMI) into two obese and non-obese groups. Waist:hip ratio (WHR), plasma epinephrine level was estimated, sympathetic skin response (SSR); postural orthostatic tachycardia syndrome, heart rate variability (HRV), and valsalva ratio were measured in both groups.”
“Compared to the control group, obese PCOS patients demonstrated higher BMI and WHR, reduced palmar SSR latency and higher amplitude, altered HRV, higher plasma epinephrine level, and rapid pulse rate. Moreover, non-obese patients show reduced palmar SSR latency and higher amplitude, higher plasma epinephrine level, and higher pulse rate. BMI and WHR of the patients were positively correlated with plasma epinephrine level; while the HRV was negatively correlated WHR.”
“The BMI and WHR were significantly higher in the PCOS patients compared to the control group 36.63±4.23 kg/m2 vs. 34.14±3.39 kg/m2 (p=0.041) and 0.88±0.05 compared to 0.79±0.11 (p=0.001), respectively.”

“We demonstrated high plasma epinephrine level during lying and standing positions in PCOS patients. This could be of obesogenic origin as we noticed a positive correlation between plasma epinephrine level and both of BMI and WHR. PCOS patients of this study exhibited central abdominal obesity and the mechanisms by which central obesity drive an increase in sympathetic activity are not entirely clear. Yet, the fat cells have increased sensitivity to lipolytic agents and/or the factors inducing fat mobilization are turned on (16). This was further supported that adipocytes isolated from the visceral fat depot of women with PCOS had increased catecholamine-stimulated lipolysis (17).”

Nice boy hips. Don’t try for kids. (Goes for all races, Spartans forced girls to be lightly athletic to be ready for childbirth as a woman, that broadens hips beyond racial average).
And when the NHS totally fails, picture the fatal correction to reality when these women expect childbirth interventions. No waist? No taste.

Old expression.

It’s genetic. They’re gonna get fat – or the kids will. We’ve all seen them. I’m just saying, the signs were there. Choosing a woman with a shit tier WHR is like electing for a manlet over the average height. It could rarely work out for health, but rarely. Don’t get angry at me.

https://www.europeanreview.org/wp/wp-content/uploads/4755-4761-Metabolic-parameters-in-PCOS-and-abdominal-obesity.pdf

“RESULTS: Women with WHR ≥0.8 had higher concentration of glucose and insulin (both fasting and after 120 min of oral administration of 75 g glucose), as well as HOMA-IR value, than women with WHR value < 0.8. Also, abdominal obesity disorders hormonal parameters. Higher free androgen index and lower concentration of sex hormone binding globulin and dehydroepiandrosterone sulfate were found in female with WHR ≥ 0.8.

There’ll still be guys like “WHR doesn’t matter, medically”.

Muh dudebros going, “at least they’re skinny”. But they’re not?

“Women with WHR ≥0.8 had… abdominal obesity disorders hormonal parameters.”

They’re literally not. Chemically. You can biopsy the tissue and test it.

the fat cells have increased sensitivity to lipolytic agents and/or the factors inducing fat mobilization are turned on”

My feels have zero to do with that, dude. It’s genes?

NOBODY is jealous. You keep your secret fatty.

I implore you to marry the future whale and learn the hard way. They’re a puffer-fish.

Whatever their race. But the shorter they are, the worse it is. Short women should have an even SMALLER waist, since it’s skeletal. My own is far smaller than most Asians, for instance, despite being taller than most of them as white. If you want to piss them off, say (honestly) that men like small waists. Just generally. Gets them every time, although most people wouldn’t say they had a large one (not really looking and they don’t dress for it). They know they’re broad and they hate women who dress to show any different, including lucky exceptions in their own race, since it’s a countersignal. Namely: I can afford to have a smaller midsection, less running and foraging is required.

[If I want to dress to piss off a group of women, bodycon but for the waist only. It’s subtle and you’d imagine as a man they would neither notice nor care. Great way to tell a woman’s natural WHR – do they like bodycon? It needn’t be tight on T&A, actually that’s better, it’s actually about waist fit. Pill women also get larger round the middle, any weight gain is there and ruins WHR so it’s visual slut shaming too. Love it.]

Follicular stimulating hormone, luteinizing hormone, androstenedione, and 17-beta-estradiol, were on similar level in both groups. Elevation in triglycerides, total cholesterol, and low-density lipoprotein levels, as well as decrease in high density lipoprotein level in serum of women with WHR value ≥0.8, were found when compared to women with WHR < 0.8. A statistically significant correlation was found between WHR value and glucose, insulin, sex hormone binding globulin, free androgen index and lipid profile parameters.”

Hips don’t lie because biochemistry.

“CONCLUSIONS: Abdominal obesity causes additional disorders in metabolic and hormonal parameters in PCOS women, which confirmed changes in analyzed parameters between PCOS women with WHR < 0.8 and WHR ≥ 0.8 and statistically significant correlations between WHR value and analyzed parameters.”

ADD meds lower testosterone

Wait, people didn’t know this?

Fake diagnosis, real hormonal nuke.

Amphetamines (and any other drugs that block dopamine indirectly) cause prolactin (yes, the milk hormone) levels to rise. Higher prolactin levels cause a reduced libido (your body* thinks it’s pregnant or postpartum) and tank your testosterone levels.

*Men are mutated women so yes this applies to you. Biology, bitch. If the template for human weren’t female (at least one X), we couldn’t give birth.

Why else do you think they wanna foist those drugs onto little boys?

Why do you think there’s a random surge in boys actually believing their girls, because the parents cleared use of amphetamines? We literally give kids amphetamines. We live in a society. This permanently alters their brain development, irreversible in adulthood.

They also want to ruin marriage.

https://www.adhdmarriage.com/content/low-testosterone-adhd-and-adderall

Enough foreplay.

Onto the SCIENCE!

https://www.ncbi.nlm.nih.gov/pubmed/6642414

“These results indicate that Amph is a poor PRL suppressor in either normo- or hyperprolactinemic subjects. It is proposed that this may be due to the drug’s ability to effect release of dopamine mainly from a non-granular pool of the amine.”

https://en.wikipedia.org/wiki/Prolactin_release-inhibiting_factor

“The prolactin cells that produce prolactin, in the absence of dopamine, secrete prolactin continuously;

dopamine inhibits this secretion. In the context of regulating prolactin secretion, dopamine is occasionally called prolactin-inhibiting factor, prolactin-inhibiting hormone, or prolactostatin.”

