Self-reported vaccination for the current season was associated with a trend (P < 0.10) toward higher viral shedding in fine aerosol samples; vaccination with both the current and previous year’s seasonal vaccines, however, was significantly associated with greater fine-aerosol shedding in unadjusted and adjusted models (P < 0.01). In adjusted models, we observed 6.3 (95% CI 1.9–21.5) times more aerosol shedding among cases with vaccination in the current and previous season compared with having no vaccination in those two seasons.”
“The association of current and prior year vaccination with increased shedding of influenza A might lead one to speculate that certain types of prior immunity promote lung inflammation, airway closure, and aerosol generation. This first observation of the phenomenon needs confirmation. If confirmed, this observation, together with recent literature suggesting reduced protection with annual vaccination, would have implications for influenza vaccination recommendations and policies.”
“Prior immunity” promotes lung inflammation… Now I wanted to explain this part so that people wouldn’t be misled. Immunity and vaccination are not synonymous, in fact, this study provides the very evidence to prove that vaccinations like the influenza vaccine don’t provide immunity,
herd immunity is also a myth (search bar it)
hence the vaccinated being infected and spread it more. The correct wording should have read that “prior VACCINATIONS promote lung inflammation” as this fact has been shown in animal studies.
This means that when you receive a flu vaccine, elucidated by this study and animal studies, the lungs are damaged. Hence the scientific term/phrase “vaccine-associated enhanced respiratory disease” which in animal studies
has shown that flu vaccinations damage lung tissue of the vaccinated and distort or weaken the natural immunity of the host, person or animal. Could this be just one more reason more people are developing severe lung diseases like COPD or why the rates of asthma in America are increasing in the vaccinated?
The short and simple: The research shows…
The vaccine doesn’t protect one from infection.
That the vaccinated are “shedding”/spreading more virus simply by breathing.
That prior vaccination has weakened the immune systems of those who got the shot.
Individuals who receive the flu vaccine are placing others around them at greater risk than the unvaccinated.
In this article, we review the clinical management of deliberate infection with several pathogens of greatest bioweapons concern. On the basis of historical incidents coupled with information on ease of dissemination, contagiousness, mortality rates, public health impact, ability to engender panic, and the need for special preparedness,1-3 the Centers for Disease Control and Prevention (CDC) stratifies pathogens and toxins into three risk categories — A, B, and C — with category A meriting the highest level of concern and preparedness.4,5 In this review, we consider diseases that are caused by category A agents for which there are high-quality clinical data in the unclassified literature (see the Supplementary Appendix, available with the full text of this article at NEJM.org). The category A viral hemorrhagic fever viruses are beyond the scope of this review.
Pneumonic plague is caused by infection with the fleaborne bacteriumYersinia pestis. This organism, found worldwide and responsible for the “Black Death,” can cause several forms of illness: bubonic (the most common) (Figure 1D), septicemic, and pneumonic plague.41Because of the focus of this review, only pneumonic plague is discussed.
How fucking fascinating.
CARDINAL FEATURES OF PNEUMONIC PLAGUE
In a deliberate attack, primary pneumonic plague — rather than secondary spread from bubonic or septicemic forms — would occur 1 to 3 days after inhalation of the released bacterium or after droplet transmission from another infected person. The initial presentation of pneumonic plague is nonspecific and is difficult to differentiate from an ordinary pneumonia in its early stages. Hemoptysis, a unique feature, might be present, and rapid progression to respiratory failure and death would occur with greater frequency than in ordinary pneumonias.41
I imagine this would make the death count impossible to distinguish from regular pneumonia.
Remember: “Hemoptysis, a unique feature, might be present…”
“Maintaining that the 2019-nCoV may cause mild to severe respiratory disease, initially clinically presented as fever, dry cough, myalgia (muscle pain), fatigue and gradually progressing to a more severe productive cough that produces phlegm, episodic headaches, hemoptysis (coughing up blood) and occasional diarrhoea.”