Of course you won’t find it online until some MGTOW blog rips me off. Again.

It isn’t explained this way, the real way, in textbooks. So I know.

I see you.

inb4

https://www.rxlist.com/adderall-drug.htm

Adderall® tablets contain d-amphetamine and l-amphetamine salts in the ratio of 3:1.”

Cause of the gamma plague?

Mixed women just look higher sexed

worlds away from considerations of beauty, which, for women, involve a narrower than group average face – NOT broader, which is high T aka libido
https://www.forbes.com/sites/zhanavrangalova/2017/09/27/what-the-shape-of-your-face-says-about-your-sex-drive/#1efda635564f

Can we call this r-face?

R-types would logically have the sluttier face.

Past research has consistently found that men with shorter and wider faces are more aggressive, more prejudiced, more likely to deceive others, more dominant, and more driven to succeed compared to men with longer and narrower faces.

Low trust, sluttier, violent, compulsive lying – r-types in a nutshell.

This is even true for non-human primates, and among women this link is found for dominance, but not aggression. Furthermore, other people pick up on these facial cues, perceiving wide-faced men and women as more masculine, dominant and threatening.

R-selected women, more aggressive as sole protectors of their spawn.

It all FITS.

Why would all these personality traits be related to this seemingly random facial feature, also known as facial width-to-height ratio (FWHR)? Because they are all thought to be influenced, at least partly, by exposure to the masculinizing effects of testosterone. Although data are mixed as to the exact timing of this exposure (in utero, at puberty or in adulthood), more testosterone generally means higher behavioral masculinity, dominance, aggression and also a higher facial width-to-height ratio.

(polite coughing)

Examples of measurement of the FWHR in faces with relatively low and high FWHRs.

 FROM GENIOLE ET AL., 2015, PLOS ONE

Find the more attractive woman, I fucking dare you.

Also applies to retro women:

A long face is more attractive in women (read: feminine).
As history shows, feminine women like Vivien Leigh can have low testosterone but a fiiiiine sex life.
[despite gaslighting from a gay bitch of a husband]

https://www.sciencedaily.com/releases/2014/04/140423101718.htm

Feminine face = better WHR measures, so best of both worlds, actually!

Wide hips – higher sex drive, partnered or not.

Long face in women is not horse face.

Except SJP but she isn’t white so… who cares?

Now, a set of two new Canadian studies find that FHWR is also linked to several aspects of people’s sexual psychology (sex drive, casual sex and infidelity), also known to be influenced by testosterone.

Water is wet, go on.

In the first study, 145 heterosexual Canadian students (52% female, 82% White, mean age = 22) currently in romantic relationships answered questions regarding their sex drive (like how often they experience sexual desire or how often they masturbate). They then had their face photos taken by the researchers (under identical distance, lighting, and backdrop conditions for all participants), and those photos were later carefully measured by two raters for width, height and some other facial features like cheekbone prominence.

Note: libido doesn’t equal promiscuity in women, which warrants separate study so hot does not equal slut. Discernment among the most attractive women in studies prove this (the sluttiest women are average or slightly above to hook more attractive mates). Yet it does explain partially why men would find certain women better options sexually if not marriage material (sexy over beauty). There’s a mutual exclusive at play here between availability (broad, manjaw) and beauty (dimorphic female, narrow face and soft jaw).

Because their average genes want to survive with anyone v no one.

And naturally propaganda and rhetoric factor below awareness.

This must change based on r/K cycling, what percentage of men find mannish women ‘hot’ rather than meh or repulsive (probably charts onto the man’s wing leaning too).

Could she be a porn star and how good (you imagine) at it? That’s sexy in our gross pornified culture and a 10 for Hotness.

Could she be portrayed as an ancient goddess in a sculpture? That’s beauty and a 10.

Beautiful, but by modern standards (which are dumb and/or anti-white, generally) – not hot.

sorry about the meme but you get the point

There’s a distinct look, defined or softer.

Like popular celebrities

Beautiful face + voluptuous body = Ideal.

By comparison, note how the limbs look thin? Odd side effect.

Shows natural fat % (so limbs must be less to be within range) but not obesity (female fat deposition, see WHR posts).

Rarely…. a woman is both.

QE-fightme-D:

Fuck you, Monica. Fuck you and your flawlessness.

The analyses confirmed the researchers’ hypothesis: Wider-faced men and women reported higher sex drive than their counterparts with narrower faces. This link remained even when other facial features were statistically controlled for.

A second sample of 314 students (57% female, 91% White, 93% heterosexual, mean age = 20) from a different Canadian university confirmed these findings regarding sex drive. This second study also asked participants about their desire for, attitudes towards and experiences with casual hookups and their expectations of future infidelity to a partner. The men with higher facial width-to-height ratios reported greater openness to hookups and a higher likelihood they’d be unfaithful to a partner compared to their narrow-faced peers. A link between FWHR and propensity for casual sex or infidelity was not found for the women in this sample.

potential

but libido finding is solid

Together with prior research on this facial feature, this study suggests that the facial width-to-height ratio, and our ability to perceive and interpret it, may be part of an evolved system that emerged long ago in our evolutionary past in order to help us figure out who may be dangerous vs. safe, trustworthy and faithful vs. likely to deceive or cheat on us, or more likely to stick around and raise babies vs. “hit it and quit it.”

Maybe r-oriented men don’t care if the woman is available to other men because, in times of r, any port in a storm, right?

R-selected men = low investment, might as well be low EFFORT.

Sorry but…. true, right?

Whites on average have the narrowest faces plus highest trust societies.

Japs are the closest non-whites. Says it all.

This is the first study to ever link FWHR to an aspect of human sexual psychology, so the findings need to be replicated in other samples and more diverse samples, including teens and older adults, non-whites, nonheterosexual and trans populations and using additional measures of sexual psychology before we can accept them.

Those studies can’t be published, you’ll kill them.

They’re in a drawer somewhere. PC people, you play yourself.

We should also remember that even if there is some positive correlation between FWHR and these personality and sexual traits, that this correlation is far from perfect. Not every person with a wide, short face is likely to be violent, cheat, hook up or crave sex several times a day. Being aware of our perceptual biases is an important first step in correcting our automatic tendency to stereotype someone based on the shape of their face.

Why did Marilyn make her face look broader with hair?

Estrogen and lifelong brain health, testosterone fraud

https://www.ncbi.nlm.nih.gov/pubmed/26109339

Estrogen facilitates higher cognitive functions by exerting effects on brain regions such as the prefrontal cortex and hippocampus. Estrogen induces spinogenesis and synaptogenesis in these two brain regions and also initiates a complex set of signal transduction pathways via estrogen receptors (ERs). Along with the classical genomic effects mediated by activation of ER α and ER β, there are membrane-bound ER α, ER β, and G protein-coupled estrogen receptor 1 (GPER1) that can mediate rapid nongenomic effects. All key ERs present throughout the body are also present in synapses of the hippocampus and prefrontal cortex. This review summarizes estrogen actions in the brain from the standpoint of their effects on synapse structure and function, noting also the synergistic role of progesterone. We first begin with a review of ER subtypes in the brain and how their abundance and distributions are altered with aging and estrogen loss (e.g., ovariectomy or menopause) in the rodent, monkey, and human brain. As there is much evidence that estrogen loss induced by menopause can exacerbate the effects of aging on cognitive functions, we then review the clinical trials of hormone replacement therapies and their effectiveness on cognitive symptoms experienced by women. Finally, we summarize studies carried out in nonhuman primate models of age- and menopause-related cognitive decline that are highly relevant for developing effective interventions for menopausal women. Together, we highlight a new understanding of how estrogen affects higher cognitive functions and synaptic health that go well beyond its effects on reproduction.

Men dosing testosterone are called meatheads for a reason, they would logically throw off their other hormones and functionally retard themselves. Because this ONE time, you can trust the pill people. No IQ studies in testosterone supplementing men, I guess there’s a good reason. They just forgot?

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4330791/

The results are, unfortunately, controversial and puzzling. Dosing, timing, even the application route seem to considerably affect the outcomes. 

You’re not trying to rig it at all, huh?

Reduction to dihydrotestosterone by 5-alpha reductase increases the androgen activity; conversion to estradiol by aromatase converts the androgen to estrogen activity.

Holy shit.

That’s hilarious.

Recently, the non-genomic effects of testosterone on behavior bypassing the nuclear receptors have attracted the interest of researchers. This review tries to summarize the current understanding of the complexity of the effects of testosterone on brain with special focus on their role in the known sex differences.

Not known.

a very important study in rhesus monkeys showed that pharmacological castration reduced and testosterone supplementation normalized anxiety levels (Suarez-Jimenez et al., ).

on the other hand, in both men and women, testosterone supplementation leads to improvement of depressive symptoms (Pope et al., ; Miller et al., ). However, not all interventional studies confirmed the anti-depressant effect of testosterone. At least in one published randomized controlled trial, the effects of testosterone were comparable to placebo effects (Seidman et al., ). Similarly, not all observational studies show a consistent picture. At least in one small study, depressive women had higher testosterone (Weber et al., ).

Another experiment on intact rats revealed that the effect of testosterone on depression is dose-dependent (Buddenberg et al., ).

Over the counter won’t work.

During the productive ages and even in early adulthood, men generally outperform women in spatial abilities (Linn and Petersen, )

Even for a few years you’re in college? Is that it?

Spatial thing is probably due to error, thus would be discounted under a valid method.

Error rate as well as the reaction time negatively correlated with testosterone (Hooven et al., ).

In another study, actual testosterone was not associated with spatial abilities, but prenatal testosterone correlated positively with spatial abilities in women (Kempel et al., ).

Congratulations, you’re on the female level. Apply your non-toxic internet cream.

In line with these findings is the lack of an association between actual salivary testosterone levels and mental rotation in men and women (Puts et al., ).

Actual science, no replication issue.

However, in a large observational study analyzing spatial abilities in adult men from various age categories, low testosterone was associated with better spatial visualization (Yonker et al., ).

Actual science, no replication issue.

Plus multiple ages in ADULT men, important.

Good work.

In a very interesting study, it was found that in men, the pubertal concentrations of testosterone are negatively associated with mental rotation in the adulthood (Vuoksimaa et al., ). In the same paper, the comparison of twins is reported. The twin with higher testosterone scored worse in the mental rotation tests. The results are contradictory, but may depend on the test used for the assessment of spatial abilities.

Counting fluke correct answers and not errors to force a finding is scientism. Bad method.

When virtual Morris water maze was used, a positive correlation between testosterone and spatial navigation was found in women, but not in men (Burkitt et al., ). The size of the corpus callosum seems to add complexity in the relationship between spatial abilities and testosterone (Karadi et al., ). This might be one of the causes for negative findings in studies where some of the determinants are missing (Kubranska et al., ). Another cause is likely the selection of the tested population. In gifted children, a negative correlation between salivary testosterone and spatial abilities was found (Ostatnikova et al., ).

FFS.

Negative findings are real science.

And that’s important.

In Chinese men, the accuracy in mental rotation tests was comparable to Americans, but the reaction times were longer indicating that cultural differences could add to the variability of published results (Yang et al., ).

No they didn’t rush the test out of boredom. Lower error rate, I’d bet.

Last but not least, genetic factors likely modulate the effect of testosterone. We have previously shown that at least in gifted boys, genetic polymorphisms influencing testosterone metabolism affect also its relationship to mental rotation (Celec et al., ). Especially, the CAG short tandem repeat in the exon 1 of the androgen receptor gene seems to be important for the action of testosterone and its metabolites (Nowak et al., ). Despite all complexity, the current picture indicates that the association between testosterone and spatial abilities is curvilinear and sex-dependent.

aka more is NOT better and it’s genetic, morons

In women higher testosterone is associated with better mental rotation, in men lower testosterone is associated with better spatial abilities. This seems to be true both for actual testosterone (Moffat and Hampson, ) and for prenatal testosterone (Grimshaw et al., ). Supplementation of testosterone in older men results in improvement of spatial abilities, but it is accompanied with changes in estradiol metabolism and it is likely that this interferes with modifications of spatial abilities (Janowsky et al., ).