What PP is versus what we’re told CV is by the MSM.
“Hemoptysis, a unique feature…”
Why has nobody else done it this way? aka the empirical one
look at signs, look at symptoms
u n i q u e f e a t u r e
I can’t be the only smartass.
But if I must.
DIAGNOSIS OF PNEUMONIC PLAGUE
Because the clinical features of pneumonic plague are nonspecific, diagnosis is largely based on the results of culture. Sputum, blood, or lymph-node aspirates could yield positive culture results. Chest radiography would reveal a severe pneumonic process. Serologic testing can also be useful but would not play much of a role during acute illness.41 Rapid antigen tests are available in regions in which plague is endemic, but none are FDA-approved.
So we’d see tests be useless early on…
and… a shortage of testing kits…
especially for America… who I imagine would call some state of emergency.
TREATMENT AND PREVENTION OF PNEUMONIC PLAGUE
The treatment of pneumonic plague involves a 10-day course of an aminoglycoside antibiotic agent, such as streptomycin or gentamicin. Doxycycline is considered a second-line treatment.41 However, a randomized, controlled trial of potential treatments for bubonic plague revealed equivalency between gentamicin and oral doxycycline; it is unclear whether these results can be extrapolated to pneumonic plague.42 There has been increased interest in the use of fluoroquinolones as primary treatment in mass-casualty settings.42 A 7-day course of doxycycline or ciprofloxacin would be used as postexposure prophylaxis.41No vaccine against plague is available. Because pneumonic plague can be transmitted from person to person through respiratory droplets, droplet precautions must be implemented for all patients.41
Why do the Chicoms want disease-ridden people in hospitals that can supposedly do nothing for them?
Yersinia pestis is the causative agent of plague, a zoonotic disease transmitted to humans through flea bites and typically characterized by the appearance of a tender and swollen lymph node, the bubo. Human-to-human transmission can occur, through either the bite of fleas (bubonic plague) or respiratory droplets, causing an overwhelming infection called pneumonic plague.
Our history books missed out that part.
Suddenly those masks don’t look so stupid.
The last plague pandemic began in Hong Kong in 1894 and spread throughout the world, establishing many endemic foci. Antibiotics and enforcement of public health measures significantly decreased the morbidity and mortality associated with the disease but did not allow its eradication. In fact, plague is now considered a reemerging disease1 ….
We report high-level resistance to multiple antibiotics, including all the drugs recommended for plague prophylaxis and therapy, in a clinical isolate of Y. pestis. The resistance genes were carried by a plasmid that could conjugate to other Y. pestis isolates. This report should serve as a warning of the risk of the spread of resistance in Y. pestis, a species previously considered universally susceptible to antibiotics.
…Strain 17/95 was resistant not only to all the antibiotics recommended for therapy (chloramphenicol, streptomycin, and tetracycline) and prophylaxis (sulfonamides and tetracycline) of plague4 but also to drugs that may represent alternatives to classic therapy, such as ampicillin, kanamycin, spectinomycin, and minocycline. The isolate remained susceptible to cephalosporins, other aminoglycosides, quinolones, and trimethoprim, and treatment with trimethoprim, despite its lack of synergism with sulfonamides, most likely led to the patient’s recovery….
The initial cases of novel coronavirus (2019-nCoV)–infected pneumonia (NCIP) occurred in Wuhan, Hubei Province, China, in December 2019 and January 2020. We analyzed data on the first 425 confirmed cases in Wuhan to determine the epidemiologic characteristics of NCIP.
identified by local hospitals using a surveillance mechanism for “pneumonia of unknown etiology” that was established in the wake of the 2003 severe acute respiratory syndrome (SARS) outbreak with the aim of allowing timely identification of novel pathogens such as 2019-nCoV.4 In recent days, infections have been identified in other Chinese cities and in more than a dozen countries around the world.5 Here, we provide an analysis of data on the first 425 laboratory-confirmed cases in Wuhan to describe the epidemiologic characteristics and transmission dynamics of NCIP.