They only studied spatial, not global, I checked.

Even in rats, testosterone administration affects the strategy of the animals in spatial tasks (Spritzer et al., ). However, the interaction between testosterone and mental rotation tests is bidirectional. It has been shown that mental rotation testing affects testosterone, at least in women (Durdiakova et al., ).

Doing smart things causes the brain to…. adapt? Really?

Does our pulse increase when we run? Some Sherlock Holmes do a fucking study.

MRI

Moreover, participants with complete androgen insensitivity syndrome presented with female-like neural activation pattern in the parietal lobe, indicating that gonadal hormone exposure rather than genetic sex itself plays role in brain functions (Van Hemmen et al., ).

Supplementing won’t work, you’re worse than the trannies. They don’t claim brain benefits.

The menstrual cycle and thus the involvement of sex hormones, including testosterone, in spatial abilities was further confirmed by Pletzer et al. In their study, error rates linked with deactivation of inferior parietal lobes and prefrontal lobes were higher during luteal phase for verbal tasks, while in the follicular phase, spatial abilities in females were confirmed (Pletzer et al., ).

One of the major factors that might explain the differences between the results of various studies is the variability of the examined populations. As mentioned above, the cultural differences, sex and age have all been shown to impact the physiological effects of testosterone. 

Duh.

Standardization in this area would surely improve our understanding of the neuroendocrinology of testosterone. More systematic research using the whole spectrum of available tools and looking at the various physiological aspects is needed. However, to be able to publish such research, journals should accept manuscripts based on the design and not on the results. Otherwise, the publication bias that is obvious in the so far published literature will continue to be a big issue. Many researchers in this field complain about negative results that are very difficult to publish in the relevant journals. The number of such unpublished observations and experiments is unknown. But based on our humble experience, the negative results will probably be more common than the published positive ones. And if the contradictory published findings are added, the picture gets even more confusing. Large systematic research projects with more cooperation between the most productive research teams is definitely needed.

You can’t because the low IQ men will complain if you publish negative findings.

e.g.

https://ohsu.pure.elsevier.com/en/publications/testosterone-influences-spatial-cognition-in-older-men-2

Testosterone plays a role in the organization of behavior during development. The authors examined whether testosterone could play a maintenance role in behavior as well. In a double-blind manner, verbal and visual memory, spatial cognition, motor speed, cognitive flexibility, and mood in a group of healthy older men who were supplemented for 3 months with testosterone were assessed. The increase in testosterone levels to 150% of baseline levels resulted in a significant enhancement of spatial cognition, but no change in any other cognitive domain was found.

You’ll be slightly more able to parallel park. Like when you were young.

Louder for the slow:

no change in any other

cognitive domain was found

NO IMPROVEMENT IN MEMORY

NO IMPROVEMENT IN MOTOR SPEED

NO IMPROVEMENT IN COGNITIVE FLEXIBILITY

NO IMPROVEMENT IN MOOD.

Did I stutter?

Testosterone supplementation influenced the endogenous production of estradiol, and estradiol was found to have an inverse relationship to spatial cognitive performance. These results suggest that testosterone supplementation can modify spatial cognition in older men; however, it is likely that this occurs through testosterone’s influence on estrogen.

I’m sure the health effects will be golden.

Waist-Hip Ratio and female beauty

The sexual dimorphism for this metric is obviously lowest on Asians.

https://www.ncbi.nlm.nih.gov/pubmed/8366421

Evidence is presented showing that body fat distribution as measured by waist-to-hip ratio (WHR) is correlated with youthfulness, reproductive endocrinologic status, and long-term health risk in women. Three studies show that men judge women with low WHR as attractive. Study 1 documents that minor changes in WHRs of Miss America winners and Playboy playmates have occurred over the past 30-60 years. Study 2 shows that college-age men find female figures with low WHR more attractive, healthier, and of greater reproductive value than figures with a higher WHR. In Study 3, 25- to 85-year-old men were found to prefer female figures with lower WHR and assign them higher ratings of attractiveness and reproductive potential. It is suggested that WHR represents an important bodily feature associated with physical attractiveness as well as with health and reproductive potential. A hypothesis is proposed to explain how WHR influences female attractiveness and its role in mate selection.

Hello sexual selection, tied intimately to natural selection.

PDF here: https://pdfs.semanticscholar.org/05d6/0e201efb208e8561641d13df30fc6ba3bc1a.pdf

also connected to “desire and capability for having childrenp7 or 299.

so K-type women may have better WHR.

Normal weight women have the most positive attributes associated.

Overweight category was universally unattractive.
It’d be nice to see a male study on this. I think Western women would want more children if fewer men were obese.

Why Asians are considered youthful but not sexy (they’d usually fall in the underweight group):

The variables of attractiveness, sexiness, and good health were located close to each other, suggesting that subjects perceived them to be closely related.

Attributes of desire and capability for having children were located close to each other in the solution space but farther from attractiveness, sexiness, and good health, implying that subjects did not perceive a great similarity between these two sets of attributes.

Finally, the attribute of youthfulness was located alone and away from both sets of other attributes. Thus, subjects apparently did not perceive youthfulness to be related to any other measured attributes of good health, sexiness, attractiveness, and desire and capability for having children.

So there’s that. Nobody’s jealous.

Figure N7 was located closer to attractiveness, sexiness, and good health as well as desire and capability for having children than any other Figure.

Normal weight for frame (and race) + most nubile WHR would make sense.
More of those genes survived.

Figure N9 was located closest to desire and capability for having children, whereas Figure N8 was located between Figure N7 and Figure N9. The figure N10 was grouped along with overweight figures, which were not perceived to be closely associated with any of the attributes under investigation. Underweight female figures, U7 and U8, were associated only with youthfulness. However, underweight figures with high WHR (U9 and U10) were perceived as neither youthful nor healthy, in spite of the fact that their depicted body weight was quite similar to figures with lower WHR.