Furthermore, children might be less likely to become infected or, if infected, may show milder symptoms, and either of these situations would account for underrepresentation in the confirmed case count. Serosurveys after the first wave of the epidemic would clarify this question.
re Pneumonic plague above: “Serologic testing can also be useful but would not play much of a role during acute illness.”
If it looks like a duck, walks like a duck and quacks like a duck – sure, it could be a chicken in a duck suit for kinky reasons but I’m inclined on probability to call it a fucking duck.
delays to hospitalization were much longer, with 89% of patients not being hospitalized until at least day 5 of illness (Figure 2). This indicates the difficulty in identifying and isolating cases at an earlier stage of disease.
re pneumonic plague, above: “The initial presentation of pneumonic plague is nonspecific and is difficult to differentiate from an ordinary pneumonia in its early stages.”
It’s the same hymn sheet. I can’t be the only one seeing this.
Our preliminary estimate of the incubation period distribution provides important evidence to support a 14-day medical observation period or quarantine for exposed persons.
re pneumonic plague, above: “In a deliberate attack, primary pneumonic plague — rather than secondary spread from bubonic or septicemic forms — would occur 1 to 3 days after inhalation of the released bacterium or after droplet transmission from another infected person.”
“The treatment of pneumonic plague involves a 10-day course of an aminoglycoside antibiotic agent, such as streptomycin or gentamicin. ”
“Because pneumonic plague can be transmitted from person to person through respiratory droplets, droplet precautions must be implemented for all patients.41“
That takes about…. 14 days.
Our study suffers from the usual limitations of initial investigations of infections with an emerging novel pathogen, particularly during the earliest phase, when little is known about any aspect of the outbreak and there is a lack of diagnostic reagents.
re pneumonic plague: “The initial presentation of pneumonic plague is nonspecific and is difficult to differentiate from an ordinary pneumonia in its early stages.”
re pneumonic plague: “diagnosis is largely based on the results of culture”
Furthermore, the initial focus of case detection was on patients with pneumonia, but we now understand that some patients can present with gastrointestinal symptoms, and an asymptomatic infection in a child has also been reported.17
“Pneumonic plague: Patients develop fever, headache, weakness, and a rapidly developing pneumonia with shortness of breath, chest pain, cough, and sometimes bloody or watery mucous. Pneumonic plague may develop from inhaling infectious droplets or may develop from untreated bubonic or septicemic plague after the bacteria spread to the lungs. The pneumonia may cause respiratory failure and shock. Pneumonic plague is the most serious form of the disease and is the only form of plague that can be spread from person to person (by infectious droplets).”
“Pneumonic plague affects the lungs. It’s the least common variety of plague but the most dangerous, because it can be spread from person to person via cough droplets. Signs and symptoms can begin within a few hours after infection, and may include:
Cough, with bloody mucus (sputum)
Nausea and vomiting
Pneumonic plague progresses rapidly and may cause respiratory failure and shock within two days of infection. Pneumonic plague needs to be treated with antibiotics within a day after signs and symptoms first appear, or the infection is likely to be fatal.”
repeating coronavirus paper:
Although delays between the onset of illness and seeking medical attention were generally short, with 27% of patients seeking attention within 2 days after onset.
Back to CV paper generally
Early infections with atypical presentations may have been missed, and it is likely that infections of mild clinical severity have been under-ascertained among the confirmed cases.18 We did not have detailed information on disease severity for inclusion in this analysis.
In conclusion, we found that cases of NCIP have been doubling in size approximately every 7.4 days in Wuhan at this stage. Human-to-human transmission among close contacts has occurred since the middle of December and spread out gradually within a month after that. Urgent next steps include identifying the most effective control measures to reduce transmission in the community. The working case definitions may need to be refined as more is learned about the epidemiologic characteristics and outbreak dynamics. The characteristics of cases should continue to be monitored to identify any changes in epidemiology — for example, increases in infections among persons in younger age groups or health care workers. Future studies could include forecasts of the epidemic dynamics and special studies of person-to-person transmission in households or other locations, and serosurveys to determine the incidence of the subclinical infections would be valuable.14
re Pneumonic plague above: “Serologic testing can also be useful but would not play much of a role during acute illness.”