Women with an atrocious WHR (boy hips, no waist) and under or overweight for their skeleton are objectively unattractive from an evolutionary standpoint. This would apply whether it’s a Jap, a Ruskie or an American.

Stop calling sexy science ‘racist’ because it doesn’t share your fetish.

This chart drags you harder than I ever could.

Your Asian girlfriend with the boy hips is approximately as attractive to the world as the average WHR white fat chick. That’s your level, accept it.

It’s also a fact we cannot accurately perceive attractiveness of the racial outgroup as well as our own, so an awareness of ingroup flaws changes nothing.

Most modern women straight up don’t look healthy, whether they’re American, European or, yes, Asian.

Stop trying to make boy hips = sexy happen. It’s not going to happen.

Look at the damn gradient on that underweight thing. The solution to fat women isn’t anorexia. That also suggests bad genes. In fact, at least the fat percentage on slightly overweight 0.7 WHR women suggests femininity and fertility.

“Overall, it seems that subjects inferred reproductive capability from body fat”

What does a foetus feed from?

“Thus, it seems that although WHR is related to health and attractiveness, body weight is perceived to be related to reproductive capability”

Obviously.

“As a group, underweight figures were assigned the lowest reproductive capability, followed by overweight figures and then normal weight figures.”

Suck on that, soyboys.

You actually tend to downgrade. That’s why the Democrat-voting soyboys all want an Asian girlfriend and expressly don’t want kids with it.

“Overall, it appears that both fatness and thinness are perceived as unattractive, and such figures are not perceived as having especially high reproductive potential. “

Not womanly. Remember that word? This:

Not girly, not sexy, not cute, not hot. Womanly.

You can’t discuss women in a reproductive, evolutionary context without it.

Thus, consistent with the present findings, men did not find thin or underweight figures attractive.

If you only care for other male opinions.

There is some evidence that suggests that being extremely underweight or overweight can have adverse effects on female reproductive functions.

Ya don’t say?

A critical body mass has been shown to be significantly related to the onset of menstrual cycle and its maintenance (Frisch & McCarthur, 1974), although recent evidence (DeRidder et al., 1990) suggests that it is the body fat distribution, rather than body fat mass or body weight, that is related to early pubertal development.

Distribution varies by race.

Africans are the most pronounced in women then Europeans shapely but delicate then Asians last – no shape, very yang flesh (broad but flat or full in the middle like cortisol fat) and almost nothing to distribute.

Am I imagining all of this?

Underweight females (15% below ideal body weight) have been reported to have a higher incidence of oligomenorrhea (menses 35 days or more apart) and amenorrhea and to have a higher prevalence of ovu-latory infertility than normal weight females (Green, Weiss, & Daling, 1986).

Underweight women also give birth to infants who are small and growth delayed, and such infants often have permanently impaired intellectual and physical development (Supy, Steer, McCusker, Steele, & Jacobs, 1988).

Menstrual dysfunction and ovulatory infertility also occur more often in females who are 20% above ideal body weight (Green et al., 1986). Morbid obesity in females with high WHR has been shown to increase the degree of androgenicity (increased percentage of free testosterone) and associated menstrual and ovulatory problems (Kirschner & Samojilik, 1991). Thus, the reproductive success of a woman may be low in spite of a high level of fat deposits if the regional distribution of fat is not appropriate, that is, gynoid.

=Womanly.

Finally, the finding that underweight figures were assigned high rankings for youthfulness but not for attractiveness (or other attributes related to reproductive potential) is difficult to reconcile with some evolutionarily based mate selection hypotheses.

Normal men aren’t pedos.

Youthfulness and health have been proprosed as absolute criteria for female attractiveness (Symons, 1987).

Stick with health.

Health has good or bad, you have no negative way to assess youth e.g. immature.

Features of physical appearance associated with youth supposedly provide the strongest and most reliable cues for female reproductive potential. The present finding illustrates that the relationship of youthfulness and attractiveness is quite complex.

Not really.

A woman who is judged to be attractive is also found to be youthful; however, youthfulness alone does not make a woman attractive. Apparently, youthfulness is a necessary, but not a sufficient condition, for determination of female physical attractiveness.

crazed pointing-

also, don’t try to chalk this up to taste:

“Furthermore, if the ideal of female attractiveness is arbitrary and ever changing, no evidence of transgenerational stability in the meaning of WHR should be found, as older men are more likely to be exposed to different ideals of attractiveness than are younger men.”

but

“Older men did not associate health with underweight figures, including those with lower WHR.”

TLDR: Pedos are wrong. Underweight, waistless wonders are not attractive.

Study 2, rubbing salt in that fact.

https://journals.plos.org/plosone/article?id=10.1371/journal.pone.0009042

Optimal Waist-to-Hip Ratios in Women Activate Neural Reward Centers in Men

Secondary sexual characteristics convey information about reproductive potential. In the same way that facial symmetry and masculinity, and shoulder-to-hip ratio convey information about reproductive/genetic quality in males, waist-to-hip-ratio (WHR) is a phenotypic cue to fertility, fecundity, neurodevelopmental resources in offspring, and overall health, and is indicative of “good genes” in women. Here, using fMRI, we found that males show activation in brain reward centers in response to naked female bodies when surgically altered to express an optimal (∼0.7) WHR with redistributed body fat, but relatively unaffected body mass index (BMI). Relative to presurgical bodies, brain activation to postsurgical bodies was observed in bilateral orbital frontal cortex. While changes in BMI only revealed activation in visual brain substrates, changes in WHR revealed activation in the anterior cingulate cortex, an area associated with reward processing and decision-making. When regressing ratings of attractiveness on brain activation, we observed activation in forebrain substrates, notably the nucleus accumbens, a forebrain nucleus highly involved in reward processes.

These findings suggest that an hourglass figure (i.e., an optimal WHR) activates brain centers that drive appetitive sociality/attention toward females that represent the highest-quality reproductive partners. This is the first description of a neural correlate implicating WHR as a putative honest biological signal of female reproductive viability and its effects on men’s neurological processing.

Quality.