These initial inferences have been made on a “line list” that includes detailed individual information on each confirmed case, but there may soon be too many cases to sustain this approach to surveillance, and other approaches may be required.19
Study allowed by Chinese Government, who arrested 8 doctors for trying to release some piece of information, can’t imagine what.
If they’d been told to give people antibiotics for a virus though, I imagine they had some pointed questions.
Especially if the Chicoms wanted to buy time to buy up global supply and produce more.
“Pneumonic plague is a severe lung infection caused by the bacterium Yersinia pestis. Symptoms include fever, headache, shortness of breath, chest pain, and cough. They typically start about three to seven days after exposure.”
Long lag time.
PP can cause meningitis, which might explain the headache.
from CV article above: “”Common coronavirus symptoms can include: — Fever — Dry cough — Shortness of breath — Aching muscles — Fatigue”
“For confirmed 2019-nCoV infections, reported illnesses have ranged from people with little to no symptoms to people being severely ill and dying. Symptoms can include:
Shortness of breath”
All three just so happen to be also the symptoms of pneumonic plague.
“CDC believes at this time that symptoms of 2019-nCoV may appear in as few as 2 days or as long as 14 after exposure”
So three, three days on the low end. How familiar.
About dat headache symptom…
Coronaviruses as Encephalitis
-Inducing Infectious Agents
so why is headache not described as a symptom?
“In acute encephalitis, viral replication occurs in the brain tissue itself, possibly causing destructive lesions of the gray matter, as was described after herpes simplex virus (HSV), rabies, or some arbovirus infections. ”
Yes, herpes can reach the brain. Again, I must remind you. Yes, it can.
This concludes why I’m banned from appearing on tv (pretty much).
A comparison between eight individual samples demonstrated that the Asian male one has an extremely large number of ACE2-expressing cells in the lung. This study provides a biological background for the epidemic investigation of the 2019-nCov infection disease, and could be informative for future anti-ACE2 therapeutic strategy development.
We may dub it The Elliot gene.
…..We also noticed that the only Asian donor (male) has a much higher ACE2-expressing cell ratio than white and African American donors (2.50% vs. 0.47% of all cells). This might explain the observation that the new Coronavirus pandemic and previous SARS-Cov pandemic are concentrated in the Asian area….
Almost like the Chinese wanted to wipe out most of their own race IF there were a war.
Scientists must question everything and especially what they love the most, i.e. their own discoveries and ideas. This basic rule of scientific research helps
avoid erroneous developments and reveals the ones
that already exist. Also, we must all be allowed to question the status quo, otherwise we would live in a dictatorship.
but muh muh scientism! – redditfags
Moreover, science cannot be limited to a
selected number of institutions and experts. Science can and must be conducted by anyone who has the necessary knowledge and the appropriate methods.
Science can be considered science only if its claims are verifiable, reproducible and if they allow predictions. Science also needs external control, because, as we will see, a part of the medical sciences has lost touch with reality for quite some time.
They believe in invisible leprechaun atoms floating in nothing, popping out of existence. Also tiny strings. They can’t tell you what a field is. They’re mad, mad as hatters. But they make up equation models that can’t be verified and their real world studies make no sense. The world does make sense, they’re just wrong. It’s human error.
Square peg, they are wrong.
Anyone who has knowledge of biology and the genesis of life, of the development and functions of the tissue, of the body and of the brain, will automatically question the assumptions about viruses.
In the reality of the body and of its mechanisms, there is no place for hypothetical malignant processes.
You must be possessed. Tiny demons have besieged your body.
All biological processes, including those that can end in suffering, pain and death, are originally meant to be useful. A different approach to the virus phenomenon is possible and necessary: any layman with some background knowledge reading scientific papers about pathogenic viruses can realize that such viruses do not exist and what is being described are only typical components and characteristics of cells. This background knowledge will be provided in this article.
Remember, ebola is a virus. And they happened to have a vaccine, ready to go!
What ARE the odds?
It’s incredible, if not impossible.
Forget the delay of at least two years before you’re set for human trials, why can’t they do that process with every damn thing?
Biotech should be criminally accountable for with-holding cures, that’s all I’ll say. Currently, they are not.
They can also use corporate espionage and political contacts to with-hold funding from small competition who want a cure.
Inc. gems like:
The search for these pathogenic poisons remains to date fruitless, however, when bacteria were discovered, it was assumed that they were producing the pathogenic poisons. This supposition, called “the germ theory”, was immediately accepted and remains very successful up to the present time.
This theory is so successful that the majority of the people are still not aware of the fact that the so-called bacterial toxins are actually normal enzymes, which either cannot appear in a human
being, or, if they do, they never appear in such an amount as to make them dangerous.
Before it could be established that the “bacterial viruses” cannot kill natural bacteria, but they are instead helping them to live and that bacteria themselves emerge from such structures, these
“phages” were already used as models for the alleged human and animal viruses. It was assumed
that the human and animal viruses looked like the “phages”, were allegedly killing cells and thereby
causing diseases, while at the same time producing new disease poisons and in this way transmitting
the diseases. To date, many new or apparently new diseases have been attributed to viruses if their origin is unknown or not acknowledged.
cough military cough
This reflex found an apparent confirmation in the discovery of the “bacterial viruses.
Don’t take the vaccine, unless you voted Hillary in which case go ahead sweetie. Take two.
All the recent MSM articles and hints in the Guardian about a virus.
This is the plan.
Plain sight. Outbreak of NWO.
…Don’t drink the fucking water.
Why tax cars into oblivion, forcing everyone on the Tube?
Why build houses so close together they catch fire? How far can a virus travel? Some buildings in London you cannot develop until they are destroyed, they are worth more burnt to a crisp. That was the rationale for WW2 too. You buy it cheap with no planning permission and then it skyrockets in value once the insurance must be unfortunately claimed.
GMO labelling. Viruses affect livestock, a fine excuse to ban meat and get us all on slave diets, eh, Soros?
Viruses can even be dropped into very white houses. They run tours.
Anonymous was an …interesting inversion.
A mirror held up to a mirror. Shows how hollow and empty the evil is, philosophically.
Satan creates nothing, he imitates, copies and inverts. Ape of Thoth. The best thing is use their tactics against them, as their own books advise. [Rules for Radicals] Because the only people trying to control human agency, are the Satanists gaslighting you it’s somebody else. Never them.
A digital rebellion long before Gamergate or Brexit…. People over self-styled “Elite” PC NWO.
Back when I was in school, actually. Computers were rarer, good ones.
For inciting rebellions, Satan sure doesn’t like it up ‘im.
The web of control (spider) becomes free information and cooperation from people who reject these would-be rulers and associated despots.
A great way to beat “cancel culture”… I am Spartacus.
If only Anonymous had gone quiet to hack various celebrity’s twitter feeds.
Actually, how many of them DON’T have it at this point?
Among other inversions, tarnishing a Christian’s name to harm (sorry, “heal”) the world.
We believe there is scientific and epidemiologic evidence that Ebola virus has the potential to be transmitted via infectious aerosol particles both near and at a distance from infected patients, which means that healthcare workers should be wearing respirators, not facemasks.1
There has been a lot of on-line and published controversy about whether Ebola virus can be transmitted via aerosols. Most scientific and medical personnel, along with public health organizations, have been unequivocal in their statements that Ebola can be transmitted only by direct contact with virus-laden fluids2,3 and that the only modes of transmission we should be concerned with are those termed “droplet” and “contact.”