Study 3

https://www.sciencedirect.com/science/article/pii/S0018506X08002298

Men report stronger attraction to femininity in women’s faces when their testosterone levels are high

Many studies have shown that women’s judgments of men’s attractiveness are affected by changes in levels of sex hormones. However, no studies have tested for associations between changes in levels of sex hormones and men’s judgments of women’s attractiveness. To investigate this issue, we compared men’s attractiveness judgments of feminized and masculinized women’s and men’s faces in test sessions where salivary testosterone was high and test sessions where salivary testosterone was relatively low.

This is why we need studies on men too.

Men reported stronger attraction to femininity in women’s faces in test sessions where salivary testosterone was high than in test sessions where salivary testosterone was low. This effect was found to be specific to judgments of opposite-sex faces. The strength of men’s reported attraction to femininity in men’s faces did not differ between high and low testosterone test sessions, suggesting that the effect of testosterone that we observed for judgments of women’s faces was not due to a general response bias. Collectively, these findings suggest that changes in testosterone levels contribute to the strength of men’s reported attraction to femininity in women’s faces and complement previous findings showing that testosterone modulates men’s interest in sexual stimuli.

Study 4

https://www.sciencedirect.com/science/article/pii/S0191886906004995

Beauty is in the eye of the plastic surgeon: Waist–hip ratio (WHR) and women’s attractiveness

Attractiveness conveys reliable information about a woman’s age, health, and fertility. Body fat distribution, as measured by waist-to-hip ratio (WHR), is a reliable cue to a woman’s age, health, and fertility, and affects judgment of women’s attractiveness. WHR is positively correlated with overall body weight or body mass index (BMI). Some researchers have argued that BMI, rather than WHR, affects judgments of female attractiveness. To evaluate the role of WHR, independent of BMI, we secured photographs of pre- and post-operative women who have undergone micro-fat grafting surgery. In this surgery, surgeons harvest fat tissue from the waist region and implant it on the buttocks. Post-operatively, all women have a lower WHR but some gain weight whereas others lose body weight. Results indicate that participants judge post-operative photographs as more attractive than pre-operative photographs, independent of post-operative changes in body weight or BMI. These results indicate that WHR is a key feature of women’s attractiveness.

Duh.

Let’s look historically. Study 5

https://academic.oup.com/ajcn/article/72/6/1436/4729453

Trends in waist-to-hip ratio and its determinants in adults in Finland from 1987 to 1997

Background: Although abdominal obesity has been shown to be an important risk factor for cardiovascular disease and a variety of other diseases, secular changes in fat distribution in populations have rarely been documented.

Objective: Our objective was to assess trends in waist-to-hip ratio (WHR) in the Finnish population during a 10-y period. In addition, we investigated the associations of WHR with body mass index (BMI), age, education, and lifestyle factors.

Design: Three independent cross-sectional surveys were carried out at 5-y intervals between 1987 and 1997. Altogether, 15096 randomly selected men and women aged 25–64 y participated in these surveys.

Results: The WHR increased in both men and women during the 10-y period (P< 0.0001). In men, the strongest upward trend took place in the first 5-y period and then seemed to plateau; in women, the WHR continued to increase into the 1990s. In both sexes, the most prominent increase was observed in subjects aged ≥45 y. The WHR increased in all education-level groups, the lowest WHR being among those with the highest education. Age (18% in men, 12% in women) and BMI (33% in men, 25% in women) accounted for most of the variation in WHR, whereas only 3% was explained by education and lifestyle factors.

Conclusions: Abdominal obesity is a growing problem in Finland, especially in persons aged ≥45 y. These adverse changes in body shape continued to take place, particularly in women, in the 1990s.

Something in the food?

More history, prehistoric. Study 6

https://journals.plos.org/plosone/article?id=10.1371/journal.pone.0123284

Preferred Women’s Waist-to-Hip Ratio Variation over the Last 2,500 Years

The ratio between the body circumference at the waist and the hips (or WHR) is a secondary sexual trait that is unique to humans and is well known to influence men’s mate preferences. Because a woman’s WHR also provides information about her age, health and fertility, men’s preference concerning this physical feature may possibly be a cognitive adaptation selected in the human lineage. However, it is unclear whether the preferred WHR in western countries reflects a universal ideal, as geographic variation in non-western areas has been found, and discordances about its temporal consistency remain in the literature. We analyzed the WHR of women considered as ideally beautiful who were depicted in western artworks from 500 BCE to the present. These vestiges of the past feminine ideal were then compared to more recent symbols of beauty: Playboy models and winners of several Miss pageants from 1920 to 2014. We found that the ideal WHR has changed over time in western societies: it was constant during almost a millennium in antiquity (from 500 BCE to 400 CE) and has decreased from the 15th century to the present. Then, based on Playboy models and Miss pageants winners, this decrease appears to slow down or even reverse during the second half of the 20th century. The universality of an ideal WHR is thus challenged, and historical changes in western societies could have caused these variations in men’s preferences. The potential adaptive explanations for these results are discussed.

Should’ve controlled for race.

Why not look at male WHR? Plus sperm health? Found:

https://www.drelist.com/blog/bmi-waist-circumference-semen-quality/

  • The volume of ejaculate decreases in a linear fashion with increasing BMI (suggesting an inverse relationship).
  • The sperm quality and viability declines with increasing waist circumference.
  • Investigators also discovered that quality of semen decreases (such as sperm viability, motility, semen volume) with increasing body size; however, no relationship was observed between sperm DNA fragmentation index and physical activity or obesity.

Latter requires time.

Various research and clinical studies suggests that subfertility in men is multifactorial i.e. several factors can impact the quality of reproductive health.

  • Abnormal sperm production: Study conducted by Jensen and associates (2) suggested that abnormal BMI is very strongly linked to impaired sperm production. One of the many reasons is, abnormal metabolism of testosterone (which plays a key role in the production of healthy and viable sperms).
  • Abdominal obesity and risk of metabolic disorders: According to a new study reported in the Human Reproduction (3), investigators provided statistical evidence that abnormal BMI and abdominal obesity is very strongly linked to a number of health issues (such as cardiovascular dysfunction, atherosclerosis, type 2 diabetes, hypertension and others). Needless to say that these health issues have a deleterious effect on the sexual health regardless of the body-mass index (or BMI).
  • Obesity, physical activity and testosterone: Testosterone levels tends to decline in males who have a sedentary lifestyle. Various research and clinical studies indicates that aerobic activity or exercise can improve testosterone metabolism in males significantly.

1. Eisenberg, M. L., Kim, S., Chen, Z., Sundaram, R., Schisterman, E. F., & Louis, G. M. B. (2014). The relationship between male BMI and waist circumference on semen quality: data from the LIFE study. Human Reproduction, 29(2), 193-200.

2. Jensen, T. K., Andersson, A. M., Jørgensen, N., Andersen, A. G., Carlsen, E., & Skakkebæk, N. E. (2004). Body mass index in relation to semen quality and reproductive hormones among 1,558 Danish men. Fertility and sterility, 82(4), 863-870.

3. Hammiche, F., Laven, J. S., Twigt, J. M., Boellaard, W. P., Steegers, E. A., & Steegers-Theunissen, R. P. (2012). Body mass index and central adiposity are associated with sperm quality in men of subfertile couples. Human reproduction, 27(8), 2365-2372.

Yet they don’t tell men this information.

Back to women

https://www.ncbi.nlm.nih.gov/pubmed/24789138

Cross-cultural variation in men’s preference for sexual dimorphism in women’s faces.

Both attractiveness judgements and mate preferences vary considerably cross-culturally.

Racially.

We investigated whether men’s preference for femininity in women’s faces varies between 28 countries with diverse health conditions by analysing responses of 1972 heterosexual participants. Although men in all countries preferred feminized over masculinized female faces, we found substantial differences between countries in the magnitude of men’s preferences. Using an average femininity preference for each country, we found men’s facial femininity preferences correlated positively with the health of the nation, which explained 50.4% of the variation among countries. The weakest preferences for femininity were found in Nepal and strongest in Japan. As high femininity in women is associated with lower success in competition for resources and lower dominance, it is possible that in harsher environments, men prefer cues to resource holding potential over high fecundity.

Asia is weird for dimorphism studies.

Hence the focus on health.

While the economy is bad, it isn’t surprising men prefer manly looking women.

It’s temporary. There’ll be a flood of divorces as the economy improves. Men will suddenly see how mannish the wife has been and be repulsed. Menopause also makes women look more mannish, including higher WHR. So much for a youth argument there.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5177465/pdf/nihms827194.pdf

Factors Underlying the Temporal Increase in Maternal Mortality in the United States

They don’t say more non-white mothers or more mixed race babies, so it’s wrong. They guess.

Right not to see degeneracy

We have one.

Cinemas still have age-restrictions, this is no different.

Sites like google put porn in totally innocent homework searches.

Child porn addicts:
In the UK, PornHub was the 35th most visited website for children ages 6 to 14 in 2013.
Children and Parents: Media Use and Attitudes Report (2014, October) (p. 232).

ofcom.org.uk/binaries/research/media-literacy/media-use-attitudes-14/Childrens_2014_Report.pdf

Dead link now, weird that.

Check yourbrainonporn.com for addiction and neuroscience information.

Child locks on ISPs have always existed, nothing is banned. The UK shouldn’t be producing this stuff. America won’t stop.

What would they say if children were smoking? They used to.

Let’s go back to this bullshit because Sargon doesn’t want to verify his age at the off-license ONE TIME.

You do that every single time you want to drink or smoke but this is clearly a bridge too far for the responsible lefty adults with muh freedoms.

Don’t BOTHER bringing up the fact this policy will cease enabling child addicts.

Don’t even BOTHER. Because you checked, right? You know?

And the clear social message? Yeah, what deterrent? This degeneracy should be underground, I don’t wanna see that. What about my rights? Children shouldn’t be able to view things online they cannot also legally view in person. They can’t go to strip clubs or BDSM shows, they shouldn’t be able to attend virtually.

I love how libertarians assume most people in society are degenerates. No.

Kids are also grooming one another on school wifis and encouraged to groom more kids in a cycle of abuse by adult pedos. It happens.

The latest report:
https://www.ofcom.org.uk/research-and-data/media-literacy-research/childrens/children-and-parents-media-use-and-attitudes-report-2018
They don’t really look into it, weirdly.

“Around one in five 12-15s (22%) who opted to answer the question said they had been contacted
online by someone they didn’t know, and one in ten (9%) said they had seen something of a sexual
nature that made them feel uncomfortable, either online or on their mobile phone.”

Mentally scarring a pre-schooler (other studies asked younger) is nothing to mildly inconveniencing Sargon one bloody time.

If you wonder why soaps and TV are so bad, we removed restrictions on theatre, film and TV for (((no reason))). Covered that UK theatrical statute before, there’s a right not to see this shit. Or at least be warned, which current numerical guidelines do not (I want warning of full nudity and specific acts before paying to see it, as consumer choice).

There’s no such thing as a hooker, Sargon*, that’s what Rotherham social workers say when women and little girls are raped. The psychiatric outcomes of prostitutes have also been studied, there is no excuse for this with Muh Consent because consensus morality doesn’t exist, that’s moral relativist bullshit. Humans don’t have owners, even if you call it a ‘pimp’. Neither is a profession, sex slave owner or sex slave. It’s an affront to human dignity (illegal under the human rights you claim to know and love).

Filming it doesn’t make the situation acceptable, it’s up there with the theatre’s constant abuse of ‘nudity’ scenes**. If adult people wanted to put that adult material online, they’d do it for free! But why do the prostitution/white slavery rings want children to be able to view it?

It’s a good question, innit?

To groom the next crop of recruits. Children have no context to understand, dismiss or deny adult themes. Bobo dolls. They lack the brain development to question what they see, consider it normal and COPY IT.