These statements are based on two lines of reasoning. The first is that no one located at a distance from an infected individual has contracted the disease, or the converse, every person infected has had (or must have had) “direct” contact with the body fluids of an infected person. [DS: “must have”, comforting]
This reflects an incorrect and outmoded understanding of infectious aerosols, which has been institutionalized in policies, language, culture, and approaches to infection control. We will address this below. Briefly, however, the important points are that virus-laden bodily fluids may be aerosolized and inhaled while a person is in proximity [CDC paper] to an infectious person and that a wide range of particle sizes can be inhaled and deposited throughout the respiratory tract.
The second line of reasoning is that respirators or other control measures for infectious aerosols cannot be recommended in developing countries because the resources, time, and/or understanding for such measures are lacking.4
…Medical and infection control professionals have relied for years on a paradigm for aerosol transmission of infectious diseases based on very outmoded research and an overly simplistic interpretation of the data….
Early aerobiologists were not able to measure small particles near an infectious person and thus assumed such particles existed only far from the source. They concluded that organisms capable of aerosol transmission (termed “airborne”) can only do so at around 3 feet or more from the source. [DS: touchable surfaces do not exist, apparently] Because they thought that only larger particles would be present near the source, they believed people would be exposed only via large “droplets” on their face, eyes, or nose.
Modern research, using more sensitive instruments and analytic methods, has shown that aerosols emitted from the respiratory tract contain a wide distribution of particle sizes—including many that are small enough to be inhaled.5,6 Thus, both small and large particles will be present near an infectious person.
As noted by early aerobiologists, liquid in a spray aerosol, such as that generated during coughing or sneezing, will quickly evaporate,7 which increases the concentration of small particles in the aerosol. Because evaporation occurs in milliseconds, many of these particles are likely to be found near the infectious person.
The current paradigm also assumes that only “small” particles (less than 5 micrometers [mcm]) can be inhaled and deposited in the respiratory tract. This is not true. Particles as large as 100 mcm (and perhaps even larger) can be inhaled into the mouth and nose. Larger particles are deposited in the nasal passages, pharynx, and upper regions of the lungs, while smaller particles are more likely to deposit in the lower, alveolar regions. And for many pathogens, infection is possible regardless of the particle size or deposition site.
It’s time to abandon the old paradigm of three mutually exclusive transmission routes for a new one that considers the full range of particle sizes both near and far from a source. In addition, we need to factor in other important features of infectivity, such as the ability of a pathogen to remain viable in air at room temperature and humidity and the likelihood that systemic disease can result from deposition of infectious particles in the respiratory system or their transfer to the gastrointestinal tract.
We recommend using “aerosol transmissible” rather than the outmoded terms “droplet” or “airborne” to describe pathogens that can transmit disease via infectious particles suspended in air.
…Being at first skeptical that Ebola virus could be an aerosol-transmissible disease, we are now persuaded by a review of experimental and epidemiologic data that this might be an important feature of disease transmission, particularly in healthcare settings.
Some pathogens are limited in the cell type and location they infect. …
HIV infects T-helper cells in the lymphoid tissues and is primarily a bloodborne pathogen with low probability for transmission via aerosols. [Throwaway Q: What’s to stop it hooking up with HIV or some other virus?]
Ebola virus, on the other hand, is a broader-acting and more non-specific pathogen that can impede the proper functioning of macrophages and dendritic cells—immune response cells located throughout the epithelium.15,16Epithelial tissues are found throughout the body, including in the respiratory tract.
…Many body fluids, such as vomit, diarrhea, blood, and saliva, are capable of creating inhalable aerosol particles in the immediate vicinity of an infected person. (e.g.)…The act of vomiting produces an aerosol and has been implicated in airborne transmission of gastrointestinal viruses. Regarding diarrhea, even when contained by toilets, toilet flushing emits a pathogen-laden aerosol that disperses in the air.
…..These rates indicate that 99% loss in aerosol infectivity would occur in 93, 104, and 162 minutes, respectively. [DS: I feel comforted, do you feel comforted?]
…In still air, 3-mcm particles can take up to an hour to settle. With air currents, these and smaller particles can be transported considerable distances before they are deposited on a surface…. There is also some experimental evidence that Ebola and other filoviruses can be transmitted by the aerosol route.
Zaire Ebola viruses have also been transmitted in the absence of direct contact among pigs25 and from pigs to non-human primates,26 which experienced lung involvement [sweet term] in infection. Persons with no known direct contact with Ebola virus disease patients or their bodily fluids have become infected.12
[sum: Direct transmission is direct]…However, the respiratory and gastrointestinal systems are not complete barriers to Ebola virus. Experimental studies have demonstrated that it is possible to infect non-human primates and other mammals with filovirus aerosols. …Altogether, these epidemiologic and experimental data offer enough evidence to suggest that Ebola and other filoviruses may be opportunistic with respect to aerosol transmission.28 That is, other routes of entry may be more important and probable, but, given the right conditions, it is possible that transmission could also occur via aerosols.
As for public protection:
Facemasks, however, do not offer protection against inhalation of small infectious aerosols, because they lack adequate filters and do not fit tightly against the face.1 Therefore, a higher level of protection is necessary.
Why not disinfect, you ask?
For a risk group 4 organism, any activity that has the potential for aerosolizing liquid body fluids, such as medical or disinfection procedures, should be avoided, if possible. Our risk assessment indicates that a PAPR with a full facepiece (APF = 50) or a hood or helmet (APF = 25) would be a better choice for patient care during epidemic conditions.
They’re beginning to treat it like a risk group 4 (the highest).
Wearing this type of respirator minimizes the need for other types of PPE, such as head coverings and goggles.
We’ve been telling you for awhile now that the government and healthcare providers were not being honest about how Ebola can spread. Over and over again, government officials and healthcare experts have insisted Ebola “can only be spread through direct contact.” Those same people have also insisted that infected people “are not contagious until they show symptoms. CDC now admits those claims were FALSE!
Why is it always from Africa?
All the deadliest diseases in the entire world, always them.
Casual contact is defined as a) being within approximately 3 feet (1 meter) or within the room or care area for a prolonged period of time (e.g., healthcare personnel, household members) while not wearing recommended personal protective equipment (i.e., droplet and contact precautions–see Infection Prevention and Control Recommendations); or b) having direct brief contact (e.g., shaking hands) with an EVD case while not wearing recommended personal protective equipment (i.e., droplet and contact precautions–see Infection Prevention and Control Recommendations). At this time, brief interactions, such as walking by a person or moving through a hospital, do not constitute casual contact.
In my academic experience, when it comes to liars, the good stuff is always in the footnotes.
In fact, if I sense someone is lying to me, it’s the first place I go. Man, do they sweat when they see me flip those pages.
We don’t know why West Africa is currently suffering from the largest outbreak of Ebola, but humans were almost certainly first infected through contact with “bush meat”.
Food critic Charles Campion, who has investigated the sale of bush meat in London markets, says that African immigrants buy the black-market meats for a taste of home.
Ebola is a Class A bioterror weapon.
These people are so stupid they know this and keeping eating it.
But carrying bush meat into Britain is illegal for health and safety reasons and to protect animal welfare. There’s no oversight into cleanliness of the meat, and the methods of transportation are often unsafe.
Well, at least they’re not keeping it around other people…
A 2010 investigation into bush meat in Europe found270 tonnescoming through Paris’s Charles de Gaulle airport alone. Researchers discovered 11 different types of bush meat from African forests, including whole sheep and calves that were wrapped in plastic and kept in holdalls during flights.
Once the bush meat arrives in Britain, street markets and Africa restaurants are thought to stock the black market goods, keeping them hidden under the counter for familiar customers. Six butchers and food stores in Ridley Road Market, Dalston, were discovered selling illicit rat meat by a BBCinvestigation in 2010, and Campion says he knows of Hendon restaurants that sell bush meat.