*(who’s currently triggered at the prospect of protecting children from Silicon Valley pedos and protecting non-addicts by default)

**Apparently public nudity is illegal unless you’re charging and on a stage. WTF. That’s prostitution. This is why we censored productions. It isn’t art. We censor plenty of ‘art’.

other related info copied from

Sources, links and further reading: Rockefeller Foundation and Kinseyhttps://rockfound.rockarch.org/kinsey-reports This Is Why I Quit Porn https://www.youtube.com/watch?v=ljBZ0_iNqrs&feature=youtu.be The Jewish Role in the Porn Industry https://www.youtube.com/watch?v=gwd_Iofr6ZQ Porn Sites Get More Visitors Each Month Than Netflix, Amazon And Twitter Combined (Dec 2017) https://www.huffingtonpost.com/2013/05/03/internet-porn-stats_n_3187682.html Pornhub wants to be your one-stop shop for sex ed (Feb 2017)http://college.usatoday.com/2017/02/02/pornhub-sex-ed/ Internet pornography by the numbers; a significant threat to society https://www.webroot.com/us/en/home/resources/tips/digital-family-life/internet-pornography-by-the-numbers How Does The Porn Industry Actually Make Money Today? AUGUST 11, 2017 https://fightthenewdrug.org/how-does-the-porn-industry-actually-make-money-today/ How the Internet Changed Porn September 12, 2017 https://www.rollingstone.com/culture/features/how-the-internet-changed-porn-w502441 Three in Traci Lords Sex Film Case Indicted, March 06, 1987 :http://articles.latimes.com/1987-03-06/local/me-5028_1_traci-lords Sugar and Spice and all things not so nice Garry Gross, Richard Prince and the story behind the Brooke Shields photograph (Oct. 2009) https://www.theguardian.com/theguardian/2009/oct/03/brooke-shields-nude-child-photograph Manwin Says Its Porn Business Is Legal Mar. 18, 2013 –https://video.foxbusiness.com/v/2235795190001/?#sp=show-clips Manwin takes over Playboy TV operations October 31, 2011 https://web.archive.org/web/20131203003713/http://www.broadbandtvnews.com/2011/10/31/manwin-takes-over-playboy-tv-operations/ 7 of the most famous Jews in porn By Gabe Friedman June 12, 2015 https://www.jta.org/2015/06/12/news-opinion/the-telegraph/7-of-the-most-famous-jews-in-porn Nathan Abrams on Jews in the American porn industry https://www.jewishquarterly.org/issuearchive/articled325.html?articleid=38 Men’s testosterone levels declined in last 20 years https://uk.reuters.com/article/health-testosterone-levels-dc/mens-testosterone-levels-declined-in-last-20-years-idUKKIM16976320061031 A Population-Level Decline in Serum Testosterone Levels in American Men, January 2007 http://jcem.endojournals.org/cgi/content/abstract/92/1/196 Research related to women working in various aspect of the sex industry http://iamatreasure.com/about/stats/Pornography Stats 2013 (PDF) http://blog.clinicalcareconsultants.com/wp-content/uploads/2013/12/porn_stats_2013_covenant_eyes.pdf Sex, Lies And Statistics (Nov 2005) https://www.forbes.com/2005/11/22/internet-pornography-children-cz_sl_1123internet.html#6522094a51ba Want to Stop Sex Trafficking? Look to America’s Porn Addiction https://www.huffingtonpost.com/johnhenry-westen/want-to-stop-sex-traffick_b_6563338.html Why Consuming Porn Is An Escalating Behavior August 2017: https://fightthenewdrug.org/why-consuming-porn-is-an-escalating-behavior/ When a person is sexually aroused, it builds new brain maps for both what they think is sexy and what they expect from their partner.”James G. Pfaus, “Who, What, Where, When (and Maybe Even Why)? How the Experience of Sexual Reward Connects Sexual Desire, Preference, and Performance,” Archives of Sexual Behavior 41 (2012): 31–62. When adolescents, both male and female, are exposed to sexualized media, they are more likely to have stronger notions of women being sex objects. L. Monique Ward and Kimberly Friedman, “Using TV as a Guide: Associations Between Television Viewing and Adolescents’ Sexual Attitudes and Behavior,” Journal of Research on Adolescents 16, no. 1 (2006): 133-156. Endocrine response to masturbation-induced orgasm in healthy men following a 3-week sexual abstinence. https://www.ncbi.nlm.nih.gov/pubmed/11760788 In the UK, PornHub was the 35th most visited website for children ages 6 to 14 in 2013. Children and Parents: Media Use and Attitudes Report (2014, October) (p. 232). http://stakeholders. ofcom.org.uk/binaries/research/media-literacy/media-use- attitudes-14/Childrens_2014_Report.pdf.  Donna M. Hughes, “Sex Trafficking of Women for the Production of Pornography,” Citizens Against Trafficking (July 1, 2010), “If a trafficking victim is forced to engage in a sex act that is filmed or photographed for sale as pornography, then the production of pornography itself becomes a severe form of trafficking in persons that is subject to criminal liability.” Robert W. Peters, Laura J. Lederer, and Shane Kelly, “The Slave and the Porn Star: Sexual Trafficking and Pornography,” The Protection Project: Journal of Human Rights and Civil Society, Issue 5 (Johns Hopkins University: Fall 2012). The Porn Myth: Exposing the Reality Behind the Fantasy of Pornography (March 2017): https://www.amazon.com/Porn-Myth-Exposing-Reality-Pornography/dp/162164006X

Nurture theories clearly demonstrate that porn is propaganda. It changes the otherwise healthy way you view women, family, babymaking into Hollywood’s push for resentful soullessness. Why don’t Americans wanna breed anymore?

Brain damage during a closing window of critical biological development is abuse. If we let Google groom kids, we’re enabling child abuse. There’s no such thing as a free lunch or free porn. We know addiction operates by escalation, including, ironically, to watching child porn. What about other kids watching it, viewing the rape and molestation of other kids?

Nothing else permanently alters the brain like viewing sexual content. Even Peterson had to point this out.

It seems to be ruining T-levels in men but propping up Viagra sales! Great for the Israeli companies making it.

It’s the ultimate r-selection tool. I have a right not to be exposed by default, same with not tuning into the radio with the BBC blasting crappy rap songs, same with not attending strip clubs and massage brothels, same with public nudity being illegal because the public doesn’t wanna see it and same thing with needing to consent to pay and prove age to buy a cinema ticket to view what’s effectively a gradual push of softcore porn into even PG kid’s films.